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1 n the brains of SAMP8 and SAMR1 mice than in C57BL mice.
2 ed 99Tc-G extravasation in the dura mater of C57BL mice.
5 killing by AMs may explain the resistance of C57BL mice and the susceptibility of C3H mice to mycopla
6 ly decreased in both Swiss-Webster (S-W) and C57bl mice (approx. cell loss of 40% and 45%, respective
7 ere that B10.BR mice, which are derived from C57BL mice but have the same MHC locus (H-2(k)) as susce
9 AM depletion exacerbated the infection in C57BL mice by reducing killing of the organism to a leve
11 sent studies the immune response in BALB and C57BL mice during the acute phase of E-55+ murine leukem
13 oE synthesis were also observed in castrated C57BL mice given either physiological or pharmacological
14 The results showed that visceral AT from old C57BL mice had significantly higher mRNA expression of t
15 transgenic and GFP-CETN2-expressing CB6F1 or C57BL mice identify polynuclear primary spermatocytes wi
16 inal-RPE separation was created in wild-type C57BL mice, IL-6(-/-) mice, and Brown Norway rats by sub
17 especially in aging control and 22A-infected C57BL mice, predominantly in the stratum radiatum of the
18 Under normoxic conditions, macrophages from C57BL mice produce low levels of vascular endothelial gr
20 R mutant of Ec1a was similarly attenuated in C57BL mice, regardless of the p47(phox) genotype, as in
23 -10 and IL-4 adversely affect the ability of C57BL mice to resist infection with C. immitis, but IL-1
26 than CBA/J mice, wild-type and p47(phox-/-) C57BL mice were similarly susceptible, and the oxyR muta
28 C57BL/6J-scid/scid (C57-SCID), and C57BL/6N (C57BL) mice with Mycoplasma pulmonis and at 14 and 21 da
29 J mice had higher peak parasitemias than did C57BL mice, with mortality rates of 40, 50, and 50%, res