ライフサイエンスコーパス: C6

1 on occurs with retention of configuration at C6'.

2 mic-like parallel factor analysis component (C6).

3 with knockout (KO) of complement protein 6 (C6).

4 y a modest effect on the replication of HAdV-C6.

5 en a dimethylallyl moiety and the FMN N5 and C6.

6 sts, but not with single-antigen tests using C6.

7 %, C1: 26%, C2: 19%, C3: 8%, C4: 4%, C5: 1%, C6: 0.42%.

8 and perfluoroalkyl carboxylic acids (PFCAs; C6-15) along with six perfluoro-octane sulfonic acid (PF

9 ing significant amounts of C4 (35.8% yield), C6 (28.2% yield), and C8 (9.4% yield) alcohols.

10 The cDNA library was transfected into C6/36 (Aedes) and Vero (primate) cells, with subsequent

11 d 3 pools yielded YFV following culture in a C6/36 cell line.

12 d in naive A. albopictus (C6/36) and wMelPop-C6/36 cells by RNAseq.

13 The viral RNA evolved from wMelPop-C6/36 cells contained low-frequency mutations (~25%) wit

14 syncytium formation, DENV2-infected wMelPop-C6/36 cells did not.

15 Moreover, while DENV2-infected naive C6/36 cells showed syncytium formation, DENV2-infected w

16 ither naive A. albopictus (C6/36) or wMelPop-C6/36 cells were infected with DENV serotype 2 (DENV2).

17 V2 can readily infect and replicate in naive C6/36 cells, whereas in mutant DENV2-infected BHK-21 or

18 restored siRNA production in Ae. albopictus C6/36 cells, which are dicer-2 defective.

19 on profiles and morphology of DENV2-infected C6/36 cells.

20 uences were analyzed in naive A. albopictus (C6/36) and wMelPop-C6/36 cells by RNAseq.

21 this mechanism, either naive A. albopictus (C6/36) or wMelPop-C6/36 cells were infected with DENV se

22 e collected in 2012; 13 PFASs (C6-C12 PFCAs; C6, 8, 10 PFSAs; MeFOSAA and EtFOSAA; and FOSA) were rou

23 The approximately 9-14% yearly decline of C6-8 perfluoroalkyl sulfonates (PFSAs) following the cea

24 yl sulfonates (PFSAs) following the cease in C6-8 PFSA precursor production in 2001 indicates that th

25 dual dipolar couplings (DeltaRDCs) involving C6/8-H6/8, C3'-H3' and C4'-H4' vectors are correlated to

26 Knockout of C6, a component of the membrane attack complex, prevente

27 dation during VACV infection by VACV protein C6, a multifunctional IFN antagonist that coprecipitates

28 the ABCC6 (ATP binding cassette transporter C6) ABC transporter are associated with pseudoxanthoma e

29 (t.Bu)ArO)(3)tacn)U(III)((Me)cy-C6)].((Me)cy-C6) adduct.

30 Continuous sampling of C4-AHL, C6-AHL, C8-AHL, C6-OXO-AHL, and C12-OXO-AHL was achieved

31 C6 alcohols also influenced the vegetal notes of BRS Vio

32 A total of 22 free VOCs were identified with C6 alcohols and esters being the major compounds.

33 6 aldehydes that seem to be more stable than C6 alcohols and esters.

34 one leaf removal had no consistent impact on C6 alcohols, volatile phenols, lactones, fermentation-de

35 increase in particular important phenols and C6 alcohols, while C6 aldehydes mostly decreased.

36 In addition, C6-alcohols accumulated before veraison and decreased to

37 lar important phenols and C6 alcohols, while C6 aldehydes mostly decreased.

38 ted by the genetic origin of the olives with C6 aldehydes that seem to be more stable than C6 alcohol

39 dified water contained low concentrations of C6 aldehydes, isothiocyanates, nitriles, and sulfides, a

40 products of the lipoxygenase pathway (mainly C6-aldehydes) and of glucosinolate hydrolysis (mainly is

41 n separation of a 11-component mixture of C1-C6 alkanes, the hierarchical phase outperforms the struc

42 photochromic hydrazone functionalized with a C6 alkyl thiolate spacer (C6 HAT) was characterized on a

43 GEOV1, MERIS, and MODIS C6 also exhibited an increasing trend, but to a much sma

44 Mice deficient for complement component 6 (C6), an essential component for TCC assembly, and mice w

45 nsporter genes in tomato cv. Moneymaker, ABC-C6 and ABC-G33, alters the composition of semi-volatile

46 C6 and C4 were also downregulated for cellular and mitoc

47 Relative to the dosing solution, C6 and C7 perfluoroalkyl sulfonates (PFSAs) were enriche

48 rparts 1-4 were proven to self-assemble into C6 and C7 pseudocycles by intramolecular H-bonds in solu

49 s the attachment of two sulfhydryl groups to C6 and C8 of a pendant octanoyl chain.

50 thway, the attachment of two sulfur atoms at C6 and C8 of an n-octanoyllysyl chain, is catalyzed by l

51 rtion of two sulfur atoms at the unactivated C6 and C8 positions of a protein-bound octanoyl chain to

52 The expressed DbpA/C6 and OspC proteins were useful in detecting anti-Borre

53 ting the more readily accessible hydroxyl at C6 and the more reactive anomeric hydroxyl.

54 e invariable region 6 of the vlsE gene (DbpA/C6) and (b) the full-length ospC gene (OspC).

55 Rodent (C6) and human (U251) glioma cell lines, and non-tumor hu

56 cis stereochemistry at the ring junction (C5-C6) and trans at the site of nucleophilic attack (C6-C7)

57 ing pathogenic variants clustered in the C3, C6, and C10 domains (18 of 22, 82%, P<0.001 versus Genom

58 MyBP-C with variants in the C3, C6, and C10 domains was expressed in rat ventricular myo

59 By contrast, anti-C6 antibodies were detected in pretreatment sera from 17

60 B. miyamotoi antibodies can cross-react with C6 antigen testing for B. burgdorferi, the causative age

61 ophen (N-acetyl-p-aminophenol, APAP) and (13)C6-APAP were incubated with rat liver microsomes, which

62 This included complement components C3, C5, C6, apolipoproteins A-I, A-IV, C-I and M, histidine-rich

63 Short-chain FA-VOCs (C5 and C6) are among the most abundant and important volatile c

64 An efficient, direct C6-arylation of 2-pyridones has been successfully accomp

65 esent hydroxytyrosol synthetic derivative HT C6 as a new antiangiogenic compound and as a good candid

66 to diversify the stereochemical outcomes at C6 as well as C10a.

67 +1) ( n = 1-4, HA = CH(3)(CH(2))(5)NH(3)(+), C6) as the organic spacer molecules between the inorgani

68 chain lengths, with perfluorohexanoic acid (C6) as the predominant one.

69 ed to assess METH toxicity in differentiated C6 astroglia-like cells through biochemical and toxicity

70 d KMO, but not IDO, in vitro in BDV-infected C6 astroglioma cells.

71 ensity-based functionals, (ii) semiclassical C6-based, and (iii) one-electron effective potentials.

72 1), whereas the corresponding process in the C6-benzyloxypurine is 56 times slower.

73 ceptor at C2 through the formation of the C1-C6 bond.

74 Specifically, selective C3 and C6 bromination has been achieved as well as C8 borylatio

75 uent Friedel-Crafts alkylation of the flavin C6, but can be rescued by addition of (pyro)phosphate.

76 ing closures and clade II enzymes catalyzing C6-C1 closures.

77 N5 mechanism in which 5 binds to S2 with its C6-C11 group poised to attack C1 in GPP to form the moen

78 er samples were collected in 2012; 13 PFASs (C6-C12 PFCAs; C6, 8, 10 PFSAs; MeFOSAA and EtFOSAA; and

79 separation of 50 analytes, including alkane (C6-C12), alkene, alcohol, aldehyde, ketone, cycloalkane,

80 pyro)phosphate leaving group is required for C6-C3' bond formation, resembling pyrophosphate initiate

81 C8/C7 and C6/C5 polyfluorinated sulfonates from br-PFOS and br-PFH

82 n of C6, C8, and C10 monoalkylated PFPAs and C6/C6, C6/C8, and C8/C8 dialkylated PFPiAs was investiga

83 assembly of soluble complement proteins C5b, C6, C7, C8 and C9, but little is known about the rate-li

84 (C4-C5-C6 in aldoheptoses and higher and C5-C6-C7 in sialic and ulosonic acids) to that of the simpl

85 nd trans at the site of nucleophilic attack (C6-C7) on the three contiguous stereogenic centers in go

86 hydroxytyrosyl alkyl ethers (HT C1, C2, C4, C6, C8 and C12).

87 The sorption of C6, C8, and C10 monoalkylated PFPAs and C6/C6, C6/C8, an

88 increasing chain length for C8-C14 PFCAs and C6-C8 perfluoroalkyl sulfonates.

89 ned during the last two days of the culture (C6-C8) resulted in a significant decrease in PF formatio

90 , C8, and C10 monoalkylated PFPAs and C6/C6, C6/C8, and C8/C8 dialkylated PFPiAs was investigated in

91 reactions (J-K), for the formation of the C5-C6, C9-C10, and C17-C18 double bonds, a Suzuki-Molander

92 membrane lipid order disruption byd-ceramide-C6 causes Golgi pH alkalization.

93 The C6-CD(3)O redirected metabolism away from a problematic

94 The C7-fluoro, C6-CD(3)O substitution pattern of the P2* isoquinoline h

95 The viability of C6 cells remained unchanged, indicating that these surfa

96 rigenic process and increased the ability of C6 cells to form colonies in soft agar or spheres when g

97 C6 ceramide increases IQGAP1 protein levels by preventin

98 We report here that C6 ceramide increases serum-stimulated ERK1/2 activation

99 osmotic shock, bacterial sphingomyelinase or C6 ceramide.

100 igenic lipid, and treatment with short-chain C6-ceramide decreased the number of ovarian cancer cells

101 ed that long-chain ceramide regenerated from C6-ceramide through the salvage/recycling pathway, at le

102 nanoparticle-based delivery, fluorescent NBD C6-ceramide was efficiently converted to NBD C6-glucosyl

103 ay, at least in part, mediated the action of C6-ceramide.

104 Azido-functionalized C6-ceramides were incorporated into and localized within

105 ome P (CYP) 450 inhibition observed with the C6-CH(3)O prototype.

106 C6 compounds such as 1-hexanol, (E)-2-hexenal, (E)-2-hex

107 ds studied, benzenoid compounds, esters, and C6 compounds, were found in greater quantity in the samp

108 the highest acetaldehyde, ethyl acetate and C6-compounds levels, and had increased ester levels in r

109 DBBM grafts containing the C3 and C6 conjugated drugs showed significantly more bone forma

110 The C6 containing DBBM demonstrated the highest percentage o

111 (1) animals maintained as separate C6 WT and C6-D homozygous colonies; and (2) establishment of a het

112 a heterozygous colony to generate C6 WT and C6-D littermate controls.

113 NMR studies of dimerization in C6 D6 find aromaticity-modulated H-bonding (AMHB) energy

114 as the identification of RNA modification by C6 deamination of adenosine (A) to produce inosine (I) i

115 nies is inadequate for testing the effect of C6 deficiency on locomotor and histological recovery aft

116 We compare the effect of C6 deficiency on recovery of locomotor function and hist

117 Under homeostatic conditions, C6-deficient mice displayed a reduced bone mass, mainly

118 was eventually obtained as a 1:1 mixture of C6 diastereomers, which were readily separated by chroma

119 ed, C2-substituted, and C5-substituted or C5,C6-disubstituted NBEs, as well as their synthetic applic

120 We then compared the performance of the C6 EIA alone and as a first-tier test followed by immuno

121 The C6 EIA alone had sensitivity similar to that of standard

122 undergoing evaluation for Lyme disease, the C6 EIA could guide initial clinical decision making, alt

123 However, the C6 EIA has not been extensively studied in pediatric pat

124 We performed a C6 EIA on all collected specimens, followed by a supplem

125 followed by a supplemental immunoblot if the C6 EIA result was positive but the whole-cell sonicate E

126 l immunoblot improved the specificity of the C6 EIA to 98.6%.

127 tal immunoblot) to MTTT (WCS EIA followed by C6 EIA) using McNemar's test to evaluate for agreement b

128 thm, where immunoblotting is replaced by the C6 EIA, performs as well or better than STTT.

129 with both a whole-cell sonicate (WCS) and a C6 EIA, with a supplemental immunoblot if either EIA was

130 T) approach where the second-tier test was a C6 EIA.

131 equence, expressed (VlsE) CLIA followed by a C6 EIA.

132 whole-cell sonicate (WCS) EIA followed by a C6 EIA; (2) a WCS EIA followed by a VlsE chemiluminescen

133 be induced by remote participation of C4 or C6 ester groups.

134 A biogenetically inspired dehydrative C6-etherification reaction proved highly effective to se

135 Cys residues (four disulfide bonds), whereas C6 evasins have only three of these disulfide bonds.

136 these assays, hydroxytyrosyl hexyl ether (HT C6) exhibited the most potent inhibitory activity in vit

137 isplays an additional, stabilizing Cu-Cipso (C6 F5 ) interaction.

138 is(alkyl) hydridoborato Ce{C(SiHMe2 )3 }2 HB(C6 F5 )3 (2).

139 ers enable C-C bond formation as catalytic B(C6 F5 )3 can be used to effect formal 1,5-alkyl migratio

140 ation of alkyne-containing substrates with B(C6 F5 )3 enabled the first catalytic intramolecular cycl

141 l-known frustrated Lewis pair (FLP) PtBu3 /B(C6 F5 )3 in varying stoichiometries.

142 reaction with the hard Lewis acidic borane B(C6 F5 )3 initiates a cascade reaction to yield a complex

143 complex Ce{C(SiHMe2 )3 }3 (1) reacts with B(C6 F5 )3 to produce the zwitterionic bis(alkyl) hydridob

144 Magnetic measurements reveal that [2Dy ][B(C6 F5 )4 ] is an SMM with a record anisotropy barrier up

145 (2Dy ) as a salt of the non-coordinating [B(C6 F5 )4 ](-) anion.

146 15), divided in 3 models: brain tumors (9 L, C6, F98), permanent stroke, and a control group of healt

147 MR approaches in healthy tissues and in the C6, F98, and permanent stroke models.

148 , are demonstrated to undergo protonation at C6 followed by regioselective amination at C5 with a var

149 products (GEOV1, MERIS, MODIS C5, and MODIS C6) from 2003 through 2011 to help clarify the performan

150 erted a dipeptide precursor into the desired C6-functionalized azabicycloalkane scaffolds.

151 +) exchanger NHE5 is abundantly expressed in C6 glioma cells and plays an important part in regulatin

152 able differences in channel activity between C6 glioma cells and tsA201 cells expressing L166Q and A1

153 and the formation of cell-cell junctions by C6 glioma cells seeded on top of electrodes.

154 C6 glioma cells were treated with 10 uM ketamine for 15

155 acellular Ca(2+) concentration in 1321N1 and C6 glioma cells without altering TRAP-6 and carbachol Ca

156 reduced proliferation and increased death of C6 glioma cells, effects that can be partially rescued b

157 In rat C6 glioma cells, leptin pre-treatment enhanced Ca(2+) mo

158 ronic antidepressant treatment of rats or of C6 glioma cells, tracks with the delayed onset of therap

159 o activity in a highly aggressive orthotopic C6 glioma model demonstrated a greater than 25% long-ter

160 April and July 2012, 32 rats implanted with C6 glioma received two intravenous injections at a 24-ho

161 ormed in athymic mice bearing NIS-expressing C6-glioma subcutaneous xenografted tumors to determine t

162 was observed in human NIS (hNIS)-expressing C6-glioma xenografts as well as expected NIS-mediated up

163 , oral glucose absorption (RaO) with [U-(13) C6 ]-glucose, and hepatic glucose production (EGP) with

164 ns in mouse tissues during 12 to 48 h of (13)C6-glucose feeding.

165 e, we report noninvasive introduction of (13)C6-glucose via a stress-free, ad libitum liquid diet.

166 , 2) 10% D-[(13)C6]glucose, and 3) 1% D-[(13)C6]glucose co-provisioned with 10% sucrose.

167 es for 7 days: 1) 10% sucrose, 2) 10% D-[(13)C6]glucose, and 3) 1% D-[(13)C6]glucose co-provisioned w

168 C6-ceramide was efficiently converted to NBD C6-glucosylceramide in live cells or in mouse tissues, w

169 enabled detection and quantification of NBD C6-glucosylceramide in the low-femtomolar range.

170 labeled metabolites, namely, (14)C- and (13)C6-glucuronides and sulfates.

171 The lack of a C6 H-bond donor while maintaining A3AR affinity and effi

172 The base and sugar (H6,C6, H1',C1') chemical shifts of C43 for the dominant con

173 dipolar zwitterionic p-benzoquinonemonoimine C6 H2 (-cdots, three dots, centered NH2 )2 (-cdots, thre

174 2 ; R=1,12-xanthendiyl spacer; Mes=2,4,6-Me3 C6 H2 ), acting as a frustrated Lewis pair (FLP) in smal

175 diazametallocyclobutene 8 (Ar=4-Ph-2,6-iPr2 C6 H2 ).

176 (kappa(2) -N,O-AZADO) ((Ar) L=1,9-(2,4,6-Ph3 C6 H2 )2 -5-mesityldipyrromethene).

177 f saturated NHC SIMes (SIMes=[:C{N(2,4,6-Me3 C6 H2 )CH2 }2 ]) with Group 1 alkyl bases suggest this d

178 Addition of N3 Ar(X3) (Ar(X3) =2,4,6-X3 C6 H2 ; X=Cl or Me) to [(i) Pr2 NN]Cu(NCMe) results in t

179 reated with NaOH/Na(+) BArf (-) (BArf =B(3,5-C6 H3 (CF3 )2 )4 ) to give lipophilic Lambda- and Delta-

180 he dicopper(II) diketimide 4 (Ar=2,6-(i) Pr2 C6 H3 ) or undergo nitrile insertion to give diazametall

181 ] (cAACMe =:C(CMe2 )2 (CH2 )NAr, Ar=2,6-iPr2 C6 H3 ) with H2 SiI2 in a 3:1 molar ratio in DME afforde

182 here PO(Bp,OMe) =(2-MeOC6 H4 )(2-{2,6-(MeO)2 C6 H3 }C6 H4 )(2-SO3 -5-MeC6 H3 )P, inserts vinyl fluori

183 lSi: ((TMS) L=N(SiMe3 )Dipp; Dipp=2,6-Pr(i)2 C6 H4 ) and two NHC ligands (N-heterocyclic carbene=:C[(

184 (Bp,OMe) =(2-MeOC6 H4 )(2-{2,6-(MeO)2 C6 H3 }C6 H4 )(2-SO3 -5-MeC6 H3 )P, inserts vinyl fluoride (VF)

185 Me3 )ClSi:-->Ni(NHC)2 ] (1; Dipp=2,6-(i) Pr2 C6 H4 ; N-heterocyclic carbene=C[((i) Pr)NC(Me)]2 ) with

186 e CAr -H bond activation product [1-F-2-IMe -C6 H4 ](+) I(-) (3).

187 taining the redox-active ligand [{2-(tBuN(O))C6 H4 CH2 }3 N](3-) .

188 and lactam functionalities using IrCl(CO)[P(C6 H5 )3 ]2 (Vaska's complex) in the presence of tetrame

189 ed Cu(II) complex Cu{N(SiMe3 )Dipp}2 (1 Dipp=C6 H5 -2,6Pr(i) 2 ) and its Cu(I) counterpart [Cu{N(SiMe

190 not detected with reactive cations such as [C6 H5 ](+) .

191 Treatment of Zr2 with one equivalent of KCH2 C6 H5 and two equivalents of benzo-15-crown-5 ether (B15

192 1, Tren(TIPS) =N(CH2 CH2 NSiPr(i)3 )3 ) with C6 H5 CH2 K and [U(Tren(TIPS) )(THF)][BPh4 ] (2) afforde

193 Treatment of 3 with C6 H5 CH2 K and two equivalents of benzo-15-crown-5 ethe

194 ddition of H3 BNMeR'H2 to [Rh(L(R) )(eta(6) -C6 H5 F)][BAr(F) 4 ].

195 C6 had the lowest natural killer infiltration rate and w

196 din A by taking advantage of a direct indole C6 halogenation of the related ketopremalbrancheamide.

197 found that the thermal isomerization rate of C6 HAT drastically increases on metal surfaces, the ther

198 C6 HAT exhibits a half-life of 789 years in solution.

199 ization mechanism to explain the behavior of C6 HAT on these different metal surfaces.

200 used to study the photoisomerization of the C6 HAT self-assembled monolayers (SAMs) on Au, Ag, and C

201 It was found that C6 HAT switches on Au and Cu surfaces when irradiated wi

202 ctionalized with a C6 alkyl thiolate spacer (C6 HAT) was characterized on a number of metal surfaces.

203 d medium-chain acylcarnitines, in particular C6-hexanoylcarnitine, C8-octanoylcarnitine, C9-nonanoylc

204 roduction of a vinyl group as a masked OH at C6, (iii) an oxymercurative aldol to synthesize the tric

205 point and of the two flanking centers (C4-C5-C6 in aldoheptoses and higher and C5-C6-C7 in sialic and

206 in, and transient receptor potential channel C6 in the podocyte was significantly altered in 1,25-vit

207 granules containing conjugated drugs C3 and C6 induced greater new bone volume generated and increas

208 The virus burdens in the livers of HAdV-C6-infected hamsters are higher than the virus burdens i

209 In addition, compounds HT C1, C2, C4 and C6 inhibited endothelial cell migration and formation of

210 Knockdown of ABC-C6 inhibits egg hatching of Meloidogyne and Globodera sp

211 Experiments were performed using a C6 intracranial glioma tumor model in 37 male Sprague Da

212 Similarly to mouse models, in hamsters, HAdV-C6 is sequestered by macrophages to a lesser degree than

213 radio ((14)C) isotopologue and a stable ((13)C6) isotopologue of acetaminophen as substrates for in v

214 The radiocalibrated (13)C6-isotopologues were in turn used to quantitate acetami

215 ABC-C6 knockdown has no impact on the attraction of the plan

216 During the pulse, rats (n = 5) were fed (13)C6-labeled lysine ("heavy") feed for 23 days to label pr

217 Experiments with (13)C6 labelled Gal and Glc showed that both monosaccharides

218 The performance of 8Ah C6/LiFePO4 pouch cells were measured following periods o

219 conformation for the fluoromethyl group (C5-C6 linkage) was ascertained for congeners with a fluorin

220 amsters are infected intravenously with HAdV-C6, live, infectious virus can be isolated from the lung

221 The commercially-available C6 Lyme enzyme immunoassay (EIA) has been approved to re

222 ns in the [(((t.Bu)ArO)(3)tacn)U(III)((Me)cy-C6)].((Me)cy-C6) adduct.

223 Of these, the C6-Me-(2-phenylethynyl) and C2-(5-chlorothienylethynyl)

224 andards, traditionally used to calibrate (13)C6-metabolites via liquid chromatography-tandem mass spe

225 phy-tandem mass spectrometry (LC-MS/MS), (13)C6-metabolites were radiocalibrated by their (14)C-isoto

226 ase are used iteratively to introduce C5 and C6 methoxy groups.

227 These data indicate that C6(-/-) mice have reduced innate immune responses that r

228 a model of acute lung injury induced by LPS, C6(-/-) mice showed reduced PMN buildup and less lung ep

229 In addition, in C6(-/-) mice, the NLRP3 inflammasome was defective both

230 rates were accelerated by ~90%, while C3 and C6 mutant MyBP-C incorporated normally with degradation

231 entified in the genes coding for C5 (n = 4), C6 (n = 5), C7 (n = 12), C8 (n = 7), and C9 (n = 2).

232 with DBBM Control, DBBM with C3 or DBBM with C6 (n = 8 defects per group/ each timepoint).

233 cyclization of a N-chloroamine to forge the C6-N bond, a transannular Mannich-type reaction of a cyc

234 ratio in the dorsal nerve root ganglion and C6 nerve (P < .001) with the multiecho TSE-mDixon sequen

235 e we show that the nonspecific phospholipase C6 (NPC6) promotes seed oil production in the Brassicace

236 en in cells overexpressing leptin receptors (C6-ObRb).

237 onation at N5 of H2F and hydride transfer to C6 occur in a stepwise mechanism.

238 f the cluster becomes covalently attached to C6 of the octanoyl substrate.

239 The structure revealed that the C6 of tryptophan is positioned next to the e-amino group

240 al isomers (C1alpha, C1beta, C2, C3, C4, and C6) of a Glc-Pt.

241 lso the first to alter the mode (N1/N4 vs C3/C6) of cycloaddition.

242 e anabolic conjugated drugs, known as C3 and C6, of an inactive bisphosphonate and a bone activating

243 gioselectively a fructosyl residue to either C6-OH group of the glucose residues in Tre.

244 or five-membered H-bonded pseudocycles (C8, C6 or C5), depending on the steric bulk of both N(alpha)

245 nd forearm flexors when using all 5, 2 (C5 + C6) or 1 (isolated C6) spinal nerve as the donor nerves.

246 brominate trypto-phan's indole moiety at C5, C6, or C7.

247 by using either the mice deficient in C5 and C6, or MAC's regulator CD59.

248 However, the DbpA/C6-OspC ELISA was markedly better (80% versus 63%) than

249 The two methods (DbpA/C6-OspC versus 2-tier test) were equivalent in identifyi

250 ntinuous sampling of C4-AHL, C6-AHL, C8-AHL, C6-OXO-AHL, and C12-OXO-AHL was achieved over a period o

251 The presence of a conformationally flexible C6 oxygen atom in the sugar-derived lactol donors is req

252 rformance is comparable to the commonly used C6 peptide by lab-based ELISA.

253 omer sulfonamide alkylamine (FTAA) and C3 to C6 perfluoroalkane sulfonamido amphoterics.

254 activities were attributed to samples C1 and C6 (PGS 7/8) and samples C2 (PGS 5/6) and C4 (PGS 6/7),

255 5-hydroxymethyl furfural is an underutilized C6-platform chemical derived from cellulose that is idea

256 ansient receptor potential (TRP) ion channel C6 plays a pivotal role in hereditary and sporadic glome

257 enabled their regiospecific ring-opening at C6 position by organometallic nucleophiles.

258 ations that could generate a new bond at the C6 position of a dienoate are particularly desirable bec

259 ew azabicycloalkane scaffolds endowed at the C6 position with a para-substituted phenethyl side chain

260 be used as a handle for cyclization onto the C6 position, using Bu(3)SnH-mediated radical cyclization

261 o the major product PA-A is oxidation at the C6 position.

262 a regioselective fashion at the challenging C6-position of 2-pyridone intermediates.

263 ion of imidazo[1,2-a]pyrimidine cores on the C6-position.

264 7 surface may accept galactosylations at the C6 positions of mannose adjacent to the mannose residue

265 ) the number of fluorine atoms at the C2 and C6 positions, and (3) the number of pairs of fluorine at

266 C6-propyl analogue of 2 (6) displayed 700-fold selectivi

267 the ligand and reaction conditions used, the C6 regioisomer a can be obtained in 4:1 ratio and excell

268 ty relationships identified sites in the C4'-C6' region on ring I that reduced ototoxicity while pres

269 pes belonging to species C, HAdV-C5 and HAdV-C6, replicate to significantly different extents and cau

270 ignificantly different severities, with HAdV-C6 replicating better and inducing more severe and more

271 ABC-C6 represents a promising target for breeding or biotech

272 l carboxylates and sulfonates (>/=C8 and >/= C6, respectively).

273 cules adsorb bowl opening-up with the center C6 ring parallel to the surface.

274 SpnF because interconversion between the C5-C6 s-trans and s-cis conformers is facile.

275 started with C3-chalcogenation, followed by C6 selanylation, activated in acidic medium.

276 tion of the metalation conditions for C2 and C6, separately and iteratively, is presented.

277 ength conservation, is predicted with C3 and C6 smoothness between and within segments, using only 2

278 sis") to evaluate tissue samples from the C2-C6 spinal cord 3 days after a C3/C4 hemi-crush injury (C

279 when using all 5, 2 (C5 + C6) or 1 (isolated C6) spinal nerve as the donor nerves.

280 quency) and sham-TESS applied between C5 and C6 spinous processes in males and females with and witho

281 the transacylation process are governed by a C6-substituent on the purine ring (R = NH(2), Me, NHBz,

282 al-group tolerance and notably proceeds with C6 substituted indoles.

283 nd quenching with an electrophile to furnish C6-substituted derivatives which, in the presence of a c

284 evelopment of catalysts and processes for C5-C6 sugar reforming into chemical intermediates and produ

285 drate polymers into their constituent C5 and C6 sugars, and subsequent heterogeneously catalyzed tran

286 ituted hydrocarbons, with chain lengths from C6 to C11, exhibit a dramatic odd-even effect in helical

287 mination was almost completely switched from C6 to C7 as in RebH.

288 depleted of individual complement components C6 to C9.

289 ogenase, which oxidizes the alcohol group at C6 to the COO(-) group.

290 95 (C4), Cys148 (C5) to Cys219 (C8), Cys151 (C6) to Cys243 (C9), and Cys161 (C7) to Cys266 (C10).

291 DPP) chromophores with methyl (Me), n-hexyl (C6), triethylene glycol (TEG), and 2-ethylhexyl (EH) sub

292 Transient receptor potential channel C6 (TRPC6) gain-of-function mutations and increased TRPC

293 85:1 preference for H atom abstraction from C6 versus C7; however, this inverts to a 1:16 preference

294 especially for 3,4-DHPEA-EDA, p-HPEA-EDA and C6 volatiles.

295 on from each of two adjacent carbon centers (C6 vs C7) in the substrate.

296 rthermore, the antiangiogenic activity of HT C6 was confirmed in vivo in the chick chorioallantoic me

297 ied serotype; when serotypes C1, C2, C5, and C6 were detected, they were responsible for symptoms in

298 ry and recovery, we generated littermate PVG C6 wildtype and deficient rats and tested functional and

299 nditions: (1) animals maintained as separate C6 WT and C6-D homozygous colonies; and (2) establishmen

300 ishment of a heterozygous colony to generate C6 WT and C6-D littermate controls.