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1                                              CALI did not negatively impact survival.
2                                              CALI do not negatively impact long-term prognosis, but t
3                                              CALI of MAG permitted significant regrowth of retinal ax
4                                              CALI of MII reduced neurite outgrowth and growth cone ar
5                                              CALI of myosin-V in growth cones of chick dorsal root ga
6                                              CALI of purified chick brain myosin-V absorbed onto nitr
7                                              CALI provides an approach to inactivate in vivo function
8                                              CALI worsen the short-term outcomes of liver resection,
9                        Among 78 patients (39 CALI and 39 matched control lung transplant patients), t
10          EGFP has been used effectively as a CALI chromophore, and its cotranslational attachment to
11 r concentrations of protein, turbidity after CALI increased significantly indicating cross-linking of
12        All specimens were reviewed to assess CALI, TRG, and micrometastases.
13                                  ReAsH-based CALI is genetically targeted, requires no antibodies or
14                           We now demonstrate CALI of connexin43 (Cx43) and alpha1C L-type calcium cha
15                                      At E12, CALI of ephrin-A5 did not affect the extent of axon outg
16 re bleaching and those that escape to effect CALI of proximate proteins.
17                              Performing EGFP-CALI experiments in deficient cells rescued with functio
18                 A potential drawback to EGFP-CALI is that the CALI phenotype can be obscured by the e
19 nglet oxygen respectively inhibit or enhance CALI.
20           One-year mortality was similar for CALI and control groups (12.8% vs 10.3%, respectively).
21  Patients who received a lung transplant for CALI are more deconditioned with prolonged hospital stay
22  patients who received a lung transplant for CALI have, to date, not looked at the infectious outcome
23 ients who underwent lung transplantation for CALI (case) and for non-COVID-19 end-stage lung disease
24                    Patients transplanted for CALI had higher rates of infection pre-transplant (66.7%
25 n mediated by fluorescent protein CALI ([FP]-CALI), the activities of purified glutathione-S-transfer
26                          Here, we generalize CALI so that it can be applied to a wider range of tasks
27 nder (FlAsH) results in significantly higher CALI efficiency than any of the fluorescent proteins (XF
28     Chromophore-assisted laser inactivation (CALI) is a light-mediated technique that offers precise
29     Chromophore assisted laser inactivation (CALI) is a technique that uses irradiation of chromophor
30 sed chromophore-assisted laser inactivation (CALI) to generate acute loss of ephrin-A5 function in lo
31 ted chromophore-assisted laser inactivation (CALI) to instantly and specifically inactivate it.
32 ing chromophore-assisted laser inactivation (CALI).
33 ing chromophore-assisted laser inactivation (CALI).
34  by chromophore-assisted laser inactivation (CALI).
35  to chromaphore-assisted light inactivation (CALI) , which resulted in the unexpected dissipation of
36 uct chromophore assisted light inactivation (CALI) of synaptic proteins.
37     Chromophore-assisted light inactivation (CALI) offers the only method capable of modulating speci
38 zes chromophore-assisted light inactivation (CALI) to inactivate presynaptic neurotransmitter release
39     Chromophore-assisted light inactivation (CALI) uses photochemically generated, reactive oxygen sp
40     Chromophore-Assisted Light Inactivation (CALI) using genetically-encoded photosensitizers provide
41  as chromophore-assisted light inactivation (CALI) with fluorescein derivatives, have been limited by
42 s as well as the removal of oxygen inhibited CALI, indicating the involvement of a reactive oxygen sp
43 pact of chemotherapy-related liver injuries (CALI), pathological tumor regression grade (TRG), and mi
44 ease 2019 (COVID-19)-associated lung injury (CALI).
45 ed green fluorescent protein (EGFP) mediated CALI has been used to inactivate EGFP-fusion proteins in
46              In conclusion, miniSOG-mediated CALI is a novel genetic platform for acute inactivation
47                               ReAsH-mediated CALI acts largely via singlet oxygen because quenchers o
48                                        Micro-CALI of L1 causes neurite retraction after a 10 min lag
49                                        Micro-CALI of M1c reduced retrograde bead flow by 76%, whereas
50                                        Micro-CALI of MII caused a rapid reduction in local lamellipod
51                                        Micro-CALI of radixin targeted to the middle of the leading ed
52                 Episodes of asymmetric micro-CALI of myosin 1c (or myosin 1c and V together) caused s
53  measured trajectories from asymmetric micro-CALI of myosin 1c-treated and untreated growth cones to
54  contribution to turning by asymmetric micro-CALI of myosin isoforms that causes localized lamellipod
55        Functional ablation of ezrin by micro-CALI (chromophore-assisted laser inactivation) blocked p
56 symmetric inactivation of myosin 1c by micro-CALI.
57                           In contrast, micro-CALI of NCAM-180 causes rapid growth cone retraction but
58 romophore-assisted laser inactivation (micro-CALI) of radixin in growth cones causes a 30% reduction
59 romophore-assisted laser inactivation (micro-CALI) of these proteins to perturb their functions at pr
60                  In contrast, repeated micro-CALI of myosin V or irradiation without added antibody d
61 e behavior of growth cones after these micro-CALI treatments resemble the drug-induced perturbation o
62                    This is opposite to micro-CALI of M1c, which caused an increase in lamellipodial p
63 d retrograde bead flow by 76%, whereas micro-CALI of MII or the MIIB isoform did not.
64 illumination to examine the effectiveness of CALI targeted to kinesin.
65     Finally, we apply this implementation of CALI to an in vitro system of motor proteins and microtu
66                     In contrast, labeling of CALI of MAG-treated crushed optic nerve showed significa
67 inactivation mediated by fluorescent protein CALI ([FP]-CALI), the activities of purified glutathione
68 luorescein, comparing them with the standard CALI dye, malachite green; and we study the relative eff
69                              In this system, CALI can effectively perturb local structure formation b
70                          We demonstrate that CALI of EGFP-CapZbeta increases uncapped actin filaments
71                     We also demonstrate that CALI of EGFP-Mena in Mena/VASP-deficient cells stabilize
72                                 We show that CALI can destroy kinesin activity in at least two ways:
73                   Specifically, we show that CALI can work with a genetically inserted epitope tag; w
74 trol lung transplant patients), those in the CALI cohort were less likely to be vaccinated pre-transp
75  potential drawback to EGFP-CALI is that the CALI phenotype can be obscured by the endogenous, unlabe
76 ferent FP mutants fused to GST vary in their CALI efficiency in the order enhanced green fluorescent
77 ition to globally inhibiting actin turnover, CALI of cofilin generated several profound effects on th
78 e this technique Inhibition of Synapses with CALI (InSynC).