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1                                              CCAP activated pyloric rhythms in most silent preparatio
2                                              CCAP affected each of the four functional groups of moto
3                                              CCAP exposure also actively terminated pre-ecdysis burst
4                                              CCAP KO animals showed specific defects at ecdysis, yet
5                                              CCAP seems to activate slow intrinsic oscillations in th
6                                              CCAP, the largest European protistan culture collection,
7 even after targeted ablation of all other 42 CCAP neurons.
8                                We isolated a CCAP-related peptide (conoCAP-a, for cone snail CardioAc
9 rsor of conoCAP-a encodes for two additional CCAP-like peptides: conoCAP-b and conoCAP-c.
10                                     Although CCAP KO populations exhibited circadian eclosion rhythms
11                       Consequently, although CCAP neither changes retraction duration nor alters GPR
12 ila larvae, demonstrating that conoCAP-a and CCAP have opposite effects.
13 contrasting cardiac effects of conoCAP-a and CCAP indicate that molluscan CCAP-like peptides have fun
14  activated during pre-ecdysis; EH, CCAP, and CCAP/MIP neurons are active prior to and during ecdysis;
15 ating to neuropeptide surges of both CHH and CCAP were seen during larval hatching, when compared to
16  and bursicon in Onychophora and EH, ETH and CCAP in Tardigrada suggests that the pathway was present
17  ingestion of sugar and water, with IPCs and CCAP neurons oppositely regulating sugar and water inges
18           Preliminary analyses using PCR and CCAP immunohistochemistry suggested that, whereas the DN
19                                Proctolin and CCAP both act on the lateral pyloric neuron and the infe
20 The motor patterns produced in proctolin and CCAP differed quantitatively in a number of ways.
21 the motor patterns produced by proctolin and CCAP.
22                                 Bath-applied CCAP also excites both LG and Int1, but selectively prol
23 d pharmacologically by sequentially applying CCAP and glutamate to the heart.
24                                   All 12 are CCAP efferents that exit the central nervous system.
25                              While arthropod CCAP is a cardio-accelerator, we found that conoCAP-a de
26 se ecdysis motor bursts persisted as long as CCAP was present and could be reinduced by successive ap
27              Furthermore, flies lacking both CCAP and bursicon show much more severe defects at ecdys
28  was distinguishable from CCAP and RPCH, but CCAP and RPCH were not distinguishable from each other.
29 (MI) in LG, the parallel G(MI) activation by CCAP reduces the impact of GPR regulation of this conduc
30       The changes in motor pattern evoked by CCAP produced significant changes in LP-innervated muscl
31 e movements were additionally potentiated by CCAP applications to isolated nerve-muscle preparations.
32 hat the functions thought to be subserved by CCAP are partially effected by bursicon, and that bursic
33 ur neuron types in the core pyloric circuit, CCAP and RPCH target the same subset of only two neurons
34 Neurons IN704 in abdominal ganglia coexpress CCAP and MIPs, whose joint actions initiate the ecdysis
35 xcitability of a group of neurons containing CCAP.
36 e for carbohydrates, and Cyclotella cryptica CCAP 1070/2, with utility for EPA production and N-assim
37                       A subset of the doomed CCAP neurons in the ventral nerve cord also expressed th
38 eurons are activated during pre-ecdysis; EH, CCAP, and CCAP/MIP neurons are active prior to and durin
39                        Identification of EH, CCAP and bursicon in Onychophora and EH, ETH and CCAP in
40  a pivotal downstream circuit neuron enables CCAP to weaken or eliminate sensory regulation of motor
41                            The threshold for CCAP action was approximately 10(-10) M, with increasing
42 ed expression but no IMI responses, we found CCAP modulation of synaptic currents.
43 cuit output in PROC was distinguishable from CCAP and RPCH, but CCAP and RPCH were not distinguishabl
44                                         High CCAP concentrations sometimes resulted in modification o
45 10(-10) M, with increasing effects at higher CCAP concentrations.
46 y when challenged with persisting (hormonal: CCAP) or acute (sensory: GPR neuron) metabotropic influe
47                                           In CCAP, the pyloric rhythms were characterized by long lat
48 hose release of EH increases excitability in CCAP/MIP neurons.
49                 We show that null mutants in CCAP express no apparent defects in ecdysis and postecdy
50 exhibited similar disruptions as observed in CCAP neuron-knockout Drosophila larvae.
51 ed that both domains are necessary to induce CCAP cell death.
52 ts non-fruiting relatives Rosculus 'ithacus' CCAP 1571/3, R. terrestris n. sp. and R. elongata n. sp.
53                   Transgenic flies that lack CCAP neurons also lacked bursicon bioactivity.
54 n additional cluster composed of four large, CCAP-positive neurons innervates the terminal chamber.
55 re pupal ecdysis by the emergence of 12 late CCAP neurons.
56             Our evidence indicates that late CCAP neurons are derived from early, likely embryonic, l
57                      Importantly, these late CCAP neurons were found to be entirely sufficient for wi
58            It also was more sensitive to low CCAP concentrations and showed saturation at lower conce
59           This distinction results from I(MI-CCAP) being regulated only by postsynaptic voltage, wher
60                                    With I(MI-CCAP) continually present, the impact of the feedback in
61    However, the CCAP-activated current (I(MI-CCAP)) and MCN1-activated current (I(MI-MCN1)) exhibit d
62                                    Molluscan CCAP-like peptides sequences, while homologous, differ b
63 is analogous to recently predicted molluscan CCAP-like preprohormones, and suggests a mechanism for t
64 f conoCAP-a and CCAP indicate that molluscan CCAP-like peptides have functions that differ from those
65                       In dfmr1 null mutants, CCAP/bursicon neurons also exhibit significantly delayed
66                                    Because N(CCAP) mediates environmentally insensitive ecdysis-relat
67 atterns can be initiated by stimulation of N(CCAP), a small network of central neurons that regulates
68  bearing targeted ablations of CCAP neurons (CCAP KO animals) to investigate the role of CCAP in the
69 ance within a defined group of neuropeptide (CCAP) -containing neurons of the ventral nervous system
70 e screen identified Nannochloropsis oceanica CCAP 849/10 and a marine isolate of Chlorella vulgaris C
71 sed Drosophila bearing targeted ablations of CCAP neurons (CCAP KO animals) to investigate the role o
72 prior to and during ecdysis; and activity of CCAP/MIP/bursicon neurons coincides with postecdysis.
73 MI, showed that saturating concentrations of CCAP activated all available IMI in LP, but only approxi
74 rsicon is believed to then act downstream of CCAP to inflate, pigment, and harden the exoskeleton of
75             The cardioacceleratory effect of CCAP release at this location may modulate the propertie
76  believed to be critical for the function of CCAP efferent neurons in ecdysis.
77 tic approach to investigate the functions of CCAP and bursicon in Drosophila ecdysis.
78 f this work is that the primary functions of CCAP as well as its importance in the control of ecdysis
79 , may also regulate ecdysis independently of CCAP.
80 ood pressure in contrast to the injection of CCAP, which did not elicit any cardiac effect.
81 eased sensitivity to sugar; however, loss of CCAP, or CCAP-R in 2 dorsal median NPF neurons, inhibite
82                             Notably, loss of CCAP, or knocking down the CCAP receptor (CCAP-R) in 2 d
83                              The majority of CCAP KO animals died at the pupal stage from the failure
84                       The primary mission of CCAP is to maintain and distribute defined cultures and
85 ic-clamp manipulations, that the presence of CCAP weakens or eliminates the GPR effect on the gastric
86 to the PD and PY neurons, in the presence of CCAP, and converted the CCAP rhythm into a rhythm that w
87  nervous system, consistent with the role of CCAP in a range of different behaviors.
88 (CCAP KO animals) to investigate the role of CCAP in the execution and circadian regulation of ecdysi
89  actions have always been placed upstream of CCAP, may also regulate ecdysis independently of CCAP.
90 sitivity to sugar; however, loss of CCAP, or CCAP-R in 2 dorsal median NPF neurons, inhibited sugar s
91 s bearing targeted ablations of either EH or CCAP neurons, or ablations of both together, to reevalua
92 uggest that crustacean cardioactive peptide (CCAP) activates the ecdysis motor program; the hormone b
93 y examining crustacean cardioactive peptide (CCAP) and bursicon circuits, which are similarly develop
94 rmone (EH), Crustacean cardioactive peptide (CCAP) and Bursicon.
95             Crustacean cardioactive peptide (CCAP) and related peptides are multifunctional regulator
96 the hormone crustacean cardioactive peptide (CCAP) and the gastropyloric receptor (GPR) proprioceptor
97 e (ETH) and crustacean cardioactive peptide (CCAP) elicit the first two motor behaviors, the pre-ecdy
98             Crustacean cardioactive peptide (CCAP) elicited expression of the motor pattern that driv
99 e (ETH) and crustacean cardioactive peptide (CCAP) evolved in the bilaterian last common ancestor (LC
100 t expresses crustacean cardioactive peptide (CCAP) has been shown previously to make the hormone burs
101 orealis, by crustacean cardioactive peptide (CCAP) is described.
102             Crustacean cardioactive peptide (CCAP) neurons and the peptide hormones they secrete are
103 lls (IPCs), crustacean cardioactive peptide (CCAP) neurons, and CCHamide-2 receptor isoform RA (CCHa2
104 function of crustacean cardioactive peptide (CCAP) neurons, either by directly acting on their nicoti
105  peripheral crustacean cardioactive peptide (CCAP) neurons, which potentiate the anterograde beat.
106 ptides from crustacean cardioactive peptide (CCAP) neurons.
107 europeptide Crustacean cardioactive peptide (CCAP) plays a key role in the initiation of the ecdysis
108 milarity to crustacean cardioactive peptide (CCAP) receptors in insects and mammalian neuropeptide S
109 lin (PROC), crustacean cardioactive peptide (CCAP), and red pigment concentrating hormone (RPCH) acti
110 atostatins, crustacean cardioactive peptide (CCAP), calcitonin-like diuretic hormone, CRF-like diuret
111  respond to crustacean cardioactive peptide (CCAP), corazonin, or adipokinetic hormone (AKH), none of
112 de hormone, crustacean cardioactive peptide (CCAP), modulates the biphasic (protraction/retraction) g
113 rmone (EH), crustacean cardioactive peptide (CCAP), myoinhibitory peptides (MIP), and bursicon.
114 peptide Ia, crustacean cardioactive peptide (CCAP), red pigment-concentrating hormone, TNRNFLRFamide,
115 ulting; and crustacean cardioactive peptide (CCAP), which is involved in stereotyped ecdysis behaviou
116 ered that 2 crustacean cardioactive peptide (CCAP)-expressing neurons in Drosophila adults regulate f
117             Crustacean cardioactive peptide (CCAP)-expressing neurons undergo programmed cell death (
118 ositive for crustacean cardioactive peptide (CCAP).
119 europeptide crustacean cardioactive peptide (CCAP).
120 ressed with crustacean cardioactive peptide (CCAP).
121 e (EH), and crustacean cardioactive peptide (CCAP).
122 rossed to a crustacean cardioactive peptide (CCAP)/bursicon neuron-specific Gal4 driver.
123 whereas the native disulfide-bonded peptides CCAP-vil, mu-conotoxin KIIIA, and human insulin were use
124 ical applications e.g. Dunaliella polymorpha CCAP 19/14, significantly the most productive for carboh
125 response of peptidergic neurons that produce CCAP (crustacean cardioactive peptide), which are key ta
126 y Map, Cancer Chromosome Aberration Project (CCAP) pages, Entrez Genomes, Clusters of Orthologous Gro
127 dbSNP, Cancer Chromosome Aberration Project (CCAP), Entrez Genomes and related tools, the Map Viewer,
128 y Map, Cancer Chromosome Aberration Project (CCAP), Entrez Genomes and related tools, the Map Viewer,
129 y Map, Cancer Chromosome Aberration Project (CCAP), Entrez Genomes, Clusters of Orthologous Groups (C
130 y Map, Cancer Chromosome Aberration Project (CCAP), Entrez Genomes, Clusters of Orthologous Groups (C
131 of CCAP, or knocking down the CCAP receptor (CCAP-R) in 2 dorsal median neurons, inhibits the release
132 eceptors of inhibitory neurons that regulate CCAP activity.
133 rdinately in response to hormonally released CCAP.
134  carbon core metabolic network for D. salina CCAP 19/18 based on the recently published nuclear genom
135 e formation or maintenance of adult-specific CCAP/bursicon cell projections during metamorphosis.
136           In the absence of GPR stimulation, CCAP does not alter retraction duration and modestly pro
137 tic and optogenetic studies demonstrate that CCAP signaling is necessary and sufficient to stimulate
138 ynamic-clamp manipulations to establish that CCAP prolongs the gastric mill protractor (LG) phase and
139              These results are evidence that CCAP acts at two levels: activation of local premotor ci
140                Here we present evidence that CCAP-expressing neurons (NCCAP) consist of two functiona
141                           We also found that CCAP immunoreactivity decreases centrally during normal
142 s and its circadian regulation indicate that CCAP is a key regulator of the behavior.
143                                          The CCAP-activated G(MI) thus counteracts the GPR-mediated d
144 , in the presence of CCAP, and converted the CCAP rhythm into a rhythm that was statistically similar
145  Notably, loss of CCAP, or knocking down the CCAP receptor (CCAP-R) in 2 dorsal median neurons, inhib
146 in the CNS; by autocrine influences from the CCAP neurons themselves; and by inhibitory actions media
147                                 However, the CCAP-activated current (I(MI-CCAP)) and MCN1-activated c
148 sing number of fully sequenced protists, the CCAP is striving to provide targeted services and suppor
149 te from its regulation of NPF signaling, the CCAP peptide also regulates triglyceride levels.
150       In this study, we demonstrate that the CCAP neuron network is remodeled immediately before pupa
151               These data illustrate that the CCAP peptide is a central regulator of feeding behavior
152  to their cGMP response at ecdysis and their CCAP-IR.
153 th a rise in intracellular cGMP within these CCAP neurons.
154                                         This CCAP action results from its ability to activate the sam
155  lepidopteran pupal ecdysis directly through CCAP neurons or by activating their upstream efferent in
156 bset had previously been shown to respond to CCAP with the activation of a modulator-activated inward
157 d, desheathed abdominal ganglia responded to CCAP by generating rhythmical ecdysis bursts.
158 concentration dependence of IMI responses to CCAP application in two identified neurons, the lateral
159 0 and a marine isolate of Chlorella vulgaris CCAP 211/21A as the best lipid producers.
160  flies, in which most of the 50 neurons were CCAP-IR, although none showed increases in cGMP at ecdys
161 GMP-IR at ecdysis, although the neurons were CCAP-IR.
162                                         When CCAP was superfused in a low Ca2+ saline that blocked ch
163                                         When CCAP was superfused, the membrane potentials of these ne
164                          Conversely, whereas CCAP-IR was severely reduced in the thoracic and subesop
165 ion that innervates swimmerets, neurons with CCAP-like immunoreactivity sent processes to the lateral
166                               Hence, without CCAP, retraction and protraction duration are determined

 
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