ライフサイエンスコーパス: CDS

1 CDS activity maintains phosphoinositide 4,5 bisphosphate

2 CDS and PDS neurons-selected based on their responses to

3 CDS neurons were strongly tuned for the direction of mot

4 ulated; (ii) 360 (99%) of the arrayed PBCV-1 CDSs were expressed at some time in the virus life cycle

5 Here we study a set of 176 CDS time series of financial institutions from 2002 to 2

6 e virus DNA synthesis begins; (iv) these 227 CDSs were grouped into two classes: 127 transcripts disa

7 There are 26 CDSs that have no viral homolog, and many did not necess

8 Additionally, 27 CDSs were identified which were present in all 47 low-pa

9 physical activity counseling (CDS arm, 45%; CDS + coaching arm, 25%; P < .001 compared with usual ca

10 ent of 39.2% and encompassed a total of 5362 CDS.

11 not support the hypothesis that the 7q22/5A3 CDS interval contains a myeloid TSG.

12 nt, we confirmed somatic deletion of the 5A3 CDS in the hematopoietic stem cell compartment.

13 ive ShE.coli meta-array gene sequences (6239 CDS) were used for in silico hybridization analysis.

14 mine whether haploinsufficiency for the 7q22 CDS contributes to myeloid leukemogenesis, we performed

15 al regions of interest in recently abstinent CDSs (n = 24) and healthy control subjects (n = 36) both

16 ethyltetrahydro-beta-carboline in the active CDS extract.

17 lear localization causes elimination of Adi3 CDS activity and induction of cell death.

18 After CDS implementation, quarterly use decreased 20.1%, from

19 y angiography were compared before and after CDS implementation in August 2007.

20 nations per 1000 admissions before and after CDS, respectively; P = .008) observed 1 month after CDS

21 monary angiographic imaging before and after CDS.

22 spectively; P = .008) observed 1 month after CDS implementation was sustained over the ensuing 32-mon

23 hic examinations positive for acute PE after CDS (P = .65).

24 However, not all CDS systems are effective.

25 principle, the network of correlations among CDS spread time series could at least display some form

26 ions of CON1 dosage with SDS (beta=0.18) and CDS (beta=0.13).

27 ed odds ratio, 2.28 [95% CI, 1.15-4.53]) and CDS + coaching arm (adjusted odds ratio, 2.60 [95% CI, 1

28 density and average nucleotide diversity and CDS (coding sequences) the lowest, in both humans and ma

29 For both DV and CDS, there was evidence of linkage to the same region on

30 negative relationship between expression and CDS length.

31 C Known Genes offers the highest genomic and CDS coverage among major human and mouse gene sets, more

32 nd 19.89% based on the ATP III, IDF, JIS and CDS criterion, respectively.

33 text of 5'- and 3'-untranslated regions, and CDS sections of aligned mRNA sequences.

34 RNA degradome data, we discovered uORFs and CDS regions potentially associated with stacked ribosome

35 s with independently altered codon usage and CDS structure, we find that the structure of the CDS reg

36 mportance of separately examining 3' UTR and CDS sequences in gene expression analyses.

37 unbalanced expression of cognate 3' UTR and CDS sequences; many genes show high 3' UTR relative to C

38 the ratio of translation over 5' leaders and CDSes is conserved between human and mouse, and correlat

39 als that sequence features at both uORFs and CDSes modulate the translation of CDSes.

40 and dynamics comparable to that of annotated CDSs.

41 arify the boundaries of previously annotated CDSs in Trypanosoma brucei.

42 e facilitated by useful and widely available CDS.

43 the KVP40 genome is coding, with an average CDS size of 587 bp.

44 Implementation of evidence-based CDS for inpatients was associated with a 12.3% immediate

45 Implementation of evidence-based CDS in the ED was associated with a significant decrease

46 onary angiography use increased 82.1% before CDS implementation, from 14.5 to 26.4 examinations per 1

47 The Winspit BoMYB29 CDS was cloned and overexpressed in Winspit and in the D

48 nothing is known about the single T. brucei CDS gene (Tb927.7.220/EC 2.7.7.41) or its activity.

49 mentation of an interaction with a certified CDS system using approved appropriate use criteria will

50 his meta-array lacked probes for many CFT073 CDS.

51 e shown to coelute with the active classical CDS extracts.

52 lan represent active components of classical CDS.

53 on 12 microsatellite loci and 1715 combined CDSs from whole-genome sequencing revealed three subclus

54 An intervention that included computerized CDS for pediatric clinicians and support for self-guided

55 imulated data set created from the Consensus CDS project and two experimental RNA-seq data sets gener

56 r nutrition or physical activity counseling (CDS arm, 45%; CDS + coaching arm, 25%; P < .001 compared

57 Crude CDS from bovine lung was purified by reverse-phase high-

58 h shifted codon triplet usage to a different CDS with a hydrophilic C terminus, changing the canonica

59 toxin and immunity proteins, did not display CDS, whereas a strain of Burkholderia dolosa producing a

60 'UTR, CDS and 3'UTR and subsequently divides CDS introns into three categories, such as phase 0, phas

61 dex of antisocial substance dependence (DV + CDS).

62 Our composite index (DV + CDS) yielded the strongest evidence for linkage (logarit

63 genome: the exome-plus design and the exome-CDS design.

64 This reduced the size of the exome-CDS to approximately 71 Mb.

65 The exome-CDS was designed as a subset of the exome-plus by omitti

66 s, we estimated the performance of the exome-CDS.

67 ith full-length protein coding sequences (FL-CDS).

68 ders overrode alerts than when they followed CDS guidelines.

69 , and a novel region on chromosome 17q12 for CDS.

70 had the highest accuracy in rice (0.5648 for CDS, 0.4468 for exon, and 0.6695 nucleotide structure) a

71 otide structure) and A. thaliana (0.5808 for CDS, 0.5955 for exon, and 0.8839 nucleotide structure).

72 ion of Adi3 directs nuclear localization for CDS and that disruption of Adi3 nuclear localization may

73 on of PEMT and thereby put women at risk for CDS.

74 s to establish clear physiological roles for CDS.

75 by coverage depth, with poor sensitivity for CDS at < 5X depth of sequencing, but have excellent PPV.

76 set of premenopausal women is protected from CDS.

77 accurately distinguish canonical ncRNAs from CDSs and UTRs (accuracies: >92% in human, mouse, worm an

78 suppresses the mitophagy induced by fundc1 (CDS+3'UTR) but not fundc1 (CDS) overexpression.

79 nduced by fundc1 (CDS+3'UTR) but not fundc1 (CDS) overexpression.

80 number of processed pseudogenes and the gene CDS length (R = -0.40; p < 0.001), i.e., the relatively

81 ith a total of 3,015 protein-encoding genes (CDS), and a GC content of 47.7%.

82 es to the PBCV-1 365 protein-encoding genes (CDSs) was constructed.

83 se was less common (47% for patients who had CDS and 31% for those who did not).

84 igh 3' UTR relative to CDS, others show high CDS to 3' UTR.

85 Considering that human CDS are devoid of >4 consecutive AAA codons, sensing of

86 ferent computational methods for identifying CDS from whole-genome shotgun sequencing is not fully es

87 34 behaved as an apparent inverse agonist in CDS assays involving TM cells but as a neutral prostamid

88 On average, responses began 6 ms earlier in CDS neurons than in PDS neurons.

89 It has been suggested that methylation in CDS slows down translation elongation.

90 n providers adhered to evidence presented in CDS were nearly double those seen when providers overrod

91 We compared the time course of responses in CDS and PDS neurons in opiate-anesthetized macaques, usi

92 portals adapted specifically for targets in CDS.

93 gnificantly lower in the cortical regions in CDSs compared with healthy control subjects.

94 (2/3) receptors and less dopamine release in CDSs were not associated with performance on working mem

95 cal D(2/3) receptors and dopamine release in CDSs.

96 reveals that rare variants tend to reside in CDSs, whereas common variants tend to reside in the 3' U

97 No studies in CDSs have reported the in vivo status of D(2/3) and dopa

98 lassified as lncRNAs due to their incomplete CDS, leading to higher false positive rate for lncRNA id

99 e the burden placed on users; 6) incorporate CDS success factors identified in the literature, in par

100 s the first good candidate domains for intra-CDS pairing of a miRNA in mammals.

101 p between Adi3 cellular localization and its CDS activity.

102 ear localization of Adi3 is required for its CDS activity and loss of nuclear localization causes eli

103 Four widely used criteria (ATPIII IDF JIS CDS) were used to measure the prevalence of MetS.

104 At least 99 KVP40 CDSs have homologs in the T4 genome (Blast alignments of

105 ymerase II (Pol II) progression through lacZ CDS in vivo.

106 ered late); and (vi) expression of most late CDSs is inhibited by adding the DNA replication inhibito

107 nd of the gene, and isolated the full-length CDS of the mNLRC5 of about 6 kb through conventional RT-

108 or patients with a Wells score of 4 or less, CDS alerts suggested d-dimer testing because acute PE is

109 th our previous proposal that miRNA-mediated CDS-targeting operates in this green alga.

110 ed miRNA:gene interactions from DIANA-microT-CDS for 4 species (human, mouse, nematode and fruit fly)

111 In silico analyses with DIANA-microT-CDS highlighted 5 putative targets in common between two

112 ithms (TargetScan, miRanda, and DIANA-microT-CDS), miR-18a-5p was predicted to be the top candidate m

113 Modern CDS systems are computer-based tools designed to improve

114 o whether patients belonged to the CDS or no CDS group.

115 primary-cancer-directed surgery (CDS) or no CDS within 4 months of diagnosis.

116 ecificity for coding sequence (CDS) over non-CDS, and increased tolerance to base-substituting and fr

117 years versus 5.6 years for patients with non-CDS QOL.

118 trategy to identify hundreds of noncanonical CDSs that are essential for cellular growth and whose di

119 an fibroblasts identifies thousands of novel CDSs, including micropeptides and variants of known prot

120 These results suggest that availability of CDS-controlled resynthesis of phosphoinositides is essen

121 ) most highly linked with the development of CDS (p < 0.00006) was located within 1 kb of the critica

122 rate that the elongation-promoting effect of CDS methylation requires the RNA helicase-containing m(6

123 centered interface design, (3) evaluation of CDS systems and rules, and (4) standards-based developme

124 llus thuringiensis (Bt) tested the impact of CDS recoding using different codon bias tables.

125 re, in particular the automatic provision of CDS as a part of clinician workflow; 7) ensure that the

126 ths scarcely change over the entire range of CDS values.

127 state until 50-75 ms after the responses of CDS neurons had stabilized.

128 nducible PEMT expression, conferring risk of CDS in women.

129 pairs was designed based on the sequences of CDS, 5'-UTR and 3'-UTR region covering 34%, 14%, 23%, re

130 mRNAs nor the physiological significance of CDS m(6)A has been clearly defined.

131 stablished the physiological significance of CDS methylation and uncovered non-overlapping function o

132 eting differences between miRNAs in terms of CDS versus 3'-untranslated region targeting, and stable

133 olyadenylation signals leading to the use of CDS protein-coding region poly(A) sites are distinct fro

134 needed to enable and support this vision of CDS-facilitated care optimization, but limited guidance

135 uORFs and CDSes modulate the translation of CDSes.

136 Functional analysis of CDSs specific of the different subclusters suggested pos

137 number variation, absence/introgressions of CDSs and relative polymorphism frequency.

138 pared in patients in whom providers overrode CDS alerts (by performing CT pulmonary angiography in pa

139 ly double those seen when providers overrode CDS alerts.

140 or non-conserved microRNAs, whereas PACCMIT-CDS addresses the lack of similar portals adapted specif

141 d with a cohort of discordant sibling pairs (CDS) to derive a comparative similarity score.

142 pchA cis-interactions by binding to the pchA CDS, thereby unmasking the pchA RBS and promoting PchA e

143 ls of transposition than the native piggyBac CDS, whereas in the absence of induction its activity is

144 ine B-Raf pseudogene Braf-rs1 or its pseudo "CDS" or "3' UTR" develop an aggressive malignancy resemb

145 of the Panel Study of Income Dynamics (PSID-CDS).

146 en at 33.2% (95% CI: 22.4, 46.1) in the PSID-CDS and 26.2% (95% CI: 20.2, 33.2) in the NLSY79-Child.

147 d and 20.6% (95% CI: 16.0, 26.3) in the PSID-CDS.

148 Evidence from high-quality CDS studies is needed to enable and support this vision

149 ercent (6,469 of 9,011) of patients received CDS, and their 30-day postoperative mortality was 10%.

150 slated regions (UTRs) and the coding region (CDS).

151 A residues located within the coding region (CDS).

152 Unexpectedly, removing CDS m(6)A from these transcripts results in a further de

153 ons for designing, conducting, and reporting CDS studies to: 1) ensure that EHR data to inform the CD

154 n the literature for designing and reporting CDS studies.

155 lained by their conserved role in repressing CDS translation.

156 ta=0.24) and communicative diagnostic score (CDS) (beta=0.23).

157 A 2.5-Mb commonly deleted segment (CDS) of chromosome band 7q22 is implicated as harboring

158 tive (PDS) or component direction selective (CDS).

159 MT component direction-selective (CDS) neurons respond to the individual gratings of a pla

160 s carrying a wild-type TP53 coding sequence (CDS) and 1 or more 3'UTR SNVs had a better 5-year surviv

161 ger RNAs (mRNAs) consist of coding sequence (CDS) and 5' and 3' UTRs, typically expected to show simi

162 et gene which truncates the coding sequence (CDS) and targets the corresponding transcript for degrad

163 arative methods can improve coding sequence (CDS) annotation, the SSA method is extended to a Phylo-H

164 Mechanistically, the pchA coding sequence (CDS) base pairs with the 5' untranslated region of the m

165 hat miR-107 paralogs target coding sequence (CDS) of mRNA; (iii) biochemical and computational studie

166 located in the 3'UTR or the coding sequence (CDS) of targets.

167 e increased specificity for coding sequence (CDS) over non-CDS, and increased tolerance to base-subst

168 translated region (3' UTR), coding sequence (CDS) region and 5' untranslated region (5' UTR) of targe

169 translated region (UTR) and coding sequence (CDS) sites exhibiting stronger interaction.

170 rsion of the PB transposase coding sequence (CDS) which provides transposition levels greater than th

171 st mostly downstream of the coding sequence (CDS), indicating the presence of high sequence diversity

172 located within the protein coding sequence (CDS).

173 on structure in the deduced coding sequence (CDS).

174 ntained a highly structured coding sequence (CDS).

175 capacity of a genome [i.e., coding sequence (CDS)] as a reliable measure of proteome diversity, we fi

176 prising 116 098 ordered coding DNA sequence (CDS) gene models.

177 unctional allele with a coding DNA sequence (CDS) of 1521 bp and a promoter adjacent to the CDS.

178 n system to deliver the coding DNA sequence (CDS) of either full-length human dystrophin (DYS: 11.1 k

179 The upstream coding sequences (CDS) present a significant challenge for the translation

180 by cataloging the protein-coding sequences (CDS) present in the metagenome of the community.

181 Protein-coding sequences (CDS) that are contiguous on annotated reference template

182 ection of 82,356 annotated coding sequences (CDS) to identify 2,325 patterns of presence or absence a

183 tic polyadenylation within coding sequences (CDS) triggers ribosome-associated quality control (RQC),

184 tion from long or G+C-rich coding sequences (CDS) without affecting the associated GAL1 promoter acti

185 were annotated into 11,676 coding sequences (CDS), which were manually curated into 116 different pro

186 nd its individual genes or coding sequences (CDS).

187 ontigs, identifying 18,857 coding sequences (CDS).

188 at uORFs are depleted near coding sequences (CDSes) and have initiation contexts that diminish their

189 ed to identify new protein-coding sequences (CDSs) and clarify the boundaries of previously annotated

190 the divergence time in the coding sequences (CDSs) and intergenic sequences (IGSs) is the same betwee

191 by comparing the predicted coding sequences (CDSs) in all 15 loci for synteny and homology.

192 d mRNA ends that mapped to coding sequences (CDSs) of Arabidopsis thaliana, rice (Oryza sativa), and

193 ces revealed 125 predicted coding sequences (CDSs) present in all highly pathogenic strains but absen

194 nd using native microalgal coding sequences (CDSs) with higher GC content improved transgene expressi

195 constrained for SNPs than coding sequences (CDSs), but more constrained than a neutral reference.

196 assumptions about protein-coding sequences (CDSs), e.g., they are conserved, do not overlap, and exc

197 39.3% G+C content, and 925 coding sequences (CDSs).

198 407 kbp, consisting of 485 coding sequences (CDSs).

199 of more than 450 predicted coding sequences (CDSs).

200 ation outside of canonical coding sequences (CDSs).

201 ition to annotated protein coding sequences (CDSs).

202 The shared CDSs represent 36% of all T4 CDSs but only 26% of those

203 total of 27%, 3.2%, and 5.2% of E. sibiricum CDS spotted on the DNA microarray detected differentiall

204 Termed contact-dependent signaling (CDS), this response promotes biofilm formation and other

205 enomenon called contact-dependent signaling (CDS).

206 m(6)A in Snail CDS, but not 3'UTR, triggers polysome-mediated translati

207 that dark causality dominates the sovereign CDS network, indicating interdependencies which require

208 mission of 10,932 novel tissue/cell specific CDS to the NCBI database for this important tick species

209 PNI0373 showed only 15 lineage specific CDS when compared to the pan-genome of pneumococcus.

210 All these GIs had many strain-specific CDS, anomalous GC contents and/or significant dinucleoti

211 ells using cellular dielectric spectroscopy (CDS).

212 TBNA) or conventional diagnosis and staging (CDS), for further investigation and staging.

213 The Cornea Donor Study (CDS) showed that donor age is not a factor in survival o

214 amine release in cocaine-dependent subjects (CDSs).

215 nding sites, clonidine-displacing substance (CDS), has been a major goal for many years.

216 edical fields to be important for successful CDS system implementation are (1) integration into clini

217 in four regions in Brazil: Cachoeira do Sul (CDS), Santo Augusto (STA), Sao Gabriel (SAG), and Vacari

218 mandating use of clinical decision support (CDS) by all providers who order advanced imaging examina

219 g, computer-based clinical decision support (CDS) could unlock widespread benefits from the significa

220 of evidence-based clinical decision support (CDS) for ordering computed tomographic (CT) pulmonary an

221 Clinical decision support (CDS) systems could be used to accomplish this.

222 The cell death suppression (CDS) activity of Adi3 requires phosphorylation by 3-phos

223 er they had primary-cancer-directed surgery (CDS) or no CDS within 4 months of diagnosis.

224 Credit Default Swaps (CDS) spreads should reflect default risk of the underlyi

225 a network of sovereign credit default swaps (CDS).

226 ed indexes of DV, conduct disorder symptoms (CDS), and a composite index of antisocial substance depe

227 atic steatosis (choline deficiency syndrome (CDS)).

228 Canine cognitive impairment syndrome (CDS) represents a group of symptoms related to the aging

229 aryotes, being produced by CDP-DAG synthase (CDS).

230 m of the enzyme CDP-diacylglycerol synthase (CDS), which catalyzes the formation of CDP-diacylglycero

231 Chrysanthemyl diphosphate synthase (CDS), the first enzyme involved in the synthesis of tran

232 ntral role of CDP-diacylglycerol synthetase (CDS) in the regulation of VEGFA signaling and angiogenes

233 by developing a Closed Deliquescence System (CDS) consisting of a mixture of desiccated Martian Regol

234 The shared CDSs represent 36% of all T4 CDSs but only 26% of those from KVP40.

235 Here, we found that CDS m(6)A leads to ribosome pausing in a codon-specific

236 We also found that CDS was characterized by the presence of reactive and se

237 Actually, it has been recognized that CDS spread time series did not anticipate but only follo

238 sis of RNA structural datasets revealed that CDS m(6)A positively regulates translation by resolving

239 The CDS + coaching arm had a smaller magnitude of effect (-0

240 The CDS intervention, implemented after a baseline observati

241 but even among those older than age 75, the CDS rate was 69% (3,378 of 4,909).

242 feasible; 10) think about how to deploy the CDS beyond the original host organization; 11) report th

243 metagenome analysis tools for extracting the CDS content of a microbial community.

244 or and similar recipient criteria as for the CDS PKP procedures, performed by 68 surgeons.

245 to the unrelated unaffected sibling from the CDS and then, sum over such individuals and pairs.

246 T-guided biopsy sample; the patient from the CDS group needed intercostal drainage and was admitted t

247 t of childhood obesity HEDIS measures in the CDS arm (adjusted odds ratio, 2.28 [95% CI, 1.15-4.53])

248 e arm, BMI increased less in children in the CDS arm during 1 year (-0.51 [95% CI, -0.91 to -0.11]).

249 sulted from an insertional disruption in the CDS by Sal-PIF, a 3100-bp PIF/Harbinger-like DNA transpo

250 ial images by the reading center used in the CDS Specular Microscopy Ancillary Study.

251 andomly permuting the affected status in the CDS.

252 ian cell loss after PK by 68 surgeons in the CDS.

253 es to: 1) ensure that EHR data to inform the CDS are available; 2) choose decision rules that are con

254 t the right users and workflows; 4) make the CDS easy to access and use; 5) minimize the burden place

255 o the affected than to the unaffected of the CDS (P value approximately 0.00001).

256 ith increased structure just upstream of the CDS being the major limiting factor in translation, whic

257 es represented 10.6%, 0.89%, and 2.3% of the CDS differentially expressed when grown at -2.5 degrees

258 structure, we find that the structure of the CDS regulates protein expression through changes in func

259 us positions of the 5'-terminal 30 nt of the CDS: in mammalian mRNAs, the 5'-UTR shows a greater cons

260 5'-UTR shows a greater conservation than the CDS, whereas the opposite trend holds for yeast mRNAs.

261 rt of clinician workflow; 7) ensure that the CDS rules are adequately tested; 8) select meaningful ev

262 other Vps factors can be cross-linked to the CDS of genes induced by Gcn4 or Gal4 in a manner depende

263 NAs plays a role in guiding miR-103/7 to the CDS of targets; and (iv) there are major sequence-specif

264 tionship to whether patients belonged to the CDS or no CDS group.

265 statistical models applied previously to the CDS PK data were considered.

266 S) of 1521 bp and a promoter adjacent to the CDS.

267 r, intercistronic distance, overlap with the CDS, and initiation context most strongly influence tran

268 shift from a common to rare codon within the CDS facilitated miRNA binding downstream of the SNP, dra

269 considered early/late); (v) 133 (36%) of the CDSs were expressed after virus DNA synthesis begins (co

270 le in the laboratory; (iii) 227 (62%) of the CDSs were expressed before virus DNA synthesis begins; (

271 While 65% of the CDSs were unique to KVP40 and had no known function, the

272 ore extreme conservation patterns than their CDSs.

273 A great proportion (49.1%) of these CDSs are organized into paralogous gene families, which

274 ents to treatment: 66 to EBUS-TBNA and 67 to CDS (one later withdrew consent).

275 der for pulmonary angiography was exposed to CDS on the basis of the Wells criteria.

276 prokaryotes clearly evolve in proportion to CDS values, whereas those of eukaryotes are more randoml

277 In 5 practices randomized to CDS + coaching, decision support was augmented by indivi

278 In 5 practices randomized to CDS, pediatric clinicians received decision support on o

279 ces; many genes show high 3' UTR relative to CDS, others show high CDS to 3' UTR.

280 r period were positive for PE; subsequent to CDS implementation, yield by quarter increased 69.0%, fr

281 rained for indels than for SNPs, compared to CDSs.

282 f 19 genes) shows a broad range of 3' UTR-to-CDS expression ratios across neurons and tissues.

283 Further, for two genes examined, a 3' UTR-to-CDS ratio above a particular threshold in any given neur

284 ed poorer OS for patients with a mutant TP53 CDS.

285 reduce NuA4 occupancy in certain transcribed CDS.

286 e that the sequence composition of transgene CDSs can directly impact silencing, providing design str

287 sequencing reads for which we know the true CDS content of the underlying communities, to determine

288 ival rate than patients carrying a wild-type CDS and the reference 3'UTR, yet there is no statistical

289 cloetide (nt) sequence overlapping the UL136 CDS supported expression of a downstream cistron in a bi

290 included several previously uncharacterized CDSs that may encode novel virulence determinants includ

291 oeconomic status were less likely to undergo CDS; but even among those older than age 75, the CDS rat

292 f the mRNA [5'-untranslated region (5'-UTR), CDS and 3'-UTR] preferentially fold onto themselves, whi

293 and for all three regions of a mRNA (3' UTR, CDS and 5' UTR), STarMir output includes the computed bi

294 es introns into three groups, such as 5'UTR, CDS and 3'UTR and subsequently divides CDS introns into

295 y characteristics and features of the 5'UTR, CDS and 3'UTR.

296 Loss of CDS2, 1 of 2 vertebrate CDS enzymes, results in vascular-specific defects in zeb

297 (s), a Gene Transfer Format (.GTF) file with CDS information and a SNP report(s) in an increasing sel

298 The median survival for patients with CDS QOL was 1.6 years versus 5.6 years for patients with

299 EBUS-TBNA (14 days; 95% CI 14-15) than with CDS (29 days; 23-35) resulting in a hazard ratio of 1.98

300 nal by complementation of a non-viable yeast CDS null strain and that it is essential in the bloodstr