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1                                              CERT downregulation in breast cancer cells enhanced ErbB
2                                              CERT has a C-terminal START domain for ceramide binding
3                                              CERT mediates nonvesicular Cer transport from the endopl
4                                              CERT mutant embryos accumulate ceramide in the ER but al
5                                              CERT transfers ceramide from the endoplasmic reticulum t
6                                              CERT's N-terminal pleckstrin homology (PH) domain target
7                  However, yeast cells lack a CERT homolog, and the mechanism of ER to Golgi ceramide
8 affinity and ceramide transfer activity of a CERT-serine-rich phosphorylation mimic.
9  We propose that ceramide transfer via VAP-A-CERT linkages drives the biogenesis of a select RNA-cont
10           In spite of ceramide accumulation, CERT mutant mice do not die as a result of enhanced apop
11  of pUL21 that lacks the ability to activate CERT.
12 t pUL21-mediated dephosphorylation activates CERT and accelerates Cer-to-SM conversion.
13 inferred from ectopic expression of IncD and CERT in the host cell.
14 CV secretion/release by attenuating OSBP and CERT functions by phosphorylation inhibition.
15 ed in lipid transfer and metabolism, such as CERT.
16                                         Both CERT isoforms, when immobilized, were found to bind the
17          PKD attenuates the function of both CERT and OSBP by phosphorylation at their respective Ser
18 al levels of ceramide phosphoethanolamine by CERT in vivo is required to prevent oxidative damages to
19  PtdIns(4)P binding and ceramide transfer by CERT.
20 rs of University of California, Riverside/CE-CERT environmental chamber data on 17 aromatic hydrocarb
21 or Environmental Research and Technology (CE-CERT) Atmospheric Processes Lab using two biomass fuel s
22                                    Combining CERT inhibitor with FLT3 inhibitor exhibits synergistic
23 after irradiation, consistent with decreased CERT function, whereas the CERT inhibitor N-(3-hydroxy-1
24 tation on the surface of pUL21 that disrupts CERT binding in vitro and in cultured cells.
25             Here, we show that knocking down CERT inhibits the growth and promotes the apoptosis of A
26 nalyze a human behavior-based dataset (i.e., CERT) that reports users' Information Technology (IT) ac
27  Implantable Cardioverter Defibrillators (EU-CERT-ICD), a prospective, investigator-initiated, non-ra
28 lanogaster mutant flies lacking a functional CERT (Dcert) protein using chemical mutagenesis and a We
29 e in vivo consequences of loss of functional CERT protein.
30 xplored the function of Gpbp and GpbpDelta26/CERT during embryogenesis in zebrafish.
31 type is mediated by Gpbp but not GpbpDelta26/CERT, suggesting that Gpbp is an important factor for no
32 olesterol and cholesterol depletion, impairs CERT Golgi localization, and promotes Golgi fragmentatio
33  at the plasma membrane, thereby implicating CERT loss in the progression of TNBCs.
34 integrity caused by ceramide accumulation in CERT mutant mice primarily affects organogenesis rather
35 ing UVB irradiation, revealing that inactive CERT can attenuate a key metabolic protective mechanism
36 trachomatis at that time, this model of IncD-CERT interaction was inferred from ectopic expression of
37 B, and we determined that, at the inclusion, CERT-VAPB interaction relied on the FFAT domain of CERT.
38                            Thus, UVB-induced CERT homotrimer formation accounts, at least in part, fo
39               Purified pUL21 and full-length CERT interact with submicromolar affinity, and we solve
40 dence that by regulating cellular SM levels, CERT determines the signaling output of the EGF receptor
41 ot significantly altered when pUL21-mediated CERT dephosphorylation is abolished.
42 ression of nonphosphorylatable serine mutant CERT S132A or OSBP S240A.
43 nal filling of multivesicular bodies (MVBs), CERT localizes to MVBs, and the ceramide-generating enzy
44 at the massive IncD-dependent association of CERT with the inclusion led to an increased recruitment
45 VB-treated keratinocytes, HPA-12 blockade of CERT function increased keratinocyte apoptosis, decrease
46 APB interaction relied on the FFAT domain of CERT.
47 protein CERT that relied on the PH domain of CERT.
48  this article, we report a novel function of CERT in the innate immune response.
49 mutations increase the Golgi localization of CERT inside the cell, consistent with enhanced PtdIns(4)
50 nt structural insight into the regulation of CERT function and localization.
51  4-kinases are involved in the regulation of CERT-mediated ceramide transport.
52 e function of ceramide transport protein (or CERT) required for sphingomyelin synthesis occur(s) in U
53 TGoCS, such as the ceramide transfer protein CERT and several members of the oxysterol binding protei
54 he recruitment of the lipid transfer protein CERT and the ER-resident protein VAPB to the inclusion.
55  expression of the ceramide transfer protein CERT is reduced in TNBCs.
56  the VAP-A-binding ceramide transfer protein CERT led to similar defects in EV RNA content.
57 ve recruitment of the lipid transfer protein CERT that relied on the PH domain of CERT.
58 he recruitment of the lipid transfer protein CERT to the inclusion.
59 se IIIbeta and the ceramide transfer protein CERT, mediate PKD signaling to influence vesicle traffic
60 he cellular ceramide (Cer) transport protein CERT.
61                   Ceramide transfer protein (CERT) and oxysterol-binding protein (OSBP) play a crucia
62                   Ceramide Transfer Protein (CERT) determines the ratio of ceramide and sphingomyelin
63 nt recruitment of ceramide transfer protein (CERT) for sphingomyelin synthesis.
64                   Ceramide transfer protein (CERT) functions in the transfer of ceramide from the end
65                   Ceramide transfer protein (CERT) transfers ceramide from the endoplasmic reticulum
66 ltidomain protein ceramide transfer protein (CERT).
67 by the essential ceramide transport protein (CERT) in mammalian cells.
68            The ceramide transporter protein (CERT) and its longer splicing isoform CERTL are known to
69 cently identified ceramide transfer protein, CERT, is responsible for the bulk of ceramide transport
70 also function as ceramide transfer proteins (CERT).
71 inding activity was decreased in recombinant CERT proteins containing the UVB-induced homotrimer.
72 also induced the rapid formation of a stable CERT homotrimer complex in keratinocytes as determined b
73                                    Targeting CERT sensitizes solid cancer cells to chemotherapy.
74                   However, whether targeting CERT to induce ceramide accumulation thereby improving A
75                  In this study, we show that CERT is an essential gene for mouse development and embr
76 ane sensing process are conserved across the CERT, OSBP and FAPP family.
77              The middle region domain of the CERT protein was required for the homotrimer formation,
78  part of ceramide in the START domain of the CERT protein.
79                                Tested on the CERT insider threat dataset, the DS-IID achieved 97% acc
80 nt with decreased CERT function, whereas the CERT inhibitor N-(3-hydroxy-1-hydroxymethyl-3-phenylprop
81  pUL21 C-terminal domain in complex with the CERT Pleckstrin homology and steroidogenic acute regulat