ライフサイエンスコーパス: CLA

1 CLA and CNA significantly reduced body weight and fat ma

2 CLA and CNA significantly reduced serum leptin and tumou

3 CLA and OCS act non-additively to activate atNHE1, indic

4 CLA attenuated the increase in BMI (0.5 +/- 0.8) compare

5 CLA exerts its anti-carcinogenic effect by reducing VEGF

6 CLA resistance and the polymorphisms of CYP2C19 were det

7 CLA significantly increased bone markers without major c

8 CLA supplementation for 7 +/- 0.5 mo decreased body fatn

9 CLA supplementation significantly prevented ovariectomy-

10 CLA treatment increased serum parathyroid hormone (PTH)

11 CLA was microencapsulated by spray drying in ten varied

12 CLA(+) is expressed on the surface of circulating CD45RO

13 CLA(+) TH2 T cells were markedly expanded in both childr

14 CLA+ and alpha4beta7+ memory T cells were isolated and c

15 CLA-rich eggs and soy control eggs were produced by addi

16 CLA-yolk mayonnaise was more viscous, had greater storag

17 CLA/ESL mediates adhesion of T cells to inflamed vascula

18 CLA:M:PPC (1:1:3) microparticles demonstrated better mor

19 CLA:M:PPC (1:1:3) showed the most promising results, thu

20 bo: 0.033 +/- 0.003; Vit D: 0.034 +/- 0.002; CLA: 0.029 +/- 0.005; Vit D + CLA: 0.038 +/- 0.005 %.h-1

21 had increased CLA(+) /CLA(-) Th2 (P < .007), CLA(+) Tc2 (P = .04), and CLA(+) Th22 (P < .05) frequenc

22 e members of the SR-B family, namely, CLA-1, CLA-2, and CD36, mediate recognition of bacteria not onl

23 soy control eggs were produced by adding 10% CLA-rich soy oil or 10% of control unmodified soy oil to

24 nd 36 +/- 11; Vit D, 38 +/- 8 and 34 +/- 10; CLA, 50 +/- 14 and 51 +/- 16; Vit D + CLA, 29 +/- 15 and

25 from mountain areas showed average c-9, t-11 CLA content higher than those from prairie districts.

26 ilk yogurts showed lower values of c-9, t-11 CLA content on lipid basis compared to full-fat yogurts.

27 erse association between the cis-9, trans-11 CLA in adipose tissue and diabetes risk is consistent wi

28 The cis-9, trans-11 CLA isomer was associated with a lower risk of diabetes.

29 determine the association between the 9c,11t-CLA isomer in adipose tissue and risk of MI.

30 Adipose tissue 9c,11t-CLA was associated with a lower risk of MI in basic and

31 9c,11t-CLA, which is present in meaningful amounts in the milk

32 The trans-10, cis-12 CLA isomer was not detected in adipose tissue.

33 The cis-9, trans-11 and trans-10, cis-12 CLA isomers in adipose tissue and 48 other fatty acids w

34 nflammatory proteins were increased by 10,12-CLA compared with bovine serum albumin vehicle in the ad

35 ns-10,cis-12-conjugated linoleic acid (10,12-CLA) activate the inflammatory signaling that promotes i

36 10,12-CLA-induced IL-8, IL-6, IL-1beta, and COX-2 mRNA levels

37 ized that they played the same role in 10,12-CLA-mediated inflammation.

38 Consistent with these data, 10,12-CLA-mediated secretions of IL-8 and IL-6 from AD50 cultu

39 d the inflammatory capacity of CM from 10,12-CLA-treated cultures.

40 te supplementation in rodents elevates NO(2)-CLA levels in plasma, urine, and tissues, which in turn

41 se to 100% molar yield of modified PC (72.3% CLA) with Duolite-Lecitase in 24 h, the highest yield re

42 almost quantitative yields of PC with 74.4% CLA content has been obtained using OM-Lecitase.

43 rom feeding rape (+43% MUFA, +10% PUFA, +40% CLA), combining to produce milk 16% lower SFA and higher

44 erexpression abrogated cell surface HECA-452/CLA expression, reduced the number of rolling leukocytes

45 specially formulated diet consisting of 0.5% CLA for 24 days.

46 re randomly assigned to receive 3 g/d of 80% CLA (50:50 cis-9,trans-11 and trans-10,cis-12 isomers) o

47 Our results define a CLA long-range inhibitory circuit (CLA-SOM inhibitory pr

48 with the phosphatase inhibitor calyculin-A (CLA) increase Na(+) transport capacity without affecting

49 gher percentage of conjugated linoleic acid (CLA(cis-9, trans-11)) as well as higher percentages of b

50 Conjugated linoleic acid (CLA) and conjugated nonadecadienoic acid (CNA) have been

51 imental studies on conjugated linoleic acid (CLA) and insulin regulation suggested that CLA could be

52 of 2 dietary oils, conjugated linoleic acid (CLA) and safflower oil (SAF), on body weight and composi

53 ch in trans, trans conjugated linoleic acid (CLA) are significantly more viscous, have more phospholi

54 Conjugated linoleic acid (CLA) content of ruminant milk reported in published rese

55 n for the elderly; conjugated linoleic acid (CLA) has been shown to improve overall bone mass when ca

56 Conjugated linoleic acid (CLA) has the unique property of inducing regression of p

57 tes the effects of conjugated linoleic acid (CLA) in preventing bone loss, using an ovariectomised mo

58 Many studies with conjugated linoleic acid (CLA) indicate that it has a protective effect against ma

59 Conjugated linoleic acid (CLA) is a family of positional and geometric isomers wit

60 Conjugated linoleic acid (CLA) is a supplemental dietary fatty acid that decreases

61 Conjugated Linoleic Acid (CLA) is fatty acid found endogenously in food sources th

62 Herein, conjugated linoleic acid (CLA) is identified as the primary endogenous substrate f

63 GC-FID), including conjugated linoleic acid (CLA) isomeric profile (Ag(+)-HPLC), and nutritional valu

64 identification of conjugated linoleic acid (CLA) isomers has been developed in which silver ion liqu

65 e development of a conjugated linoleic acid (CLA) oil-in-water beverage emulsion containing acacia gu

66 n the synthesis of conjugated linoleic acid (CLA) partial glycerides, which presented nutraceutical p

67 sity is to consume conjugated linoleic acid (CLA) supplements containing isomers cis-9, trans-11 and

68 n omega-3 and +12% conjugated linoleic acid (CLA)) appear complementary to those from feeding rape (+

69 aturated FA (UFA), conjugated linoleic acid (CLA), n-3 FA, and C18:1cis9 to C16 ratio.

70 ty acids including conjugated linoleic acid (CLA), polyunsaturated fatty acids C18:2(n-6) and C18:3(n

71 ration products of conjugated linoleic acid (CLA).

72 the main source of conjugated linoleic acid (CLA; 18:2n-7t), which is produced by the ruminal biohydr

73 pids (PL) rich in conjugated linolenic acid (CLA) have important health effects.

74 id, docosapentaenoic acid, arachidonic acid, CLA:9c11t and gamma linolenic acid.

75 g more n-3 FA and conjugated linoleic acids (CLA) than conventionally produced dairy products.

76 oth vitamin D and conjugated linoleic acids (CLAs) stimulate muscle protein synthesis.

77 either a study of skin lesions or activated CLA(+) T-cell subsets in peripheral blood.

78 ho were receiving either 4 g/d of 78% active CLA isomers (3.2 g/d: 39.2% cis-9,trans-11 and 38.5% tra

79 Chaperone-like activity (CLA) was evaluated by measuring the ability of alpha-cry

80 s, indicating BX-independent defense against CLA.

81 e not only the CLnA concentrations, but also CLA in egg-yolk lipids.

82 Although CLA(+) T(H)1 frequencies were significantly lower in inf

83 .001]), with less significant effects among CLA(+) T cells (IL-22: 11 vs 7.5, P = .04).

84 uencies, which were highly significant among CLA(-) cells (IL-22: 3.7 vs 1.7 [P < .001] and IL-17: 1.

85 group than in the PCV group (P = .014); and CLA was expressed more frequently in the pneumonia group

86 -) Th2 (P < .007), CLA(+) Tc2 (P = .04), and CLA(+) Th22 (P < .05) frequencies than controls.

87 SFA and higher in MUFA (43%), PUFA (55%) and CLA (59%).

88 gut (alpha4beta7, CCR6) and skin (CCR10 and CLA).

89 d the skin-homing receptors CCR4, CCR10, and CLA and migrated in response to CCL17/CCL27.

90 n of other skin addressins (CCR6, CCR10, and CLA).

91 tin and CD27 but strongly expressed CCR4 and CLA, a phenotype suggestive of skin resident effector me

92 d CD8(+) T cells were compared in CLA(-) and CLA(+) populations.

93 g CLA per day; 3) both Vit D (2000 IU/d) and CLA (4000 mg/d); or 4) placebo for 8 wk.

94 and the synergistic interactions of OPDA and CLA feeding significantly induced the expression of the

95 aneous lymphocyte antigen (CLA)-positive and CLA(-) T-cell subsets in patients with AD and control su

96 Cutaneous lymphocyte-associated antigen (CLA(+) ) T cells are specialized for skin homing and rep

97 hat cutaneous lymphocyte-associated antigen (CLA), a functional E-selectin ligand (ESL), is selective

98 oming markers, cutaneous lymphocyte antigen (CLA) and alpha4beta7 integrin, are used to determine whe

99 expression of cutaneous lymphocyte antigen (CLA) in ILCs.

100 -selectin, and cutaneous lymphocyte antigen (CLA) on S. pneumoniae-specific plasmablasts was examined

101 s positive for cutaneous lymphocyte antigen (CLA) were increased fourfold in all CD4+ and CD8+ T cell

102 T-cell, cutaneous lymphocyte antigen (CLA)(+) and CCR4(+) T-cell and cytokine responses were s

103 rities between cutaneous lymphocyte antigen (CLA)(+) polarized T-cell subsets in children versus adul

104 od skin-homing/cutaneous lymphocyte antigen (CLA)(+) T cells emerge as biomarkers of cutaneous immune

105 contained few cutaneous lymphocyte antigen (CLA)(+) T cells, the cell type thought to provide cutane

106 in skin-homing/cutaneous lymphocyte antigen (CLA)(+) versus systemic/CLA(-) T cells.

107 CXCR3, but not cutaneous lymphocyte antigen (CLA), on circulating T cell subsets was associated with

108 ubsets in both cutaneous lymphocyte antigen (CLA)-positive and CLA(-) T-cell subsets in patients with

109 The corn leaf aphid (CLA; Rhopalosiphum maidis) is a phloem sap-sucking insec

110 mir1-mediated resistance to corn leaf aphid (CLA; Rhopalosiphum maidis), a phloem sap-sucking insect

111 yvitamin D3 concentration was not changed by CLA.

112 Furthermore, feeding by CLA on Mp708 plants enhanced callose deposition, a poten

113 Furthermore, foliar feeding by CLA rapidly sends defensive signal(s) to the roots that

114 Feeding by CLA triggers the rapid accumulation of mir1 transcripts

115 corporating polarized Th1/Th17 cells or CCR6+CLA+ T cells derived from psoriasis patients into the co

116 We report circulating skin-homing CD22+CLA+B cells in healthy volunteers and melanoma patients

117 define a CLA long-range inhibitory circuit (CLA-SOM inhibitory projections -> LA principal neurons)

118 Circulating CLA(+) T cells may be a reliable surrogate marker of the

119 PTDM (N = 24), the frequency of circulating CLA(+) (skin-homing) Tregs was decreased (1.53% vs 3.99%

120 e latest advancements reached on circulating CLA(+) in AD and the great potential they harbor in unde

121 Isomerization of cis,trans and trans,cis CLA to trans,trans isomers was observed mainly for the m

122 AA patients had increased CLA(+) /CLA(-) Th2 (P < .007), CLA(+) Tc2 (P = .04), and CLA(+)

123 ents with Vitiligo showed the highest CLA(+)/CLA(-) T(H)1/type 1 cytotoxic T-cell polarization, with

124 itors (PPI), amoxicillin and clarithromycin (CLA) has been the standard in Latin America.

125 However, the function of the claustrum (CLA) and its neural projections remains largely unknown.

126 the analysis of CLA isomers in a commercial CLA supplement, milk fat, and the lipid extract from a L

127 single-isomer studies, and results comparing CLA isomers were inconclusive.

128 due to inflammatory signaling and considers CLA's linkage with lipogenesis, lipolysis, thermogenesis

129 The mobile phase containing CLA isomers eluting from the Ag(+)-LC column flows throu

130 , neither Vit D nor CLA nor combined Vit D + CLA supplementation affected the basal myofibrillar prot

131 /- 10; CLA, 50 +/- 14 and 51 +/- 16; Vit D + CLA, 29 +/- 15 and 35 +/- 8).

132 034 +/- 0.002; CLA: 0.029 +/- 0.005; Vit D + CLA: 0.038 +/- 0.005 %.h-1), and hyperinsulinemia-hypera

133 - 0.006%/h; CLA: 0.039 +/- 0.006%/h; Vit D + CLA: 0.040 +/- 0.007%/h) or the hyperinsulinemia-hyperam

134 djustment to the median dose of 3.2 g CLA/d, CLA was effective and produced a reduction in fat mass f

135 001) CONCLUSION: Given at a dose of 3.2 g/d, CLA produces a modest loss in body fat in humans.

136 The aim of this study was to determine CLA's efficacy with regard to change in fat and body mas

137 Dietary CLA and nitrite supplementation in rodents elevates NO(2

138 intravital microscopy, we show that, during CLA-induced regression of pre-established atherosclerosi

139 direct toxic effect, contributes to enhanced CLA resistance in Mp708.

140 was moderate, and the proportion expressing CLA was low.

141 nzymatic and cellular mechanisms account for CLA nitration, including reactions catalyzed by mitochon

142 esented describe a novel functional role for CLA in the regulation of monocyte adhesion, polarization

143 itioned medium (CM) model, CM collected from CLA-treated AD50 but not AD0 cultures induced IL-8 and I

144 After adjustment to the median dose of 3.2 g CLA/d, CLA was effective and produced a reduction in fat

145 y 0.128-1.501, 0.405-1.250 and 0.433-0.976 g CLA/100 g fat.

146 loss compared with placebo was -0.024 kg x g CLA(-1) x wk(-1) (P=0.03).

147 P-selectin glycoprotein ligand-1 glycoform "CLA," and CD43.

148 ine secretion in the order of CD36 > CLA-2 > CLA-1 in HEK293 cells.

149 of cytokine secretion in the order of CD36 > CLA-2 > CLA-1 in HEK293 cells.

150 040 +/- 0.004%/h; Vit D: 0.044 +/- 0.006%/h; CLA: 0.039 +/- 0.006%/h; Vit D + CLA: 0.040 +/- 0.007%/h

151 enhanced callose accumulation and heightened CLA resistance.

152 After only 3h, OM-RML gave the highest CLA conversion (54% at 40 degrees C with 1:3M ratio of g

153 Patients with Vitiligo showed the highest CLA(+)/CLA(-) T(H)1/type 1 cytotoxic T-cell polarization

154 nt groups and Th2 skewing in the skin-homing CLA+ cells of peanut allergic patients.

155 roliferation predominates in the skin-homing CLA+ subset, whilst peanut-tolerant groups have a mixed

156 aimed to compare frequencies of skin homing (CLA(+) ) vs systemic (CLA(-) ) "polar" CD4(+) and CD8(+)

157 tion markers and frequencies of skin-homing (CLA(+)) versus systemic (CLA(-)) "polar" CD4 and CD8 T-c

158 okine production by circulating skin-homing (CLA(+)) versus systemic (CLA(-)) "polar" CD4(+)/CD8(+) r

159 However, CLA resistance is a rising problem affecting eradication

160 etabolites were not significantly altered in CLA-infested Mp708 plants, indicating BX-independent def

161 g CD4(+) and CD8(+) T cells were compared in CLA(-) and CLA(+) populations.

162 those in control subjects, but decreases in CLA(+) TH1 T-cell numbers were greater in children with

163 ke in adults, no imbalances were detected in CLA(-) T cells from pediatric patients with AD nor were

164 ol subjects, with significant differences in CLA(+) T-cell numbers (P < .01).

165 with AD, with no significant differences in CLA(-) T-cell numbers.

166 of cortical long-range GABAergic neurons in CLA communication.SIGNIFICANCE STATEMENT It is very well

167 sn-1 mono and sn-1,3 diacylglycerols rich in CLA, with a ratio of sn-1,3/sn-1,2 regioisomers of 21.8,

168 AA patients had increased CLA(+) /CLA(-) Th2 (P < .007), CLA(+) Tc2 (P = .04), and

169 ly, use of bifidobacteria slightly increased CLA relative content in the conventional fermented milks

170 After infancy, CLA(-) T(H)2 frequencies were increased in patients with

171 oxidative stability of conjugated linoleic (CLA) and linoleic (LA) acids in different chemical forms

172 g cis-9 trans-11, C18:2 conjugated linoleic (CLA-1.4 times), and alpha-linolenic acids (ALA-1.6 times

173 We recruited 23 subjects from our main CLA efficacy study who were receiving either 4 g/d of 78

174 d were analyzed to estimate the overall mean CLA value.

175 Meanwhile, CLA significantly reduced femur tartrate resistant acid

176 the sympathetic nervous system in mediating CLA's antiobesity properties.

177 0 IU vitamin D-3 (Vit D) per day; 2) 4000 mg CLA per day; 3) both Vit D (2000 IU/d) and CLA (4000 mg/

178 , whilst peanut-tolerant groups have a mixed CLA/alpha4beta7 response (P = 0.008).

179 In model 1, a differentiation model, CLA activation of MAPK and induction of interleukin-8 (I

180 Pasture-grazing dairy cows have more CLA in their milk than do grain-fed cows.

181 at three members of the SR-B family, namely, CLA-1, CLA-2, and CD36, mediate recognition of bacteria

182 rtico-lateral-amygdala somatostatin neurons (CLA-SOM), has a direct inhibitory influence on the outpu

183 Nitro-conjugated linoleic acid (NO2-CLA) is preferentially formed, constitutes the most abun

184 y, human serum albumin was found to bind NO2-CLA both non-covalently and to form covalent adducts at

185 presence of two electrophilic centers in NO2-CLA located on the beta- and delta-carbons with respect

186 new insights into the chemical basis of NO2-CLA signaling actions.

187 this work, we examined the reactions of NO2-CLA with low molecular weight thiols (glutathione, cyste

188 pwise mechanisms with thiolate attack on NO2-CLA as rate-controlling step.

189 Compared with placebo, neither Vit D nor CLA nor combined Vit D + CLA supplementation affected th

190 Although the ability of CLA to inhibit angiogenesis in the peripheral nervous sy

191 n (+/-SD) percentage of total fatty acids of CLA for the cis-9, trans-11 isomer in adipose tissue was

192 the study was to test whether the amount of CLA in adipose tissue is associated with risk of diabete

193 /O3-MS method was applied to the analysis of CLA isomers in a commercial CLA supplement, milk fat, an

194 A meta-analysis of CLA resistance studies among adult residents in Santiago

195 However, causality of CLA-mediated responses to body fat loss, particularly th

196 tives were (1) compare the FA composition of CLA-rich yolk granules and plasma, relative to standard

197 evalence of CLA resistance, consideration of CLA free therapies in Santiago is warranted.

198 ariation of pooled estimated mean content of CLA in milk among the study regions and were highest in

199 The contents of CLA and n-3 FA in a serving of whole milk (3.25% fat) in

200 Nitroalkene derivatives of CLA and their metabolites are detected in the plasma of

201 sed for the direct and fast determination of CLA isomers at low concentrations and in complex lipid m

202 The effect of CLA in lowering BMI was detected during the last 8 wk of

203 to demonstrate the anti-angiogenic effect of CLA in the brain, and suggests that CLA be explored as a

204 However, potential effects of CLA and calcium on bone mass during a period of bone los

205 l models have reported beneficial effects of CLA on atherosclerosis.

206 investigation of the safety and efficacy of CLA supplementation in children is recommended.

207 ing in the skin, and thus, the evaluation of CLA(+) T cells in the blood may eliminate the need for s

208 Except of CLA(-) Tc1 cells (P = .03), IFN-gamma levels were mostly

209 The expansion of CLA-expressing effector memory CD8+ T cells in response

210 s was observed mainly for the methyl form of CLA.

211 all older patients (P < .01), frequencies of CLA(+) T(H)2 T cells were similarly expanded across all

212 A significantly lower frequency of CLA(+) IFN-gamma-producing cells was observed in patient

213 In this way, de novo identification of CLA positional isomers, i.e. without requiring compariso

214 Proposed antiobesity mechanisms of CLA include regulation of (a) adipogenesis, (b) lipid me

215 d n = 194) yielded to a pooled prevalence of CLA resistance of 31.3% (95% CI 23.9-38.7).

216 adication rate was 63% and the prevalence of CLA resistance was 26%.

217 Given the high pooled prevalence of CLA resistance, consideration of CLA free therapies in S

218 had higher Bet v 1-specific proliferation of CLA(+) and CCR4(+) T cells compared with patients with b

219 ility (CLA isomer profile, quantification of CLA and volatile compounds by SPME coupled with CG-MS) d

220 E-selectin on tumor vessels, recruitment of CLA(+) CD8(+) T cells, and histological evidence of tumo

221 In contrast to the role of CLA-PFC pathway in selectively regulating impulsivity, a

222 ts are the most important dietary sources of CLA, we have investigated the CLA concentrations and add

223 did not influence the oxidative stability of CLA, however its presence improved physical-chemical cha

224 gs and (3) compare the emulsion stability of CLA-yolk mayonnaise.

225 d, double-blind, placebo-controlled trial of CLA in 62 prepubertal children aged 6-10 y who were over

226 CELL." E-Ig reactivity was most prominent on CLA in mouse cells and on HCELL in human cells.

227 Multivariable analyses showed that only CLA resistance was significantly and inversely associate

228 uencies of TH22 T cells within the CLA(+) or CLA(-) compartments.

229 l women with type 2 diabetes received SAF or CLA (8 g oil/d) during two 16-wk diet periods separated

230 In only 2 h, with a 1/12 PC/CLA molar ratio at 50 degrees C, similar almost quantita

231 < .0001), and frequencies of IL-13-producing CLA(+) cells were also correlated with IgE levels and SC

232 gC tagged alphaB-crystallin displayed robust CLA.

233 here were no differences in Bet v 1-specific CLA(+) and CCR4(+) proliferation and cytokine secretion

234 state of PA correlates with peanut-specific CLA responses, with tolerance associated with predominan

235 cal characteristics and oxidative stability (CLA isomer profile, quantification of CLA and volatile c

236 aled that it is possible to produce a stable CLA oil-in-water emulsion for using in beverages.

237 cts of the addition of CPP on the structure, CLA, and cell transduction properties of alphaB-crystall

238 ies of skin-homing (CLA(+)) versus systemic (CLA(-)) "polar" CD4 and CD8 T-cell subsets in patients w

239 lating skin-homing (CLA(+)) versus systemic (CLA(-)) "polar" CD4(+)/CD8(+) ratio and activated T-cell

240 encies of skin homing (CLA(+) ) vs systemic (CLA(-) ) "polar" CD4(+) and CD8(+) and activated T-cell

241 lymphocyte antigen (CLA)(+) versus systemic/CLA(-) T cells.

242 c9,t11-CLA lowered triacylglycerol (P </= 0.01) and had no effe

243 and cell studies suggest that VA and c9,t11-CLA may be hypocholesterolemic and antiatherogenic, epid

244 -9,trans-11 conjugated linoleic acid (c9,t11-CLA), are less clear.

245 We determined the effects of VA, c9,t11-CLA, and iTFA, in the context of highly controlled diets

246 , with the requirement of VA, but not c9,t11-CLA, to be listed under TFA on the Nutrition Facts Panel

247 y 3% VA, approximately 3% iTFA, or 1% c9,t11-CLA.

248 g two months of storage at room temperature, CLA:PPC (1:4) was selected for comparisons.

249 In this study, we demonstrate that CLA inhibits CXCR4 expression, resulting in a failure of

250 tic-adhesion assay, we provide evidence that CLA prevents monocytes from binding to ICAM-1 and subseq

251 risk is consistent with the hypothesis that CLA may be involved in insulin regulation.

252 Our results indicate that CLA administration significantly reduces angiogenesis in

253 We show that CLA inhibits human peripheral blood monocyte cell adhesi

254 Our study shows that CLA resistance is associated with failure of H. pylori e

255 These data suggest that CLA instigates the release of inflammatory signals from

256 The data suggest that CLA, along with dietary calcium, has great potential to

257 (CLA) and insulin regulation suggested that CLA could be associated with risk of diabetes, but epide

258 ffect of CLA in the brain, and suggests that CLA be explored as a therapeutic treatment for cancer an

259 The CLA content of ruminant milk samples was grouped accordi

260 The CLA(+) TH1/TH2 and TC1/TC2 ratio was highly imbalanced i

261 The CLA, vaccenic acid, C18:39c12c15c, total C18:1 trans and

262 and produced a reduction in fat mass for the CLA group alone (0.05 +/- 0.05 kg/wk; P<0.001) and for t

263 e (0.05 +/- 0.05 kg/wk; P<0.001) and for the CLA group compared with placebo (0.09 +/- 0.08 kg/wk; P<

264 the role of the neural projections from the CLA to the PFC in regulating impulsivity in male rats.

265 We first identified the CLA-PFC pathway by retrograde tracer and virus expressio

266 al body weight was smaller (P = 0.02) in the CLA group (-0.09 +/- 0.9%) than in the placebo group (0.

267 bsorptiometry was smaller (P = 0.001) in the CLA group (-0.5 +/- 2.1%) than in the placebo group (1.3

268 creased significantly more (P = 0.05) in the CLA group (-5.1 +/- 7.3 mg/dL) than in the placebo group

269 ineral accretion was lower (P = 0.04) in the CLA group (0.05 +/- 0.03 kg) than in the placebo group (

270 subjects completed the trial (n = 28 in the CLA group, n = 25 in the placebo group).

271 factors have been reported to influence the CLA content of milk, the effect of the "geographical ori

272 ary sources of CLA, we have investigated the CLA concentrations and additionally the fatty acid profi

273 The physical-chemical properties of the CLA microparticles were characterised by core retention,

274 The predominance of the CLA+ response to peanut in peanut allergic patients is c

275 hemogenetic activation and inhibition of the CLA-PFC pathway increased and reduced the impulsive-like

276 Furthermore, chemogenetic inhibition of the CLA-PFC pathway prevented methamphetamine-induced impuls

277 hemogenetic activation and inhibition of the CLA-PFC pathway reduced and increased overall activity o

278 These results indicate that the CLA-PFC pathway is essential for impulsivity.

279 tered frequencies of TH22 T cells within the CLA(+) or CLA(-) compartments.

280 somers, i.e. without requiring comparison to CLA standards, was achieved.

281 40 degrees C with 1:3M ratio of glycerol to CLA).

282 eters aphid settling)-mediated resistance to CLA compared with B73 and Tx601 maize susceptible inbred

283 JA, contributed to heightened resistance to CLA in maize.

284 gesting that the OPDA-mediated resistance to CLA is independent of the jasmonic acid pathway.

285 se accumulation and heightened resistance to CLA, suggesting that the OPDA-mediated resistance to CLA

286 sition by providing heightened resistance to CLA.

287 nt breeding, provides enhanced resistance to CLA.

288 Mp708 provides phloem-mediated resistance to CLA.

289 ir1-Cys Protease provides direct toxicity to CLA.

290 evated ratios of alpha(4)beta(7)(+) Tregs to CLA(+) Tregs (odds ratio, 18.1; P = .020).

291 The content of total CLA and the percentage of its t11,c13 isomer were higher

292 The total CLA content of milk samples from cows, sheep, goats, yak

293 al properties or emulsion stability by using CLA-rich eggs.

294 ace method was used and 10% w/w AG, 3.5% w/w CLA and 0.3% w/w XG was introduced as the optimum formul

295 l properties are needed to establish whether CLA(+) memory subsets can be used as biomarkers and a su

296 This review examines whether CLA's antiobesity properties are due to inflammatory sig

297 ll characterized, it remains unknown whether CLA also affects vascular morphology in the central nerv

298 e identified a novel mechanism through which CLA alters monocyte function.

299 Understanding the mechanisms through which CLA mediates its atheroprotective effect may help to ide

300 of phosphatidylcholine (PC) acidolysis with CLA use to be limited to <30%, due to competitive side-h

301 gical properties of mayonnaise prepared with CLA-rich eggs to control eggs and (3) compare the emulsi

302 Supplementation with CLA and SAF exerted different effects on BMI, total and

303 Supplementation with CLA reduced body mass index (BMI) (P = 0.0022) and total

304 aegypti mosquitoes infected with the wMelPop-CLA strain of Wolbachia and in Drosophila melanogaster a