ライフサイエンスコーパス: CMD

1 CMD 50 D was activated by EDC/NHS for immobilization of

2 CMD can be detected by applying machine learning to EEG

3 CMD is caused by a complex comprised of 11 recognized vi

4 CMD is highly prevalent among at-risk individuals and is

5 CMD is one of the most important diseases of cassava and

6 CMD treatment reduced hydroperoxide generation and blunt

7 CMD was an independent predictor of a lower rate of left

8 CMD was defined as impaired endothelium-independent and/

9 CMD was estimated via quadriceps electromyogram.

10 CMD was highly prevalent both in men and women (51% and

11 CMD was invasively assessed by continuous intracoronary

12 CMD was measured using the General Health Questionnaire.

13 CMD-35647 is a pharmocological compound that can neutral

14 A total of 123 CMD patients were investigated using the traditional app

15 in the country and exhibits the higher SO(2) CMD at national level.

16 ; 62% women) underwent random assignment (57 CMD group and 30 reference group).

17 odontic movement in a mouse model carrying a CMD knock-in (KI) mutation (Phe377del) in the Ank gene.

18 pression and/or anxiety as an indicator of a CMD (n = 2698, 14%).

19 The primary outcome was the presence of a CMD, as defined by the Revised Clinical Interview Schedu

20 Most individuals with a CMD reported visiting nature >= once a week.

21 ce of blocked group III/IV muscle afferents, CMD is less inhibited but O(2) transport compromised and

22 NGS, we performed mutation detection in all CMD genes of 26 samples and compared the results with Sa

23 that show C-H activation proceeds by an AMLA/CMD mechanism.

24 p Program for Common Metabolic Diseases (AMP CMD).

25 no association between physical activity and CMD 10 years later.

26 s, were associated with both birthweight and CMD at a false discovery rate of 5%.

27 3)) in loci influencing both birthweight and CMD.

28 l muscular dystrophy (CMD), Fukuyama CMD and CMD type 1C (MDC1C) are caused by mutations in the genes

29 Both CMD and CMD-b-PEG decreased the charge of alpha-lac below pH 5.5

30 atrophy [at least 250 mum in diameter], and CMD).

31 e in moesin levels in the muscles of DMD and CMD mice.

32 and congenital muscular dystrophies (DMD and CMD, respectively) and dysferlinopathy, but not in the w

33 e and congenital muscle dystrophies (DMD and CMD, respectively), and dysferlinopathy.

34 was created using the combination of HRL and CMD scores.

35 nemia and reproductive tract infections) and CMD.

36 een alpha-lac and CMD-b-PEG or alpha-lac and CMD was determined following acidification by light scat

37 Formation of complexes between alpha-lac and CMD-b-PEG or alpha-lac and CMD was determined following

38 nd congenital muscular dystrophies (LGMD and CMD) are characterized by skeletal muscle weakness and d

39 nterventions into both health management and CMD management.

40 ce rates increased with age for both MDS and CMD (P < .05) and were highest among whites and non-Hisp

41 nual age-adjusted incidence rates of MDS and CMD for 2001 through 2003 were 3.3 and 2.1 per 100,000,

42 S population, we evaluated trends in MDS and CMD incidence, estimated case numbers for the entire Uni

43 l relative 3-year survival rates for MDS and CMD were 45% and 80%, respectively, with males experienc

44 jected future precipitation, temperature and CMD at our study locations for four scenarios selected t

45 enarios projected increasing temperature and CMD in coming decades (e.g., from 4.5% to 29.5% higher C

46 reased INFLA-score with the risk of CMMs and CMDs.

47 ty, insulin resistance, type 2 diabetes, and CMDs.

48 is-/trans-gene regulation across tissues and CMDs.

49 cted from male rhesus macaques classified as CMD or CHD after 12 months of voluntary ethanol self-adm

50 erformance of acetate vs carbonate to assist CMD in the azole series reveals also considerable change

51 f CLIs on overweight, obesity and associated CMDs, in controlled studies and in routine care.

52 In recent years, severe, atypical CMD symptoms have been observed occasionally on resistan

53 The annual average CMD in Ekibastuz reaches 2.5 x 10(-5) kg/m(2), whereas f

54 Both CMD and CMD-b-PEG decreased the charge of alpha-lac belo

55 Mutational analyses showed that both CMD-1 and HTH-4 are also necessary for activation of the

56 where production is severely constrained by CMD.

57 lbumin (alpha-lac) and carboxymethyldextran (CMD) in acidic solutions lead to phase-separated complex

58 molecules supported on carboxymethyldextran (CMD)-coated surfaces measured using surface plasmon reso

59 agnoses had variants in genes known to cause CMD (76%), and 11 of 59 (19%) had variants in genes asso

60 dystroglycan in mice is sufficient to cause CMD-like brain malformations, including disarray of cere

61 up of laminin alpha 2-chain-positive classic CMD likely is heterogeneous.

62 or breakthrough involves a type of "classic" CMD with abnormalities of the white matter on magnetic r

63 ng of the limb-girdle (LGMD) and congenital (CMD) muscular dystrophies have led to a new, genetically

64 from DMD, Ullrich CMD, and merosin-deficient CMD patients, all of which present high levels of fibros

65 large gradient in climatic moisture deficit (CMD) from warm and dry low elevation sites to relatively

66 d formaldehyde (HCHO) column mass densities (CMD) are analyzed for the period 2005-2016 over the atmo

67 Endothelium-dependent CMD (ie, abnormal coronary vasoreactivity) was identifie

68 Concerted metalation deprotonation (CMD) approach with appropriate proton shuttle precursor,

69 t with a concerted metalation deprotonation (CMD) pathway.

70 upported concerted metalation deprotonation (CMD) steps and the unique role of dimethyl sulfoxide (DM

71 Although concerted metalation-deprotonation (CMD) is favored under acetate assistance at the C5 site

72 via the concerted metalation-deprotonation (CMD) mechanism in all types of reactions, the subsequent

73 ion by a concerted metalation-deprotonation (CMD) mechanism involves precoordination of the C-H bond

74 on via a concerted metalation-deprotonation (CMD) mechanism, and (5) reprotonation of the amide direc

75 cal of a concerted metalation-deprotonation (CMD) mechanism, were observed in the case of a bromopyro

76 ion of a concerted metalation-deprotonation (CMD) mechanism.

77 p by the concerted metalation-deprotonation (CMD) pathway is important in further development of cros

78 d in the concerted metalation-deprotonation (CMD) process.

79 upport a concerted metalation-deprotonation (CMD)-type ruthenation mode and shed light on the unusual

80 A) and "concerted metalation deprotonation" (CMD) concepts.

81 Noninvasive cardiac MRI MPRI can detect CMD defined by invasive CRT.

82 al in CMD, cardiac MRI may be able to detect CMD in this population.

83 At each d(mob), the count median diameter (CMD) of the rBC cores was essentially the same as their

84 Contemporary mortgage discrimination (CMD) scores were calculated, and PMD index was created u

85 ramides (Cer) drive cardiometabolic disease (CMD) by promoting lipotoxicity and oxidative stress, yet

86 ing risk factor for cardiometabolic disease (CMD) in the United States, but its economic costs are un

87 Cardiometabolic disease (CMD) is a leading cause of death worldwide and encompass

88 ntified hundreds of cardiometabolic disease (CMD) risk loci.

89 ch IE fat blends on cardiometabolic disease (CMD) risk markers remain unclear.

90 ifferential risk of cardiometabolic disease (CMD), which can be predicted by epigenetic marks in prox

91 ssification: coronary microvascular disease (CMD group) if CFR<2.5 and reference group if CFR>=2.5.

92 Coronary microvascular disease (CMD) causes myocardial ischemia in a variety of clinical

93 Coronary microvascular disease (CMD), characterized by impaired coronary flow reserve (C

94 ociated with coronary microvascular disease (CMD), where narrowing of the small blood vessels can lea

95 viruses (CMBs) cause cassava mosaic disease (CMD) across Africa and the Indian subcontinent.

96 ude diseases such as Cassava Mosaic Disease (CMD) and Cassava Bacterial Blight (CBB), drought, and ac

97 diseases, including Cassava Mosaic Disease (CMD) and Cassava Brown Streak Disease (CBSD), pose signi

98 01, resistant to the cassava mosaic disease (CMD) and the cassava bacterial blight (CBB), and MECU72,

99 y analysis targeting cassava mosaic disease (CMD) identified 27 genes observed in the plant-pathogen

100 Cassava mosaic disease (CMD) is one of the most important viral diseases of cass

101 o be associated with cassava mosaic disease (CMD) resistance.

102 Cassava mosaic disease (CMD) suppresses cassava yields across the tropics.

103 Cassava mosaic disease (CMD), which is caused by single-stranded DNA begomovirus

104 iota are linked to cardiometabolic diseases (CMD), it is unclear if they similarly predict CMD mortal

105 Cardiometabolic diseases (CMDs) affect significant numbers of adults in the United

106 Cardiometabolic diseases (CMDs) have shared properties and causes.

107 ral infections and cardiometabolic diseases (CMDs), controversy still lingers.

108 ood/beverage intake, common mental disorder (CMD) and depression and to examine the role of reverse c

109 local information on common mental disorder (CMD) and substance use by comparing data from the South

110 hysical activity and common mental disorder (CMD), defined as anxiety and depression, using data from

111 Common mental disorders (CMD), which include depression and anxiety, are prevalen

112 ower risk of common mental health disorders (CMDs), such as depression and anxiety, but we know littl

113 predicts postnatal common mental disorders (CMDs), anxiety and depression.

114 high prevalence of common mental disorders (CMDs).

115 s) and chronic myeloproliferative disorders (CMDs) to population-based cancer registries in the Unite

116 Cognitive motor dissociation (CMD) can improve the accuracy to predict recovery of beh

117 sciousness, or cognitive motor dissociation (CMD), is present in up to 15-20% of patients with DoC an

118 d characterization of patterns with distinct CMD in patients with HF and could identify patients with

119 age has two community medicine distributors (CMDs), who are the seed nodes and responsible for admini

120 trophic muscles of the murine models of DMD, CMD, and dysferlinopathy were the source of Cthrc1.

121 d cardiac muscles of mice, representing DMD, CMD, and dysferlinopathy.

122 the boundaries of this conserved Mga domain (CMD-1) spanning from residues 10 to 15 of the protein, w

123 In contrast, chronic moderate drinking (CMD) has been associated with improved cardiovascular he

124 on peripheral fatigue, central motor drive (CMD) and endurance capacity during high-intensity leg-cy

125 Classical Molecular Dynamics (CMD) simulations were used to investigate the chemistry

126 spasm or coronary microvascular dysfunction (CMD) due to microvascular spasm.

127 Coronary microvascular dysfunction (CMD) encompasses several pathogenetic mechanisms involvi

128 cator of coronary microvascular dysfunction (CMD) in patients with ischemia with nonobstructive coron

129 Coronary microvascular dysfunction (CMD) is a prevalent and prognostically important finding

130 Coronary microvascular dysfunction (CMD) is defined by diminished flow reserve.

131 Coronary microvascular dysfunction (CMD) is involved in heart failure (HF) onset and progres

132 CAD) and coronary microvascular dysfunction (CMD) may contribute to the pathophysiologic characterist

133 ten have coronary microvascular dysfunction (CMD), diagnosed by invasive coronary reactivity testing

134 arker of coronary microvascular dysfunction (CMD), is independently associated with body mass index (

135 ted with coronary microvascular dysfunction (CMD).

136 Craniometaphyseal dysplasia (CMD) is a rare genetic disorder encompassing hyperostosi

137 Craniometaphyseal dysplasia (CMD) is a rare genetic disorder with hyperostosis of cra

138 Craniometaphyseal dysplasia (CMD) is a rare skeletal disorder characterized by progre

139 inosis (CCAL2), craniometaphyseal dysplasia (CMD), mental retardation, deafness and ankylosis syndrom

140 The congenital muscular dystrophies (CMD) are a heterogeneous group of autosomal recessive di

141 hy types of congenital muscular dystrophies (CMDs) and ocular abnormalities termed muscle-eye-brain d

142 en in those congenital muscular dystrophies (CMDs) caused by mutations in genes involved in O-mannosy

143 The congenital muscular dystrophies (CMDs) comprise a heterogeneous group of heritable muscle

144 re group of congenital muscular dystrophies (CMDs) known as dystroglycanopathies.

145 rs known as congenital muscular dystrophies (CMDs) with associated CNS abnormalities.

146 yndrome are congenital muscular dystrophies (CMDs) with associated developmental brain defects.

147 LGMD2I) and congenital muscular dystrophies (CMDs) with brain malformations and mental retardation.

148 Congenital muscular dystrophy (CMD) is a group of clinically and genetically heterogene

149 form of human congenital muscular dystrophy (CMD) is caused by mutations in the laminin-alpha2 gene.

150 Congenital muscular dystrophy (CMD) is characterized by severe muscle wasting, prematur

151 vere forms of congenital muscular dystrophy (CMD) such as Fukuyama type congenital muscular dystrophy

152 Two forms of congenital muscular dystrophy (CMD), Fukuyama CMD and CMD type 1C (MDC1C) are caused by

153 Both SEGS enhanced CMD symptoms in cassava (Manihot esculentaCrantz) when c

154 lia-associated chronic myeloid disorder (eos-CMD) characterized by the t(5;12)(q33;p13) cytogenetic a

155 ate the therapeutic value of imatinib in eos-CMD that is not associated with t(5;12)(q33;p13).

156 l men, median age = 46 years) and 2 with eos-CMD (both men, aged 45 and 58 years) were treated with i

157 3 weeks), 2 patients with HES and 1 with eos-CMD have achieved complete clinical remission and 1 addi

158 valence of vasomotor dysfunction (especially CMD) compared with male patients.

159 nd experiences of those already experiencing CMDs.

160 (59%) with a pathological ACH test, 33% for CMD and 26% for epicardial vasospasm.

161 Density functional calculations for CMD C-H activation at C6, C5, C8, and C2' sites imply th

162 tate ligand, which is the base of choice for CMD, was found inefficient for nCMD and the comparative

163 insights into gene-regulatory mechanisms for CMD risk loci, facilitating their translation into oppor

164 is in homozygous Ank KI/KI mice, a model for CMD, showed that molars can be moved by orthodontic forc

165 ignificant with a female-male odds ratio for CMD and epicardial vasospasm of 4.2 (95% confidence inte

166 s independently associated with the risk for CMD were factors indicative of gender disadvantage, part

167 ctice guidelines support routine testing for CMD in patients with ischemia with nonobstructive corona

168 ructive coronary artery disease, testing for CMD may play a role in patients with acute myocardial in

169 age cultures, peripheral blood cultures from CMD patients exhibited reduced osteoclastogenesis.

170 can lead to an improvement in recovery from CMD among patients attending public primary care facilit

171 Fukuyama CMD, muscle-eye-brain disease and Walker-Warburg syndrom

172 ongenital muscular dystrophy (CMD), Fukuyama CMD and CMD type 1C (MDC1C) are caused by mutations in t

173 Functional CMD is characterized by elevated resting flow that is li

174 ymethyl-dextran-block-poly(ethylene glycol) (CMD-b-PEG), the PEG block was hypothesized to reduce int

175 Of the 86 patients, 46 had CMD (28 functional, 18 structural), and 40 patients form

176 The highest HCHO CMD (9 x 10(15) molecules/cm(2)) appears in the summerti

177 very sparse at the warmest and driest (high CMD) sites, particularly when combined with high topogra

178 ch as Almaty and Nur-Sultan, experience high CMDs of SO(2) in particular years, such as 2008.

179 ing decades (e.g., from 4.5% to 29.5% higher CMD by the 2080's compared to the 1971-2000 average), ev

180 obilize BSA, carboxymethyl dextran hydrogel (CMD) Au chip was used.

181 ction pathway, several previously identified CMD resistance genes, and two peroxidase family proteins

182 cation on gated computed tomography imaging, CMD was common in both sexes, despite normal stress perf

183 This translates to $50.4 billion in CMD costs (18.2% of total) for the whole population, of

184 ugh initially similar, the percent change in CMD was 9 +/- 3% higher at end-exercise with fentanyl vs

185 icated that the rumen bacterial community in CMD-fed lambs had lower diversity and a higher relative

186 anism of altered resting and maximal flow in CMD endotypes.

187 ing the alpha2-chain of laminin 2 (LAMA2) in CMD patients.

188 oninvasive stress imaging is often normal in CMD, cardiac MRI may be able to detect CMD in this popul

189 ity was associated with a small reduction in CMD over 5 years.

190 l and functional brain abnormalities seen in CMD.

191 The onset of symptoms in CMD is within the first few months of life, whereas in L

192 as episomes and is packaged into virions in CMD-infected cassava and viruliferous whiteflies.

193 Furthermore, their absence in CMDs suggests that hypoglycosylation of RPTPzeta/phospha

194 verages had a 23% increased odds of incident CMD after 5 years (95% CI: 1.02, 1.48) independent of he

195 djustment, MIP was associated with increased CMD mortality risk (HR per 1-SD: 2.10 [1.30-3.38]).

196 Additionally, genetics influences CMDs, and this creates a confounding relationship among

197 ltaneous screening of mutations in all known CMD-associated genes would be a more effective diagnosti

198 rmed at pH values near pH 6, while alpha-lac/CMD complexes formed at pH 5.5.

199 Hydrodynamic radii of alpha-lac/CMD-b-PEG complexes was between 11 and 20 nm, which incr

200 separation, size, and structure of alpha-lac/CMD-b-PEG complexes were characterized by turbidimetry,

201 icrographs that were attributed to alpha-lac/CMD-b-PEG complexes.

202 Complexes of alpha-lac/CMD-b-PEG formed at pH values near pH 6, while alpha-lac

203 LAMA2-CMD patients exhibit hypotonia from birth and progressiv

204 related congenital muscular dystrophy (LAMA2-CMD) is a fatal muscle disease caused by mutations in th

205 ective protein replacement therapy for LAMA2-CMD.

206 ective protein-replacement therapy for LAMA2-CMD.

207 tly no cure or effective treatment for LAMA2-CMD.

208 fter disease onset in a mouse model of LAMA2-CMD.

209 Since LAMA2-CMD patients are often diagnosed after advanced disease,

210 At the genus level, CMD mediated specific shifts from Prevotella, Alloprevot

211 ck from group III/IV muscle afferents limits CMD but also minimizes locomotor muscle fatigue developm

212 CMD2 is a major CMD resistance locus that has been deployed in many cass

213 as defined as the coexistence of two or more CMDs, such as coronary artery disease, type 2 diabetes m

214 Neither CMD nor depression predicted intake changes.

215 A group of non-CMD patients was identified with injury to the left thal

216 c to CMD ratio, while the relative amount of CMD-b-PEG did not significantly influence either.

217 The clinical assessment of CMD in women must include exploration of violence and ge

218 ditional education and provider awareness of CMD and its role in cardiovascular disease is needed to

219 % of patients with DoC and that detection of CMD in the intensive care unit can predict functional re

220 Here, we investigated the effects of CMD-mutant ANK on mineralization and bone mass at a cell

221 glycol) (PEG) chain onto the reducing end of CMD, forming carboxymethyl-dextran-block-poly(ethylene g

222 gical symptoms may be somatic equivalents of CMD in women in Asian cultures.

223 SELCoH sample prevalence estimates of CMD were nearly twice that of the APMS England sample es

224 Dental abnormalities are features of CMD that have been little discussed in the literature.

225 n gene (FKRP) is mutated in a severe form of CMD (MDC1C), characterized by the inability to walk, leg

226 in this autosomal recessive type 1A form of CMD results in neuromuscular dysfunction and, often, ear

227 the locus for the autosomal dominant form of CMD to an approximately 5-cM interval on chromosome 5p,

228 in the pathogenesis of the 'classic' form of CMD.

229 rter analyses demonstrated the importance of CMD-1 for activation of Pemm and autoactivation of Pmga

230 noninvasive identification and management of CMD patients.

231 of somatic gene therapy in a mouse model of CMD.

232 in-alpha2 deficiency in the murine models of CMD.

233 ty leisure-time activity had reduced odds of CMD 5 years later (below median vs. none: adjusted odds

234 hysically demanding jobs had reduced odds of CMD after 5 years, and there was no association between

235 FKRP may play a role in the pathogenesis of CMD and potentially explain why the allelic disorder LGM

236 erve <2.0 was used to define the presence of CMD.

237 information for estimating the prevalence of CMD and substance use is essential for surveillance and

238 The high prevalence of CMD in both sexes suggests that it may be a useful targe

239 The prevalence of CMD was 6.6% (95% confidence interval [CI], 5.7%-7.6%).

240 Due to the emerging role of CMD as common denominator in different clinical phenotyp

241 ents with a clinical phenotype suggestive of CMD, with muscle biopsy reserved as a second-tier invest

242 8 month treatment of CMD-35647 improved muscle function in aged mice independ

243 8 month treatment of CMD-35647 mitigated muscle atrophy in tibialis anterior

244 Among the group of CMDs with abnormalities of brain formation and mental re

245 is a pivotal molecule in the pathogenesis of CMDs.

246 her oral infections can increase the risk of CMDs independent of the aforementioned confounding facto

247 , 87 687 participants developed at least one CMD, 14 164 developed CMM, and 17 541 died afterwards.

248 to changing pH, covalently immobilized onto CMD-coated supports.

249 affect total:HDL cholesterol ratio or other CMD risk markers compared with stearic acid-rich fats.

250 The main outcome, CMD, was indicated by a score of 12 or above on the Revi

251 so presented, including the formation of pre-CMD intermediates and the analysis of bonding interactio

252 MD), it is unclear if they similarly predict CMD mortality.

253 in childhood may have utility in predicting CMD risk.

254 ple behaviors, have the potential to prevent CMD, but their implementation, reach and effectiveness i

255 Annual diet-related CMD costs were $301/person (95% CI $287-$316).

256 an immunolabelling, unlinked to any reported CMD loci.

257 driven RNFL metabolic state model stratifies CMD risk with up to 21.8-fold enrichment between risk de

258 Functional and structural CMD endotypes have recently been described, with normal

259 unctional group compared with the structural CMD and reference groups, respectively.

260 k CBF was lowest in patients with structural CMD compared with the functional and reference groups.

261 Patients with structural CMD have endothelial dysfunction, which leads to diminis

262 Serial Sanger sequencing of suspected CMD genes, while the current molecular diagnostic method

263 RI was performed in 118 women with suspected CMD who had undergone CRT and 21 asymptomatic reference

264 A previous study showed that CMD symptom severity and viral DNA accumulation increase

265 dysgalactiae subsp. dysgalactiae showed that CMD-1 and HTH-4 are critical for transcriptional activat

266 Here, we show that CMDs with tightly knit (clustered) friendship connection

267 The CMD pathway to the C-H bond cleavage for different class

268 Most of the CMD cases are caused by genetic mutations of laminin-alp

269 Successful validation and application of the CMD NGS panel to improve the diagnostic yield in a clini

270 boxylic groups, BSA was immobilized onto the CMD chip through covalent amide binding formation.

271 to quantify the various contributions to the CMD transition state (TS).

272 r accelerating the C-H deprotonation via the CMD mechanism; and (4) revealing the roles of the CsF ba

273 Thus, the CMDs are a model disorder group for development and vali

274 lycosylated in a mouse model of one of these CMDs, known as muscle-eye-brain disease, a disorder that

275 s defined as the coexistence of two or three CMDs, including ischaemic heart disease (IHD), stroke, a

276 g brain and eye malformations in addition to CMD.

277 tion were both sensitive to the alpha-lac to CMD ratio, while the relative amount of CMD-b-PEG did no

278 which increased with increasing alpha-lac to CMD-b-PEG ratio and with decreasing pH.

279 pyrophosphate (PPi) transporter ANK leads to CMD-like features in an Ank(KI/KI) mouse model.

280 graphic and socioeconomic factors related to CMD and substance use across samples.

281 hat enhance symptoms and break resistance to CMD.

282 mon soil theory, the continuum of obesity to CMDs is the same pathology at different time points, and

283 With regard to CMDs, the major breakthrough involves a type of "classic

284 eight and 15 cardiometabolic disease traits (CMD).

285 in muscle biopsy specimens from DMD, Ullrich CMD, and merosin-deficient CMD patients, all of which pr

286 motor commands with motor outputs underlies CMD.

287 c review of the literature to assess whether CMDs can be modulated via dietary interventions targetin

288 The relative extent to which CMD affects both sexes is largely unknown.

289 cortical network function is associated with CMD detection.

290 Gene expression traits associated with CMD risk single-nucleotide polymorphism (SNPs) identifie

291 med dentofacial examination of patients with CMD and evaluated consequences of orthodontic movement i

292 ns for the dental treatment of patients with CMD are provided.

293 d as the reference group or as patients with CMD by a coronary flow reserve threshold of 2.5; functio

294 alometric analysis showed that patients with CMD tend to have a short anterior cranial base, short up

295 700 patients with MDS and 6300 patients with CMD were estimated for the entire United States in 2004.

296 d effective diagnostic tool in patients with CMD.

297 Aged mice treated with CMD-35647 had greater respiration in red gastrocnemius m

298 Individuals with CMDs seem to be using nature for self-management, but 'g

299 or all, though slightly lower for those with CMDs.

300 sions were seen in patients with and without CMD.