ライフサイエンスコーパス: CRP

1 CRP also showed a negative association with the "felt lo

2 CRP and Ki-67 index were also prognostic and remained in

3 CRP and PCT were tested simultaneously in the same study

4 CRP appears to be a peripheral biomarker that reflects p

5 CRP concentrations were measured from prediagnosis serum

6 CRP is a non-specific systemic marker of chronic inflamm

7 CRP is a nonspecific systemic marker of chronic inflamma

8 CRP testing using a cutoff of >=80 mg/L, integrated into

9 CRP, WBC and percentage of neutrophils were analyzed in

10 IL-2, IL-4, IL-5, IL-6, IL-8, IL-10, IL-12, CRP, TNF-alpha, IFN-gamma, GM-CSF, MIP-1alpha, and Eotax

11 djustment for any inflammation marker (IL-6, CRP [C-reactive protein], TNF-alpha R1, D-dimer, and fib

12 IL-6, CRP and triglycerides are likely to be causally linked w

13 A CRP concentration >10 mg/L was positively associated wit

14 to determine the diagnostic performance of a CRP enzyme-linked immunosorbent assay (ELISA) (Eurolyser

15 ssor, named ErfA, which together with Vfr, a CRP-like activator, controls exlBA expression in P. aeru

16 sensitive (LOD = 0.80 mug/mL), and accurate CRP determination (E(r) = 1%) in hardly available preter

17 e sensitive (LOD = 0.54 mug/mL) and accurate CRP determination using very low volume preterm neonatal

18 Adding CRP to prediction models based on established risk facto

19 l oxygen upon hospitalization, and admission CRP were independent predictors for the development of s

20 At the time of admission, CRP level was 12.5mg/dl and the remarkably increased exp

21 nates had higher levels of IP-10, TNF-alpha, CRP, sCD14, and BAFF than United States neonates.

22 dent of CRP genotype combinations that alter CRP levels and more biologically plausible than other cl

23 Although CRP is known to facilitate the clearance of cell debris

24 was a significantly higher WCC(P=0.014) and CRP(P=0.004) on admission in P2.

25 The respective AUC values for IL-6 and CRP levels in the evaluation cohort were 0.97 and 0.86,

26 renal injury (OR, 2.7; 95% CI, 1.3-5.6), and CRP on admission (OR, 1.006; 95% CI, 1.001-1.01).

27 n to examine the association between AMH and CRP without and with adjustment for age, race, body mass

28 rman correlation occurred between SES-CD and CRP (r = 0.525), fCal (r = 0.450), and CDAI (r = 0.407),

29 sal estimates of "felt loved as a child" and CRP on PTSD.

30 ur results revealed that higher cortisol and CRP levels were significantly associated with persistent

31 s of subunits and domains are different, and CRP is asymmetric.

32 d testosterone, free androgen index, HDL and CRP (P < 0.01).

33 n the UK cohort whilst testosterone, HDL and CRP were higher in the Middle East population.

34 Interestingly, Ki-67 index and CRP influenced the prognostic power of PD-L1.

35 Human milk leptin, insulin, and CRP concentrations were higher in OW mothers compared wi

36 an 8.5 g/dL as highly predictive of IRIS and CRP>106 ug/ml and BMI < 15.6 kg/m2 as predictive of deat

37 th decreased serum concentrations of MBL and CRP and increased serum levels of SIRT1.

38 rkedly decreased the mRNA levels of MCP1 and CRP and both mRNA and protein levels of TNF-alpha.

39 evaluation cohort (IL-6 level > 80 pg/mL and CRP level > 97 mg/L) both correctly classified 80% of pa

40 ng ICI tended to have a longer median OS and CRP - but not PD-L1 - was a significant prognosticator i

41 ctional genetic association between PTSD and CRP, also suggesting a potential role of SES in the inte

42 ificant negative association between PZC and CRP or AGP, application of the RC approach is supported.

43 The ability of a weekly DBS sampling and CRP test regime to detect flare outside the clinic was a

44 Significant reductions in temperature and CRP were seen post-tocilizumab.

45 Levels of IL6, TNFalpha, VEGF, and CRP were significantly higher in psychosis probands comp

46 who had higher inflammatory markers (WBC and CRP), HDL and insulin resistance (p < 0.001).

47 of serum ferritin (SF), vitamin A, zinc, and CRP measured using different assays with variable LLOQs.

48 to measure 13 proteins in blood (ANG1, ANG2, CRP, SAA1, IL7, EMMPRIN, MMP1, MMP2, MMP3, MMP9, TGFA, C

49 Anti-CRP functionalized micromotors (anti-CRP-rGO(reduced gra

50 st efficient and reproducible (CV = 9%) anti-CRP functionalization, controlled stopped-flow operation

51 Anti-CRP functionalized micromotors (anti-CRP-rGO(reduced graphene oxide)/Ni/PtNPs (platinum nanop

52 o calculate CFs, inflammation was defined as CRP >5 mg/L or AGP >1 g/L, or both.

53 s of inflammation and organ function such as CRP, platelet count and serum lactate have to be taken i

54 cyte (M) and platelet (P) counts, as well as CRP (C) and albumin (A) levels were recorded.

55 Baseline DBS ELISA assay CRP measures yielded a mean positive bias of 2.693 +/- 8

56 ntify flares, was greater than 'minimal' AUC CRP data (54.5%).

57 y of 'continuous' area under the curve (AUC) CRP data (72.7%) to identify flares, was greater than 'm

58 Patients with the highest level of baseline CRP or IL6, both downstream of IL1beta signaling, trende

59 of CRP and lower platelet count at baseline (CRP 262 mg/L (IQR 101-307) vs. 94 mg/L (IQR 26-153): p =

60 udy was designed to assess agreement between CRP values obtained by dried blood spot (DBS) versus con

61 ium (OC3) to examine the association between CRP and epithelial ovarian cancer risk overall, by histo

62 observed a bidirectional association between CRP and PTSD (CRP -> PTSD: beta = 0.065, p = 0.015; PTSD

63 Correlation coefficients between CRP and vitamin B-12 for WRA and PSC ranged from -0.25 t

64 Similarly, correlations between CRP and serum folate ranged from -0.13 to 0.08, and corr

65 e weak and inconsistent correlations between CRP or AGP and vitamin B-12 or folate biomarkers, there

66 Spearman correlations between CRP or AGP and vitamin B-12 or folate concentrations wer

67 Furthermore, increased peripheral blood CRP in MDD has been associated with altered reward circu

68 Genetic effects on BMI, CRP and risk-taking were all positively correlated, and

69 lso found significant associations with both CRP and IL-6.

70 The maximal level of IL-6, followed by CRP level, was highly predictive of the need for mechani

71 vers of systemic inflammation as measured by CRP.

72 vers of systemic inflammation as measured by CRP.

73 ong Chinese adults, who have low mean LDL-C, CRP, but not fibrinogen, was independently associated wi

74 el ICF algorithms inclusive of point-of-care CRP-based TB screening and confirmatory testing with uri

75 ociated with the proportion of left-censored CRP data, whereas these were not associated in the proba

76 1.6 mg/dL (24.0) and geometric mean (95% CI) CRP and fibrinogen were 0.90 mg/L (0.87-0.93) and 3.01 g

77 omparing two proposed thresholds to classify CRP concentrations as low or high to guide antibiotic tr

78 CSF CRP in turn correlated with CSF cytokine receptors/antag

79 correlation was found between plasma and CSF CRP (r = 0.855, p < 0.001).

80 Associations among plasma and CSF CRP and plasma and CSF inflammatory cytokines (interleuk

81 een-group differences in changes in hs-cTnI, CRP, uric acid, or urine protein-creatinine ratio were o

82 al pro-B-type natriuretic peptide), hs-cTnT, CRP (C-reactive protein), and circulating oxidative stre

83 orrelated with levels of NT-proBNP, hs-cTnT, CRP, or oxidative stress biomarkers.

84 In reference to M. tuberculosis culture, CRP had a sensitivity of 78% (95% confidence interval [C

85 istress, immune cell proportions, cytokines, CRP and serum cortisol were measured at baseline and dur

86 DAS28(CRP) 3.2 or lower was met by 42 (19%) of 216 (95% CI 14-

87 ving clinical remission according to a DAS28-CRP of less than 2.6.

88 ed with clinical remission, defined as DAS28-CRP of 3.2 or less and no swollen joints.

89 From baseline DAS28-CRP values of 5.70 in the upadacitinib group and 5.88 in

90 based on the C-reactive protein level (DAS28-CRP; range, 0 to 9.4, with higher scores indicating more

91 ept in the change from baseline in the DAS28-CRP and the achievement of remission at week 12 but was

92 ept in the change from baseline in the DAS28-CRP and the percentage of patients having clinical remis

93 tivity Score using C-reactive protein (DAS28[CRP]) of 3.2 or lower, both with non-responder imputatio

94 Despite, CRP antigen was further detected in human serum by spiki

95 Elevated CRP, a lower BI, a history of malignancy, and elevated h

96 ients with pericarditis pain and an elevated CRP level were enrolled in the run-in period.

97 that older males with insomnia and elevated CRP may be particularly vulnerable to deficits in reward

98 ESR, CRP, fibrinogen, ferritin, and procalcitonin were higher

99 of insulin, leptin, and, to a lesser extent, CRP.

100 ations for WRA ranged from -0.07 to 0.08 for CRP and -0.04 for AGP (1 country).

101 e validation cohorts from SAGES (n = 150 for CRP, IL-6; n = 126 for CHI3L1).

102 ical distribution of SF after correction for CRP and AGP by the BRINDA method was comparable with the

103 eremia, 30-day rates of clinical failure for CRP-guided antibiotic treatment duration and fixed 7-day

104 n the physiological concentration ranges for CRP (0.1-50 mg/L), NT-proBNP (50-10,000 pg/mL), cTnI (1-

105 icantly higher AUROC value than the test for CRP (0.876, P < .001 in validation cohort 1 and 0.624, P

106 velopmental programs along trajectories from CRP to mature venous and arterial populations.

107 symptoms were associated with higher future CRP/IL-6 in both unadjusted and adjusted analyses - this

108 RP -> PTSD: beta = 0.065, p = 0.015; PTSD -> CRP: beta = 0.008, p = 0.009).

109 , blood pressure, biomarkers (lipids, HbA1c, CRP) and 24-hour Holter monitoring were obtained at base

110 ificant effects were observed for TG, HbA1c, CRP, ALT, and AST.

111 RT, baseline low BMI and hemoglobin and high CRP and D-dimer levels may be clinically useful predicto

112 flammatory (n=83; 55% obese), and obese high CRP (C-reactive protein; n=89; 98% obese).

113 The obese high CRP phenotype resembled the noninflammatory phenotype ex

114 Higher CRP/IL-6 were associated with future depressive symptoms

115 rs, with adjusted ORs (95% CI) per SD higher CRP of 1.67 (1.44-1.94) for MCE and 1.22 (1.10-1.36) for

116 hs-CRP was a significant predictor of increased salivary an

117 )D was inversely associated with sICAM-1, hs-CRP, and AGP, which were positively associated with BMI.

118 els, CP (P = 0.034), CHD (P < 0.001), and hs-CRP (P < 0.001) were significantly associated with serum

119 on and the interplay between IL-1beta and hs-CRP concentrations on the risk of premature death.

120 Moreover, periodontitis and hs-CRP were the only significant predictors of the augmente

121 25(OH)D and higher PTH, HOMA-IR, HOMA-B, hs-CRP, and eGFR than white women (all P values < 0.0001).

122 nic acid ratio was associated with higher hs-CRP (P < .001) and neopterin concentrations (P < .001),

123 cardiac troponin I) 6.3 (3.4-13.0) ng/L, hs-CRP (high sensitivity C-reactive protein) 2.8 (1.3-6.1)

124 kynurenic acid was associated with lower hs-CRP (P = .025) and neopterin concentrations (P = .034).

125 um ADMA, whereas in a multivariate model, hs-CRP remained a significant predictor of serum ADMA (P <

126 Levels of hs-CRP did not differ among diets.

127 y Check Index (QUICKI) (0.004) and plasma hs-CRP (-0.222 mg/L) and reduction in liver fat content (1.

128 -1), high sensitivity C-reactive protein (hs-CRP), and alpha1-acid glycoprotein (AGP)] for which obse

129 ids, high-sensitivity C-reactive protein (hs-CRP), and for plasma and salivary suPAR levels.

130 rin, high-sensitivity C-reactive protein (hs-CRP), and lipids.

131 ding high-sensitivity C-reactive protein (hs-CRP), estimated glomerular filtration rate (eGFR), and h

132 ated high-sensitivity C-reactive protein (hs-CRP).

133 and high-sensitivity C-reactive protein (hs-CRP).

134 ate regression analysis demonstrated that hs-CRP (P <.001) and periodontitis (P <.001) had a signific

135 21 to 3.06; p = 0.005), regardless of the hs-CRP concentration.

136 t predictors of salivary ADMA levels were hs-CRP (P < 0.001) and education socioeconomic status (P =

137 redictor of plasma suPAR (P = .035) while hs-CRP was the only significant predictor of salivary suPAR

138 lack women and positively associated with hs-CRP in white women, independently of 25(OH)D.

139 598 patients in CRP group A, 593 in CRP group B, and 767 in the control group had follow-up

140 n three different activation regions (AR) in CRP that are ~30 angstrom apart.

141 be needed for those showing slow declines in CRP.

142 63 (5.5%) in the 14-day group (difference in CRP vs 14-day group, -3.1% [1-sided 97.5% CI, -infinity

143 Every 1-unit increase in CRP increased the risk of severe disease by 0.06%.

144 598 patients in CRP group A, 593 in CRP group B, and 767 in the control

145 Significant reductions in CRP (C-reactive protein), NT-proBNP (N-terminal pro-B-ty

146 of zinc deficiency increased with increasing CRP deciles, and to a lesser extent, with increasing AGP

147 zation in a 1:1:1 ratio to an individualized CRP-guided antibiotic treatment duration (discontinuatio

148 (IL-8), systemic and vascular inflammation (CRP, ICAM-1, VCAM-1), and SAA (all P < .001).

149 nclusion, among HIV-seronegative inpatients, CRP testing performed substantially below targets for a

150 higher insulin, insulin resistance, leptin, CRP, IL-1RA, and IL-6, and lower ghrelin than subjects i

151 recorded and serum C-reactive protein level (CRP) was assessed.

152 lele of rs181704186 is associated with lower CRP levels and decreased transcriptional activity and pr

153 C use (P = 0.002) were associated with lower CRP levels in postmenopausal women, whereas duration of

154 OC use (p=0.002) were associated with lower CRP levels in postmenopausal women, whereas only OC dura

155 ignificant increase in inflammation markers (CRP and IL-6) after stopping CPT compared to those who c

156 mechanical ventilation, followed by maximal CRP level.

157 als homozygous for rs181704186-G have a mean CRP level of 0.23 mg/L, in contrast to individuals heter

158 duals heterozygous for rs181704186 with mean CRP of 2.97 mg/L and major allele homozygotes with mean

159 mg/L and major allele homozygotes with mean CRP of 4.11 mg/L.

160 st for both full broadband- and NIR-mediated CRP in aqueous conditions.

161 In mixed models, CRP was associated with ferritin (positive) and serum ir

162 d the energy landscapes of WT and 14 mutated CRPs to determine how a homodimeric protein can distingu

163 regression indicated that the association of CRP and future depression was larger in older samples an

164 We investigated associations of CRP and fibrinogen with risks of incident major coronary

165 ximately log-linear positive associations of CRP with each outcome, which persisted after adjustment

166 Notably, the associations of CRP with LOY were similar in magnitude compared to those

167 Notably, the associations of CRP with LOY were similar in magnitude to associations w

168 EP groups have higher mean concentrations of CRP and, as predicted by the theory, absolute differenti

169 Genetic correlations of CRP were observed with PTSD (rg = 0.16, p = 0.026) and t

170 5 ng/mL was obtained during the detection of CRP in serum, and a large working range (1 ng/mL - 100 m

171 precision for the simultaneous detection of CRP.

172 lowed the fast and accurate determination of CRP in hardly available clinical samples as those coming

173 eneral, apiaries within foraging distance of CRP lands showed improved performance and higher gene ex

174 y phenotype except for isolated elevation of CRP and lower functional performance.

175 The optimal binding frequency of CRP-phosphocholine interaction was determined to be 100

176 chical cluster assignment was independent of CRP genotype combinations that alter CRP levels and more

177 significantly associated with both levels of CRP (Z = 0.12; 95% CI, 0.09-0.16) and IL-6 (Z = 0.15; 95

178 esponders had significantly higher levels of CRP and lower platelet count at baseline (CRP 262 mg/L (

179 Our study highlights the potential of CRP lands to improve pollinator health and the utility o

180 nd bacteria by phagocytic cells, the role of CRP in additional immunological functions is less clear.

181 The sensitivities of CRP (cut-off 6.5 mg/L) and PCT (cut-off 0.025 ng/mL) wer

182 lower-income settings supporting the use of CRP tests to rationalise antibiotic use in primary care

183 settings are needed to assess the utility of CRP for TB screening.

184 ite sex role of periodontitis and obesity on CRP levels were confirmed.

185 tic treatment duration (discontinuation once CRP declined by 75% from peak; n = 170), fixed 7-day tre

186 is suggests the possibility of using IL-6 or CRP level to guide escalation of treatment in patients w

187 o platelet activation by PAR1-AP, PAR4-AP or CRP-XL.

188 entation on serum lipids, blood pressure, or CRP in healthy 5- to 7-y-old children.

189 providing host defense against bacteria, PC:CRP-induced inflammation may also exacerbate pathology i

190 theless, the relationship between peripheral CRP and other peripheral and central markers of inflamma

191 e we report an oxygen tolerant, photoinduced CRP approach which readily affords quantitative yields o

192 Plasma CRP was correlated with multiple plasma inflammatory mar

193 Hair cortisol (N = 4761) and plasma CRP (N = 5784) were measured in wave 6 (2012-13).

194 arkers that were associated with high plasma CRP (>3 mg/L) and correlated with depressive symptom sev

195 rol over controlled radical polymerizations (CRP).

196 s, including a capillary resident precursor (CRP) that displays stem cell and migratory gene signatur

197 d RR 1.11, 95% CI 1.04-1.18); POD2 and PREOP CRP difference was associated with discharge to postacut

198 dehydrogenase (LDH), high Creactive protein (CRP) and chest radiographic appearance exceeding one-lun

199 56 [95% CI, 1.27-9.97]), C-reactive protein (CRP) >100 mg/L (adjusted OR, 2.71 [95% CI, 1.26-5.86]),

200 s predictive of IRIS and C-reactive protein (CRP) >106 mug/mL and BMI <15.6 kg/m2 as predictive of de

201 ically required range of C-reactive protein (CRP) (0.005 - 500 mg L(-1); r(2) = 0.993) levels with a

202 inflammation biomarkers C-reactive protein (CRP) and alpha1-acid glycoprotein (AGP) and serum vitami

203 of measurement of serum C-reactive protein (CRP) and fecal calprotectin.

204 plasma concentrations of C-reactive protein (CRP) and fibrinogen, is associated with increased risk o

205 els of markers including C-reactive protein (CRP) and Interleukin 6 (IL-6) measured in the same indiv

206 ic inflammatory markers, C-reactive protein (CRP) and interleukin-6 (IL-6).

207 itionally, the impact of C-reactive protein (CRP) and platelet count was also analysed.

208 the sensitivity of blood C-reactive protein (CRP) and procalcitonin (PCT) measured by Point-of-Care t

209 els of quantification of C-reactive protein (CRP) and recovery in spiked serum samples of ~98%.

210 mation biomarkers, e.g., C-reactive protein (CRP) and/or alpha1-acid-glycoprotein (AGP).

211 nterleukin-6 (IL-6), and C-reactive protein (CRP) are likely causal risk factors for depression.

212 tic performance of serum C-reactive protein (CRP) as a triage test for tuberculosis (TB) among HIV-se

213 mptoms worsen and plasma C-reactive protein (CRP) becomes elevated.

214 martly been designed for C-reactive protein (CRP) determination in plasma of preterm infants with sep

215 ed as a new approach for C-reactive protein (CRP) determination in serum and preterm neonatal plasma

216 nflammatory markers like C-reactive protein (CRP) have been associated with post-traumatic stress dis

217 f the detection of human C-reactive protein (CRP) in clinically relevant fluids.

218 n cholesterol (LDLc) and C-reactive protein (CRP) in patients with chronic kidney disease (CKD) are p

219 through the detection of C-reactive protein (CRP) in serum.

220 d immediate detection of C-reactive protein (CRP) in the wide concentration range of clinical interes

221 Peripheral blood C-reactive protein (CRP) is a biomarker used clinically to measure systemic

222 ammatory load and serum C- reactive protein (CRP) level, and infant birth weight.

223 5; 95% CI 1.15-1.81) and C-reactive protein (CRP) levels (HR 1.02; 95% CI 1.00-1.04) were significant

224 as associated with lower C-reactive protein (CRP) levels in both premenopausal (difference = -11.5%,

225 , cytokine profiles, and C-reactive protein (CRP) levels pre- and post-tocilizumab treatment.

226 Point-of-care testing of C-reactive protein (CRP) may be a way to reduce unnecessary use of antibioti

227 ociation between PZC and C-reactive protein (CRP) or alpha-1-acid glycoprotein (AGP) was observed: 1)

228 l was to explore whether C-reactive protein (CRP) testing at point of care could rationalise antibiot

229 te counts, and increased C-reactive protein (CRP) than patients with nonsevere infection.

230 ty and efficacy of using C-reactive protein (CRP) to decide on antibiotic prescription among febrile

231 terol, apoA-I, apoB, and C-reactive protein (CRP) were analyzed and non-HDL cholesterol calculated at

232 Levels of ADMA and C-reactive protein (CRP) were assessed with a commercially available kit.

233 Leptin, insulin, and C-reactive protein (CRP) were measured using electrochemiluminescence immuno

234 hair cortisol and plasma C-reactive protein (CRP) with the longitudinal persistence and dimensions (c

235 d glycoprotein (AGP) and C-reactive protein (CRP)) were measured at 6 and 18 mo.

236 genetic determinants of C-reactive protein (CRP), a biomarker of chronic inflammation, have been ext

237 We measured C-reactive protein (CRP), a marker of systemic inflammation and risk of infl

238 ce (HOMA-IR), uric acid, C-reactive protein (CRP), alanine transaminase (ALT), aspartate transaminase

239 periodontal parameters, C-reactive protein (CRP), and fibrinogen as markers of inflammation.

240 ndex (CDAI), fCal, serum C-reactive protein (CRP), and haemogram.

241 like protein 1 (CHI3L1), C-reactive protein (CRP), and interleukin-6 (IL-6) were independently valida

242 nd circulating levels of C-reactive protein (CRP), counter to their hypothesis.

243 with older age; elevated C-reactive protein (CRP), D-dimer, and fibrinogen levels; tachycardia; throm

244 s, circulating levels of C-reactive protein (CRP), IL6, IL18, IL1 receptor antagonist, TNFalpha, lept

245 cluding high-sensitivity C-reactive protein (CRP), interleukin (IL)-6, interferon-gamma inducible pro

246 the inflammatory markers C-reactive protein (CRP), interleukin 6 (IL-6), and tumor necrosis factor al

247 Serum Amyloid A (SAA), C - reactive protein (CRP), Procalcitonin (PCT) and Lipopolysaccharide-binding

248 lood pressure, increased C-reactive protein (CRP), reduced HDL, insulin resistance as well as increas

249 e was the serum level of C-reactive protein (CRP), serum amyloid P (SAP), and alpha-2-macroglobulin (

250 s were changes in plasma C-reactive protein (CRP), the diversity of the faecal microbiota, quality of

251 nding protein (LBP), and C-reactive protein (CRP), were elevated in sera, and correlated positively w

252 es for the assessment of C-reactive protein (CRP).

253 iR-93-3p correlated with C-reactive protein (CRP).

254 for the determination of C-reactive Protein (CRP).

255 r-Alpha (TNF-alpha), and C-reactive protein (CRP)] in community samples, both unadjusted and adjusted

256 markers of inflammation (C-reactive protein [CRP] level and erythrocyte sedimentation rate [ESR]), ca

257 (as shown by an elevated C-reactive protein [CRP] level).

258 -1, procalcitonin [PCT], C-reactive protein [CRP]) activation pathways were determined in consecutive

259 in acute phase proteins (C-reactive protein [CRP], alpha-1-acid glycoprotein [AGP]) and biomarkers of

260 irectional association between CRP and PTSD (CRP -> PTSD: beta = 0.065, p = 0.015; PTSD -> CRP: beta

261 rence method, especially at low to mid-range CRP values.

262 However, 4 patients did not show rapid CRP declines, of whom 3 had poorer outcomes.

263 DBS sample CRP measurements were made by enzyme-linked immunosorben

264 between 'minimal' baseline and 6 week sample CRP data and the 'continuous' weekly CRP data.

265 flammation (interleukin-6 and high-sensitive CRP) confirmed results.

266 s of IL (interleukin)-6 and high-sensitivity CRP (C-reactive protein).

267 High-sensitivity CRP and low-density lipoprotein cholesterol (LDL-C) were

268 I score (P = 0.039), BOP% (P = 0.023), serum CRP level (P < 0.001) and oral polymorphonuclear neutrop

269 We investigated the relationship among serum CRP, PTSD, and traits related to traumatic events and so

270 y oPMN (beta = - 0.244, P = 0.021) and serum CRP (beta = - 0.226, P = 0.019) were included in the bes

271 nd between IBW and OIL (P = 0.006) and serum CRP (P < 0.001).

272 t of FC and higher than measurement of serum CRP.

273 The serum CRP and SAP increases correlated with tau post-CPB level

274 s further detected in human serum by spiking CRP to run-through the detection with the physiologicall

275 0.81-0.92) and was significantly better than CRP (P < .0001) and PCT (P = .0001).

276 Our results indicate that CRP and PCT are not suitable to distinguish UTI and ASB

277 Additionally, we show that CRP regulates expression of sirA via the newly identifie

278 Our results suggest that it is unlikely that CRP (C-reactive protein) and vitamin D play causal roles

279 dence for eight distinct associations at the CRP locus, including two variants that have not been ide

280 Median antibiotic duration in the CRP group was 7 (interquartile range, 6-10; range, 5-28)

281 occurred in 4 of 164 (2.4%) patients in the CRP group, 11 of 166 (6.6%) in the 7-day group, and 9 of

282 occurred in 10 of 143 patients (7.0%) in the CRP group, 16 of 151 (10.6%) in the 7-day group, and 16

283 and wide range of treatment durations in the CRP-guided group.

284 and the median time to normalization of the CRP level was 7 days.

285 that in ventricles, inversely associated to CRP and IL (interleukin)-1 changes in acute infection pa

286 were typically higher in colonies exposed to CRP environments.

287 longed but rapidly normalized in parallel to CRP (C-reactive protein) and cytokine level reduction.

288 er SD higher usual levels of log-transformed CRP and fibrinogen.

289 ase in genetically-predicted log-transformed CRP was 1.18 (95% CI 1.07-1.29; p = 0.0009).

290 sample CRP data and the 'continuous' weekly CRP data.

291 When CRP was evaluated using tertiles, no associations with o

292 ients who were prescribed an antibiotic when CRP concentrations were above and below the 20 mg/L or 4

293 les 95% CI [0.072, 0.775], particularly when CRP was also elevated 95% CI [0.672, 1.551].

294 ion of OC use was positively associated with CRP levels in premenopausal women (P < 0.0001).

295 order to discover novel loci associated with CRP levels, we examined a multi-ancestry population (n =

296 ing osteopontin alone or in combination with CRP modestly improved the predictive ability of ACS beyo

297 prospective associations of depression with CRP/CRP with depression were substantially attenuated an

298 P) was observed: 1) exclude individuals with CRP > 5 mg/L or AGP > 1 g/L; 2) apply arithmetic correct

299 al variables such as probing depth (PD) with CRP and fibrinogen.

300 In WRA, the association of PZC with CRP and AGP was weak and inconsistent.