ライフサイエンスコーパス: CSH

1 CSH also enhances NE inhibition of glutamate release fro

2 CSH enhanced the alpha2-adrenoreceptor agonist clonidine

3 CSH exposure increased evoked EPSC (eEPSC) amplitude via

4 CSH is associated with a significantly increased risk of

5 CSH was defined as a post-procedure hematoma requiring f

6 CSH was the only independent predictor and was associate

7 o retained heterozygosity (0.91; 0.56-1.48), CSH p=0.039.

8 A CSH approach should be used in conjunction with overall

9 xed-mode reversed-phase chromatography, on a CSH Phenyl-Hexyl column, to accomplish mixed-mode anion-

10 drenoreceptor antagonist yohimbine abolished CSH-induced enhancement of NE inhibition of eEPSCs.

11 H2O2 oxidizes capping agent CSH, modulating the growth of CSH-stabilized cadmium sul

12 y (CSH) influences virulence of C. albicans, CSH properties of C. dubliniensis were investigated and

13 oxic controls; i.e. acute hypoxia in CON and CSH, and normoxia in CSH.

14 increased ventilation 25% and 50% in CON and CSH, respectively, while NMDA doubled ventilation in bot

15 metabolomic platforms (GC-TOF MS, HILIC- and CSH-QExactive MS/MS), freely available metabolite annota

16 This column chemistry, known as CSH (charged-surface hybrid) C18, improves upon an alrea

17 ionary phase with a charged surface, such as CSH C18, holds significant promise for facilitating chal

18 nding the ASH domain and CoA binding the ASH-CSH interface to produce acetyl-CoA and oxaloacetate pro

19 eins, the lack of these virulence-associated CSH entities in C. dubliniensis could contribute to its

20 ospectively examined the association between CSH and subsequent device infection.

21 en non-accidental falls and cardioinhibitory CSH.

22 tly sensitized PN-, walnut- (WN) and cashew (CSH)-allergic mice received 1-day PN/WN/CSH rush OIT plu

23 -free and selective detection of cysteamine (CSH).

24 nsor for the electrooxidation of l-cysteine (CSH) in aqueous media.

25 ystem is based on the oxidation of cysteine (CSH) with hydrogen peroxide (H2O2) enzymatically generat

26 dentify prognostic factors for the following CSH rates: intestinal graft failure (IGF)/death due to r

27 The detection limit for CSH is 150nM (S/N=3).

28 methanol + NH(3) modifier, particularly for CSH PFP and, unexpectedly, also for PGC.

29 ther developed as a new detection scheme for CSH by chronoamperometry method and under optimized cond

30 he BSAGNCs allow sensitive detection of free CSH in the range of 500-10,000nM.

31 The use of a cause-specific hazard (CSH) approach may provide more precise identification an

32 A multivariable cause-specific hazard (CSH) rate analysis using Cox stepwise regression was per

33 e of clinically significant pocket hematoma (CSH).

34 on reduced clinically significant hematomas (CSH) by 80% compared with heparin bridging (3.5% versus

35 tios with the chi(2) test for heterogeneity (CSH).

36 motetrameric core citrate synthase homology (CSH) module flanked by acyl-CoA synthetase homology (ASH

37 amer with a rigid citrate synthase homology (CSH) module, flanked by four flexible acetyl-CoA synthet

38 in and human chorionic somatotropin hormone (CSH) produced by the placental trophoblastic cells.

39 layer, concurrent calcium silicate hydrate (CSH) alteration to an amorphous zeolite and Ca-carbonate

40 Calcium silicate hydrate (CSH) is the main binding phase of Portland cement, the s

41 As cell surface hydrophobicity (CSH) influences virulence of C. albicans, CSH properties

42 ioinhibitory carotid sinus hypersensitivity (CSH).

43 ter a chronic exposure to sustained hypoxia (CSH) (7-8 d at 10% FIO(2)).

44 With chronic sustained hypoxia (CSH), such as during acclimatization to high altitude, a

45 bably glutamate receptor modification, as in CSH rats the expression of phosphorylated NR1 and GluR1

46 use, there was no significant difference in CSH observed between direct oral anticoagulant use compa

47 No difference in CSH was found between direct oral anticoagulant versus c

48 oM) elicited greater inhibition of eEPSCs in CSH cells (63 +/- 2%; n = 16) than NORM cells (45 +/- 3%

49 ats, and completely blocked the acute HVR in CSH rats but had no effect on ventilation in normoxia.

50 (50) of NE inhibition of eEPSCs was lower in CSH cells (3.0 +/- 0.9 microM; n = 5) than in normoxic (

51 cute hypoxia in CON and CSH, and normoxia in CSH.

52 e and bonding of a large subset of the known CSH minerals.

53 ining heterozygosity than in those with LOH (CSH p=0.02).

54 1) the use of a new charge-surface-modified (CSH) C18 stationary phase to mitigate the challenges of

55 eated rats held in normoxia (CON) or 10% O2 (CSH) for 7 days and measured ventilation in conscious, u

56 Co/GCE) improves electrochemical behavior of CSH oxidation, as compared to the GO and PcCo.

57 ue to the complex structure and chemistry of CSH at various length scales, the focus has progressivel

58 a synergic effect in the electrooxidation of CSH.

59 capping agent CSH, modulating the growth of CSH-stabilized cadmium sulphide quantum dots (CdS QDs).

60 ant antiplatelet therapy doubled the risk of CSH during device surgery.

61 CSH, and (2) understand the relative risk of CSH in patients treated with direct oral anticoagulant v

62 CT01675076) observed a similarly low risk of CSH when comparing continued versus interrupted direct o

63 Placental secretion of CSH into maternal circulation was reduced by 80% compare

64 The sensing mechanism is based on CSH etching-induced fluorescence quenching of the bovine

65 ffect of concomitant antiplatelet therapy on CSH, and (2) understand the relative risk of CSH in pati

66 ), charged surface hybrid pentafluorophenyl (CSH PFP), and porous graphitic carbon (PGC) as a nonsili

67 ental nutrient transport function, placental CSH production and uterine blood flow.

68 d in 11% of patients (7 of 66) with previous CSH and in 1.5% (9 of 593) without CSH.

69 al secretion of chorionic somatomammotropin (CSH), and targeted placental gene expression were measur

70 d on separations of a nine-peptide standard, CSH C18 was found to exhibit improved loadability, great

71 tural amino acids do not interfere with such CSH-induced etching process.

72 These results demonstrate that CSH increases evoked excitatory inputs to NTS neurons re

73 It is found that CSH can etch the Au25 nanoclusters, exhibiting the poten

74 Previous studies have shown that CSH status of C. albicans involves multiple surface prot

75 The CSH approach should be more widely used in investigation

76 temperature is one factor which affects the CSH status of stationary-phase C. albicans.

77 homology (ASH) domains; CoA is bound at the CSH-ASH interface in mutually exclusive productive or un

78 The SEM confirmed the CSH gel formation in the mixtures containing bentonite a

79 this data support an allosteric role for the CSH module in ACLY catalysis.

80 main, with an additional oxaloacetate in the CSH domain, which could function in ACLY autoinhibition.

81 nd can load CoA and unload acetyl-CoA in the CSH module.

82 Since protein glycosylation influences the CSH status of C. albicans, we compared the cell wall man

83 The specific catalytic role of the CSH module and an essential D1026A residue contained wit

84 ed out towards a better understanding of the CSH-induced fluorescence quenching of the BSAGNCs.

85 associated with graft/patient survival, the CSH analysis more precisely identified their prognostic

86 nges represent central neural adaptations to CSH.

87 econdary increase persists after exposure to CSH and involves plasticity within the circuits in the c

88 krusei, C. parapsilosis, and C. tropicalis), CSH status correlated with coaggregation with the anaero

89 ition (40 microM; NORM, 36 +/- 2%, n = 11 vs CSH, 26 +/- 4%, n = 6; p < 0.05).

90 ibition (3 microM; NORM, 23 +/- 2%, n = 5 vs CSH, 44 +/- 5%, n = 4; p < 0.05) but attenuated alpha1-a

91 The primary outcome for both trials was CSH.

92 ough application to peptide mapping, wherein CSH C18 was found to aid the development of a high-resol

93 elet use (versus nonuse) was associated with CSH in 9.8% versus 4.3% of patients (P<0.001), and remai

94 NTS contribute to plasticity in the HVR with CSH.

95 previous CSH and in 1.5% (9 of 593) without CSH.

96 hew (CSH)-allergic mice received 1-day PN/WN/CSH rush OIT plus 3 weeks of maintenance dosing, with or

97 An XSelect CSH C18 column with gradient elution using phosphate buf