ライフサイエンスコーパス: CVA

1 CVA estimates those multivariate patterns of activation

2 CVA is a physiological phenomenon of importance to healt

3 CVA was used to extract the duration of time the child o

4 CVAs were noted in 6 cases.

5 dex (h(s)2 = 0.212, SE = 0.1347, P = 0.0611; CVA = 0.1826).

6 els in 102 eyes (88%), AVD in 81 eyes (74%), CVA in 107 eyes (94%), and CVH in 100% of affected eyes.

7 ienced CVAs, as compared with 3 deaths and 9 CVAs in the 25 patients who remained on intensified anti

8 A CVA occurred in 92 patients (0.30% of procedures).

9 A CVA occurred in the 12 months following HZO in 14 patien

10 A CVA was defined as a composite of transient ischemic att

11 with the experimental design; the order of a CVA model is then determined by the number of significan

12 rtality for BD and cerebrovascular accident (CVA) for mixed SMI.

13 failure (CHF), or cerebrovascular accident (CVA) from 1991 to 1994.

14 s had at least one cerebrovascular accident (CVA) in follow-up; 35 patients had at least one CVA or t

15 and prevalence of cerebrovascular accident (CVA) in paced versus unpaced patients during admission t

16 gher prevalence of cerebrovascular accident (CVA) in patients with SEC when compared with controls wi

17 Cerebrovascular accident (CVA) is a major complication of sickle cell disease.

18 irst occurrence of cerebrovascular accident (CVA) or death.

19 CVIs, freedom from cerebrovascular accident (CVA) or transient ischemic attack (TIA), and 30-day mort

20 increased risk of cerebrovascular accident (CVA) remains uncertain.

21 9), and history of cerebrovascular accident (CVA) was significantly inversely related to SFCT (coeffi

22 ication were prior cerebrovascular accident (CVA), chronic obstructive pulmonary disease (COPD), type

23 d occlusion, prior cerebrovascular accident (CVA), or SPs less than 35 mmHg.

24 tive/postoperative cerebrovascular accident (CVA), postoperative bleeding, and sternal wound infectio

25 nfarction (MI), or cerebrovascular accident (CVA).

26 increased risk of cerebrovascular accidents (CVA) in individuals with PHACES, yet the precise causes

27 arctions (MI) and cerebrovascular accidents (CVA) in patients with diabetic macular edema (DME) compa

28 Cerebrovascular accidents (CVA) or ischemic heart disease (IHD) after SJS/TEN survi

29 </= 70 years from cerebrovascular accidents (CVA) or trauma that were correlated with eligible deaths

30 ry disease (CAD), cerebrovascular accidents (CVA), chronic obstructive pulmonary disease (COPD), asth

31 ry disease (CAD), cerebrovascular accidents (CVA), diabetes mellitus (DM), and end-stage renal diseas

32 isease (CVD), and cerebrovascular accidents (CVA).

33 increased risk of cerebrovascular accidents (CVA).

34 l implications of cerebrovascular accidents (CVAs) after percutaneous coronary interventions (PCIs).

35 red high-risk for cerebrovascular accidents (CVAs) in PHACES patients.

36 the incidence of cerebrovascular accidents (CVAs).

37 ime (CFIX), and amoxicillin-clavulanic acid (CVA-AMPC), 21.2% were confirmed to be authentic, 91.3% m

38 t visual acuity (LCVA), color visual acuity (CVA), and visual fields (VF).

39 hara test) and a sub-sample of 6 CVD adults (CVA-UMinho colour discrimination and colour naming tasks

40 24 months by traditional increment analysis (CVA & FOTI at the D(3) (dentin only) threshold + radiogr

41 ) combined with Canonical Variance Analysis (CVA) revealed that this association was mediated indirec

42 t controls using Canonical Variate Analysis (CVA) provides several distinct clusters for each scenari

43 lution based on Canonical Variates Analysis (CVA) model scoring at the subject level and random effec

44 ovel methods using computer vision analysis (CVA) for automatically quantifying patterns of attention

45 On multivariate analysis, CVA was independently associated with in-hospital death

46 (ANOVA) approach, report on the analytical (CVA), intraindividual (CVI), and between individual (CVG

47 to choroidal intervortex venous anastomoses (CVAs) accompanied by choroidal vascular hyperpermeabilit

48 AHR, 1.28; 95% CI, 1.21-1.42; P < .001), and CVA (AHR, 1.22; 95% CI, 1.17-1.33; P < .001) compared wi

49 alence of pachyvessels (94%), AVD (67%), and CVA (90%) was similar to affected eyes, whereas CVH was

50 Increase of isolation of EV-A71 and CVA--the predominant pathogens for the hand, foot, and m

51 iac surgery who have risk factors for AF and CVA.

52 e frequencies of hospitalization for AMI and CVA did not differ from those of the comparison group, b

53 Although pachyvessels, AVD, and CVA are observed frequently, CVH was observed in all aff

54 ubstantially increased risk of AMI, CHF, and CVA.

55 t hypothermia and normoxia, TVR was high and CVA unit activity was present, with marked respiratory m

56 ntrast, during hyperthermia, TVR was low and CVA unit activity was absent.

57 adjusted rate ratios of hospitalized MI and CVA events were then calculated.

58 decrease of >/= 7 mum predicted poor VF and CVA (sensitivity of 78% and 100% and specificity of 63%

59 sed for FHP using the craniovertebral angle (CVA) and separated into either the normal head posture (

60 g the chorioallantois vaccinia virus Ankara (CVA) vaccine strain in chicken embryo fibroblasts during

61 innervating the caudal ventral tail artery (CVA) of anaesthetised rats can still be recorded followi

62 units supplying tail caudal ventral artery (CVA) in spontaneously breathing anaesthetized rats, whil

63 was assessed by clinical visual assessment (CVA-simplified version of Dundee Selectable Threshold Me

64 least one CVA or transient ischemic attack (CVA/TIA).

65 employed in cerebrovascular autoregulation (CVA) tests on the brain, without contact and in real tim

66 migration along the mouse crypt-villus axis (CVA).

67 ere in sickle cell anemia (SS) patients, but CVA occurred in all common genotypes.

68 th Fast Fourier Transform (FFT), followed by CVA treatment.

69 ither the normal head posture (NHP) group by CVA > 50 degrees or the FHP group by CVA < 50 degrees .

70 roup by CVA > 50 degrees or the FHP group by CVA < 50 degrees .

71 e assay was compared with culture on a Campy-CVA plate (Remel, Lenexa, Kans.) and blood-free campylob

72 A prognostic risk score (CARRS [CVA, albumin, re-HTx, renal dysfunction, and sternotomie

73 P; deaths from CVA: 34-118 PMP; and combined CVA and trauma: 91-229 PMP.

74 mized and propensity-score studies comparing CVA in patients without AF undergoing cardiac surgery wi

75 s, Donor type (DCD or DBD), cause of Death = CVA, serum creatinine, Age, height, and weight (length).

76 8]) and death due to cardiovascular disease (CVA: HR, 1.69; 95% CI, 1.46-1.96; IHD: HR, 1.55; 95% CI,

77 nd contralateral vestibuloacoustic efferent (CVA) neurons are incorrectly specified.

78 nt, the incidence of cerebrovascular events (CVA), a history of deep venous thrombosis (DVT) and pulm

79 treatment, 1 patient died and 2 experienced CVAs, as compared with 3 deaths and 9 CVAs in the 25 pat

80 ntly associated with actuarial rate of first CVA included hypertension (P = .002), age (P < .0001), c

81 = 2.40 (95% CI: 2.25 to 2.55, p < 0.001) for CVA in schizophrenia.

82 interval (CI): 1.33 to 1.81, p < 0.001), for CVA in people with BD to HR/rate ratio = 2.40 (95% CI: 2

83 o 2.37) for CVA and 1.68 (1.003 to 2.06) for CVA/TIA.

84 ncidence ratios were 1.74 (0.94 to 2.37) for CVA and 1.68 (1.003 to 2.06) for CVA/TIA.

85 to 2.46) for MI, 1.28 (CI, 1.06 to 1.54) for CVA, and 2.13 (CI, 1.61 to 2.81) for PVD.

86 isk per 1,000 patients per year was 1.67 for CVA and 2.76 for CVA/TIA.

87 ients per year was 1.67 for CVA and 2.76 for CVA/TIA.

88 The Cox regression analysis for CVA in patients undergoing LAAO reported a hazard ratio

89 anatomic distribution, and risk factors for CVA in patients with UC.

90 idence and mortality of and risk factors for CVA in sickle cell disease patients in the United States

91 nd atrial fibrillation were risk factors for CVA in the m-UC cohort, but only age was associated with

92 nd atrial fibrillation were risk factors for CVA in univariate analysis for both sexes.

93 1-1.49), and the risk of hospitalization for CVA was not significantly different from those in the co

94 HR for MI, 11.89 [CI, 2.40 to 59.00]; HR for CVA, 3.93 [CI, 1.76 to 8.79]; HR for PVD, 3.86 [CI, 0.78

95 The adjusted rate ratio for CVA was 1.98 (95% CI: 1.39-2.83, p < 0.001) for DME vers

96 The HR/rate ratios for CVA and CHD were larger in studies with outcomes occurri

97 lar assist device patients at major risk for CVA and death.

98 UC patients have an increased risk for CVA, with women over 80 demonstrating the highest risk.

99 The most common locations for CVAs in UC patients were frontal and occipital lobes (19

100 OPOs: trauma deaths: 44-118 PMP; deaths from CVA: 34-118 PMP; and combined CVA and trauma: 91-229 PMP

101 The 1-year freedom from CVA or death was 87.5% and 49.5% in the surgical and med

102 sis, there was no difference in freedom from CVA or TIA for the 2 groups (P = .07).

103 ion, but there were no consistent changes in CVA unit firing rate or T-rhythm frequency, although res

104 The incidence of infarctive CVA was lowest in SS patients 20 to 29 years of age and

105 There were 19 ipsilateral CVAs (2.7%).

106 s is associated with increased risks for MI, CVA, and PVD.

107 n age, 73 years), the incidence rates of MI, CVA, and PVD were 10.0, 8.0, and 4.2 events per 1000 per

108 lyses were conducted for the outcomes of MI, CVA, and PVD.

109 In wild-type mice CVA neurons migrate from r4 into the contralateral side,

110 ad higher risks of cardiovascular morbidity (CVA: HR, 1.65 [95% CI, 1.57-1.72] and subdistribution HR

111 es more likely to be hospitalized because of CVA (95% CI 1.17-2.93).

112 n independent and more powerful correlate of CVA than reduced LAAEV or atrial size.

113 prompt acyclovir had a 76.2% lower hazard of CVA (0.9% vs 2.6%, P = .022) on multivariate analysis.

114 Hazard of CVA was highest immediately following HZO, with median t

115 ex interaction, pseudophakia, and history of CVA were significantly associated with SFCT in the elder

116 mia, Charlson comorbidity scores, history of CVA, hyperlipidemia, and other cerebrovascular diseases.

117 used to estimate the hazard ratios (HRs) of CVA and IHD morbidity and mortality after SJS/TEN surviv

118 also associated with increased incidence of CVA and CHD relative to control participants since the 1

119 At 10 years, the cumulative incidence of CVA was 6.3% (4.6%-8.1%) versus 3.7% (2.9%-4.5%) in pati

120 The incidence of CVA, CVD events, and heart failure in SMI was higher tha

121 e of anticoagulation and higher incidence of CVA.

122 Age was the only significant predictor of CVA alone (P < .001).

123 pAF was an independent predictor of CVA at 10 years (hazard ratio, 1.53 [95% CI, 1.06-2.23];

124 Predictors of CVA events were cardiac arrhythmia, Charlson comorbidity

125 P = .003) remained significant predictors of CVA/TIA.

126 The highest rates of prevalence of CVA (4.01%) and incidence (0.61 per 100 patient-years) w

127 The prevalence of CVA in UC patients (4.7%, 95% CI 3.9-5.6) was higher tha

128 patients with SEC had a higher prevalence of CVA than controls, p = 0.03.

129 SR is associated with a higher prevalence of CVA.

130 The rate of CVA or TIA in the iBCVI group was 5.1% compared with 15.

131 e-specific prevalence and incidence rates of CVA in patients with the common genotypes of sickle cell

132 etic retinopathy (NPDR) had a higher risk of CVA (hazard ratio [HR], 1.31; 95% confidence interval [C

133 ssociation between pAF and long-term risk of CVA by performing a post hoc analysis of 10-year outcome

134 matologic and clinical events on the risk of CVA were analyzed.

135 significantly associated with future risk of CVA, MI, CHF, and death, with higher degrees of retinopa

136 LAAO was associated with a decreased risk of CVA, no difference in the incidence of postoperative atr

137 the association between pAF and the risk of CVA.

138 artery bypass grafting are at higher risk of CVA.

139 seline hypertension led to increased risk of CVA/TIA (log-rank P < .0001).

140 The most common type of CVA was ischemic stroke (77.7%).

141 Prevalence of CVAs in patients with UC was compared to that of the gen

142 ) in follow-up; 35 patients had at least one CVA or transient ischemic attack (CVA/TIA).

143 Event rates of MI or CVA were higher in patients with DME than in diabetes co

144 ated with an increased risk of death, MI, or CVA compared with patients who were aspirin sensitive (2

145 vere NPDR had a higher risk of each outcome (CVA: HR, 1.56; 95% CI, 1.29-1.89; MI: HR, 1.92; 95% CI,

146 Among them, 19 had clinically overt CVA, 20 had life-threatening hypoxic episodes or soft ne

147 ient (FSIQ) was associated with either overt CVA or silent ischemic lesions.

148 Among the 30 subjects with no overt CVA, 25 (83%) demonstrated imaging abnormalities based o

149 Of the 19 subjects with overt CVA, 17 had abnormal MRI (89%), 17 had abnormal PET (89%

150 PC-CVA improved clustering, provided clearer loadings, and

151 al components-canonical variate analysis (PC-CVA), and Random Forests (RF) data analysis with the aim

152 The combination of PC-CVA and RF allowed very subtle differences between bacte

153 umbers and while showing overlap with the PC-CVA, produced additional peaks of interest.

154 cteristics of those who had a periprocedural CVA were compared with those who did not.

155 s ratio [OR], 1.16; P=.261) or postoperative CVA (adjusted OR, 1.06; P=.765), risks of sternal wound

156 1.9, 95% CI 1.1 to 3.3; p = 0.033), previous CVA (OR 2.3, 95% CI 1.3 to 4.0; p = 0.0059), and creatin

157 nd renal disease) and 4 (older with previous CVA), and those subgroups had a higher frequency of pre-

158 The prevalence of prior CVA was not significantly different between the three gr

159 arin is contraindicated and history of prior CVA was studied in three groups: 1) group A with continu

160 nts with proliferative diabetic retinopathy (CVA: HR, 2.53; 95% CI, 1.84-3.48; MI: HR, 1.89; 95% CI,

161 ) there was recommencement of firing in some CVA units, at low discharge rate, with respiratory modul

162 ACE), coronary artery disease (CAD), stroke (CVA), heart failure (HF), renal failure (RF), diabetic n

163 risk for cerebral vasculopathy with stroke (CVA) and cognitive impairment.

164 nt for HZO may reduce the risk of subsequent CVA when given within 72 hours of rash onset.

165 hihara and Farnsworth-Munsell 100 hue tests, CVA-UMinho colour discrimination and colour naming tasks

166 The CVA cohort included 10 571 SJS/TEN survivors (mean [SD]

167 The CVA rates at 5 years were 6.8+/-1.0% in the no-LAAO grou

168 ession of lineage-specific markers along the CVA consistent with transcription site repression of the

169 the normal reprogramming of cells along the CVA was dampened in the Apc(1638N/+) mice, with an overr

170 mutants, motor neurons differentiate but the CVA and FBM neurons fail to migrate into their proper po

171 flow and vascular resistance (TVR) from the CVA, under conditions of modest hypothermia and hyperthe

172 thy status and the 5-year risk of first-time CVA, MI, CHF, and all-cause mortality was investigated u

173 xia in TVR and sympathetic nerve activity to CVA are dependent on core temperature.

174 This approach can also be applied to CVA models with a fixed number of canonical vectors but

175 HACES syndrome as a potential contributor to CVA.

176 mediately following HZO, with median time to CVA of 2.3 months (IQR 0.8-5.9 months).

177 Of the 15 total CVAs or TIAs, 11 (73.3%) resulted from carotid injury an

178 s suggest acceptable analytical variability (CVA < or = 1/2 CVI) for all assays.

179 1.53 [95% CI, 1.06-2.23]; P=0.025) even when CVAs that occurred during the index admission were exclu

180 No increased risk was seen for adults with CVA.

181 -UC cohort, but only age was associated with CVA in f-UC.

182 , SEC in NSR was found to be associated with CVA, larger LA size and decreased mean LAAEV.

183 Factors associated with CVA/TIA were hypertension (P < .001), coronary artery di

184 p = 0.0285; data not shown) co-existed with CVA incidence.

185 adults with DM or those aged <65 years with CVA.

186 ent arteries (p = 0.01) were associated with CVAs, with severe tortuosity being the strongest predict

187 olution of a primary hemolysis event without CVA or death occurred in 21/24 patients treated with sur