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1 ly eliminates tumor cells overexpressing the CXCR4 receptor.
2 cally targeting the ligand binding pocket of CXCR4 receptor.
3 ds, confirming a direct interaction with the CXCR4 receptor.
4 CD39 and the multifunctional, mTOR-inducing CXCR4 receptor.
5 y, was also dependent on the presence of the CXCR4 receptor.
6 ive to TFF2 treatment upon expression of the CXCR4 receptor.
7 acts with the G protein, seven-transmembrane CXCR4 receptor.
8 (SDF-1) is a CXC chemokine that binds to the CXCR4 receptor.
9 of CD45 and induced its association with the CXCR4 receptor.
10 y virus gp120-induced internalization of the CXCR4 receptor.
11 at least overlapping binding site(s) on the CXCR4 receptor.
12 F-1) is an alpha chemokine that binds to the CXCR4 receptor.
13 l line-adapted, strains (X4 strains) use the CXCR4 receptor.
14 ocytic leukemia cell lines all expressed the CXCR4 receptor.
15 ng of free SDF1 or the fusion protein to the CXCR4 receptor.
16 in injured renal tubules overexpressing the CXCR4 receptor.
17 hemokine heterodimer binds and activates the CXCR4 receptor.
18 dimeric beta1-adrenoreceptor, mu-opioid, and CXCR4 receptors.
19 ndocytosis and signaling of WT and WHIM-like CXCR4 receptors.
20 periphery: endocytosis of TCRs and chemokine CXCR4 receptors.
21 potentially capable of interacting with two CXCR4 receptors.
22 bodies (IgM-ALA) bind to CD3, CD4, CCR5, and CXCR4 receptors.
23 sis as well as for physical interaction with CXCR4 receptors.
24 tly evolve to T-tropic viruses (X4) that use CXCR4 receptors.
25 ulated the proportion of cells with CCR5 and CXCR4 receptors.
26 , HUT78, CEM, and Sup-T1 for the presence of CXCR4 receptors.
27 s T-cell-tropic and dual-tropic isolates use CXCR4 receptors.
29 se ligation of the CD4 receptor alone or the CXCR4 receptor alone causes p38 activation and apoptosis
31 ngs suggest that in addition to CXCR4, a non-CXCR4 receptor and cell-surface heparans also play an im
33 tic structural motifs that interact with the CXCR4 receptor and induce apoptosis in CD4(+) lymphocyte
34 L12 is a human chemokine that recognizes the CXCR4 receptor and is involved in immune responses and m
35 ecules that will enable further study of the CXCR4 receptor and may contribute to the development of
40 ther GPCRs such as beta2-adrenergic, FSH and CXCR4 receptors, but does not affect the beta-arrestin-i
44 tin polymerization may directly regulate the CXCR4 receptor during viral entry and is involved in vir
45 and in vitro and explored whether SDF-1alpha/CXCR4 receptor engagement promotes HSC activation, fibro
46 unit of NF-kappaB in PC-3 cells up-regulated CXCR4 receptor expression and increased the adhesion and
47 lpha-induced NF-kappaB reporter activity and CXCR4 receptor expression as shown by flow cytometry.
50 We found that CLL B cells express functional CXCR4 receptors for the chemokine stromal cell-derived f
51 e cannabinoid system can negatively modulate CXCR4 receptor function and perhaps tumor progression.
52 oth the human and the murine variants of the CXCR4 receptor (half-maximal inhibitory concentration, 8
54 ent probes that show specific binding to the CXCR4 receptor in a novel fluorescence-based NanoBRET bi
60 opulation of progenitor cells that expressed CXCR4 receptors in vitro and differentiated into neurons
62 kine expression in HPC progenies showed that CXCR4 receptor is detected on the majority of MKs from e
65 Fc to primary T cells, suggesting that a non-CXCR4 receptor is involved in the binding of FIV SU.
66 derived factor-1 (SDF-1), the ligand for the CXCR4 receptor, is a highly efficacious chemoattractant
67 tor 1 (SDF-1), the only known ligand for the CXCR4 receptor, is expressed close to these migration si
71 ine CXCL12 via a concentration-dependent and CXCR4 receptor-mediated mechanism, termed chemorepulsion
72 We studied the effects of Lyn ablation on CXCR4 receptor-mediated migration and adhesion of hemato
73 se results suggest that JAK2 is required for CXCR4 receptor-mediated signaling that regulates cytoske
75 f CXCR4 disrupts the interaction between the CXCR4 receptor on hematopoietic stem cells (HSCs) and th
77 aqueous liposome chamber and then bound with CXCR4 receptors on the tumor cell surface to inhibit met
78 tor, site-specific mutagenesis, hybrid CXCR3/CXCR4 receptors, pharmacological reagents, peptide array
79 rove useful as a diagnostic tool to identify CXCR4 receptor-positive tumor cells in culture and tumor
81 eceptor CCR7, and the down-regulation of the CXCR4 receptor provide a mechanistic basis of Ab-induced
83 or AMD3100, to delineate the role of CD4 and CXCR4 receptors, respectively, in gp120-induced p38 acti
86 myces cerevisiae that expresses a functional CXCR4 receptor, site-specific mutagenesis, hybrid CXCR3/
87 l small molecule selective antagonist of the CXCR4 receptor that increases mobilization and trafficki
88 as been shown to be an imaging agent for the CXCR4 receptor that is likely to be applicable across a
90 l role of signaling molecules connecting the CXCR4 receptor to the process of hematopoietic migration
92 To target tumor cells that overexpress the CXCR4 receptor, we linked the CXCR4 DV3 ligand to two tr
93 ometry and lipoparticles containing only the CXCR4 receptor, we quantified the binding affinity for t
94 rom the N-terminal extracellular tail of the CXCR4 receptor, we show that the principal determinants
96 Models of the adenosine A2AR and chemokine CXCR4 receptors were first ranked in GPCR-DOCK blind pre
98 utgrowth of viruses capable of utilizing the CXCR4 receptor (X4 viruses), the existence of three dist