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1 CYP11A1 (rate-limiting enzyme) was expressed in cancerou
2 CYP11A1 and CYP27A1 hydroxylate tachysterol(3) , a photo
3 CYP11A1 expression was stable, CYP17A1 increased, and HS
4 CYP11A1 is involved in the first step in the steroidogen
5 CYP11A1 primarily binds its substrate 20R,22R-dihydroxyc
6 CYP11A1, CYP17A1, HSD3beta, and HSD17beta3 were identifi
9 R), cytochrome P450 family 11, subfamily A1 (CYP11A1) and 3 beta-hydroxysteroid dehydrogenase type 1
12 reduced androgen biosynthesis and CYP17 and CYP11A1 mRNA when added to the medium of cultured PCOS t
13 in a PCOS phenotype of augmented CYP17A1 and CYP11A1 gene transcription, mRNA abundance, and androgen
15 xides induced spectral shifts in CYP27A1 and CYP11A1 but glycol metabolites were detected only with C
16 25-hydroperoxide to the enzymes CYP27A1 and CYP11A1 induced well-defined spectral changes while gene
18 ightly associated with the membrane, such as CYP11A1, can be quantified in the total tissue membrane
20 the context of a modeled interaction between CYP11A1 and the reduced (functional) form of Adx in whic
21 gated whether there exist contact(s) between CYP11A1 and Adx that are differentially influenced by ch
22 be the metabolism of 7-dehydrocholesterol by CYP11A1 to a single product identified as 7-dehydropregn
24 oduction in glial cells was not inhibited by CYP11A1 inhibitors DL-aminoglutethimide and ketoconazole
25 rocholesterol (7DHC) metabolism initiated by CYP11A1 and previously characterized in vitro, occur in
27 novel pathways of D3 metabolism initiated by CYP11A1, with the product profile showing organ/cell typ
29 red for androgen synthesis from cholesterol (CYP11A1, CYP17A1, HSD3beta, HSD17beta3) were investigate
30 m pathways with highly negatively correlated CYP11A1 and CYP1B1 expression in breast tissue from post
31 nd M77S and mutagenesis of the corresponding CYP11A1 contact (W418A) revealed differential effects to
32 mRNA levels of CGA, CGB, PPARG, CYP19A1, CYP11A1, PTGS2, EREG, and the intracellular beta subunit
34 CPR and FdR) and tightly associated (CYP7B1, CYP11A1, CYP27A1, and CYP46A1) membrane proteins were qu
36 ygen species, (2) metabolism via a different CYP11A1 isoform, and (3) metabolism via another CYP450 e
37 steroidogenesis pathway initiated by enzyme CYP11A1, and via the acidic bile acid pathway, which is
38 ntial actions of the cytochrome P450 enzymes CYP11A1 and CYP17A1, so that CYP17A1 inhibitors such as
42 ubstrate-specific contacts are important for CYP11A1 monooxygenation of vitamin D3 or side chain clea
43 ween genotypes of the cytochrome P-450 genes CYP11A1 (-528[TTTTA]n) or CYP17A1 (-34T/C) or the 17beta
44 SLC2A1, CYP17A1, RB1, IDS, GUSB, DMD, GLUD1, CYP11A1, GALNS, CPS1, PLPBP, ALDH7A1, SLC26A3, SLC25A15,
45 In placenta and adrenal glands with high CYP11A1 expression, the predominant pathway was D3 --> 2
49 controlling endogenous estrogen metabolism, CYP11A1 harbors common variants that may influence expre
50 Comparisons with cholesterol-metabolizing CYP11A1 and cortisol-producing CYP11B1, which also bind
51 dies have shown that substrates can modulate CYP11A1 protein-protein interactions with the redox part
53 lt, protein levels and enzymatic activity of CYP11A1, a steroidogenic enzyme regulating CD8(+) T-cell
60 n, we present the 2.5-A crystal structure of CYP11A1 in complex with the first reaction intermediate,
61 rome P450 side-chain cleavage enzyme (SCC or CYP11A1) catalyzes cholesterol to pregnenolone conversio
64 g purified mitochondrial cytochrome P450scc (CYP11A1) reconstituted with the iron-sulfer protein, adr
65 xpression of the steroidogenic gene products CYP11A1 and StAR in both H295R adrenal and MA-10 Leydig
66 ence that the circadian network may regulate CYP11A1 activity and by inference, the production of dow
77 tion of the role of genetic variation of the CYP11A1 gene in breast cancer risk in a study of 1193 br
79 Local androgen regulation in the skin of the CYP11A1 gene, which encodes a crucial enzyme that metabo
82 l steroid dependent and mediated by upstream CYP11A1-dependent intraturmoral pregnenolone/progesteron
83 expression was significantly correlated with CYP11A1 (P = 0.0009), HSD3beta (P = 0.0297), and HSD17be