ライフサイエンスコーパス: CaCl2

1 CaCl2 also improved the retention of Zn and P in the fle

2 CaCl2 destabilized GO more aggressively than MgCl2 and N

3 CaCl2 had no effect on troponin binding to the thin fila

4 CaCl2 improved both systolic and diastolic function and

5 CaCl2, but not neomycin or KC1, could overcome the inhib

6 mposition of 0.5% alginate, 400ml oil, 0.05M CaCl2 and 100ml surfactant was recognized as the optimiz

7 treatment of the wild-type T=3 virus with 1M CaCl2 and crystallized from sodium malonate.

8 c activity above 80% in the presence of 20mM CaCl2.

9 the selective precipitation of RNA with 1.4M CaCl2, followed by a final selective precipitation of th

10 Yield of lipid increased when 50mM CaCl2 was combined with CPE.

11 rodeposition of high-quality Si films from a CaCl2 -based molten salt.

12 Comparative metabolite profiling of a CaCl2 extract of the PRX33/PRX34 knockdown lines showed

13 eap and environmentally friendly Lewis acid, CaCl2, was used as a catalyst.

14 heat stability in autumn, whereas with added CaCl2, the best heat stability was observed in spring.

15 ion in cells shocked in buffer without added CaCl2 than in cells shocked in 200 or 500 microM CaCl2 o

16 ose in cells shocked in buffer without added CaCl2 were measured in cells following acid shock in buf

17 Adding CaCl2 had no effect on bioenergetics, except for a small

18 Adding CaCl2 made the milk more unstable to UHT than to in-cont

19 dependent increase in [Ca2+](m) after adding CaCl2 equivalent to 20, 114, and 485 nM extramatrix free

20 work, the impact was evaluated of alginate, CaCl2, oil and surfactant content on the size and encaps

21 study the influence of pH (3.5 and 7.0) and CaCl2 and MgCl2 addition on heat-set gelation of a quino

22 ion processes using wood ashes, Ca(OH)2, and CaCl2 on the physicochemical properties of beans.

23 ch as calcium gluconate, calcium acetate and CaCl2, had greater effects on the rate and extent of fre

24 tes in the presence or absence of CaCl2, and CaCl2 could not reduce pre-formed aggregates.

25 EDTA and CaCl2 gave a slight inhibition and DTT a slight enhancem

26 Forskolin and CaCl2 increased contraction similarly in WT and Gsalpha

27 idity of both modified starches improved and CaCl2 caused more evident changes.

28 sed (quadratic) as a function of the KCl and CaCl2 concentrations in the initial brines.

29 cts of the substitution of NaCl with KCl and CaCl2 on the physicochemical, mineral and sensory profil

30 in aqueous solutions by HCl, NaCl, KCl, and CaCl2 and in acetonitrile by CF3COOH and the supporting

31 ozonation and chloride salts (NaCl, KCl, and CaCl2).

32 Divalent cations such as MgCl2 and CaCl2 stimulate the binding of 125I-glucagon to the wild

33 In contrast, the MgCl2 and CaCl2 surface hydrate crystals are interdispersed with n

34 is active only in the presence of MgCl2 and CaCl2.

35 with individual salts (NaCl, KCl, MgCl2, and CaCl2) using deionized water with a known stable isotope

36 on effect) and salts (LiCl, NaCl, MgCl2, and CaCl2).

37 for four electrolytes: NaCl, KCl, MgCl2, and CaCl2.

38 Finally, prothrombin (1.2 microM) and CaCl2 (2 mM) could substitute for HK (45 nM) and ZnCl2 (

39 ininogen (HK, 45 nM), ZnCl2 (25 microM), and CaCl2 (2 mM), conditions optimal for factor XI binding t

40 In addition, MgCl2, MnCl2, and CaCl2 allowed some peptidase and caseinase activity in t

41 nder two relative flow rates with Na2CO3 and CaCl2 in the inner and outer fluids, respectively.

42 were determined in the presence of NaCl and CaCl2 (0, 50, 100, 150 and 200mM).

43 es the effect of including hydrated NaCl and CaCl2 (up to 6% w/w) on the physicochemical properties o

44 logenin as a function of increasing NaCl and CaCl2 concentration beginning with solution conditions o

45 ited on silica surfaces at elevated NaCl and CaCl2 concentrations before being rinsed with eluents of

46 subjected to transport with varying NaCl and CaCl2 concentrations.

47 stabilizing" effect of cooking with NaCl and CaCl2 on bean proteins.

48 Antiplasticising effects of NaCl and CaCl2 on the non-phosphorylated tapioca starch are indis

49 NaCl and CaCl2 water subphases are simulated.

50 n a variety of salt solutions (KCl, NaCl and CaCl2) at low salt concentrations (<10(-4) M) showed sev

51 reased with increasing electrolyte (NaCl and CaCl2) concentrations.

52 in the presence of different salts (NaCl and CaCl2), ionic strengths, pHs, and temperatures were cons

53 da tympani responses to NaCl, KCl, NH4Cl and CaCl2 were recorded in Sprague-Dawley rats, and in wild-

54 studies, dialysis in the presence of OP and CaCl2 removes only the catalytic zinc atom.

55 CaM Mr in presence of synthetic peptide and CaCl2 in 4 M urea polyacrylamide gel electrophoresis.

56 ctivation in the presence of prothrombin and CaCl2.

57 However, the presence of both sucrose and CaCl2 restored the ABA response to 20-40% of the maximum

58 f the maximum value measured in sucrose- and CaCl2-free media.

59 er incubation in media amended with urea and CaCl2 and/or SrCl2 , calcite (CaCO3 ), strontianite (SrC

60 l filtration with a combination of anhydrous CaCl2 (100mg) and primary-secondary amine (PSA, 25mg) as

61 re, hydrocooling cherry fruit in appropriate CaCl2 solutions (i.e., 0.2-0.5%) for 5 min and then pass

62 theart' and 39-188% linearly for 'Lapins' as CaCl2 rate increased from 0.2% to 2.0% at 0 degrees C fo

63 ersely, favorable deposition was observed at CaCl2 concentrations above 0.5 mM when the charge of SLB

64 fficient to mobilize all O-MWCNTs studied at CaCl2 concentrations as high as 10 mM.

65 etraacetic acid, blocked the effects of both CaCl2 and Pfr on the ATPase.

66 NaCl increased the wrinkles but CaCl2 softened the surface of granules.

67 able oils containing surfactant, followed by CaCl2 addition resulting in hardened particles.

68 ing reaction in EDTA followed immediately by CaCl2, the looped DNA was blocked from recombination whi

69 Pedicel browning was inhibited by CaCl2 at 0.2% and 0.5%, but increased by higher rates at

70 This effect was not mimicked by CaCl2, CdCl2, or CoCl2.

71 capsules cross-linked with divalent cationic CaCl2 salt (MCS), and the third group consisted of contr

72 s avium L.), the effect of calcium chloride (CaCl2) added to hydro-cooling water on physiological and

73 enzylaminopurine (BAP) and calcium chloride (CaCl2) could induce tolerance to chilling and could cons

74 with low concentrations of calcium chloride (CaCl2) induced chronic degeneration and calcification of

75 agnesium chloride (MgCl2), calcium chloride (CaCl2)], and electrolyte mixtures (KCl + CaCl2) through

76 as extracted from aqueous samples containing CaCl2 and FeCl3.

77 d periadventitial application of crystalline CaCl2 (0.5 g).

78 r a wide range of mono- (NaCl) and divalent (CaCl2) electrolytes and using time-resolved dynamic ligh

79 ic), CaCO3 (ecological), CaSO4 (ecological), CaCl2 (ecological), and Ca(C2H5COO)2 (ecological).

80 cubated in urea-amended media and equivalent CaCl2 and SrCl2 concentrations.

81 lectrolyte components showed that especially CaCl2 improved the oxidation rate of GSH.

82 Addition of exogenous CaCl2 also depressed the ATPase activity, and the kineti

83 incubation of the cell lysate with exogenous CaCl2.

84 e, diazoxide, tolbutamide, and extracellular CaCl2 omission or excess.

85 he role of calcium during APC, extracellular CaCl2 was decreased to 0.5 mM but only during the APC ep

86 Decreases in extracellular CaCl2 during APC blocked the recovery of EPs after 'test

87 ip (r(2) = 0.92 for NaCl and r(2) = 0.96 for CaCl2) between food quantity and Cl LC50.

88 he adhesion force profile to be distinct for CaCl2 compared with the other electrolytes.

89 (matching DLVO predictions) and 10.6 mM for CaCl2, respectively.

90 of 80-200 mM for sucrose and 0.76-20 mM for CaCl2.

91 Masa of flours from CaCl2 and Ca(C2H5COO)2 had the lowest values of G' and G

92 type with an overall trend of NaCl > MgCl2 > CaCl2.

93 alt concentrations in concentrated LiCl-HCl, CaCl2-HCl, and AICl3-HCl solutions, respectively.

94 d C(salt), in concentrated aqueous LiCl-HCl, CaCl2-HCl, and AlCl3-HCl solutions (C(acid) at 5-6 M; C(

95 ually no response to stimuli (including high CaCl2 concentration) and inhibitors (CaCl2 omission bein

96 iated Cl(-), which gain prevalence at higher CaCl2 concentration.

97 in the saline-treated controls to 9+/-3% in CaCl2-treated dogs (P<.05).

98 ted and measured dissolved concentrations in CaCl2 soil extracts, using the different extractions as

99 h MWNTs and silica surfaces with the drop in CaCl2 concentration likely resulted in the decrease in t

100 nced colloidal stability of nanoparticles in CaCl2 solution but rapid nanoparticle-nanoparticle aggre

101 and determined using 500 and 600 MHz NMR in CaCl2/methanol solution.

102 gible amount of deposited GO was released in CaCl2 under favorable conditions due to the binding of G

103 CFM reveals stronger adhesion on silica in CaCl2 compared with the other electrolytes, and shows a

104 2 Hz) was deposited on the silica surface in CaCl2 due to the bridging ability of Ca(2+) ions with GO

105 quadratic polynomial manner with increasing CaCl2 concentration from 0.2% to 2.0% in cold water (0 d

106 With increasing CaCl2 concentrations, a similar stepwise change in molec

107 ng high CaCl2 concentration) and inhibitors (CaCl2 omission being paradoxically stimulatory).

108 In this work, the effect of an inorganic (CaCl2) and an organic (tetraalkylphosphonium chloride) m

109 In isotonic CaCl2 solution, the slope conductance of the channel was

110 de (CaCl2)], and electrolyte mixtures (KCl + CaCl2) through a gated nanofluidic device was performed.

111 s in aqueous solutions containing NaCl, KCl, CaCl2, and Tris buffer show that the magnitude of the ef

112 5-minute intracoronary infusion of 20 mmol/L CaCl2 or saline before undergoing 1 hour of coronary occ

113 were diluted in a buffer containing 5 mmol/L CaCl2, phospholipid vesicles (10 micromol/L), and APC.

114 t (TIMP-1-/-) mice were exposed to 0.5 mol/L CaCl2 for 15 minutes, with terminal studies performed at

115 d Cu, and then extracted using either 0.01 M CaCl2 or 0.005 M diethylenetriaminepentaacetic acid (DTP

116 and soil in synthetic soil solution (0.01 M CaCl2), but in synthetic earthworm guts desorption was h

117 mmon leaching solutions (0.5 M K2SO4, 0.01 M CaCl2, 2 M KCl, and H2O) were used to extract WSOM from

118 , 8, and 16 weeks) post-TAA induction (0.5 M CaCl2, 15 minutes).

119 ic chemistries (pH, salt types (NaCl, MgCl2, CaCl2), ionic strength) relevant to natural and engineer

120 on of three commonly prevalent salts, MgCl2, CaCl2, and NaCl, next to a sapphire substrate using surf

121 MgCl2-CaCl2 composite salts were selected, and their fluxing e

122 ric monitoring of NADH removal in 100 microM CaCl2 at ionic strength 0.15 M, pH 7.0 and 21 degreesC.

123 g acid shock in buffer containing 200 microM CaCl2 and supplemented with neomycin, ruthenium red, or

124 ocked in 500 microM CaCl2 than in 200 microM CaCl2, although the accumulation durations were similar.

125 2 than in cells shocked in 200 or 500 microM CaCl2 or in the presence of CdCl2.

126 greater in cells acid shocked in 500 microM CaCl2 than in 200 microM CaCl2, although the accumulatio

127 not enhanced by incorporating 10 millimolar CaCl2 into the receiver block.

128 um; HC = imbibed and raised in 10 millimolar CaCl2), in the absence of additional Ca2+ (intermediate

129 ion in the receiver up to 5 to 10 millimolar CaCl2.

130 M HEPES-buffered saline (pH 8.4) with 0.1 mM CaCl2 and 0.08% sodium azide.

131 ckground electrolyte of 100 mM NaCl and 1 mM CaCl2 in equilibration with 400 ppmv CO2(g) ([U(VI)] = 4

132 With 1 mM CaCl2 in the extracellular solution, the channel conduct

133 in the presence of 10 mM NaCl, adding 0.1 mM CaCl2 reduced stability of GO with increased pH.

134 Addition of 1 mM CaCl2 to the cell wall preparation increased the transit

135 nce (>8000 Omega cm2) was achieved when 1 mM CaCl2 was included in the culture medium.

136 ations ranging from 0% to 65% (w/w) in 10 mM CaCl2 solution, thus producing formulations with a total

137 Cl2 and a binding isotherm measured in 10 mM CaCl2 using gel filtration to separate enzyme-bound and

138 The concentrations of Tl in 10 mM CaCl2-extracts increased with increasing soil Tl content

139 In 2 mM CaCl2, the affinity of the farnesylated protein to a syn

140 In the presence of 2 mM CaCl2, the far UV circular dichroism spectra of thrombos

141 In 2 mM CaCl2, the near UV circular dichroism spectra of thrombo

142 ered solution (pH(o) 7.40) containing 1.2 mM CaCl2.

143 00, and 70 mM NaCl and 10, 12, 6, and 7.5 mM CaCl2 for nC60, nC70, nC76, and nC84, respectively.

144 nts were conducted in the presence of 0.5 mM CaCl2 to ensure maximal activity.

145 ffer (pH 7.2) containing 0.14 M NaCl, 0.5 mM CaCl2, and 0.15 mM MgCl2 or with this buffer and either

146 M reduced and oxidized glutathione, and 5 mM CaCl2, followed by buffer exchange to remove denaturant

147 10 mM Tris.HCl, 10 mM KCl, 10 mM MgCl2, 5 mM CaCl2, pH 7.0, 22 degrees C).

148 e pH was decreased from 7.1 to 4.0 at 1.5 mM CaCl2.

149 10 mM sodium phosphate buffer, pH 7.2/0.5 mM CaCl2/0.15 mM MgCl2 (MBC50 range, 2.8-11.5 mM).

150 der favorable deposition conditions (1.50 mM CaCl2 and pH 7.1) and the deposited MWNTs were then rins

151 tant, occurs when cells are grown in 5-50 mM CaCl2.

152 han the thrombospondin-2 construct at 0.6 mM CaCl2.

153 IC50 of 1.79 and 0.47 mM when 10 and 1.8 mM CaCl2, respectively, was used as the charge carrier.

154 GO were determined to be 44 mM NaCl, 0.9 mM CaCl2, and 1.3 mM MgCl2.

155 istribution, and protein solubility in model CaCl2 solutions (50 mmol L(-1)) or rennet casein dispers

156 bons through cathodic polarization in molten CaCl2 at temperatures of about 1100 K, which generates p

157 omparing sorption from 0.01 N KCl and 0.01 N CaCl2; however, no significant differences were observed

158 tion was observed in the presence of Na2SO4, CaCl2, and NaHCO3.

159 ynthetic formation brine, and synthetic NaCl+CaCl2 brine were collected at the pressures from 100 to

160 ly represented by that in the synthetic NaCl+CaCl2 brines.

161 ncentrations (CCC) were determined for NaCl, CaCl2, and MgCl2 electrolytes.

162 a function of GO concentration and in NaCl, CaCl2, and MgCl2 as a function of ionic strength (IS).

163 After gradual salinization (NaCl:CaCl2 = 5.7:1, mol/mol), CMO mRNA, protein, and enzyme l

164 The peak response was caused by net CaCl2 and fluid gain via PIEZO1 channels driven by the s

165 of aggregates in the presence or absence of CaCl2, and CaCl2 could not reduce pre-formed aggregates.

166 f gyration of bacterial EPS with addition of CaCl2 was 20 times larger than that with the addition of

167 se parameters were enhanced with addition of CaCl2 while NaCl exhibited an opposite trend for all of

168 radius of bacterial EPS with the addition of CaCl2, but no change was observed after addition of MgCl

169 nor in the dark after subsequent addition of CaCl2.

170 rocket leaves, and stabilized by addition of CaCl2.

171 As the amount of CaCl2 added was increased, however, these changes became

172 We used periaortic application of CaCl2 to induce abdominal aortic aneurysm (AAA) and foun

173 rysm formation induced by the application of CaCl2 to the aortic surface was almost completely ablate

174 erve as an alternative to the application of CaCl2 used to extend the shelf life of numerous agricult

175 stablished AAA model in rats on the basis of CaCl2-mediated aortic injury.

176 the application method and concentration of CaCl2 applied periarterially.

177 The same concentration of CaCl2 induced approximately 25% of NFs to form longitudi

178 n with urea and equivalent concentrations of CaCl2 and SrCl2 was identified as hydrated Ca and Sr car

179 the presence of increasing concentrations of CaCl2, but the opposite trend was observed in the presen

180 w at pH > 7 or in elevated concentrations of CaCl2.

181 n media containing varying concentrations of CaCl2.

182 y the re-addition of calcium (in the form of CaCl2), but not by the addition of magnesium (MgCl2).

183 With the inclusion of CaCl2 in the receiver blocks, gravity-induced polar move

184 Brief intracoronary infusion of CaCl2 mimicked, whereas treatment with dimethylamiloride

185 By making iontophoretic injections of CaCl2 we have investigated calcium muffling, the sum of

186 KO, n =11) mice were exposed to 0.5 mol/L of CaCl2 for 15 minutes with terminal studies performed at

187 ed by other agents except for high levels of CaCl2.

188 and surfactant contents, higher molarity of CaCl2 and lower alginate concentrations.

189 ) and PF1.2 (but not PF1) in the presence of CaCl2 (2 mM) were able to replace HK (45 nM) in the pres

190 The presence of CaCl2 and CaSO4, even at low ionic strengths (i.e. 10 mM

191 egation by alpha-thrombin in the presence of CaCl2 but not with EDTA, suggesting that fibrinogen cros

192 ivation by alpha-thrombin in the presence of CaCl2 compared with that present in EDTA.

193 An increasing presence of CaCl2 in the initial brines improved the colour index, L

194 e-derived ligand provided in the presence of CaCl2 Michael adducts in moderate to high enantioselecti

195 r proteolytic degradation in the presence of CaCl2 than in the presence of EDTA, demonstrating that c

196 In the presence of CaCl2, CCC values of GO and rGO are quite similar, howev

197 nd fucosylated FVII EGF-1 in the presence of CaCl2.

198 ymerized at similar rates in the presence of CaCl2.

199 as significantly enhanced in the presence of CaCl2.

200 Removal of CaCl2 from the Krebs solution, disruption of the endothe

201 tenuated LV dilatation, and had no effect on CaCl2-mediated contraction.

202 he prevalence of these sites depends only on CaCl2 concentration.

203 viour when exposed to abscisic acid (ABA) or CaCl2 .

204 t crosslinking of proteins by citric acid or CaCl2 resulted in the formation of cold-set hydrogels.

205 se, in buffers that contained either EDTA or CaCl2 so as to preclude DNA cleavage by the endonuclease

206 e shell cross-linking ability of laccase, or CaCl2, on microcapsules.

207 ite polyamide membranes with either MgCl2 or CaCl2 draw solution.

208 ial EPS with the addition of either MgCl2 or CaCl2 solution.

209 ion of millimolar concentrations of MgCl2 or CaCl2 to the trans solution.

210 y energy minimum when the background NaCl or CaCl2 concentrations were decreased at pH 7.1.

211 trength to 1 at conditions with high NaCl or CaCl2 concentrations.

212 Salt type (NaCl or CaCl2) did not affect the Cl LC50, Daphnia life history

213 mized (TPTX) rats generated by either PTH or CaCl2 infusion.

214 icated that soil characteristics, namely, pH(CaCl2) for Fe and Cr, silt and fine-sand for Al and V, O

215 of FKS2 expression in response to pheromone, CaCl2, or loss of FKS1 function requires the Ca2+/calmod

216 groups were also prepared-calcium phosphate (CaCl2.2H2O + K2HPO4 in buffer solution) and SDF (Ag[NH3]

217 using low-melting-point ternary molten salts CaCl2 -MgCl2 -NaCl, which still retains high CaSiO3 solu

218 eed oil/pectin) and a cross-linked solution (CaCl2).

219 alues obtained suggests the presence of some CaCl2/lactose aggregates in the media, which are influen

220 The CaCl2 and MgCl2 addition increased the gel strength duri

221 The CaCl2 system, despite its simplicity, was a good indicat

222 nhibitors of elastin degradation) before the CaCl2-mediated injury significantly reduced elastin calc

223 The analysis of the CaCl2 diffusion coefficient values obtained suggests the

224 Characterization of the CaCl2-induced model revealed that HMGB1 and RAGE, both l

225 on aortic dilatation were measured using the CaCl2-induced AAA model in mice.

226 When the CaCl2 concentration in the eluent was decreased, a large

227 reatment in Ca2+-free medium) and exposed to CaCl2 or MnCl2 had a greater rate of Ca2+ entry (18.6 +/

228 Peak [Ca2+]i in cells exposed to CaCl2 was also enhanced (869.4 +/- 114.7 vs. 562.6 +/- 6

229 (but not wild-type W3110) hypersensitive to CaCl2 at 5 mM or higher.

230 und that MMP-knockout mice were resistant to CaCl2-mediated aortic injury and did not develop elastin

231 When CaCl2 was incorporated into the receivers, movement of l

232 ned the surface of PG starch particles while CaCl2 did not bring about obvious changes on their morph

233 were embedded in alginate cross-linked with CaCl2 and cultured in modified Ham's F10 medium with 10%

234 ipoprotein E-deficient (apoE(-/-)) mice with CaCl2-induced carotid aneurysm (n = 11).

235 ted when mungbean cuttings were treated with CaCl2, while treatment with MgCl2 had no effect.