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1  nt cytoplasmic RNA that is conserved across Caenorhabditis and possesses three essential secondary s
2 es on stomatal shapes in the model nematodes Caenorhabditis and Pristionchus.
3                                              Caenorhabditis bovis is a particularly unusual species t
4           A new study analyzes the genome of Caenorhabditis bovis, a nematode that may be evolving a
5 ht C. elegans (Orsay virus) and its relative Caenorhabditis briggsae (Santeuil virus, Le Blanc virus,
6 and Santeuil, which both specifically infect Caenorhabditis briggsae, were also found to form fibrous
7  Drosophila, Escherichia coli (E. coli), and Caenorhabditis elegans (C. elegan) datasets.
8 omatin and nucleoli during cell division and Caenorhabditis elegans (C. elegans) growth.
9 ied by the dauer larva stage of the nematode Caenorhabditis elegans (C. elegans).
10  we report that mitochondrial dysfunction in Caenorhabditis elegans activates RNAi-directed silencing
11                                 We find that Caenorhabditis elegans aggregates are observed in large
12 rmation, and emergence of swarming in active Caenorhabditis elegans aggregates.
13                                              Caenorhabditis elegans alternate between local search an
14 s involved in glial size regulation, we used Caenorhabditis elegans amphid sheath (AMsh) glia as a mo
15                    Using an in vivo model of Caenorhabditis elegans anchor cell invasion, we characte
16  of the dosage compensation complex (DCC) in Caenorhabditis elegans and demonstrate that loop anchors
17 e mechanisms underlying UPR(mt) signaling in Caenorhabditis elegans and discuss emerging connections
18 as leveraged simple model organisms, such as Caenorhabditis elegans and Drosophila melanogaster larva
19 d aging, including stem cell dysfunction, in Caenorhabditis elegans and Drosophila melanogaster model
20  (mice and humans) as well as invertebrates (Caenorhabditis elegans and Drosophila melanogaster).
21 ulfide reduction exclusively in endosomes in Caenorhabditis elegans and identified that exchange is m
22 e paralysis, we conducted genetic screens in Caenorhabditis elegans and isolated mutants that became
23                In numerous species including Caenorhabditis elegans and mammals, neural serotonin sig
24 al to organismal health in both the nematode Caenorhabditis elegans and mammals.
25 fication of Abeta oligomers in vitro, and in Caenorhabditis elegans and mouse hippocampal tissues.
26 mporal regulation of LAG-1/CSL expression in Caenorhabditis elegans and observed that an increase in
27                                              Caenorhabditis elegans and related Caenorhabditis spp. c
28 ptide F (NPF)-related neuropeptide system in Caenorhabditis elegans and show that this FLP-34/NPR-11
29 vensis is a natural pathogen of the nematode Caenorhabditis elegans and that parental exposure of ani
30 t method relying on the free-living nematode Caenorhabditis elegans and the infection of those nemato
31 mental inhibitor of the free-living nematode Caenorhabditis elegans and the plant-parasitic nematode
32 on of a long-term swim exercise protocol for Caenorhabditis elegans and we demonstrate its benefits t
33 ultured cells, acute mouse brain slices, and Caenorhabditis elegans and Xenopus laevis in vivo.
34 couple these key events of the cell cycle in Caenorhabditis elegans and zebrafish through live-cell i
35           Laboratory-conditioned 'wild-type' Caenorhabditis elegans are different from wild-isolated
36 rpose, we have designed a pipeline that uses Caenorhabditis elegans as a genetic model to screen for
37                                  We employed Caenorhabditis elegans as a heterologous host to underst
38                                        Using Caenorhabditis elegans as a model, here we report that h
39 ral, biochemical, and functional analyses in Caenorhabditis elegans As capsaicin elicits heat and pai
40                                          The Caenorhabditis elegans AWC olfactory neuron pair asymmet
41                   RNAi-mediated knockdown of Caenorhabditis elegans bcat-1 is known to recapitulate P
42                            We identified the Caenorhabditis elegans BK channel SLO-1 as a molecular t
43 ogical mechanisms that delay neuron aging in Caenorhabditis elegans by asking how neuron morphologica
44  We developed a glutamate spillover model in Caenorhabditis elegans by inactivating the conserved gli
45                However, for the infection of Caenorhabditis elegans by P. aeruginosa, the precise pat
46 ibed a physiological program in the nematode Caenorhabditis elegans called the intracellular pathogen
47 how that bloating of the intestinal lumen of Caenorhabditis elegans caused by microbial colonization
48 fied a gain-of-function (gf) mutation in the Caenorhabditis elegans CaV2 channel alpha1 subunit, UNC-
49 oteins and an unexpected direct link between Caenorhabditis elegans CFP-1 and an Rpd3/Sin3 small (SIN
50                                  Here, using Caenorhabditis elegans chemosensory neurons as a model s
51                                              Caenorhabditis elegans consumes bacteria, which can supp
52                              We show that in Caenorhabditis elegans COQ-2e is required for efficient
53          Here the role of human AMPK and its Caenorhabditis elegans counterpart AAK-2 was explored up
54  This subunit, dSol-1, is the homolog of the Caenorhabditis elegans CUB (Complement C1r/C1s, Uegf, Bm
55 e atomic cryo-EM structures of a full-length Caenorhabditis elegans cyclic GMP-activated channel TAX-
56 actors implicated in translation fidelity in Caenorhabditis elegans decreases lifespan, and eEF2K is
57                     Research in the nematode Caenorhabditis elegans demonstrates that retrieval of av
58                             We show that the Caenorhabditis elegans DIP-2 maintains morphology of mat
59                                              Caenorhabditis elegans early embryos generate cell-speci
60      We followed the first nine divisions of Caenorhabditis elegans embryo development and demonstrat
61                                          The Caenorhabditis elegans embryo is an important model for
62                        Here we use the early Caenorhabditis elegans embryo to explore how the actin f
63 ce in situ hybridization (smFISH) studies in Caenorhabditis elegans embryogenesis.
64 nvestigated cytokinesis during the invariant Caenorhabditis elegans embryonic divisions and found sev
65 ression levels of APA isoforms in individual Caenorhabditis elegans embryos at different stages throu
66 robe and control the cell-division timing in Caenorhabditis elegans embryos using a combination of lo
67                                           In Caenorhabditis elegans embryos, chromodomain protein CEC
68              Here, we show that the nematode Caenorhabditis elegans exhibits the key features of alco
69  report that the noncanonical sHsp HSP-17 of Caenorhabditis elegans facilitates aggregation of model
70                Apoe(-/-) mice expressing the Caenorhabditis elegans Fat-1 transgene (Fat-1(tg)xApoe(-
71 ng, we performed a forward genetic screen in Caenorhabditis elegans for genes involved in body size r
72 on developmental gene expression dataset for Caenorhabditis elegans from Bao, Murray, Waterston et al
73 lar range cysteamine bitartrate treatment in Caenorhabditis elegans gas-1(fc21) RC complex I (NDUFS2-
74                   We deleted mafr-1 from the Caenorhabditis elegans genome and found that animals lac
75 H Oligopaint library that targets the entire Caenorhabditis elegans genome at chromosome, three megab
76 onserved homeodomain proteins encoded by the Caenorhabditis elegans genome(2), that the complete set
77                             Here, we use the Caenorhabditis elegans germline as a model to investigat
78 ins FBF-1 and FBF-2 have opposite effects on Caenorhabditis elegans germline stem cell dynamics: FBF-
79                                       In the Caenorhabditis elegans germline, fem-3 Binding Factor (F
80 e-like glia, which extended the life span in Caenorhabditis elegans Glial XBP-1s initiated a robust c
81                                       In the Caenorhabditis elegans gonad, the broad outlines of germ
82 pling between the soma and germ cells in the Caenorhabditis elegans gonad.
83 rain to induce CA production directly in the Caenorhabditis elegans gut, we reveal the local effect o
84                                 The nematode Caenorhabditis elegans has an alternate dispersal life s
85                                              Caenorhabditis elegans has long been a laboratory model
86                       Some nematodes such as Caenorhabditis elegans have an XO sex determination syst
87 sed by all neurons in model systems, such as Caenorhabditis elegans have left still unresolved how ne
88    Hawaiian isolates of the nematode species Caenorhabditis elegans have long been known to harbor ge
89 netic studies on the excretory canal cell of Caenorhabditis elegans have revealed many proteins that
90                                        Young Caenorhabditis elegans hermaphrodites use their own sper
91  TFEB and TFE3, as well as their ortholog in Caenorhabditis elegans HLH-30, play an important role in
92 nd genetic analysis, we found that catp-6, a Caenorhabditis elegans homolog of ATP13A2, was responsib
93                               UNC-104 is the Caenorhabditis elegans homolog of kinesin-3 KIF1A known
94 ere, we study the effect of different crh-1 [Caenorhabditis elegans homolog of mammalian cAMP respons
95                         Here we show that in Caenorhabditis elegans HS induces up- and downregulation
96 regulates EV levels in the sensory organs of Caenorhabditis elegans in a cilia specific manner.
97 mechanical properties associated with ageing Caenorhabditis elegans in addition to capturing high-res
98 successfully reduced the fungal burdens in a Caenorhabditis elegans infection model by up to 96%.
99                           Additionally, in a Caenorhabditis elegans infection model, the ospemifene-i
100 utative FAM173B orthologue from the nematode Caenorhabditis elegans Interestingly, lack of Lys-43 met
101                                 The nematode Caenorhabditis elegans is a bacterivore filter feeder.
102                     The free-living nematode Caenorhabditis elegans is a key laboratory model for met
103                  The pharynx of the nematode Caenorhabditis elegans is a simple neuromuscular organ w
104                                 The nematode Caenorhabditis elegans is a useful model for studying th
105                                              Caenorhabditis elegans is a valuable model organism in b
106                                              Caenorhabditis elegans is an animal with few cells but a
107                           The model nematode Caenorhabditis elegans is ideal to study these interacti
108                          The endoderm GRN in Caenorhabditis elegans is initiated by the maternally su
109                                              Caenorhabditis elegans is used extensively as a medical
110              Upon sensing starvation stress, Caenorhabditis elegans larvae (L2d) elicit two seemingly
111 can modulate the developmental trajectory of Caenorhabditis elegans larvae by promoting entry into da
112      We generate CRISPR knockout strains for Caenorhabditis elegans lincRNAs and evaluate their pheno
113 -shared neurons in the nervous system of the Caenorhabditis elegans male is controlled by the tempora
114 g a conspicuous muscle change that occurs in Caenorhabditis elegans males.
115 tion channel complex in the sensory cilia of Caenorhabditis elegans mechanoreceptor neurons.
116 ayer in coordinating these key events during Caenorhabditis elegans meiosis.
117  specific isoform of CREB1/CRH-1, CRH-1e, in Caenorhabditis elegans memory formation and chemosensati
118 -helix transcription factor 30 (hlh-30), the Caenorhabditis elegans MiT/TFE ortholog, to starvation f
119 beta-induced paralysis in a transgenic Abeta Caenorhabditis elegans model and specifically target and
120  To this end, we have generated a transgenic Caenorhabditis elegans model expressing both human Abeta
121                   Previously, we generated a Caenorhabditis elegans model of ALS, in which the expres
122 ogical tau protein, we employed a transgenic Caenorhabditis elegans model of human tauopathy exhibiti
123                    A genome-wide screen in a Caenorhabditis elegans model of SOD1-linked ALS identifi
124 re, we develop an experimental handle in the Caenorhabditis elegans model system, in which we uncover
125 MP resistance show enhanced virulence in the Caenorhabditis elegans model, an effect that is abolishe
126 en suggested to regulate tau inclusions in a Caenorhabditis elegans model.
127         In both amyloid-beta (Abeta) and tau Caenorhabditis elegans models of AD, mitophagy stimulati
128 hes to better understand the function of the Caenorhabditis elegans MORC-1 protein.
129 pmental expression of three conserved genes, Caenorhabditis elegans mpk-1, lag-1, and lag-3/sel-8, wh
130 e diseases, and we show that the lack of the Caenorhabditis elegans Msp1 homologue triggers an import
131 hondria are arrayed in a periodic pattern in Caenorhabditis elegans muscle, but this pattern is disru
132                                              Caenorhabditis elegans must distinguish pathogens from n
133                            Here, we analyzed Caenorhabditis elegans mutants deleted of the sole SEIPI
134 d nucleus deformation dynamics within living Caenorhabditis elegans nematodes.
135 e retraction is shown to be operative in the Caenorhabditis elegans nervous system.
136  to visualize alternative splicing in single Caenorhabditis elegans neurons, identifying complex spli
137 e in primary fibroblasts, murine neurons and Caenorhabditis elegans neurons.
138 ant for controlling levels of DCV cargoes in Caenorhabditis elegans neurons.
139  we discovered a novel coordinated action of Caenorhabditis elegans neuropeptide Y/neuropeptide F and
140                                          The Caenorhabditis elegans NPY/NPF ortholog FLP-34 displays
141   Here we report our investigations on three Caenorhabditis elegans orthologous proteins involved in
142                      We also report that the Caenorhabditis elegans orthologs of 14-3-3zeta and MSec
143 subunits partially rescue sensory defects in Caenorhabditis elegans osm-9 and tax-4 knock-out strains
144 of Alzheimer's disease in the model organism Caenorhabditis elegans Our results pose and answer the q
145 e we describe the emerging model system of a Caenorhabditis elegans polymodal neuron named PVD, whose
146                                              Caenorhabditis elegans possess a mild inflammatory respo
147                                 The nematode Caenorhabditis elegans possesses glial types similar to
148 of integrin-mediated collagen recruitment to Caenorhabditis elegans postembryonic gonadal and pharyng
149                                              Caenorhabditis elegans primordial germ cells (PGCs) jett
150                                 The nematode Caenorhabditis elegans produces a broad family of pherom
151                       Here we identified the Caenorhabditis elegans protein huntingtin-interacting pr
152 re, we report the identification of PLP-1, a Caenorhabditis elegans protein related to the human sing
153                           Here we describe a Caenorhabditis elegans quiescent behavior, post-response
154                       The spermatheca of the Caenorhabditis elegans reproductive system enables study
155  dbt-1 gene underlies natural differences in Caenorhabditis elegans responses to the toxin arsenic.
156  The expression of chimeric hnRNPA2 D290V in Caenorhabditis elegans results in stress-induced glutama
157 R-generated, fluorescently tagged endogenous Caenorhabditis elegans RHEB-1 and DAF-15/Raptor are expr
158                                          The Caenorhabditis elegans RIG-I-like receptor DRH-1 promote
159 colleagues provide evidence that exposure of Caenorhabditis elegans roundworms to 2 simple nutrients,
160 l-angle x-ray scattering) and in vivo tools (Caenorhabditis elegans sarcomere structure).
161 repeated induction of the flight response in Caenorhabditis elegans shortens lifespan and inhibits co
162 gene expression analyses in P. univalens and Caenorhabditis elegans show that the intestine is the ma
163                                           In Caenorhabditis elegans sleep occurs during a larval tran
164                                       During Caenorhabditis elegans sleep, energetic stores are alloc
165        We use the myoepithelial cells of the Caenorhabditis elegans spermatheca to study the mechanis
166 to a brief 2 degrees C temperature increase, Caenorhabditis elegans spermatocytes exhibit up to a 25-
167 bservations were recapitulated in vivo, in a Caenorhabditis elegans strain deficient in the ATP13A2 o
168                               A new study in Caenorhabditis elegans suggests the ubiquitin-proteasome
169 e for the whole-life culture of the nematode Caenorhabditis elegans that allows the scoring of animal
170  identify a neuronal circuit in the nematode Caenorhabditis elegans that processes information percei
171 he model agrees with experiments in the worm Caenorhabditis elegans that show the following: Life spa
172 which male-specific neurons are generated in Caenorhabditis elegans through the direct transdifferent
173 omeric organization and muscle function from Caenorhabditis elegans to humans.
174                      We have used RNA-seq in Caenorhabditis elegans to produce transcription profiles
175                                Here, we used Caenorhabditis elegans to systematically functionally ch
176        For example, in the annotation of the Caenorhabditis elegans transcriptome, more than half of
177 come well established that the nematode worm Caenorhabditis elegans triggers innate immune responses
178 s controlled by Munc18 (mammalian homolog of Caenorhabditis elegans uncoordinated gene 18) proteins.
179                                          The Caenorhabditis elegans vulva has been a paradigm for und
180                                              Caenorhabditis elegans was the first multicellular eukar
181 regulates its trafficking and degradation in Caenorhabditis elegans We found that the WD40-repeat pro
182 multipotent vulval precursor cells (VPCs) of Caenorhabditis elegans We have previously shown that thi
183 ned by the number of traps after exposure to Caenorhabditis elegans While some strains were highly se
184  neuronal tissue, mouse embryonic tissue and Caenorhabditis elegans whole embryos.
185 riation in the behavioral flexibility of two Caenorhabditis elegans wild strains.
186  measured touch-induced mechanical strain in Caenorhabditis elegans worms.
187        During the asymmetric division of the Caenorhabditis elegans zygote, germ (P) granules are dis
188                   Using the newly fertilized Caenorhabditis elegans zygote, we show that the mitotic
189                                           In Caenorhabditis elegans zygotes, PAR-1 localizes to the p
190 EB) transcription factor significantly slows Caenorhabditis elegans' reproductive decline, an early h
191 transposon silencing and RNA interference in Caenorhabditis elegans(1-4).
192 on in HD models (cells, primary neurons, and Caenorhabditis elegans) increases mutant HTT exon 1 phos
193 dopsis thaliana), rice (Oryza sativa), worm (Caenorhabditis elegans), and human (Homo sapiens) cells
194 erves this function in human cells (LEM-3 in Caenorhabditis elegans).
195 obes in a complex multicellular model (i.e., Caenorhabditis elegans).
196 ere we develop tests for IIA in the nematode Caenorhabditis elegans, an animal with only 302 neurons,
197                      Remarkably, RAD-51 from Caenorhabditis elegans, an organism without Dmc1, has ac
198 t organisms, including Arabidopsis thaliana, Caenorhabditis elegans, and Danio rerio.
199 bolism without affecting eating behaviors in Caenorhabditis elegans, and identified specific odors th
200                                           In Caenorhabditis elegans, and possibly other organisms, wh
201 e, whereas Smn from Drosophila melanogaster, Caenorhabditis elegans, and Schizosaccharomyces pombe wa
202  of a related nonparasitic nematode species, Caenorhabditis elegans, and the parasitic nematode Haemo
203 europeptide Y/neuropeptide F in the nematode Caenorhabditis elegans, and we discovered that it is req
204                                           In Caenorhabditis elegans, ascaroside pheromones can dictat
205                              In the nematode Caenorhabditis elegans, axonal regeneration can proceed
206 tant proteins remain enriched at synapses in Caenorhabditis elegans, but show defects in active-zone
207 nd prodigiosin antibiotics, and infection in Caenorhabditis elegans, but up-regulating flagellar moti
208 herefore, simple organisms like the nematode Caenorhabditis elegans, combining relevant advantages of
209 ry element activities across five tissues of Caenorhabditis elegans, covering ~90% of cells.
210                                           In Caenorhabditis elegans, deficiency of DCAP-1/DCP1, the e
211 is regard, we summarize recent studies using Caenorhabditis elegans, Drosophila melanogaster, Danio r
212 ets across species inclusive of Arabidopsis, Caenorhabditis elegans, Drosophila, human, mouse, rat, y
213 erotonin-dependent behavior of the roundworm Caenorhabditis elegans, egg laying, to perform a behavio
214 ically infects the laboratory model nematode Caenorhabditis elegans, encodes a fibrous protein delta
215 hat govern the formation of gap junctions in Caenorhabditis elegans, five of which are already suppor
216 zing proteome-wide mass spectrometry data of Caenorhabditis elegans, however, we show that the levels
217 cells as well as Drosophila melanogaster and Caenorhabditis elegans, indicating suitability for use i
218                                           In Caenorhabditis elegans, Katanin MT-severing activity is
219                                           In Caenorhabditis elegans, low vitamin B12, or genetic pert
220                                           In Caenorhabditis elegans, MET-2, the homologue of mammalia
221                                           In Caenorhabditis elegans, neural excitation increases with
222                                           In Caenorhabditis elegans, piRNA loci are clustered within
223                                           In Caenorhabditis elegans, RBPs control the translation, st
224                                           In Caenorhabditis elegans, reduced mitochondrial electron t
225                                           In Caenorhabditis elegans, regulation of piRNA target genes
226                                           In Caenorhabditis elegans, RNA interference (RNAi) response
227                                           In Caenorhabditis elegans, RNAi can be achieved by feeding
228                              We show that in Caenorhabditis elegans, serotonin released by maternal n
229 velopment in a subpopulation of synchronized Caenorhabditis elegans, sets processes in motion that in
230                                           In Caenorhabditis elegans, signals from the germline initia
231                                           In Caenorhabditis elegans, the avoidance response to gentle
232                        Here, we show that in Caenorhabditis elegans, the axons of the ALA neuron cont
233                                           In Caenorhabditis elegans, the BMP ligand DBL-1 is a regula
234                                           In Caenorhabditis elegans, the closest BUBR1 orthologue lac
235                                           In Caenorhabditis elegans, the heterochronic pathway contro
236                        To test this, we used Caenorhabditis elegans, the intestinal cells of which ar
237 ntrations in most studied species, including Caenorhabditis elegans, the molecular mechanisms behind
238                         Here we show that in Caenorhabditis elegans, the neuromodulator tyramine prod
239                                           In Caenorhabditis elegans, the transcription factor HBL-1 (
240                                           In Caenorhabditis elegans, the transcription factor, SKN-1,
241                              In the nematode Caenorhabditis elegans, the transcription regulators DAF
242                              In the nematode Caenorhabditis elegans, this highly efficient regenerati
243 icellular settings, from the model organism, Caenorhabditis elegans, to humans.
244 lls, a population of epidermal stem cells in Caenorhabditis elegans, to study the influence of temper
245 usly shown that 2 paralogous tetraspanins in Caenorhabditis elegans, TSP-12 and TSP-14, function redu
246                              In the nematode Caenorhabditis elegans, UNC-87 is a calponin-related pro
247                                           In Caenorhabditis elegans, unc-89 encodes a set of giant mu
248   Using the touch receptor neurons (TRNs) of Caenorhabditis elegans, we find that mec-15(-) mutants d
249 ructions of spermatocyte meiotic spindles in Caenorhabditis elegans, we find the lagging X chromosome
250 ors of heat-shock-induced gene expression in Caenorhabditis elegans, we found a new allele of hsf-1 t
251               Using the in vivo model system Caenorhabditis elegans, we found that the highly conserv
252                                  Here, using Caenorhabditis elegans, we generated the first organism
253 n coupled with deep-sequencing (TRAP-seq) in Caenorhabditis elegans, we have obtained high coverage p
254 encing and genomic and genetic approaches in Caenorhabditis elegans, we reveal an unprecedented role
255                        Using optogenetics in Caenorhabditis elegans, we solved the presynaptic circui
256  and siRNAs in regulating gene expression in Caenorhabditis elegans, we subjected small RNAs and mRNA
257               In a forward genetic screen in Caenorhabditis elegans, we uncover that TALP-3, a homolo
258 increased in young amyloid B (AB) expressing Caenorhabditis elegans, whereas it is not in wild type s
259 odels zebrafish, Drosophila melanogaster and Caenorhabditis elegans, which command superb research re
260                  In simple organisms such as Caenorhabditis elegans, whole brain imaging has been per
261                                           In Caenorhabditis elegans, wounding the epidermis triggers
262 we address this question using heteroplasmic Caenorhabditis elegans.
263 tivity in the nervous system of the nematode Caenorhabditis elegans.
264 f two new SC components, SYP-5 and SYP-6, in Caenorhabditis elegans.
265  of proteins to chemosensory cilia in living Caenorhabditis elegans.
266  homologous recombination-mediated repair in Caenorhabditis elegans.
267 rous iron accumulates over adult lifetime in Caenorhabditis elegans.
268 tol's effects on fatty acid metabolism using Caenorhabditis elegans.
269 e molecular bases of this organization using Caenorhabditis elegans.
270 g m(5)C at position C2381 on the 26S rRNA in Caenorhabditis elegans.
271 ll ferroptosis and sterility in the nematode Caenorhabditis elegans.
272 cleavage to attract glia during migration in Caenorhabditis elegans.
273 during embryonic development of the nematode Caenorhabditis elegans.
274 ween Pseudomonas aeruginosa and the nematode Caenorhabditis elegans.
275 n an in vivo system using the model organism Caenorhabditis elegans.
276 e been best described for the model organism Caenorhabditis elegans.
277 mentally identified piRNA targeting sites in Caenorhabditis elegans.
278 ting the speed of locomotion of the nematode Caenorhabditis elegans.
279 educe lipid storage and promote longevity in Caenorhabditis elegans.
280 n structural analysis in vivo (IV-FPOP) with Caenorhabditis elegans.
281 , followed by functional characterization in Caenorhabditis elegans.
282  thereof on the reproduction of the nematode Caenorhabditis elegans.
283 with the membraneless organelle P granule in Caenorhabditis elegans.
284 hoprim exhibited increased virulence against Caenorhabditis elegans.
285 tic protein phosphofructokinase-1/PFK-1.1 in Caenorhabditis elegans.
286 y small silencing RNAs in the model nematode Caenorhabditis elegans.
287 A (ncRNA) essential for axon regeneration in Caenorhabditis elegans.
288 nactive one, controls checkpoint strength in Caenorhabditis elegans.
289 terograde synaptic organizer in the nematode Caenorhabditis elegans.
290 scales of hierarchical locomotion control in Caenorhabditis elegans.
291                                          The Caenorhabditis hosts were found defective in degrading e
292 imized to measure activity and quiescence in Caenorhabditis larvae and adults and in Drosophila larva
293                           Several species of Caenorhabditis nematodes have evolved a mating system in
294 omosome in hemizygous (XY) males, whereas in Caenorhabditis nematodes, expression is halved for both
295 ing cell fate among vulva precursor cells in Caenorhabditis nematodes, that of P3.p.
296 logenetic relationships of C. bovis to other Caenorhabditis species and reveal the changes in genome
297                               All of the ~65 Caenorhabditis species currently in culture are free-liv
298 preserving, are more highly conserved across Caenorhabditis species, and are enriched in specific cis
299 ncement in our understanding of this unusual Caenorhabditis species.
300           Caenorhabditis elegans and related Caenorhabditis spp. contain LTR retrotransposons and, as

 
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