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1 erves this function in human cells (LEM-3 in Caenorhabditis elegans).
2 obes in a complex multicellular model (i.e., Caenorhabditis elegans).
3 f two new SC components, SYP-5 and SYP-6, in Caenorhabditis elegans.
4  of proteins to chemosensory cilia in living Caenorhabditis elegans.
5  homologous recombination-mediated repair in Caenorhabditis elegans.
6 rous iron accumulates over adult lifetime in Caenorhabditis elegans.
7 tol's effects on fatty acid metabolism using Caenorhabditis elegans.
8 e molecular bases of this organization using Caenorhabditis elegans.
9 g m(5)C at position C2381 on the 26S rRNA in Caenorhabditis elegans.
10 ll ferroptosis and sterility in the nematode Caenorhabditis elegans.
11 cleavage to attract glia during migration in Caenorhabditis elegans.
12 during embryonic development of the nematode Caenorhabditis elegans.
13 ween Pseudomonas aeruginosa and the nematode Caenorhabditis elegans.
14 n an in vivo system using the model organism Caenorhabditis elegans.
15 e been best described for the model organism Caenorhabditis elegans.
16 mentally identified piRNA targeting sites in Caenorhabditis elegans.
17 ting the speed of locomotion of the nematode Caenorhabditis elegans.
18 educe lipid storage and promote longevity in Caenorhabditis elegans.
19 n structural analysis in vivo (IV-FPOP) with Caenorhabditis elegans.
20  alpha-synuclein-driven neurodegeneration in Caenorhabditis elegans.
21 , followed by functional characterization in Caenorhabditis elegans.
22  thereof on the reproduction of the nematode Caenorhabditis elegans.
23 with the membraneless organelle P granule in Caenorhabditis elegans.
24 tic protein phosphofructokinase-1/PFK-1.1 in Caenorhabditis elegans.
25 hoprim exhibited increased virulence against Caenorhabditis elegans.
26 y small silencing RNAs in the model nematode Caenorhabditis elegans.
27 A (ncRNA) essential for axon regeneration in Caenorhabditis elegans.
28 nactive one, controls checkpoint strength in Caenorhabditis elegans.
29 terograde synaptic organizer in the nematode Caenorhabditis elegans.
30 scales of hierarchical locomotion control in Caenorhabditis elegans.
31 we address this question using heteroplasmic Caenorhabditis elegans.
32 tivity in the nervous system of the nematode Caenorhabditis elegans.
33 transposon silencing and RNA interference in Caenorhabditis elegans(1-4).
34  we report that mitochondrial dysfunction in Caenorhabditis elegans activates RNAi-directed silencing
35                                 We find that Caenorhabditis elegans aggregates are observed in large
36 rmation, and emergence of swarming in active Caenorhabditis elegans aggregates.
37                                              Caenorhabditis elegans alternate between local search an
38 s involved in glial size regulation, we used Caenorhabditis elegans amphid sheath (AMsh) glia as a mo
39 ere we develop tests for IIA in the nematode Caenorhabditis elegans, an animal with only 302 neurons,
40                      Remarkably, RAD-51 from Caenorhabditis elegans, an organism without Dmc1, has ac
41                    Using an in vivo model of Caenorhabditis elegans anchor cell invasion, we characte
42  of the dosage compensation complex (DCC) in Caenorhabditis elegans and demonstrate that loop anchors
43 e mechanisms underlying UPR(mt) signaling in Caenorhabditis elegans and discuss emerging connections
44 as leveraged simple model organisms, such as Caenorhabditis elegans and Drosophila melanogaster larva
45 d aging, including stem cell dysfunction, in Caenorhabditis elegans and Drosophila melanogaster model
46  (mice and humans) as well as invertebrates (Caenorhabditis elegans and Drosophila melanogaster).
47 vertebrate experimental organisms, including Caenorhabditis elegans and Drosophila melanogaster.
48 ulfide reduction exclusively in endosomes in Caenorhabditis elegans and identified that exchange is m
49 e paralysis, we conducted genetic screens in Caenorhabditis elegans and isolated mutants that became
50                In numerous species including Caenorhabditis elegans and mammals, neural serotonin sig
51 al to organismal health in both the nematode Caenorhabditis elegans and mammals.
52 fication of Abeta oligomers in vitro, and in Caenorhabditis elegans and mouse hippocampal tissues.
53 mporal regulation of LAG-1/CSL expression in Caenorhabditis elegans and observed that an increase in
54                                              Caenorhabditis elegans and related Caenorhabditis spp. c
55 ptide F (NPF)-related neuropeptide system in Caenorhabditis elegans and show that this FLP-34/NPR-11
56 vensis is a natural pathogen of the nematode Caenorhabditis elegans and that parental exposure of ani
57 t method relying on the free-living nematode Caenorhabditis elegans and the infection of those nemato
58 mental inhibitor of the free-living nematode Caenorhabditis elegans and the plant-parasitic nematode
59 on of a long-term swim exercise protocol for Caenorhabditis elegans and we demonstrate its benefits t
60 ultured cells, acute mouse brain slices, and Caenorhabditis elegans and Xenopus laevis in vivo.
61 couple these key events of the cell cycle in Caenorhabditis elegans and zebrafish through live-cell i
62 dopsis thaliana), rice (Oryza sativa), worm (Caenorhabditis elegans), and human (Homo sapiens) cells
63 t organisms, including Arabidopsis thaliana, Caenorhabditis elegans, and Danio rerio.
64 bolism without affecting eating behaviors in Caenorhabditis elegans, and identified specific odors th
65 ciated protein misfolding toxicity in yeast, Caenorhabditis elegans, and mammalian cells.
66                                           In Caenorhabditis elegans, and possibly other organisms, wh
67 e, whereas Smn from Drosophila melanogaster, Caenorhabditis elegans, and Schizosaccharomyces pombe wa
68  of a related nonparasitic nematode species, Caenorhabditis elegans, and the parasitic nematode Haemo
69 europeptide Y/neuropeptide F in the nematode Caenorhabditis elegans, and we discovered that it is req
70               Indeed, in ARX/alr-1-deficient Caenorhabditis elegans animals, SAHA was shown to counte
71           Laboratory-conditioned 'wild-type' Caenorhabditis elegans are different from wild-isolated
72 rpose, we have designed a pipeline that uses Caenorhabditis elegans as a genetic model to screen for
73                                  We employed Caenorhabditis elegans as a heterologous host to underst
74                                        Using Caenorhabditis elegans as a model, here we report that h
75 ral, biochemical, and functional analyses in Caenorhabditis elegans As capsaicin elicits heat and pai
76                                           In Caenorhabditis elegans, ascaroside pheromones can dictat
77                                          The Caenorhabditis elegans AWC olfactory neuron pair asymmet
78                              In the nematode Caenorhabditis elegans, axonal regeneration can proceed
79                   RNAi-mediated knockdown of Caenorhabditis elegans bcat-1 is known to recapitulate P
80                            We identified the Caenorhabditis elegans BK channel SLO-1 as a molecular t
81 tant proteins remain enriched at synapses in Caenorhabditis elegans, but show defects in active-zone
82 nd prodigiosin antibiotics, and infection in Caenorhabditis elegans, but up-regulating flagellar moti
83 ogical mechanisms that delay neuron aging in Caenorhabditis elegans by asking how neuron morphologica
84  We developed a glutamate spillover model in Caenorhabditis elegans by inactivating the conserved gli
85                However, for the infection of Caenorhabditis elegans by P. aeruginosa, the precise pat
86  Drosophila, Escherichia coli (E. coli), and Caenorhabditis elegans (C. elegan) datasets.
87 omatin and nucleoli during cell division and Caenorhabditis elegans (C. elegans) growth.
88 ctivation of the UPR(ER) in the intestine of Caenorhabditis elegans (C. elegans) through release of a
89 ied by the dauer larva stage of the nematode Caenorhabditis elegans (C. elegans).
90 ibed a physiological program in the nematode Caenorhabditis elegans called the intracellular pathogen
91 how that bloating of the intestinal lumen of Caenorhabditis elegans caused by microbial colonization
92 fied a gain-of-function (gf) mutation in the Caenorhabditis elegans CaV2 channel alpha1 subunit, UNC-
93 oteins and an unexpected direct link between Caenorhabditis elegans CFP-1 and an Rpd3/Sin3 small (SIN
94                                  Here, using Caenorhabditis elegans chemosensory neurons as a model s
95 herefore, simple organisms like the nematode Caenorhabditis elegans, combining relevant advantages of
96                                              Caenorhabditis elegans consumes bacteria, which can supp
97                              We show that in Caenorhabditis elegans COQ-2e is required for efficient
98          Here the role of human AMPK and its Caenorhabditis elegans counterpart AAK-2 was explored up
99 ry element activities across five tissues of Caenorhabditis elegans, covering ~90% of cells.
100  This subunit, dSol-1, is the homolog of the Caenorhabditis elegans CUB (Complement C1r/C1s, Uegf, Bm
101 e atomic cryo-EM structures of a full-length Caenorhabditis elegans cyclic GMP-activated channel TAX-
102 ve L3 larvae, an arrested stage analogous to Caenorhabditis elegans dauer larvae.
103 actors implicated in translation fidelity in Caenorhabditis elegans decreases lifespan, and eEF2K is
104                                           In Caenorhabditis elegans, deficiency of DCAP-1/DCP1, the e
105                     Research in the nematode Caenorhabditis elegans demonstrates that retrieval of av
106                             We show that the Caenorhabditis elegans DIP-2 maintains morphology of mat
107 is regard, we summarize recent studies using Caenorhabditis elegans, Drosophila melanogaster, Danio r
108 ets across species inclusive of Arabidopsis, Caenorhabditis elegans, Drosophila, human, mouse, rat, y
109                                              Caenorhabditis elegans early embryos generate cell-speci
110 namic responses of touch receptor neurons in Caenorhabditis elegans (Eastwood et al., 2015), which su
111 erotonin-dependent behavior of the roundworm Caenorhabditis elegans, egg laying, to perform a behavio
112      We followed the first nine divisions of Caenorhabditis elegans embryo development and demonstrat
113                                          The Caenorhabditis elegans embryo is an important model for
114                        Here we use the early Caenorhabditis elegans embryo to explore how the actin f
115 ce in situ hybridization (smFISH) studies in Caenorhabditis elegans embryogenesis.
116 nvestigated cytokinesis during the invariant Caenorhabditis elegans embryonic divisions and found sev
117 ression levels of APA isoforms in individual Caenorhabditis elegans embryos at different stages throu
118 robe and control the cell-division timing in Caenorhabditis elegans embryos using a combination of lo
119                                           In Caenorhabditis elegans embryos, chromodomain protein CEC
120 ically infects the laboratory model nematode Caenorhabditis elegans, encodes a fibrous protein delta
121              Here, we show that the nematode Caenorhabditis elegans exhibits the key features of alco
122  report that the noncanonical sHsp HSP-17 of Caenorhabditis elegans facilitates aggregation of model
123                     We show that FAMK-1, the Caenorhabditis elegans Fam20C orthologue, contributes to
124                Apoe(-/-) mice expressing the Caenorhabditis elegans Fat-1 transgene (Fat-1(tg)xApoe(-
125 hat govern the formation of gap junctions in Caenorhabditis elegans, five of which are already suppor
126 ng, we performed a forward genetic screen in Caenorhabditis elegans for genes involved in body size r
127 on developmental gene expression dataset for Caenorhabditis elegans from Bao, Murray, Waterston et al
128 lar range cysteamine bitartrate treatment in Caenorhabditis elegans gas-1(fc21) RC complex I (NDUFS2-
129                               We performed a Caenorhabditis elegans genetic screen to find cellular f
130                   We deleted mafr-1 from the Caenorhabditis elegans genome and found that animals lac
131 H Oligopaint library that targets the entire Caenorhabditis elegans genome at chromosome, three megab
132 onserved homeodomain proteins encoded by the Caenorhabditis elegans genome(2), that the complete set
133                             Here, we use the Caenorhabditis elegans germline as a model to investigat
134 ins FBF-1 and FBF-2 have opposite effects on Caenorhabditis elegans germline stem cell dynamics: FBF-
135                                       In the Caenorhabditis elegans germline, fem-3 Binding Factor (F
136 e-like glia, which extended the life span in Caenorhabditis elegans Glial XBP-1s initiated a robust c
137                                       In the Caenorhabditis elegans gonad, the broad outlines of germ
138 pling between the soma and germ cells in the Caenorhabditis elegans gonad.
139 rain to induce CA production directly in the Caenorhabditis elegans gut, we reveal the local effect o
140                                 The nematode Caenorhabditis elegans has an alternate dispersal life s
141                                              Caenorhabditis elegans has long been a laboratory model
142                       Some nematodes such as Caenorhabditis elegans have an XO sex determination syst
143 sed by all neurons in model systems, such as Caenorhabditis elegans have left still unresolved how ne
144    Hawaiian isolates of the nematode species Caenorhabditis elegans have long been known to harbor ge
145 netic studies on the excretory canal cell of Caenorhabditis elegans have revealed many proteins that
146                                        Young Caenorhabditis elegans hermaphrodites use their own sper
147  TFEB and TFE3, as well as their ortholog in Caenorhabditis elegans HLH-30, play an important role in
148 nd genetic analysis, we found that catp-6, a Caenorhabditis elegans homolog of ATP13A2, was responsib
149                               UNC-104 is the Caenorhabditis elegans homolog of kinesin-3 KIF1A known
150 ere, we study the effect of different crh-1 [Caenorhabditis elegans homolog of mammalian cAMP respons
151 zing proteome-wide mass spectrometry data of Caenorhabditis elegans, however, we show that the levels
152                         Here we show that in Caenorhabditis elegans HS induces up- and downregulation
153 regulates EV levels in the sensory organs of Caenorhabditis elegans in a cilia specific manner.
154 mechanical properties associated with ageing Caenorhabditis elegans in addition to capturing high-res
155 on in HD models (cells, primary neurons, and Caenorhabditis elegans) increases mutant HTT exon 1 phos
156 cells as well as Drosophila melanogaster and Caenorhabditis elegans, indicating suitability for use i
157 successfully reduced the fungal burdens in a Caenorhabditis elegans infection model by up to 96%.
158                           Additionally, in a Caenorhabditis elegans infection model, the ospemifene-i
159 utative FAM173B orthologue from the nematode Caenorhabditis elegans Interestingly, lack of Lys-43 met
160                                 The nematode Caenorhabditis elegans is a bacterivore filter feeder.
161                     The free-living nematode Caenorhabditis elegans is a key laboratory model for met
162  on the host strain.IMPORTANCE The roundworm Caenorhabditis elegans is a laboratory model organism in
163                  The pharynx of the nematode Caenorhabditis elegans is a simple neuromuscular organ w
164                                 The nematode Caenorhabditis elegans is a useful model for studying th
165                                              Caenorhabditis elegans is a valuable model organism in b
166                                              Caenorhabditis elegans is an animal with few cells but a
167                           The model nematode Caenorhabditis elegans is ideal to study these interacti
168                          The endoderm GRN in Caenorhabditis elegans is initiated by the maternally su
169                                              Caenorhabditis elegans is used extensively as a medical
170                                           In Caenorhabditis elegans, Katanin MT-severing activity is
171              Upon sensing starvation stress, Caenorhabditis elegans larvae (L2d) elicit two seemingly
172 can modulate the developmental trajectory of Caenorhabditis elegans larvae by promoting entry into da
173      We generate CRISPR knockout strains for Caenorhabditis elegans lincRNAs and evaluate their pheno
174                                           In Caenorhabditis elegans, low vitamin B12, or genetic pert
175 -shared neurons in the nervous system of the Caenorhabditis elegans male is controlled by the tempora
176 g a conspicuous muscle change that occurs in Caenorhabditis elegans males.
177 tion channel complex in the sensory cilia of Caenorhabditis elegans mechanoreceptor neurons.
178 ayer in coordinating these key events during Caenorhabditis elegans meiosis.
179  specific isoform of CREB1/CRH-1, CRH-1e, in Caenorhabditis elegans memory formation and chemosensati
180                                           In Caenorhabditis elegans, MET-2, the homologue of mammalia
181 -helix transcription factor 30 (hlh-30), the Caenorhabditis elegans MiT/TFE ortholog, to starvation f
182 beta-induced paralysis in a transgenic Abeta Caenorhabditis elegans model and specifically target and
183  To this end, we have generated a transgenic Caenorhabditis elegans model expressing both human Abeta
184                   Previously, we generated a Caenorhabditis elegans model of ALS, in which the expres
185 ogical tau protein, we employed a transgenic Caenorhabditis elegans model of human tauopathy exhibiti
186                    A genome-wide screen in a Caenorhabditis elegans model of SOD1-linked ALS identifi
187 re, we develop an experimental handle in the Caenorhabditis elegans model system, in which we uncover
188 MP resistance show enhanced virulence in the Caenorhabditis elegans model, an effect that is abolishe
189 rough CRISPR/Cas9-mediated gene editing in a Caenorhabditis elegans model.
190 en suggested to regulate tau inclusions in a Caenorhabditis elegans model.
191         In both amyloid-beta (Abeta) and tau Caenorhabditis elegans models of AD, mitophagy stimulati
192 hes to better understand the function of the Caenorhabditis elegans MORC-1 protein.
193 pmental expression of three conserved genes, Caenorhabditis elegans mpk-1, lag-1, and lag-3/sel-8, wh
194 e diseases, and we show that the lack of the Caenorhabditis elegans Msp1 homologue triggers an import
195 hondria are arrayed in a periodic pattern in Caenorhabditis elegans muscle, but this pattern is disru
196                                              Caenorhabditis elegans must distinguish pathogens from n
197                            Here, we analyzed Caenorhabditis elegans mutants deleted of the sole SEIPI
198                                           In Caenorhabditis elegans, mutations in WDR-5 and other com
199 d nucleus deformation dynamics within living Caenorhabditis elegans nematodes.
200 e retraction is shown to be operative in the Caenorhabditis elegans nervous system.
201                                           In Caenorhabditis elegans, neural excitation increases with
202  to visualize alternative splicing in single Caenorhabditis elegans neurons, identifying complex spli
203 e in primary fibroblasts, murine neurons and Caenorhabditis elegans neurons.
204 ant for controlling levels of DCV cargoes in Caenorhabditis elegans neurons.
205  we discovered a novel coordinated action of Caenorhabditis elegans neuropeptide Y/neuropeptide F and
206                                          The Caenorhabditis elegans NPY/NPF ortholog FLP-34 displays
207   Here we report our investigations on three Caenorhabditis elegans orthologous proteins involved in
208                      We also report that the Caenorhabditis elegans orthologs of 14-3-3zeta and MSec
209 subunits partially rescue sensory defects in Caenorhabditis elegans osm-9 and tax-4 knock-out strains
210 of Alzheimer's disease in the model organism Caenorhabditis elegans Our results pose and answer the q
211                                           In Caenorhabditis elegans, piRNA loci are clustered within
212 e we describe the emerging model system of a Caenorhabditis elegans polymodal neuron named PVD, whose
213                                              Caenorhabditis elegans possess a mild inflammatory respo
214                                 The nematode Caenorhabditis elegans possesses glial types similar to
215 of integrin-mediated collagen recruitment to Caenorhabditis elegans postembryonic gonadal and pharyng
216                                              Caenorhabditis elegans primordial germ cells (PGCs) jett
217                                 The nematode Caenorhabditis elegans produces a broad family of pherom
218                       Here we identified the Caenorhabditis elegans protein huntingtin-interacting pr
219 re, we report the identification of PLP-1, a Caenorhabditis elegans protein related to the human sing
220                           Here we describe a Caenorhabditis elegans quiescent behavior, post-response
221                                           In Caenorhabditis elegans, RBPs control the translation, st
222                                           In Caenorhabditis elegans, reduced mitochondrial electron t
223                                           In Caenorhabditis elegans, regulation of piRNA target genes
224                       The spermatheca of the Caenorhabditis elegans reproductive system enables study
225 EB) transcription factor significantly slows Caenorhabditis elegans' reproductive decline, an early h
226  dbt-1 gene underlies natural differences in Caenorhabditis elegans responses to the toxin arsenic.
227  The expression of chimeric hnRNPA2 D290V in Caenorhabditis elegans results in stress-induced glutama
228 R-generated, fluorescently tagged endogenous Caenorhabditis elegans RHEB-1 and DAF-15/Raptor are expr
229                                          The Caenorhabditis elegans RIG-I-like receptor DRH-1 promote
230                                           In Caenorhabditis elegans, RNA interference (RNAi) response
231                                           In Caenorhabditis elegans, RNAi can be achieved by feeding
232 colleagues provide evidence that exposure of Caenorhabditis elegans roundworms to 2 simple nutrients,
233 l-angle x-ray scattering) and in vivo tools (Caenorhabditis elegans sarcomere structure).
234                              We show that in Caenorhabditis elegans, serotonin released by maternal n
235 velopment in a subpopulation of synchronized Caenorhabditis elegans, sets processes in motion that in
236 repeated induction of the flight response in Caenorhabditis elegans shortens lifespan and inhibits co
237 gene expression analyses in P. univalens and Caenorhabditis elegans show that the intestine is the ma
238                                           In Caenorhabditis elegans, signals from the germline initia
239                                           In Caenorhabditis elegans sleep occurs during a larval tran
240                                       During Caenorhabditis elegans sleep, energetic stores are alloc
241        We use the myoepithelial cells of the Caenorhabditis elegans spermatheca to study the mechanis
242 to a brief 2 degrees C temperature increase, Caenorhabditis elegans spermatocytes exhibit up to a 25-
243 bservations were recapitulated in vivo, in a Caenorhabditis elegans strain deficient in the ATP13A2 o
244                               A new study in Caenorhabditis elegans suggests the ubiquitin-proteasome
245                              For example, in Caenorhabditis elegans, SUN protein UNC-84 binds to two
246 e for the whole-life culture of the nematode Caenorhabditis elegans that allows the scoring of animal
247  identify a neuronal circuit in the nematode Caenorhabditis elegans that processes information percei
248 he model agrees with experiments in the worm Caenorhabditis elegans that show the following: Life spa
249                    We have recently shown in Caenorhabditis elegans that transcript levels of sqst-1/
250                                           In Caenorhabditis elegans, the avoidance response to gentle
251                        Here, we show that in Caenorhabditis elegans, the axons of the ALA neuron cont
252                                           In Caenorhabditis elegans, the BMP ligand DBL-1 is a regula
253                                           In Caenorhabditis elegans, the closest BUBR1 orthologue lac
254                                           In Caenorhabditis elegans, the heterochronic pathway contro
255                        To test this, we used Caenorhabditis elegans, the intestinal cells of which ar
256 ntrations in most studied species, including Caenorhabditis elegans, the molecular mechanisms behind
257                         Here we show that in Caenorhabditis elegans, the neuromodulator tyramine prod
258                                           In Caenorhabditis elegans, the transcription factor HBL-1 (
259                                           In Caenorhabditis elegans, the transcription factor, SKN-1,
260                              In the nematode Caenorhabditis elegans, the transcription regulators DAF
261                              In the nematode Caenorhabditis elegans, this highly efficient regenerati
262 which male-specific neurons are generated in Caenorhabditis elegans through the direct transdifferent
263 omeric organization and muscle function from Caenorhabditis elegans to humans.
264                      We have used RNA-seq in Caenorhabditis elegans to produce transcription profiles
265                                Here, we used Caenorhabditis elegans to systematically functionally ch
266 icellular settings, from the model organism, Caenorhabditis elegans, to humans.
267 lls, a population of epidermal stem cells in Caenorhabditis elegans, to study the influence of temper
268        For example, in the annotation of the Caenorhabditis elegans transcriptome, more than half of
269 come well established that the nematode worm Caenorhabditis elegans triggers innate immune responses
270 usly shown that 2 paralogous tetraspanins in Caenorhabditis elegans, TSP-12 and TSP-14, function redu
271                              In the nematode Caenorhabditis elegans, UNC-87 is a calponin-related pro
272                                           In Caenorhabditis elegans, unc-89 encodes a set of giant mu
273 s controlled by Munc18 (mammalian homolog of Caenorhabditis elegans uncoordinated gene 18) proteins.
274                                          The Caenorhabditis elegans vulva has been a paradigm for und
275                                              Caenorhabditis elegans was the first multicellular eukar
276 regulates its trafficking and degradation in Caenorhabditis elegans We found that the WD40-repeat pro
277 multipotent vulval precursor cells (VPCs) of Caenorhabditis elegans We have previously shown that thi
278   Using the touch receptor neurons (TRNs) of Caenorhabditis elegans, we find that mec-15(-) mutants d
279 ructions of spermatocyte meiotic spindles in Caenorhabditis elegans, we find the lagging X chromosome
280 ors of heat-shock-induced gene expression in Caenorhabditis elegans, we found a new allele of hsf-1 t
281               Using the in vivo model system Caenorhabditis elegans, we found that the highly conserv
282                                  Here, using Caenorhabditis elegans, we generated the first organism
283 n coupled with deep-sequencing (TRAP-seq) in Caenorhabditis elegans, we have obtained high coverage p
284                    Using forward genetics in Caenorhabditis elegans, we identified a conserved orphan
285 encing and genomic and genetic approaches in Caenorhabditis elegans, we reveal an unprecedented role
286                        Using optogenetics in Caenorhabditis elegans, we solved the presynaptic circui
287  and siRNAs in regulating gene expression in Caenorhabditis elegans, we subjected small RNAs and mRNA
288               In a forward genetic screen in Caenorhabditis elegans, we uncover that TALP-3, a homolo
289 increased in young amyloid B (AB) expressing Caenorhabditis elegans, whereas it is not in wild type s
290 odels zebrafish, Drosophila melanogaster and Caenorhabditis elegans, which command superb research re
291 ned by the number of traps after exposure to Caenorhabditis elegans While some strains were highly se
292  neuronal tissue, mouse embryonic tissue and Caenorhabditis elegans whole embryos.
293                  In simple organisms such as Caenorhabditis elegans, whole brain imaging has been per
294 riation in the behavioral flexibility of two Caenorhabditis elegans wild strains.
295  measured touch-induced mechanical strain in Caenorhabditis elegans worms.
296                                           In Caenorhabditis elegans, wounding the epidermis triggers
297        During the asymmetric division of the Caenorhabditis elegans zygote, germ (P) granules are dis
298                   Using the newly fertilized Caenorhabditis elegans zygote, we show that the mitotic
299                                           In Caenorhabditis elegans zygotes, PAR-1 localizes to the p
300                                 In wild-type Caenorhabditis elegans zygotes, symmetry breaking during

 
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