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1 erves this function in human cells (LEM-3 in Caenorhabditis elegans).
2 obes in a complex multicellular model (i.e., Caenorhabditis elegans).
3 f two new SC components, SYP-5 and SYP-6, in Caenorhabditis elegans.
4 of proteins to chemosensory cilia in living Caenorhabditis elegans.
5 homologous recombination-mediated repair in Caenorhabditis elegans.
6 rous iron accumulates over adult lifetime in Caenorhabditis elegans.
7 tol's effects on fatty acid metabolism using Caenorhabditis elegans.
8 e molecular bases of this organization using Caenorhabditis elegans.
9 g m(5)C at position C2381 on the 26S rRNA in Caenorhabditis elegans.
10 ll ferroptosis and sterility in the nematode Caenorhabditis elegans.
11 cleavage to attract glia during migration in Caenorhabditis elegans.
12 during embryonic development of the nematode Caenorhabditis elegans.
13 ween Pseudomonas aeruginosa and the nematode Caenorhabditis elegans.
14 n an in vivo system using the model organism Caenorhabditis elegans.
15 e been best described for the model organism Caenorhabditis elegans.
16 mentally identified piRNA targeting sites in Caenorhabditis elegans.
17 ting the speed of locomotion of the nematode Caenorhabditis elegans.
18 educe lipid storage and promote longevity in Caenorhabditis elegans.
19 n structural analysis in vivo (IV-FPOP) with Caenorhabditis elegans.
20 alpha-synuclein-driven neurodegeneration in Caenorhabditis elegans.
21 , followed by functional characterization in Caenorhabditis elegans.
22 thereof on the reproduction of the nematode Caenorhabditis elegans.
23 with the membraneless organelle P granule in Caenorhabditis elegans.
24 tic protein phosphofructokinase-1/PFK-1.1 in Caenorhabditis elegans.
25 hoprim exhibited increased virulence against Caenorhabditis elegans.
26 y small silencing RNAs in the model nematode Caenorhabditis elegans.
27 A (ncRNA) essential for axon regeneration in Caenorhabditis elegans.
28 nactive one, controls checkpoint strength in Caenorhabditis elegans.
29 terograde synaptic organizer in the nematode Caenorhabditis elegans.
30 scales of hierarchical locomotion control in Caenorhabditis elegans.
31 we address this question using heteroplasmic Caenorhabditis elegans.
32 tivity in the nervous system of the nematode Caenorhabditis elegans.
34 we report that mitochondrial dysfunction in Caenorhabditis elegans activates RNAi-directed silencing
38 s involved in glial size regulation, we used Caenorhabditis elegans amphid sheath (AMsh) glia as a mo
39 ere we develop tests for IIA in the nematode Caenorhabditis elegans, an animal with only 302 neurons,
42 of the dosage compensation complex (DCC) in Caenorhabditis elegans and demonstrate that loop anchors
43 e mechanisms underlying UPR(mt) signaling in Caenorhabditis elegans and discuss emerging connections
44 as leveraged simple model organisms, such as Caenorhabditis elegans and Drosophila melanogaster larva
45 d aging, including stem cell dysfunction, in Caenorhabditis elegans and Drosophila melanogaster model
48 ulfide reduction exclusively in endosomes in Caenorhabditis elegans and identified that exchange is m
49 e paralysis, we conducted genetic screens in Caenorhabditis elegans and isolated mutants that became
52 fication of Abeta oligomers in vitro, and in Caenorhabditis elegans and mouse hippocampal tissues.
53 mporal regulation of LAG-1/CSL expression in Caenorhabditis elegans and observed that an increase in
55 ptide F (NPF)-related neuropeptide system in Caenorhabditis elegans and show that this FLP-34/NPR-11
56 vensis is a natural pathogen of the nematode Caenorhabditis elegans and that parental exposure of ani
57 t method relying on the free-living nematode Caenorhabditis elegans and the infection of those nemato
58 mental inhibitor of the free-living nematode Caenorhabditis elegans and the plant-parasitic nematode
59 on of a long-term swim exercise protocol for Caenorhabditis elegans and we demonstrate its benefits t
61 couple these key events of the cell cycle in Caenorhabditis elegans and zebrafish through live-cell i
62 dopsis thaliana), rice (Oryza sativa), worm (Caenorhabditis elegans), and human (Homo sapiens) cells
64 bolism without affecting eating behaviors in Caenorhabditis elegans, and identified specific odors th
67 e, whereas Smn from Drosophila melanogaster, Caenorhabditis elegans, and Schizosaccharomyces pombe wa
68 of a related nonparasitic nematode species, Caenorhabditis elegans, and the parasitic nematode Haemo
69 europeptide Y/neuropeptide F in the nematode Caenorhabditis elegans, and we discovered that it is req
72 rpose, we have designed a pipeline that uses Caenorhabditis elegans as a genetic model to screen for
75 ral, biochemical, and functional analyses in Caenorhabditis elegans As capsaicin elicits heat and pai
81 tant proteins remain enriched at synapses in Caenorhabditis elegans, but show defects in active-zone
82 nd prodigiosin antibiotics, and infection in Caenorhabditis elegans, but up-regulating flagellar moti
83 ogical mechanisms that delay neuron aging in Caenorhabditis elegans by asking how neuron morphologica
84 We developed a glutamate spillover model in Caenorhabditis elegans by inactivating the conserved gli
88 ctivation of the UPR(ER) in the intestine of Caenorhabditis elegans (C. elegans) through release of a
90 ibed a physiological program in the nematode Caenorhabditis elegans called the intracellular pathogen
91 how that bloating of the intestinal lumen of Caenorhabditis elegans caused by microbial colonization
92 fied a gain-of-function (gf) mutation in the Caenorhabditis elegans CaV2 channel alpha1 subunit, UNC-
93 oteins and an unexpected direct link between Caenorhabditis elegans CFP-1 and an Rpd3/Sin3 small (SIN
95 herefore, simple organisms like the nematode Caenorhabditis elegans, combining relevant advantages of
100 This subunit, dSol-1, is the homolog of the Caenorhabditis elegans CUB (Complement C1r/C1s, Uegf, Bm
101 e atomic cryo-EM structures of a full-length Caenorhabditis elegans cyclic GMP-activated channel TAX-
103 actors implicated in translation fidelity in Caenorhabditis elegans decreases lifespan, and eEF2K is
107 is regard, we summarize recent studies using Caenorhabditis elegans, Drosophila melanogaster, Danio r
108 ets across species inclusive of Arabidopsis, Caenorhabditis elegans, Drosophila, human, mouse, rat, y
110 namic responses of touch receptor neurons in Caenorhabditis elegans (Eastwood et al., 2015), which su
111 erotonin-dependent behavior of the roundworm Caenorhabditis elegans, egg laying, to perform a behavio
112 We followed the first nine divisions of Caenorhabditis elegans embryo development and demonstrat
116 nvestigated cytokinesis during the invariant Caenorhabditis elegans embryonic divisions and found sev
117 ression levels of APA isoforms in individual Caenorhabditis elegans embryos at different stages throu
118 robe and control the cell-division timing in Caenorhabditis elegans embryos using a combination of lo
120 ically infects the laboratory model nematode Caenorhabditis elegans, encodes a fibrous protein delta
122 report that the noncanonical sHsp HSP-17 of Caenorhabditis elegans facilitates aggregation of model
125 hat govern the formation of gap junctions in Caenorhabditis elegans, five of which are already suppor
126 ng, we performed a forward genetic screen in Caenorhabditis elegans for genes involved in body size r
127 on developmental gene expression dataset for Caenorhabditis elegans from Bao, Murray, Waterston et al
128 lar range cysteamine bitartrate treatment in Caenorhabditis elegans gas-1(fc21) RC complex I (NDUFS2-
131 H Oligopaint library that targets the entire Caenorhabditis elegans genome at chromosome, three megab
132 onserved homeodomain proteins encoded by the Caenorhabditis elegans genome(2), that the complete set
134 ins FBF-1 and FBF-2 have opposite effects on Caenorhabditis elegans germline stem cell dynamics: FBF-
136 e-like glia, which extended the life span in Caenorhabditis elegans Glial XBP-1s initiated a robust c
139 rain to induce CA production directly in the Caenorhabditis elegans gut, we reveal the local effect o
143 sed by all neurons in model systems, such as Caenorhabditis elegans have left still unresolved how ne
144 Hawaiian isolates of the nematode species Caenorhabditis elegans have long been known to harbor ge
145 netic studies on the excretory canal cell of Caenorhabditis elegans have revealed many proteins that
147 TFEB and TFE3, as well as their ortholog in Caenorhabditis elegans HLH-30, play an important role in
148 nd genetic analysis, we found that catp-6, a Caenorhabditis elegans homolog of ATP13A2, was responsib
150 ere, we study the effect of different crh-1 [Caenorhabditis elegans homolog of mammalian cAMP respons
151 zing proteome-wide mass spectrometry data of Caenorhabditis elegans, however, we show that the levels
154 mechanical properties associated with ageing Caenorhabditis elegans in addition to capturing high-res
155 on in HD models (cells, primary neurons, and Caenorhabditis elegans) increases mutant HTT exon 1 phos
156 cells as well as Drosophila melanogaster and Caenorhabditis elegans, indicating suitability for use i
157 successfully reduced the fungal burdens in a Caenorhabditis elegans infection model by up to 96%.
159 utative FAM173B orthologue from the nematode Caenorhabditis elegans Interestingly, lack of Lys-43 met
162 on the host strain.IMPORTANCE The roundworm Caenorhabditis elegans is a laboratory model organism in
172 can modulate the developmental trajectory of Caenorhabditis elegans larvae by promoting entry into da
173 We generate CRISPR knockout strains for Caenorhabditis elegans lincRNAs and evaluate their pheno
175 -shared neurons in the nervous system of the Caenorhabditis elegans male is controlled by the tempora
179 specific isoform of CREB1/CRH-1, CRH-1e, in Caenorhabditis elegans memory formation and chemosensati
181 -helix transcription factor 30 (hlh-30), the Caenorhabditis elegans MiT/TFE ortholog, to starvation f
182 beta-induced paralysis in a transgenic Abeta Caenorhabditis elegans model and specifically target and
183 To this end, we have generated a transgenic Caenorhabditis elegans model expressing both human Abeta
185 ogical tau protein, we employed a transgenic Caenorhabditis elegans model of human tauopathy exhibiti
187 re, we develop an experimental handle in the Caenorhabditis elegans model system, in which we uncover
188 MP resistance show enhanced virulence in the Caenorhabditis elegans model, an effect that is abolishe
193 pmental expression of three conserved genes, Caenorhabditis elegans mpk-1, lag-1, and lag-3/sel-8, wh
194 e diseases, and we show that the lack of the Caenorhabditis elegans Msp1 homologue triggers an import
195 hondria are arrayed in a periodic pattern in Caenorhabditis elegans muscle, but this pattern is disru
202 to visualize alternative splicing in single Caenorhabditis elegans neurons, identifying complex spli
205 we discovered a novel coordinated action of Caenorhabditis elegans neuropeptide Y/neuropeptide F and
207 Here we report our investigations on three Caenorhabditis elegans orthologous proteins involved in
209 subunits partially rescue sensory defects in Caenorhabditis elegans osm-9 and tax-4 knock-out strains
210 of Alzheimer's disease in the model organism Caenorhabditis elegans Our results pose and answer the q
212 e we describe the emerging model system of a Caenorhabditis elegans polymodal neuron named PVD, whose
215 of integrin-mediated collagen recruitment to Caenorhabditis elegans postembryonic gonadal and pharyng
219 re, we report the identification of PLP-1, a Caenorhabditis elegans protein related to the human sing
225 EB) transcription factor significantly slows Caenorhabditis elegans' reproductive decline, an early h
226 dbt-1 gene underlies natural differences in Caenorhabditis elegans responses to the toxin arsenic.
227 The expression of chimeric hnRNPA2 D290V in Caenorhabditis elegans results in stress-induced glutama
228 R-generated, fluorescently tagged endogenous Caenorhabditis elegans RHEB-1 and DAF-15/Raptor are expr
232 colleagues provide evidence that exposure of Caenorhabditis elegans roundworms to 2 simple nutrients,
235 velopment in a subpopulation of synchronized Caenorhabditis elegans, sets processes in motion that in
236 repeated induction of the flight response in Caenorhabditis elegans shortens lifespan and inhibits co
237 gene expression analyses in P. univalens and Caenorhabditis elegans show that the intestine is the ma
242 to a brief 2 degrees C temperature increase, Caenorhabditis elegans spermatocytes exhibit up to a 25-
243 bservations were recapitulated in vivo, in a Caenorhabditis elegans strain deficient in the ATP13A2 o
246 e for the whole-life culture of the nematode Caenorhabditis elegans that allows the scoring of animal
247 identify a neuronal circuit in the nematode Caenorhabditis elegans that processes information percei
248 he model agrees with experiments in the worm Caenorhabditis elegans that show the following: Life spa
256 ntrations in most studied species, including Caenorhabditis elegans, the molecular mechanisms behind
262 which male-specific neurons are generated in Caenorhabditis elegans through the direct transdifferent
267 lls, a population of epidermal stem cells in Caenorhabditis elegans, to study the influence of temper
269 come well established that the nematode worm Caenorhabditis elegans triggers innate immune responses
270 usly shown that 2 paralogous tetraspanins in Caenorhabditis elegans, TSP-12 and TSP-14, function redu
273 s controlled by Munc18 (mammalian homolog of Caenorhabditis elegans uncoordinated gene 18) proteins.
276 regulates its trafficking and degradation in Caenorhabditis elegans We found that the WD40-repeat pro
277 multipotent vulval precursor cells (VPCs) of Caenorhabditis elegans We have previously shown that thi
278 Using the touch receptor neurons (TRNs) of Caenorhabditis elegans, we find that mec-15(-) mutants d
279 ructions of spermatocyte meiotic spindles in Caenorhabditis elegans, we find the lagging X chromosome
280 ors of heat-shock-induced gene expression in Caenorhabditis elegans, we found a new allele of hsf-1 t
283 n coupled with deep-sequencing (TRAP-seq) in Caenorhabditis elegans, we have obtained high coverage p
285 encing and genomic and genetic approaches in Caenorhabditis elegans, we reveal an unprecedented role
287 and siRNAs in regulating gene expression in Caenorhabditis elegans, we subjected small RNAs and mRNA
289 increased in young amyloid B (AB) expressing Caenorhabditis elegans, whereas it is not in wild type s
290 odels zebrafish, Drosophila melanogaster and Caenorhabditis elegans, which command superb research re
291 ned by the number of traps after exposure to Caenorhabditis elegans While some strains were highly se