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1 eneralist lineages of the zoonotic bacterium Campylobacter.
2 ng that two resistance mechanisms existed in Campylobacter.
3  adequate for protecting against exposure to Campylobacter.
4 ylobacter, indicating their functionality in Campylobacter.
5  involved in generating the helical shape of Campylobacter.
6 t-of-care screening of chicken carcasses for Campylobacter.
7 uding Helicobacter, Chlamydia, Brucella, and Campylobacter.
8  of infection is an estimated 1 in 23500 for Campylobacter, 1 in 5050000 for Cryptosporidium, and 1 i
9 pathogens Shigella (36%), Giardia (33%), and Campylobacter (30%) predominated, but their presence was
10                          Presently, the anti-Campylobacter activity of thymol was compared against th
11 e results identify a key virulence factor in Campylobacter and a potential target for the control of
12 erience diarrhea show increased abundance of Campylobacter and a reduction in Helicobacter macacae.
13 can be extended to include other loci within Campylobacter and developed for molecular epidemiology s
14 erythromycin, a total of eight strains (four Campylobacter and four Enterococcus) obtained macrolide-
15  acute gastroenteritis and culture-confirmed Campylobacter and nontyphoidal Salmonella infections in
16 n reportable enteric pathogen detected after Campylobacter and the most detected diarrheagenic E. col
17  motors that produce different torques, from Campylobacter and Vibrio species.
18  TM7, and 11 genera, including Anaeroplasma, Campylobacter, and Clostridium, were correlated with app
19 er in Actinomyces, Selenomonas, Veillonella, Campylobacter, and TM7 [G-1] than the Swedish groups.
20 lla, specific multidrug-resistant strains of Campylobacter are circulating by sexual transmission in
21 lla, specific multidrug-resistant strains of Campylobacter are circulating by sexual transmission in
22          Pathogenic species within the genus Campylobacter are responsible for a considerable burden
23 ed to estimate aerobic plate count (APC) and Campylobacter as well as Salmonella prevalence.
24       Several reports have demonstrated that Campylobacter bacteriophage DNA is refractory to manipul
25                                  Among them, Campylobacter, Bacteroides, Porphyromonas, Lachnospirace
26         In microaerophilic organisms such as Campylobacter, biofilms play a key role in transmission
27 d 13 bacterial taxa indicative of cumulative Campylobacter burden and 14 taxa significantly associate
28                                       A high Campylobacter burden was associated with a lower length-
29 identified taxa specifically associated with Campylobacter burden.
30 omach, including Fusobacterium, Megasphaera, Campylobacter, Capnocytophaga, and Dialister.
31          We propose to rename Tlp11 as CcrG, Campylobacter ChemoReceptor for Galactose.
32 ins that were similar to those identified in Campylobacter chromosomal DNA.
33             We purified R.PabI homologs from Campylobacter coli (R.CcoLI) and Helicobacter pylori (R.
34 p., Shigella spp., Campylobacter jejuni, and Campylobacter coli and an EIA for Shiga toxins 1 and 2.
35               When treated with 1 mM thymol, Campylobacter coli and jejuni were reduced during pure o
36 rolide-resistant mutants were induced in one Campylobacter coli and one Enterococcus faecium strain,
37                     Campylobacter jejuni and Campylobacter coli are zoonotic pathogens once considere
38 ry, clinical and environmental C. jejuni and Campylobacter coli contained genetic changes within the
39  of 2 clonal lineages of multidrug-resistant Campylobacter coli from MSM in Seattle and Montreal.
40 f two clonal lineages of multidrug-resistant Campylobacter coli from MSM in Seattle, Washington and M
41 ferase situated in a CRISPR array locus in a Campylobacter coli isolate.
42 or the detection of Campylobacter jejuni and Campylobacter coli, leading global causes of bacterial g
43 /89 (89.9% sensitivity) Campylobacter jejuni/Campylobacter coli-positive cases.
44 ood-borne pathogens Campylobacter jejuni and Campylobacter coli.
45 n order to determine the natural patterns of Campylobacter colonization over a period of 63 weeks.
46                                              Campylobacter concisus is an emergent pathogen that play
47 s of PglC, a prototypic dual domain PGT from Campylobacter concisus Using a luminescence-based assay,
48                  We used reference pathogens Campylobacter, Cryptosporidium, and rotavirus as conserv
49  enterotoxigenic Escherichia coli, Shigella, Campylobacter, Cryptosporidium, norovirus GII, and adeno
50                                              Campylobacter data collected from several urban stormwat
51 s positively correlated with the presence of Campylobacter, Deinococcus, and Sulfurospirillum Finally
52 rch is required to better define whether the Campylobacter detectable in stormwater are pathogenic to
53    Factors associated with a reduced risk of Campylobacter detection included exclusive breastfeeding
54 ed excellent sensitivity and specificity for Campylobacter detection with low cross reactivity for ot
55 ntigen CIDTs compared to culture and PCR for Campylobacter detection.
56        Given the relatively low incidence of Campylobacter disease and the generally poor diagnostic
57            After immobilization of a genomic Campylobacter DNA onto a paper membrane, and addition of
58 lts suggest that there is natural control of Campylobacter dynamics within a flock which could potent
59                    A better understanding of Campylobacter epidemiology in commercial chicken flocks
60  for 9 bacterial enteropathogens: Aeromonas, Campylobacter, Escherichia coli O157, other Shiga toxin-
61 osis cases are acute and self-limiting, with Campylobacter excretion ceasing a few weeks after sympto
62                 In Montevideo (2013-2018), 8 Campylobacter fetus extraintestinal infections were repo
63 4 to December 2016, a cluster of 13 cases of Campylobacter fetus intestinal and extraintestinal infec
64                                              Campylobacter fetus is a venereal pathogen of cattle and
65 he substantial burden of apparently sporadic Campylobacter from cattle where transmission routes are
66 upled to biotin-Si-NPs successfully detected Campylobacter from naturally contaminated chicken meat,
67                                    A rise in Campylobacter gastroenteritis in an isolated population
68 versity in farm-derived samples and revealed Campylobacter genotypes that would not be detected using
69 m, Eikenella corrodens, Eubacterium nodatum, Campylobacter gracilis, Capnocytophaga sputigena, and Ve
70 ture-independent detection tests (CIDTs) for Campylobacter have become an area of intense controversy
71 single-stranded DNA-binding protein (SSB) of Campylobacter hominis Our findings indicate that SSB pos
72  the FlgK junction onto the structure of the Campylobacter hook provides some clues about its diverge
73 uctural constraints, due to the structure of Campylobacter hook, causes divergence of one element of
74        Retrospective study of MSM-associated Campylobacter in Seattle and Montreal with phenotypic an
75  was a retrospective study of MSM-associated Campylobacter in Seattle, Washington and Montreal, Quebe
76 ts, as standalone tests for the detection of Campylobacter in stool is increasing.
77  as standalone tests for direct detection of Campylobacter in stool.
78                       Structured sampling of Campylobacter in the clinic and from potential reservoir
79  on the epidemiology of antibiotic-resistant Campylobacter in the ruminant reservoir.
80 ndings reveal the rising prevalence of FQ(R) Campylobacter in the U.S. and provide novel information
81  the TA systems, pVir was readily cured from Campylobacter, indicating their functionality in Campylo
82 ion of two novel DNA modification systems in Campylobacter-infecting phages, which replace all guanos
83 rade 3]; diarrhoea and fever associated with Campylobacter infection [grade 3]; recurrence of abdomin
84 ntibiotic treatment may reduce the burden of Campylobacter infection and improve growth in children i
85 , we describe the epidemiology and impact of Campylobacter infection in the first 2 years of life.
86                                              Campylobacter infection is associated with impaired grow
87                                              Campylobacter infection is common in this cohort and ass
88                                              Campylobacter infection is the most commonly notified ba
89                                              Campylobacter infection was also associated with increas
90 echanisms, we evaluated associations between Campylobacter infection, linear growth, and fecal microb
91 oxacin, an antibiotic of choice for treating Campylobacter infection, through the pore of MOMP reveal
92 of the gastrointestinal tract as a result of Campylobacter infection.
93 dentify risk factors for sporadic intestinal Campylobacter infections and to determine the relative i
94 only 9% of nontyphoidal Salmonella and 4% of Campylobacter infections compared with 59% and 55% among
95              We describe the epidemiology of Campylobacter infections in the United States during 200
96 icken meat represents an important source of Campylobacter infections of humans world-wide.
97 out were the most important risk factors for Campylobacter infections.
98                                              Campylobacter is a pathogen frequently detected in urban
99  macrolides, in the major foodborne pathogen Campylobacter is considered a serious threat to public h
100                                              Campylobacter is the most common cause of foodborne bact
101 ntibiotic resistance genes have spread among Campylobacter isolated from humans, animals and the envi
102                                      Sixteen Campylobacter isolates were collected from the patient d
103                        Cultured Shigella and Campylobacter isolates were frequently resistant to mult
104  a patient with fecal specimens positive for Campylobacter jejuni (ST45) intermittently during a 10-y
105  to bacteria such as Pseudomonas aeruginosa, Campylobacter jejuni and Agrobacterium tumefaciens, whic
106          Human campylobacteriosis, caused by Campylobacter jejuni and C. coli, remains a leading caus
107                                              Campylobacter jejuni and Campylobacter coli are zoonotic
108 t targets mapA and ceuE for the detection of Campylobacter jejuni and Campylobacter coli, leading glo
109 resistance (AMR) in the food-borne pathogens Campylobacter jejuni and Campylobacter coli.
110     Monomeric OTases, such as the PglBs from Campylobacter jejuni and Campylobacter lari, catalyze tr
111                                              Campylobacter jejuni AR101 (Cj-P0) was introduced to chi
112 inked protein glycosylation (Pgl) pathway of Campylobacter jejuni are evaluated for their tolerance f
113                    The central enzyme in the Campylobacter jejuni asparagine-linked glycosylation pat
114  Here we report the cryo-EM structure of the Campylobacter jejuni cap complex, which reveals that Fli
115                                          The Campylobacter jejuni capsular polysaccharide is importan
116    Here, we report the crystal structures of Campylobacter jejuni Cas9 (CjCas9), one of the smallest
117                           To investigate how Campylobacter jejuni causes the clinical symptoms of dia
118 graphene quantum dot with surface protein in Campylobacter jejuni cell membrane.
119                                              Campylobacter jejuni Cj-P1-DCA-Anaero was isolated from
120 he structures and functional dynamics of the Campylobacter jejuni CmeB multidrug efflux pump.
121    Here the authors present the structure of Campylobacter jejuni CmeB pump combined with functional
122 udy, the effect of sustainable probiotics on Campylobacter jejuni colonization and gut microbiome com
123 NA (sgRNA)-guided and catalytically impaired Campylobacter jejuni CRISPR-associated protein 9-fused a
124                   One phase-variable gene of Campylobacter jejuni encodes a homologue of an unusual T
125 acterial oligosaccharyltransferase, PglB, of Campylobacter jejuni favors acceptor proteins with conse
126                           We discovered that Campylobacter jejuni FlhG is at the center of a multipar
127 ublic Health England received 25 isolates of Campylobacter jejuni from an individual with combined va
128 ced downregulation of NEIL2 was specific, as Campylobacter jejuni had no such effect.
129                                              Campylobacter jejuni harbors a branched electron transpo
130        A lack of relevant disease models for Campylobacter jejuni has long been an obstacle to resear
131                                              Campylobacter jejuni helical shape is important for colo
132 treptococcus pneumoniae in mice, and against Campylobacter jejuni in chicken.
133             Also, this method for monitoring Campylobacter jejuni in poultry liver was applied and re
134   Guillain-Barre syndrome is often caused by Campylobacter jejuni infection that has induced antibodi
135                                              Campylobacter jejuni is a commensal bacterium in the int
136                                              Campylobacter jejuni is a helix-shaped enteric bacterial
137                                              Campylobacter jejuni is a leading cause of bacterial gas
138                                              Campylobacter jejuni is a leading cause of enteric bacte
139                                              Campylobacter jejuni is a leading cause of foodborne ill
140                                              Campylobacter jejuni is a major cause of bacterial gastr
141                                              Campylobacter jejuni is a major zoonotic pathogen, and i
142                                              Campylobacter jejuni is a microaerophilic foodborne path
143                                              Campylobacter jejuni is a prevalent enteric pathogen tha
144                                              Campylobacter jejuni is a prevalent foodborne pathogen m
145                                              Campylobacter jejuni is a zoonotic pathogen and is one o
146                                              Campylobacter jejuni is a zoonotic pathogen, and a hyper
147                                              Campylobacter jejuni is an important zoonotic pathogen t
148                                              Campylobacter jejuni is one of the leading causes of bac
149                                              Campylobacter jejuni is one of the leading infectious ca
150                                              Campylobacter jejuni is the leading cause of foodborne b
151                                              Campylobacter jejuni is the leading cause of human bacte
152                   The Gram-negative organism Campylobacter jejuni is the major cause of food poisonin
153                                              Campylobacter jejuni is the most common cause of bacteri
154                sourceR is demonstrated using Campylobacter jejuni isolate data collected in New Zeala
155 he genetic basis of biofilm formation in 102 Campylobacter jejuni isolates.
156 pecific binding of graphene quantum dot with Campylobacter jejuni membrane leads to generate a distan
157                                              Campylobacter jejuni monitors intestinal metabolites pro
158 o-L-gluco-heptopyranose residue found in the Campylobacter jejuni NCTC11168 (HS:2) capsular polysacch
159                                   In lack of Campylobacter jejuni or in existence of other bacterial
160                            The characterized Campylobacter jejuni phages fall into two phylogenetic g
161 ml and ability of this FRET immunosensor for Campylobacter jejuni sensing in comparison with other ba
162 ber 2013, sexual transmission of 2 clades of Campylobacter jejuni subspecies jejuni isolates resulted
163                                           In Campylobacter jejuni the periplasmic binding protein Ceu
164      The microaerophilic food-borne pathogen Campylobacter jejuni uses complex cytochrome-rich respir
165 n resonance (SPR) for the rapid detection of Campylobacter jejuni was developed.
166 oxide and graphene quantum dot for detection Campylobacter jejuni whole cell in food samples was desi
167 nd Staphylococcus spp.; a zoonotic pathogen: Campylobacter jejuni) and antimicrobial resistance (AMR)
168 he most common food-born bacterial pathogen (Campylobacter jejuni) in the most prolific agricultural
169                                              Campylobacter jejuni, a leading bacterial cause of foodb
170                            Here we show that Campylobacter jejuni, a leading bacterial cause of human
171                                              Campylobacter jejuni, a leading cause of bacterial gastr
172                   We examined the TsdAs from Campylobacter jejuni, a microaerobic human pathogen, and
173 detection of Salmonella spp., Shigella spp., Campylobacter jejuni, and Campylobacter coli and an EIA
174 classes of inhibitors of Bacillus anthracis, Campylobacter jejuni, and Clostridium perfringens IMPDHs
175  Enterovirus, adenovirus A, Salmonella spp., Campylobacter jejuni, bovine polyomavirus, and bovine ro
176              The leading foodborne pathogen, Campylobacter jejuni, can carry multiple plasmids associ
177                  Another foodborne pathogen, Campylobacter jejuni, can mimic the GM1 ganglioside rece
178 erial species (Bacteroides thetaiotaomicron, Campylobacter jejuni, Enterococcus faecalis, Escherichia
179 d to spray-irrigated dairy manure containing Campylobacter jejuni, enterohemorrhagic Escherichia coli
180 n pathogens, such as Helicobacter pylori and Campylobacter jejuni, have escaped TLR5 activation by mu
181                                              Campylobacter jejuni, known for being a major cause of b
182 d the highest antimicrobial activity against Campylobacter jejuni, L. monocytogenes, and Pseudomonas
183 toxigenic Escherichia coli, Vibrio cholerae, Campylobacter jejuni, norovirus) in cohorts from Haiti,
184 s, Vibrio cholerae, Helicobacter pylori, and Campylobacter jejuni, organisms from three classes of Pr
185      The foodborne microaerophilic pathogen, Campylobacter jejuni, possesses a periplasmic formate de
186                                              Campylobacter jejuni, the leading cause of human bacteri
187                                              Campylobacter jejuni, the most common cause of bacterial
188                                              Campylobacter jejuni, the most frequent cause of food-bo
189 previously identified in invasive strains of Campylobacter jejuni, the most prevalent cause of bacter
190 ember of the cell-shape-determining class of Campylobacter jejuni, the peptidoglycan peptidase 3 (Pgp
191 ugh frequent recombination with it, while in Campylobacter jejuni, we find a minority population we p
192 y and kinetic parameters of PglC, a PGT from Campylobacter jejuni, were quickly established using thi
193 that the periplasmic binding protein CeuE of Campylobacter jejuni, which was previously thought to bi
194 omic DNA extracted from Escherichia coli and Campylobacter jejuni, with the concentration as low as 2
195  involved in biosynthesis of UDP-diNAcBac in Campylobacter jejuni.
196 he phospholipidome of the bacterial pathogen Campylobacter jejuni.
197 unosensor for the detection of food pathogen Campylobacter jejuni.
198 ies in the Gram-negative intestinal pathogen Campylobacter jejuni.
199 ectious neuropathy most frequently caused by Campylobacter jejuni.
200 ure alone detected 80/89 (89.9% sensitivity) Campylobacter jejuni/Campylobacter coli-positive cases.
201 rate per 100 child-months of infections with Campylobacter jejuni/coli and Campylobacter species duri
202 pylobacter species by enzyme immunoassay and Campylobacter jejuni/coli by quantitative PCR in stool s
203 e 6 bacterial enteric pathogens tested, only Campylobacter jejuni/coli detection was significantly re
204 d unimproved sanitation were associated with Campylobacter jejuni/coli infection.
205                The cumulative burden of both Campylobacter jejuni/coli infections and Campylobacter s
206 oth all Campylobacter species infections and Campylobacter jejuni/coli infections on growth and enter
207 ruses, enterotoxigenic Escherichia coli, and Campylobacter jejuni/coli.
208 lear whether that association is specific to Campylobacter jejuni/coli.
209 oncoding RNAs in the major zoonotic pathogen Campylobacter jejuni; however, few have been functionall
210 h as the PglBs from Campylobacter jejuni and Campylobacter lari, catalyze transfer of glycans from me
211 animals serve as a significant reservoir for Campylobacter, limited information is available on antib
212 e intensified along the food chain to reduce Campylobacter load, especially on chicken meat.
213              In summary, the T6SS encoded by Campylobacter megaplasmids mediates lysis of RBCs and li
214          To induce abortion, orally ingested Campylobacter must translocate across the intestinal epi
215 rmation is available on antibiotic-resistant Campylobacter of bovine origin.
216                  The annual median number of Campylobacter outbreaks increased from 28 in 2004-2006 t
217                    Similarly, Prevotella and Campylobacter (P < 0.05) decreased in stratum 3 after tr
218  a means to evaluate the function of pVir in Campylobacter pathobiology.
219 entative members of this superfamily are the Campylobacter PglCs, which initiate N-linked glycoprotei
220 NA demonstrated to play a functional role in Campylobacter physiology to date.
221                            The T6SS genes in Campylobacter plasmids encoded genes and proteins that w
222 al susceptibility testing suggested that the Campylobacter population developed resistance to several
223 consistently colonized with organisms from a Campylobacter population that adapted to the internal en
224 iotic therapy and long-term excretion on the Campylobacter population.
225 and the effect of antimicrobial treatment on Campylobacter populations in this unusual situation of l
226 e was a substantial reduction in predominant Campylobacter populations proposing that SUCRAM suppleme
227 ted positive using additional assays for 0/2 Campylobacter-positive specimens, 0/4 Escherichia coli O
228 ith most children (n = 1606; 84.9%) having a Campylobacter-positive stool sample by 1 year of age.
229   Ninety-three percent (251) of children had Campylobacter present in asymptomatic fecal samples duri
230 icola (94%/74%), Parvimonas micra (86%/62%), Campylobacter rectus (90%/76%), Eubacterium nodatum (64%
231 omitans (Aa), Porphyromonas gingivalis (Pg), Campylobacter rectus (Cr), and Tannerella forsythia (Tf)
232 omonas gingivalis, Tannerella forsythia, and Campylobacter rectus (P </=0.05).
233 sobacterium nucleatum, Parvimonas micra, and Campylobacter rectus were elevated in AgP in a few studi
234 ing association of Prevotella intermedia and Campylobacter rectus with the etiology of peri-implantit
235 terium nucleatum, Prevotella intermedia, and Campylobacter rectus), two red-complex periodontal patho
236 idis, including Aggregatibacter aphrophilus, Campylobacter rectus, Catonella morbi, Haemophilus haemo
237         We found that Prevotella intermedia, Campylobacter Rectus, Factor 2 (Pi/Prevotella nigrescens
238                                Antibodies to Campylobacter rectus, Veillonella parvula, Prevotella me
239 while both Amplon and PAA yielded detectable Campylobacter reductions at all steps.
240    In particular, questions remain about how Campylobacter resistomes interact between species and ho
241 method, we determined that seasonal peaks of Campylobacter, Salmonella, and Shiga toxin-producing Esc
242 were only 19/196 (10%) positive cultures for Campylobacter, Salmonella, or Shigella entero-pathogens
243 ss with the following methods: four types of Campylobacter selective media, four commercial stool ant
244               To better understand how these campylobacters sense nutrient availability, we examined
245                                              Campylobacter sequence types (STs) were not mutually exc
246 lus, Corynebacterium, Cellulosimicrobium and Campylobacter showed lower abundances in OLP patients, a
247 umors followed by Pseudomonas aeruginosa and Campylobacter sp.
248 GI panel were Clostridium difficile (55.0%), Campylobacter species (20.9%), Salmonella species (12.4%
249 imental model for the study of diarrheagenic Campylobacter species and will be useful in exploring th
250                                              Campylobacter species are among the most common causes o
251 D birth cohort study, including detection of Campylobacter species by enzyme immunoassay and Campylob
252 thogen Detection database revealed extensive Campylobacter species clusters carrying multiple drug re
253 nfections with Campylobacter jejuni/coli and Campylobacter species during 1-24 month follow up were 1
254                           Of these, emerging Campylobacter species have been found to be more prevale
255 s to assess the association between both all Campylobacter species infections and Campylobacter jejun
256                                              Campylobacter species infections have been associated wi
257 lphia region were prospectively analyzed for Campylobacter species other than C. jejuni and C. coli u
258 vasion, and intracellular survival, emerging Campylobacter species should be investigated as etiologi
259 oth Campylobacter jejuni/coli infections and Campylobacter species were associated with poor growth a
260 ent isolation of C. concisus The majority of Campylobacter species were not clinically significant.
261  = 0.08), but the relative abundances of two Campylobacter species, along with another 33 gut bacteri
262     Of 225 samples tested, 13 (5.8%) yielded Campylobacter species, with frequent isolation of C. con
263 a to evaluate a PCR assay for distinguishing Campylobacter species.
264  95% CI 3.0-7.1), rotavirus (4.8%, 4.5-5.0), Campylobacter spp (3.5%, 0.4-6.3), astrovirus (2.7%, 2.2
265 th diarrhoea in the second year of life were Campylobacter spp (7.9%, 3.1-12.1), norovirus GII (5.4%,
266 oody diarrhoea was primarily associated with Campylobacter spp and Shigella spp, fever and vomiting w
267 bstrates was fabricated for the detection of Campylobacter spp in food matrices.
268 -acquired infections such as Salmonella spp, Campylobacter spp, N gonorrhoeae, and H pylori.
269 bacter pylori, and fluoroquinolone-resistant Campylobacter spp, Neisseria gonorrhoeae, and Salmonella
270 rus 40/41, ST-ETEC, Cryptosporidium spp, and Campylobacter spp.
271 f the most common poultry meat contaminants: Campylobacter spp.
272 erichia coli (OR: 1.39; 95% CI: 1.05, 1.83), Campylobacter spp. (OR: 1.46; 95% CI: 1.11, 1.91), heat-
273                                              Campylobacter spp. are among the most common causes of e
274                                              Campylobacter spp. are responsible for acute bacterial d
275 ed for the concentrations of PCR amplicon of Campylobacter spp. between 1 and 25 nM with a limit of d
276 thogen Detection database revealed extensive Campylobacter spp. clusters carrying multiple drug resis
277 lification, and electrochemical detection of Campylobacter spp. in raw poultry meat samples.
278                       The high prevalence of Campylobacter spp. in retail liver products was previous
279 , which helps explain the high prevalence of Campylobacter spp. in retail liver products.
280                              Megaplasmids in Campylobacter spp. likely play important roles in antibi
281 s pathogenic strains of Escherichia coli and Campylobacter spp. that declined in prevalence based on
282 llected and tested by enzyme immunoassay for Campylobacter Stool and blood samples were assayed for m
283 port comprehensive performance data for four Campylobacter stool antigen CIDTs versus culture and mol
284 pecificity, and positive predictive value of Campylobacter stool antigen tests were highly variable.
285 l liver and meat juices enhanced survival of Campylobacter strains at low temperatures and increased
286                                   All tested Campylobacter strains showed higher survival in beef liv
287                          The cytotoxicity of Campylobacter strains towards the human embryonic kidney
288                                              Campylobacter STs typically reached a peak in prevalence
289  the method preferable to hqSNP analysis for Campylobacter surveillance and cluster detection.
290                        Here we show that, in Campylobacter, the 3D structure of FlgK differs from tha
291 nserved DNA methyltransferase, which we term Campylobacter transformation system methyltransferase (c
292 ne copies/swab, respectively; P < .001), and Campylobacter ureolyticus (1.7 x 10(5) and 1.6 x 10(7)16
293                                              Campylobacter was identified using a broadly reactive, g
294                                              Campylobacter was prevalent across diverse settings and
295 26 267 nondiarrheal stool samples tested for Campylobacter We describe a high prevalence of infection
296 onine production and chicken colonization by Campylobacter, we constructed two mutants for phenotypic
297                       High concentrations of Campylobacter were found, being the lowest in the lake (
298 ggretative, enteropathogenic), Shigella, and Campylobacter were the most commonly detected bacteria (
299 health risks associated with the exposure to Campylobacter when harvesting urban stormwater for toile
300 sor would help in the sensitive detection of Campylobacter which can result in reducing pre-enrichmen

 
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