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1 eneralist lineages of the zoonotic bacterium Campylobacter.
2 ng that two resistance mechanisms existed in Campylobacter.
3 adequate for protecting against exposure to Campylobacter.
4 ylobacter, indicating their functionality in Campylobacter.
5 involved in generating the helical shape of Campylobacter.
6 t-of-care screening of chicken carcasses for Campylobacter.
7 uding Helicobacter, Chlamydia, Brucella, and Campylobacter.
8 of infection is an estimated 1 in 23500 for Campylobacter, 1 in 5050000 for Cryptosporidium, and 1 i
9 pathogens Shigella (36%), Giardia (33%), and Campylobacter (30%) predominated, but their presence was
11 e results identify a key virulence factor in Campylobacter and a potential target for the control of
12 erience diarrhea show increased abundance of Campylobacter and a reduction in Helicobacter macacae.
13 can be extended to include other loci within Campylobacter and developed for molecular epidemiology s
14 erythromycin, a total of eight strains (four Campylobacter and four Enterococcus) obtained macrolide-
15 acute gastroenteritis and culture-confirmed Campylobacter and nontyphoidal Salmonella infections in
16 n reportable enteric pathogen detected after Campylobacter and the most detected diarrheagenic E. col
18 TM7, and 11 genera, including Anaeroplasma, Campylobacter, and Clostridium, were correlated with app
19 er in Actinomyces, Selenomonas, Veillonella, Campylobacter, and TM7 [G-1] than the Swedish groups.
20 lla, specific multidrug-resistant strains of Campylobacter are circulating by sexual transmission in
21 lla, specific multidrug-resistant strains of Campylobacter are circulating by sexual transmission in
27 d 13 bacterial taxa indicative of cumulative Campylobacter burden and 14 taxa significantly associate
34 p., Shigella spp., Campylobacter jejuni, and Campylobacter coli and an EIA for Shiga toxins 1 and 2.
36 rolide-resistant mutants were induced in one Campylobacter coli and one Enterococcus faecium strain,
38 ry, clinical and environmental C. jejuni and Campylobacter coli contained genetic changes within the
40 f two clonal lineages of multidrug-resistant Campylobacter coli from MSM in Seattle, Washington and M
42 or the detection of Campylobacter jejuni and Campylobacter coli, leading global causes of bacterial g
45 n order to determine the natural patterns of Campylobacter colonization over a period of 63 weeks.
47 s of PglC, a prototypic dual domain PGT from Campylobacter concisus Using a luminescence-based assay,
49 enterotoxigenic Escherichia coli, Shigella, Campylobacter, Cryptosporidium, norovirus GII, and adeno
51 s positively correlated with the presence of Campylobacter, Deinococcus, and Sulfurospirillum Finally
52 rch is required to better define whether the Campylobacter detectable in stormwater are pathogenic to
53 Factors associated with a reduced risk of Campylobacter detection included exclusive breastfeeding
54 ed excellent sensitivity and specificity for Campylobacter detection with low cross reactivity for ot
58 lts suggest that there is natural control of Campylobacter dynamics within a flock which could potent
60 for 9 bacterial enteropathogens: Aeromonas, Campylobacter, Escherichia coli O157, other Shiga toxin-
61 osis cases are acute and self-limiting, with Campylobacter excretion ceasing a few weeks after sympto
63 4 to December 2016, a cluster of 13 cases of Campylobacter fetus intestinal and extraintestinal infec
65 he substantial burden of apparently sporadic Campylobacter from cattle where transmission routes are
66 upled to biotin-Si-NPs successfully detected Campylobacter from naturally contaminated chicken meat,
68 versity in farm-derived samples and revealed Campylobacter genotypes that would not be detected using
69 m, Eikenella corrodens, Eubacterium nodatum, Campylobacter gracilis, Capnocytophaga sputigena, and Ve
70 ture-independent detection tests (CIDTs) for Campylobacter have become an area of intense controversy
71 single-stranded DNA-binding protein (SSB) of Campylobacter hominis Our findings indicate that SSB pos
72 the FlgK junction onto the structure of the Campylobacter hook provides some clues about its diverge
73 uctural constraints, due to the structure of Campylobacter hook, causes divergence of one element of
75 was a retrospective study of MSM-associated Campylobacter in Seattle, Washington and Montreal, Quebe
80 ndings reveal the rising prevalence of FQ(R) Campylobacter in the U.S. and provide novel information
81 the TA systems, pVir was readily cured from Campylobacter, indicating their functionality in Campylo
82 ion of two novel DNA modification systems in Campylobacter-infecting phages, which replace all guanos
83 rade 3]; diarrhoea and fever associated with Campylobacter infection [grade 3]; recurrence of abdomin
84 ntibiotic treatment may reduce the burden of Campylobacter infection and improve growth in children i
85 , we describe the epidemiology and impact of Campylobacter infection in the first 2 years of life.
90 echanisms, we evaluated associations between Campylobacter infection, linear growth, and fecal microb
91 oxacin, an antibiotic of choice for treating Campylobacter infection, through the pore of MOMP reveal
93 dentify risk factors for sporadic intestinal Campylobacter infections and to determine the relative i
94 only 9% of nontyphoidal Salmonella and 4% of Campylobacter infections compared with 59% and 55% among
99 macrolides, in the major foodborne pathogen Campylobacter is considered a serious threat to public h
101 ntibiotic resistance genes have spread among Campylobacter isolated from humans, animals and the envi
104 a patient with fecal specimens positive for Campylobacter jejuni (ST45) intermittently during a 10-y
105 to bacteria such as Pseudomonas aeruginosa, Campylobacter jejuni and Agrobacterium tumefaciens, whic
108 t targets mapA and ceuE for the detection of Campylobacter jejuni and Campylobacter coli, leading glo
110 Monomeric OTases, such as the PglBs from Campylobacter jejuni and Campylobacter lari, catalyze tr
112 inked protein glycosylation (Pgl) pathway of Campylobacter jejuni are evaluated for their tolerance f
114 Here we report the cryo-EM structure of the Campylobacter jejuni cap complex, which reveals that Fli
116 Here, we report the crystal structures of Campylobacter jejuni Cas9 (CjCas9), one of the smallest
121 Here the authors present the structure of Campylobacter jejuni CmeB pump combined with functional
122 udy, the effect of sustainable probiotics on Campylobacter jejuni colonization and gut microbiome com
123 NA (sgRNA)-guided and catalytically impaired Campylobacter jejuni CRISPR-associated protein 9-fused a
125 acterial oligosaccharyltransferase, PglB, of Campylobacter jejuni favors acceptor proteins with conse
127 ublic Health England received 25 isolates of Campylobacter jejuni from an individual with combined va
134 Guillain-Barre syndrome is often caused by Campylobacter jejuni infection that has induced antibodi
156 pecific binding of graphene quantum dot with Campylobacter jejuni membrane leads to generate a distan
158 o-L-gluco-heptopyranose residue found in the Campylobacter jejuni NCTC11168 (HS:2) capsular polysacch
161 ml and ability of this FRET immunosensor for Campylobacter jejuni sensing in comparison with other ba
162 ber 2013, sexual transmission of 2 clades of Campylobacter jejuni subspecies jejuni isolates resulted
164 The microaerophilic food-borne pathogen Campylobacter jejuni uses complex cytochrome-rich respir
166 oxide and graphene quantum dot for detection Campylobacter jejuni whole cell in food samples was desi
167 nd Staphylococcus spp.; a zoonotic pathogen: Campylobacter jejuni) and antimicrobial resistance (AMR)
168 he most common food-born bacterial pathogen (Campylobacter jejuni) in the most prolific agricultural
173 detection of Salmonella spp., Shigella spp., Campylobacter jejuni, and Campylobacter coli and an EIA
174 classes of inhibitors of Bacillus anthracis, Campylobacter jejuni, and Clostridium perfringens IMPDHs
175 Enterovirus, adenovirus A, Salmonella spp., Campylobacter jejuni, bovine polyomavirus, and bovine ro
178 erial species (Bacteroides thetaiotaomicron, Campylobacter jejuni, Enterococcus faecalis, Escherichia
179 d to spray-irrigated dairy manure containing Campylobacter jejuni, enterohemorrhagic Escherichia coli
180 n pathogens, such as Helicobacter pylori and Campylobacter jejuni, have escaped TLR5 activation by mu
182 d the highest antimicrobial activity against Campylobacter jejuni, L. monocytogenes, and Pseudomonas
183 toxigenic Escherichia coli, Vibrio cholerae, Campylobacter jejuni, norovirus) in cohorts from Haiti,
184 s, Vibrio cholerae, Helicobacter pylori, and Campylobacter jejuni, organisms from three classes of Pr
185 The foodborne microaerophilic pathogen, Campylobacter jejuni, possesses a periplasmic formate de
189 previously identified in invasive strains of Campylobacter jejuni, the most prevalent cause of bacter
190 ember of the cell-shape-determining class of Campylobacter jejuni, the peptidoglycan peptidase 3 (Pgp
191 ugh frequent recombination with it, while in Campylobacter jejuni, we find a minority population we p
192 y and kinetic parameters of PglC, a PGT from Campylobacter jejuni, were quickly established using thi
193 that the periplasmic binding protein CeuE of Campylobacter jejuni, which was previously thought to bi
194 omic DNA extracted from Escherichia coli and Campylobacter jejuni, with the concentration as low as 2
200 ure alone detected 80/89 (89.9% sensitivity) Campylobacter jejuni/Campylobacter coli-positive cases.
201 rate per 100 child-months of infections with Campylobacter jejuni/coli and Campylobacter species duri
202 pylobacter species by enzyme immunoassay and Campylobacter jejuni/coli by quantitative PCR in stool s
203 e 6 bacterial enteric pathogens tested, only Campylobacter jejuni/coli detection was significantly re
206 oth all Campylobacter species infections and Campylobacter jejuni/coli infections on growth and enter
209 oncoding RNAs in the major zoonotic pathogen Campylobacter jejuni; however, few have been functionall
210 h as the PglBs from Campylobacter jejuni and Campylobacter lari, catalyze transfer of glycans from me
211 animals serve as a significant reservoir for Campylobacter, limited information is available on antib
219 entative members of this superfamily are the Campylobacter PglCs, which initiate N-linked glycoprotei
222 al susceptibility testing suggested that the Campylobacter population developed resistance to several
223 consistently colonized with organisms from a Campylobacter population that adapted to the internal en
225 and the effect of antimicrobial treatment on Campylobacter populations in this unusual situation of l
226 e was a substantial reduction in predominant Campylobacter populations proposing that SUCRAM suppleme
227 ted positive using additional assays for 0/2 Campylobacter-positive specimens, 0/4 Escherichia coli O
228 ith most children (n = 1606; 84.9%) having a Campylobacter-positive stool sample by 1 year of age.
229 Ninety-three percent (251) of children had Campylobacter present in asymptomatic fecal samples duri
230 icola (94%/74%), Parvimonas micra (86%/62%), Campylobacter rectus (90%/76%), Eubacterium nodatum (64%
231 omitans (Aa), Porphyromonas gingivalis (Pg), Campylobacter rectus (Cr), and Tannerella forsythia (Tf)
233 sobacterium nucleatum, Parvimonas micra, and Campylobacter rectus were elevated in AgP in a few studi
234 ing association of Prevotella intermedia and Campylobacter rectus with the etiology of peri-implantit
235 terium nucleatum, Prevotella intermedia, and Campylobacter rectus), two red-complex periodontal patho
236 idis, including Aggregatibacter aphrophilus, Campylobacter rectus, Catonella morbi, Haemophilus haemo
240 In particular, questions remain about how Campylobacter resistomes interact between species and ho
241 method, we determined that seasonal peaks of Campylobacter, Salmonella, and Shiga toxin-producing Esc
242 were only 19/196 (10%) positive cultures for Campylobacter, Salmonella, or Shigella entero-pathogens
243 ss with the following methods: four types of Campylobacter selective media, four commercial stool ant
246 lus, Corynebacterium, Cellulosimicrobium and Campylobacter showed lower abundances in OLP patients, a
248 GI panel were Clostridium difficile (55.0%), Campylobacter species (20.9%), Salmonella species (12.4%
249 imental model for the study of diarrheagenic Campylobacter species and will be useful in exploring th
251 D birth cohort study, including detection of Campylobacter species by enzyme immunoassay and Campylob
252 thogen Detection database revealed extensive Campylobacter species clusters carrying multiple drug re
253 nfections with Campylobacter jejuni/coli and Campylobacter species during 1-24 month follow up were 1
255 s to assess the association between both all Campylobacter species infections and Campylobacter jejun
257 lphia region were prospectively analyzed for Campylobacter species other than C. jejuni and C. coli u
258 vasion, and intracellular survival, emerging Campylobacter species should be investigated as etiologi
259 oth Campylobacter jejuni/coli infections and Campylobacter species were associated with poor growth a
260 ent isolation of C. concisus The majority of Campylobacter species were not clinically significant.
261 = 0.08), but the relative abundances of two Campylobacter species, along with another 33 gut bacteri
262 Of 225 samples tested, 13 (5.8%) yielded Campylobacter species, with frequent isolation of C. con
264 95% CI 3.0-7.1), rotavirus (4.8%, 4.5-5.0), Campylobacter spp (3.5%, 0.4-6.3), astrovirus (2.7%, 2.2
265 th diarrhoea in the second year of life were Campylobacter spp (7.9%, 3.1-12.1), norovirus GII (5.4%,
266 oody diarrhoea was primarily associated with Campylobacter spp and Shigella spp, fever and vomiting w
269 bacter pylori, and fluoroquinolone-resistant Campylobacter spp, Neisseria gonorrhoeae, and Salmonella
272 erichia coli (OR: 1.39; 95% CI: 1.05, 1.83), Campylobacter spp. (OR: 1.46; 95% CI: 1.11, 1.91), heat-
275 ed for the concentrations of PCR amplicon of Campylobacter spp. between 1 and 25 nM with a limit of d
276 thogen Detection database revealed extensive Campylobacter spp. clusters carrying multiple drug resis
281 s pathogenic strains of Escherichia coli and Campylobacter spp. that declined in prevalence based on
282 llected and tested by enzyme immunoassay for Campylobacter Stool and blood samples were assayed for m
283 port comprehensive performance data for four Campylobacter stool antigen CIDTs versus culture and mol
284 pecificity, and positive predictive value of Campylobacter stool antigen tests were highly variable.
285 l liver and meat juices enhanced survival of Campylobacter strains at low temperatures and increased
291 nserved DNA methyltransferase, which we term Campylobacter transformation system methyltransferase (c
292 ne copies/swab, respectively; P < .001), and Campylobacter ureolyticus (1.7 x 10(5) and 1.6 x 10(7)16
295 26 267 nondiarrheal stool samples tested for Campylobacter We describe a high prevalence of infection
296 onine production and chicken colonization by Campylobacter, we constructed two mutants for phenotypic
298 ggretative, enteropathogenic), Shigella, and Campylobacter were the most commonly detected bacteria (
299 health risks associated with the exposure to Campylobacter when harvesting urban stormwater for toile
300 sor would help in the sensitive detection of Campylobacter which can result in reducing pre-enrichmen