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1 virulence of the human gut mucosal pathogen Campylobacter jejuni .
2 ed sites (eg, Aeromonas, Vibrio cholerae O1, Campylobacter jejuni).
3 with the periplasmic binding protein CeuE of Campylobacter jejuni.
4 91% for C. difficile A/B toxins; and 90% for Campylobacter jejuni.
5 the human pathogens Helicobacter pylori and Campylobacter jejuni.
6 of the major human gastrointestinal pathogen Campylobacter jejuni.
7 ies in the Gram-negative intestinal pathogen Campylobacter jejuni.
8 a part of a controlled human infection with Campylobacter jejuni.
9 f the metabolic capacity and epidemiology of Campylobacter jejuni.
10 the asaccharolytic food-borne human pathogen Campylobacter jejuni.
11 variable genomic regions between strains of Campylobacter jejuni.
12 on of sigma(54)-dependent flagellar genes in Campylobacter jejuni.
13 ssion of the multidrug efflux pump CmeABC in Campylobacter jejuni.
14 ed intragastrically with increasing doses of Campylobacter jejuni.
15 involved in biosynthesis of UDP-diNAcBac in Campylobacter jejuni.
16 he phospholipidome of the bacterial pathogen Campylobacter jejuni.
17 unosensor for the detection of food pathogen Campylobacter jejuni.
18 ectious neuropathy most frequently caused by Campylobacter jejuni.
20 We report isolation and characterization of Campylobacter jejuni 81-176 lgtF and galT lipooligosacch
24 mine whether H. pylori can transfer DNA into Campylobacter jejuni, a closely related species of the C
32 (NrfA) reductases have been investigated in Campylobacter jejuni, a microaerophilic food-borne patho
34 ereas previous endeavors have focused on the Campylobacter jejuni acetyltransferase (PglD) from the N
35 specific inhibitor of PFOR of H. pylori and Campylobacter jejuni, also inhibited NADP reduction in c
36 to bacteria such as Pseudomonas aeruginosa, Campylobacter jejuni and Agrobacterium tumefaciens, whic
37 s used to detect the presence of cmeC in 131 Campylobacter jejuni and C. coli strains isolated from v
39 rized two FeEnt receptors (CfrA and CfrB) in Campylobacter jejuni and C. coli, the enteric human path
43 ovide routine definitive characterization of Campylobacter jejuni and Campylobacter coli for clinical
44 true but also, in fact, the clearly distinct Campylobacter jejuni and Campylobacter coli species may
45 closely related zoonotic pathogenic species, Campylobacter jejuni and Campylobacter coli, are converg
46 athogens associated with diarrhoea-including Campylobacter jejuni and Campylobacter coli, Cryptospori
48 t targets mapA and ceuE for the detection of Campylobacter jejuni and Campylobacter coli, leading glo
49 hough cdtB gene sequences were found in both Campylobacter jejuni and Campylobacter coli, the product
51 Monomeric OTases, such as the PglBs from Campylobacter jejuni and Campylobacter lari, catalyze tr
57 genes with higher sequence diversity in the Campylobacter jejuni and Neisseria meningitidis genomes
59 says demonstrate that LpxJ and homologues in Campylobacter jejuni and Wolinella succinogenes can act
60 erborne illness caused almost exclusively by Campylobacter jejuni and, to a lesser extent, by Campylo
61 nd Staphylococcus spp.; a zoonotic pathogen: Campylobacter jejuni) and antimicrobial resistance (AMR)
62 detection of Salmonella spp., Shigella spp., Campylobacter jejuni, and Campylobacter coli and an EIA
64 classes of inhibitors of Bacillus anthracis, Campylobacter jejuni, and Clostridium perfringens IMPDHs
65 ichia coli (ETEC), enteropathogenic E. coli, Campylobacter jejuni, and Giardia lamblia document heter
66 ne contained contaminating moieties (such as Campylobacter jejuni antigens that mimic human gangliosi
68 inked protein glycosylation (Pgl) pathway of Campylobacter jejuni are evaluated for their tolerance f
71 arahemeolyticus, Pseudomonas aeruginosa, and Campylobacter jejuni, as well as an additional 12 bacter
73 icated a moderate probability of illness for Campylobacter jejuni at the study beaches, especially wh
74 Enterovirus, adenovirus A, Salmonella spp., Campylobacter jejuni, bovine polyomavirus, and bovine ro
75 nsible for high-affinity iron acquisition in Campylobacter jejuni but also is essential for C. jejuni
76 nic Escherichia coli, Shigella flexneri, and Campylobacter jejuni, but not Neisseria gonorrhoeae, cle
77 IA (ProSpecT), and duplex PCR to distinguish Campylobacter jejuni/C. coli and non-jejuni/coli Campylo
79 us GII, rotavirus, and sapovirus), bacteria (Campylobacter jejuni/C. coli, Clostridium difficile, Sal
80 l specimens, including pathogenic Aeromonas, Campylobacter jejuni, Campylobacter coli, Salmonella, Sh
81 ase 3, sequences from Clostridium difficile, Campylobacter jejuni, Campylobacter concisus, and Salmon
82 ure alone detected 80/89 (89.9% sensitivity) Campylobacter jejuni/Campylobacter coli-positive cases.
85 Here we report the cryo-EM structure of the Campylobacter jejuni cap complex, which reveals that Fli
88 Here, we report the crystal structures of Campylobacter jejuni Cas9 (CjCas9), one of the smallest
94 ed with those of three bacterial IMPDHs from Campylobacter jejuni, Clostridium perfringens, and Vibri
96 Here the authors present the structure of Campylobacter jejuni CmeB pump combined with functional
97 oli (STEC), Shigella spp. , Salmonella spp , Campylobacter jejuni/coli , and methicillin-resistant St
98 rate per 100 child-months of infections with Campylobacter jejuni/coli and Campylobacter species duri
99 pylobacter species by enzyme immunoassay and Campylobacter jejuni/coli by quantitative PCR in stool s
100 e 6 bacterial enteric pathogens tested, only Campylobacter jejuni/coli detection was significantly re
103 oth all Campylobacter species infections and Campylobacter jejuni/coli infections on growth and enter
106 udy, the effect of sustainable probiotics on Campylobacter jejuni colonization and gut microbiome com
108 of antimicrobial agents and is essential for Campylobacter jejuni colonization of the animal intestin
112 claimed to be responsible for inhibition of Campylobacter jejuni could not be detected either direct
113 NA (sgRNA)-guided and catalytically impaired Campylobacter jejuni CRISPR-associated protein 9-fused a
114 actinomycetemcomitans, Escherichia coli, and Campylobacter jejuni differ in their abilities to intoxi
118 ubform acute motor axonal neuropathy (AMAN), Campylobacter jejuni enteritis triggers the production o
119 erial species (Bacteroides thetaiotaomicron, Campylobacter jejuni, Enterococcus faecalis, Escherichia
120 d to spray-irrigated dairy manure containing Campylobacter jejuni, enterohemorrhagic Escherichia coli
121 ty against Gram-negative bacteria, including Campylobacter jejuni, Escherichia coli O157:H7, and mult
123 of Sn in Mphi interactions with heat-killed Campylobacter jejuni expressing a GD1a-like, sialylated
124 acterial oligosaccharyltransferase, PglB, of Campylobacter jejuni favors acceptor proteins with conse
126 unogenicity and protective efficacy of three Campylobacter jejuni flagellum-secreted proteins, FlaC,
129 ublic Health England received 25 isolates of Campylobacter jejuni from an individual with combined va
130 radish peroxidase (HRP), myoglobin (Mb), and Campylobacter jejuni globin (Cgb) are compared and signi
132 d export; examples include Escherichia coli, Campylobacter jejuni, Haemophilus influenzae, Neisseria
141 s, we show that the flagellar filaments from Campylobacter jejuni have seven protofilaments rather th
142 n pathogens, such as Helicobacter pylori and Campylobacter jejuni, have escaped TLR5 activation by mu
144 and Salmonella typhimurium, the flagella of Campylobacter jejuni, Helicobacter pylori and Vibrio cho
145 in (Ig) G and IgA to Haemophilus influenzae, Campylobacter jejuni, Helicobacter pylori, Streptococcus
146 oncoding RNAs in the major zoonotic pathogen Campylobacter jejuni; however, few have been functionall
150 osphorylase (PNPase) facilitates survival of Campylobacter jejuni in low temperatures and favors swim
151 ude an increased appreciation of the role of Campylobacter jejuni in postinfectious sequelae, a broad
153 he most common food-born bacterial pathogen (Campylobacter jejuni) in the most prolific agricultural
154 he simplest system is that of the bacterium, Campylobacter jejuni, in which a heptasaccharide glycan
155 ertoire of glycoconjugates that are found in Campylobacter jejuni includes lipooligosaccharides mimic
156 zation domain-containing protein 2 (NOD2) in Campylobacter jejuni-induced intestinal inflammation.
157 the contribution of PI3K-gamma signaling in Campylobacter jejuni-induced neutrophil accumulation and
161 Guillain-Barre syndrome is often caused by Campylobacter jejuni infection that has induced antibodi
163 1221 identified four large genomic elements, Campylobacter jejuni-integrated elements (CJIEs), that w
206 acter-associated abortion in sheep; however, Campylobacter jejuni is increasingly associated with she
212 c acid cycle in the microaerophilic pathogen Campylobacter jejuni is potentially vulnerable, as it em
227 receptor CfrA is present in the majority of Campylobacter jejuni isolates and is responsible for hig
233 d the highest antimicrobial activity against Campylobacter jejuni, L. monocytogenes, and Pseudomonas
234 O157), shiga-toxin producing E. coli (stx2), Campylobacter jejuni (mapA), Shigella spp. (ipaH), and a
236 B1a protein of the gastrointestinal pathogen Campylobacter jejuni mediates interactions with epitheli
238 pecific binding of graphene quantum dot with Campylobacter jejuni membrane leads to generate a distan
240 oss of the gne gene in the expression of the Campylobacter jejuni N-glycosylation system in E. coli i
242 o-L-gluco-heptopyranose residue found in the Campylobacter jejuni NCTC11168 (HS:2) capsular polysacch
244 toxigenic Escherichia coli, Vibrio cholerae, Campylobacter jejuni, norovirus) in cohorts from Haiti,
246 s, Vibrio cholerae, Helicobacter pylori, and Campylobacter jejuni, organisms from three classes of Pr
249 ein (MOMP) typing as a screen to compare the Campylobacter jejuni porA gene sequences of clinical out
252 The foodborne microaerophilic pathogen, Campylobacter jejuni, possesses a periplasmic formate de
253 OTase, and PglB(Cj), the N-linked OTase from Campylobacter jejuni, preferred the native N. gonorrhoea
254 mmensalism in animals and disease in humans, Campylobacter jejuni produces a flagellar organelle for
255 75-year-old man was diagnosed with probable Campylobacter jejuni prosthetic knee infection after a d
258 ssion essential for formation of flagella in Campylobacter jejuni requires the components of the inne
259 eropathogenic E. coli (EPEC), Shigella spp., Campylobacter jejuni, Salmonella enterica, and Aeromonas
260 rrhea-causing pathogens, including bacteria (Campylobacter jejuni, Salmonella spp., Shigella spp., en
261 ml and ability of this FRET immunosensor for Campylobacter jejuni sensing in comparison with other ba
262 y of a recombinant truncated multifunctional Campylobacter jejuni sialyltransferase CstII mutant, Cst
263 a patient with fecal specimens positive for Campylobacter jejuni (ST45) intermittently during a 10-y
264 Initial steps in flagellar biosynthesis in Campylobacter jejuni stimulate phosphotransfer from the
265 he complete flagellin glycosylation locus of Campylobacter jejuni strain 81-176 revealed a less compl
268 Analysis of the complete genomic sequence of Campylobacter jejuni strain RM1221 identified four large
271 genetically diverse Campylobacter fetus and Campylobacter jejuni strains have been implicated in suc
272 breoganii 1C-10, Bacillus cereus ATCC 10987, Campylobacter jejuni subsp. jejuni 81-176 and C. jejuni
273 n (strain VPI-5482) [PDB:3KZT], Cj0202c from Campylobacter jejuni subsp. jejuni serotype O:2 (strain
274 ber 2013, sexual transmission of 2 clades of Campylobacter jejuni subspecies jejuni isolates resulted
275 ae, Treponema pallidum, Helicobacter pylori, Campylobacter jejuni, Synechocystis sp., and Mycobacteri
276 859c, or FspA, is a small, acidic protein of Campylobacter jejuni that is expressed by a sigma(28) pr
277 llar motility is an important determinant of Campylobacter jejuni that is required for promoting inte
282 previously identified in invasive strains of Campylobacter jejuni, the most prevalent cause of bacter
283 ember of the cell-shape-determining class of Campylobacter jejuni, the peptidoglycan peptidase 3 (Pgp
284 transfer of an N-glycosylation pathway from Campylobacter jejuni to Escherichia coli in 2002 can be
285 owth of the microaerophilic mucosal pathogen Campylobacter jejuni under oxygen-limited conditions was
286 The microaerophilic food-borne pathogen Campylobacter jejuni uses complex cytochrome-rich respir
287 me loci as a previously described system for Campylobacter jejuni was developed for Campylobacter col
292 ugh frequent recombination with it, while in Campylobacter jejuni, we find a minority population we p
293 The FlhF proteins of Bacillus subtilis and Campylobacter jejuni were recently shown to have GTPase
294 y and kinetic parameters of PglC, a PGT from Campylobacter jejuni, were quickly established using thi
295 method for determination of capsule types of Campylobacter jejuni which is simpler and more affordabl
296 the surface of the food-borne human pathogen Campylobacter jejuni, which has a major role in adherenc
297 that the periplasmic binding protein CeuE of Campylobacter jejuni, which was previously thought to bi
298 oxide and graphene quantum dot for detection Campylobacter jejuni whole cell in food samples was desi
299 omic DNA extracted from Escherichia coli and Campylobacter jejuni, with the concentration as low as 2