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1 d with histologically confirmed, symptomatic Castleman disease.
2 red by patients with idiopathic multicentric Castleman disease.
3 between PEL and KSHV-associated multicentric Castleman disease.
4 (IL)-6 related KSHV-associated multicentric Castleman disease.
5 osi sarcoma, body cavity-based lymphoma, and Castleman disease.
6 primary effusion lymphoma, and multicentric Castleman disease.
7 ary effusion lymphoma (PEL) and multicentric Castleman disease.
8 TbetaRII in Kaposi sarcoma and multicentric Castleman disease.
9 primary effusion lymphoma, and some cases of Castleman disease.
10 posi sarcoma, primary effusion lymphoma, and Castleman disease.
11 ry effusion lymphoma (PEL), and multicentric Castleman disease.
12 ry effusion lymphoma (PEL), and multicentric Castleman disease.
13 entation as having localized or multicentric Castleman disease.
14 s primary effusion lymphoma and multicentric Castleman disease.
17 herapies successful in the treatment of both Castleman disease and associated autoimmune disease.
18 hieving long-term remission in patients with Castleman disease and associated autoimmune features.
19 This review focuses on the intersection of Castleman disease and autoimmunity with an emphasis on s
22 ase (2 with visceral KS, 1 with multicentric Castleman disease, and 1 with primary effusion lymphoma)
25 er primary effusion lymphoma or multicentric Castleman disease B-cell malignancies, and from polyclon
32 etermine the cellular and molecular basis of Castleman Disease (CD), we analyze the spatial proteome
33 recent progress has been coordinated by the Castleman Disease Collaborative Network (CDCN), and furt
35 ry effusion lymphoma (PEL), and multicentric Castleman disease, encodes four interferon regulatory fa
36 ganomegaly, potentially including coexisting Castleman disease, endocrinopathy, monoclonal plasma cel
38 ng of pathogenic cell types and cytokines in Castleman disease have allowed the development of target
41 ces in understanding the biological basis of Castleman disease have provided new targets for therapeu
43 ful treatment of HIV-associated multicentric Castleman disease (HIV+MCD) with rituximab-based approac
51 clinical subtype of idiopathic multicentric Castleman disease (iMCD), consists of a constellation of
53 tent lymphadenopathy resembling multicentric Castleman disease in rhesus macaques (RMs) coinfected wi
54 HIV-associated plasmablastic multicentric Castleman disease is an increasingly frequent diagnosis.
57 oma (KS) herpesvirus-associated multicentric Castleman disease (KSHV-MCD) is a lymphoproliferative di
58 a herpesvirus (KSHV)-associated multicentric Castleman disease (KSHV-MCD) is characterized by severe
63 enesis, both involved in the pathogenesis of Castleman disease, may provide additional unique therape
64 reported as a heralding sign of multicentric Castleman disease (MCD) and other lymphoproliferative di
65 erpesvirus 8 is associated with multicentric Castleman disease (MCD) and primary effusion lymphoma (P
66 ive agent of two B cell tumors, multicentric Castleman disease (MCD) and primary effusion lymphoma (P
67 recognition of KSHV-associated multicentric Castleman disease (MCD) as a distinct lymphoproliferativ
68 a herpesvirus (KSHV)-associated multicentric Castleman disease (MCD) is a lymphoproliferative disorde
70 a herpesvirus (KSHV)-associated multicentric Castleman disease (MCD) is a lymphoproliferative inflamm
71 a herpesvirus (KSHV)-associated multicentric Castleman disease (MCD) is a polyclonal B-cell lymphopro
74 gent of Kaposi sarcoma (KS) and multicentric Castleman disease (MCD), a life-threatening, virally ind
75 effusion lymphoma (PEL) and/or multicentric Castleman disease (MCD), and in 18 (60.0%) of 30 HIV(+),
76 lymphoma (PEL), KSHV-associated multicentric Castleman disease (MCD), and KS inflammatory cytokine sy
77 rimary effusion lymphoma (PEL), multicentric Castleman disease (MCD), and MCD-associated plasmablasti
78 vestigated in patients with KS, multicentric Castleman disease (MCD), and primary effusion lymphoma.
79 itary (EC) PEL, KSHV-associated multicentric Castleman disease (MCD), KSHV+ diffuse large B-cell lymp
92 t-line treatment for idiopathic multicentric Castleman disease on the basis of durable efficacy and s
93 rus (HIV)-infected men with KS, multicentric Castleman disease, or primary effusion lymphoma and 8 HI
94 ional criteria were included: a bone lesion, Castleman disease, organomegaly (or lymphadenopathy), en
95 tuximab in posttrans-plantation multicentric Castleman disease patients and non-neoplastic HHV8-assoc
96 nd at least 1 of the other 3 major criteria (Castleman disease, sclerotic bone lesions, or elevated V
99 on lymphoma and KSHV-associated multicentric Castleman disease, which are B-cell lymphoproliferative
101 aposi sarcoma, HHV-8-associated multicentric Castleman disease with microlymphomas and a severe hemop