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1 we evaluated XRCC1-deficient and -proficient Chinese hamster and human cancer cells for synthetic let
2 ive replication of several OPXV species in a Chinese hamster cell line was caused by a species-specif
3 ray repair complementing defective repair in Chinese hamster cells 3 (XRCC3), 4 CpGs) birth weight.
6 g in potent inhibition against FR-expressing Chinese hamster cells and human KB tumor cells in cultur
7 in binding in G1-synchronized populations of Chinese hamster cells harboring amplified copies of the
8 (CHO-S and CHO DG44) and compared with seven Chinese hamster (Cricetulus griseus) tissues (brain, hea
9 t a 2.4-Gb draft genome sequence of a female Chinese hamster, Cricetulus griseus, harboring 24,044 ge
11 ompetitive binding assay was performed using Chinese hamster lung (CHL) cells transfected with GLP-1R
12 ompetitive binding assay was performed using Chinese hamster lung (CHL) cells transfected with the GL
13 type 2, early onset- (BRCA2-) deficient V79 Chinese hamster lung fibroblast cell line derivative (VC
18 ized LDL and acLDL to CR1 on CR1-transfected Chinese Hamster Ovarian cells (CHO-CR1) was tested by fl
20 has been demonstrated from both E. coli and Chinese hamster ovaries (CHO) cell expression platforms;
22 for inhibition of proliferation in isogenic Chinese hamster ovary (CHO) and HeLa cells expressing PC
23 bit the import of spermidine in DFMO-treated Chinese hamster ovary (CHO) and L3.6pl human pancreatic
25 city of the nine NOCs was quantified using a Chinese hamster ovary (CHO) cell assay, and the descendi
26 re, we present an effort to create a "clean" Chinese hamster ovary (CHO) cell by disrupting multiple
30 e human amyloid precursor protein (APP) in a Chinese hamster ovary (CHO) cell line by cleaving APP at
31 s/mL) perfusion culture of an IgG1-producing Chinese hamster ovary (CHO) cell line for 18-25 days.
33 d-type and glycosaminoglycan (GAG)-deficient Chinese hamster ovary (CHO) cell lines and soluble GAGs,
35 al binding and transduction assays on mutant Chinese hamster ovary (CHO) cell lines defective in vari
36 vity were evaluated as expressed in a set of Chinese hamster ovary (CHO) cell lines under conditions
37 , we engineered and characterized a panel of Chinese hamster ovary (CHO) cell lines with inducible tr
38 ifficult in the case of genetically unstable Chinese hamster ovary (CHO) cell lines with only draft g
41 he heavy chain and the light chain (LC) of a Chinese hamster ovary (CHO) cell-expressed monoclonal an
44 om Schistosoma mansoni (Platyhelminthes), in Chinese hamster ovary (CHO) cells and use fluorescence-b
45 etection platform to image endogenous H2S in Chinese hamster ovary (CHO) cells and use the developed
46 fractions, here we show that, when parental Chinese hamster ovary (CHO) cells are briefly exposed to
47 e a broad host range in mammalian cells, but Chinese hamster ovary (CHO) cells are nonpermissive for
51 ckdown with small interfering RNA (siRNA) in Chinese hamster ovary (CHO) cells augments M3-MR signali
52 o generate any current when transfected into Chinese hamster ovary (CHO) cells but, surprisingly, exe
53 The McKbac virus entered efficiently into Chinese hamster ovary (CHO) cells constitutively express
55 sibility of a nuclear-translocation assay in Chinese hamster ovary (CHO) cells expressing an NFkappaB
56 the binding of 5,6-EET-EA to membranes from Chinese hamster ovary (CHO) cells expressing either reco
57 shift in neonatal mouse cardiac myocytes or Chinese hamster ovary (CHO) cells expressing the mouse o
58 o demonstrate that somatostatin treatment of Chinese hamster ovary (CHO) cells expressing the wild ty
60 ion of PS on the plasma membrane of isolated Chinese Hamster Ovary (CHO) cells following exposure to
61 hich KCNQ subunits are targeted by AKAP79 in Chinese hamster ovary (CHO) cells heterologously express
62 activation of human DORs stably expressed in Chinese hamster ovary (CHO) cells increased AMPK activit
63 this study, we evaluated the genotoxicity to Chinese hamster ovary (CHO) cells induced by municipal s
64 how that overexpression of TREK-1 in NPE and Chinese hamster ovary (CHO) cells leads to a significant
66 e of exporting the diamine putrescine in the Chinese hamster ovary (CHO) cells selected for resistanc
68 n embryonic kidney (HEK) 293, C6 glioma, and Chinese hamster ovary (CHO) cells stably expressing this
70 ition, compounds 3-6 inhibited the growth of Chinese hamster ovary (CHO) cells that expressed FRs but
71 beta-GlcNAc on the complex N-glycans of Lec8 Chinese hamster ovary (CHO) cells that lack UDP-Gal tran
72 ronic cytotoxicity and acute genotoxicity in Chinese hamster ovary (CHO) cells to compare the toxicit
74 an amphotericin B loss-of-function screen in Chinese hamster ovary (CHO) cells using insertional muta
77 LDL increased cytosolic G protein by 350% in Chinese hamster ovary (CHO) cells with genetically induc
78 accomplish glycomic survey of bioengineered Chinese Hamster Ovary (CHO) cells with knock-in/out enzy
80 er cells were created by stably transfecting Chinese Hamster Ovary (CHO) cells with plasmids encoding
83 and duration of reporter gene expression in Chinese Hamster Ovary (CHO) cells, Human Immortalized My
84 ter) obtained from red blood cells (RBC), or Chinese hamster ovary (CHO) cells, were immobilized on t
85 re assayed as luciferase reporter fusions in Chinese hamster ovary (CHO) cells, where the putative ci
86 re caused by EGFR expression, we transfected Chinese hamster ovary (CHO) cells, which lack EGFR expre
87 Here we describe a procedure to vesiculate Chinese hamster ovary (CHO) cells, widely used for the e
110 89%) of mouse-human heterohybridoma (HH) and Chinese hamster ovary (CHO) mAb-Ds blocked ADCC and clea
112 These analogues inhibited proliferation of Chinese hamster ovary (CHO) sublines expressing folate r
113 alysis was performed on mouse myeloma SP2/0, Chinese hamster ovary (CHO), and human embryonic kidney
114 the first N-glycosylation gene, DPAGT1, from Chinese hamster ovary (CHO), Madin-Darby canine kidney (
118 RNA-based pathway inhibition in recombinant Chinese hamster ovary (CHO)-S1P2 cells as well as human
122 AIRS enzyme by analysis of mutations in the Chinese hamster ovary (CHO-K1) cell that require purines
124 study, we found that knockdown of cofilin in Chinese hamster ovary 7WD10 cells and primary neurons si
125 s (c13C6, h-13F6, and c6D8) were produced in Chinese hamster ovary and in whole plant (Nicotiana bent
126 CR3 affects the generation of cAMP, we used Chinese hamster ovary and K562 cells transfected to expr
128 in relation to endogenous Notch receptors of Chinese hamster ovary and murine embryonic stem (ES) cel
129 at were and were not associated with CSBS in Chinese Hamster Ovary and T84 cells and generated a zebr
130 was assayed in vitro by the transduction of Chinese hamster ovary and trabecular meshwork cells.
132 on the stabilities of Escherichia coli- and Chinese hamster ovary cell (CHO)-derived IgG1 Fc high-or
134 1 antibody, generated during production in a Chinese hamster ovary cell culture, was observed in the
137 in a human retinal cell line (ARPE-19) and a Chinese hamster ovary cell line (CHO-K1) to study the fu
138 ath, we generated and characterized a mutant Chinese hamster ovary cell line that is resistant to pal
140 his mutated form of XPF in the XPF-deficient Chinese hamster ovary cell line, UV41, only partially re
141 at transduction occurs efficiently in mutant Chinese hamster ovary cell lines deficient in glycosamin
142 promoter trap mutagenesis to generate mutant Chinese hamster ovary cell lines resistant to lipotoxic
145 p91(phox) and p22(phox), we demonstrate in a Chinese hamster ovary cell model system and in RAW 264.7
150 ished potencies based on the comet assay for Chinese hamster ovary cells (assesses the level of DNA s
151 liquids (ILs) on zebrafish (Danio rerio) and Chinese hamster ovary cells (CHO) was investigated with
153 of the three homologs that are expressed in Chinese hamster ovary cells (DPY19L1, DPY19L3, and DPY19
154 fragments [Fab, F(ab')(2)] were expressed in Chinese hamster ovary cells and evaluated in vitro and i
155 acromolecular channel complex in transfected Chinese hamster ovary cells and found a dominant-negativ
156 position tool, and we tested the adhesion of Chinese Hamster Ovary cells and Human Embrionic Kidney c
158 P9 significantly reduced Abeta generation in Chinese hamster ovary cells and in primary neurons, demo
160 of extracellular signal-regulated kinase in Chinese hamster ovary cells and permits chemokine and pr
161 resistant to c-Cbl-mediated degradation, in Chinese hamster ovary cells and the UMSCC11B human head
164 clamp recordings from channels expressed in Chinese Hamster Ovary Cells at different temperatures (3
165 iggered gating of BKCa channels expressed in Chinese hamster ovary cells at distinct membrane potenti
168 oprecipitated with the I(Ks) channel only in Chinese hamster ovary cells co-expressing AKAP-9, and PD
169 When ADAP was heterologously expressed in Chinese hamster ovary cells co-expressing alphaIIbbeta3,
170 assays that use human receptors expressed in Chinese hamster ovary cells demonstrate that NDD-713 and
171 nant human beta-gal (rhbeta-gal) produced in Chinese hamster ovary cells enabled direct and precise r
172 nant human Beta-Gal (rhBeta-Gal) produced in Chinese hamster ovary cells enabled direct and precise r
173 lite trends from a bioreactor cultivation of Chinese hamster ovary cells expressing a recombinant ant
176 ning this exon promote serotonin uptake into Chinese hamster ovary cells expressing either vesicular
177 was evaluated before and after adsorption to Chinese hamster ovary cells expressing human TSHRs using
181 a synthetic naive human scFv library against Chinese hamster ovary cells expressing the oncogenic tar
182 r Abeta binding and uptake were confirmed in Chinese hamster ovary cells genetically deficient in HSP
183 ibodies as well as significantly enhanced in Chinese hamster ovary cells genetically modified to expr
184 well as in human embryonic kidney cells and Chinese hamster ovary cells heterologously expressing hu
185 eta3 enhanced IGF-1-induced proliferation of Chinese hamster ovary cells in serum-free conditions (in
186 t transient expression of kindlin-1 or -2 in Chinese hamster ovary cells inhibits the activation of e
187 etabolism, using RNA interference to deplete Chinese hamster ovary cells of NPC1 alone or in combinat
188 -linked fluorophore to these compartments in Chinese hamster ovary cells or Jurkat lymphocytes, membr
189 n wild-type and D1275N channels expressed in Chinese hamster ovary cells or tsA201 cells in the absen
191 emperature dependence of hERG kinetics using Chinese hamster ovary cells overexpressing hERG1a on the
192 ents as well as single-channel recordings on Chinese hamster ovary cells overexpressing I(Ks) channel
193 ses formed between primary T lymphocytes and Chinese hamster ovary cells presenting major histocompat
194 TGF-mediated control of beta-AR sensitivity, Chinese hamster ovary cells pretreated with rec-hCTGF di
197 b/IIIa (integrin alphaIIbbeta3) expressed on Chinese hamster ovary cells promoted melanoma cell adhes
198 ene promoter; overexpression of HNF4alpha in Chinese hamster ovary cells re-established transcription
199 mpetition assays at membrane preparations of Chinese hamster ovary cells recombinantly expressing the
200 n, and competition assays using membranes of Chinese hamster ovary cells recombinantly expressing the
201 ivated Teffs with Sn(+) macrophages or Sn(+) Chinese hamster ovary cells resulted in increased cell d
202 at the human beta1-adrenoceptor expressed in Chinese hamster ovary cells revealed negative cooperativ
204 Galpha(q) and Galpha(i) pathways in vitro on Chinese hamster ovary cells stably expressing FFA2.
205 substrate for PKC, and this was confirmed in Chinese hamster ovary cells stably expressing full-lengt
206 ated patch-clamp platform, by applying it to Chinese hamster ovary cells stably expressing hERG1a.
207 or binding of fluorescently labeled Abeta to Chinese hamster ovary cells stably expressing human CD36
208 stimulated cAMP accumulation was measured in Chinese hamster ovary cells stably expressing the human
209 agonist 5'-(N-ethylcarboxamido)adenosine in Chinese hamster ovary cells stably transfected with the
210 a membrane of intact rabbit erythrocytes and Chinese hamster ovary cells that can be explained by the
211 ection technique to generate a population of Chinese hamster ovary cells that display a global defici
212 t a comprehensive gene engineering screen in Chinese hamster ovary cells that enables production of l
213 horage-independent conditions in transformed Chinese hamster ovary cells that express alphavbeta3 (be
215 ) and selective inhibitory activities toward Chinese hamster ovary cells that expressed FRalpha or FR
216 ulated release of arachidonic acid (AA) from Chinese hamster ovary cells that expressed M(3) muscarin
218 to block the binding of ligands to pgsA-745 Chinese hamster ovary cells that overexpress GPIHBP1.
221 ession of TREM-2-DAP12 enables nonphagocytic Chinese hamster ovary cells to internalize bacteria.
222 lations to bind and/or infect HL-60 cells or Chinese hamster ovary cells transfected to express PSGL-
223 ndent decrease in the luciferase activity in Chinese Hamster Ovary cells transfected with CACNA1C and
224 tent stem cell-derived cardiomyocytes and in Chinese hamster ovary cells transfected with SCN5A, enco
227 to inhibit MO-evoked calcium accumulation in Chinese hamster ovary cells transfected with TRPV1 and T
228 Forster resonance energy transfer (FRET) on Chinese hamster ovary cells under total internal refecti
229 nt protein-tagged KCNQ subunits expressed in Chinese hamster ovary cells under total internal reflect
230 ools to analyze proteins in situ in cultured Chinese hamster ovary cells using fluorescence recovery
231 upernatant following transient expression in Chinese hamster ovary cells were analyzed by immunoblot
233 leasing intracellular proteins from adherent Chinese hamster ovary cells while preserving the cell vi
234 imulated 2D diffusion conditions and in live Chinese hamster ovary cells with a GFP-tagged transmembr
238 nescence and storage lesion, was assessed in Chinese hamster ovary cells with/without functional GPIb
239 CaK in fusion with enhanced GFP in mammalian Chinese hamster ovary cells' plasma membrane gave rise t
240 ukaryotic cells (up to approximately 36% for Chinese hamster ovary cells) and bacterial cells (up to
241 efficiency for transfection (up to ~71% for Chinese hamster ovary cells) and permeabilization sugges
242 al epithelial cells) and a cancer cell line (Chinese hamster ovary cells) were exposed to liquid and
243 cular, in both Xenopus oocytes and mammalian Chinese hamster ovary cells, AaTXKbeta(2)(-)(6)(4), but
244 ly localized to the endoplasmic reticulum in Chinese hamster ovary cells, and this intracellular loca
246 of various cell types in culture, including Chinese hamster ovary cells, chicken DF1 fibroblasts, pr
247 etylglucosaminyltransferase I-deficient Lec1 Chinese hamster ovary cells, indicating that N-glycosyla
248 nnels, which are heterologously expressed in Chinese hamster ovary cells, into the depolarizing direc
250 Ibbeta3 integrins by talin head fragments in Chinese hamster ovary cells, nor do I observe affinity i
251 We expressed mutant forms of GPIHBP1 in Chinese hamster ovary cells, rat and human endothelial c
253 capability of our setup on fixed and living Chinese hamster ovary cells, showing the cytoskeleton dy
254 development of filopodia and lamellipodia in Chinese hamster ovary cells, stimulate their motility, a
255 increased plasmalogen synthesis in wild-type Chinese hamster ovary cells, strongly suggesting that Fa
256 tes and its binding to the LDLR expressed on Chinese hamster ovary cells, suggesting a direct interac
257 nt Kv3.4 channel heterologously expressed in Chinese hamster ovary cells, supporting our findings in
258 Th2 receptor (1.14 +/- 0.44 nM) expressed in Chinese hamster ovary cells, the binding being reversibl
260 when coexpressing ficolin-2 and ficolin-3 in Chinese hamster ovary cells, we could detect ficolin-2/-
261 ocell" particles are taken up efficiently by Chinese hamster ovary cells, where, due to a reduced pH
262 -1 K(+) channels heterologously expressed in Chinese hamster ovary cells, which are silent in physiol
282 ans to modify protein glycosylation, we used Chinese hamster ovary ldl-D cells defective in UDP-Gal/U
283 hanced binding to FcgammaRIIIa compared with Chinese hamster ovary or wild-type plant-derived 2G12.
284 which endogenously expresses DRD2) and CHO (Chinese hamster ovary) cell lines, decreasing luciferase
285 e the uptake of silver nanoparticles by CHO (Chinese hamster ovary) cells and their subsequent fate a
286 ssessed in the arrestin recruitment assay in Chinese hamster ovary-K(1) cells expressing the long iso
289 g to platelets from P1 but absent binding to Chinese hamster ovary-K1 cells expressing variant D304N
290 Ca(2+) upregulation; however, in HEK293T or Chinese hamster ovary-K1 cells overexpressing M3R, piloc
291 single-point mutations were transfected into Chinese hamster ovary-K1 cells, and affinity and functio
294 tion of human erythrocytes when expressed in Chinese hamster ovary-K1, but not in human endothelial k
296 screening assay using the GeneBridge4 human/Chinese hamster radiation hybrid panel and found to be t
299 rget cells (Salmonella typhimurium TA100 and Chinese hamster V79) of standard mutagenicity tests trea
300 region of the genome, in two cell lines, the Chinese hamster V79-derived G12 and G10 cells, respectiv