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1 ibutions of caspase-1 and -11 during genital Chlamydia infection.
2 nd ovary/oviduct microbiome with and without Chlamydia infection.
3 l as essential for endometrial repair during Chlamydia infection.
4 tor-6alpha (ATF6alpha)-were activated during Chlamydia infection.
5 ology and decreased fertility rate following Chlamydia infection.
6 . trachomatis organism load in human genital chlamydia infection.
7 gulated over the course of the intracellular Chlamydia infection.
8 roduction, revealing that retromer restricts Chlamydia infection.
9 fficacy of azithromycin for the treatment of chlamydia infection.
10 ily (7 days) for the treatment of urogenital chlamydia infection.
11 is heterogeneity in the risk of PID after a chlamydia infection.
12 azithromycin for the treatment of urogenital chlamydia infection.
13 ihood of developing more severe disease from Chlamydia infection.
14 null clones restored full susceptibility to Chlamydia infection.
15 , respectively, as important in facilitating Chlamydia infection.
16 molecule that plays a role in resistance to Chlamydia infection.
17 Golgi apparatus-independent pathway during a Chlamydia infection.
18 between host and bacterial metabolism during Chlamydia infection.
19 d pregnancy outcome in women with persistent chlamydia infection.
20 the incomplete protection provided by prior Chlamydia infection.
21 t dampens the host innate immune response to Chlamydia infection.
22 , and mediators and may reduce pregnancy and chlamydia infection.
23 wer regions of the GT respond differently to Chlamydia infection.
24 sceptibility and immune responses to genital Chlamydia infection.
25 endocarditis and chronicity is a hallmark of Chlamydia infection.
26 lighting a possible way to selectively treat Chlamydia infections.
27 on and thereby shape macrophage responses to Chlamydia infections.
28 ng inflammation and adaptive immunity during Chlamydia infections.
29 thological scarring seen as a consequence of Chlamydia infections.
30 participate in the immunopathology seen with Chlamydia infections.
31 innate and adaptive immune responses during Chlamydia infections.
32 nt for the ability of mutant mice to resolve Chlamydia infections.
33 and DQB1*06) to be associated with recurrent Chlamydia infection (adjusted relative odds [RO], >2.0;
34 study aims to determine whether the time of Chlamydia infection affects the genital tract microbiome
35 doxycycline for the treatment of urogenital chlamydia infection among adolescents in youth correctio
36 tcome was the community prevalence of ocular chlamydia infection among children aged 0-9 years at mon
38 dose azithromycin in the treatment of rectal chlamydia infection among men who have sex with men.
40 mmatory mediators by epithelial cells during Chlamydia infection and highlight the contribution of TL
41 sible association between complications from chlamydia infection and host circadian clock, which may
43 quirement for MyD88 in CD4(+) T cells during Chlamydia infection and indicate that the importance of
45 ncy, and tubal factor infertility) following chlamydia infection and repeat infection hampers the des
47 that being African American, having previous chlamydia infection, and having less than a high-school
48 al epithelial cells released IL-1alpha after Chlamydia infection, and increased secretion of the proi
49 both groups, treated baseline gonorrhoea and chlamydia infections, and assessed 9-month gonorrhoea an
50 me has been implicated in the progression of Chlamydia infections, and with chlamydial infections at
51 cus because more than 85% of men with rectal chlamydia infection are asymptomatic, and clinical guide
52 nd 53% to 100% of extragenital gonorrhea and chlamydia infections are asymptomatic or associated with
53 s estimates for clearance rates of untreated chlamydia infections are important for understanding chl
54 er, these results suggest that P2X7R affects Chlamydia infection by directly inhibiting infection in
55 tested the requirement for these proteins in Chlamydia infection by microinjecting anti-Pls1 and anti
57 did not result in significantly less ocular chlamydia infections compared with untreated communities
58 did not affect T cell-mediated clearance of Chlamydia infection, consistent with a B cell-specific r
59 endent designs that a concurrent Candida and Chlamydia infection could not accelerate or modulate the
62 in meters squared), parity, insurance type, Chlamydia infection during pregnancy, smoking, and rural
63 g all sexually active adolescent females for chlamydia infection every 6 months, regardless of sympto
65 D8+ T cells, the response of CD8+ T cells to Chlamydia infection has been explored in a number of stu
68 herosclerosis suggests an etiologic role for Chlamydia infection in the development of coronary ather
71 ay reduce the likelihood of continued ocular chlamydia infection in the setting of mass antibiotic tr
72 ot reduce the rate of new HIV, gonorrhea, or chlamydia infection in this group of sex workers who use
80 volved in humoral protection against genital chlamydia infection is crucial to development of an effe
82 f antibodies in protection against a primary Chlamydia infection is unclear and was a focus of this w
83 that the robust type 2 immunity elicited by Chlamydia infection of human genital tissue may analogou
84 genes that were defectively regulated during Chlamydia infection of the TLR3-deficient OE cells, and
86 um is commonly used as a model for ascending Chlamydia infections of the human female genital tract.
87 Possible factors, such as time of day of chlamydia infection on chlamydial pathogenesis has not b
88 d cervical cancer may be due to an effect of chlamydia infection on persistence of high-risk HPV.
89 These findings suggest that the effect of a Chlamydia infection on trophoblast secretion of chemokin
90 demonstrated that interleukin-8 induction by Chlamydia infection or PGE2 treatment was dependent on e
91 dy discharge was the strongest predictor for Chlamydia infection (OR = 20.99, 95% CI: 6.09-72.29, p <
95 thelial (OE) cells that is stimulated during Chlamydia infection, resulting in the synthesis of beta
96 transcripts were compared among subgroups of chlamydia infection status and ocular disease presentati
98 les analyzed individually identified 3% more chlamydia infections than pooled (99.3% and 96.0%; P = .
99 les analysed individually identified 3% more chlamydia infections than pooled (99.3%, 96.0%; p=0.027)
101 g directly observed treatment for urogenital chlamydia infection, the efficacy of azithromycin was 97
102 is no obvious dsRNA molecule associated with Chlamydia infection, the requirement for TLR3 in Chlamyd
104 ydia replication and restored the ability of Chlamydia infection to induce IL-6, CXCL10, and CCL5 exp
106 udies that showed extant upper genital tract Chlamydia infection was associated with increased co-exp
107 We have previously shown that the time of Chlamydia infection was crucial in determining the chlam
109 uently suffer from ocular diseases caused by Chlamydia infection, we also examined the eye microbiome
110 migrate to the genital tract in response to Chlamydia infection, we generated retrogenic mice that e
111 ow CD4 count (<350 cells/mm(3)) and previous chlamydia infection were associated with an increased ri
112 etween condom use and incident gonorrhea and chlamydia infection were compared between case-crossover
113 niasis, bacterial vaginosis, gonorrhoea, and chlamydia infection were significantly lower in the inte
114 metrial leukocyte (constitutively and during Chlamydia infection), whereas studies with eosinophil-de
115 type had elevated IL-10 concentrations after Chlamydia infection, which may reflect involvement of a
116 ted endocervical specimens for gonorrhea and chlamydia infection with DNA probes, tested for HIV infe
117 IFN-gamma-/- mice have in the resolution of Chlamydia infection, yet IFN-gamma production by CTL is