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1 tis mouse pneumonitis and L3 serovars and to Chlamydia pneumoniae.
2 chlamydial species Chlamydia trachomatis or Chlamydia pneumoniae.
3 udies that have targeted patients exposed to Chlamydia pneumoniae.
4 bacterial DNA from several oral species and Chlamydia pneumoniae.
5 much interest has focused on infection with Chlamydia pneumoniae.
6 zed to contain nucleic acid sequences of the Chlamydia pneumoniae 16S rRNA primers in a position flan
10 ystemic bacterial-entry network initiated by Chlamydia pneumoniae, a widespread opportunistic pathoge
12 Intraperitoneal inoculation of mice with Chlamydia pneumoniae, after immunization with neural ant
13 mplement activation occurs on the surface of Chlamydia pneumoniae, an obligate intracellular Gram-neg
15 ms accounting for the potential link between Chlamydia pneumoniae and atherosclerosis are unknown.
16 o establish a causative relationship between Chlamydia pneumoniae and atherosclerosis, animal models
17 otentially strong association exists between Chlamydia pneumoniae and atherosclerosis, but the clinic
18 possible association of prior infection with Chlamydia pneumoniae and atherosclerotic risk, the contr
19 ions, have the capacity to support growth of Chlamydia pneumoniae and be activated to secrete proinfl
20 investigate a proposed relationship between Chlamydia pneumoniae and coronary heart disease, coronar
22 plex virus type 1 (HSV1), HSV type 2 (HSV2), Chlamydia pneumoniae and Helicobacter pylori, and C-reac
23 but new methods to detect S. pneumoniae, or Chlamydia pneumoniae and Mycoplasma pneumoniae may facil
25 wever, prospective data relating exposure to Chlamydia pneumoniae and risks of future myocardial infa
26 NA genes specific for Chlamydia trachomatis, Chlamydia pneumoniae, and Chlamydia psittaci as improved
27 tein gene (omcB) from Chlamydia trachomatis, Chlamydia pneumoniae, and Chlamydia psittaci were analyz
28 ether seropositivity to Helicobacter pylori, Chlamydia pneumoniae, and cytomegalovirus (CMV) is an in
29 cal evidence on CHD and Helicobacter pylori, Chlamydia pneumoniae, and cytomegalovirus (CMV), as well
31 to determine the presence of M. pneumoniae, Chlamydia pneumoniae, and seven respiratory viruses thro
32 present in all Chlamydia species except for Chlamydia pneumoniae, and their catalytic domains bear s
33 gionella antigens, Mycoplasma pneumoniae and Chlamydia pneumoniae antibodies from paired serums, and
35 Recent observations have shown that both Chlamydia pneumoniae antigens and DNA may be found withi
36 matis (C/TW-3/OT, E/UW-5/Cx, and L2/434/Bu), Chlamydia pneumoniae (AR-39), and Chlamydia psittaci (6B
39 hogenesis of AF, and Helicobacter pylori and Chlamydia pneumoniae are two bacteria that have aroused
40 Chlamydia trachomatis and Chlamydophila (Chlamydia) pneumoniae are known triggers of reactive art
42 f tumor necrosis factor alpha (TNF-alpha) in Chlamydia pneumoniae atherogenesis was evaluated in TNF-
43 med using primers for Chlamydia trachomatis, Chlamydia pneumoniae, Borrelia burgdorferi, and pan bact
44 GF2) was shown to enhance the infectivity of Chlamydia pneumoniae but not C. trachomatis in endotheli
45 fic to Chlamydiales and were confirmed to be Chlamydia pneumoniae by a C. pneumoniae-specific ompA-ba
46 t studies have suggested that infection with Chlamydia pneumoniae (C pneumoniae) may contribute to th
49 cterial sexually transmitted infections, and Chlamydia pneumoniae causes community-acquired respirato
50 the same tube) and could discriminate among Chlamydia pneumoniae, Chlamydia psittaci, and Chlamydia
56 nce of the association between elevated anti-Chlamydia pneumoniae (Cp) antibody titres and coronary h
62 It has been suggested that infection with Chlamydia pneumoniae(CPn) can trigger inflammatory mecha
64 nome of the obligate intracellular bacterium Chlamydia pneumoniae CWL029 encodes a family of 21 prote
65 [CRP]) and infection (eg, seropositivity to Chlamydia pneumoniae, cytomegalovirus [CMV], and Helicob
68 previously shown that different isolates of Chlamydia pneumoniae display heterogeneity in the in vit
69 d strain 129 mice infected intranasally with Chlamydia pneumoniae displayed a moderate-to-severe infl
70 Mouse models were used to determine whether Chlamydia pneumoniae establishes chronic infection of th
71 ata have shown that the respiratory pathogen Chlamydia pneumoniae expresses an altered gene transcrip
73 e need to understand the transmissibility of Chlamydia pneumoniae from systemic infections in order t
94 us other infectious agents (cytomegalovirus, Chlamydia pneumoniae, Helicobacter pylori, and herpes si
95 obulin-G antibody titers to cytomegalovirus, Chlamydia pneumoniae, Helicobacter pylori, hepatitis A v
96 serological evidence of prior infection with Chlamydia pneumoniae, herpes simplex virus type 1 (HSV-1
97 rterectomy were examined for the presence of Chlamydia pneumoniae, human cytomegalovirus, and bacteri
98 timulated with human cytomegalovirus (HCMV), Chlamydia pneumoniae, human heat-shock protein 60 (hHSP6
101 we found no evidence of association between Chlamydia pneumoniae IgG seropositivity and risks of fut
102 relative risks of future MI associated with Chlamydia pneumoniae IgG titers >/=1:16, 1:32, 1:64, 1:1
105 we measured IgG antibodies directed against Chlamydia pneumoniae in blood samples collected at basel
110 lity" criterion of a role for infection with Chlamydia pneumoniae in the pathogenesis of human athero
111 licated specific infectious agents including Chlamydia pneumoniae in the progression of atherogenesis
114 udies implicate infectious agents, including Chlamydia pneumoniae, in the pathogenesis of atheroscler
116 amydia trachomatis, Chlamydia muridarum, and Chlamydia pneumoniae inclusions, whereas GFP-Rab5, GFP-R
117 t was found that the intracellular bacterium Chlamydia pneumoniae induces foam cell formation by huma
120 progression and the influence of infection (Chlamydia pneumoniae-infected vs. uninfected control mic
121 FP) transgenic mice were used to analyze how Chlamydia pneumoniae infection affects the adherence of
124 ing evidence supports an association between Chlamydia pneumoniae infection and atherosclerosis.
125 iological evidence of an association between Chlamydia pneumoniae infection and coronary artery disea
127 us work has suggested an association between Chlamydia pneumoniae infection and coronary atherosclero
128 To examine a possible relationship between Chlamydia pneumoniae infection and multiple sclerosis (M
136 thologic, and animal model studies show that Chlamydia pneumoniae infection is associated with corona
137 o scarring of ocular or genital tissues, and Chlamydia pneumoniae infection is associated with the de
139 , properdin-depleted serum could not control Chlamydia pneumoniae infection of HEp-2 cells compared w
141 merous studies have suggested a link between Chlamydia pneumoniae infection, atherosclerosis, and cor
142 dy showed that in contrast to infection with Chlamydia pneumoniae, infection of the lung and aorta wi
143 alence of synovial Chlamydia trachomatis and Chlamydia pneumoniae infections in patients with chronic
154 lopmentally regulated intracellular pathogen Chlamydia pneumoniae is a natural tryptophan auxotroph.
166 ether past use of antibiotics active against Chlamydia pneumoniae is associated with a decrease in th
168 gic studies suggest that past infection with Chlamydia pneumoniae is associated with clinical and sub
173 trast, it has been reported that the MOMP of Chlamydia pneumoniae is not surface exposed and is immun
175 ntly that the bacterial respiratory pathogen Chlamydia pneumoniae is present in the cerebrospinal flu
178 The intracellular bacterium Chlamydophila ("Chlamydia") pneumoniae is a pathogen for several respira
181 ance has not yet been described in wild type Chlamydia pneumoniae isolates, nor has selective emergen
182 the immune and disease responses to repeated Chlamydia pneumoniae lung infection by multivariate mode
183 based on the hypothesis that infection with Chlamydia pneumoniae may be causally associated with car
185 ecognize corresponding peptides derived from Chlamydia pneumoniae MOMP, further suggesting that they
186 s been implicated in asthma inception, while Chlamydia pneumoniae, Mycoplasma pneumoniae, and latent
190 nfection with very common organisms, such as Chlamydia pneumoniae or cytomegalovirus, may lead to a l
191 utions of the antibiotics, and infected with Chlamydia pneumoniae or stimulated with tumor necrosis f
192 history of infection with pathogens such as Chlamydia pneumoniae, or other systemic inflammation, ca
194 t 30 publications from 1992 to 1999 describe Chlamydia pneumoniae organisms in atherosclerotic lesion
195 f the major outer membrane protein (MOMP) of Chlamydia pneumoniae, peptides representing these areas
201 tudy was to examine the relationship between Chlamydia pneumoniae seropositivity and aortic atheroscl
202 Associations have been reported between Chlamydia pneumoniae seropositivity and both acute and c
203 quence comparison between C. trachomatis and Chlamydia pneumoniae showed less conservation between sp
204 onitis (MoPn) strain Nigg (1 069 412 nt) and Chlamydia pneumoniae strain AR39 (1 229 853 nt) were det
205 cans (GAGs) in the invasion of host cells by Chlamydia pneumoniae strains TW-183 and A-03 was investi
206 rms accelerates complement activation on the Chlamydia pneumoniae surface, as measured by C3b and C9
207 inis-like mycoplasma from a stock culture of Chlamydia pneumoniae TW-183 obtained from the American T
213 However, the regulation of infection with Chlamydia pneumoniae, which is a ubiquitous pneumonia-ca