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1 tis mouse pneumonitis and L3 serovars and to Chlamydia pneumoniae.
2  chlamydial species Chlamydia trachomatis or Chlamydia pneumoniae.
3 udies that have targeted patients exposed to Chlamydia pneumoniae.
4  bacterial DNA from several oral species and Chlamydia pneumoniae.
5  much interest has focused on infection with Chlamydia pneumoniae.
6 zed to contain nucleic acid sequences of the Chlamydia pneumoniae 16S rRNA primers in a position flan
7                               Infection with Chlamydia pneumoniae, a causative agent of acute and chr
8                                              Chlamydia pneumoniae, a common cause of respiratory infe
9                               Infection with Chlamydia pneumoniae, a human respiratory pathogen, has
10 ystemic bacterial-entry network initiated by Chlamydia pneumoniae, a widespread opportunistic pathoge
11         We have previously demonstrated that Chlamydia pneumoniae accelerates plaque formation in apo
12     Intraperitoneal inoculation of mice with Chlamydia pneumoniae, after immunization with neural ant
13 mplement activation occurs on the surface of Chlamydia pneumoniae, an obligate intracellular Gram-neg
14                Infections with the bacterium Chlamydia pneumoniae, an RNA virus, and herpesviruses al
15 ms accounting for the potential link between Chlamydia pneumoniae and atherosclerosis are unknown.
16 o establish a causative relationship between Chlamydia pneumoniae and atherosclerosis, animal models
17 otentially strong association exists between Chlamydia pneumoniae and atherosclerosis, but the clinic
18 possible association of prior infection with Chlamydia pneumoniae and atherosclerotic risk, the contr
19 ions, have the capacity to support growth of Chlamydia pneumoniae and be activated to secrete proinfl
20  investigate a proposed relationship between Chlamydia pneumoniae and coronary heart disease, coronar
21                               Moreover, both Chlamydia pneumoniae and cytomegalovirus exacerbate lesi
22 plex virus type 1 (HSV1), HSV type 2 (HSV2), Chlamydia pneumoniae and Helicobacter pylori, and C-reac
23  but new methods to detect S. pneumoniae, or Chlamydia pneumoniae and Mycoplasma pneumoniae may facil
24                                              Chlamydia pneumoniae and Mycoplasma pneumoniae were eval
25 wever, prospective data relating exposure to Chlamydia pneumoniae and risks of future myocardial infa
26 NA genes specific for Chlamydia trachomatis, Chlamydia pneumoniae, and Chlamydia psittaci as improved
27 tein gene (omcB) from Chlamydia trachomatis, Chlamydia pneumoniae, and Chlamydia psittaci were analyz
28 ether seropositivity to Helicobacter pylori, Chlamydia pneumoniae, and cytomegalovirus (CMV) is an in
29 cal evidence on CHD and Helicobacter pylori, Chlamydia pneumoniae, and cytomegalovirus (CMV), as well
30 ionella pneumophila, Haemophilus influenzae, Chlamydia pneumoniae, and Mycoplasma pneumoniae.
31  to determine the presence of M. pneumoniae, Chlamydia pneumoniae, and seven respiratory viruses thro
32  present in all Chlamydia species except for Chlamydia pneumoniae, and their catalytic domains bear s
33 gionella antigens, Mycoplasma pneumoniae and Chlamydia pneumoniae antibodies from paired serums, and
34 lamydia psittaci, whereas the titers of anti-Chlamydia pneumoniae antibodies were the highest.
35     Recent observations have shown that both Chlamydia pneumoniae antigens and DNA may be found withi
36 matis (C/TW-3/OT, E/UW-5/Cx, and L2/434/Bu), Chlamydia pneumoniae (AR-39), and Chlamydia psittaci (6B
37                      Helicobacter pylori and Chlamydia pneumoniae are both pathogenic to humans.
38  mitochondrial DNA and viral factors such as Chlamydia pneumoniae are reviewed.
39 hogenesis of AF, and Helicobacter pylori and Chlamydia pneumoniae are two bacteria that have aroused
40     Chlamydia trachomatis and Chlamydophila (Chlamydia) pneumoniae are known triggers of reactive art
41                    We found that recombinant Chlamydia pneumoniae ArgR functions as an arginine-depen
42 f tumor necrosis factor alpha (TNF-alpha) in Chlamydia pneumoniae atherogenesis was evaluated in TNF-
43 med using primers for Chlamydia trachomatis, Chlamydia pneumoniae, Borrelia burgdorferi, and pan bact
44 GF2) was shown to enhance the infectivity of Chlamydia pneumoniae but not C. trachomatis in endotheli
45 fic to Chlamydiales and were confirmed to be Chlamydia pneumoniae by a C. pneumoniae-specific ompA-ba
46 t studies have suggested that infection with Chlamydia pneumoniae (C pneumoniae) may contribute to th
47                                              Chlamydia pneumoniae can infect arterial cells.
48                                              Chlamydia pneumoniae causes community-acquired pneumonia
49 cterial sexually transmitted infections, and Chlamydia pneumoniae causes community-acquired respirato
50  the same tube) and could discriminate among Chlamydia pneumoniae, Chlamydia psittaci, and Chlamydia
51           Heat shock protein 60 derived from Chlamydia pneumoniae (cHSP60) activates Toll-like recept
52                                              Chlamydia pneumoniae commonly causes respiratory infecti
53 ly of polymorphic membrane proteins (Pmp) in Chlamydia pneumoniae consists of 21 members.
54                   Heterologous expression of Chlamydia pneumoniae CopN in yeast and mammalian cells r
55                                          The Chlamydia pneumoniae CopN protein is a member of the Yop
56 nce of the association between elevated anti-Chlamydia pneumoniae (Cp) antibody titres and coronary h
57                    Cytomegalovirus (CMV) and Chlamydia pneumoniae (CP) possibly contribute to atheros
58                              IgA antibody to Chlamydia pneumoniae (CP)>or=1:64 was considered indicat
59                        In the mouse model of Chlamydia pneumoniae (Cpn) infection, this immunity is c
60 nity to the intracellular bacterial pathogen Chlamydia pneumoniae (Cpn).
61  cells play an essential role in immunity to Chlamydia pneumoniae (Cpn).
62    It has been suggested that infection with Chlamydia pneumoniae(CPn) can trigger inflammatory mecha
63 were not serovar restricted or confounded by Chlamydia pneumoniae cross-reactivity.
64 nome of the obligate intracellular bacterium Chlamydia pneumoniae CWL029 encodes a family of 21 prote
65  [CRP]) and infection (eg, seropositivity to Chlamydia pneumoniae, cytomegalovirus [CMV], and Helicob
66                                              Chlamydia pneumoniae, cytomegalovirus and Helicobacter p
67 ms of human properdin bind to the surface of Chlamydia pneumoniae directly.
68  previously shown that different isolates of Chlamydia pneumoniae display heterogeneity in the in vit
69 d strain 129 mice infected intranasally with Chlamydia pneumoniae displayed a moderate-to-severe infl
70  Mouse models were used to determine whether Chlamydia pneumoniae establishes chronic infection of th
71 ata have shown that the respiratory pathogen Chlamydia pneumoniae expresses an altered gene transcrip
72            Three methods for the recovery of Chlamydia pneumoniae from spiked nasopharyngeal and bloo
73 e need to understand the transmissibility of Chlamydia pneumoniae from systemic infections in order t
74                             The isolation of Chlamydia pneumoniae from the cerebrospinal fluid (CSF)
75                                              Chlamydia pneumoniae has been associated with atheroscle
76                                              Chlamydia pneumoniae has been associated with atheroscle
77                                              Chlamydia pneumoniae has been detected in atheroscleroti
78                                              Chlamydia pneumoniae has been found within atherosclerot
79                                              Chlamydia pneumoniae has been hypothesized to play a rol
80                                              Chlamydia pneumoniae has been identified in coronary ath
81                               Infection with Chlamydia pneumoniae has been implicated as a potential
82               Because chronic infection with Chlamydia pneumoniae has been implicated in the pathogen
83                                              Chlamydia pneumoniae has been postulated to cause system
84                                              Chlamydia pneumoniae has been reported as a possible con
85                                              Chlamydia pneumoniae has been shown to accelerate athero
86                                              Chlamydia pneumoniae has been shown to possess at least
87                                              Chlamydia pneumoniae has recently been associated with a
88                    One particular bacterium, Chlamydia pneumoniae, has been observed at high frequenc
89               Both cytomegalovirus (CMV) and Chlamydia pneumoniae have been associated with atheroscl
90                                              Chlamydia pneumoniae have been demonstrated in atheroscl
91   To date attempts to use this approach with Chlamydia pneumoniae have failed.
92                 Certain pathogens, including Chlamydia pneumoniae, have evolved mechanisms to subvert
93     Many pathogens, including Chlamydophila (Chlamydia) pneumoniae, have been associated with MS.
94 us other infectious agents (cytomegalovirus, Chlamydia pneumoniae, Helicobacter pylori, and herpes si
95 obulin-G antibody titers to cytomegalovirus, Chlamydia pneumoniae, Helicobacter pylori, hepatitis A v
96 serological evidence of prior infection with Chlamydia pneumoniae, herpes simplex virus type 1 (HSV-1
97 rterectomy were examined for the presence of Chlamydia pneumoniae, human cytomegalovirus, and bacteri
98 timulated with human cytomegalovirus (HCMV), Chlamydia pneumoniae, human heat-shock protein 60 (hHSP6
99                                              Chlamydia pneumoniae IgG antibodies (titers >or=1:16) we
100                                              Chlamydia pneumoniae IgG antibody titers are not associa
101  we found no evidence of association between Chlamydia pneumoniae IgG seropositivity and risks of fut
102  relative risks of future MI associated with Chlamydia pneumoniae IgG titers >/=1:16, 1:32, 1:64, 1:1
103                                              Chlamydia pneumoniae illness is poorly characterized, pa
104              We investigated the presence of Chlamydia pneumoniae in 81 normal and pathological speci
105  we measured IgG antibodies directed against Chlamydia pneumoniae in blood samples collected at basel
106                              The presence of Chlamydia pneumoniae in many atherosclerotic lesions rai
107               Recent evidence has implicated Chlamydia pneumoniae in the aggravation of atheroscleros
108          In literature in which detection of Chlamydia pneumoniae in the artery is described, the met
109                        Our identification of Chlamydia pneumoniae in the cerebrospinal fluid (CSF) of
110 lity" criterion of a role for infection with Chlamydia pneumoniae in the pathogenesis of human athero
111 licated specific infectious agents including Chlamydia pneumoniae in the progression of atherogenesis
112 ant percentage of healthy blood donors carry Chlamydia pneumoniae in their blood.
113 succinate on the growth of three isolates of Chlamydia pneumoniae in vitro.
114 udies implicate infectious agents, including Chlamydia pneumoniae, in the pathogenesis of atheroscler
115               Here we report that Cpn0585, a Chlamydia pneumoniae inclusion membrane protein (Inc), i
116 amydia trachomatis, Chlamydia muridarum, and Chlamydia pneumoniae inclusions, whereas GFP-Rab5, GFP-R
117 t was found that the intracellular bacterium Chlamydia pneumoniae induces foam cell formation by huma
118                                              Chlamydia pneumoniae induces macrophage foam cell format
119 en reading frame Cpn0797 in the cytoplasm of Chlamydia pneumoniae-infected host cells.
120  progression and the influence of infection (Chlamydia pneumoniae-infected vs. uninfected control mic
121 FP) transgenic mice were used to analyze how Chlamydia pneumoniae infection affects the adherence of
122 suggest that there is an association between Chlamydia pneumoniae infection and atherogenesis.
123 evidence have implied an association between Chlamydia pneumoniae infection and atherogenesis.
124 ing evidence supports an association between Chlamydia pneumoniae infection and atherosclerosis.
125 iological evidence of an association between Chlamydia pneumoniae infection and coronary artery disea
126          Evidence for an association between Chlamydia pneumoniae infection and coronary artery disea
127 us work has suggested an association between Chlamydia pneumoniae infection and coronary atherosclero
128   To examine a possible relationship between Chlamydia pneumoniae infection and multiple sclerosis (M
129                                              Chlamydia pneumoniae infection can exacerbate atheroscle
130                                              Chlamydia pneumoniae infection has been associated with
131                       Serologic diagnosis of Chlamydia pneumoniae infection has been based on the mic
132                                      Chronic Chlamydia pneumoniae infection has been implicated in th
133 ed an in vitro model of long-term continuous Chlamydia pneumoniae infection in HEp-2 cells.
134                                              Chlamydia pneumoniae infection induces inflammatory chan
135                                              Chlamydia pneumoniae infection is associated with athero
136 thologic, and animal model studies show that Chlamydia pneumoniae infection is associated with corona
137 o scarring of ocular or genital tissues, and Chlamydia pneumoniae infection is associated with the de
138                                              Chlamydia pneumoniae infection is implicated in atherosc
139 , properdin-depleted serum could not control Chlamydia pneumoniae infection of HEp-2 cells compared w
140       Experimental and clinical studies link Chlamydia pneumoniae infection to atherogenesis and athe
141 merous studies have suggested a link between Chlamydia pneumoniae infection, atherosclerosis, and cor
142 dy showed that in contrast to infection with Chlamydia pneumoniae, infection of the lung and aorta wi
143 alence of synovial Chlamydia trachomatis and Chlamydia pneumoniae infections in patients with chronic
144                                              Chlamydia pneumoniae is a causative agent for many respi
145                                              Chlamydia pneumoniae is a common cause of pulmonary infe
146                                              Chlamydia pneumoniae is a common cause of upper and lowe
147                                              Chlamydia pneumoniae is a common human respiratory patho
148                                              Chlamydia pneumoniae is a common respiratory pathogen as
149                                              Chlamydia pneumoniae is a common respiratory pathogen th
150                                              Chlamydia pneumoniae is a common respiratory pathogen th
151                                              Chlamydia pneumoniae is a common respiratory pathogen, w
152                                              Chlamydia pneumoniae is a human pathogen that primarily
153                                              Chlamydia pneumoniae is a major human pathogen that is w
154 lopmentally regulated intracellular pathogen Chlamydia pneumoniae is a natural tryptophan auxotroph.
155                                              Chlamydia pneumoniae is a respiratory pathogen that has
156                                              Chlamydia pneumoniae is a respiratory tract pathogen but
157                                              Chlamydia pneumoniae is an important human pathogen, but
158                                              Chlamydia pneumoniae is an important respiratory pathoge
159                                              Chlamydia pneumoniae is an intracellular bacterium respo
160                                              Chlamydia pneumoniae is an obligate intracellular human
161                                              Chlamydia pneumoniae is an obligate intracellular parasi
162                                              Chlamydia pneumoniae is an obligate intracellular pathog
163                                              Chlamydia pneumoniae is an obligate intracellular pathog
164                                              Chlamydia pneumoniae is an obligate intracellular prokar
165                                              Chlamydia pneumoniae is an omnipresent obligate intracel
166 ether past use of antibiotics active against Chlamydia pneumoniae is associated with a decrease in th
167                                              Chlamydia pneumoniae is associated with atherosclerosis
168 gic studies suggest that past infection with Chlamydia pneumoniae is associated with clinical and sub
169                                              Chlamydia pneumoniae is associated with coronary artery
170       Infection with Helicobacter pylori and Chlamydia pneumoniae is associated with coronary heart d
171                                              Chlamydia pneumoniae is commonly detected in atheroscler
172                                              Chlamydia pneumoniae is detected by macrophages and othe
173 trast, it has been reported that the MOMP of Chlamydia pneumoniae is not surface exposed and is immun
174                                              Chlamydia pneumoniae is one possible pathogen.
175 ntly that the bacterial respiratory pathogen Chlamydia pneumoniae is present in the cerebrospinal flu
176 and harvest of purified elementary bodies of Chlamydia pneumoniae is presented.
177                                              Chlamydia pneumoniae is the causative agent of respirato
178  The intracellular bacterium Chlamydophila ("Chlamydia") pneumoniae is a pathogen for several respira
179              This toxin gene, not present in Chlamydia pneumoniae, is a member of the YopT effector f
180                               Replication of Chlamydia pneumoniae isolates A-03 and BAL-16 was inhibi
181 ance has not yet been described in wild type Chlamydia pneumoniae isolates, nor has selective emergen
182 the immune and disease responses to repeated Chlamydia pneumoniae lung infection by multivariate mode
183  based on the hypothesis that infection with Chlamydia pneumoniae may be causally associated with car
184           Recent reports have suggested that Chlamydia pneumoniae may play a role in the pathogenesis
185 ecognize corresponding peptides derived from Chlamydia pneumoniae MOMP, further suggesting that they
186 s been implicated in asthma inception, while Chlamydia pneumoniae, Mycoplasma pneumoniae, and latent
187                                Attributes of Chlamydia pneumoniae of potential importance to a relati
188                 The effect of infection with Chlamydia pneumoniae on host messenger RNA expression in
189          The hypothetical protein encoded by Chlamydia pneumoniae open reading frame cpn0308 was dete
190 nfection with very common organisms, such as Chlamydia pneumoniae or cytomegalovirus, may lead to a l
191 utions of the antibiotics, and infected with Chlamydia pneumoniae or stimulated with tumor necrosis f
192  history of infection with pathogens such as Chlamydia pneumoniae, or other systemic inflammation, ca
193            We therefore conclude that viable Chlamydia pneumoniae organisms are frequently present in
194 t 30 publications from 1992 to 1999 describe Chlamydia pneumoniae organisms in atherosclerotic lesion
195 f the major outer membrane protein (MOMP) of Chlamydia pneumoniae, peptides representing these areas
196              The common respiratory pathogen Chlamydia pneumoniae recently has been implicated as a r
197               Exposure of human monocytes to Chlamydia pneumoniae resulted in a significant enhanceme
198               During invasion of host cells, Chlamydia pneumoniae secretes the effector protein CPn06
199                       We show that AmiA from Chlamydia pneumoniae separates daughter cells in an Esch
200     We also assessed Helicobacter pylori and Chlamydia pneumoniae serology.
201 tudy was to examine the relationship between Chlamydia pneumoniae seropositivity and aortic atheroscl
202      Associations have been reported between Chlamydia pneumoniae seropositivity and both acute and c
203 quence comparison between C. trachomatis and Chlamydia pneumoniae showed less conservation between sp
204 onitis (MoPn) strain Nigg (1 069 412 nt) and Chlamydia pneumoniae strain AR39 (1 229 853 nt) were det
205 cans (GAGs) in the invasion of host cells by Chlamydia pneumoniae strains TW-183 and A-03 was investi
206 rms accelerates complement activation on the Chlamydia pneumoniae surface, as measured by C3b and C9
207 inis-like mycoplasma from a stock culture of Chlamydia pneumoniae TW-183 obtained from the American T
208            To date, use of MIF serology with Chlamydia pneumoniae (TWAR) antigen has provided the mos
209                                              Chlamydia pneumoniae (TWAR), a common cause of acute res
210                                              Chlamydia pneumoniae was cultured from atherosclerotic p
211                                              Chlamydia pneumoniae was the causative pathogen of this
212 required for infection by C. trachomatis and Chlamydia pneumoniae were characterized.
213    However, the regulation of infection with Chlamydia pneumoniae, which is a ubiquitous pneumonia-ca

 
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