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1 mycin antibiotic treatment and opportunistic Clostridium difficile infection.
2 contributes to the pathology observed during Clostridium difficile infection.
3 idian Bioscience, Inc.) for the diagnosis of Clostridium difficile infection.
4 rectal procedures without increased risk of Clostridium difficile infection.
5 bleed, ventilator-associated pneumonia, and Clostridium difficile infection.
6 a transplantation for treatment of recurrent Clostridium difficile infection.
7 those at high risk of complications such as Clostridium difficile infection.
8 onia, ICU mortality, ICU length of stay, and Clostridium difficile infection.
9 ) in an investigation of the transmission of Clostridium difficile infection.
10 n of outbreaks is crucial for the control of Clostridium difficile infection.
11 n was recently approved for the treatment of Clostridium difficile infection.
12 aboratories from utilizing real-time PCR for Clostridium difficile infection.
13 ce, severity, and high rate of recurrence of Clostridium difficile infection.
14 mples for CCCA did not increase detection of Clostridium difficile infection.
15 G II plays in the rabbit ileal-loop model of Clostridium difficile infection.
16 the responses of luminal epithelial cells to Clostridium difficile infection.
17 ntestinal symptoms, rash, kidney injury, and Clostridium difficile infection.
18 idity, development of ileus, reoperation and Clostridium difficile infection.
19 xolidinone antibiotic developed for treating Clostridium difficile infection.
20 nsecutive, evaluable patients with recurrent Clostridium difficile infection.
21 and fidaxomicin are therapies of choice for Clostridium difficile infection.
22 ns about promoting antibiotic resistance and Clostridium difficile infection.
23 , ventilator-associated pneumonia (VAP), and Clostridium difficile infections.
24 antimicrobial use and adverse events such as Clostridium difficile infections.
25 r understanding the changing epidemiology of Clostridium difficile infections.
27 ently encountered infectious etiologies were Clostridium difficile infection (13.3% and 11.8%, respec
29 s in Ahrr(-/-) mice caused susceptibility to Clostridium difficile infection and dextran sodium-sulfa
30 ts it has been successfully used in cases of Clostridium difficile infection and IBD, although contro
32 al microbiota transplantation to face severe Clostridium difficile infections and to perform decoloni
33 ated charges for inflammatory bowel disease, Clostridium difficile infection, and chronic liver disea
34 with outcomes (antibiotic-days, incidence of Clostridium difficile infection, and in-hospital mortali
35 numbers of Staphylococcus aureus bacteremia, Clostridium difficile infection, and vancomycin-resistan
36 han 9000 nosocomial infections, 1000 to 5000 Clostridium difficile infections, and 2 to 6 cases of an
37 ctors affecting a person's susceptibility to Clostridium difficile infection are well-understood, lit
39 tic withdrawal regimen may resolve recurrent Clostridium difficile infection as effectively as fecal
40 sistently toxin-negative patients tested for Clostridium difficile infection between February 1998 an
42 s is not observed in subjects with recurrent Clostridium difficile infection but is observed in the s
43 rded as the primary virulence determinant in Clostridium difficile infection, but naturally occurring
44 ffective in treating relapsing or refractory Clostridium difficile infection, but practical barriers
51 plification tests (NAATs) do not distinguish Clostridium difficile infection (CDI) and asymptomatic C
53 e advances in the diagnosis and treatment of Clostridium difficile infection (CDI) and prevention eff
55 n to be noninferior to vancomycin for curing Clostridium difficile infection (CDI) and superior for r
56 f reduced response to standard therapies for Clostridium difficile infection (CDI) and the risk for r
57 s of Staphylococcus aureus bacteremia (SAB), Clostridium difficile infection (CDI) and vancomycin-res
60 es suggest that most cases of hospital-onset Clostridium difficile infection (CDI) are unrelated to o
61 methods may underestimate the true burden of Clostridium difficile infection (CDI) because they fail
66 tic increase in morbidity and mortality from Clostridium difficile infection (CDI) due to the recent
67 The dramatic changes in the epidemiology of Clostridium difficile infection (CDI) during recent year
74 dences of antibiotic-associated diarrhea and Clostridium difficile infection (CDI) has been demonstra
76 icrobiota transplantation (FMT) in recurrent Clostridium difficile infection (CDI) has been limited t
77 igh sensitivity of PCR assays for diagnosing Clostridium difficile infection (CDI) has greatly reduce
78 splantation (FMT) as treatment for recurrent Clostridium difficile infection (CDI) has increased rapi
79 osing to, or modulating disease severity in, Clostridium difficile infection (CDI) has not been inves
86 id suppression medication is associated with Clostridium difficile infection (CDI) in adults and is i
87 ent approved by the FDA for the treatment of Clostridium difficile infection (CDI) in adults over the
88 dies on risk factors for and transmission of Clostridium difficile infection (CDI) in China have been
89 of revised clinical practice guidelines for Clostridium difficile infection (CDI) in February 2018.
90 This article defines the risk factors for Clostridium difficile infection (CDI) in hospitalized ch
92 ) is highly effective for treating recurrent Clostridium difficile infection (CDI) in observational s
94 e clinical and laboratory characteristics of Clostridium difficile infection (CDI) in patients with d
95 e an appropriate therapeutic option for mild Clostridium difficile infection (CDI) in select patients
97 and influenza are associated with nosocomial Clostridium difficile infection (CDI) incidence, but the
123 mal therapy for critically ill patients with Clostridium difficile infection (CDI) is not known.
124 f fecal microbiota transplantation (FMT) for Clostridium difficile infection (CDI) is not well-known.
142 rganism bloodstream infection (MDRO-BSI) and Clostridium difficile infection (CDI) rates in the 12 mo
144 innate immune response to the resolution of Clostridium difficile infection (CDI) remains incomplete
150 of anaerobic microbiota during treatment of Clostridium difficile infection (CDI) than vancomycin an
151 crobiota transplant (FMT) is recommended for Clostridium difficile infection (CDI) treatment; however
154 are few high-quality studies of the costs of Clostridium difficile infection (CDI), and the majority
155 iable option for the treatment of refractory Clostridium difficile infection (CDI), and, more recentl
156 for the efficacy of probiotics in preventing Clostridium difficile infection (CDI), but guidelines do
157 (FT) is a promising treatment for recurrent Clostridium difficile infection (CDI), but its true effe
158 to increase the risk of community-associated Clostridium difficile infection (CDI), but the associati
159 polymorphism rs4073/-251T >A predisposes to Clostridium difficile infection (CDI), but this associat
160 catheter-associated urinary tract infection, Clostridium difficile infection (CDI), central line-asso
161 oratory tests are available for diagnosis of Clostridium difficile infection (CDI), delays in complet
162 effective alternative therapy for recurrent Clostridium difficile infection (CDI), is infrequently u
166 es have evaluated risk factors for recurrent Clostridium difficile infection (CDI), the vast majority
167 ens, including acute kidney injury (AKI) and Clostridium difficile infection (CDI), were also conside
194 trum antibiotic approved for Clostridioides (Clostridium) difficile infection (CDI) in adults, is ass
197 ed hospitalization, and hospitalization with Clostridium difficile infection [CDI]) were associated w
200 d dysbiosis is a key predisposing factor for Clostridium difficile infections (CDIs), which cause int
201 (IRR 0.79; 95% CI, 0.67-0.92; P = .004) and Clostridium difficile infections (CDIs; IRR 0.46; 95% CI
203 viously used to cure patients with recurrent Clostridium difficile infection, could also protect agai
204 n-resistant Staphylococcus aureus (MRSA) and Clostridium difficile infections declined across the UK
205 biotic-based strategies for the treatment of Clostridium difficile infections disrupt indigenous micr
206 currently used for diarrhea associated with Clostridium difficile infection, effectively resensitize
209 den of antimicrobial-resistant organisms and Clostridium difficile infections, halting unnecessary an
216 h care-onset health care facility-associated Clostridium difficile infection (HO-CDI) is overdiagnose
217 actam (PIP/TAZO) shortage and hospital-onset Clostridium difficile infection (HO-CDI) risk in 88 US m
219 been successfully applied to treat recurrent Clostridium difficile infection in humans, but a precise
223 recognition of several serious outbreaks of Clostridium difficile infection in the industrialized wo
226 antations for different disorders, including Clostridium difficile infection, irritable bowel syndrom
230 common misconceptions lead to misdiagnosis: Clostridium difficile infection is a possibility when th
234 eudomembranous enterocolitis associated with Clostridium difficile infection is an important cause of
236 om recent experiments on antibiotic-mediated Clostridium difficile infection is analyzed to quantify
244 Their use as probiotics for prevention of Clostridium difficile infection is prevalent among consu
254 or-associated complication or pneumonia, and Clostridium difficile infection; minor outcomes included
256 mine whether the reductions in recurrence of Clostridium difficile infection observed with fidaxomici
258 pment for therapy of systemic infections and Clostridium difficile infection of the gastrointestinal
260 reports found addressed the use of FMTs for Clostridium difficile infection or inflammatory bowel di
261 osure (OR 3.24; 95% CI, 2.99-3.52), and late Clostridium difficile infection (OR 1.60; 95% CI, 1.37-1
262 for the same infection, acute kidney injury, Clostridium difficile infection, or drug-related adverse
263 riptions) ADRs, with Clostridiodes (formerly Clostridium) difficile infections pivotal to its ADR pro
264 with their use including increased risk for Clostridium difficile infection, pneumonia, and fracture
265 dmission rates, central venous catheter use, Clostridium difficile infection rates, and hospital leng
271 ast 50 years for the treatment of refractory Clostridium difficile infections (RCDIs) in adults, it h
272 py, and frequency of complications including Clostridium difficile infection, readmission, and all-ca
273 e-level association test of the reduction in Clostridium difficile infection recurrence in patients t
274 le in developing the IDSA/SHEA guidelines on Clostridium difficile infection (see Wilcox and Planche'
276 treatment of portal systemic encephalopathy, Clostridium difficile infection, small bowel intestinal
277 Whereas many antibiotics increase risk of Clostridium difficile infection through dysbiosis, epide
278 treated population, the overall incidence of Clostridium difficile infection was 1.7% in the ertapene
280 as oral non-systemic antibacterial drugs for Clostridium difficile infections were active against pat
281 ation (FMT) is effective in the treatment of Clostridium difficile infection, where efficacy correlat
282 fective for treating patients with recurrent Clostridium difficile infection who are left with few cl
283 cohort of 109 subjects treated for recurrent Clostridium difficile infection with fecal microbiota tr
284 t serious cephalosporin-associated ADRs were Clostridium difficile infection within 90 days (0.91%),
285 acious and inexpensive therapy for recurrent Clostridium difficile infection, yet its safety is thoug