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1 (Mycobacterium tuberculosis) and diphtheria (Corynebacterium diphtheriae).
2 iphtheria is an infectious disease caused by Corynebacterium diphtheriae.
3 ection of exotoxin from toxigenic strains of Corynebacterium diphtheriae.
4 on of heme and hemoglobin as iron sources by Corynebacterium diphtheriae.
5 iron-dependent, negative global regulator in Corynebacterium diphtheriae.
6 respiratory disease caused by the bacterium Corynebacterium diphtheriae.
7 man pathogens Mycobacterium tuberculosis and Corynebacterium diphtheriae.
8 theria is a toxin-mediated disease caused by Corynebacterium diphtheriae.
9 ctrometry (MALDI-TOF MS) were conclusive for Corynebacterium diphtheriae.
10 e regulation of virulence and other genes in Corynebacterium diphtheriae.
11 idence for sortase reaction intermediates in Corynebacterium diphtheriae.
12 s associated with ferrous ion homeostasis in Corynebacterium diphtheriae.
13 nd reliable recognition of the toxin gene in Corynebacterium diphtheriae.
14 rimination as mammalian HO-1 and the HO from Corynebacterium diphtheriae.
15 tor that has been linked to the virulence of Corynebacterium diphtheriae.
16 th siderophore biosynthesis and transport in Corynebacterium diphtheriae.
17 r of iron homeostatic and virulence genes in Corynebacterium diphtheriae.
18 tional regulator of known virulence genes in Corynebacterium diphtheriae.
19 to mammalian HO than another studied HO from Corynebacterium diphtheriae.
20 atal respiratory disease caused by toxigenic Corynebacterium diphtheriae.
21 nt in the control of iron-sensitive genes in Corynebacterium diphtheriae.
22 transition metal ion-activated repressor in Corynebacterium diphtheriae.
23 of pathogens Mycobacterium tuberculosis and Corynebacterium diphtheriae.
24 tion from iron-regulated promoters (IRPs) in Corynebacterium diphtheriae.
25 in transport system previously identified in Corynebacterium diphtheriae.
26 ose obtained by nose swab to be positive for Corynebacterium diphtheriae.
27 is an acute, communicable disease caused by Corynebacterium diphtheriae.
28 based study of tonsillectomy and immunity to Corynebacterium diphtheriae (1931), 2 papers from a long
29 entifications for the clinically significant Corynebacterium diphtheriae (4 of 4) and Corynebacterium
31 xotoxin, diphtheria toxin (Dtx) expressed by Corynebacterium diphtheriae also can function as part of
35 cted defined deletions in the hmuO gene from Corynebacterium diphtheriae and Corynebacterium ulcerans
37 lus subunit SpaA of the SpaA-type pilus from Corynebacterium diphtheriae and FimA of the type 2 pilus
38 enase (HmuO) from the gram-positive pathogen Corynebacterium diphtheriae and for eukaryotic heme oxyg
39 strial production of amino acids, as well as Corynebacterium diphtheriae and Mycobacterium tuberculos
40 ally modified to diphthamide, the target for Corynebacterium diphtheriae and Pseudomonas aeruginosa t
41 totypical models, the heterotrimeric pili in Corynebacterium diphtheriae and the heterodimeric pili i
42 ogens such as Mycobacterium tuberculosis and Corynebacterium diphtheriae are characterized by their c
44 , the diphtheria toxin repressor (DtxR) from Corynebacterium diphtheriae, are iron-dependent regulato
46 actinomycetes Mycobacterium tuberculosis and Corynebacterium diphtheriae, but unlike the linear chrom
47 ease in diphtheria cases caused by toxigenic Corynebacterium diphtheriae (C diphtheriae) was reported
48 s old were immunized with either pentavalent Corynebacterium diphtheriae C7 (beta197) cross-reactive
49 nant-positive dtxR(E175K) mutant allele from Corynebacterium diphtheriae can be expressed in Mycobact
51 O, a heme degradation enzyme in the pathogen Corynebacterium diphtheriae, catalyzes the oxygen-depend
55 kDa protein secreted by lysogenic strains of Corynebacterium diphtheriae, causes the disease diphther
57 ionine sulfoxide reductase A of the pathogen Corynebacterium diphtheriae (Cd-MsrA) and shown that thi
61 oregulatory protein distantly related to the Corynebacterium diphtheriae diphtheria toxin repressor (
62 e, designated ScaR, with 26% identity to the Corynebacterium diphtheriae diphtheria toxin repressor (
65 nces which are homologous to elements of the Corynebacterium diphtheriae DtxR regulon, which controls
69 Mycobacterium smegmatis (EsxA and EsxB), and Corynebacterium diphtheriae (EsxA and EsxB) are heterodi
73 ethods for the molecular characterization of Corynebacterium diphtheriae has become a priority in ord
76 oxygenase gene from the pathogenic bacterium Corynebacterium diphtheriae has been subcloned and expre
78 to a scarcity of tools, genetic analysis of Corynebacterium diphtheriae has primarily relied on anal
79 oxygenase (HO) from the pathogenic bacterium Corynebacterium diphtheriae, have been investigated by (
84 es the expression of iron-sensitive genes in Corynebacterium diphtheriae, including the diphtheria to
85 has been the cornerstone of the treatment of Corynebacterium diphtheriae infection for more than 100
88 ideR, like DtxR, represses transcription of Corynebacterium diphtheriae iron-regulated promoters in
90 The diphtheria toxin repressor (DtxR) from Corynebacterium diphtheriae is a divalent metal-activate
93 rulent phenotype of the pathogenic bacterium Corynebacterium diphtheriae is conferred by diphtheria t
95 ependent transcriptional regulator DtxR from Corynebacterium diphtheriae is the prototype for a famil
96 The diphtheria toxin repressor (DtxR) from Corynebacterium diphtheriae is the prototypic member of
97 lar characterization of 53 U.S. and Canadian Corynebacterium diphtheriae isolates by multilocus enzym
99 is a hallmark of important pathogens (e.g., Corynebacterium diphtheriae, Mycobacterium tuberculosis)
100 mately 4% of them were infected by toxigenic Corynebacterium diphtheriae of both mitis and gravis bio
102 sion of safA from not only A. oris, but also Corynebacterium diphtheriae or Corynebacterium matruchot
106 overexpression of MdbA or a counterpart from Corynebacterium diphtheriae, rescues the Deltavkor mutan
107 Expression of HmuO, the heme oxygenase from Corynebacterium diphtheriae, restores iron and heme leve
108 n TRX-1 protects nematodes from infection by Corynebacterium diphtheriae, revealing the importance of
109 Numerous pathogenic bacteria, including Corynebacterium diphtheriae, Salmonella enterica, and Vi
112 y fatal infection mostly caused by toxigenic Corynebacterium diphtheriae strains and occasionally by
115 pidemic areas, and the emergence of epidemic Corynebacterium diphtheriae strains globally have highli
116 x) and its regulatory element (dtxR) from 72 Corynebacterium diphtheriae strains isolated in Russia a
118 The remaining eight (40.0%) were caused by Corynebacterium diphtheriae strains; six were biovar mit
119 global iron-dependent regulatory protein in Corynebacterium diphtheriae that controls gene expressio
120 ia toxin (DT) is an extracellular protein of Corynebacterium diphtheriae that inhibits protein synthe
121 tein is a global iron-dependent repressor in Corynebacterium diphtheriae that regulates transcription
122 production method for the fusion toxin using Corynebacterium diphtheriae that secretes fully folded,
123 Da protein, secreted by lysogenic strains of Corynebacterium diphtheriae, that causes the disease dip
130 ) and B (TcdB) and binary toxin (CDTa-CDTb), Corynebacterium diphtheriae toxin (DT), and Pseudomonas
131 hemical studies of a heterotrimeric pilus in Corynebacterium diphtheriae, uncovering the molecular sw
132 The diphtheria toxin repressor (DtxR) of Corynebacterium diphtheriae uses Fe(2+) as a corepressor
136 htheria toxin regulatory protein, DtxR, from Corynebacterium diphtheriae was identified from T. palli
138 ed to build the prototypical SpaA pilus from Corynebacterium diphtheriae We show that sortase-catalyz
139 ane, is caused by toxin-producing strains of Corynebacterium diphtheriae, with similar illness produc
140 18.5 days (95% CI, 17.7-19.4), and 95% clear Corynebacterium diphtheriae within 48 days (95% CI, 46-5
141 terval [CrI], 17.7-19.4 days), and 95% clear Corynebacterium diphtheriae within 48 days (95% CrI, 46-