ライフサイエンスコーパス: Cryptococcus

1 orphotype required for sexual development in Cryptococcus.

2 o have in-vitro and in-vivo activity against cryptococcus.

3 rine infection for the human fungal pathogen Cryptococcus.

4 n >10% of patients with KS, tuberculosis, or Cryptococcus.

5 shown in vitro and in vivo activity against cryptococcus.

6 pulation dynamics, and lineage divergence in Cryptococcus.

7 , 16% (4/25) for tuberculosis, 14% (1/7) for Cryptococcus, 10% (1/10) for Mycobacterium avium complex

8 Western blot analysis showed that Cryptococcus also induces phosphorylation of focal adhes

9 Cryptococcus amylolentus is the most closely known relat

10 e the related, but thermo-intolerant species Cryptococcus amylolentus to demonstrate that this respon

11 species complexes in the large fungal genus Cryptococcus and are responsible for potentially lethal

12 Invasive fungal diseases (IFD) caused by Cryptococcus and dimorphic fungi are associated with sig

13 Transcript leaders in Cryptococcus and other fungi are substantially longer an

14 d growth of other clinically-relevant yeast (Cryptococcus) and molds (Aspergillus) at a concentration

15 aths are attributable to species of Candida, Cryptococcus, and Aspergillus Treating fungal infections

16 cally relevant organisms, including Candida, Cryptococcus, and Aspergillus, in subjects with increasi

17 ens: Aspergillus, Blastomyces, Coccidioides, Cryptococcus, and Histoplasma species.

18 e host defense against Candida, Aspergillus, Cryptococcus, and others, with specific elements of the

19 Detection of Cryptococcus antigen (CrAg) is invaluable for establishi

20 Patients infected with the fungal pathogen Cryptococcus are most effectively treated with a combina

21 ptococcal meningitis places them at risk for Cryptococcus-associated immune reconstitution inflammato

22 e for the exploration of the relationship in Cryptococcus between cellular morphology and pathogenesi

23 Previous studies have demonstrated that Cryptococcus binding and invasion of human brain microva

24 ciated with host immune response rather than Cryptococcus burden.

25 Cryptococcus can cause meningoencephalitis (CM) among pr

26 of fungal antigens in body fluids, including cryptococcus capsular polysaccharide, histoplasma antige

27 Cryptococcus cells encounter high copper levels in the l

28 Here, we discovered that Cryptococcus cells polyploidize in response to genotoxic

29 xamined the relationship between rate of CSF Cryptococcus clearance (EFA) and mortality through 18 we

30 l trials, early fungicidal activity (EFA) of Cryptococcus clearance from cerebrospinal fluid (CSF) is

31 e aspirate of the chest lesions demonstrated Cryptococcus coinfection.

32 the recruitment of AIC components to nascent Cryptococcus-containing vacuoles (CnCVs) regulates the i

33 Cryptococcus contains more than 50 genes sharing high se

34 Here we demonstrate that two Cryptococcus copper transporters, Ctr1 and Ctr4, differe

35 Pichia angusta VKM Y-2559 and the oleaginous Cryptococcus curvatus VKM Y-3288 yeast cells were immobi

36 Using Cryptococcus deneoformans in a murine model of infection

37 me resequencing of the human fungal pathogen Cryptococcus deuterogattii identified an outbreak lineag

38 Here we show that resistance to 5FC in Cryptococcus deuterogattii is acquired more frequently i

39 The human fungal pathogen Cryptococcus deuterogattii is RNAi-deficient and lacks a

40 high rates of HIV-related mortality, yet the Cryptococcus factors influencing patient outcome are not

41 Cryptococcus gattii (Cg) infection emerged in British Co

42 enomenon in Cryptococcus neoformans (Cn) and Cryptococcus gattii (Cg), the significance of which has

43 Cryptococcus neoformans or, less frequently, Cryptococcus gattii are an important cause of mortality

44 Cryptococcus neoformans and Cryptococcus gattii are two species complexes in the lar

45 he prediction of Cryptococcus neoformans and Cryptococcus gattii in Europe is an important tool to un

46 erapy and management of complications due to Cryptococcus gattii infection in 86 Australian patients

47 Cryptococcus gattii is an emerging fungal pathogen on th

48 Cryptococcus gattii is an emerging intracellular pathoge

49 Cryptococcus gattii is responsible for a large outbreak

50 es of strains of Cryptococcus neoformans and Cryptococcus gattii isolated from serial episodes of cry

51 lecular types of the Cryptococcus neoformans/Cryptococcus gattii species complex that infect dogs and

52 lentulus and Neosartorya udagawae and yeast Cryptococcus gattii VGII (implicated in the outbreak in

53 la-zoster virus, and Cryptococcus neoformans/Cryptococcus gattii We describe a multicenter evaluation

54 Cryptococcus gattii, an emerging fungal pathogen in the

55 isease caused by Cryptococcus neoformans and Cryptococcus gattii.

56 bout the virulence of the molecular types in Cryptococcus gattii.

57 fungal species: Cryptococcus neoformans and Cryptococcus gattii.

58 significant evolutionary changes within the Cryptococcus genus, including transition from the ancest

59 pecies of Candida, Aspergillus, Exserohilum, Cryptococcus, Histoplasma, and Coccidioides, were studie

60 Cryptococcus pseudolongus, from the Cryptococcus humicola species complex, was more abundant

61 Heterologous expression of Cryptococcus HXS1 rendered the S. cerevisiae mutant lack

62 Fungal and bacteriological studies showed Cryptococcus in 64 (19.5%) patients, pneumococcus in 8 (

63 Rho pulldown assays revealed that Cryptococcus induces activation of three members of RhoG

64 Fungal culture or histopathology confirmed Cryptococcus infection for 20 patients (52.6%), and CrAg

65 Patient outcome following Cryptococcus infection is linked to initial fungal burde

66 hoproteomic analysis of the host response to Cryptococcus infection.

67 t common life-threatening diseases caused by Cryptococcus infection.

68 Cryptococcus is a major fungal pathogen that frequently

69 Cryptococcus is an emerging global health threat that is

70 hat temperature-dependent TE mobilization in Cryptococcus is an important mechanism that enhances mic

71 Cryptococcus is the most common cause of adult meningiti

72 Cryptococcus is the most common cause of meningitis in a

73 unistic infections, such as tuberculosis and cryptococcus, is crucial because of the high mortality a

74 Znf2, a master regulator of morphogenesis in Cryptococcus, is necessary and sufficient for the produc

75 those with negative CSF CrAg, 9% (5/54) had Cryptococcus isolated on CSF culture (n = 3) or PCR (n =

76 Cryptococcus isolates from 15 dogs and 27 cats were type

77 th molecular type, but the susceptibility of Cryptococcus isolates from dogs and cats is largely unkn

78 ts that triggered genomic changes within the Cryptococcus/Kwoniella species complex and may contribut

79 nsistent with the role of Fbp1 in regulating Cryptococcus-macrophage interaction and fungal virulence

80 llular proliferation after phagocytosis in a Cryptococcus-macrophage interaction assay, which likely

81 Interestingly, Cryptococcus-mediated IL-25 signaling suppressed the exp

82 133/153) specificity, while another study of Cryptococcus meningitis found 89% (69/78) sensitivity an

83 tory blood immune signature, possibly due to Cryptococcus modulation of the host immune response.

84 erable genetic diversity compared with other Cryptococcus molecular types and could be divided into t

85 Numerous Cryptococcus mRNAs encode predicted dual-localized prote

86 uconazole monotherapy was suboptimal despite Cryptococcus-negative CSF.

87 nfected with a moderately virulent strain of Cryptococcus neoformans (52D), which resulted in prolong

88 Cryptococcus neoformans (C. neoformans var. grubii) is a

89 rain - is a recently described phenomenon in Cryptococcus neoformans (Cn) and Cryptococcus gattii (Cg

90 Cryptococcus neoformans (Cn) is a common facultative int

91 Cryptococcus neoformans (Cn) is a deadly fungal pathogen

92 iodiomycete forming an induced DOPA-melanin, Cryptococcus neoformans (CN); and the slow-growing envir

93 Here, we focus on the Prp8 intein from Cryptococcus neoformans (Cne), a human fungal pathogen.

94 ith Pneumocystis jirovecii (pneumocystosis), Cryptococcus neoformans (cryptococcosis), Histoplasma ca

95 Isavuconazole showed good activities against Cryptococcus neoformans (MIC90, 0.12 mug/ml) and other n

96 s presenting with P. marneffei (n = 719) and Cryptococcus neoformans (n = 1598) infection to the Hosp

97 Cryptococcus neoformans admissions were not seasonal, an

98 es for melanization of the pathogenic fungus Cryptococcus neoformans also offers unique opportunities

99 d specificity were as follows: 100% each for Cryptococcus neoformans and C. gattii, 98.6% and 100% fo

100 ally expressed by the four main serotypes of Cryptococcus neoformans and C. gattii, is heterogeneousl

101 inent function in the capture of circulating Cryptococcus neoformans and Candida albicans, thereby re

102 e of macrophage autophagy in the response to Cryptococcus neoformans and Candida albicans, two import

103 ectivity for the Hsp90 isoforms expressed by Cryptococcus neoformans and Candida albicans, two pathog

104 ns (Aspergillus fumigatus, Candida albicans, Cryptococcus neoformans and Coccidioides immitis).

105 Cryptococcus neoformans and Cryptococcus gattii are two

106 Fundamental niche prediction of Cryptococcus neoformans and Cryptococcus gattii in Europ

107 ompare the molecular genotypes of strains of Cryptococcus neoformans and Cryptococcus gattii isolated

108 Cryptococcosis is a fungal disease caused by Cryptococcus neoformans and Cryptococcus gattii.

109 marily caused by two related fungal species: Cryptococcus neoformans and Cryptococcus gattii.

110 rabidopsis thaliana, Caenorhabditis elegans, Cryptococcus neoformans and Drosophila melanogaster.

111 erol endoperoxide, which were active against Cryptococcus neoformans and methicillin-resistance Staph

112 H)2) cell bias upon pulmonary infection with Cryptococcus neoformans and other non-T(H)2 stimuli.

113 in GM-CSF are susceptible to infections with Cryptococcus neoformans and other opportunistic fungi.

114 e infections with pathogenic species such as Cryptococcus neoformans Because the purine biosynthesis

115 Infection with Cryptococcus neoformans begins when desiccated yeast cel

116 se, and catalytically dead Tps2PD(D24N) from Cryptococcus neoformans bound to trehalose-6-phosphate (

117 ved (Blastomyces dermatitidis by culture and Cryptococcus neoformans by PLEX-ID).

118 Upon ingestion by macrophages, Cryptococcus neoformans can survive and replicate intrac

119 ificity of V region-identical IgE and IgA to Cryptococcus neoformans capsular polysaccharide and foun

120 Cryptococcus neoformans causes life-threatening meningit

121 Cryptococcus neoformans causes life-threatening meningoe

122 xin, Srx1, in oxidative stress resistance of Cryptococcus neoformans causing fungal meningoencephalit

123 n macrophages that had ingested live or dead Cryptococcus neoformans cells, or inert beads.

124 l-deficient mice have demonstrated increased Cryptococcus neoformans dissemination from lungs to brai

125 The human fungal pathogen Cryptococcus neoformans encodes many Cu-responsive genes

126 Eumelanins produced by pathogenic Cryptococcus neoformans fungi are virulence factors that

127 AfTOK1 (Aspergillus fumigatus), and H99TOK (Cryptococcus neoformans grubii) are K(+) -selective and

128 The fungus Cryptococcus neoformans has emerged as a major cause of

129 ot been studied in the human fungal pathogen Cryptococcus neoformans Here, we identified and characte

130 nistic fungal pathogens Candida albicans and Cryptococcus neoformans However, the molecular mechanism

131 e mAbs to the capsule of the fungal pathogen Cryptococcus neoformans impaired yeast budding by trappi

132 The burden of Cryptococcus neoformans in cerebrospinal fluid (CSF) pre

133 tes transmigration of the neurotropic fungus Cryptococcus neoformans into the brain parenchyma after

134 The mechanisms by which Cryptococcus neoformans invades the brain are largely un

135 Cryptococcus neoformans is a basidiomycete fungus that i

136 The fungal meningitis pathogen Cryptococcus neoformans is a central driver of mortality

137 Lethal disease caused by the fungus Cryptococcus neoformans is a consequence of the combined

138 Cryptococcus neoformans is a facultative intracellular f

139 Cryptococcus neoformans is a facultative intracellular p

140 Cryptococcus neoformans is a fatal fungal pathogen of hu

141 Cryptococcus neoformans is a fungal pathogen that causes

142 Cryptococcus neoformans is a fungal pathogen that kills

143 Cryptococcus neoformans is a fungal pathogen with a uniq

144 Cryptococcus neoformans is a fungal pathogen with worldw

145 Cryptococcus neoformans is a global human fungal pathoge

146 Cryptococcus neoformans is a human fungal pathogen that

147 Cryptococcus neoformans is a human fungal pathogen that

148 Cryptococcus neoformans is a pathogenic yeast capable of

149 Cryptococcus neoformans is a pathogenic yeast that can i

150 Cryptococcus neoformans is a significant fungal pathogen

151 Cryptococcus neoformans is a ubiquitous environmental fu

152 Cryptococcus neoformans is a ubiquitous, opportunistic f

153 Cryptococcus neoformans is an encapsulated fungal pathog

154 Cryptococcus neoformans is an encapsulated fungus that c

155 Cryptococcus neoformans is an important human, fungal pa

156 Cryptococcus neoformans is an opportunistic fungal patho

157 Cryptococcus neoformans is an opportunistic fungal patho

158 Cryptococcus neoformans is an opportunistic fungal patho

159 Cryptococcus neoformans is an opportunistic fungal patho

160 Cryptococcus neoformans is an opportunistic fungal patho

161 Cryptococcus neoformans is an opportunistic fungal patho

162 Cryptococcus neoformans is an opportunistic human pathog

163 Cryptococcus neoformans is an opportunistic pulmonary fu

164 Cryptococcus neoformans is an opportunistic yeast that i

165 Cryptococcus neoformans is an unconventional dimorphic f

166 ity to progressive infection with the fungus Cryptococcus neoformans is associated with an allergic p

167 Cryptococcus neoformans is the leading cause of death by

168 Cryptococcus neoformans is the most common cause of adul

169 d trait of the opportunistic fungal pathogen Cryptococcus neoformans is the production of melanin pig

170 C. glabrata, and 53 C. krusei isolates), 35 Cryptococcus neoformans isolates, and 191 other clinical

171 virulence factor laccase to the cell wall in Cryptococcus neoformans Laccase activity was perturbed,

172 on doubled the time-to-endpoint of mice with Cryptococcus neoformans meningitis.

173 Numerous virulence factors expressed by Cryptococcus neoformans modulate host defenses by promot

174 rable to infection by the encapsulated yeast Cryptococcus neoformans Most commonly found in the envir

175 The pathogenic fungus Cryptococcus neoformans must adapt to glucose-limited co

176 The pathogenic fungus Cryptococcus neoformans must overcome multiple stressors

177 of 1201 signature-tagged deletion strains of Cryptococcus neoformans mutants to identify previously u

178 CaTOK (Candida albicans) and CnTOK (Cryptococcus neoformans neoformans) pass both K(+) and N

179 seminated infections with the fungal species Cryptococcus neoformans or, less frequently, Cryptococcu

180 The Cryptococcus neoformans polysaccharide capsule is a well

181 xylomannan (GXM), the major component of the Cryptococcus neoformans polysaccharide capsule, hydrolyz

182 The opportunistic fungal pathogen Cryptococcus neoformans possesses a polysaccharide capsu

183 The pathogenic fungus Cryptococcus neoformans produces melanin within its cell

184 The pathogenic fungus Cryptococcus neoformans produces PGE2, and we found that

185 The basidiomycete fungal pathogen Cryptococcus neoformans requires the PUF protein, Pum1,

186 of C57BL/6 mice with the moderately virulent Cryptococcus neoformans strain 52D models the complex ad

187 murine model of C57BL/6J mice infected with Cryptococcus neoformans strain 52D.

188 ential of these genes by comparing wild-type Cryptococcus neoformans strain H99 with deletant and com

189 yptococcal meningitis patients infected with Cryptococcus neoformans strains with the same multilocus

190 oducibility strains (4 Candida species and 6 Cryptococcus neoformans strains), and 746 isolates of Ca

191 otective immune responses to highly virulent Cryptococcus neoformans strains, such as H99, are associ

192 etrotransposons in the human fungal pathogen Cryptococcus neoformans The resulting DSBs were repaired

193 In order for Cryptococcus neoformans to invade the central nervous sy

194 ability of the opportunistic fungal pathogen Cryptococcus neoformans to resist oxidative stress is on

195 Cryptococcus neoformans var. grubii is the causative age

196 Compared to other well-studied Cryptococcus neoformans virulence factors such as the po

197 the cell wall of the human pathogenic fungus Cryptococcus neoformans We observed that melanin is asse

198 ns, Candida krusei, Candida parapsilosis and Cryptococcus neoformans were investigated.

199 ave directly correlated phenotypic traits of Cryptococcus neoformans with clinical outcome of infecte

200 ) exhibit potent antifungal activity against Cryptococcus neoformans with high selectivity.

201 of lipid droplets during the interaction of Cryptococcus neoformans with macrophages in the presence

202 vestigated the outcome of the interaction of Cryptococcus neoformans with murine macrophages using la

203 rcular (Histoplasma capsulatum) to punctate (Cryptococcus neoformans) to labeling at the bud sites (C

204 relevant yeast species (Candida glabrata and Cryptococcus neoformans), is shown.

205 a spp., 146 from 9 Aspergillus spp., 84 from Cryptococcus neoformans, 40 from 23 other mold species,

206 trophils have been shown to efficiently kill Cryptococcus neoformans, a causative agent of meningoenc

207 Human infection with Cryptococcus neoformans, a common fungal pathogen, follo

208 c functional analysis of the phosphatases in Cryptococcus neoformans, a fungal pathogen that causes l

209 Here, we demonstrate that Cryptococcus neoformans, a model eukaryotic pathogen, re

210 One such organism is Cryptococcus neoformans, a pathogenic yeast that causes

211 It has shown potent activity against Cryptococcus neoformans, a yeast that can affect immunoc

212 ons against Candida albicans, Candida auris, Cryptococcus neoformans, and Aspergillus fumigatus (Sigm

213 The fungal pathogens Candida albicans, Cryptococcus neoformans, and Aspergillus fumigatus have

214 sential for virulence of the fungal pathogen Cryptococcus neoformans, and bacterial P5CDHs have been

215 hly active against a second fungal pathogen, Cryptococcus neoformans, and moderately active against a

216 In the yeast Cryptococcus neoformans, CG methylation occurs in transp

217 n of clinically significant Candida species, Cryptococcus neoformans, Histoplasma capsulatum, and Bla

218 l pathogens of environmental origin, such as Cryptococcus neoformans, is their ability to adapt to ma

219 mune response against the pathogenic fungus, Cryptococcus neoformans, is unknown.

220 Compared to Cryptococcus neoformans, little is known about the virul

221 e fungal pathogens Aspergillus fumigatus and Cryptococcus neoformans, little was known about their in

222 Pathogens included Balamuthia mandrillaris, Cryptococcus neoformans, lymphocytic choriomeningitis vi

223 In the pathogenic fungus Cryptococcus neoformans, noncanonical Gbeta Gib2 promote

224 on and analysis of SRP in the human pathogen Cryptococcus neoformans, providing the first description

225 he major human opportunistic fungal pathogen Cryptococcus neoformans, remains unknown.

226 Candida albicans, Aspergillus fumigatus and Cryptococcus neoformans, result in more deaths annually

227 Cryptococcus neoformans, the causative agent of cryptoco

228 pathogens Candida albicans, C. glabrata and Cryptococcus neoformans, the food spoilage organism Zygo

229 In the human fungal pathogen Cryptococcus neoformans, the homeodomain transcription f

230 Finally, in Cryptococcus neoformans, the loss of a cytosine DNA meth

231 Cryptococcus neoformans, the predominant etiological age

232 In the human fungal pathogen Cryptococcus neoformans, the Rim101 protein retains cons

233 Using the yeast Cryptococcus neoformans, we describe a mechanism by whic

234 sis is important for virulence of the fungus Cryptococcus neoformans, which can cause lethal meningoe

235 ypic diversity in the human pathogenic yeast Cryptococcus neoformans, which is globally distributed a

236 fection with environmental organisms such as Cryptococcus neoformans, which require no host in their

237 Candida species, Aspergillus fumigatus, and Cryptococcus neoformans.

238 sponse to infection with the fungal pathogen Cryptococcus neoformans.

239 nsing pathway in the human pathogenic fungus Cryptococcus neoformans.

240 t regulates virulence in the fungal pathogen Cryptococcus neoformans.

241 IP5/IP7, which is essential for virulence of Cryptococcus neoformans.

242 central role in regulating the virulence of Cryptococcus neoformans.

243 d with the distantly related fungal pathogen Cryptococcus neoformans.

244 of H3K27 methylation (H3K27me) in the yeast Cryptococcus neoformans.

245 nown whether such an association exists with Cryptococcus neoformans.

246 for virulence of the human pathogenic fungus Cryptococcus neoformans.

247 th a similar function in the related species Cryptococcus neoformans.

248 mall-RNA biogenesis in the pathogenic fungus Cryptococcus neoformans.

249 ual development of the human fungal pathogen Cryptococcus neoformans.

250 ntainment following pulmonary challenge with Cryptococcus neoformans.

251 C57BL/6 mice after pulmonary infection with Cryptococcus neoformans.

252 pC) and with the LPMO-like protein Bim1 from Cryptococcus neoformans.

253 al3b (CNAG_07348) from the pathogenic fungus Cryptococcus neoformans.

254 Molecular types of the Cryptococcus neoformans/Cryptococcus gattii species comp

255 an parechovirus, varicella-zoster virus, and Cryptococcus neoformans/Cryptococcus gattii We describe

256 ction with the opportunistic fungal pathogen Cryptococcus neoformans; however, the role of plasmacyto

257 molecule and the preferred carbon source for Cryptococcus, plays a critical role in fungal developmen

258 ics and outcomes of persons with HIV-related Cryptococcus presenting with higher CD4 counts.

259 Cryptococcus pseudolongus, from the Cryptococcus humicol

260 h from infection (including tuberculosis and cryptococcus) shortly after the initiation of antiretrov

261 MICs varied with Cryptococcus species and molecular type in dogs and cats

262 Cryptococcus species are associated with invasive fungal

263 Cryptococcus species are known agents of opportunistic i

264 bisexual and unisexual cycles of pathogenic Cryptococcus species are stimulated by properties associ

265 Cryptococcus species are yeastlike fungi that cause leth

266 We evaluated Cryptococcus species as a cause of acute respiratory inf

267 Species within the human pathogenic Cryptococcus species complex are major threats to public

268 Recent genomic studies in the Cryptococcus species complex revealed that chromosome tr

269 sely known related species of the pathogenic Cryptococcus species complex, and it is non-pathogenic.

270 We sequenced isolates from two Cryptococcus species complexes, C. gattii and C. neoform

271 Additionally, while pathogenic Cryptococcus species have bipolar mating systems with a

272 The yeast genera Kondoa might be protective; Cryptococcus species might also affect asthma severity.

273 etween C. amylolentus and related pathogenic Cryptococcus species provide evidence that multiple chro

274 e evolution of mating and MAT in this genus, Cryptococcus species provide key insights into the evolu

275 Cryptococcus species utilize a variety of sexual reprodu

276 Cryptococcus species were the most common pathogens dete

277 to bipolar mating systems in the pathogenic Cryptococcus species, as well as its possible link with

278 led that, similar to those of the pathogenic Cryptococcus species, C. amylolentus has regional centro

279 T locus that is now extant in the pathogenic Cryptococcus species.

280 g peptide-MHC class II molecules to identify Cryptococcus-specific Treg cells combined with genetic f

281 age-gated K(v) channel subunits are found in Cryptococcus spp.

282 ght patients received ISAV for IFD caused by Cryptococcus spp. (n = 9), Paracoccidioides spp. (n = 10

283 Cryptococcus spp. cause fungal meningitis, a life-threat

284 though the FDA black box warning states that Cryptococcus spp. do not make (1,3)-beta-d-glucan, the c

285 Heteroresistance was detectable in Cryptococcus spp. in the CSF of all patients at baseline

286 Total and resistant subpopulations of Cryptococcus spp. were quantified directly from patient

287 of 28 fungal strains including Candida spp., Cryptococcus spp., Aspergillus spp., and Fusarium spp.

288 The genomes of Aspergillus spp. and Cryptococcus spp., but not those of S. cerevisiae or the

289 he Madison Chamber during exposure can alter Cryptococcus survival and dose retained in mice.

290 cerebrospinal fluid (CSF) clearance rate of cryptococcus, termed early fungicidal activity, measured

291 transporter-like proteins (Hxs1 and Hxs2) in Cryptococcus that share the highest sequence identity wi

292 Upon pulmonary infection with Cryptococcus, Treg cells accumulated in the lung parench

293 Here we describe the first case of Cryptococcus uzbekistanensis causing bone marrow infecti

294 rationale to support continued investment in Cryptococcus vaccine research, potential challenges that

295 hat an F-box protein, Fbp1, is essential for Cryptococcus virulence independent of the classical viru

296 transporters is induced and is critical for Cryptococcus virulence.

297 of the peripheral CD4(+) T-cell response to Cryptococcus was associated with disease severity and ou

298 ) and 5-fluorocytosine (5FC) were found when Cryptococcus was incubated at 37 degrees compared to 30

299 We tested this concept in two pathogenic Cryptococcus yeast species by genome-wide mapping of tra

300 nce procedures exist for Candida species and Cryptococcus yeasts; however, no standardized methods ha