ライフサイエンスコーパス: Cushing syndrome

1 nd syndromes including Addison's disease and Cushing syndrome.

2 thy and were less likely to develop signs of Cushing syndrome.

3 bone density reported here parallel those of Cushing syndrome.

4 lowed by resolution of signs and symptoms of Cushing syndrome.

5 methasone (Dex) for 14 d provided a model of Cushing syndrome.

6 tions and leads to adrenocortical tumors and Cushing syndrome.

7 causes of primary hyperaldosteronism and the Cushing syndrome.

8 dular adrenal hyperplasia (PMAH), a cause of Cushing syndrome.

9 gery for a lung neuroendocrine tumor causing Cushing syndrome.

10 a greater index of suspicion for subclinical Cushing syndrome.

11 s cortisol secretion (MACS) without signs of Cushing syndrome.

12 a previously unidentified approach to treat Cushing syndrome.

13 ypertension, myopathy, and dermal atrophy of Cushing syndrome.

14 roidism (2.58; 1.35 to 4.92; p-value 0.004), Cushing syndrome (1.97; 1.06 to 3.65; p-value 0.03), Cro

15 gnificantly higher prevalence of subclinical Cushing syndrome (21.7% [5 of 23] vs 6.2% [7 of 112]) (P

16 , weight gain (4% in MCALP vs 19% in FP) and Cushing syndrome (3% in MCALP vs 18% in FP).

17 opathy, myopathy, polyneuropathy, and pseudo-Cushing syndrome, among others.

18 ore likely to be associated with subclinical Cushing syndrome and less likely to be pheochromocytomas

19 three men, nine women) with ACTH-independent Cushing syndrome and with bilateral nonpigmented multino

20 Skin and cardiac myxomas, Cushing syndrome, and acromegaly were present in 62, 30,

21 hormone excess, like primary aldosteronism, Cushing syndrome, and pheochromocytoma, claim thousands

22 c hormone secretion from lung tumors causing Cushing syndrome are associated with high rates of morbi

23 Evaluation of patients with possible Cushing syndrome begins with ruling out exogenous steroi

24 Management of Cushing syndrome begins with surgery to remove the sourc

25 uishes such patients from those who have the Cushing syndrome caused by other primary adrenal disorde

26 Cushing syndrome characteristically presents with skin c

27 d and endogenous glucocorticoid (GC) excess, Cushing syndrome, create a significant health burden.

28 Cushing syndrome (CS) is a constellation of signs and sy

29 >138 nmol/L and absence of typical clinical Cushing syndrome [CS] features, definitive MACS [MACS-2]

30 it memory deficits have been demonstrated in Cushing syndrome, depression, and posttraumatic stress d

31 in approximately 60% to 70% of patients with Cushing syndrome due to endogenous cortisol production.

32 nous steroid use, the estimated incidence of Cushing syndrome due to endogenous overproduction of cor

33 First-line therapy for Cushing syndrome due to endogenous overproduction of cor

34 The incidence of Cushing syndrome due to endogenous overproduction of cor

35 Pediatric endogenous Cushing syndrome (eCs) is mainly caused by pituitary cor

36 roendocrine pulmonary tumors associated with Cushing syndrome had increased nodal metastasis, higher

37 Until recently, the prevalence of endogenous Cushing syndrome has been considered to be low.

38 nally, in subcutaneous AT from patients with Cushing syndrome, higher VEGFA expression was associated

39 ed AEs (HR, 2.2; 95% CI, 1.7-2.8), including Cushing syndrome (HR, 11.8; 95% CI, 1.4-97.2), hyperthyr

40 ed nodular adrenocortical disease causes the Cushing syndrome in children and young adults and is mos

41 We investigated the genetic source of Cushing syndrome in individuals with adrenocortical hype

42 Finally, Armc5 haploinsufficiency leads to Cushing syndrome in mice, but only later in life, and th

43 ypokalemia (in patients with aldosteronoma), Cushing syndrome (in the patient with cortisol-secreting

44 Cushing syndrome is associated with hyperglycemia, prote

45 Cushing syndrome is defined as a prolonged increase in p

46 Although the most frequent cause of Cushing syndrome is exogenous steroid use, the estimated

47 ssive glucocorticoid hormone, as occurs with Cushing syndrome, is known to be associated with altered

48 types of adrenocortical tumors that lead to Cushing syndrome may be caused by aberrant cyclic AMP (c

49 lysis, age, stage, presence of virilization, Cushing syndrome, or hypertension, germline TP53 status,

50 at ranges from rare clinically overt adrenal Cushing syndrome to the much more prevalent mild autonom

51 in states of glucocorticoid excess, such as Cushing syndrome, which are associated with frank dyslip

52 the evidence for hippocampal atrophy in (1) Cushing syndrome, which is characterized by a pathologic

53 AIMAH is a rare cause of ACTH-independent Cushing syndrome, with characteristic CT findings of mas