ライフサイエンスコーパス: D-glucose

1 cid and phospholipid biosynthesis from (13)C D-glucose.

2 ounds in 16-21 % overall yield starting from D-glucose.

3 dynamic PET imaging of [(18)F]fluoro-2-deoxy-D-glucose.

4 T5, as well as a XylE mutant that transports D-glucose.

5 either [(13)C(6)]-myo-inositol or [(13)C(6)]-D-glucose.

6 ugar, 4,6-dideoxy-4-(3-hydroxybutanoylamino)-D-glucose.

7 ters that are insensitive to the presence of D-glucose.

8 l tissues were measured using [(3)H]-2-deoxy-D-glucose.

9 lytic inhibitor WP1122, a prodrug of 2-deoxy-d-glucose.

10 sence of the glycosylation inhibitor 2-deoxy-d-glucose.

11 zation of [U-(13) C(6) ,1,2,3,4,5,6,6-d(7) ]-d-glucose.

12 dTDP-3-amino-2,3,6-trideoxy-4-keto-3-methyl-D-glucose.

13 suspension of quiescent E. coli metabolizing d-glucose.

14 pSenLys cells that synthesized l-lysine from d-glucose.

15 id, L-arabinose, L-rhamnose, D-galactose and D-glucose.

16 h a 1.7% overall yield from d-cellobiose and d-glucose.

17 ate cleavage reactivity toward l-fucose over d-glucose.

18 ntial phosphoryl transfer steps using a beta-D-glucose 1,6-bisphosphate (betaG16BP) intermediate.

19 e ribose 5-phosphate and the activator alpha-D-glucose 1,6-bisphosphate (glucose 1,6-bisphosphate), a

20 se (betaPGM) catalyzes isomerization of beta-D-glucose 1-phosphate (betaG1P) into D-glucose 6-phospha

21 e evaluated as potential inhibitors of alpha-D-glucose 1-phosphate thymidylyltransferase (Cps2L), the

22 e phosphorolysis of GDP-D-glucose to GDP and D-glucose 1-phosphate.

23 cultured cells with 2-deoxy-2-[(14)C]carbon-D-glucose ([(14)C]2DG) support at least 35% higher [(18)

24 ografts in mice using 2-deoxy-2-[F-18]fluoro-D-glucose ((18)F-FDG) PET imaging.

25 actate production and (18)F-2-fluoro-2-deoxy-D-glucose ((18)F-FDG) uptake.

26 er clinical trial using (18)F-fluoro-2-deoxy-d-glucose ((18)F-FDG), (18)F-fluoromisonidazole ((18)F-F

27 The PET tracers 2-(18)F-fluoro-2-deoxy-d-glucose ((18)F-FDG), 3'-deoxy-3'-(18)F-fluorothymidine

28 elling cells with 2'-[(18)F]-fluoro-2'-deoxy-D-glucose ((18)F-FDG).

29 pic imaging (MRSI) and [(18)F]fluoro-2-deoxy-D-glucose ((18)FDG) positron emission tomography (PET) t

30 emission tomography (PET) and [(18)F]fluoro-D-glucose ((18)FDG) to measure brain glucose metabolism

31 The glucose analog [(18)F]fluoro-2-deoxy-2-d-glucose ([(18)F]-FDG) is commonly used in PET/CT that

32 ted macrophages with 2-deoxy-2-[(18)F]fluoro-D-glucose ([(18)F]FDG) has been proposed for identificat

33 ured BAT activity by 2-deoxy-2-[(18)F]fluoro-d-glucose ([(18)F]FDG) positron emission tomography/comp

34 d 15% per MBq/ml of 2-deoxy-2-[(18)F]-fluoro-d-glucose ([(18)F]FDG).

35 20 years after Fischer's first synthesis of (D)-glucose (1890), we are witnessing important developme

36 vity as measured by [18F]-2-fluoro-d-2-deoxy-d-glucose (18F-FDG) PET/CT.

37 tol (1-[(18)F]FDAM), 2-deoxy-2-[(18)F]fluoro-d-glucose (2-[(18)F]FDG), and 6-deoxy-6-[(18)F]fluoro-d-

38 The combination of 2-deoxy-D-glucose (2-DG) and UA-4 induced cell cycle arrest in G

39 The glucose analog 2-deoxy-D-glucose (2-DG) mimics CR effects in several animal mod

40 ex 1 and is efficiently inhibited by 2-deoxy-d-glucose (2-DG) or by glucose starvation.

41 Inhibition of glycolysis with 2-deoxy-D-glucose (2-DG) reduced osteoclast formation and activi

42 eted drugs (MTD) in combination with 2-deoxy-d-glucose (2-DG), a compound that inhibits glycolysis.

43 apy using periocular carboplatin and 2-deoxy-d-glucose (2-DG).

44 2-Deoxy-2-(18)F-fluoro-d-glucose (2-FDG) with PET is undeniably useful in the c

45 2-Deoxy-2-[18F]fluoro-D-glucose (2-FDG) with positron emission tomography (2-F

46 SGLTs and GLUTs; and 2-deoxy-2-[F-18]-fluoro-d-glucose (2-FDG), a substrate for GLUTs.

47 obenz-2-oxa-1, 3-diazol-4-yl) amino]-2-deoxy-D-glucose (2-NBDG) reports on glucose uptake and Tetrame

48 1) pivotal for intestinal mass absorption of d-glucose, 2) triggers the glucose-induced secretion of

49 Cs) treated with DL-Hcy (500 micromol/L) and d-glucose (25 mmol) for 48 h.

50 cemia and to a blunted CRR caused by 2-deoxy-d-glucose (2DG) administration.

51 with low doses of the glucose analog 2-deoxy-d-glucose (2DG) on ADPKD progression in orthologous and

52 ither been treated or untreated with 2-deoxy-d-glucose (2DG), a pharmaceutical that targets cell meta

53 ycolysis and N-linked glycosylation, 2-deoxy-D-glucose (2DG), potently inhibited surface expression o

54 ples were collected from control and 2-deoxy-d-glucose (2DG)-injected rats for Western blot analysis

55 zole (NDI) with glycolytic inhibitor 2-deoxy-d-glucose (2DG).

56 ivated by glucoprivation, induced by 2-deoxy-D-glucose (2DG).

57 Concurrent inhibition of glycolysis (2-deoxy-D-glucose, 2DG) and mitochondrial respiration (rotenone)

58 iosynthetic enzyme (rmlC; TDP-4-keto-6-deoxy-d-glucose 3,5-epimerase), the ATP binding cassette (ABC)

59 substrate for SGLTs; 4-deoxy-4-[F-18]-fluoro-d-glucose (4-FDG), a substrate for SGLTs and GLUTs; and

60 -6-phosphate into mixtures of labeled methyl d-glucose-4,6-phosphates, which were analyzed by (31)P N

61 ical allosteric mechanism in human UDP-alpha-d-glucose 6-dehydrogenase (hUGDH) based on an easily acq

62 of beta-D-glucose 1-phosphate (betaG1P) into D-glucose 6-phosphate (G6P) via sequential phosphoryl tr

63 olites, adenosine 5'-monophosphate (AMP) and d-glucose 6-phosphate (Glucose-P), were detected in a su

64 ent equatorial transamination of 3-oxo-alpha-D-glucose 6-phosphate to form alpha-D-kanosamine 6-phosp

65 (2-[(18)F]FDG), and 6-deoxy-6-[(18)F]fluoro-d-glucose (6-[(18)F]FDG) was studied in EMT6 cells, tumo

66 her non-specific glycolytic proteins such as d-glucose-6-phosphate dehydrogenase.

67 in combination with conversion of the formed d-glucose-6-phosphate into mixtures of labeled methyl d-

68 othelial cell (HUVECs), challenged with high D-glucose (60% inhibition), hydrogen peroxide (80% inhib

69 cose (UDP-viosamine) from UDP-4-keto-6-deoxy-D-glucose, a key compound involved also in the biosynthe

70 rystalline form 3-nitro-3,4,6-trideoxy-alpha-D-glucose, a nitro sugar stereochemically homologous to

71 nvolved in the synthesis of 1-O-galloyl-beta-d-glucose, a precursor for the synthesis of hydrolysable

72 Because intestinal D-glucose absorption is mediated by SGLT1 localized in t

73 In wild-type mice, passage of D-glucose across the intestinal BBM was predominantly me

74 The comparison of growth on L-fucose and D-glucose allows first insights into the genome-wide cha

75 In contrast, in GA cockroaches, D-glucose also stimulated bitter-GRNs and suppressed the

76 The insulin secretagogue D-glucose also stimulates beta-cell p38 MAPK phosphoryla

77 that varying the position of substitution of d-glucose alters not only the cellular uptake and cytoto

78 Using 2-deoxy-d-glucose, an inhibitor of glucose uptake, and compound

79 enumber shifts were 50cm(-1)/mM obtained for d-(+)-glucose and 96cm(-1)/mug/mL for Cy5-conjungated Ra

80 e gut microbiota metabolites modulated (14)C-D-glucose and (14)C-deoxy-D-glucose uptake into hepatic

81 s an unbranched heteropolymer with repeating d-glucose and 6-deoxy-l-talose residues in which the 6-d

82 phase diagrams of the food ingredients alpha-d-glucose and citric acid, along with sodium sulfate, we

83 Isotopic studies with (13)C(6)-d-glucose and D(2)O unambiguously confirmed the source o

84 e real-time monitoring of sucrose, sorbitol, d-glucose and d-fructose concentrations gave unique resu

85 r real-time monitoring of sucrose, sorbitol, d-glucose and d-fructose concentrations is reported.

86 tyl-D-glucosamine, N-acetyl-D-galactosamine, D-glucose and D-galactose, present on the cell surface.

87 ting that the tricyclic core is derived from d-glucose and d-ribose, whereas the tiglyl moiety is der

88 asons hampering simultaneous fermentation of D-glucose and D-xylose, two primary sugars present in li

89 lucose negatively affected transport of both D-glucose and D-xylose.

90 ich is selectively fueled by the presence of D-glucose and ethyl butyrate.

91 Insulin secretion induced by d-glucose and forskolin is amplified by overexpressing i

92 1,3-dioxane derivative was synthesized from d-glucose and found to be a highly stereoselective templ

93 ristic structures in melanoidins formed from d-glucose and l-alanine between 130 and 200 degrees C.

94 ompared by detecting two different analytes: d-glucose and l-cysteine under nonspecific and specific

95 There is no interference from d-glucose and l-glutamic acid, ascorbic acid and o-nitro

96 he elaboration of biosensors for D-sorbitol, D-glucose and L-lactate with using D-sorbitol dehydrogen

97 occluded conformations present multiple beta-d-glucose and maltose interaction sites, whereas inward-

98 clearance of non-metabolizable [(3) H]methyl-d-glucose and placental SLC2A8 (glucose transporter 8) g

99 composed of two units of beta-(1-->6)-linked d-glucose and resembles the carbohydrate moiety of lipid

100 hat secrete insulin/C-peptide in response to D-glucose and theophylline.

101 se, or d-galactose (D-Gal) are replaced with d-glucose and/or l-rhamnose.

102 We monitor environmentally (2-deoxy-D-glucose) and genetically (DeltaPFK2) perturbed Sacchar

103 conditions mimicking diabetes mellitus (high D-glucose) and ischemia-associated starvation (low growt

104 oice between metabolizable sugar (sucrose or D-glucose) and nonmetabolizable (zero-calorie) sugar (su

105 rmer presents overlapping endofacial WZB117, d-glucose, and CB binding envelopes.

106 ncluding L-arabinose, D-fucose, D-galactose, D-glucose, and D-xylose.

107 levels of O-ethyl beta-d-glucopyranoside and d-glucose, and lower levels of malic, quinic and ascorbi

108 d-glucosamine versus 2,3-diamino-2,3-dideoxy-d-glucose, and phosphorylation status all correlated wit

109 A-RFs are filled with different solutions of d-glucose, and the Deltalambdapeak is measured in real t

110 te between lactulose, l-rhamnose, 3-O-methyl-d-glucose, and xylose.

111 For example, metabolisms viable on D-glucose are 1835 times more likely to give rise to met

112 e details of the ROA spectrum of methyl-beta-D-glucose are found to be highly sensitive to solvation

113 lacental uptake and clearance of (3)H-methyl-D-glucose at D19.

114 h dynamic PET imaging of [18F]fluoro-2-deoxy-D-glucose at two occasions with 24-hour interval between

115 polymerizations (ROP) of six-membered cyclic d-glucose-based carbonates was found to result in signif

116 fter an injection of [(18)F]2-fluoro-2-deoxy-d-glucose before the OGTT, and the rate of glucose absor

117 ted that the major neutral sugars were alpha-D-glucose, beta-D-glucose, rhamnose and D-glucuronic aci

118 er Bragg gratings (TFBGs) functionalized for D-glucose biosensing through polydopamine (PDA)-immobili

119 antiomeric block co-beta-peptide, poly(amido-D-glucose)-block-poly(beta-L-lysine), with high yield an

120 ynamic [(15)O]H2O and [(18)F]-fluoro-2-deoxy-d-glucose brain positron emission tomography scans to me

121 ucose metabolism, as measured by [18F] deoxy-D-glucose brain positron emission tomography.

122 esigned and synthesized with mono- (BTA-beta-d-glucose; BTA-Glc and BTA-alpha-d-mannose; BTA-Man) or

123 s likely that alterations sterically prevent D-glucose but not D-xylose from entering the pocket.

124 observed that platelet exposure to 25 mmol/L d-glucose, but not to iso-osmolar mannitol, 1) reduced t

125 with D-ribo configuration, was prepared from D-glucose by inverting the C-3 stereocenter to introduce

126 PET with [18F]fluoro-2-deoxy-D-glucose can be used to image cellular metabolism, whic

127 PET with [(18)F]fluoro-2-deoxy-D-glucose can be used to image cellular metabolism, whic

128 eutron scattering (SANS) experiments of poly(d-glucose carbonate) block copolymers in solution that e

129 was observed for naturally derived poly(4,6-d-glucose carbonate)s (PGCs) containing carbonate side c

130 accharides (L-arabitol, D-fructose, sucrose, D-glucose, cellotetraose, cellulose, and starch) were tr

131 this work, two DESs have been characterized: d-glucose:choline chloride:water (GCH) and d-glucose:cit

132 : d-glucose:choline chloride:water (GCH) and d-glucose:citric acid:water (GCiH).

133 19, but not D16, transplacental (3) H-methyl-d-glucose clearance was reduced by 33% in corticosterone

134 d increase in food consumption, (3) H-methyl-d-glucose clearance was similar to the controls.

135 ication process of a functional human sodium/D-glucose co-transporter 1 (hSGLT1) in Pichia pastoris a

136 ransduction, develop a robust preference for d-glucose compared with isocaloric l-serine independentl

137 The beta-d-glucose-containing compound 3, bearing 2-chlorothiophe

138 shown to induce downregulation of the sodium-D-glucose cotransporter 1 (SGLT1) and of the concentrati

139 Na(+)-d-glucose cotransporter 1 (SGLT1) is rate-limiting for g

140 like hRS1-Reg(S20E) and whether human Na(+)-d-glucose cotransporter hSGLT2 and the human glucose sen

141 lates the plasma membrane abundance of Na(+)-d-glucose cotransporter SGLT1 by blocking the exocytotic

142 To clarify the physiological role of Na(+)-D-glucose cotransporter SGLT1 in small intestine and kid

143 found were d-xylose, d-galactose, d-mannose, d-glucose, d-arabinose, d-rhamnose and d-glucuronic acid

144 To address this question, d-glucose, d-fructose and l-ascorbic acid were incubated

145 formed from different carbohydrates, such as d-glucose, d-fructose, and d-xylose, and their typical d

146 biologically relevant underivatized hexoses, d-glucose, d-galactose, d-mannose, and d-fructose, using

147 esis of beta-C-glycopyranosyl aldehydes from D-glucose, D-mannose, and D-galactose.

148 dition of a flavour enhancer solution (FES) (d-glucose, d-ribose, l-cysteine and thiamin) and of sous

149 hough biomass-derived carbohydrates (such as D-glucose, D-xylose and D-galactose) are extracted on co

150 lactate with using D-sorbitol dehydrogenase, D-glucose dehydrogenase and L-lactate dehydrogenase resp

151 nd an inhibitor of energy metabolism 2-deoxy-D-glucose (DeOGlc) + sodium iodoacetate (IAc), on the tr

152 d-glucose-dependent suppression of BKCa channel beta1 su

153 aminocyclization/lactamization of d-mannose/D-glucose derived C5-gamma-azido esters as a key step wh

154 hly diastereoselective Barbier reaction on a d-glucose-derived aldehyde.

155 an lead to inaccuracies in amperometric beta-D-glucose determinations.

156 1.5 uM starch plus 2.5 uM D-glucose displayed significantly better results as per

157 Moreover, d-glucose-evoked increases in cytosolic ATP and d-glucos

158 apsed disease, using 2-deoxy-2-[(18)F]fluoro-D-glucose (FDG) and 3-deoxy-3[(18)F]fluorothymidine (FLT

159 ed with the glucose analog, 2-fluoro-2-deoxy-D-glucose (FDG) in vivo.

160 tumor cells take up 2-[(18)F]fluoro-2-deoxy-D-glucose (FDG) is heterogeneous and influenced by a mul

161 d in real time using 2-deoxy-2-[(18)F]fluoro-d-glucose (FDG) positron emission tomography (PET)/compu

162 Although 2[18F]fluoro-2-deoxy-d-glucose (FDG) uptake during positron emission tomograp

163 e origin of the (18)fluorine-labeled 2-deoxy-D-glucose (FdG) uptake signals observed clinically.

164 eled glucose analogue 2[(18)F]fluoro-2-deoxy-D-glucose (FDG).

165 ly allylated sugar derivatives, derived from D-glucose, followed by a sequential ring-closing metathe

166 cultured in 5 mm normal glucose, 25 mm l- or d-glucose for 48 h (osmotic control and high glucose tre

167 ose phosphorylase may function to remove GDP-D-glucose formed by GDP-D-mannose pyrophosphorylase, an

168 ivity of the P3/P4 and propylene-linked beta-d-glucose fragments, stronger in fIIa (15.5 kJ.mol(-1))

169 s of melanoidin formed at 160 degrees C from d-glucose (Glc) and l-alanine (Ala) as well as from fruc

170 unnatural glycosides such as 6-azido-6-deoxy-d-glucose/glucosamine to lead to beta-d-galactopyranosyl

171 Additionally, stimulation with TNF-alpha and D-glucose had an additive effect on RUNX1 expression, wh

172 d-Glucose has been identified as an efficient C1 synthon

173 High D-glucose (HDG) significantly increased PLK2 expression

174 Fungal beta-glucans are comprised of d-glucose homopolymers containing beta-1,3-linked glucos

175 lication of 5 mM QEP in the presence of 5 mM d-glucose, hSGLT1-mediated AMG uptake into small intesti

176 0 M, with a low detection limit of 0.01 M of d-glucose (i.e., 1.80 ppm), a sensitivity of 4.93 nm M(-

177 n using PET imaging of [(18)F]fluoro-2-deoxy-D-glucose in a porcine experimental model of early acute

178 croinjections of 2-deoxy-D-glucose or 5-thio-D-glucose in anesthetized, euglycemic rats.

179 hydrates in ionic liquids, the solubility of d-glucose in four ionic liquids was measured within a te

180 of beta1,4-mannobiose to 4-O-beta-d-mannosyl-d-glucose in mannan metabolism.

181 n-natural phenyl cyclopropanes directly from D-glucose in single-vessel fermentations.

182 oxy-d-glucose to form TDP-3-keto-4,6-dideoxy-d-glucose in the biosynthesis of TDP-d-desosamine.

183 The concentration of D-glucose in the blood following glucose gavage increase

184 gnificantly, we found an accumulation of GDP-D-glucose in the C10F3.4 mutant worms, suggesting that t

185 -dideoxy-D-glucose to TDP-3-keto-4,6-dideoxy-D-glucose in the desosamine biosynthetic pathway.

186 esized in cells incubated in 5 mM [U-(13) C]-D-glucose in the presence and absence of unlabelled mann

187 eated with the hexokinase inhibitor, 2-deoxy-d-glucose, indicating that a functional glycolytic pathw

188 s enhanced by glucose and reduced by 2-deoxy-D-glucose-induced starvation.

189 ments, the receptor region was identified by D-glucose infusion of isolated regions.

190 before repeating experiments using water or D-glucose infusion.

191 ments performed following [U-(2)H7, U-(13)C]-D-glucose injections.

192 ard conformations present only a single beta-d-glucose interaction site.

193 natively triggers the inversion of one alpha-d-glucose into a 5-C-acetoxy-beta-l-idose unit possessin

194 dTDP-3-amino-2,3,6-trideoxy-4-keto-3-methyl-d-glucose into its C-3' nitro derivative.

195 Thus, D-glucose is processed as both a phagostimulant and dete

196 T) with FDG-glucose (2-[(18)F]fluoro-2-deoxy-d-glucose) is already being used as a metabolic imaging

197 As a result of this analysis, l-glucose, d-glucose, l-allose, d-allose, d-gulose, d-galactose, an

198 trations and perform steady-state [U-(13) C]-D-glucose labelling.

199 Infusion of D-glucose led to 2.9-fold up-regulation in SGLT1 compare

200 tional capacities of ECs cultured under high D-glucose/low growth factors.

201 SEC analyses of d-glucose model reactions with and without l-pyroglutami

202 Heating aqueous d-glucose model reactions with l-glutamine and l-alanine

203 opy (ROESY) NMR experiment, wherein the beta-d-glucose moiety of 1 was used as an internal probe to u

204 (SS)-catalyzed assembly of (alpha1-4)-linked d-glucose molecules into maltodextrins generally agree t

205 X-ray diffraction analysis identified d-Glucose monohydrate as the main crystalline component.

206 larimetric measurements showed a doubling of d-glucose mutarotation velocity and HPLC analyses of d-f

207 interact with the C6 hydroxymethyl group of D-glucose negatively affected transport of both D-glucos

208 ad a significant effect on [F]fluoro-2-deoxy-D-glucose net uptake rate Ki in high-strain lipopolysacc

209 the glycation process occurs as a result of d-glucose nonenzymatically reacting with proteins such a

210 Cross-reactivity with d-lactose and d-(+)-glucose occurred only at concentrations >10(4)-fol

211 M was elicited by microinjections of 2-deoxy-D-glucose or 5-thio-D-glucose in anesthetized, euglycemi

212 ltures grown on rich media supplemented with d-glucose or glycerol produce H2 and simultaneously cons

213 nteers incubated for 60 min with 5-25 mmol/L d-glucose or iso-osmolar mannitol, we evaluated the infl

214 of B6.Sle1Sle2.Sle3 mice with either 2-deoxy-D-glucose or metformin were sufficient to prevent autoim

215 -matched controls using 18F-2-fluoro-2-deoxy-d-glucose PET (n = 20 per group) and voxel-based morphom

216 eletion strain was found to totally lack GDP-D-glucose phosphorylase activity; this activity was also

217 10F3.4 mutant worms, suggesting that the GDP-D-glucose phosphorylase may function to remove GDP-D-glu

218 The highest expression of GDP-D-glucose phosphorylase was found in the nervous and mal

219 C15orf58 gene expression products as the GDP-D-glucose phosphorylases of these organisms.

220 the rate of intracellular [F]fluoro-2-deoxy-D-glucose phosphorylation.

221 and is exploited with (18)F-2-fluoro-2-deoxy-d-glucose positron emission tomography ((18)F-FDG-PET) t

222 gnostic role of interim [18F]-fluoro-2-deoxy-D-glucose positron emission tomography (FDG-PET) perform

223 brain glucose concentrations) with 1-[(11)C]-d-glucose positron emission tomography during hyperinsul

224 a phenomenon used in 2-[(18)F]fluoro-2-deoxy-D-glucose positron emission tomography imaging of solid

225 ormal subjects with [(18)F]-2-fluoro-2-deoxy-D-glucose positron emission tomography imaging.

226 he utility of 2-[fluorine-18]-fluoro-2-deoxy-D-glucose positron emission tomography integrated with c

227 [F] fluoro-D-glucose positron emission tomography/computed tomograp

228 e subjects by means of [(18)F]fluoro-2-deoxy-d-glucose Positron Emission Tomography/Computed Tomograp

229 ied by FDG-PET-CT (2-[(1)(8)F]fluoro-2-deoxy-D-glucose-positron emission tomography combined with com

230 glucose uptake with [(18) F]2-fluoro-2-deoxy-D-glucose/positron emission tomography, lipolysis (RaGly

231 5,6-dihydrouracil, hesanal, cis-olefin, beta-D-glucose, propanal and some unassigned species.

232 N-acetylornithine, D-glucose, putrescine, and L-acetylcarnitine are consume

233 tyl-d-quinovosamine (2-acetamido-2,6-dideoxy-d-glucose, QuiNAc) occurs in the polysaccharide structur

234 fically, we find that high concentrations of D-glucose rapidly signal through AtWNK8 and AtWNK10, whe

235 to the C2 atom of the intermediate to form a d-glucose residue.

236 close analogues of cyclodextrins composed of d-glucose residues and triazole units bound together thr

237 lobiose 2-epimerase (CE) reversibly converts d-glucose residues into d-mannose residues at the reduci

238 Glucoamylases (GAs) from a wild and a deoxy-d-glucose-resistant mutant of a locally isolated Aspergi

239 oduct was formed, whereas the use of 2-deoxy-d-glucose resulted in reduced chemo- and stereoselectivi

240 or neutral sugars were alpha-D-glucose, beta-D-glucose, rhamnose and D-glucuronic acid.

241 probes in the presence and absence of excess d-glucose, "saturated cypate" and palmitic acid in two n

242 ion tomography with [(18)F]-2-fluoro-2-deoxy-D-glucose scan in addition to noncontrast computed tomog

243 nical ventilation, dynamic [F]fluoro-2-deoxy-D-glucose scans were acquired to quantify metabolic acti

244 equimolar concentration of L-asparagine and D-glucose showed a significant inhibition of acrylamide

245 After gavage of d-glucose, small intestinal glucose absorption across th

246 alose, d-(+)-xylose, d-fructose, 1-thio-beta-d-glucose sodium salt, d-(+)-galactose, sorbitol, glycer

247 Tests performed in different D-Glucose solutions have revealed a limit of detection c

248 1.0, 1.5 and 2.0 uM of starch with 2.5 uM D-glucose solutions were coated over freshly harvested c

249 V7 is still highly beta-D-glucose specific, highly active with the quinone diimi

250 is an oxidoreductase exhibiting a high beta-D-glucose specificity and high stability which renders g

251 cose-averse (GA) cockroaches, D-fructose and D-glucose stimulated sugar-gustatory receptor neurons (G

252 lucose-evoked increases in cytosolic ATP and d-glucose-stimulated insulin secretion were diminished i

253 ibitors were also effective at stopping high D-glucose-stimulated RUNX1 expression.

254 hus, our findings are in accord with 2-Deoxy-D-glucose studies performed in V1 of macaques and studie

255 s the synthesis of all positional isomers of d-glucose substitution for platinum warheads with detail

256 In this study, we investigated the effect of d-glucose substitution position on the biological activi

257 ibution analysis using 2-deoxy-2-[18F]fluoro-D-glucose that reprogramming of intestinal glucose metab

258 Aqueous-phase isomerization of d-glucose to d-fructose and l-sorbose is catalyzed in pa

259 ersion of dTDP-3-amino-2,3,6-trideoxy-4-keto-D-glucose to dTDP-3-amino-2,3,6-trideoxy-4-keto-3-methyl

260 s the deamination of TDP-4-amino-4,6-dideoxy-d-glucose to form TDP-3-keto-4,6-dideoxy-d-glucose in th

261 y these enzymes is the phosphorolysis of GDP-D-glucose to GDP and D-glucose 1-phosphate.

262 inated C-linked glycosides of D-galactose or D-glucose to L-cysteine using thiol-ene "click" chemistr

263 s the deamination of TDP-4-amino-4,6-dideoxy-D-glucose to TDP-3-keto-4,6-dideoxy-D-glucose in the des

264 vidual component polyphenols inhibited (14)C-D-glucose transport across differentiated Caco-2/TC7 cel

265 ylococcus epidermidus, homologs of the human D-glucose transporters, the GLUTs (SLC2), provide inform

266 diated cells even in glucose-free or 2-deoxy-D-glucose-treated conditions.

267 2+) uptake in response to d-glucose, whereas d-glucose-triggered cytosolic Ca(2+) oscillations remain

268 1.1.22) catalyze the conversion of UDP-alpha-d-glucose (UDP-Glc) to the key metabolic precursor UDP-a

269 yze the formation of UDP-4-amino-4,6-dideoxy-D-glucose (UDP-viosamine) from UDP-4-keto-6-deoxy-D-gluc

270 se-free medium or in the presence of 2-deoxy-D-glucose upon CCCP treatment.

271 nine (Q282A) doubled the Km(app) for 2-deoxy-d-glucose uptake and eliminated cis-allostery (stimulati

272 A 2-deoxy-d-glucose uptake assay indicates that depletion of septi

273 es modulated (14)C-D-glucose and (14)C-deoxy-D-glucose uptake into hepatic HepG2 cells.These data ind

274 idal strain enhances local [F]fluoro-2-deoxy-D-glucose uptake primarily by increasing the rate of int

275 [18F]fluoro-2-deoxy-D-glucose uptake rate was computed for the total lung, f

276 [(18)F]fluoro-2-deoxy-D-glucose uptake rate was computed for the whole lung, f

277 Overload-induced [(3)H]-2-deoxy-d-glucose uptake was not inhibited by d-fructose, demons

278 s, muscle weights and ex vivo [(3)H]-2-deoxy-d-glucose uptake were assessed.

279 porters (GLUTs) were monitored by 3-O-methyl-D-glucose uptake.

280 dal hyperinflation had [(18)F]fluoro-2-deoxy-D-glucose uptakes similar to controls.

281 te gravitational zones [(18)F]fluoro-2-deoxy-D-glucose uptakes were higher in ventilator-induced lung

282 aboration to their C14-epimers starting from d-glucose using beta-glycosylation and Grubbs olefin cro

283 e was synthesized from the bis(acetonide) of d-glucose using dicyclopentadienylzirconium(0)-mediated

284 ptical activity (ROA) spectra of methyl-beta-D-glucose utilizing density functional theory combined w

285 inct hexoses including the key carbon source D: -glucose, various glucose epimers, and several acetyl

286 as performed with injection of 100 mL of 20% d-glucose via the cubital vein.

287 es were lower than maximal rates for 2-deoxy-d-glucose (Vmax of 224 and 32 pmol/min/oocyte for GLUT2

288 sis to produce [2,3,4,6,6-(2)H5, 3,4-(13)C2]-D-glucose was developed to improve the (13)C signal-to-n

289 When d-glucose was used as a starting material, only the fura

290 omography (PET) with [(18)F]2-fluoro-2-deoxy-D-glucose was used to measure changes in regional brain

291 channels in patch-clamp experiments, whereas D-glucose was without effect.

292 Starting from d-glucose, we developed a divergent synthetic route to t

293 and beta-casein formed during glycation with d-glucose were identified and monitored in binary system

294 d mitochondrial Ca(2+) uptake in response to d-glucose, whereas d-glucose-triggered cytosolic Ca(2+)

295 cheap synthesis of 99.4% pure L-glucose from D-glucose which requires purification of neither interme

296 etabolism, and a treatment combining 2-deoxy-D-glucose, which inhibits glucose metabolism, and metfor

297 transporters are competitively inhibited by D-glucose, which is one of the major reasons hampering s

298 lation of hydroxy protected 1,6-anhydro-beta-d-glucose with arylalanes to provide beta-C-arylglucosid

299 ileged sites for the nonenzymatic binding of d-glucose with HSA.

300 s were incubated ex vivo with [(3)H]-2-deoxy-d-glucose, with or without insulin or AICAR, before isol