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1 hese cells is at least in part driven by the D5 receptor.
2 holinergic interneurons express the dopamine D5 receptor.
3 brain, and diencephalon express the dopamine D5 receptor.
4 ne acetyltransferase (ChAT) and the dopamine D5 receptor.
5 atial object recognition via the dopamine D1/D5 receptor.
6 ether it depends on dopamine acting on D1 or D5 receptors.
7 t depends on hippocampal dopamine D1 but not D5 receptors.
8                                 The dopamine D5 receptor, a D1-class receptor subtype, potentiates ac
9  of EPSCs, yet slightly reducing release, D1/D5 receptor activation selectively enhanced sustained sy
10  protein synthesis, we show that dopamine D1/D5 receptor activation stimulates local protein synthesi
11                                       The D1/D5 receptor agonist dihydrexidine (DHX) (0.01-10 microM;
12 nhance social learning, we administered a D1/D5 receptor agonist during the exposure sessions in whic
13 the hyperactivity-inducing effects of the D1/D5 receptor agonist SKF 81297.
14              We discovered compound 24, a D1/D5 receptor agonist with beta-arrestin recruitment and p
15           Administration of the D1-like (D1, D5) receptor agonist SKF38393 had no such effect.
16                             Both dopamine D1/D5 receptor agonists, which are positively coupled to ad
17                   We investigated whether D1/D5 receptors also affect depotentiation, the reversal of
18     To better understand the function of the D5 receptor and how it affects cholinergic interneurons
19         Sustained CREB phosphorylation by D1/D5 receptor and L-type channel agonists was targeted to
20 specific enhancement of early LTP through D1/D5 receptors and cAMP.
21  part mediated by dopamine acting through D1/D5 receptors and involves an increase in neuronal synchr
22 -133 phosphorylation of CREB via dopamine D1/D5 receptors and L-type calcium channels in organotypic
23 DRD5 and SLC6A3 which encode dopamine D4 and D5 receptors and the dopamine transporter, respectively.
24 f mature receptors, abrogated binding of the D5 receptor antagonist [(3)H]SCH23390, suggesting that w
25  stimulation were blocked by the dopamine D1/D5 receptor antagonist SCH 23390.
26                                       The DA D5 receptors appeared particularly important in regulati
27              To determine whether the D1 and D5 receptors are localized to separate populations of sy
28  the binding and functional properties of D1/D5 receptors, as well as effector proteins within their
29 toactivation are sensitive to hippocampal D1/D5 receptor blockade and resistant to adrenoceptor block
30      Dopamine or SKF 81297 (an agonist at D1/D5 receptors), caused inhibition of both inward and outw
31 s reversed by SCH 23390 (an antagonist at D1/D5 receptors), confirming that it was mediated by activa
32  mutagenesis was used to generate a panel of D5 receptors containing mutations in the three predicted
33                                 The dopamine D5 receptor (D(5)R) interacts with sorting nexin 1 (SNX1
34                                 The dopamine D5 receptor (D5R) is a Galpha(s)-coupled dopamine recept
35 eceptor subtype mediating this effect is the D5 receptor (D5R).
36                        Moreover, the role of D5 receptors (D5Rs) has not been addressed so far.
37  Here we tested the hypothesis that dopamine D5 receptors (D5Rs), characterized by a high constitutiv
38                             We find that the D5 receptor-deficient mice are viable and fertile and ap
39 ecently, we have shown that the dopamine D1B/D5 receptor displays binding and coupling properties tha
40    Moreover, dopaminergic projections and D1/D5 receptor distributions display a layer-dependent orga
41 ther critical brain areas also show dopamine D5 receptor expression.
42 , alongside changes in CB1, CB2, D1, D2, and D5 receptors expression.
43        We advanced known noncatecholamine D1/D5 receptor G protein-biased agonists and found that rem
44 rt the functional importance of the dopamine D5 receptor in the modulation of acetylcholine release t
45          Dopamine acts mainly through the D1/D5 receptor in the prefrontal cortex (PFC) to modulate n
46 ne the effects of local antagonism of D2 and D5 receptors in the AH on adolescent AAS-induced aggress
47        Together, these results indicate that D5 receptors in the LAH modulate non-GABAergic pathways
48 distribution of GAD neurons colocalized with D5 receptors in the LAH.
49 ce of GAD (a marker for GABA production) and D5 receptors in the lateral subdivision of the AH (LAH).
50 dition, was found to prevent localization of D5 receptors in the plasma membrane.
51  antagonists implicate the involvement of D1/D5 receptors in various pavlovian conditioning tasks in
52            Pharmacological stimulation of D1/D5 receptors increased auditory-evoked synaptic currents
53 ially overlapping populations, such that the D5 receptor is found in a subpopulation of those spines
54               These results suggest that the D5 receptor is not essential for many dopamine-mediated
55 idate a plausible mechanism for the observed D5 receptor mediation of AAS-induced aggression, brains
56 ated that endogenous activity of both D1 and D5 receptors modulated plasticity in the mPP.
57                             Also in support, D5 receptor mRNA antisense microinjected into the VMN bl
58               Indeed, the stimulation of the D5 receptor on rIL-2-activated NK cells inhibits the bin
59 area 9; rather, dopamine can activate D1 and D5 receptors on the same spines, plus an additional grou
60 the reported association of calcyon with the D5 receptor, or in addition, may suggest that calcyon ha
61 n of residue N7 was the major determinant of D5 receptor plasma membrane localization.
62       Immunoblots confirmed that D1, D2, and D5 receptor proteins were present from stomach through d
63 macological antagonism of spinal dopamine D1/D5 receptors reversed priming, whereas D1/D5 agonists in
64 a chronic pain state in both sexes; however, D5 receptors seem to play a critical role in males where
65 ies with excellent D2 versus D1, D3, D4, and D5 receptor selectivity.
66 ceptive substratum in the brain and a unique D5 receptor-specific signaling paradigm.
67 uts onto layer V PFC pyramidal neurons by D1/D5 receptor stimulation.
68                         Dopamine D1, D2, D3, D5 receptor subtype mRNAs and dopamine transporter mRNA
69 d in terms of physiological functions is the D5 receptor subtype.
70 istent with this model, the activation of D1/D5 receptors that couple to PKA in dopamine neurons anta
71  activation state of hippocampal dopamine D1/D5 receptors, the memory of a novel object presented dur
72 a key role in the cell surface expression of D5 receptors, they do not appear to contribute to the re
73                  In normal ears, D1, D2, and D5 receptors were detected in microdissected immature (p
74                                     Dopamine D5 receptors were localized on the somata, dendrites, an
75 ins of the embryonic forebrain, D3-, D4- and D5-receptors were predominant.
76  partially prevented by protection of the D1/D5-receptor with the agonist (R)-SKF 38393 or the specif