ライフサイエンスコーパス: DBP

1 DBP and RPP significantly higher in order "A" compared t

2 DBP concentrations in real samples ranged from 40 to 17

3 DBP exposure of recipient mice reduced androgen-dependen

4 DBP increased the recruitment of BAL total macrophages b

5 DBP/SBP of reference charts for all women and for each e

6 DBPs consisted of mostly brominated species, including b

7 DBPs, including haloacetic acids and trihalomethanes, ar

8 dataset (SBP [beta(GSMR) = -0.12, p = .02], DBP [beta(GSMR) = -0.10, p = .05]) and to the paternal f

9 dataset (SBP [beta(GSMR) = -0.16, p = .02], DBP [beta(GSMR) = -0.24, p = 7.4 x 10(-4)]) showed the s

10 ptoelectronic properties of two different [2]DBP[12]CPP nanohoops with electronically modifying subst

11 porating two dibenzo[a,e]pentalene units: [2]DBP[12]CPP nanohoops.

12 and is required for uptake of the 25(OH)D(3)-DBP complex.

13 1alpha,25(OH)(2)D(3) and for the 25(OH)D(3)/DBP complex to stimulate vitamin D receptor targets and

14 However, 7 DBPs (mostly brominated and nitrogenous) increased in fo

15 the combined meta-analysis also confirmed a DBP effect on AD (beta(GSMR) = -0.14, p = .03).

16 her GAC can make drinking water safer from a DBP perspective.

17 a 37% increased risk for HF, and young adult DBP >=80 mm Hg (compared with <80 mm Hg) was associated

18 responsiveness was increased by 48.1% after DBP exposure in participants without baseline hyperrespo

19 d immune cell phenotypes were assessed after DBP exposure.Measurements and Main Results: DBP exposure

20 uction of toxicity pathways observed for all DBPs caution of additive effects of mixtures and suggest

21 an expected based on prior work, whereas all DBPs induced notable effects on transcription of genes r

22 h SBP (SMD: -0.20; 95% CI: -0.37, -0.03) and DBP (SMD: -0.27; 95% CI: -0.52, -0.03) than did the cont

23 ssolved organic matter (DOM) composition and DBP formation is investigated using lake water collected

24 (2)O(2) AOP on the elemental composition and DBP formation potential of two DOM isolates by using ult

25 t its effectiveness for unregulated DBPs and DBP-associated toxicity is unclear.

26 The RO fouling and DBP formation behavior of anaerobic SMPs were similar to

27 ] >=90 mm Hg) and normal (SBP <140 mm Hg and DBP <90 mm Hg) blood pressure.

28 o SBP (high >=120 mm Hg, low <120 mm Hg) and DBP (high >70 mm Hg, low <=70 mm Hg).

29 eactions that leads to disinfectant loss and DBP formation.

30 identified no shared pathways between Nb and DBP-FITC, but revealed a type-I IFN (IFN-I) signature un

31 ll as confirmatory evidence that obesity and DBP influence RCC risk.

32 a predict both microcystin-LR occurrence and DBP formation potential (DBPfp) in lake water.

33 A decrease in PWV(CF) , PWV(CR) , SBP and DBP (-25%, -17%, -4% and -8%, respectively; P < 0.05) wa

34 ge 53 years and greater increases in SBP and DBP between 43 and 53 years were positively associated w

35 In the order "A", the SBP and DBP increased at the midpoint of the session.

36 In the order "B", the SBP and DBP increased only immediately after the end of the sess

37 t diets also significantly decreased SBP and DBP levels.

38 Heritability of SBP and DBP ranged from 16.8% to 40.4% for daytime, sleeping, 24

39 Mean baseline SBP and DBP were 139.7+/-15.6 and 78.1+/-11.9 mm Hg, respectivel

40 ere positively associated with their SBP and DBP.

41 Lastly, we show that an artificial DBP cocktail simulating the environmental concentrations

42 and autochthonous DOM as well as associated DBP formation are changed during an entire algal life cy

43 tions, time-dependent AVS, and using average DBP and RHR (all p <= 0.02).

44 coli DBP, SrmB, represents a model bacterial DBP whose absence impairs formation of the large ribosom

45 ggers (symptoms, LVEF, and LVESDi), baseline DBP (adjusted-hazard ratio [HR]: 0.79 [95% confidence in

46 mong individuals with low values of baseline DBP.

47 R1) was significantly associated with better DBP response to CTD (p = 5.76 x 10(-6), beta = -15.75) i

48 ods to test the shape of association between DBP and cardiovascular disease (CVD).

49 rts suggested a J-shaped association between DBP and MI.

50 onlinear J- or U-shaped relationship between DBP and adverse CVD outcomes; including MI.

51 inearity in the genetic relationship between DBP and CVD events.

52 tween systolic BP (SBP) and/or diastolic BP (DBP) and risk of Alzheimer's disease (AD).

53 for both systolic BP (SBP) and diastolic BP (DBP), and further assessed the direction of the variatio

54 ntile for systolic BP (SBP) or diastolic BP (DBP).

55 131/78 mm Hg systolic BP (SBP)/diastolic BP (DBP)], there was strong evidence of a linear dose-respon

56 correlations of migraine with diastolic BP (DBP, r(g) = 0.11, P = 3.56 x 10(-06)) and systolic BP (S

57 scenarios to elucidate the mechanisms of Br-DBP formation.

58 ribed brominated disinfection byproducts (Br-DBPs).

59 es, BAC and GAC treatment favored brominated DBP species by removing DOC but not bromide.

60 concentrations of the more toxic brominated DBP species, BAC and GAC treatment favored brominated DB

61 he fate of these newly identified brominated DBPs are unknown.

62 rison, small RNAs were minimally affected by DBP exposure.

63 ne mediator levels were modestly affected by DBP.Conclusions: DBP exposure augmented allergen-induced

64 hether cutaneous vitamin D is transported by DBP, we utilized DBP(-/-) mice that were made vitamin D-

65 Disinfection byproduct (DBP) exposure has been linked to multiple adverse health

66 sinfectant decay and disinfection byproduct (DBP) formation are necessary for predicting water qualit

67 nsequently impacting disinfection byproduct (DBP) formation in finished water; however, it remains un

68 t, but their role in disinfection byproduct (DBP) formation is unclear.

69 matter (DOM) and the disinfection byproduct (DBP) formation potential may also be altered.

70 lated drinking water disinfection byproduct (DBP)-can stimulate natural transformation rates in the m

71 oncentrations of 46 disinfection byproducts (DBPs) after treatment by chlorine or chloramines weighte

72 Disinfection byproducts (DBPs) and algal toxins can be expensive to monitor and r

73 tics of chlorinated disinfection byproducts (DBPs) and exacerbate the burden of DBP control for water

74 Disinfection byproducts (DBPs) are a ubiquitous source of chemical exposure in dr

75 precursors to form disinfection byproducts (DBPs) during disinfection (e.g., chloramination).

76 to the formation of disinfection byproducts (DBPs) that have negative human-health effects.

77 ation of brominated disinfection byproducts (DBPs) upon chlorination.

78 nated and iodinated disinfection byproducts (DBPs) when treated.

79 gulated halogenated disinfection byproducts (DBPs), and 8 N-nitrosamines after chloramination.

80 ntrolling regulated disinfection byproducts (DBPs), but its effectiveness for unregulated DBPs and DB

81 eads to exposure to disinfection byproducts (DBPs), including trihalomethanes (THMs), which have been

82 formation of toxic disinfection byproducts (DBPs).

83 itiates exposure to disinfection byproducts (DBPs).

84 he production of 43 disinfection byproducts (DBPs).

85 ter to form harmful disinfection byproducts (DBPs).

86 nt classes of toxic disinfection byproducts (DBPs).

87 ds, nitrogenous DBPs, and other carbonaceous DBPs.

88 During the bonne chauffe, DBP and DEHP accumulated in the secondes more than in th

89 posure to six brominated and two chlorinated DBPs: bromoacetic acid (BAA), bromoacetonitrile (BAN), 2

90 ains novel high molecular weight chlorinated DBPs that are detected via high-resolution mass spectrom

91 The Escherichia coli DBP, SrmB, represents a model bacterial DBP whose absenc

92 s were modestly affected by DBP.Conclusions: DBP exposure augmented allergen-induced lung function de

93 wo currently unregulated nitrogen-containing DBP (N-DBP) groups commonly found in water disinfected w

94 for most detected DBPs; disulfur-containing DBPs, like bromosultone sulfonate and bromohydrin disulf

95 dinated, brominated, and nitrogen-containing DBPs.

96 Over 300 sulfur-containing DBPs, with 43 unique molecular formulas, were found by h

97 As controls, DBP(+/+) animals were vitamin D depleted and made equall

98 ed in each zone to link to the corresponding DBPs.

99 f recombinant DBP to the vitamin D-deficient DBP(-/-) mice restored the response to UV light.

100 ation of olefin sulfonate surfactant-derived DBPs from laboratory-disinfected gas extraction wastewat

101 nate as a likely precursor for most detected DBPs; disulfur-containing DBPs, like bromosultone sulfon

102 together with systolic (SBP) and diastolic (DBP) blood pressure.

103 tion, whereas DDM (dichlorodiphenylmethane), DBP (dichlorobenzophenone), and DDA (dichlorodiphenylace

104 e (DBP) and corresponding MSNA at rest (i.e. DBP 'operating pressure' and MSNA 'operating point'), as

105 degrade three different plasticizers (i.e., DBP, DEHP, and ATBC) via a comprehensive proteogenomic a

106 The contribution of each DBP class to the cumulative toxicity was estimated by we

107 ion, to measure 32 conventional and emerging DBPs in different produce types including lettuce, cabba

108 By using novel engineered DBP immunogens, we validate that the prime targets of pr

109 e the dominant contributors to the estimated DBP-associated toxicity.

110 oxidative stress in other studies exploring DBP toxicity.

111 be useful tools for future research on food DBPs.

112 g/100 mmol SR (95% CI: -10.4, -5.0), and for DBP it was -3.0 mm Hg/100 mmol SR (95% CI: -4.6, -1.4).

113 Hg/100 mmol SR (95% CI: -2.7, -0.20) and for DBP it was: -0.07 mm Hg/100 mmol SR (95% CI: -1.5, 1.4).

114 hese results reveal a specific mechanism for DBP regulation of ribosomal assembly, indirectly mediate

115 mHg for SBP and from -2.50 to -4.22 mmHg for DBP.

116 Similar findings were observed for DBP variation.

117 These results demonstrate a requirement for DBP to utilize cutaneously produced vitamin D.

118 Halogenated ions were assigned in four DBP elemental groups including CHOBr (180), CHONBr (13),

119 ; increase in mean WMHV per 10 mm Hg greater DBP 15%, 4-27, p=0.0057; increase in mean WMHV per one S

120 1 years of age (-6.9 mL per 10 mm Hg greater DBP, -11.9 to -1.9, p=0.0068), as were greater increases

121 M4 rarely exceeded international guidelines, DBPs of greater toxicological concern were observed in h

122 Regardless, the reduction in halogenated DBP formation during postchloramination achieved by BAF

123 C was more effective at reducing halogenated DBP formation during postchloramination.

124 ts show the formation of complex halogenated DBPs that are formed in the treatment of water with a no

125 Unregulated, halogenated DBPs were the dominant contributors to the estimated DBP

126 rein, we report surface modification of a Hf-DBP nMOF for the co-delivery of a hydrophobic small-mole

127 Upon X-ray irradiation, IMD@Hf-DBP/alphaCD47 effectively modulates the immunosuppressiv

128 esting that exposure to or removal from high DBP produces effects that require longer than one sperma

129 biological pathways, initiation on the high-DBP condition activated oxidative stress and DNA damage

130 Higher DBP at 43 years of age was associated with smaller whole

131 HT-DBP is a microscopy-based, single-cell resolution assay

132 HT-DBP requires only 24 hours of ex vivo culture, which ena

133 Effective compounds identified by HT-DBP induced tumor regression in genetically engineered a

134 ed high-throughput dynamic BH3 profiling (HT-DBP).

135 ncer patients, our data demonstrated that HT-DBP could be used to generate personalized pharmacotypes

136 Thus, HT-DBP appears to be an ex vivo functional method with suff

137 n in many source waters, enhancing Br- and I-DBP formation.

138 12; increase in mean WMHV per 1 SD change in DBP 15%, 3-30, p=0.017).

139 decreases consistently per unit decrease in DBP, even among individuals with low values of baseline

140 The more severe testis dysgenesis in DBP-MPW animals may result from the presence of basally

141 azard ratio, 1.07 per unit mm Hg increase in DBP; P<0.001).

142 1.9, p=0.0068), as were greater increases in DBP between 36 and 43 years of age (-6.5 mL per 1 SD cha

143 lamina, whereas GC migration was minimal in DBP-FW animals.

144 .8, -0.5; p = 0.027) in RT; and -3.2 mmHg in DBP (95%CI -7.9, 1.5; p = 0.001) in CT.

145 ilar findings were observed for variation in DBP.

146 demonstrated an adverse effect of increasing DBP increments on CVD outcomes, including MI (MI hazard

147 es to allow the analysis of thermally labile DBPs.

148 lts of this study suggest that mutagens like DBPs may play an important role in enhancing the fixatio

149 ent train does not necessarily result in low DBP concentrations in the basin of a pool.

150 F), LV end-systolic diameter-index (LVESDi), DBP, and RHR were univariable predictors of all-cause mo

151 Maintaining mean DBP >/=25 mm Hg in infants and >/=30 mm Hg in children >

152 namically significant AR, routinely measured DBP and RHR demonstrate a robust association with all-ca

153 n all-cause mortality and routinely measured DBP and RHR was examined.

154 waters may switch to chloramination to meet DBP regulations.

155 recoveries (50-130%) were achieved for most DBPs in the different produce.

156 Method detection limits for most DBPs were between 15 and 100 ng/L, and relative standard

157 ently unregulated nitrogen-containing DBP (N-DBP) groups commonly found in water disinfected with mon

158 ) M(-1) s(-1)) to form a recently reported N-DBP, the N-haloacetamide N,2,2-trichloroacetamide.

159 Instead of DCAM, a previously unreported N-DBP, N-chloro-2,2-dichloroacetamide (N-Cl-DCAM), was con

160 ihalomethanes, haloacetic acids, nitrogenous DBPs, and other carbonaceous DBPs.

161 In patients 60 years or older, SBP but not DBP was associated with 10-year survival, an effect that

162 o contributes to enhanced formation of novel DBPs during chlorine photolysis.

163 udy sought to investigate the association of DBP and RHR with all-cause mortality in patients with AR

164 ter-specific blood and urinary biomarkers of DBP were associated with small for gestational age (SGA)

165 products (DBPs) and exacerbate the burden of DBP control for water utilities.

166 erm birth across tertiles (or categories) of DBP biomarker concentrations measured across pregnancy t

167 The documentation of DBP species other than THM4 is rare in low-income countr

168 dies targeting a conserved binding domain of DBP.

169 ndomization (MR) estimates for the effect of DBP on CVD.

170 In this analysis of the genetic effect of DBP, we found no evidence for a nonlinear J- or U-shaped

171 n reveals stronger instrumental estimates of DBP (OR [95% CI] = 1.20 [1.15-1.25]/10 mmHg; P = 5.57 x

172 Comprehensive MR interrogation of DBP required us to also model systolic blood pressure, g

173 UV irradiation of DBP(+/+) animals restored serum calcium and serum 25(OH)

174 g water in the European Union as a marker of DBP exposure and estimated the attributable burden of bl

175 Measurements of DBP-EGFP extravasation in plvapb mutants provided direct

176 These findings support a critical role of DBP in migraine susceptibility and shared biology underl

177 Second, we created polygenic risk scores of DBP and systolic blood pressure and generated linear Men

178 Here, we report the structures of DBP region II in complex with human-derived, neutralizin

179 nd serum 25(OH)D while the same treatment of DBP(-/-) animals failed to show either a serum calcium o

180 It was also demonstrated higher values of DBP and minor mean R-R intervals in order "B" at 10 min-

181 d to engage multiple polymorphic variants of DBP.

182 sed risk of SGA, whereas other biomarkers of DBPs examined across pregnancy were not associated with

183 s were applied to lower the concentration of DBPs and their precursors in pool water by using a pilot

184 ved acceleration of facilitated diffusion of DBPs by molecular crowding agents and crowder-concentrat

185 sis of the enhanced facilitated diffusion of DBPs inside a crowded cellular milieu through, to our kn

186 sition of DOM and the formation potential of DBPs.

187 single extraction method for a wide range of DBPs, producing the lowest method detection limits to-da

188 sults demonstrated that effective removal of DBPs across the treatment train does not necessarily res

189 ) attack during the AOP and the influence on DBP formation.

190 eatment, the effect of the produced water on DBP formation was largely eliminated.

191 participants were exposed to control air or DBP for 3 hours in an environmental chamber followed imm

192 ta-analysis of 299,024 individuals of SBP or DBP as exposure variables against three different outcom

193 which have the potential to serve as organic DBP precursors in OGW-impacted water sources.

194 an decouple the formation of THM4 from other DBP classes that are more potent toxins.

195 but ozonation was less significant for other DBPs.

196 may not be an adequate indicator of overall DBP exposure in impaired water supplies prevalent in som

197 lomethanes (THM4) as an indicator of overall DBP exposure.

198 analysis identified >100 unique targets per DBP.

199 ylphenol (BHT) and 2,4-di-tert-butyl-phenol (DBP), in humans (fat tissues, serum, urine, breast milk,

200 reated with vehicle or di-n-butyl-phthalate (DBP, a plasticising chemical known to induce testicular

201 sticizers (PAEs), such as dibutyl phthalate (DBP) and bis(2-ethyl hexyl) phthalate (DEHP), are now in

202 dication is monomeric in dibutyl phthalate (DBP) matrix at low temperatures, and it has a half-life

203 However, effects of dibutyl phthalate (DBP) or other phthalates found in high concentrations in

204 Administration of dibutyl phthalate (DBP) to pregnant rats causes reproductive disorders in m

205 benzyl phthalate [BBzP], dibutyl phthalate [DBP], di(2-ethylhexyl) phthalate [DEHP]) decreased, whil

206 a known concentration of a single phthalate, DBP.Methods: In a randomized crossover study, 16 allerge

207 While toxicology studies have pinpointed DBPs with the greatest toxic potency, analytical methods

208 scopy, extending previous work on predicting DBPs alone.

209 GAC also effectively removed preformed DBPs at plants using prechlorination, including highly t

210 atest reduction in diastolic blood pressure (DBP) (P = 0.02) but also the most pronounced increase in

211 he relationship of diastolic blood pressure (DBP) and corresponding MSNA at rest (i.e. DBP 'operating

212 od pressure (SBP), diastolic blood pressure (DBP) and mean arterial pressure (MAP) were significantly

213 ic significance of diastolic blood pressure (DBP) and resting heart rate (RHR) in patients with hemod

214 pressure (SBP) and diastolic blood pressure (DBP) are important predictors of graft and patient survi

215 pressure (SBP) and diastolic blood pressure (DBP) at age 53 years and greater increases in SBP and DB

216 and systolic (SBP)/diastolic blood pressure (DBP) in individuals with prehypertension or hypertension

217 tolic (SBP) and/or diastolic blood pressure (DBP) levels.

218 est that excessive diastolic blood pressure (DBP) lowering might increase the risk of myocardial infa

219 rload, whereas low diastolic blood pressure (DBP) may lead to impaired coronary perfusion.

220 180 mm Hg, office diastolic blood pressure (DBP) of 90 mm Hg or greater, and a mean 24-h ambulatory

221 (SBP) or >= 10 in diastolic blood pressure (DBP) upon standing classified subjects as OH positive (O

222 glucose, HbA1c and diastolic blood pressure (DBP), and positively correlated with age, known diabetes

223 systolic (SBP) and diastolic blood pressure (DBP), high-density-lipoprotein cholesterol (HDL-C), and

224 systolic (SBP) and diastolic blood pressure (DBP), rate-pressure product (RPP) oxygen saturation (SpO

225 systolic (SBP) and diastolic blood pressure (DBP), total cholesterol (TC), LDL and HDL cholesterol, t

226 s for variation in diastolic blood pressure (DBP).

227 pressure (SBP) and diastolic blood pressure (DBP).

228 CI 1.30-2.55) and diastolic blood pressure (DBP; ORSD: 1.28, 95% CI 1.11-1.47), but not systolic blo

229 BP] >=140 mm Hg or diastolic blood pressure [DBP] >=90 mm Hg) and normal (SBP <140 mm Hg and DBP <90

230 Some disinfection by-products (DBPs) are reproductive and developmental toxicants in la

231 Ms) are widespread disinfection by-products (DBPs) in drinking water, and long-term exposure has been

232 formation of toxic disinfection by-products (DBPs) is among the main concerns in the use of chlorine

233 mation of numerous disinfection by-products (DBPs), some of which are cytotoxic, mutagenic, genotoxic

234 l priming measured by dynamic BH3 profiling (DBP) identifies drugs that are persistently active in BH

235 washing with chlorine significantly promoted DBPs' formation and concentrations in the produce.

236 ion of the genetically tagged plasma protein DBP-EGFP, we show that the developmental acquisition of

237 mbrane receptor for serum d-binding protein (DBP) in kidney cells and is required for uptake of the 2

238 The Plasmodium vivax Duffy-binding protein (DBP) is a prime target of the protective immune response

239 he circulation by vitamin D binding protein (DBP), but experimental evidence is lacking.

240 st association between DNA-binding proteins (DBPs) and DNA is explained by a facilitated diffusion me

241 DEAD-box proteins (DBPs) comprise a large family of proteins that most comm

242 oclonal anti-DARC antibodies and recombinant DBP to CD71(high)/RNA(high) reticulocytes was significan

243 us injection of small amounts of recombinant DBP to the vitamin D-deficient DBP(-/-) mice restored th

244 ive for removing DBP precursors and reducing DBP formation and total organic halogen, even after >22

245 controlling 61 unregulated DBPs, 9 regulated DBPs, and speciated total organic halogen (total organic

246 o be more cyto- and genotoxic than regulated DBPs.

247 The remaining DBPs were analyzed using a GC-single quadrupole mass spe

248 Overall, GAC was effective for removing DBP precursors and reducing DBP formation and total orga

249 DBP exposure.Measurements and Main Results: DBP exposure increased the early allergic response (21.4

250 s, improvements in TC, HDL cholesterol, SBP, DBP, HOMA-IR, and acute/chronic FMD remained significant

251 ity duration was largely attenuated for SBP, DBP, and HDL-C.

252 GSH levels correlated negatively with SBP, DBP and MBP values in all participants (p = 0.0010; p =

253 vels of GSSG correlated positively with SBP, DBP and MBP values in all participants (p = 0.0410; p =

254 ts, for all women and each ethnic group, SBP/DBP at 12 and 20 weeks gestation was similar before risi

255 Compared to WB, P women had lower SBP/DBP at 12, 20 and 37 weeks gestation.

256 y-two participants (54 +/- 11 y, resting SBP/DBP 137 +/- 9/86 +/- 6 mmHg) were randomly allocated int

257 ification for 61 toxicologically significant DBPs from 7 different chemical classes, including unregu

258 However, studies of trimester-specific DBP exposure on adverse birth outcomes in humans are inc

259 g analysis indicated one-third of the target DBPs were found in unwashed produce, and washing with ch

260 The DBP analysis in the combined meta-analysis also confirme

261 As the DBP level was altered, numerous sperm RNA elements (REs)

262 or pilot-scale reuse trains to estimate the DBP-associated toxicity of their effluents.

263 This raises the importance of the DBP formation potential of the organic precursors, which

264 Environmental Protection Agency revised the DBP regulations starting in 1998 to further limit levels

265 The results indicated that while the DBP-associated quality of MF/RO/AOP-based reuse waters c

266 Therefore, DBP and RHR should be integrated into comprehensive clin

267 While these DBPs are highly toxic, the total calculated cytotoxicity

268 verse proportion (p nonlinearity = 0.002) to DBP starting at 70 mm Hg and peaking at 55 mm Hg and in

269 .0008); (3) BFM was positively correlated to DBP (R(2) = 0.1232, P = 0.02) and partially correlated t

270 y design of high or background, exposures to DBP.

271 of this effort, the effect on sperm RNAs to DBP exposure were longitudinally assessed using a cross-

272 ransformed human cells after 4 h exposure to DBPs at predetermined equipotent concentrations, identif

273 ential adverse health effects of exposure to DBPs.

274 ghted by metrics of toxic potency, the total DBP calculated toxicity was 4-fold lower than observed p

275 f pretreatment method prior to RO, the total DBP concentrations were <14 mug/L upstream of RO.

276 ation process, and chloramination, the total DBP concentrations were <=5 mug/L.

277 r quantifying complete classes of most toxic DBPs at sufficiently low quantification limits (ng/L).

278 s for rational design of strain-transcending DBP-based vaccines and therapeutics against P. vivax.

279 were sparse for outcomes related to treating DBP to a lower target or for patients older than 60 year

280 erations led to the formation of 226 unknown DBPs with decreased aromaticity indices (AI(mod)) in the

281 Links between the unknown DBPs and the corresponding precursors in DOM were visual

282 We measured THM4 and 21 unregulated DBPs in tap waters and laboratory-treated source waters

283 service lives for controlling 61 unregulated DBPs, 9 regulated DBPs, and speciated total organic halo

284 DBPs), but its effectiveness for unregulated DBPs and DBP-associated toxicity is unclear.

285 Other blood and urinary DBP biomarkers examined were unrelated to SGA, LBW, or p

286 vitamin D is transported by DBP, we utilized DBP(-/-) mice that were made vitamin D-deficient.

287 ust analytical methods for measuring various DBPs in produce have been lacking.

288 in four genes in the vitamin D pathway (VDR, DBP, CYP27B1, CYP24A1) on PCOS.

289 s (DARC) and the dimer interface of P. vivax DBP.

290 her hand, the potential presence of volatile DBPs and fractionation losses do not allow for tentative

291 p revealed higher concentrations of volatile DBPs in the pool compared to those employing GAC filtrat

292 e environmental concentrations of five water DBP classes stimulates natural transformation by almost

293 The total toxic potency-weighted DBP concentrations in some waters were elevated compared

294 relate with the total toxic potency-weighted DBP concentrations.

295 Comparing the total toxicity-weighted DBP concentration between reuse and drinking waters prov

296 These total toxicity-weighted DBP concentrations were compared to those measured in th

297 When DBP concentrations were weighted by metrics of toxic pot

298 er, the molecular initiating events by which DBPs induce their toxicities remain unclear.

299 a facilitated diffusion mechanism, in which DBPs adopt a weighted combination of three-dimensional d

300 (p = 0.04, beta = 4.36) for association with DBP.