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1                                              DCA (10-150 microM) stimulated the release of both Hbeta
2                                              DCA (50-250 micromol/L) caused profound Ca2+ release fro
3                                              DCA - RQ storage was associated with the activation of t
4                                              DCA also caused dramatic translocation of PH-PLCdelta-GF
5                                              DCA and similar approaches that use sequence information
6                                              DCA and taurodeoxycholic acid (TDCA) (100 micromol/L) ca
7                                              DCA can accurately infer spatial adjacencies between res
8                                              DCA did not affect RPP but normalized dP/dt in HYP.
9                                              DCA forces CSCs into oxidative phosphorylation but does
10                                              DCA had no effect on localization of a novel (PKCdelta)
11                                              DCA induced cAMP formation at the plasma membrane and cy
12                                              DCA induced mild, transient colonic inflammation within
13                                              DCA induces mild, transient colitis, resulting in persis
14                                              DCA involvement in these disease processes results partl
15                                              DCA is based on three features, primarily the previously
16                                              DCA is currently not approved for cancer treatment in th
17                                              DCA is shown to yield a large number of correctly predic
18                                              DCA promoted minority MOMP noted by minimal release of c
19                                              DCA showed minimal net benefit.
20                                              DCA stimulated phosphorylation of the p65 subunit of NF-
21                                              DCA stimulated TGR5 redistribution to plasma membrane mi
22                                              DCA storage reduces ACC (1-aminocyclopropane-1-carboxyla
23                                              DCA treatment restored cardiac mitochondrial membrane po
24                                              DCA was also found to induce tyrosine phosphorylation an
25                                              DCA, taurolithocholic acid, and oleanolic acid did not s
26                                              DCA-CF is efficient in quality maintenance of 'Royal Gal
27                                              DCA-RQ1.5 is the most suited for the storage of 'Fuji Su
28 ) day(-1) simvastatin + 30 mg kg(-1) day(-1) DCA (n = 9) or 88 mg kg(-1) day(-1) simvastatin + 40 mg
29 ) day(-1) simvastatin + 40 mg kg(-1) day(-1) DCA (n = 9).
30 ane (1,1,1-TCA); and 1,1-dichloroethane (1,1-DCA), range from -26.5 per thousand to -1.8 per thousand
31                     Unlike 1,1,1-TCA and 1,1-DCA, reductive dechlorination of CF by the Dehalobacter-
32 /MEL also shown to degrade 1,1,1-TCA and 1,1-DCA.
33 icroarray analyses revealed a cluster of 142 DCA- and INA-responsive genes that show a pattern of dif
34    When aseptic root cultures were fed [14C]-DCA, compared with wild types, the ugt72B1 plants showed
35                Isotopic fractionation of 1,2-DCA (epsilonbulk(C) and epsilonbulk(Cl)) for Dehalococco
36 nisms controlling natural attenuation of 1,2-DCA and to design appropriate strategies to enhance biod
37 sed to characterize dihaloelimination of 1,2-DCA by different bacteria, which needs to be confirmed i
38 rination are the proposed mechanisms for 1,2-DCA degradation by coupled nZVI-dithionite treatment.
39 tterns allow for the first time reliable 1,2-DCA degradation pathway identification in the field, whi
40 e potential of this approach to identify 1,2-DCA degradation pathways in the field.
41 of the dual isotope approach to identify 1,2-DCA degradation pathways in the field.
42 and anaerobic biodegradation pathways of 1,2-DCA in the field and suggests that this approach might a
43 t coupling nZVI with dithionite to treat 1,2-DCA is proposed in this work.
44 nZVI-dithionite was able to degrade >90% 1,2-DCA over the course of a year.
45 lcitrance toward abiotic dechlorination, 1,2-DCA remains a challenging compound for the remediation c
46 nd-specific chlorine isotope analysis of 1,2-DCA was performed for the first time, and isotope fracti
47                         Lambda values of 1,2-DCA were, for the first time, determined in two field si
48 rachloroethene (PCE)/1,2-dichloroethane (1,2-DCA) completely to ethene.
49                      1,2-Dichloroethane (1,2-DCA) is a chlorinated solvent classified as a probable h
50 ic biodegradation of 1,2-dichloroethane (1,2-DCA) using five microbial cultures.
51 ic biodegradation of 1,2-dichloroethane (1,2-DCA) via dihaloelimination by Dehalococcoides and Dehalo
52 ic biodegradation of 1,2-dichloroethane (1,2-DCA) via oxidative cleavage of a C-H bond (Pseudomonas s
53 tepwise removed halogens from 2,4,6-TCP, 1,2-DCA, PCE, PBDEs, and PCBs.
54 o achieve nearly complete degradation of 1,2-DCA.
55 etallic) has been unable to dechlorinate 1,2-DCA.
56 phere monitored by respiratory quotient 1.3 (DCA-RQ 1.3) showed lower ethylene production, respiratio
57  and its metabolite 3,4-dichloroaniline (3,4-DCA) on thyroid function and metamorphosis in tadpoles o
58 phere monitored by respiratory quotient 1.5 (DCA-RQ 1.5) increased the acetaldehyde, ethanol and ethy
59  apples can be stored at 3 degrees C using a DCA system, based either on CF or on RQ, to save electri
60  correlation with viral stability, whereas a DCA-derived metric was highly correlated with virus tran
61 ative IGF-1 receptor (K1003R) also abolished DCA-induced AKT activation.
62 ck-out or knockdown of p53 or CD95 abolished DCA + MEK1/2 inhibitor-induced PERK phosphorylation and
63 ific bile acids, and the secondary bile acid DCA in particular, in the regulation of hepatic LCFA upt
64 ) enhanced the toxicity of deoxycholic acid (DCA) + MEK1/2 inhibitor.
65 e the secondary bile acids deoxycholic acid (DCA) and lithocholic acid (LCA), which accumulate at con
66 vestigating the effects of deoxycholic acid (DCA) and ursodeoxycholic acid on the expression and rele
67  Secondary bile acids like deoxycholic acid (DCA) are well-established tumor promoters that may exert
68 eoxycholic acid (UDCA) and deoxycholic acid (DCA) as the two most potent inhibitors of the liver-spec
69                            Deoxycholic acid (DCA) is an endogenous secondary bile acid implicated in
70 ry bile acids (BA) such as deoxycholic acid (DCA) promote the development of several gastrointestinal
71 a-linolenic acid (DGLA) to deoxycholic acid (DCA) species (DCAS) was significantly increased in obese
72                            Deoxycholic acid (DCA) was instilled into the rat colon daily for 3 days a
73              Second, CDCA, deoxycholic acid (DCA), and other synthetic FXR agonists, such as GW4064,
74 uct of secondary bile acid deoxycholic acid (DCA), at as low as 50 uM, inhibited 82.8% of C. perfring
75                            Deoxycholic acid (DCA), chenodeoxycholic acid (CDCA), taurodeoxycholic aci
76 o the oncogenic bile acid, deoxycholic acid (DCA), for 1 year.
77 holic acid (T-betaMCA) and deoxycholic acid (DCA), induce proliferation and DNA damage in Lgr5(+) cel
78                       BAs (deoxycholic acid (DCA), taurolithocholic acid) and the selective agonists
79 ta-cyclodextrin suppressed deoxycholic acid (DCA)-induced apoptosis, and staining for cholesterol wit
80 revealed the presence of Dichloracetic acid (DCA) as the active antitumor constituent of Chaetomorpha
81 sulfoxide (DMSO) and 5% dichloroacetic acid (DCA) and successive heteronuclear (1)H-(15)N HSQC spectr
82 o-compartment model for dichloroacetic acid (DCA).
83 epresentative, 3,5-dichloroanthranilic acid (DCA), efficiently induced defense reactions to the phyto
84                Bile acids (deoxycholic acid, DCA; taurocholic acid, TCA) activated AKT and glycogen s
85    Conjugating lipids (e.g. docosanoic acid, DCA) to siRNA supports extrahepatic delivery, but tissue
86 s of Purkinje cells, nearly universal across DCAs, dysregulates the dentatothalamocortical network.
87    Genera previously associated with aerobic DCA biodegradation (Xanthobacter, Ancylobacter, Azoarcus
88  a difference of convex functions algorithm (DCA).
89 )-alkylmethylammonium dicyanamide ([Aliquat][DCA]) and 1-n-octyl-3-methylimidazolium dicyanamide ([Om
90                     Decision curve analyses (DCA) showed improvements of the clinical usefulness and
91 nalysis (RFA) and damping capacity analysis (DCA).
92                    Dynamic cluster analysis (DCA) is an automated, unbiased technique which can ident
93                    Direct coupling analysis (DCA) for protein folding has made very good progress, bu
94         Currently, direct coupling analysis (DCA) infers nucleotide contacts in a sequence from its h
95                    Direct-coupling analysis (DCA) is a statistical framework that captures residue co
96 consC3 outperforms direct coupling analysis (DCA) methods significantly independent on family size, s
97 cts estimated from direct coupling analysis (DCA) of co-evolving genomic sequences.
98  based entirely on Direct Coupling Analysis (DCA) of correlated mutations in multiple sequence alignm
99 t constraints from direct coupling analysis (DCA) to determine the dominant docked conformation of th
100 d this model using direct coupling analysis (DCA), a powerful statistical inference method that has b
101 ecently introduced direct-coupling analysis (DCA).
102 global statistical direct coupling analysis (DCA).
103                     Decision-curve analysis (DCA) confirmed the superiority of the SORT over other pr
104 sequent VLRAF using decision-curve analysis (DCA).
105 ic (ROC) curves and Decision Curve Analysis (DCA).
106 cid (50-200 microM) inhibited both basal and DCA-induced defensin release.
107 xycholic acid in unconjugated (CDCA, CA, and DCA) and tauro-conjugated (t-CDCA, t-CA, t-DCA) form on
108 ing ClustalW, Probcons, Muscle, T-Coffee and DCA.
109  at these sites using the Frustratometer and DCA.
110 ty related to the values obtained by RFA and DCA devices, which could create disagreements and miscom
111 is had been made in various regions; RFA and DCA should have been applied in the same implants and pe
112                                Both UDCA and DCA were able to inhibit LCFA uptake by primary hepatocy
113  which specific bile acids, such as UDCA and DCA, can impact hepatic triglyceride metabolism and may
114 al cases of a few protein families and apply DCA to a systematic large-scale study of nearly 2,000 Pf
115 ons such as dynamical cluster approximation (DCA).
116             Degenerative cerebellar ataxias (DCAs) affect up to 1 in 5,000 people worldwide, leading
117 tory quotient dynamic controlled atmosphere (DCA - RQ), which induces ethanol production through low
118 en (ULO) with dynamic controlled atmosphere (DCA) and controlled atmosphere (CA) on the post storage
119 he effects of dynamic controlled atmosphere (DCA) storage based on chlorophyll fluorescence (DCA-CF)
120 a' apples, in dynamic controlled atmosphere (DCA), treated with or without 1-methylcyclopropene (1-MC
121  TACE inhibition was sufficient to attenuate DCA-induced AREG, but not TGF-alpha shedding.
122 n and cell-cycle progression, and attenuated DCA-induced colorectal cancer or PDAC tumorigenicity.
123 ine ACR was measured by nurses using a Bayer DCA 2000 analyzer and expressed in mg/mmol (reference ra
124 Expression of dominant negative PERK blocked DCA + MEK1/2 inhibitor-induced expression of ATG5, GRP78
125 TILs) [emim][BF(4)], [emim][DCA], and [bmim][DCA] at 25 degrees C from below 1 GHz to 10 THz by ultra
126 th butylmethylpyrrolidinium-dicyanamide (BMP-DCA) IL shows high sensitivity toward ascorbic acid (AA)
127                                More broadly, DCA, LCA and their derivatives are major components of t
128 did not alter AKT or GS activation caused by DCA.
129 ocytes abolished activation of AKT and GS by DCA and TCA.
130  large interfaces are commonly identified by DCA.
131 t alter ERK1/2 and AKT activation induced by DCA or CDCA but abolished pathway activations by conjuga
132  findings suggest that contacts predicted by DCA can be used as a reliable guide to facilitate comput
133 aluate the accuracy of contact prediction by DCA for a large number of protein domains, based purely
134               A simple hybrid method, called DCA-fold, integrating DCA contacts with an accurate know
135 e older group, FMT signal in dorsal caudate (DCA) and dorsal putamen was greater with age, suggesting
136 nd the lowest by fruits stored under DCA-CF (DCA based on chlorophyll fluorescence).
137 lmitoleic acid and the deoxycholate/cholate (DCA/CA) ratio, along with the dysregulation scores of 3
138 ibitors, our findings suggest that combining DCA with a DNA methylation inhibitor would offer a means
139                                 In contrast, DCA-predicted contacts cannot be used to fold any of the
140                             Correspondingly, DCA increased cytoplasmic Ca2+ to levels that were appro
141                   The dorsal cricoarytenoid (DCA) muscles, are a fundamental component of the athleti
142 ystem based on chlorophyll fluorescence DCA (DCA-CF), and static controlled atmosphere.
143                             Dichloroacetate (DCA) is an anticancer agent that can reverse the Warburg
144                             Dichloroacetate (DCA), a compound capable of shifting metabolism from gly
145 DK) inhibitor/PDH activator dichloroacetate (DCA) normalizes HPMC metabolism, reduces lactate secreti
146 se complex (PDC) activator, dichloroacetate (DCA), would blunt activation of FOXO gene targets and re
147  and its synthetic analogue dichloroacetate (DCA).
148 Inhibiting PDK3 activity by dichloroacetate (DCA) or siRNA-mediated attenuation was sufficient to inc
149 inhibition of human PDK2 by dichloroacetate (DCA).
150 e effect of the orphan drug dichloroacetate (DCA) on survival in an animal model of hemorrhagic shock
151 he public and scientists in dichloroacetate (DCA) as a potential anticancer drug.
152 ochondrial kinase inhibitor dichloroacetate (DCA) has recently received attention in oncology due to
153 ed the potential neurotoxin dichloroacetate (DCA) was investigated using an intermediate-pressure mat
154 ained untreated or received dichloroacetate (DCA) to activate PDH and increase substrate competition
155  of PDH by cell exposure to dichloroacetate (DCA) increased production of hyperpolarized 5-(13)C-glut
156    Herein, it is shown that dichloroacetone (DCA) enhances helical secondary structures when introduc
157 e persistent pollutants 3,4-dichloroaniline (DCA) and 2,4,5-trichlorophenol (TCP).
158 ed for bioremediation of 1,2-dichloroethane (DCA) in groundwater.
159  ionic liquids (RTILs) [emim][BF(4)], [emim][DCA], and [bmim][DCA] at 25 degrees C from below 1 GHz t
160 nduced LC3-GFP vesicularization and enhanced DCA + MEK1/2 inhibitor-induced toxicity.
161 pression of p21 or p27(Kip-1) (p27) enhanced DCA + MEK1/2 inhibitor toxicity in primary hepatocytes t
162 expression of p21 or p27 profoundly enhanced DCA + MEK1/2 inhibitor-induced expression of ATG5 and GR
163 thesis of enantiomeric deoxycholic acid (ent-DCA) from achiral 2-methyl-1,3-cyclopentanedione.
164 c acid (ent-CDCA), and deoxycholic acid (ent-DCA) to induce toxicity and apoptosis in HT-29 and HCT-1
165 ve completed the successful synthesis of ent-DCA in 25 steps with a yield of 0.3% with all stereochem
166  critical micelle concentration (cmc) of ent-DCA, determined by a dye solubilization method, was iden
167 olon cancer cell lines demonstrated that ent-DCA had similar effects on proliferation, yet showed a m
168                           To further explore DCA induced changes in cell signaling, we completed a to
169 , a system based on chlorophyll fluorescence DCA (DCA-CF), and static controlled atmosphere.
170 ) storage based on chlorophyll fluorescence (DCA-CF) and respiratory quotient (DCA-RQ) on the quality
171 tient (DCA-RQ) and chlorophyll fluorescence (DCA-CF) on anaerobic metabolism, physiological storage d
172 lues for DCA, whereas binding affinities for DCA are comparable with wild-type PDK2.
173  range), although the uncertainty bounds for DCA exceeded the predicted variability.
174 stic increases in apparent IC(50) values for DCA, whereas binding affinities for DCA are comparable w
175  an electrical stimulation-based therapy for DCAs targeting the dorsal dentate nucleus.
176                                 Furthermore, DCA treatment also abrogated the clonogenic advantage co
177 onexpresser of Pathogenesis-Related genes1), DCA acts transiently and is only partially dependent on
178                                         Here DCA is applied on (i) simulated isotope patterns of the
179                                          HFD+DCA activated PDC throughout and restored whole-body CHO
180  saline (CD and HFD) or dichloroacetate (HFD+DCA).
181                                     However, DCA molecule exhibits poor bioavailability and cellular
182                                     However, DCA, associated with etoposide or irradiation, induced a
183 petition from PDH reduced anaplerosis in HYP+DCA by 18%.
184    Interestingly, reduced anaplerosis in HYP+DCA corresponded with normalized TAG (14.9+/-0.6 micromo
185 ation: HYP=1419+/-220 nmol/g dry weight; HYP+DCA=343+/-56 nmol/g dry weight.
186 cifically, we show that AREG participates in DCA-induced EGFR and STAT3 signaling, cell-cycle progres
187             In younger adults, FMT signal in DCA was lower with age, likely related to ongoing develo
188                                   Storage in DCA-CF reduces fruit ester production, especially 2-meth
189 commended for 'Fuji Suprema' apple stored in DCA conditions.
190                    The apples were stored in DCA-RQ, a new technology for storing fruits, and were co
191 amined a role for the BA transporter TGR5 in DCA-mediated EGFR and STAT3 signaling.
192 7 day initial adaptation phase of increasing DCA concentration.
193 mediated silencing of TGR5 or AREG inhibited DCA-induced EGFR, MAPK, and STAT3 signaling, blunted cyc
194 aB inhibitor, BMS-345541 (25 muM), inhibited DCA-induced HbetaD2, but not HbetaD1, release.
195  group folded only 11 of them by integrating DCA-predicted contacts into fragment-based conformation
196  hybrid method, called DCA-fold, integrating DCA contacts with an accurate knowledge of local informa
197                               Interestingly, DCA reduced C. perfringens invasion into ileum (P < 0.05
198 fects were diminished (P < 0.05) by 1.5 g/kg DCA diet.
199 A) of myoelectrical activity within the left DCA muscle in half of this horse population and the hors
200                       At the cellular level, DCA alleviated NE-associated ileal epithelial death and
201                                    Likewise, DCA was found to affect membrane distribution of caveoli
202 alogenase gene dhlA was developed to monitor DCA-degrading bacteria in the MBR, and a positive correl
203 zed compounds, we have selected the multiple DCA-loaded compound 10, characterized by a tertiary amin
204  method, was identical to the cmc of natural DCA.
205 y to induce apoptosis as compared to natural DCA.
206                                 TCA, but not DCA, activated Galpha(i) proteins in primary rat hepatoc
207                                     Notably, DCA potentiated the antitumor effects of elesclomol, a p
208 come these biases, we have synthesized novel DCA-loaded compounds.
209 cells resistant to the antitumor activity of DCA.
210                            Administration of DCA in vivo via injection or as part of a high-fat diet
211  gene abundance and the cumulative amount of DCA that had entered the MBR.
212 n studies together with a new application of DCA to the eukaryotic proteins NAF-1 and Bcl-2 provided
213                            Biodegradation of DCA in the MBR began after 26 days, and was sustained fo
214  rapid and sensitive method for detection of DCA-degrading bacteria.
215 r would offer a means to reduce the doses of DCA to avoid detrimental effects associated with high do
216 kinase resistant to the inhibitory effect of DCA, thereby uncoupling the active site from the alloste
217                  We evaluated the effects of DCA on Ca2+ signaling in BHK-21 fibroblasts using fura-2
218                                   Effects of DCA were mimicked by the Takeda GPCR 5 agonist, INT-777
219 es for ADP and ATP, mimicking the effects of DCA.
220 ack of effective mechanisms for the entry of DCA into tumor cells may underlie this phenomenon.
221 n, a Pt(II) compound, and two equivalents of DCA.
222 s it was concluded that the glucosylation of DCA may not be as effective in xenobiotic detoxification
223 ne cholesterol content observed after 4 h of DCA treatment of HCT116 cells.
224     Nonetheless, despite the clear impact of DCA and LCA on host physiology, an incomplete knowledge
225  computationally efficient implementation of DCA, which allows us to evaluate the accuracy of contact
226 fect of BBR is mediated by the inhibition of DCA biotransformation by Ruminococcus bromii.
227 l expression coinciding with the kinetics of DCA-mediated disease resistance.
228 ar differential effects on the metabolism of DCA and TCP were obtained in whole plant studies with wi
229 alogous to wild-type PDK2 in the presence of DCA and ADP.
230 tridium sporogenes, conferring production of DCA and LCA on a nonproducing commensal and demonstratin
231 Rs can be an effective method for removal of DCA from groundwater, and that the dhlA qPCR is a rapid
232 ctyl-3-methylimidazolium dicyanamide ([Omim][DCA]) inside the porous structure of ceramic membranes.
233 othioate (PS) content, linker composition-on DCA-conjugated siRNA delivery and efficacy in vivo.
234             In vitro studies using Leptin or DCA-treatment suggested causal significance of obesity-i
235                                         Oral DCA reduced peritoneal fluid lactate concentrations and
236        Importantly, mice infected with Cj-P1-DCA-Anaero showed attenuation of intestinal inflammation
237  in the colon compared to Cj-P0, while Cj-P1-DCA-Anaero showed reduction of the inflammatory gene exp
238                   Campylobacter jejuni Cj-P1-DCA-Anaero was isolated from Cj-P0-infected birds transp
239 nd then infected with Cj-P0, Cj-P1, or Cj-P1-DCA-Anaero, respectively.
240 ed to Cj-P0, which was attenuated with Cj-P1-DCA-Anaero.
241 hepatocytes with either CsA or BKA prevented DCA-induced inhibition of PTPase activity.
242  which block lipid raft formation, prevented DCA stimulation of ERK1/2.
243   In conclusion, microbial metabolic product DCA prevents NE-induced BW loss and ileal inflammation t
244 N-benzyl-3-(3,4 dihydroxyphenyl)propanamide (DCA) and phosphatidylcholine (PC) was explored in a 6-da
245 tmosphere monitored by respiratory quotient (DCA-RQ) and chlorophyll fluorescence (DCA-CF) on anaerob
246 orescence (DCA-CF) and respiratory quotient (DCA-RQ) on the quality and volatile profile of 'Royal Ga
247 tmosphere monitored by respiratory quotient (DCA-RQ) with three fruit maturity stages at harvest (ear
248                                       Regret-DCA showed that for physicians with Pt values of 3-year
249 Regret-based decision curve analysis (Regret-DCA) was performed on a Cox's regression model developed
250  function showed greater FMT signal in right DCA, independent of age effects.
251 biotic and very little metabolism to soluble DCA-glucose or associated polar conjugates.
252 ethyladenine or knockdown of ATG5 suppressed DCA + MEK1/2 inhibitor-induced LC3-GFP vesicularization
253 d DCA) and tauro-conjugated (t-CDCA, t-CA, t-DCA) form on human ENaC in its alphabetagamma- and delta
254 ECT achieves better overall performance than DCA approaches.
255 ction were less variable (2-fold range) than DCA production (5-fold range), although the uncertainty
256            In conclusion we demonstrate that DCA can be successfully used in the treatment of hemorrh
257                  We further demonstrate that DCA can differentiate between subfamilies with different
258  cell lines under BA treatment revealed that DCA and its conjugated form, TDCA, significantly inhibit
259 nally, fluorescence anisotropy revealed that DCA causes a decrease in membrane fluidity consistent wi
260                                 We show that DCA is able to shift the pyruvate metabolism in rat glio
261 ing for cholesterol with filipin showed that DCA caused a marked rearrangement of this lipid in the m
262               Molecular analysis showed that DCA significantly reduced inflammatory mediators of Infg
263         In conclusion, our data suggest that DCA-modulated anaerobes attenuate chicken-transmitted ca
264 otif in mediating communications between the DCA-, the nucleotide-, and the lipoyl domain-binding sit
265              Back-electron transfer from the DCA radical anion followed by barrierless intramolecular
266                           Dysfunction of the DCA muscle leads to arytenoid collapse during exercise,
267 he absence of fluid resuscitation; therefore DCA may be a good candidate in prolonged field care foll
268  the eight-step conversion of cholic acid to DCA.
269      One-year exposure of Barrett's cells to DCA induced a malignant phenotype noted by clone and tum
270 osure of colorectal cancer and PDAC cells to DCA resulted in colocalization of Src and TACE to the ce
271                 Minority MOMP contributes to DCA induced carcinogenesis in preneoplastic BE.
272 d sciatic nerve tissues from rats exposed to DCA.
273              Here we complete the pathway to DCA and LCA by assigning and characterizing enzymes for
274          The former phenomenon is related to DCA-induced Foxo3 and p53 expression, resulting in the o
275  in vitro antitumor activity with respect to DCA and increased in vivo stability.
276 singly, the knockouts were less sensitive to DCA.
277 dically reduced conjugating activity towards DCA and TCP and the absence of immunodetectable UGT72B1
278          Here we show that SLC5A8 transports DCA very effectively with high affinity.
279 )CHCl(2))(2)Cl(2)], mitaplatin, in which two DCA units are appended to the axial positions of a six-c
280             Contacts were predicted with two DCA methods (gplmDCA and PSICOV).
281          The storage of 'Galaxy' apple under DCA-RQ 1.3 is efficient in keeping quality regardless of
282  apples could be stored at 3 degrees C under DCA - RQ1.5.
283 r ULO; and the lowest by fruits stored under DCA-CF (DCA based on chlorophyll fluorescence).
284 h firmness was higher in fruits stored under DCA-CF and ULO differing from CA, in the year 2012, but
285  acetate), as compared to fruit stored under DCA-CF, but fruit stored under DCA-RQ 1.5 and RQ 2.0 als
286 ar quality maintenance to those stored under DCA-CF.
287  stored under DCA-CF, but fruit stored under DCA-RQ 1.5 and RQ 2.0 also showed higher amounts of key
288                           Fruit stored under DCA-RQ 2.0 accumulated the highest amounts of anaerobic
289    Data are consistent with a model in which DCA directly induces both Ca2+ release from internal sto
290         Storage at 3 degrees C combined with DCA maintained higher flesh firmness after 8.0 months st
291 ater anaerobic metabolism in comparison with DCA - CF (chlorophyll fluorescence) and controlled atmos
292                Cotreatment of melanomas with DCA and elesclomol in vivo achieved a more durable respo
293 ear DNA with cisplatin and mitochondria with DCA selectively in cancer cells.
294 ng photoinduced electron transfer (PET) with DCA as the photosensitizer.
295                             Simvastatin with DCA maintained body mass gain and food intake, abrogated
296  from Cj-P0-infected birds transplanted with DCA-modulated anaerobic microbiota.
297 to hemorrhagic injury (HI) when treated with DCA.
298                               Treatment with DCA resulted in normalization of several metabolic and m
299 mitochondria following HI and treatment with DCA.
300 um with the active open conformation without DCA and ADP.

 
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