ライフサイエンスコーパス: DCP

1 DCP also significantly reduced attack rates relative to

2 DCP EAA was significantly associated with worse visual a

3 DCP inhibitory effects were attributed to induction of a

4 DCP is typically caused by non-progressive lesions to th

5 DCP potentiated monocyte antimycobacterial activity by i

6 DCP treatment of infected monocytes resulted in a signif

7 DCP-CM is available as an online tool that can inform lo

8 DCPs ensure thereby that only mutated sequences associat

9 DCPs prevent reassociation of denatured DNA strands: the

10 7 vs 0.513 (P = .004) and 0.560 (P = .0005); DCP, 0.357 vs 0.682 (P = .016) and 0.672 (P = .0005).

11 sophila caspase, named Drosophila caspase-1 (DCP-1), was identified and found to be structurally and

12 oropicrin (CP), and 1,1-dichloropropane (1,1-DCP) after a short period of heating.

13 tic acids (HAAs), 1,1-dichloropropanone (1,1-DCP), trichloroacetaldehyde (TCAL), haloacetonitriles (H

14 the best scFv bound tightly to the UO(2)(2+)-DCP complex (K(d), 19.6 nM).

15 en cells of rabbits immunized with UO(2)(2+)-DCP conjugated to keyhole limpet hemocyanin.

16 ehalogenimonas strain is responsible for 1,2-DCP conversion in the culture.

17 d a sediment-free culture dechlorinating 1,2-DCP in the absence of methanogenesis.

18 valuable approach for monitoring in situ 1,2-DCP reductive dechlorination by Dehalogenimonas strains.

19 scribed to encode a corrinoid-containing 1,2-DCP reductive dehalogenase was detected.

20 cally dechlorinated 1,2-dichloropropane (1,2-DCP) to propene.

21 ic fractionation (epsilonC(bulk)) of the 1,2-DCP-to-propene reaction was -15.0 +/- 0.7 per thousand u

22 copies in the culture and consumption of 1,2-DCP.

23 population, while concentrations of BPA, 24-DCP, and parabens were similar.

24 s and three parabens: 2,4-dichlorophenol (24-DCP), 2,5-dichlorophenol (25-DCP), benzophenone-3 (BP-3)

25 Urinary concentrations of TCS, BP-3, and 25-DCP were higher than among women of reproductive age in

26 BP-3 had the highest (0.62), followed by 25-DCP (0.49) and TCS (0.47).

27 hlorophenol (24-DCP), 2,5-dichlorophenol (25-DCP), benzophenone-3 (BP-3), bisphenol A (BPA), triclosa

28 The 3-MCPD and 1,3-DCP contents of the analyzed food samples varied from no

29 = 43) were analyzed for their 3-MCPD and 1,3-DCP contents using a validated gas chromatography-mass s

30 risk assessment revealed that 3-MCPD and 1,3-DCP intakes in the 50th, 95th, and 99th percentiles were

31 ol (3-MCPD) and 1,3-dichloro-2-propanol (1,3-DCP) were found in domestically manufactured soy-based s

32 2,3-DCP was dechlorinated to 3-CP, and, because cultures usi

33 higher for 2,5-DCP (6.1-12.9 mug/L) than 2,4-DCP (0.8-1.0 mug/L) throughout 2003-2010.

34 ed contact time of 0.55 min, over 90% of 2,4-DCP (initially 20 muM) and 90% of adsorbable organic chl

35 6-trichlorophenol (TCP) and 2,4,5-TCP to 2,4-DCP and 3,4-DCP, respectively, and dechlorinated 2,3,6-T

36 form for the comprehensive evaluation of 2,4-DCP and posed a great potential to simplify environmenta

37 JNA converted 2,3,4-TCP to 3,4-DCP and 2,4-DCP by ortho and meta dechlorination, respectively.

38 dical oxidation by achieving much higher 2,4-DCP degradation capacity and avoiding the formation of h

39 hlorine (AOCl) can be removed at the PDS/2,4-DCP molar ratio of 1 and 4, respectively.

40 step, followed by a rapid reaction with 2,4-DCP present in the solution.

41 ully applied to evaluate the toxicity of 2,4-DCP to HepG2 cells.

42 ummed environmental phenols (2,5-DCP and 2,4-DCP) were inversely associated with age of menarche [haz

43 n, benzophenone-3, total phthalates, and 2,4-DCP) were not significantly associated with age of menar

44 2,4-Dichlorophenol (2,4-DCP), 2,5-dichlorophenol (2,5-DCP), and their precursors

45 sess the toxicity of 2,4-dichlorophenol (2,4-DCP), a priority pollutant and has potential risk to pub

46 ocatechol (3,5-DCK), 2,4-dichlorophenol (2,4-DCP), and 3,5-dichlorophenol (3,5-DCP).

47 presence of H2O2 and 2,4-dichlorophenol (2,4-DCP), is characterised via the individualised quantifica

48 ng a model compound, 2,4-dichlorophenol (2,4-DCP).

49 2,4-DCBA, 3,5-DCK, 2,4-DCP, and 3,5-DCP were quantified in >80% of all urine sa

50 inked to POD activity in the presence of 2,4-DCP.

51 bled a high electrocatalytic activity to 2,4-DCP.

52 tive concentrations of the same ([H2O2]/[2,4-DCP]/[Chl]=1:3:0.02) is crucial to explaining inhibition

53 JNA converted 2,3,4-TCP to 3,4-DCP and 2,4-DCP by ortho and meta dechlorination, respec

54 henol (TCP) and 2,4,5-TCP to 2,4-DCP and 3,4-DCP, respectively, and dechlorinated 2,3,6-TCP to 3-chlo

55 Chromosomal loci associated with 49 DCPs were confirmed by linkage analysis and tests of gen

56 ) urinary concentrations were higher for 2,5-DCP (6.1-12.9 mug/L) than 2,4-DCP (0.8-1.0 mug/L) throug

57 5-DCP) and summed environmental phenols (2,5-DCP and 2,4-DCP) were inversely associated with age of m

58 icity, we found that 2,5-dichlorophenol (2,5-DCP) and summed environmental phenols (2,5-DCP and 2,4-D

59 orophenol (2,4-DCP), 2,5-dichlorophenol (2,5-DCP), and their precursors are widely used in industry a

60 findings suggest an association between 2,5-DCP, a potential EDC, and earlier age of menarche in the

61 ted 2,3,6-TCP to 3-chlorophenol (CP) via 2,5-DCP.

62 2,4-DCBA, 3,5-DCK, 2,4-DCP, and 3,5-DCP were quantified in >80% of all urine samples with ma

63 henol (2,4-DCP), and 3,5-dichlorophenol (3,5-DCP).

64 on of a calibrated infrared pyrometer into a DCP instrument is shown to enhance the measurement capab

65 ROC curve indicated that a DCP value of 125 mAU/mL yielded the best sensitivity (89

66 quantities of beta-amyloid peptide (Abeta), DCP-LA and DHA-CP6 reduced the intracellular and secrete

67 Effective densities determined by absolute DCP for the silica particles ranged from 2.02 to 2.34 g/

68 diameter determinations provided by absolute DCP was confirmed using silica particles with nominal di

69 Advantages of absolute DCP determinations for size and density analysis relativ

70 erization of colloidal silica using absolute DCP suggests applicability of the technique to a variety

71 to 2,9-dicarboxyl-1,10-phenanthroline-acid (DCP), were produced using genetic material obtained from

72 t Reaper and Grim, but not HID, can activate DCP-1 in vivo.

73 milarly, the PKC epsilon-specific activator, DCP-LA, effectively prevents synaptic loss, amyloid plaq

74 Although active DCP-1 protein cleaves full-length DCP-1 and full-length

75 they complement AFP; and what factors affect DCP, AFP-L3%, or AFP levels.

76 AFP, DCP, and AFP-L3% levels were measured blinded to clinica

77 0 +/- 1.5% vs. 37.3 +/- 1.7%; P = 0.003) and DCP (24.4 +/- 2.3% vs. 28.0 +/- 2.3%; P < 0.001) than co

78 The SCP (P = .012) and DCP (P = .013) vessel density and perfusion density (P =

79 orrelated with SCP (r = -0.88, P = .012) and DCP capillary densities (r = -0.79, P = .048).

80 r both the SCP (42.9% vs 47.7%, P = .02) and DCP (47.4% vs 52.6%, P = .01).

81 d telangiectasias in both SCP (P = .021) and DCP (P = .042).

82 ber of ROIs were found in SCP (55 vs 39) and DCP (60 vs 49) using PLEX Elite 9000 vs AngioVue.

83 ducted to compare the performance of AFP and DCP.

84 he binding curves obtained with 8A11-Cy5 and DCP-UO22+ species changed from sigmoidal to hyperbolic a

85 drICE and DCP-1 have similar yet different enzymatic specificities

86 y which can cleave p35, lamin DmO, drICE and DCP-1 in vitro, and which can trigger chromatin condensa

87 tor caspases, including Drosophila drICE and DCP-1, suggests that in vivo activation of these group I

88 the vessel density in the perifoveal ICP and DCP and outer retinal thickness in RP patients with no h

89 ges in the perifoveal regions of the ICP and DCP in RP, with relative sparing of the SVC.

90 The ICP and DCP VD were not significantly lower in the glaucoma grou

91 ted visual acuity, PD, VD, and FD in ICP and DCP were significantly lower; and CRT, FAZ area and peri

92 mediate and deep capillary plexuses, ICP and DCP) were primarily damaged by RP, compared to superfici

93 ; and CRT, FAZ area and perimeter in ICP and DCP, and presence of cystoid macular edema, HE, and cata

94 uced retinal vessel density in SVC, ICP, and DCP compared to those in controls.

95 At baseline, EAAs for SVC, ICP, and DCP were all significantly correlated with retinopathy s

96 ion decreased significantly in SVC, ICP, and DCP.

97 n-specific immunotherapy (EPIT) with OVA and DCP also protected sensitized mice from anaphylaxis and

98 tration in both normal control (NC) rats and DCP rats, but the sensitivity of FSK on HCN channels was

99 relation was found between IOP reduction and DCP-VD at six months (p < 0.05), whereas IOP reduction c

100 CP (P < .0001 and P = .02, respectively) and DCP (P < .0001 and P = .0004, respectively) compared to

101 of retinal thinning associated with SCP and DCP flow loss (P = .03).

102 d CMT, DRIL, and DROL on SS-OCT, and SCP and DCP ischemia on SS-OCTA are significant predictors of po

103 ed CMT, DROL and DRIL on SS-OCT, and SCP and DCP ischemia on SS-OCTA contributed significantly to dim

104 The mean VD in the SCP and DCP was 46.94% (+/-3.11%) and 52.48% (+/-3.14%), respect

105 perficial and deep capillary plexus (SCP and DCP).

106 rficial and deep capillary plexuses (SCP and DCP, respectively).

107 SCP, 29 (21.5%) were visible in both SCP and DCP; and 21 (15.6%) were not visible on OCTA.

108 mproved when an appropriate adjuvant such as DCP is used.

109 eyes (28.8%) revealed capillary dilations at DCP, 15 of which were in stages 1 and 2.

110 These findings, especially at DCP, may improve our understanding about the pathogenesi

111 Two of the authors (DCP and REvdR) belong to the Coloured population, with o

112 Balanced DCP-CWGCs ensure uniform item distribution across pools,

113 t was significantly associated with baseline DCP EAA (odds ratio = 3.39, P = .002).

114 rance and rehabilitation differences between DCP-eligible vs -ineligible patients and among stratifie

115 ented work provides the missing link between DCP dispersive models and FETD and/or SETD based algorit

116 a Drosophila embryo cDNA library that block DCP-1 caspase-dependent yeast cell death.

117 Both DCP and AFP levels increased progressively from G1 to G4

118 Both DCP and AFP were tested using enzyme immunoassay methods

119 < .001), and the deep location (DCP or both DCP and SCP, P = .007) were strongly associated with the

120 Across setting-scenarios, both DCP policies led to DALYs being averted: 18 400 DALYs pe

121 s increased progressively from G1 to G4, but DCP values had less overlap among the groups than AFP.

122 dence interval [CI]: 0.77-0.84), followed by DCP (0.72, 95% CI: 0.68-0.77) and AFP-L3% (0.66, 95% CI:

123 ression but are independent of regulation by DCP-66.

124 Des-gamma-carboxyprothrombin (DCP) has been reported to be more sensitive and specific

125 Two very similar Drosophila caspases, DCP-1 and drICE, have been previously identified.

126 ertebrates and possesses two known caspases, DCP-1 and drICE.

127 d nerve agent mimic diethyl chlorophosphate (DCP) in photoluminescent (PL) spectroscopic channels.

128 onds of exposure to diethyl chlorophosphate (DCP) vapor.

129 nd 25:12.5:2.5 mole ratio of surfactant:Chol:DCP was the optimum formulation in the encapsulation of

130 -2',2'-dimethyldihydropyrano[2,3-f]chromone (DCP, 4) were designed and synthesized.

131 In conclusion, DCP was more sensitive and specific than AFP for differe

132 Dynamic cystocolpoproctography (DCP) has evolved from a method of evaluating the anorect

133 r weak contractility of Diabetic cystopathy (DCP) and to study the possible mechanism of regulating t

134 abnormalities at superficial (SCP) and deep (DCP) capillary plexuses and choriocapillaris (CC) in pat

135 e level either of superficial (SCP) or deep (DCP) capillary plexus and the presence of flow on the co

136 We detected DCPs in at least 81% of participants.

137 7 for 4-Cl-Ph-O-dG and 2,6-dichloro-Ph-O-dG (DCP-O-dG), respectively.

138 ted pentachlorophenol to 3,5-dichlorophenol (DCP) via removal of the ortho and para chlorines in all

139 ary concentrations of these dichlorophenols (DCPs) have been measured as part of four National Health

140 we tested the efficacy of dichlorphenamide (DCP; Daranide), a potent carbonic anhydrase inhibitor, i

141 simetrically detects diethylchlorophosphate (DCP), a model organophosphorous cholinesterase inhibitor

142 djuvant potential of diphenylcyclopropenone (DCP), a strong contact sensitizer, which is currently us

143 al. show that DIAP1 polyubiquitinates DRONC, DCP-1, and drICE, leading to their nondegradative inacti

144 rence (in terms of the end point) for either DCP or placebo, and 11 of these preferred DCP.

145 2-Ethyl DCP (8) exhibited the best anti-HIV activity in both ass

146 coding an Ex-1 binding transcription factor, DCP-66, using a yeast one-hybrid screen.

147 00 DALYs per year (95% CI 16 700-20 100) for DCP including cabotegravir and 56 400 DALYs per year (52

148 ts over 50 years: $8.6 million (7.7-9.4) for DCP without cabotegravir and $13.2 million (11.6-14.8) f

149 nd 56 400 DALYs per year (52 300-60 500) for DCP without cabotegravir in 10 million adults.

150 botegravir and $13.2 million (11.6-14.8) for DCP including cabotegravir.

151 ncreased the affinity of the native 8A11 for DCP-UO22+ by 3-fold.

152 sed the affinity of the primary antibody for DCP-UO22+ by 4-fold.

153 r operating characteristic curve (AUROC) for DCP was 0.99 and was significantly larger than that of A

154 the incremental cost-effectiveness ratio for DCP including cabotegravir (vs no DCP) was $234 per DALY

155 ow cutoff was 43% and 94%, respectively, for DCP and 47% and 75%, respectively, for AFP.

156 ith CFT, IRT, ORT, foveal SCP-VD, and foveal DCP-VD and a significant positive correlation with subfo

157 I, CFT, IRT, ORT, foveal SCP-VD, and foveal DCP-VD were significantly greater than those in the othe

158 less tightly (K(d), 23.5 muM) to metal-free DCP.

159 Hence, DCP acts as an immunoregulatory compound enhancing the a

160 us (DCP), and all-plexus retina (SVC + ICP + DCP).

161 tigen in the absence of protein G, and (iii) DCP-UO22+ and protein G oppose each other's binding to t

162 In DCP, two major movement disorders, dystonia and choreoat

163 iated with increased severity of ischemia in DCP.

164 m region decreased in SVC and ICP but not in DCP.

165 n HCN channels was clearly down-regulated in DCP rats.

166 dentified predictors and evaluated trends in DCP concentrations according to race/ethnicity, age, sex

167 thione S-transferase-DIAP1 directly inhibits DCP-1 caspase activity but that it had minimal effect on

168 ugh active DCP-1 protein cleaves full-length DCP-1 and full-length drICE in vitro, GMR-DeltaN-dcp-1 d

169 isibility (P < .001), and the deep location (DCP or both DCP and SCP, P = .007) were strongly associa

170 spite the advances in other imaging methods, DCP has remained a practical, cost-effective procedure f

171 n our Disease Control Priorities Cost Model (DCP-CM), developed as part of the DCP3 project to determ

172 se, only a persistent weekly use of multiple DCPs over time seemed to have an adverse effect on asthm

173 d in the detection of early HCC, but neither DCP nor AFP is optimal.

174 ratio for DCP including cabotegravir (vs no DCP) was $234 per DALY averted.

175 Compared with no DCP, there was a mean increase in annual discounted cost

176 In this study, we tested the ability of DCP to mediate inhibition of intracellular mycobacteria

177 The antimicrobial activity of DCP was comparable to that of pyrazinamide (PZA), one of

178 d Participants: A longitudinal assessment of DCP implementation and Medicaid expansion/open enrollmen

179 stic regressions to estimate associations of DCP concentrations above the 95th percentile with housin

180 conjugate with saturating concentrations of DCP-UO22+ decreased the affinity of the conjugate for pr

181 oacetate) reversed the inhibitory effects of DCP on intracellular mycobacterial growth.

182 ate, (ii) binding of the first equivalent of DCP-UO22+ to the antibody promotes the binding of the se

183 e energy binding model in which (i) 2 mol of DCP-UO22+ and 1 mol of protein G bind to each mole of th

184 ICCs-DM is important in the pathogenesis of DCP.

185 this study were to determine performance of DCP and AFP-L3% for the diagnosis of early HCC; whether

186 ace/ethnicity were significant predictors of DCP urinary concentrations above the 95th percentile.

187 tudying the wideband plasmonic properties of DCP media.

188 Prospective studies to evaluate the role of DCP in early HCC are underway.

189 The sensitivity and specificity of DCP at month 0 was 74% and 86%, respectively, at a cutof

190 The sensitivity and specificity of DCP were 96.8% and 98.3% respectively, based on a Receiv

191 target for the pharmacological treatment of DCP.

192 In conclusion, the utility of DCP for the diagnosis of HCC among Nigerian patients was

193 cuss the limitations and clinical utility of DCP.

194 survey cycle with urinary concentrations of DCPs during NHANES 2003-2010.

195 Only DCP vessel density significantly correlated with the sta

196 ary nonperfusion at the superficial (SCP) or DCP, we used the spectral-domain optical coherence tomog

197 temporary and permanent dual-chamber pacing (DCP) were evaluated in symptomatic pediatric patients wi

198 Dyskinetic cerebral palsy (DCP) is the second most common type of cerebral palsy af

199 nges in the drying/crystallization patterns (DCP) of an array of microdroplets containing solutions o

200 Dipterinyl calcium pentahydrate (DCP), a calcium-complexed pterin compound, has previousl

201 cribe the technique they use when performing DCP, define the radiographic criteria used for diagnosis

202 Permanent DCP is an effective therapy for selected pediatric patie

203 Permanent DCP pacing can reduce left ventricular outflow tract (LV

204 derwent surgical implantation of a permanent DCP system.

205 exed with 2,9-dicarboxy-1,10-phenanthroline (DCP-UO22+).

206 iated with differential chemical phenotypes (DCPs), defined as greater than or equal to fivefold diff

207 of cholesterol (Chol) and dicetyl phosphate (DCP) as well as different concentration of alpha-tocophe

208 Disc centrifuge photosedimentometry (DCP) with fluids of different densities is used to simul

209 A specific activator of PKCepsilon, DCP-LA, slowed CBF after maximal PKCepsilon activation.

210 ere assessed with the Direct Current Plasma (DCP) technique and its distribution assessed with Morin

211 cation-density based crystalline plasticity (DCP) and nonlinear finite element (FE) analysis were use

212 ascular plexus (SVP), deep capillary plexus (DCP) and choriocapillaris (CC) as detected on optical co

213 plexus (SCP) and the deep capillary plexus (DCP) did not correlate with each other in all parameters

214 lary plexus (SCP) and deep capillary plexus (DCP) in the central macula in all 6 patients were compar

215 the level of SCP and deep capillary plexus (DCP) on OCTA.

216 lary plexus (SCP) and deep capillary plexus (DCP) vessel density (VD), and foveal avascular zone (FAZ

217 lary plexus (SCP) and deep capillary plexus (DCP) were assessed using OCT angiography (OCTA).

218 pillary plexus (ICP), deep capillary plexus (DCP), and all-plexus retina (SVC + ICP + DCP).

219 pillary plexus (SCP), deep capillary plexus (DCP), and full retinal projections were obtained using a

220 pillary plexus (SCP), deep capillary plexus (DCP), choriocapillaris (CC)] was calculated at baseline

221 chemia of the retinal deep capillary plexus (DCP).

222 lary plexus (SCP) and deep capillary plexus (DCP).

223 ary plexus (ICP), and deep capillary plexus (DCP).

224 ary plexus (ICP), and deep capillary plexus (DCP).

225 perficial (SCP) and deep capillary plexuses (DCP).

226 , middle (MCP), and deep capillary plexuses (DCP): parafoveal vessel density (VD), adjusted flow inde

227 ficial (SCP-VD) and deep capillary plexuses (DCP-VD) of the foveal and parafoveal areas were examined

228 tectures comprised of Drude-Critical Points (DCP) media (e.g., gold and silver) is proposed and valid

229 ighted average equivalent exposure of 10 ppb DCP effects an irreversible change in smartphone readout

230 er DCP or placebo, and 11 of these preferred DCP.

231 egy relying on the use of DNA clutch probes (DCPs) that render specific sequences of ctDNA accessible

232 e example of the disordered contact process (DCP) of infection spreading in heterogeneous systems.

233 e pre-mRNA, the downstream cleavage product (DCP) is degraded by the 5'-3' exonuclease activity of CP

234 g cleavage, the downstream cleavage product (DCP) is rapidly degraded in vitro by a nuclease that als

235 significantly lower in all but 1 projection (DCP).

236 we show three orthologs of P body proteins, DCP-2, CAR-1 and CGH-1, and two markers of stress granul

237 ein (AFP) and des-gamma-carboxy prothrombin (DCP) in the early diagnosis of HCC.

238 Des-gamma carboxy-prothrombin (DCP) and lectin-bound AFP (AFP-L3%) are potential survei

239 Des-gamma carboxy-prothrombin (DCP) has been reported to be more sensitive and specific

240 including the Dependent Coverage Provision (DCP) and Medicaid expansion/open enrollment, and to cons

241 valuate reperfusion patterns within the SCP, DCP, and CC.

242 Several DCP analogues (4, 5, 7, 8, 13, and 17) exhibited extreme

243 Even more promisingly, some DCP analogues also showed activity against a multi-RT in

244 nd density analysis relative to standardized DCP measurements include the elimination of instrument s

245 negative surface charge than inactive strain DCP-Ps1.

246 d by metabolically active cultures of strain DCP-Ps1, which at similar values of SIcalcite, have a mo

247 The ability of Pseudomonas stutzeri strain DCP-Ps1 to drive CaCO3 biomineralization has been invest

248 and 4-methyl DCK (2), all newly synthesized DCP analogues (4-21) were screened for anti-HIV-1 activi

249 AFP was more sensitive than DCP and AFP-L3% for the diagnosis of early and very earl

250 We conclude that DCP is effective in the prevention of episodic weakness

251 These findings indicate that DCP induced mycobacterial killing via MIP-1beta- and nit

252 We now show that DCP-1 has a substrate specificity that is remarkably sim

253 aenorhabditis elegans CED-3, suggesting that DCP-1 is a death effector caspase.

254 The DCP and Chol improved the physicochemical properties of

255 The DCP-CM was informed primarily by published estimates of

256 f capillary non perfusion in the SCP and the DCP (p = 0,026 and p = 0,02 respectively) and larger FAZ

257 s had more microaneurysms in the SCP and the DCP (p = 0,039 and p = 0,024 respectively), more IRMA in

258 foveal capillary ischemia in the SCP and the DCP.

259 FAZ area was enlarged at the DCP (P = .001).

260 clease, suggesting that CPSF-73 degrades the DCP both in vitro and in vivo.

261 characterize the activity that degrades the DCP.

262 ty is processive and continues degrading the DCP substrate even after complete removal of the U7-bind

263 In HbSS eyes, VD was lower in the DCP (47.7%, P = .008) but not in the SCP (45.5%, P = .5)

264 FA (P = .016), exclusively localized in the DCP (P = .016).

265 e AngioVue in the SCP (p = 0.005) and in the DCP (p = 0.027).

266 146), while no differences were noted in the DCP (p = 0.2717) nor in the CC (p = 0.6848).

267 g substantial protein-induced changes in the DCP as "1" and those that show no or small changes as "0

268 pre-injection images, vessel density in the DCP was 68.8% and 78.6% of baseline in monkey 1 and monk

269 s (p = 0,048 in the SCP and p = 0,012 in the DCP).

270 on OCTA; 76 (56.3%) were visible only in the DCP, 9 (6.7%) only in the SCP, 29 (21.5%) were visible i

271 ted with the number of microaneurysms in the DCP, the surface of capillary non perfusion areas and th

272 n the SVP and CC appeared sooner than in the DCP.

273 spectively; P = 0.92), vessel density of the DCP (54.1+/-3.3% and 54.0+/-1.8%, respectively; P = 0.93

274 gnificant difference in the VD and PD of the DCP (VD: p = 0.015; PD: p = 0.014), and for the PD of th

275 HeLa cells indicate that degradation of the DCP does not depend on the Xrn2 5' exonuclease, suggesti

276 ASH antibodies, the 5'-3' degradation of the DCP is not affected.

277 to enhance the measurement capability of the DCP technique by correcting for the temperature dependen

278 tant in highlighting the contribution of the DCP to the oxygen requirements of the photoreceptors as

279 Before implementation of the DCP, 1 of 4 patients was uninsured.

280 ry plexus, PAMM is caused by ischemia of the DCP, and appears as hyper-reflective, band-like lesions

281 nges on SDOCT showed robust perfusion of the DCP.

282 e-mRNA and the subsequent degradation of the DCP.

283 f capillary nonperfusion at the level of the DCP.

284 pillaries, and capillary nonperfusion of the DCP.

285 VD in the 6 x 6 mm(2) SCP scans, the DCP (all scans), and FAZ metrics showed no significant d

286 nd altered patient outcomes in ways that the DCP alone was never intended to do.

287 V cross-linking studies demonstrate that the DCP and its 5'-truncated version specifically interact w

288 f value was 11 ng/mL and was inferior to the DCP value of 125 mAU/mL, the area under the ROC curves b

289 ressure declined, the flow signal within the DCP appeared initially as dots, which progressed lateral

290 Adjusted GM concentrations of the DCPs among children (6-11 years of age) and adults > 60

291 g three genes that were linked to 96% of the DCPs.

292 Addition of long-acting cabotegravir PrEP to DCP was cost-effective (vs DCP without cabotegravir); th

293 When the microbeads are subjected to DCP vapor, the conversion of FLA into a phosphoramide ca

294 Exposure to DCPs and their precursors was prevalent in the general U

295 In the PSPP trial, DCP significantly reduced attack rates relative to place

296 Furthermore, urinary DCP concentrations have showed a downward trend since 20

297 otegravir PrEP to DCP was cost-effective (vs DCP without cabotegravir); the incremental cost-effectiv

298 ional case control study to evaluate whether DCP is more sensitive and specific than AFP for differen

299 Binding curves obtained with DCP-UO22+ and the bivalent (Fab)2 or the monovalent Fab

300 488 altered the binding curves obtained with DCP-UO22+ from hyperbolic to sigmoidal, the latter chara

301 n nanotube based chemidosimetric scheme with DCP limits of detection of 28 ppb.

302 Loss of zygotic DCP-1 function in Drosophila caused larval lethality and