ライフサイエンスコーパス: DD

1 DD achieved a remission rate (RR) of 64% and a very good

2 DD offers a convenient alternative approach to BMD metho

3 DD represents an ideal human model disease for fibrosis

4 DD therapy also further downregulated BALF ILs and cytok

5 DD was considered negative (DD-) if <500 ng/mL.

6 DD, but not VD, MNs reverse their cellular polarity in a

7 DD-study: Diet or Diuretics for Salt-sensitivity in Chro

8 for ruling out AAS in patients with ADD-RS=0/DD- or ADD-RS </=1/DD-.

9 In 294 patients with ADD-RS=0/DD-, 1 case of AAS was observed.

10 Improvement in DD grade/grade 1 DD at 30 days post-TAVR was seen in 70.8% patients.

11 Improvement in DD grade/grade 1 DD at 30 days was protective for 1-year CV death/rehospi

12 with improvement in >=1 grade of DD/grade 1 DD had reduced 1-year CV death/rehospitalization (p < 0.

13 the 1,750 included, 682 (54.4%) had grade 1 DD, 352 (28.1%) had grade 2 DD, 168 (13.4%) had grade 3

14 9.9% (95% CI, 47.7-52.2) for the ADD-RS </=1/DD- strategy.

15 In 924 patients with ADD-RS </=1/DD-, 3 cases of AAS were observed.

16 in patients with ADD-RS=0/DD- or ADD-RS </=1/DD-.

17 35 subjects with first-episode psychosis (11 DD, 12 SZ, 12 other psychotic disorders) and 19 healthy

18 m June 2013 to June 2015, we identified 1768 DD livers exported to regional candidates with MELD scor

19 .4%) had grade 1 DD, 352 (28.1%) had grade 2 DD, 168 (13.4%) had grade 3 DD, and 51 (4.1%) had indete

20 Baseline grade 3 DD was a predictor of 1-year CV death/rehospitalization

21 .1%) had grade 2 DD, 168 (13.4%) had grade 3 DD, and 51 (4.1%) had indeterminate DD grade.

22 Common Terminology Criteria [CTC] grade 3/4: DD 16%, DVD 18%) and likely related to a high rate of ly

23 high rate of lymphocytopenia (CTC grade 3/4: DD 20%, DVD 17%).

24 iable method of producing cultures of >/=90% DD Mtb in starved cells.

25 P until a candidate is eligible to receive a DD-CIK.

26 tude lower than that of the corresponding AA.DD duplex.

27 ion is observed for homo-sequence 2-mers (AA.DD) where there are no competing folding equilibria.

28 e for Paired Kidney Donation, and the actual DDs from the Scientific Registry of Transplant Recipient

29 e Statistics database and included all adult DD patients who were surgically treated.

30 ystemic complications and re-operation after DD surgery.

31 On restarting SD after DD treatment, a possible carryover effect was seen for s

32 as: deceased-donor kidney transplant alone (DD-KA, 68%), living-donor kidney transplant alone (LD-KA

33 ROR: 2.24, 95% CI: 1.09-4.61, p = 0.029 and DD = +9.1 pp, ROR: 2.65, 95% CI: 1.01-6.98, p = 0.048) a

34 polysorbate 80 levels were tested by BMD and DD.

35 TP53+/+ cells compared to TP53-/- cells and DD-Cas9, suggesting that differences in break sensing ar

36 With EVD and DD, a specific clinicopathologic diagnosis was made in 2

37 opsy specimens examined in 2015 with EVD and DD.

38 lized panels; Sensititre; Thermo Fisher) and DD (30 mug; research-use-only [RUO] MASTDISCS and FDA-cl

39 d of high energy lipid classes, while HD and DD tissues contained greater proportions of structural l

40 d possible homology, between hippocampal and DD/DL circuitry.

41 16 individuals with mild to profound ID and DD and a de novo mutation in PPP2CA, encoding the cataly

42 were tested as comparator agents for MIC and DD methods, respectively.

43 ives as aromatic C(sp(2))-H nucleophiles and DDs as acceptors.

44 cts were categorized into +DD ( n = 29) and -DD ( n = 18) groups.

45 thophysiology of psychotic disorders such as DD and SZ.

46 R); and two non-arginyl dipeptides: Asp-Asp (DD) and Glu-Asp (ED).

47 een Fas death domain (DD) and Fas-associated DD (FADD).

48 In the PARTNER 2 SAPIEN 3 registry, baseline DD was a predictor of up to 2 years clinical outcomes in

49 We describe the connection between DD and global assortativity of the network from both for

50 peripherally NBN-doped PAHs to form H-bonded DD.AA- and ADDA.DAAD-type complexes with suitable comple

51 binding studies of the first double-H-bonded DD.AA-type complexes of a series of aromatic boronic aci

52 Both DD methods performed poorly for Acinetobacter baumannii

53 es were susceptible by Etest, 70% (67/96) by DD, and 88% (84/96) by agar dilution.

54 We calculated DD prevalence for each definition and determined the ove

55 Cefoxitin DD performed best when interpreted with the CLSI SOSA br

56 Likewise, oxacillin BMD and cefoxitin DD tests using the coagulase-negative Staphylococcus spe

57 . schleiferi and oxacillin BMD and cefoxitin DD tests using the CoNS breakpoints reliably identified

58 For cefoxitin DD and BMD results interpreted using S. aureus/S. lugdun

59 There were 4.9% VMEs for cefoxitin DD with 0% MEs, and 3.6% VMEs and 3.9% MEs for cefoxitin

60 connections between land cover and childhood DD, we compiled a database of 293,362 children in 35 cou

61 ier to seeking healthcare for sick children (DD = -26.4 pp, ROR: 0.23, 95% CI: 0.08-0.66, p = 0.006).

62 e "low-cost" tools to evaluate DD-a clinical DD score, the balloon expulsion test (BET) and a digital

63 , constipated patients answered the clinical DD score and were evaluated by DRE and BET [standard and

64 The clinical DD score revealed an AUC of 0.417 (SE = 0.07, p = 0.191)

65 ry) to diseased tissue on diseased colonies (DD; i.e. lesion front) was observed, demonstrating a red

66 Under these conditions, DD and DH were 39.40 and 2.11%, respectively.

67 DAPK1-DD-deficient mice were more susceptible to tumor growth

68 , which are essential for constant darkness (DD) rhythmicity.

69 ed on reaching a high degree of deamidation (DD) with a simultaneously low degree of hydrolysis (DH).

70 ; LD-KA: aHR (1.29) 1.87(2.71) ) and death (DD-KA: aHR (2.12) 3.25(5.00) ; LD-KA: aHR (1.54) 2.35(3.

71 concentration of trans,trans-2,4-decadienal (DD) during the light cycle.

72 tual disability (ID) or developmental delay (DD) and short stature.

73 ldren with ASD (N=415), developmental delay (DD) without ASD (N=188), and general population (GP) con

74 ual disability (ID) and developmental delay (DD).

75 ess than that of the unmodified drug depots (DDs).

76 tagonist SFRP4, in surgical specimen-derived DD myofibroblasts.

77 generating such "differentially detectable" (DD) Mtb in vitro would aid studies of the biology and dr

78 receiving either daratumumab/dexamethasone (DD, n = 106) or daratumumab/bortezomib/dexamethasone (DV

79 2 and two degradation domains (FKBP-DD, DHFR-DD), in multiple orientations, and determined (i) the fo

80 e measured the vertical optic disc diameter (DD) of the UK Biobank (UKBB) cohort (N = 67 040) and per

81 calization, and overexpression of a Dia1 DID-DD fragment competitively removed Dia1 and Dia2 from cel

82 In Dia1 DID-DD-overexpressing cells, Dia1 and Dia2 were mislocalized

83 s inhibitory domain-dimerization domain (DID-DD) region of Dia1 was sufficient for Dia1 localization,

84 itions of indoles with 1,2-diaza-1,3-dienes (DDs) is reported.

85 ent of resorcinols and 1,2-diaza-1,3-dienes (DDs) under acidic conditions.

86 othioamides (ATAs) and 1,2-diaza-1,3-dienes (DDs).

87 underexplored measure, 'degree difference' (DD) between vertices of an edge, to understand the local

88 Difference-in-differences (DD) estimates of impact were calculated, adjusting for c

89 Typically, edges with different DD play different structural roles and the DD distributi

90 uate oxacillin and cefoxitin disk diffusion (DD) and broth microdilution (BMD) methods for the detect

91 omycin Etest and Kirby-Bauer disk diffusion (DD) assays, and the results were compared to those obtai

92 n broth microdilution (BMD), disk diffusion (DD), and PBP2a immunoassay, and the results were compare

93 D-dimer (DD) is highly sensitive for AAS but is inadequate as a s

94 and patients grouped by this and by D-dimer (DD) levels.

95 classes of photoproducts-dibenzo-p-dioxins (DDs) and 2,2'-dihydroxylated biphenyls-was also investig

96 lopmental delay and intellectual disability (DD and ID) are heterogeneous phenotypes that arise in ma

97 mental delay and/or intellectual disability (DD and/or ID) disease phenotypes.

98 Delay discounting (DD), the tendency to discount the value of delayed versu

99 Moreover, we discovered DD-03-171, an optimized lead compound that exhibits enha

100 Individuals with Dupuytren disease (DD) are commonly seen by physicians and surgeons across

101 Salt-sensitivity in Chronic Kidney Disease (DD), NCT02875886.

102 Dupuytren's disease (DD) is a common fibro-proliferative disorder of the palm

103 ssue from patients with Dupuytren's disease (DD), a localized fibrotic disorder of the palm, we sough

104 A defecation disorder (DD) is a difficulty in evacuation documented by physiolo

105 chosis, such as between delusional disorder (DD) and schizophrenia (SZ).

106 on binding, as well as disorder-to-disorder (DD) transitions, when the conformational heterogeneity i

107 ole of RT in severe developmental disorders (DD) has not yet been explored.

108 Depressive disorders (DDs), anxiety disorders (ADs), obsessive-compulsive diso

109 7833 probands with developmental disorders (DDs) and their unaffected parents, as well as more than

110 ne individuals with developmental disorders (DDs).

111 g (MODY) and severe developmental disorders (DDs).

112 oromandibular joint (TMJ) disc displacement (DD) and degenerative joint disease (DJD) has never been

113 without (-) bilateral TMJ disc displacement (DD).

114 tative progenitors, Brachypodium distachyon (DD) and Brachypodium stacei (SS).

115 to the interphase between Fas death domain (DD) and Fas-associated DD (FADD).

116 is and cell death that recruit death domain (DD) proteins FADD and TRADD and caspases to form death-i

117 RIPK1 is a death-domain (DD) containing kinase involved in regulating apoptosis,

118 ll molecule by using a destabilizing domain (DD).

119 In this study, the module docking domain (DD) affinity prediction performance on a hold-out testin

120 ely diagnostic de novo mutations in dominant DD-associated genes at noncanonical positions in splice

121 ocation system that includes deceased donor (DD) kidneys as chain-initiating kidneys (DD-CIK) in a ki

122 Under Share 35, deceased donor (DD) livers are offered regionally to candidates with Mod

123 malizing AAT trough levels with double-dose (DD) therapy (120 mg/kg/wk) in subjects with AATD already

124 Ex vivo dermoscopy (EVD) with derm dotting (DD) improves clinicopathologic correlation and the quali

125 e association between diastolic dysfunction (DD) and outcomes in patients with aortic stenosis underg

126 Diastolic dysfunction (DD) is an independent predictor of HFpEF risk, associate

127 left ventricular (LV) diastolic dysfunction (DD), with 25% exhibiting grade III/IV restrictive DD wit

128 is often preceded by diastolic dysfunction (DD).

129 Developmental dyslexia (DD) is a learning disorder affecting the ability to read

130 of eight cases with developmental dyslexia (DD) revealed several regions shared by the affected indi

131 nto the American Society of Echocardiography DD groups.

132 whereas no effect was observed with RE, ER, DD, or ED.

133 Three "low-cost" tools to evaluate DD-a clinical DD score, the balloon expulsion test (BET)

134 her odds of delivering in a health facility (DD = +10.6 pp, ROR: 1.53, 95% CI: 1.10-2.13, p = 0.012)

135 tly higher risk of kidney allograft failure (DD-KA: aHR (1.53) 2.20(3.17) ; LD-KA: aHR (1.29) 1.87(2.

136 flurane hindered the interaction between Fas DD and FADD.

137 e is known to play a significant role in Fas DD-FADD interaction, supporting our in vitro and in vivo

138 The effect of sevoflurane on Fas DD-FADD interaction was examined using fluorescence reso

139 with ERT2 and two degradation domains (FKBP-DD, DHFR-DD), in multiple orientations, and determined (

140 wed that Shld1, the chemical inducer of FKBP-DD, does not interfere with stem cell differentiation, w

141 We found that the FKBP-DD confers the PB transposase with a higher transpositio

142 In addition, we found that the FKBP-DD regulates transposon activity in a reversible and dos

143 For DD methods, EUCAST BPs demonstrated lower susceptibility

144 ated an SNP-based heritability of 20-25% for DD, and observed significant associations of dyslexia ri

145 very major errors (VME) were calculated for DD methods.

146 nsider this gene as a putative candidate for DD.

147 39% of the study sample as indeterminate for DD.

148 ,488 adults underwent 158,119 operations for DD.

149 ing to be the most significant predictor for DD screening.

150 [SD] age, 12.9 [5.1] years); 17 studies for DD, 10 for AD, 8 for OCD, and 1 for PTSD.

151 evaluated as separate or combined tests for DD screening.

152 search that may lead to novel treatments for DD and fibrosis more generally.

153 Calculated degradation rates were slower for DDs and faster for dihydroxylated biphenyls in compariso

154 ere significantly larger (all P < 0.01) for +DD compared to -DD subjects by 1.7-, 2.5-, and 1.9-fold

155 or the photodegradation mechanism that forms DDs and dihydroxylated biphenyls.

156 ith ASD might be etiologically distinct from DD without ASD.

157 to babies with birth weights (BWs) <2,500 g (DD = -11.8, ROR: 0.29, 95% CI: 0.10-0.82, p = 0.020).

158 (WES) with Developmental Disorders G2P (G2P(DD); 2044 entries).

159 VEP-G2P(DD) shows a sensitivity/precision of 97.3%/33% for de no

160 In contrast, thioridazine did not generate DD Mtb from starved cells but killed those generated by

161 e describe 15 unrelated individuals who have DD and/or ID, central nervous system (CNS) dysfunction,

162 UCTs had a protective effect on HAZ (DD = +0.25 z-scores, 95% confidence interval [CI]: 0.01-

163 Hence, DD-treated cells utilized transcriptional and mRNA stabi

164 Patients with low ML and high DD demonstrated a hyperfibrinolytic biomarker profile, l

165 Conversely, patients with low ML but high DD levels are severely injured, functionally coagulopath

166 aumatic brain injury and had a 7-fold higher DD level (died vs. survived: 103,170 vs. 13,672 ng/mL, P

167 -BR, wet-dry hulling-WD, and dry-dry hulling-DD) and a standard roasting treatment.

168 Additionally, we identify three candidate ID/DD-associated genes of which two have an established rol

169 l disability and developmental disorders (ID/DD) and successfully identify 15 genes with clustering m

170 Response to placebo is large, especially in DD.

171 Improvement in DD grade at 30 days was associated with improvement in s

172 Improvement in DD grade/grade 1 DD at 30 days post-TAVR was seen in 70.

173 Improvement in DD grade/grade 1 DD at 30 days was protective for 1-year

174 acteraceae-affiliated sequences increased in DD tissues, suggesting opportunistic proliferation of th

175 neral population, in which mean ACE level in DD carriers is approximately twice that in II carriers,

176 ) of 44%, higher than previously reported in DD liposarcoma.

177 We also showed that 12% of the variance in DD was accounted for by genotype and that the genetic si

178 larger (all P < 0.04) by up to 8.5-fold in +DD compared to -DD subjects.

179 ficantly larger ( P = 0.012) by 1.4-fold in +DD compared to -DD subjects.

180 ess to DD-CIK of 6 months or less, including DD-CIKs increases the number of transplants by at least

181 grade 3 DD, and 51 (4.1%) had indeterminate DD grade.

182 consensus diagnosis of normal/indeterminate, DD with reduction, or DD without reduction.

183 8, p = 0.048) and household food insecurity (DD = -10.7 pp, ROR: 0.63, 95% CI: 0.43-0.91, p = 0.016).

184 Forty-seven subjects were categorized into +DD ( n = 29) and -DD ( n = 18) groups.

185 CI: 1.32-2.62, p < 0.001) and physical IPV (DD = -7.9 pp, ROR: 0.60, 95% CI: 0.36-0.99, p = 0.048).

186 rall ubiquitination pattern of RIPK1 and its DD-mediated interactions with other DD-containing protei

187 1 activation is known to be regulated by its DD-mediated interaction and ubiquitination, though under

188 or (DD) kidneys as chain-initiating kidneys (DD-CIK) in a kidney paired donation pool (KPDP), and est

189 ffects were also found on women's knowledge (DD = +14.8, ROR: 1.86, 95% CI: 1.32-2.62, p < 0.001) and

190 construct that encodes the Lhx2-HD and Ldb1-DD (dimerization domain) in a single transcript cell-aut

191 Patients presenting with low ML and low DD levels have low injury severity and normal outcomes.

192 C, including PlsX and KASII (FA metabolism), DD-transpeptidase and MurB (peptidoglycan synthesis), gl

193 compare cefiderocol disk diffusion methods (DD) to broth microdilution (BMD) for AST of Gram-negativ

194 Other genes modulating DD respecification such as lin-14, irx-1, and oig-1 are

195 e that m154 uses its cytoplasmic tail motif, DD, to interfere with the adaptor protein-1 (AP-1) compl

196 DD was considered negative (DD-) if <500 ng/mL.

197 e such tool, GeneMatcher, we identified nine DD- and/or ID-affected probands with a rare, heterozygou

198 Notably, DD is the basic unit of assortativity.

199 Application of DD-SRM to human serum and tissue provides precise quanti

200 te the different clinical characteristics of DD and schizophrenia (SZ), antipsychotics are deemed equ

201 xample, we predict that, within the class of DD motor neurons, only three (DD04, DD05, or DD06) shoul

202 niform, reliable, and accurate definition of DD for epidemiologic studies is needed.

203 concordance of 4 established definitions of DD in the Coronary Artery Risk Development in Young Adul

204 and clinical factors and the definitions of DD.

205 line joint-specific soft tissue diagnoses of DD, 598 (76%) joints showed no change; 109 (14%) demonst

206 als with a surgically validated diagnosis of DD from the UK, with replication in British, Dutch, and

207 ignificant increases in the zone diameter of DD testing for isolates that were fosA positive compared

208 BET performance regarding the evaluation of DD, with a sensitivity of 86%, specificity of 67%, PPV o

209 whether they contribute to the generation of DD Mtb or kill them.

210 Patients with improvement in >=1 grade of DD/grade 1 DD had reduced 1-year CV death/rehospitalizat

211 Incremental baseline grades of DD were associated with an increase in combined 1- and 2

212 gest genome-wide association study (GWAS) of DD to date.

213 Within 3 h of DD exposure, Lhcf2 mRNA levels and transcription were st

214 ate significant variability in prevalence of DD and its association with established risk factors acr

215 Prevalence of DD ranged from 2% to 32% across the 4 definitions, with

216 We evaluated the prevalence of DD using previously published definitions in a populatio

217 over is associated with lower probability of DD downstream.

218 y genotype and that the genetic signature of DD overlapped with attention-deficit/hyperactivity disor

219 was to assess the longitudinal stability of DD and DJD among 401 subjects.

220 rs (SNRIs), and placebo for the treatment of DD, AD, OCD, and PTSD in children and adolescents.

221 weeks of SD therapy, followed by 4 weeks of DD therapy, and 4 weeks after return to SD therapy.

222 Based on DD connectivity, we illustrate the close similarity, and

223 A model based on DD-SIMCA was also developed and applied to the control o

224 and appears to be dispensable in equinox or DD conditions.

225 ney graft and patient survival than LD-KA or DD-KA recipients.

226 ID from mothers of children with ASD-noID or DD suggests that the intellectual disability associated

227 normal/indeterminate, DD with reduction, or DD without reduction.

228 and its DD-mediated interactions with other DD-containing proteins.

229 Overall, DD is a local measure that is simple to define, easy to

230 Overall, DD surgery performed in England was safe; however, re-op

231 Oxacillin DD tests using the Clinical and Laboratory Standards Ins

232 Oxacillin DD yielded high ME rates (20.7 to 21.7%) using CLSI or E

233 Our findings demonstrate that oxacillin DD tests using the CLSI M100-S28 breakpoints for S. pseu

234 Compared to SPK,P+, DD-KA, or LD-KA recipients had significantly higher risk

235 efective p27pT157pT198 phosphomimetic (p27CK-DD), cJun is activated and interacts with p27, and p27/c

236 roduce platelet-derived growth factor (PDGF)-DD, which promotes cellular proliferation, epithelial-me

237 PDGF-DD engagement of NKp44 triggered NK cell secretion of in

238 Thus, while cancer cell production of PDGF-DD supports tumor growth and stromal reaction, it concom

239 s and innate lymphoid cells, recognizes PDGF-DD.

240 equential BET is a low cost, well-performing DD screening tool, appropriate to the Primary Care Setti

241 hine learning based algorithm for type I PKS DD affinity and pathway prediction.

242 t a high efficiency tool, (ii) the predicted DD affinity is represented by a probability (0-1), which

243 Cell body peptidoglycan contained primarily DD-crosslinks between meso-diaminopimelic acid and D-ala

244 apted to co-express, from the same promoter, DD-Cas9 with any other gene of interest without co-modul

245 Of several published DD criteria, it is unclear which, if any, are applicable

246 ed child morbidity 2 week's prior to report (DD = -3.5 pp, ROR: 0.80, 95% CI: 0.56-1.14, p = 0.214) b

247 of animal source foods (ASFs) (respectively, DD = +4.5 pp, ROR: 2.24, 95% CI: 1.09-4.61, p = 0.029 an

248 with 25% exhibiting grade III/IV restrictive DD with mitral regurgitation.

249 were obtained.Measurements and Main Results: DD therapy increased trough AAT levels to normal and, co

250 Here we show that K627 in human RIPK1-DD and its equivalent K612 in murine RIPK1-DD is a key u

251 1-DD and its equivalent K612 in murine RIPK1-DD is a key ubiquitination site that regulates the overa

252 Moreover, we show DD to be an indicator for topological robustness of scal

253 loped an antibody-independent deep-dive SRM (DD-SRM) approach that capitalizes on multidimensional hi

254 rvention arm, but had no impact on stunting (DD = -6.2 percentage points [pp], relative odds ratio [R

255 D MNs, which are divided into two subgroups: DD and VD.

256 > 0.8) with that of the previously suggested DD susceptibility genes KIAA0319, CTNND2, CNTNAP2 and GR

257 ynthesis capacity (DSC) of patients with SZ, DD, other psychotic disorders, and the DSC of healthy su

258 e duration of principal cell discharges than DD EPSPs, enabling GCs to compare contrasting versions o

259 We hypothesize that DD/DL circuitry can implement the same pattern separatio

260 These data indicate that DD, SZ, and other psychotic disorders have similar dysre

261 al and conceptual perspective, and show that DD can reveal structural properties that are not obtaine

262 ng synthetic and real networks, we show that DD distribution can be used to distinguish between diffe

263 t DD play different structural roles and the DD distribution is an important network signature.

264 on strength is around 27 +/- 1 M(-1) for the DD.AA complexes in C(6)D(6), whereas it rises to 1820 +/

265 ter in the caudate region of subjects in the DD group (ES = 0.83, corrected p = 0.048), the SZ group

266 cket to facilitate substrate turnover in the DD-CoA binding region of the protein.

267 In addition, we show that the DD subnuclei have complex reciprocal connections with su

268 We consider chains of length 2 in which the DD-CIK gives to a candidate in the KPDP, and that candid

269 Thus, DD-SRM holds great promise for precisely measuring extre

270 with compared to women without bilateral TMJ DD.

271 d 3 per month, and delay times for access to DD-CIK of 6 months or less, including DD-CIKs increases

272 Of the agents tested, rifamycins led to DD Mtb generation, an effect lacking in a rifampin-resis

273 d profiles of HD tissues was more similar to DD tissues than to HH tissues, reflecting a colony-wide

274 jects strongly to DDi, with lesser inputs to DDs and DDmg.

275 fy the contribution of splicing mutations to DDs.

276 y larger (all P < 0.01) for +DD compared to -DD subjects by 1.7-, 2.5-, and 1.9-fold for day, night,

277 0.04) by up to 8.5-fold in +DD compared to -DD subjects.

278 ( P = 0.012) by 1.4-fold in +DD compared to -DD subjects.

279 han that of hydrogel-incorporated unmodified DDs.

280 The V(DD)J recombination is currently viewed as an aberrant an

281 alian genomes, thus demonstrating that the V(DD)J recombination is not a "bug" but an important featu

282 e V(D)J recombination fails to explain the V(DD)J recombination, the molecular mechanism of tandem D-

283 e identified 81 loci (66 novel) for vertical DD.

284 or well-differentiated/de-differentiated (WD/DD) liposarcomas has been reported to be minimal, howeve

285 ics in patients with retroperitoneal (RP) WD/DD liposarcoma treated at The University of Texas MD And

286 We provide an explanation as to why DD can characterize structural heterogeneity in mixing p

287 for hemEFS (hazard ratio, 2.1 and 3.1) with DD and DVD.

288 ADD-RS (either ADD-RS=0 or ADD-RS </=1) with DD may be considered to standardize diagnostic rule out

289 rdiac organ responses were noted in 22% with DD and 26% with DVD after 6 months.

290 ation of pretest probability assessment with DD testing is feasible, but the safety and efficiency of

291 ted patients, 35 (38.9%) were diagnosed with DD according to Rome IV criteria, mainly female (n = 30,

292 Use of EVD with DD increased the detection of positive section margins i

293 ical characteristics, usefulness of EVD with DD, and turnaround times (TATs).

294 psy processing with the practice of EVD with DD.

295 red an adverse factor for OS and hemEFS with DD, whereas translocation t(11;14) was associated with a

296 arily by the placebo response: patients with DD exhibited significantly larger placebo responses (g =

297 of children with either ASD-noID, those with DD, or GP controls.

298 ed events in 9738 exome sequenced trios with DD-affected probands.

299 ical trials of SSRIs or SNRIs in youths with DD, AD, OCD, or PTSD were included.

300 genic effects of the SVs in individuals with DDs.