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2 DDH in infants younger than 3 months, and a DDH risk increase of female sex was found to be lower th
5 related the n-butane direct dehydrogenation (DDH) activity with the average coordination number (CN)
7 kene selectivity (99.0%) for n-butane direct DDH reaction at 450 C, compared to typical Pt NP and Pt
11 rtilage can be detected in chondrocytes from DDH cartilage before histological manifestations of dege
12 fold termed the disulfide-directed hairpin (DDH) motif, which is the proposed evolutionary structura
13 ology to developmental dysplasia of the hip (DDH) and a realisation that neonatal hip maturation is p
14 to treat developmental dysplasia of the hip (DDH) classified Crowe III-IV using direct anterior appro
15 ning for developmental dysplasia of the hip (DDH) has gained increasing popularity despite the lack o
16 cts with developmental dysplasia of the hip (DDH) often show early-onset osteoarthritis (OA); however
17 story of developmental dysplasia of the hip (DDH), female sex, and primiparity to increase the risk o
18 such as developmental dysplasia of the hip (DDH), the differentiation of what is normal, what is abn
19 hout the superficial and deeper dorsal horn (DDH), as well as the lateral spinal nucleus (LSN), of mo
21 e full acetabular shape, which might improve DDH developmental dysplasia of the hip assessment accura
22 These findings suggest that early events in DDH cartilage originate at the chondrocyte level and tha
23 nty-three consecutive hips with Crowe III-IV DDH who underwent DAA were retrospectively evaluated fro
25 to match patients who were DDH to those not DDH and a preference-based instrumental variable (IV) an
26 younger than 3 months in whom a diagnosis of DDH was made by hip ultrasonography using the criterion
29 4.15 [95% CI, 2.62-6.57]), family history of DDH (OR, 3.83 [95% CI, 2.05-7.15]), female sex (OR, 2.50
30 -analysis calculated the pooled incidence of DDH per 1000 newborns with 95% CIs using a random- or fi
37 ere not significantly different for patients DDH (odds ratio [OR], 1.00; 95% CI, 0.96-1.04), and ther
39 e was no difference in outcomes for patients DDH compared with ward transfer prior to discharge when
41 originate at the chondrocyte level and that DDH cartilage may provide a novel opportunity to study t
42 nce from observational studies suggests that DDH from ICU may have no difference in safety outcomes c
47 expression in yeast showed that one of these DDH isomers is converted to solanacol, one of the most a
49 and collagen II immunostaining in undamaged DDH cartilage, with no evidence of augmented cell death
51 increase in the risk of sonography-verified DDH in infants younger than 3 months, and a DDH risk inc
54 ropensity scoring to match patients who were DDH to those not DDH and a preference-based instrumental
56 d DDH was 0.5 (95% CI, 0.2-1.5) infants with DDH per 1000 newborns with clinical screening, 0.6 (95%
57 rate was 8.4 (95% CI, 4.8-14.8) infants with DDH per 1000 newborns with clinical screening, 4.4 (95%
58 creening, 4.4 (95% CI, 2.4-8.0) infants with DDH per 1000 newborns with selective ultrasonographic sc
59 creening, 0.6 (95% CI, 0.3-1.3) infants with DDH per 1000 newborns with selective ultrasonographic sc
60 g, and 23.0 (95% CI, 15.7-33.4) infants with DDH per 1000 newborns with universal ultrasonographic sc
61 ning, and 0.2 (95% CI, 0.0-0.8) infants with DDH per 1000 newborns with universal ultrasonographic sc