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1                                              DEET (N, N-diethyl-meta-toluamide) is the most effective
2                                              DEET (N,N-diethyl-3-methylbenzamide) is a 6-decade-old s
3                                              DEET (N,N-diethyl-meta-toluamide) is the world's most wi
4                                              DEET acts on insect smell [2-6] and taste [7-11], and it
5                                              DEET acts on this complex to potentiate or inhibit odour
6                                              DEET can also inhibit certain taste neurons, revealing t
7                                              DEET inhibits behavioral attraction to food odors in Dro
8                                              DEET inhibits food ingestion by Drosophila melanogaster
9                                              DEET inhibits odor-evoked currents mediated by the insec
10                                              DEET is the most widely used insect repellent worldwide.
11                                              DEET permeation through human skin in vitro was measured
12                                              DEET seems to act both at close range as a contact chemo
13                                              DEET stimulated action potentials in GRNs that respond t
14                                              DEET was potent in suppressing feeding as <0.1% DEET eli
15                                              DEET-based products provided complete protection for the
16                                  Since 0.02% DEET elicited action potentials, we conclude that DEET d
17 T was potent in suppressing feeding as <0.1% DEET elicited aversive behavior.
18 as used to randomise 795 households to a 15% DEET lotion and the remainder were given a placebo lotio
19  noninfected mosquitoes was achieved with 5% DEET, which corresponds approximately to a 30% dose in t
20 nment-issued insect repellent containing 75% DEET (N,N-diethyl-m-toluamide) in ethanol applied during
21       With the exceptions of bisphenol A and DEET, all TOrCs that were detected in the DS were well r
22 ered the sensitivity of MhOR5 to eugenol and DEET and broadly reconfigured the receptor's tuning.
23 insect odorant receptor to odour ligands and DEET.
24                We further show that 6-MH and DEET can bind simultaneously to OBPs with other ligands.
25                        We show that 6-MH and DEET can compete for the binding of attractive odorants
26 en silicone sampler levels of permethrin and DEET with their corresponding urinary metabolites (r(s)
27     Young male rats were provided PB, PM and DEET at equivalent human doses and physical restraint (t
28  many continue to have practices of applying DEET (N,N-diethyl-3-methylbenzamide) based repellents th
29 MH binds to OBP1 at exactly the same site as DEET.
30  [para-chlorobenzoic acid (p-CBA), atrazine, DEET, and ibuprofen] was not significantly inhibited in
31 ited a more robust and longer-lived aversive-DEET memory.
32 ncentrations of bitters, but rapidly avoided DEET-treated skin and did not blood feed.
33                                         Both DEET and caffeine were rapidly degraded by UV/FC and SS/
34 bition by the odour ligand and modulation by DEET.
35                            For An. coluzzii, DEET and other non-volatile repellents mask the mosquito
36 t they are rapidly repelled after contacting DEET-treated skin [6].
37                 Insect repellents containing DEET (N,N-diethyl-meta-toluamide) are highly effective,
38                                 In contrast, DEET, an active ingredient in mosquito and tick repellen
39 osquitoes use their sense of smell to detect DEET, but there are currently two hypotheses regarding i
40 ion by all tested mosquito repellents except DEET.
41 its bitterness per se does not fully explain DEET contact chemorepellency.
42 lecule that are proposed to be important for DEET binding are not involved in binding of 6-MH.
43 not an ionotropic receptor, is necessary for DEET reception and repellency in Culex mosquitoes.
44         Mixed relationships are observed for DEET, which reacts with multiple PPRI.
45 similar level of protection as observed from DEET.
46 , and single sensillum recordings identified DEET-sensitive sensilla that were nonresponders in the i
47                             Here we identify DEET-sensitive neurons in a pit-like structure in the Dr
48 re are major impediments to finding improved DEET alternatives because the receptors causing olfactor
49      Formulation B led to a 30% reduction in DEET permeation versus control.
50 oses, we observed a significant reduction in DEET-elicited protection against ZIKV-infected yellow fe
51 er characterized insect repellents including DEET.
52 on decreases effective repellency since less DEET is available for evaporation.
53 he search for molecular mechanisms mediating DEET contact chemorepellency and novel contact-based ins
54 ch chemicals, N,N-diethyl-3-methylbenzamide (DEET) and caffeine, by low pressure ultraviolet (UV) lig
55               N,N-diethyl-3-methylbenzamide (DEET) is popular insect repellent which is considered sa
56               N,N-diethyl-3-methylbenzamide (DEET) is the most effective, and best studied, insect re
57 ents, such as N,N-diethyl-3-methylbenzamide (DEET),(8)(,)(9) but how catnip triggers aversion in inse
58 e, now called N,N-diethyl-3-methylbenzamide (DEET); a repellent containing IR3535 (ethyl butylacetyla
59                         The two microcapsule DEET formulations exhibited 36-40% higher cumulative eva
60                      Currently available non-DEET repellents do not provide protection for durations
61 andidate molecular targets for the action of DEET may aid in the design of safer and more effective i
62                   To understand the basis of DEET contact chemorepellency, we carried out behavioral
63  time passed after training, the behavior of DEET-sugar-trained flies reversed from conditioned odor
64                     Higher concentrations of DEET provided longer-lasting protection.
65                   Reaction rate constants of DEET and caffeine with the respective radical species we
66                           The degradation of DEET followed pseudo-first-order kinetics, whereas the d
67 ix under UV/FC and SS/FC, the degradation of DEET was significantly inhibited, but the degradation of
68  and Cl. were responsible for degradation of DEET, whereas ClO. related reactive species (ClOrrs), ge
69  compensated by applying a 5x higher dose of DEET.
70         We suggest that the effectiveness of DEET in pest control owes to its dual action in inducing
71 t olfactory- and contact-mediated effects of DEET are mechanistically distinct.
72 isms for the observed behavioural effects of DEET in the gas phase have been proposed: that DEET inte
73 planation for the broad-spectrum efficacy of DEET against multiple insect species.
74 acted to human hosts even in the presence of DEET, but are repelled upon contact, indicating that olf
75  attracted to humans even in the presence of DEET, but they are rapidly repelled after contacting DEE
76 acted by human odors despite the presence of DEET.
77 the behaviorally relevant contact sensors of DEET.
78 shown to have an efficacy similar to that of DEET-based repellents.
79 protection for durations similar to those of DEET-based repellents and cannot be relied on to provide
80 IR40a knockdown had no significant effect on DEET detection and repellency.
81 rosophila olfactory receptor neurons (ORNs), DEET is detected through a mechanism employing the olfac
82  exposure to low doses of GWIR chemicals PB, DEET, and permethrin induced depressive- and anxiety-lik
83        A formulation containing 23.8 percent DEET had a mean complete-protection time of 301.5 minute
84 g pyridostigmine bromide (PB) and pesticides DEET and permethrin during the war has been proposed as
85  including caffeine, meprobamate, primidone, DEET, carbamazepine, dilantin, naproxen, and triclosan.
86                         The insect repellent DEET (N,N-diethyl-m-toluamide), which attenuates odor re
87 tion and sensitivity to the insect repellent DEET (N,N-diethyl-meta-toluamide).
88         How does the common insect repellent DEET modify a mosquito's ability to detect humans?
89 we discover that the common insect repellent DEET repels Anopheles coluzzii upon contact with their l
90 the odorant eugenol and the insect repellent DEET.
91 l and susceptibility to the insect repellent DEET.
92 ontaminated with the common insect repellent DEET.
93 se have led a handful of contact repellents (DEET, picaridin, IR3535) to dominate the field.
94 /day, permethrin (PM) 0.13 mg/kg/day (skin), DEET 40 mg/kg/day (skin) and were physically restrained
95                        Like bitter tastants, DEET is a feeding deterrent when ingested, but its bitte
96 sential oil samples were more effective than DEET at reducing the number of mosquito landings.
97 repellents are generally less effective than DEET-based products.
98 tem to block host odour recognition and that DEET actively repels insects by activating olfactory neu
99 elicited action potentials, we conclude that DEET directly activates of GRNs.
100                             We conclude that DEET masks host odor by inhibiting subsets of heteromeri
101                    Here, we demonstrate that DEET, picaridin, insect repellent 3535, and p-menthan-3,
102                           We determined that DEET, IR3535, and picaridin decrease the response of Orc
103 t behavioral experiments and discovered that DEET acts by three distinct mechanisms: smell, ingestion
104 urthermore, they support the hypothesis that DEET acts as a molecular 'confusant' that scrambles the
105 to proboscis, leading to the hypothesis that DEET repels on contact by activating an aversive bitter
106 ET in the gas phase have been proposed: that DEET interferes with the olfactory system to block host
107                           Here, we show that DEET blocks electrophysiological responses of olfactory
108                                 We show that DEET contact chemorepellency is mediated exclusively by
109                            Here we show that DEET functions as a modulator of the odour-gated ion cha
110                 Our measurements showed that DEET provided significantly higher protection than picar
111                         Here, we showed that DEET suppressed feeding behavior in Drosophila, and this
112           Previously, it has been shown that DEET targets multiple components of the olfactory system
113 ng GCaMP-expressing mosquitoes suggests that DEET works differently for different mosquito species.
114 nstrate that flies independently process the DEET and sugar components to form parallel aversive and
115 controlled-release formulations in which the DEET active material was temporarily sequestered within
116                                         This DEET-aversive learning required the MB-MP1 dopaminergic
117 d or Ir40a is knocked down lose avoidance to DEET.
118 rsial as to whether ORNs respond directly to DEET or whether DEET blocks the response to attractive o
119  exposures and assessed noncancer hazards to DEET, piperonyl butoxide, prometon, secbumeton, terbumet
120 des aegypti females that were insensitive to DEET, and the selection of either sensitive or insensiti
121 dy suggests that behavioral insensitivity to DEET in A. aegypti is a genetically determined dominant
122 roantennography showed a reduced response to DEET in the selected insensitive line compared with the
123 crease in electroantennographic responses to DEET and a complete lack of repellency.
124                     N,N-Diethyl-m-toluamide (DEET) is one of the most effective and commonly used mos
125  permetrim (PM) and N,N-diethyl-m-toluamide (DEET) used as protectants against insects and nerve gase
126 e times longer than N,N-diethyl-m-toluamide (DEET), the most widely used repellent throughout the wor
127 hetic repellents N,N-diethyl-meta-toluamide (DEET) and IR3535 did not activate Anopheles odorant rece
128 pyrethroids, and N,N-diethyl-meta-toluamide (DEET)) and some of their metabolites were characterized
129 lso sensitive to N,N-diethyl-meta-toluamide (DEET), a mosquito repellent.
130 ding permethrin, N,N-diethyl-meta-toluamide (DEET), and chlorpyrifos.
131 rants, including N,N-diethyl-meta-toluamide (DEET), at a distance.
132 y used repellent N,N-diethyl-meta-toluamide (DEET), on the function of specific ORs of the African ma
133 s and to be effectively repelled by volatile DEET.
134                                         When DEET-based repellents are applied in combination with pe
135         Natural products are often used when DEET cannot be afforded or accessed and when consumers c
136 her ORNs respond directly to DEET or whether DEET blocks the response to attractive odors.
137     We have investigated the extent to which DEET skin absorption can be reduced and evaporation sust
138     Moreover, differential conditioning with DEET versus shock suggests that formation of these disti

 
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