ライフサイエンスコーパス: DRM

1 DRM is also a highly endothermic reaction and requires o

2 DRM is inevitably accompanied by deactivation due to car

3 DRMs account for a small proportion of all elevated VL a

4 DRMs can revert to wild-type and remain as minority stra

5 DRMs in PR-RT and IN regions were analyzed in Stanford H

6 DRMs isolated from chondrocyte PM were enhanced in gangl

7 DRMs to NNRTIs and NRTIs were found among 65% and 51% of

8 DRMs were common, with 28 of 179 (15.6%) specimens carry

9 gically failed ART, of whom 70% carried >/=1 DRM and 49% had dual-class resistance, with an average o

10 Among participants with >/=1 DRM, HIV-1 susceptibility was reduced in 93% for efavire

11 46 (43.4%) with VL >/= 1000 cps/mL and >/=1 DRMs; no HIV RNA data was available for 32 (30.2%) parti

12 HIV-1 DRMs and phylogenetic characteristics were determined us

13 ARV association of previously reported HIV-2 DRMs and identified novel TSMs.

14 ual-class resistance, with an average of 2.4 DRMs per sequence (range, 1-8).

15 ter along with any other jobs scheduled by a DRM.

16 The evidence that a DRM-causing mutation affects CryAB muscle function and l

17 ent in DRMs, but was less abundant in abcb19 DRMs.

18 s, but virus in several patients accumulated DRMs to drugs not received, such as abacavir, didanosine

19 Overall, 7.3% transmitted and 34.3% acquired DRMs were found, including M184V, thymidine analogue mut

20 n the initial 30-day ART supply and acquired DRMs.

21 itiated ART upon enrollment, and no acquired DRMs were detected.

22 initiated ART upon enrolment and no acquired DRMs were detected.

23 For all DRMs detected, PANDAA showed a sensitivity and specifici

24 etative cells, and somatic tissues, although DRM activity extends into heterochromatin in vegetative

25 er, fluorescence probe microenvironment, and DRM formation were similar to those of cholesterol, othe

26 ount of LAT in the immunological synapse and DRM by posttranslational palmitoylation contributes to t

27 ifferent active site requirement for WGS and DRM is confirmed by the experimental evaluation of the a

28 ed rabbits were treated with 1% Brij 98, and DRMs were isolated via sucrose gradient centrifugation.

29 ybrid repressors by fusing distinct ESMs and DRMs provides a means to flexibly rewire genetic network

30 or predicting compatibility between ESMs and DRMs.

31 ane (DRM), a key site of Src inhibition, and DRMs from K6-null keratinocytes were depleted of both ke

32 Groups were compared on pre-ART DRMs and detection of antiretrovirals in stored plasma.

33 in the cohort may be attributable to pre-ART DRMs vs >90% attributable to ART nonadherence.

34 The prevalence of pre-ART DRMs was 10% in cases and 5% in controls (adjusted odds

35 cial for protein misfolding diseases such as DRM.

36 ation also impaired infectious-HCV assembly, DRM localization of NS2 and E2, and colocalization of NS

37 ins and selection pressure on TDR-associated DRMs.

38 ations, M184V/I and the tenofovir-associated DRMs K65R and K70E/Q/G/N/T accounted for 82.9%, 7.3%, an

39 suggesting that FAK activation can occur at DRM microdomains.

40 ation of increased diversity or selection at DRM sites compared to other sites and no association bet

41 amides at the ER largely decreases Sig-1R at DRMs and causes translocation of Sig-1R from the MAM to

42 timized the MAS assay to determine subtype B DRMs in dried blood spots (DBS) collected from patients

43 While compartment-based DRM discordance was largely consistent with increased dr

44 at a significant, dynamic, compartment-based DRM ratio imbalance can exist, as it has the potential t

45 Ni-based DRM catalysts are promising due to their high activity a

46 n, with 28 of 179 (15.6%) specimens carrying DRMs, including the PR N88S and RT K103N mutations, whic

47 Under current climate, DRMs performed best, although only marginally.

48 The most common DRMs were M184V (53.5%), K103N (28.9%), Y181C (15.5%), a

49 ate groups that were also reported to confer DRM association.

50 K1, RIPK3, and MLKL to caveolin-1-containing DRM vesicles without fully blocking the activation of RI

51 In contrast, DRM contained significant levels of crystalline sterol.

52 In contrast, DRM had structural properties similar to those of NR, an

53 This review will cover DRM literature for catalysts based on Rh, Ru, Pt, and Pd

54 Although the dsRNA-binding activity of CPL2 DRM has not been shown to date, T-DNA insertion mutants

55 Sig-1Rs associate with MAM-derived DRMs but not with those from microsomes.

56 Several small studies have described DRMs that emerge in patients receiving dolutegravir.

57 The new assay accurately detected DRMs, including low-abundance mutations that were often

58 Disrupting DRMs by lowering cholesterol or inhibiting de novo synth

59 nsient detergent-resistant membrane domains (DRMs) within the ER, also called the ER regulatory pool

60 n assay sensitivity, the length of time each DRM is detectable within individuals.

61 the Drosophila dREAM/Myb-MuvB and C. elegans DRM transcriptional regulatory complexes.

62 either a part of DRM (cpl2-1) or the entire DRM (cpl2-2) exhibited leaf expansion defects, early flo

63 ngs suggest that CYP1A2 and CPR reside in ER-DRMs and that the unique lipid components of these domai

64 s, while all Ni surface atoms are active for DRM.

65 The most widely used catalysts for DRM are based on Ni.

66 ctive and coke-resistant catalytic sites for DRM.

67 y increasing significantly the stability for DRM.

68 We screened for DRMs during early-acute viremia and examined the diversi

69 The majority of low-frequency DRM detected using UDS are likely errors inherent to UDS

70 In general, DRMs were enriched in saturated fatty acids when compare

71 ikely that minority viral variants harboring DRM are transmitted and maintained in the recipient host

72 de novo methyltransferase in plants, but how DRM acts mechanistically is poorly understood.

73 tor and cytochalasin D-stimulated changes in DRM lipid composition follow first-order kinetics.

74 lide, prevents antigen-stimulated changes in DRM PL composition.

75 argely prevented mitochondrial deficiency in DRM but that contractility was not improved because of m

76 ase in the recovery of polyunsaturated PL in DRM, particularly in phosphatidylinositol species.

77 tely 73% of CYP1A2 and 68% of CPR resided in DRM fractions, compared with only 33% of total ER protei

78 les on alumina, the commonly used support in DRM.

79 However, pUL37x1/vMIA was not detected in DRMs from mitochondria.

80 PECAM localized in DRMs displays reduced phosphorylation and does not suppo

81 In the wild type, PIN1 was also present in DRMs, but was less abundant in abcb19 DRMs.

82 N-cadherin and catenins are present in DRMs; however, their DRM distribution is not significant

83 Individual DRMs were identified more frequently in plasma than in p

84 y, we hypothesized that CryAB(R120G)-induced DRM would also respond favorably to prolonged voluntary

85 or nucleoside and nonnucleoside RT inhibitor DRM.

86 n-nucleotide reverse transcriptase inhibitor DRMs, sensitivity and specificity were 100% (97% to 100%

87 r nucleotide reverse transcriptase inhibitor DRMs, sensitivity and specificity were 98% (95% confiden

88 lattice opposes entry of many proteins into DRM rafts.

89 transporters, such as Abcg1, which itself is DRM associated.

90 RT initiation, 38 (18%) subjects had a major DRM and an increased proportion of resistance was seen i

91 sensitivity and specificity to detect major DRMs, PANDAA could represent a simple and rapid alternat

92 nto care, as 18% of our population had major DRMs prior to initiation of ART.

93 Interestingly, two subjects had major DRMs to NRTIs, NNRTIs, and 4 mutations in the Gag P2/NC

94 ns (DRMs) in the gag P2/NC CS, and pol major DRMs.

95 ral Society-USA list was used to score major DRMs.

96 mmonly used distributed resource management (DRM) systems, and it can be easily extended to new DRMs.

97 tal excision (PME), distal resection margin (DRM), and a composite outcome ("successful resection") w

98 ombined total of 227 drug-related materials (DRM) were detected from all eight test article incubatio

99 ory effect" of the Deese-Roediger-McDermott (DRM) paradigm: studying words that fit a common schema (

100 ssion of the mutation, CryAB(R120G)-mediated DRM is characterized by CryAB and desmin accumulations w

101 , similar to that found with plasma membrane DRMs.

102 components in detergent-resistant membrane (DRM) domains that are related to ordered membrane compar

103 nslocates to a detergent-resistant membrane (DRM) during transmigration.

104 sterols in the detergent-resistant membrane (DRM) fractions obtained from yeast and plant cells repli

105 ocalization in detergent-resistant membrane (DRM) fractions were defective in anergic T cells.

106 o Triton X-100 detergent-resistant membrane (DRM) fractions.

107 the amount of detergent-resistant membrane (DRM) material.

108 ly in platelet detergent-resistant membrane (DRM) rafts but that from Glanzmann's thrombasthenic plat

109 Studies of detergent-resistant membrane (DRM) rafts in mature erythrocytes have facilitated ident

110 or outside of detergent-resistant membrane (DRM) regions at the plasma membrane.

111 n-1-associated detergent-resistant membrane (DRM) vesicles in HT-29 cells.

112 nriched in the detergent-resistant membrane (DRM), a key site of Src inhibition, and DRMs from K6-nul

113 is enriched in detergent-resistant membrane (DRM)-forming lipids, including cholesterol and ceramide,

114 o produce (i) detergent-resistant membranes (DRM) and detergent-soluble membranes (DSM), (ii) nondete

115 zation to the detergent-resistant membranes (DRM) and HCV particle assembly.

116 sociates with detergent-resistant membranes (DRM) in a p7-dependent manner.

117 formation of detergent-resistant membranes (DRM).

118 ocated at the detergent-resistant membranes (DRM).

119 rporated into detergent-resistant membranes (DRMs) but still are delivered apically, suggesting that

120 omposition of detergent-resistant membranes (DRMs) from chondrocyte PM.

121 aised against detergent-resistant membranes (DRMs) from monocytes or activated T cells, but not resti

122 d as buoyant, detergent-resistant membranes (DRMs) in OptiPrep density gradients.

123 port that the detergent-resistant membranes (DRMs) play an important role in anchoring Sig-1Rs to the

124 c analysis of detergent-resistant membranes (DRMs) revealed that the galectin lattice opposes entry o

125 ssociate with detergent-resistant membranes (DRMs) that are believed to represent lipid rafts.

126 as defined by detergent-resistant membranes (DRMs), in non-lipid raft membranes, and in virions showe

127 bly occurs in detergent-resistant membranes (DRMs), while serine 36 phosphorylation takes place in no

128 nslocate into detergent-resistant membranes (DRMs).

129 ociation with detergent-resistant membranes (DRMs).

130 analysis using the disjunctive rule merging (DRM) approach on a large and diverse dataset compiled fr

131 production via the dry reforming of methane (DRM) is a highly endothermic process conducted under har

132 Dry (CO2) reforming of methane (DRM) is a well-studied reaction that is of both scientif

133 Dry reforming of methane (DRM) is an attractive route to utilize CO(2) as a chemic

134 The dry reforming of methane (DRM), i.e., the reaction of methane and CO2 to form a sy

135 as shift (WGS) and dry reforming of methane (DRM), two key industrial reactions with common elementar

136 DOMAINS REARRANGED METHYLTRANSFERASE (DRM) is a key de novo methyltransferase in plants, but h

137 d in detergent-resistant lipid microdomains (DRM).

138 Detergent-resistant membrane microdomains (DRM) rich in cholesterol and sphingolipid, termed lipid

139 h detergent-resistant membrane microdomains (DRMs), which are known to be involved in their biogenesi

140 -enriched, detergent-resistant microdomains (DRMs).

141 ent, the probability of detecting a minority DRM in the recipient was not increased when the same min

142 er sites and no association between minority DRM and AM.

143 was no correlation between detected minority DRM and clinical failure of first-line ART.

144 ed the association between detected minority DRM and the virologic failure of first-line antiretrovir

145 evidence for sexual transmission of minority DRM (BF = 0.02).

146 The presence of minority DRM during early infection, followed by a rapid decay, i

147 al significance of the detection of minority DRM remains controversial.

148 s variability in the proportions of minority DRM.

149 ent was not increased when the same minority DRM was detected in the source (Bayes factor [BF] = 6.37

150 ission pairs to investigate whether minority DRM detected shortly after transmission were the consequ

151 Bayesian process-based dynamic range model (DRM).

152 sing pathogen specific dose response models (DRMs).

153 g module (ESM) and a DNA recognition module (DRM) has the potential to unlock a combinatorial set of

154 Moreover, DRMs in CerS2 null mice displayed properties significant

155 ns a double-stranded (ds)-RNA-binding motif (DRM).

156 ) had at least one drug resistance mutation (DRM).

157 sence of minority drug resistance mutations (DRM) may be a consequence of sexual transmission, de nov

158 ation of minority drug resistance mutations (DRM), including among antiretroviral therapy-naive HIV-i

159 l populations with drug-resistant mutations (DRM) are inconsistent with evidence that HIV-1 infection

160 ty and drug resistance-associated mutations (DRMs) among treatment-naive blood donors is critical for

161 gical failure and drug resistance mutations (DRMs) after 12 or 24 months of ART.

162 he acquisition of drug resistance mutations (DRMs) among SD arm nonlinkers.

163 argeting priority drug resistance mutations (DRMs) are being evaluated to improve access to HIVDR tes

164 Drug resistance mutations (DRMs) can serve as distinct, nonpolymorphic markers for

165 HIV strains with drug-resistance mutations (DRMs) causes public health problems in resource-rich cou

166 apid selection of drug resistance mutations (DRMs) during monotherapy with direct-acting antivirals (

167 mined patterns of drug-resistance mutations (DRMs) in individuals with virological failure on first-l

168 ult in discordant drug resistance mutations (DRMs) in the compartments.

169 Cameroonians for drug resistance mutations (DRMs) in the gag P2/NC CS, and pol major DRMs.

170 smitted genotypic drug resistance mutations (DRMs) in this population.

171 ve proportions of drug resistance mutations (DRMs) on individual HIV-1 polymerase genomes in the cere

172 revalence and the drug resistance mutations (DRMs) responsible for TDR in the United States.

173 e emerging on the drug resistance mutations (DRMs) selected by the most widely used ARVs and on the i

174 utions of pre-ART drug resistance mutations (DRMs) vs nonadherence in the etiology of elevated VL are

175 000 cps/mL and no drug resistance mutations (DRMs), 46 (43.4%) with VL >/= 1000 cps/mL and >/=1 DRMs;

176 f HIV-1 subtype C drug resistance mutations (DRMs).

177 ceptibilities and drug resistance mutations (DRMs).

178 he acquisition of drug resistance mutations (DRMs).

179 etwork inferences, drug resistant mutations (DRMs), subtypes and HIV-1 diversity estimations were com

180 Desmin-related myofibrillar myopathy (DRM) is a cardiac and skeletal muscle disease caused by

181 ion associated with desmin-related myopathy (DRM), results in an altered sarcomeric actin pattern, in

182 ryAB(R120G)) causes desmin-related myopathy (DRM), which is characterized by the formation of desmin-

183 ryAB(R120G)) causes desmin-related myopathy (DRM).

184 ystems, and it can be easily extended to new DRMs.

185 FAK biosensor are stronger than that at non-DRM regions, suggesting that FAK activation can occur at

186 serine 36 phosphorylation takes place in non-DRMs.

187 High levels of NNRTI and NRTI DRMs among CLHIV receiving ABC/3TC/LPV/r suggests a last

188 Treatment-naive subjects harboring NRTI-DRMs had significantly lower CD4 cells than those with N

189 icantly lower CD4 cells than those with NRTI-DRMs on ART (p = 0.042).

190 No NS3 DRMs that confer resistance to GS-9256 and GS-9451 (R155

191 not suppress the identified preexisting NS3 DRMs and thus a need for a combination therapy.

192 In contrast, 100% of DRM in both matrixes were subjected to MS(2) acquisition

193 e incubations, and among those, 5% and 4% of DRM were not triggered for MS(2) acquisition with IDA an

194 al analyses revealed an exponential decay of DRM (BF = 0.05) while genetic diversity increased.

195 studies, we observed increased detection of DRM at low frequencies (average, 0.56%; 95% CI, 0.43 to

196 The clinical impact of DRM discordance on viral persistence and HAND pathogenes

197 MS(2) spectra were the real product ions of DRM detected in microsomal samples from IDA, SWATH, and

198 While the majority of DRM (>20%) were consistently transmitted from source to

199 g enzyme that is upregulated in our model of DRM.

200 press CPL2 variants lacking either a part of DRM (cpl2-1) or the entire DRM (cpl2-2) exhibited leaf e

201 od of sexual transmission and persistence of DRM was assessed using Bayesian-based statistical modeli

202 e, sequence reproducibility, and presence of DRM and accessory mutations (AM).

203 the structural and functional properties of DRM and those of the nondetergent preparations (ACR and

204 fidence intervals (CI) for the proportion of DRM at each site and fitted a binomial mixture model to

205 growth factor (PDGF), the FRET responses of DRM-targeting FAK biosensor are stronger than that at no

206 questions about the clinical significance of DRM discordance remain, in the quest for viral eradicati

207 While transmission of DRM may impact future therapy options for newly infected

208 Subsequent lipid characterization of DRMs via high performance liquid chromatography-triple q

209 more rapid, change in the PL composition of DRMs in the absence of FcepsilonRI activation, indicatin

210 significant change in the PL composition of DRMs.

211 We previously compared PL compositions of DRMs with plasma membrane vesicles and whole cell lipid

212 significant differences in the frequency of DRMs in the CSF and plasma are readily found in those on

213 Furthermore, the isolation of DRMs from purified intracellular organelles independentl

214 progressively complex in China, and lots of DRMs found in the study exacerbate the primary drug resi

215 essures in the plasma, overrepresentation of DRMs in the central nervous system (CNS) can occur.

216 nsuppressive cART, and overrepresentation of DRMs in the CNS can occur.

217 In this study, we evaluated the presence of DRMs in HCV-infected patients treated with the HCV prote

218 9 year-olds showed the highest prevalence of DRMs (22.9%).

219 The high proportion of DRMs associated with infrequently used regimens combined

220 ally significant differences in the ratio of DRMs in the CSF and plasma were readily found in those o

221 Significant differences in the ratios of DRMs in CSF and plasma were found in 3 individuals with

222 ficant temporal differences in the ratios of DRMs in the compartments.

223 Transmission of DRMs with extremely high FCs (>6%) only occurs from appr

224 Transmission of DRMs with low FCs ( approximately 0.2%) is similar to wi

225 tlenecks substantially limit transmission of DRMs with moderately high FCs ( approximately 0.6%); cha

226 ting impact of failed PMTCT interventions on DRMs.

227 o HAND subjects (2/7 = 29%) demonstrated one DRM in CSF not identified in paired plasma.

228 % of the participants contained at least one DRM.

229 411) fusion protein translocated to platelet DRM rafts on thrombin activation, but its mutant that wa

230 DeltaNI = 2.85%, PNI = 0.025), and positive DRM (RD 0.15%, 90% CI -0.58 to 0.87, DeltaNI = 1.28%, PN

231 Predominant DRMs were associated with ART regimens, but virus in sev

232 ividuals; however, the levels of preexisting DRMs are too low to be directly quantified in most patie

233 ws the progression to HF in the CryAB(R120G) DRM model and that PAO accumulation is mediated, at leas

234 lipid-sensing peptide that forms regulatory DRMs in the ER, called mitochondria-associated ER membra

235 olipids, thus forming Triton X-114-resistant DRMs.

236 We determined that although NS2's DRM localization is dependent on p7, p7 was not targeted

237 ered with other TDR strains sharing the same DRMs.

238 ers new opportunities to develop large-scale DRM processes using earth abundant catalysts.

239 ng transmission of 10 clinically significant DRMs.

240 has been hindered by the absence of suitable DRMs for ARB.

241 he catalytic domain of the Nicotiana tabacum DRM methyltransferase, which efficiently targets DNA met

242 Lipid analysis showed that DRM fractions were enriched in cholesterol and sphingomy

243 nvelopment and postenvelopment events at the DRM to generate infectious HCV production.

244 According to ab initio calculations, the DRM proceeds on Mo sites of the oxycarbide nanosheet wit

245 yclodextrin (MbetaCD) not only disrupted the DRM localization of Core, NS2, and E2 but also specifica

246 ts we demonstrate that alphaB resides in the DRM fraction.

247 background level of p7 was detectable in the DRM fractions.

248 cation-dependent accumulation of ApoE in the DRM fractions.

249 s that comprise pollen-with mutations in the DRM, CMT2, and CMT3 methyltransferases.

250 urated or monounsaturated PL decrease in the DRM, whereas concentrations of PL with two or more doubl

251 omitantly with fibrin translocation into the DRM raft of thrombin-stimulated platelets.

252 Thus, our data support the role of the DRM as a platform for particle assembly process.

253 Since disruption of the DRM localization of NS2 and E2, either due to p7 and NS2

254 Relative fitness of the DRM viruses was shown to be significantly lower than the

255 Based on the DRM growth kinetics during drug treatment, pretreated NS

256 odextrin (MbetaCD) significantly reduced the DRM association of Core, NS2, and E2 and reduced infecti

257 olume of blank rat urine (urine sample), the DRM without MS(2) acquisition increased to 29% and 18%,

258 oylation of LAT and were selective since the DRM localization and palmitoylation of Fyn were intact.

259 We show that the DRM methyltransferase double mutant drm1drm2 also presen

260 3) lipids with a composition similar to the DRM fractions (V-DRM).

261 h the DRM, recruitment of E2 and ApoE to the DRM may allow the efficient coordination of Core particl

262 NS3 autoprocessing and E2 recruitment to the DRM, are regulated by palmitoylation at NS2/C113.

263 ed that NS2 regulates E2 localization to the DRM, consistent with its role in recruiting E2 to the vi

264 or is not involved in NS2 recruitment to the DRM, despite its apparent targeting to this location.

265 pecifically inhibited E2 localization to the DRM, indicating that NS2 regulates this process.

266 ction, these proteins' associations with the DRM likely play an important role during HCV assembly.

267 ining a neuropsychological approach with the DRM task.

268 CV Core protein strongly associates with the DRM, recruitment of E2 and ApoE to the DRM may allow the

269 The DRMs could support viral RNA synthesis on both the endog

270 Recent studies have described the DRMs that emerge in patients receiving tenofovir prodrug

271 Subsequently, these proteins exited the DRMs and were recovered quantitatively in purified virio

272 catenins are present in DRMs; however, their DRM distribution is not significantly affected by lattic

273 These DRMs were found to be cholesterol-dependent: CPR and CYP

274 widely used ARVs and on the impact of these DRMs on ARV susceptibility and virological response to f

275 Bcl-2 overexpression prolonged life in this DRM model, in the absence of apoptosis, another death pa

276 mediate plant asymmetric methylation through DRM enzymes, is also unclear.

277 tracellular membranes, in marked contrast to DRM that contained the highest level of cross contaminan

278 es show that S-palmitoylation contributes to DRM association of nicastrin and APH-1.

279 n affects CryAB muscle function and leads to DRM-like phenotypes in the fly reveals a conserved stres

280 l-dependent apical sorting is not related to DRM association of GPI-APs.

281 Furthermore, fibrin translocation to DRM rafts was impaired in factor XIII A subunit-deficien

282 A new approach to DRMs is introduced to capture ARB and antibiotic-suscept

283 SOAT1/Acat1 enzyme and activity localize to DRMs.

284 wnregulated following PECAM translocation to DRMs.

285 e of the methyltransferase domain of tobacco DRM (NtDRM) and reveal a molecular basis for its rearran

286 Reverse-transcriptase DRMs demonstrate that the breadth of variants in transmi

287 upregulates the formation of these transient DRMs by inducing the internalization of extracellular ch

288 e deactivation of 2D-Mo(2)CO(x)/SiO(2) under DRM conditions can be avoided by tuning the contact time

289 dy we investigated the mechanisms underlying DRM association of gamma-secretase subunits.

290 ween CPR and CYP1A2 was measured, V-ER and V-DRM liposomes produced lower apparent K(m) values compar

291 composition similar to the DRM fractions (V-DRM).

292 bunits of the gamma-secretase associate with DRM.

293 nce of Gag, envelope fails to associate with DRMs.

294 ns also eliminated envelope association with DRMs in 293T cells and in the T-cell line, MOLT 4.

295 we show that HIV-1 envelope association with DRMs is directly influenced by its interaction with Gag.

296 lly, suggesting that GPI-AP association with DRMs is not necessary for apical targeting.

297 tial for efficient envelope association with DRMs, which in turn is essential for envelope budding an

298 us also eliminated envelope association with DRMs.

299 exosomes may reside in its association with DRMs/lipid rafts.

300 tered the association of viral proteins with DRMs and resulted in an enhanced release of virus partic