ライフサイエンスコーパス: DSE

1 DSE are common among older adults, especially those with

2 DSE can be used to predict adverse outcomes after AMI.

3 DSE in autaptic cultures is both more robust and elicite

4 DSE is a feasible, safe and accurate screening method fo

5 DSE is less important for aggregates (reduced dipoles or

6 DSE may provide a way for cells to use their firing rate

7 DSE may represent one pathway through which spirituality

8 DSE was performed in 5-min stages with infusion of intra

9 per 100 patients tested [95% CI, 0.11-0.29], DSE 24 [95% CI, 0.10-0.38], and coronary angiography 20

10 Analysis of Nank4-7* DSE structural energetics at room temperature as a funct

11 RRR: MPS, 1.09; 95% CI, 0.64-1.86; P = 0.74; DSE, 1.56; 95% CI, 0.71-3.45; P = 0.25).

12 RRR, MPS, 0.89; 95% CI, 0.38-2.10; P = 0.78; DSE, 1.09; 95% CI, 0.12-10.05; P = 0.93), and MACE (RRR:

13 Twenty-two patients had 91 DSE studies and 45 coronary angiograms.

14 aracterization of the Hrp1/Nab4 protein as a DSE-binding factor that activates NMD.

15 oup, Bigot et al. proposed the presence of a DSE motif in hAT transposases.

16 ex is active for searching and recognizing a DSE for approximately 200 nt 3' of the stop codon.

17 Hrp1p binds specifically to a DSE-containing RNA and interacts with Upf1p, a component

18 cted cardiac events after normal or abnormal DSE.

19 After DSE, subjects were monitored for TxCAD-related cardiac e

20 nts that occurred during the 12 months after DSE were tabulated: myocardial infarction (MI), cardiac

21 get HR is not uncommon despite an aggressive DSE regimen.

22 ficant changes in the body composition among DSE subjects, they experienced a decline in the Si and A

23 (RRR, 0.69; 95% CI, 0.49-0.96; P = 0.03) and DSE (RRR, 0.72; 95% CI, 0.50-1.02; P = 0.06), noninvasiv

24 ing guides secondary structure accretion and DSE contraction as solvent quality is decreased.

25 iate analysis of clinical, angiographic, and DSE variables revealed that the only independent predict

26 endocannabinoid release involved in DSI and DSE is likely to evoke endocannabinoid release in respon

27 oylshikimic acid were recovered from DSP and DSE after in-vitro digestion.

28 ugh Caco-2 monolayer was observed in DSP and DSE, while protocatechuic acid and p-hydroxybenzoic acid

29 DSI in the hippocampus, subthreshold MSE and DSE act synergistically.

30 corded from cultured hippocampal neurons and DSE evoked by a 15 s depolarization to 0 mV and MSE evok

31 Six months later, the PET and DSE studies were repeated, and the animals were euthaniz

32 00% of age-predicted maximum heart rate) and DSE (5 to 40 microg/kg/min at 3-min stages with or witho

33 during dobutamine infusion between RTCE and DSE.

34 acceptance scores were similar for TAPSE and DSE.

35 ApoAI DSE-4 is a multi-factor complex that includes an Sp/H4TF

36 complexes with the DSE (referred to as apoAI DSE-1, -2, -3, and -4).

37 Because apoAI DSE mutations revealed transcription defects in transien

38 The highest apoAI DSE-3 levels were observed with retinoic acid treated He

39 omoters and the hepatic-specific human apoAI DSE at Sp1 and H4TF2 binding sites.

40 an Sp/H4TF1 factor and either H4TF2 or apoAI DSE-3.

41 The apoAI DSE-1 and -2 complexes showed similar binding specificit

42 The apoAI DSE-1 complex was predominantly recognized by anti-Sp1 a

43 The apoAI DSE-2 complex was identified as H4TF1 and formed in the

44 The apoAI DSE-3 complex bound to a consensus GATA element within t

45 The apoAI DSE-3 complex was completely disrupted by a GATA-4 antib

46 the Sp/H4TF1 consensus site within the apoAI DSE.

47 annabinol, which has been shown to attenuate DSE, antagonizes MSE.

48 An additional requirement for autaptic DSE is filled internal calcium stores.

49 role in determining the duration of autaptic DSE.

50 of DAGLalpha substantially reduces autaptic DSE, shifting the "depolarization-response curve" from a

51 GRS) as an auxiliary downstream element (AUX DSE) which influences the processing efficiency of the S

52 This suggests that AUX DSE binding proteins may play an active role in stimulat

53 AUX DSEs, therefore, serve as a integral part of the polyade

54 First, AUX DSEs can promote processing efficiency by maintaining th

55 These novel AUX DSEs all influenced the efficiency of 3'-end processing

56 Second, AUX DSEs can enhance processing by forming a stable structur

57 Three possible mechanisms by which AUX DSEs mediate efficient in vitro 3'-end processing have b

58 Both DSE and synaptically evoked suppression of excitation (S

59 to inhibit EPSCs, yet readily occludes both DSE and EPSC inhibition by a synthetic CB1 agonist, WIN

60 cant change in rest or stress wall motion by DSE six months postoperatively in either group.

61 urgery over a three-year period, preceded by DSE, were included.

62 colonisation of Antarctic vascular plants by DSEs facilitates not only the acquisition of organic nit

63 L123 had no significant effect on cerebellar DSE in MAGL(+/+) and (-/-) mice.

64 tes support and education control condition (DSE) among 4503 US adults with body mass index of 25 or

65 We report here that CB1-dependent DSE can be elicited from autaptic cultures of excitatory

66 We have recently reported that CB1-dependent DSE can be elicited in autaptic cultures of excitatory h

67 ) will result; we demonstrate fork-dependent DSEs proximal to Mu.

68 nto the study, 72 (92%) underwent diagnostic DSE by means of a standard protocol, 4.6 +/- 1.9 years a

69 Surprisingly, DAGLbeta knockdown diminishes DSE as much or more (ED(50) 6.4 seconds), suggesting tha

70 ith low to moderate risk of cardiac disease, DSE performed as part of an evaluation for liver transpl

71 without improvement in function at low dose DSE, who demonstrated worsening of function at a high do

72 e improvement of wall thickening at low-dose DSE may be limited in hibernating myocardium by severe h

73 (CB1R)--a possible mechanism underlying DSI/DSE.

74 4 women had wall motion abnormalities during DSE.

75 0.75 vs. 1.35 +/- 0.54, p < 0.05) and during DSE (2.11 +/- 0.67 vs. 1.21 +/- 0.41, p < 0.05) late aft

76 MCE and WMA for the detection of CAD during DSE have not been studied in a large number of patients.

77 ities (WMAs) consistent with ischemia during DSE.

78 if >/=2 of 16 segments were ischemic during DSE.

79 gnificant augmentation in wall motion during DSE after revascularization (WMSI 2.16 +/- 0.50 vs. 1.60

80 n of EDWT and any contractile reserve during DSE for recovery of regional function improved the speci

81 Patients with hypertensive response during DSE are more likely to have stress-induced myocardial is

82 he frequency of abnormal BP responses during DSE and their impact on accuracy of test results.

83 test or a dobutamine stress echocardiogram (DSE).

84 sfunction underwent rest 2D echocardiograms, DSE and rest-redistribution T1-201 tomography before rev

85 ities of dobutamine stress echocardiography (DSE) and rest-redistribution thallium-201 (T1-201) scint

86 s during dobutamine stress echocardiography (DSE) are associated with abnormal test results, nor if s

87 value of dobutamine stress echocardiography (DSE) for assessment of cardiac risk before nonvascular s

88 uracy of dobutamine stress echocardiography (DSE) for evaluating posttransplant coronary artery disea

89 ility of dobutamine stress echocardiography (DSE) for evaluation of women with suspected ischemic hea

90 value of dobutamine stress echocardiography (DSE) for predicting long-term outcomes in a large cohort

91 and peak dobutamine stress echocardiography (DSE) for the diagnosis of coronary artery disease (CAD).

92 n during dobutamine stress echocardiography (DSE) has been increasingly used for detection of hiberna

93 R during dobutamine stress echocardiography (DSE) identifies viable myocardium that may improve in fu

94 f serial dobutamine stress echocardiography (DSE) in new heart transplant recipients and to examine t

95 perative dobutamine stress echocardiography (DSE) in patients who fail to achieve target heart rate (

96 ility of dobutamine stress echocardiography (DSE) in predicting cardiac events in the year after test

97 role of dobutamine stress echocardiography (DSE) in these patients, DSE was included in the preopera

98 y (MPS), dobutamine stress echocardiography (DSE) or coronary angiography, performed during preoperat

99 standard dobutamine stress echocardiography (DSE) protocol.

100 PET) and dobutamine stress echocardiography (DSE) were performed to quantitate regional myocardial bl

101 entional dobutamine stress echocardiography (DSE) without contrast.

102 n during dobutamine stress echocardiography (DSE), particularly a biphasic response, predicts recover

103 ion (ILI) or diabetes support and education (DSE) on whom 2 y of data were collected.

104 B surgery or diabetes support and education (DSE).

105 don and searches for the downstream element (DSE), whose recognition by the complex identifies the tr

106 nse codon for a downstream sequence element (DSE) associated with RNA-binding proteins.

107 romoter elements: a distal sequence element (DSE) at around -220, a proximal sequence element (PSE) a

108 at around -55 and a distal sequence element (DSE) at around -220.

109 ignal (PAS) and downstream sequence element (DSE) motifs drive broad alterations in 3' UTR isoform ex

110 transcription and a distal sequence element (DSE) required for activated transcription.

111 lar enhancers, the distal sequence elements (DSEs), and similar basal promoter elements, the proximal

112 er as the relevant endocannabinoid to elicit DSE.

113 RecA4142 was loaded at a double-strand end (DSE) of DNA.

114 s into a Mu target gap, a double strand end (DSE) will result; we demonstrate fork-dependent DSEs pro

115 es both a site-specific double-stranded end (DSE) and a Holliday junction.

116 a delay of its ending dates (dry-season end, DSE), and is accompanied by a prolonged fire season.

117 ycete fungi, termed dark septate endophytes (DSEs), frequently colonise the roots of these plant spec

118 s not the sole source of double-strand ends (DSEs) during TLD, as previously proposed; models are sug

119 BDNF treatment blocked MSE and enhanced DSE.

120 Expression of H1a enhanced DSE and inhibited MSE at the same synapse.

121 rotein AI (apoAI) 48-bp downstream enhancer (DSE) were identified and characterized by electrophoreti

122 not present in the denatured state ensemble (DSE) or in intrinsically disordered proteins.

123 The denatured state ensemble (DSE) represents the starting state for protein folding a

124 om an equilibrated denatured state ensemble (DSE), we also do not get agreement with the equilibrium

125 nteractions in the denatured state ensemble (DSE).

126 e descriptions of denatured state ensembles (DSEs) remain unresolved.

127 sfection assays, we conclude that the entire DSE sequence is required for full apoAI transcriptional

128 mechanism when starting from an equilibrated DSE, when the simulation time is long enough to sample t

129 ng pilus subunit by a donor-strand exchange (DSE) mechanism.

130 membrane usher where donor strand exchange (DSE) replaces PapD's donated beta strand with an amino-t

131 arization-induced suppression of excitation (DSE) and inhibition (DSI) are forms of short-term neuron

132 arization-induced suppression of excitation (DSE) and inhibition (DSI).

133 arization-induced suppression of excitation (DSE) and metabotropic glutamate receptor (mGluR1)-mediat

134 arization-induced suppression of excitation (DSE) and metabotropic suppression of excitation (MSE).

135 arization-induced suppression of excitation (DSE) and reduced agonist-mediated desensitization of DSE

136 arization-induced suppression of excitation (DSE) is a form of cannabinoid CB(1) receptor-mediated in

137 arization-induced suppression of excitation (DSE) is a major form of cannabinoid-mediated short-term

138 arization-induced suppression of excitation (DSE) is accompanied by altered paired-pulse plasticity,

139 arization-induced suppression of excitation (DSE) is present in both SCs and basket cells.

140 arization-induced suppression of excitation (DSE), a CB1 receptor-dependent form of synaptic plastici

141 arization-induced suppression of excitation (DSE).

142 ppression of inhibition (DSI) or excitation (DSE).

143 pression of inhibitory (DSI) and excitatory (DSE) synapses by a mechanism that does not involve mGluR

144 ished a neuronal subpopulation that exhibits DSE and a differential complement of MSE-mediating Gq-co

145 e prevalence of daily spiritual experiences (DSE) and how they may relate to physical and mental heal

146 seed pita bread (DSB) and date seed extract (DSE).

147 re- and postsynaptic machinery necessary for DSE but also that for MSE.

148 p = 0.0001) were shorter for TAPSE than for DSE.

149 gp46, but not EndoVII, regressed origin fork DSEs are processed by degradation of the DSE and a pathw

150 Frequent DSE were significantly associated with a higher number o

151 SE and white men reported the least frequent DSE (mean+/-SD 35.9+/-13.6 versus 52.2+/-19.1).

152 pain, and comorbid conditions, more frequent DSE were associated with increased energy (P<0.009) and

153 SE scores than men (reflecting more frequent DSE, mean+/-SD 37.3+/-15.0 versus 45.8+/-17.5; P=0.012).

154 an American women reported the most frequent DSE and white men reported the least frequent DSE (mean+

155 However, DSE is usually negative in women with single-vessel sten

156 gy and biochemistry, we show that changes in DSE are a result of the reduced expression of Cb1Rs and

157 he study of cannabinoid signaling, including DSE.

158 Increased DSE may be associated with more energy and less depressi

159 Increasingly, DSE has been applied to risk stratification of patients.

160 ng-range contacts; however, the urea-induced DSE deviates from a random coil.

161 In contrast, the urea-induced DSE has significantly less residual secondary structure

162 d in buffer was compared to the urea-induced DSE.

163 ed suppression of excitation and inhibition (DSE and DSI) appear to be important forms of short-term

164 he incoming Nte is able to dock and initiate DSE due to inherent dynamic fluctuations within the chap

165 Women had significantly lower DSE scores than men (reflecting more frequent DSE, mean+

166 ent with the role of bona fide eCB mediating DSE.

167 dy of the roles of each isoform in mediating DSE.

168 s with arthritis reported significantly more DSE than those without arthritis (mean+/-SD 35.2+/-12.1

169 Negative DSE predicts short-term freedom from such events.

170 A negative DSE without resting WMAs has excellent NPV regardless of

171 urred in 2 of 50 children (4%) with negative DSE, versus 6 of 22 children (27%) with positive DSE (p

172 Of 397 negative DSEs, peak HR was <85% maximum predicted in 62 (16%).

173 Drug-Side Effect Context-Sensitive Network (DSE-CSN) of 139 760 drug-side effect pairs, representing

174 t patients, all with normal or nondiagnostic DSE studies (negative predictive value 86%).

175 s DSE, and the agonist WIN 55,212-2 occludes DSE.

176 ignalling machinery: MSE mimics and occludes DSE and is itself occluded by the endocannabinoid 2-arac

177 respectively, compared with less than 2% of DSE participants (1.7% [95% CI, 1.2%-2.3%] for at least

178 wall thickening and improves the accuracy of DSE for detecting viable myocardium.

179 atergic neurons prolonged the time course of DSE in the amygdala, and impaired fear extinction in aud

180 reduced agonist-mediated desensitization of DSE.

181 The efficacy of DSE for detecting atherosclerotic coronary artery diseas

182 ansporter is necessary for the expression of DSE.

183 The transient nature of DSE--tens of seconds--is probably determined by the regu

184 r, the study provides an in-depth picture of DSE, including the first atomistic insights into the mol

185 core using clinical variables and results of DSE stratified patients into three risk groups for morta

186 The sensitivity and specificity of DSE were 72% and 80%, respectively, when compared with c

187 al and astrocytic MAGL to the termination of DSE and DSI in Purkinje cells (PCs) in cerebellar slices

188 nificantly contributes to the termination of DSE at parallel fiber (PF) to PC synapses and DSI at put

189 isk scores nor long-term prognostic value of DSE has been described in a large diabetic population.

190 The positive predictive value of DSE was calculated for each BP group.

191 Positive predictive value of DSE was similar for patients who had hypertensive and no

192 hobic clusters might be a generic feature of DSEs that play a gatekeeping role to protect against agg

193 bese patients with diabetes who were offered DSE, a progressive decline in the glucose homeostasis an

194 of these patients had inducible ischemia on DSE (sensitivity 100%, specificity 63%).

195 us were assigned to RYGB surgery (n = 30) or DSE (n = 31).

196 We compared our DSE-CSN-based model to the traditional similarity-based

197 ss echocardiography (DSE) in these patients, DSE was included in the preoperative evaluation.

198 ion scores were similar at baseline and peak DSE using both techniques.

199 at baseline (84%: Kappa = 0.59) and at peak DSE (88.9%: Kappa = 0.72).

200 agreements for detection of ischemia at peak DSE were superior for RT-3D, 92.7% compared with 84.6% f

201 n 30 s of each other at baseline and at peak DSE.

202 We performed DSE in 80 patients with CAD and LV dysfunction (ejection

203 Positive DSE identifies patients at increased risk of TxCAD-relat

204 ry angiography within 30 days after positive DSE.

205 t are not more likely to have false-positive DSE results.

206 =1905; 9%) were more likely to have positive DSE than those with normal (n=19 770; 90%) or hypotensiv

207 versus 6 of 22 children (27%) with positive DSE (p < 0.01).

208 In patients undergoing preoperative DSE, failure to achieve target HR is not uncommon despit

209 inoid CB1 receptor antagonist AM251 prevents DSE, and the agonist WIN 55,212-2 occludes DSE.

210 l)piperidine-1-carboxylate (JZL184)] prolong DSE in autaptic hippocampal neurons, whereas inhibition

211 show that genetic deletion of MAGL prolonged DSE at parallel fibre (PF) or climbing fibre (CF) to Pur

212 Hermes transposase differs from the proposed DSE motif.

213 l and anatomical evidence that MGL regulates DSE in autaptic hippocampal neurons and, taken together

214 Of 99 patients, 80% reported DSE most days and many times per day.

215 autaptic hippocampal neurons exhibit robust DSE.

216 t recipients were selected to undergo serial DSE at the time of their regularly scheduled endomyocard

217 nd processing but small changes in the short DSE severely reduced cleavage efficiency.

218 on each lithium and "dielectric solvation" (DSE, dielectric solvation energies), immersion of each m

219 re synapses but, following 4 Hz stimulation, DSE is persistently reduced and recovers more rapidly.

220 t ischemia during dobutamine-induced stress (DSE) and compares the results with conventional two-dime

221 s Ischemia Syndrome Evaluation (WISE) study, DSE was assessed in women participating at the Universit

222 he prognostic value of a negative-submaximal DSE study before noncardiac surgery is unknown.

223 isease evaluated before nonvascular surgery, DSE had incremental value over clinical, electrocardiogr

224 These downstream regulatory elements, termed DSE, can bind c-Fos and JunD and transmit protein kinase

225 MAGL plays a predominant role in terminating DSE at climbing fiber (CF) to PC synapses, while both ne

226 More TAPSE than DSE studies were called "ischemic" (37% vs. 14%; p = 0.0

227 icipants lost significantly more weight than DSE participants at year 1 (net difference, -7.9%; 95% C

228 icipants lost significantly more weight than DSE participants at year 1 (net difference, -7.9%; 95% C

229 We find that DSE is normally stable at parallel fibre synapses but, f

230 The DSE also bind related proteins of the CREB/ATF family.

231 The DSE populated in buffer was compared to the urea-induced

232 The DSE that is most relevant for folding is the ensemble po

233 The DSE, which contains an octamer motif, binds broadly expr

234 An interaction between the complex and the DSE-binding protein(s) triggers NMD.

235 sitioned nucleosome that resides between the DSE and the PSE.

236 suggests that a nucleosome lies between the DSE and the PSE.

237 RuvAB catalyzes RFR, RecJ and XonA blunt the DSE (created by the RFR), and then RecBCD loads RecA4142

238 Both the DSE and the PSE are protected from digestion, and the pa

239 ogeneous network was built by connecting the DSE-CSN and the PPIN through 3684 known DTIs.

240 NMD pathway, as a consequence of either the DSE being too far from a stop codon or the presence of t

241 taining mRNAs abolishes its affinity for the DSE and fails to interact with Upf1p.

242 -8.4%) at year 4, compared with 2.0% for the DSE group at both time points (95% CIs, 1.4%-2.6% at yea

243 ions, thus serving as useful proxies for the DSE of NTL9 in 8.3 M urea.

244 d low-likelihood long-range contacts for the DSE of NTL9.

245 traying hierarchical folding observed in the DSE at 55 degrees C are also often seen at room temperat

246 kg, respectively) but were unchanged in the DSE group (0.00 +/- 0.02 and 0.004 +/- 0.003 kg, respect

247 reveals residual secondary structure in the DSE in buffer, which is stabilized by both local and lon

248 ere are transient long-range contacts in the DSE in buffer.

249 Residual structure in the DSE influences the kinetics of protein folding, the prop

250 ficant, coupled interactions can form in the DSE of globular proteins, and can involve residues that

251 been used to infer residual structure in the DSE under nondenaturing conditions, but direct character

252 ing from higher energy subpopulations in the DSE.

253 nts from higher energy subpopulations in the DSE.

254 at residues are energetically coupled in the DSE.

255 ls, suggesting hydrophobic clustering in the DSE.

256 nsufficient to keep hydrophobes apart in the DSE.

257 ork DSEs are processed by degradation of the DSE and a pathway that includes recombination proteins.

258 icantly underestimate the variability of the DSE and DSL and their controlling processes.

259 h biases imply that the future change of the DSE and DSL may be underestimated by the climate project

260 Mutations that alter the energetics of the DSE can impact the analysis of cooperativity and folding

261 (June-August) seem to cause the delay of the DSE in austral spring (September-November).

262 study reveals the structural details of the DSE mechanism.

263 ion protection was also seen upstream of the DSE over a sequence corresponding to the binding site of

264 ortional to the inverse of the volume of the DSE, giving a compact species equivalent to a premolten

265 tion/termination cycle immediately 5' of the DSE.

266 For each drug on the DSE-CSN, its genetic targets were predicted and prioriti

267 The MC4R poly(A) site requires only the DSE and an A-rich upstream sequence to direct efficient

268 nd campaigns in years 2 to 4 (n=2241) or the DSE, which was an offer of 3 group sessions per year on

269 analysed the pattern of protection over the DSE and PSE of the U2 genes in mitotic cells.

270 transcribing polymerases accumulate over the DSE and that removal of this signal leads to transcripti

271 e pattern of methylation protection over the DSE is virtually identical to that obtained in vitro usi

272 mutant of NTL9, V3A/I4A-NTL9, populates the DSE in the absence of denaturant and is in slow exchange

273 mics simulations have been used to probe the DSE mechanism during formation of the Saf pilus from Sal

274 In this regard, the DSE behave as 12-0-tetradecanoylphorbol 13-acetate respo

275 cent pilus subunit was seen to stabilise the DSE product against unbinding, which also proceeded in t

276 Here we present data showing that the DSE of the N-terminal domain of the L9 (NTL9) ribosomal

277 era in gel shift assays, indicating that the DSE was recognized by multiple Sp family members.

278 The results suggested that the DSE-CSN-based model was able to rank known DTIs highly.

279 eractions changes little on transferring the DSE from 6 M urea to water and then to 1 M TMAO, backbon

280 d 5.2 +/- 0.7 kg less after 2 y, whereas the DSE group did not change significantly (-0.4 +/- 0.6 and

281 s the multisubunit factor SNAPc, whereas the DSE recruits Oct-1.

282 sed to form four specific complexes with the DSE (referred to as apoAI DSE-1, -2, -3, and -4).

283 bound to a consensus GATA element within the DSE that was recognized by recombinant human GATA-6 as w

284 s seen in a primary care setting about their DSE, health perceptions, pain, energy, and depression.

285 r frequency of U and GU nucleotides in their DSE compared with canonical poly(A) signals.

286 two competing polyadenylation signals, this DSE increases the utilization of upstream poly(A) sites

287 Thus, DSE and DSI at different synapses is not uniformly affec

288 Of the 12 patients who underwent TMLR, DSE was repeated at 3 months postoperatively in 11 patie

289 ery of function and is a valuable adjunct to DSE in the assessment of myocardial viability.

290 been proposed as an efficient alternative to DSE.

291 In contrast to DSE, MSE undergoes heterologous desensitization over the

292 Patients who showed a biphasic response to DSE before revascularization (n = 12) had the most impro

293 atients who underwent liver transplantation, DSE was normal in 25, nondiagnostic in 34 because of ina

294 Enrolled subjects underwent DSE using a modified protocol.

295 AND We studied 21 949 patients who underwent DSE at Mayo Clinic, Rochester, MN, grouped by peak systo

296 patients (303 men, 227 women) who underwent DSE before nonvascular surgery and did not sustain an in

297 of 1,183 consecutive patients who underwent DSE were reviewed.

298 chest pain, or age > or = 60 years underwent DSE.

299 TMAO are reported on the thermally unfolded DSE of Nank4-7*, a truncated notch ankyrin protein.

300 performed in 70 patients were compared with DSE findings.