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1 es depending upon the presence or absence of Descemet's membrane.
2 dle into the deep stroma without penetrating Descemet's membrane.
3 rior stroma and the anterior banded layer of Descemet's membrane.
4 development of guttae, focal excrescences of Descemet's membrane.
5 COL8A2 was observed in the anterior half of Descemet's membrane.
6 al stroma as well as immediately adjacent to Descemet's membrane.
7 nectin and collagen type IV, constituents of Descemet's membrane.
8 , but there is no staining in the underlying Descemet's membrane.
9 malfunctioning corneal endothelial cells and Descemet's membrane.
10 idually variable distances of keratocytes to Descemet's membrane.
11 nd abnormal assembly of collagen VIII within Descemet's membrane, a process that is presumed to begin
17 proposed that trophozoites cannot penetrate Descemet's membrane and the corneal endothelium to enter
18 e IV collagen on the endothelial face of the Descemet's membrane; and abnormal deposition of stromal
20 tures (trabecular meshwork, Schlemm's canal, Descemet's membrane), better postural comfort for the su
21 al epithelium, epithelial basement membrane, Descemet's membrane, ciliary epithelium, lens capsule, r
24 rneal epithelial basement membrane (EBM) and Descemet's membrane (DM) components exhibit heterogeneou
28 1) creation of BB followed by peeling of the Descemet's membrane (DM); (2) peeling off of the DM foll
30 ry (33.3%), corneal decompensation requiring Descemet's Membrane Endothelial Keratoplasty (20%), and
32 r treatment for endothelial dysfunction, but Descemet's membrane endothelial keratoplasty (DMEK) now
33 igher-order aberrations (HOAs) in eyes after Descemet's membrane endothelial keratoplasty (DMEK), Des
35 SAEK are comparable with those published for Descemet's membrane endothelial keratoplasty and better
36 pping automated endothelial keratoplasty and Descemet's membrane endothelial keratoplasty in their ma
41 antation, including increasing acceptance of Descemet's membrane endothelial keratoplasty, may alter
45 or toxic injury of the endothelium, stripped Descemet's membrane, epithelial toxicity and disruption,
46 or toxic injury of the endothelium, stripped Descemet's membrane, epithelial toxicity and disruption,
48 This technique facilitates the separation of Descemet's membrane from the stroma without affecting en
49 lagenase A, but not Dispase, of the stripped Descemet's membrane generated HCEC aggregates, which pre
52 tes were observed at variable distances from Descemet's membrane, increasing from 1.5 to 12 mum (mean
57 osterior stroma (P<0.0001), opacification of Descemet's membrane (P<0.0001), and corneal guttae (P<0.
58 pigmented cells on the posterior surface of Descemet's membrane, pointing to a possible iris injury.
59 age 5 demonstrates hydrops; 5a, acute onset: Descemet's membrane rupture and dilaceration of collagen
61 basement membranes in wild-type eyes, except Descemet's membrane, showed immunogold labeling with ant
62 MEK consists merely of donor endothelium and Descemet's membrane, so DMEK does not create a stromal i
63 sterior polymorphous membrane dystrophy from Descemet's membrane specimens removed from each eye.
64 the current study revealed that in the pre- Descemet's membrane stroma of the periphery, the degener
75 tes at the lower portions of guttae, next to Descemet's membrane, whereas CLU localized mostly on top
76 a was easily and reproducibly separated from Descemet's membrane with a combination of intrastromal c
80 e also greatly increased in the of posterior Descemet's membrane, yet another general feature shared