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1 s, were found with germline deletions of the E-box element.
2 ocyte target promoter despite binding to its E-box element.
3  regulation of APEG-1 transcription via this E-box element.
4 pression of reporter plasmids containing the E-box element.
5 peat SRE-1 or to a palindromic high-affinity E-box element.
6  activation of specific target genes through E box elements.
7 anscriptional control of MuRF1 via conserved E-box elements.
8 rs directed to the PER and CRY promoters via E-box elements.
9 required the activation of neuronal promoter E-box elements.
10 oter; this reduction was dependent on intact E-box elements.
11           The Cugbp1 promoter contains three E-box elements.
12  sterol regulatory element (SRE), and double E-Box elements.
13 equences but not those with mutations in the E-box elements.
14 ell as relief of repression through distinct E-box elements.
15 cally, is capable of binding to both Inr and E-box elements.
16 x but contained a TATA box and AP1, AP2, and E-box elements.
17  -43 in the Per2 promoter but not on CRE and E-box elements.
18 ng domain which recognizes the enhancer-box (E-box) element 5'-CANNTG-3' (where N = any nucleotide).
19 xpression by binding to DNA at enhancer-box (E-box) elements 5-CACGTG-3.
20 L1 heterodimers activated transcription from E-box elements, a type of transcription factor-binding s
21 vate the hGH1 promoter, and mutation of this E-box element adversely affected basal activity after ge
22               Here, we show that a conserved E-box element also contributes to control in the islet a
23 uman BMAL1/MOP4 heterodimers bound the AANAT E box element and enhanced transcription.
24  differentiation and enhances its binding to E box elements and transcriptional activity.
25 r deletion analysis, we have mapped a distal E-box element and a proximal Ikaros site that are essent
26 nimal hTERT promoter contains Myc-responsive E-box elements and recent studies have suggested a role
27               It binds with high affinity to E-box elements and, through interaction with coactivator
28 ssion: 1) CpG methylation of the core of the E-box element, and 2) binding of a distinct protein comp
29                    The enhancer contains two E-box elements, and one FTZ-F1 nuclear hormone receptor
30 ned TATA and CCAAT boxes; Sp1, AP1, AP2, and E-box elements; and erythrocyte-specific CACCC and Krupp
31 vates MTA1 transcription through a conserved E-box element at its promoter.
32 through direct Twist1 binding to a conserved E-box element at the IL-6 promoter.
33 rect c-Myc target; it contains two consensus E-box elements, both of which bind c-Myc.Max heterodimer
34 her affinity than its interaction with other E-box elements (CANNTG).
35 rt the identification of a Ca(2+)-responsive E-box element, CaRE2, within BDNF promoter III that bind
36 romoter activity, while the proximal GAS and E box elements contribute to the IFN-gamma inducibility
37 olutionarily conserved and overlaps with the E-Box element critical for BMAL1/CLOCK binding and its t
38 ed hTERT promoter fragments, we show that an E-box element downstream of the transcription initiation
39               ABF-1 is capable of binding an E-box element either as a homodimer or as a heterodimer
40 oter, the mutations in either the SRE or the E-Box elements eliminated the response to FFAs.
41 in DNA of the FT gene that possesses various E-box elements except G box.
42  the mouse OSCAR gene contains two potential E-box elements for microphthalmia transcription factor (
43 emonstrated that the betaGK and insulin gene E-box elements formed the same cell-enriched (BETA2:E47)
44                                          The E-box elements had a minimal role in E-cadherin transcri
45  expression of cathepsin B by binding to the E box element in the cathepsin B promoter as a repressor
46 ion of the hTERT promoter was dependent upon E box elements in the core promoter.
47 al cells is controlled by two well-conserved E box elements in the large first intron.
48 wist transcriptional repressors that bind to E box elements in the TNF promoter and suppress NF-kappa
49                  We identify a non-canonical E-box element in Bmal1 and demonstrate that is required
50  in a STAT3-independent manner, requiring an E-box element in the CCL2 promoter to mediate transcript
51 ecific transcription factor HNF-3beta and an E-box element in the distal enhancer adjacent to an NF-k
52              We further uncover a functional E-box element in the enhancer region, linking the expres
53 gulating HP1gamma by directly binding to the E-box element in the first intron of HP1gamma gene, and
54           Both c-Myc and NeuroD can bind the E-box element in the insulin promoter, but unlike NeuroD
55                 Previously, we identified an E-box element in the mouse APEG-1 proximal promoter, whi
56 zed by measuring their ability to bind to an E-box element in the NEUROD1 promoter in vitro and to in
57 romised the ability of NEUROG3 to bind to an E-box element in the NEUROD1 promoter.
58                           ASCL1 binds to the E-box elements in IGFBP5 and directly regulates its tran
59 ELIN, uncovered NEUROD2 binding to conserved E-box elements in multiple introns.
60 athway that augments binding of c-Myc to key E-box elements in the AEG-1 promoter, thereby regulating
61 aEF1 increased enhancer activity of upstream E-box elements in the Col1a2 gene.
62 crophthalmia transcription factor, acting at E-box elements in the Ctsk promoter.
63                                              E-box elements in the E-cadherin promoter were found to
64 gnaling induces Snail, which binds conserved E-box elements in the PGDH promoter to repress transcrip
65        Taken together, the data indicate the E-box elements in the proximal E-cadherin promoter are c
66 ial occupancy of c-Myc and Prx1 complexes at E-box elements in the prx5 gene proximal promoter.
67  is suggested that rUSF1s, which bind to two E-box elements in the SP-A gene 5'-flanking region, may
68 ptional activation by c-Myc through specific E box elements is thought to be essential for its biolog
69            The results clearly show that the E-box element is not the SREBP recognition site in this
70                                         This E-box element is required for maximum activity of the AT
71 ory factor(s) interaction with the canonical E-box element located within the alphaBC promotor.
72     c-Myc/Max heterodimers bind to canonical E-box elements located in the bax promoter region as dem
73                  We now report that a distal E-box element, located between bp -309 and -314 upstream
74                                         This E-box element mediated repression of hTERT transcription
75 iR-210-Mnt pathways caused c-Myc to bind the E-box element of cyclin D1, instead of Mnt, resulting in
76       Nuclear binding activity of Myc to the E-box element of p53 and cyclin D1 increased, whereas th
77 known mediator of ER stress and requires the E-box element of the E4bp4 promoter.
78 te IA-1 gene expression through the proximal E-box element of the IA-1 promoter.
79 anscription in vivo through both Inr and the E-box elements of the adenovirus major late promoter.
80                               Twist bound to E-box elements on AKT2 promoter and enhanced its transcr
81 moter analyses demonstrate that the proximal E box element serves as a myogenin binding site and is b
82 c-finger transcription factors known to bind E-box elements, SLUG and SNAIL, repressed E-cadherin-dri
83 ranscription factor binding sites, including E-box elements that are consensus Myc binding sites, as
84 ter regions of both genes, we identified two E-box elements that work in conjunction to activate tran
85 omoter sequences revealed an enhancer motif (E-box) element that binds the circadian transcriptional
86 tiple TATA and CCAAT boxes and Sp1, AP1, and E-box elements; the exon 1 promoter lacked a CCAAT box b
87                        CLK-CYC directly bind E box elements to activate transcription, but the mechan
88 -like sequences cooperate with the canonical E-box element to promote high-amplitude transcription, w
89 bind the hGH1 promoter region containing the E-box element was confirmed in the hGH1 transgenic mouse
90  reporters containing non-glucose-responsive E box elements were not activated by ChREBP-Mlx expressi
91 carbohydrate response and sterol-responsive (E-box) elements were present in the Mstn promoter and we
92          Mist1 also is capable of binding to E-box elements when complexed as a heterodimer with the
93                      In addition, a group of E-box elements, which are Myc binding sites, confer resp
94                      We demonstrate that the E-box elements, which we show can function as Myc-respon
95 that bind to and activate expression from an E-box element within its enhancer.
96 e findings indicate that by interacting with E-box elements within the promoter, MYCN can upregulate