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1 NA-binding affinity of the antibody with the E box site.
2 4 transcription through their binding to the E-box site.
3 ding and transactivation from MyoD-dependent E-box sites.
4 nd binding of MyoD complexes to oligomerized E-box sites.
5 LOCK and up-regulates the PAI-1 gene through E-box sites.
6 f genes, preferentially at noncanonical (NC) E-box sites.
7 hat pTa promoter contains a conserved tandem E box site activated by E protein, HEB.
8 e promoter of Tbeta4 at consensus Thing1 and E-Box sites and identify both activation and repression
9 eins since they bind the classic palindromic E-box site as well as the direct repeat SRE-1 element.
10  protein is nuclear and binds efficiently to E-box sites as a homodimer.
11 ues to dissect the functions of two proximal E-box sites at -165 and +44 nt in regulating the hTERT p
12 raction with Max for specific DNA binding at E-box sites, but have opposing transcriptional activitie
13 gh several mechanisms including occupancy of E box sites by mSharp-1 homodimers and by direct physica
14 -GGTCAC-3', which differs from the canonical E-box site, CANNTG, to which most other bHLH proteins bi
15 e octamer motifs and a single muE5 motif (an E-box site, consensus CANNTG) interact for its function.
16                             They bind to the E-box site, found in regulatory regions of B cell-specif
17 r, the possibility remains that the GATA and E box sites function in enhancer independent DV101S1 ger
18 gation, we determined the means by which the E-box site functions in this basal repression.
19 quence specificity as a factor binding to an E box site in the CD4 enhancer; thus, a member of the bH
20 s heterodimers with Max protein that bind to E-box sites in DNA and stimulate genes required for prol
21                          Mutations of Ets or E-box sites in either DNA motif abolished the activation
22  with the CLOCK:BMAL1 complex for binding to E-box sites in target genes.
23 SNAI1, SNAI2, TWIST1 and ZEB1, which bind to E-box sites in the EpCAM promoter.
24  significantly regulated through the AP-1 or E-box sites in the promoter.
25 readily homodimerizes and binds to identical E-box sites in vitro.
26 rgy was shown to be GATA site-dependent, but E-box site-independent.
27 ssociation with USF1 and accumulation at the E-box site of the C/EBPalpha promoter in differentiated
28 ted in thymocytes and is thought to regulate E-box sites present in many T cell-specific gene enhance
29 er, a nuclear factor one (NFI) site, and two E-box sites, suggesting a conserved mode of regulation.
30     ChREBP-Mlx binding discriminated between E box sites that are glucose-responsive and those that a
31  Igkappa gene locus, including NF-kappaB and E-box sites that are important for the activity.
32 finity of the CLOCKDelta19:BMAL1 complex for E-box sites, thereby permitting increased USF1 occupancy
33 7 reporter construct, containing a hexameric E-box site, was used.
34                                          Two E-box sites, which bind E protein transcription factors
35 nds the TFEB recognition sequence CACGTG (an E box site) with a 5-10-fold lower affinity than TFEB.
36 of the protein and on the sequences of three E-box sites within PHM's first intron; the sites make di
37 s are identified that interact with specific E-box sites within the Dll3-promoter in vitro.
38                                 Mutations of E-box sites within this enhancer did not result in an ap