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1                                              E. amylovora overwinters in cankers which play an import
2 encapsulated biocontrol agent, E325, against E. amylovora, and could serve as a model for further stu
3                                           An E. amylovora dspE mutant, however, elicited a hypersensi
4 t a genetic outlook on the progression of an E. amylovora infection in the host.
5 genic and epiphytic Erwinia species, such as E. amylovora; E. carotovora subsp. atroseptica, betavasc
6 observed after the addition of an autoclaved E. amylovora suspension.
7                     The response elicited by E. amylovora in its host during disease development is s
8 ction, another exopolysaccharide produced by E. amylovora.
9  homologs in E. carotovora, E. chrysanthemi, E. amylovora, E. herbicola, E. stewartii and E. rhaponti
10                                 By contrast, E. amylovora produces 430- and 300-base rsmB transcripts
11 produce antibiotics that effectively control E. amylovora, the bacterial pathogen responsible for the
12 nce activities in Xenopus oocytes and during E. amylovora and P. syringae infections.
13 ly reduced the induction of Hsr203 following E. amylovora challenge, further demonstrating a role for
14 tions, we depicted a clear invasion path for E. amylovora cells, from epiphytic growth on glandular t
15                     The CobB(L) isoform from E. amylovora restored growth of as S. enterica cobB muta
16 ivo expression technology system to identify E. amylovora genes that are activated during infection o
17                               Conversely, in E. amylovora, the homologous glucosyl transferase activi
18                     We show that rsmB(Ea) in E. amylovora positively regulates extracellular polysacc
19 e secretion of the main exopolysaccharide in E. amylovora, amylovoran, leading to increased biofilm f
20 hat three of the five predicted DGC genes in E. amylovora (edc genes, for Erwinia diguanylate cyclase
21 the first to describe a role for c-di-GMP in E. amylovora and suggest that downregulation of motility
22                           Deletion of hfq in E. amylovora Ea1189 significantly reduced bacterial viru
23 bscission is associated with the increase in E. amylovora population.
24 OmrAB and RmaA regulate swimming motility in E. amylovora.
25 onstrated that CsrA plays a critical role in E. amylovora virulence and suggested that negative regul
26 ringae were identified for the first time in E. amylovora and included HecA hemagglutinin family adhe
27 acA-Csr system regulates virulence traits in E. amylovora remains unknown.
28 saccharide production, and thus virulence in E. amylovora.
29 ce of different bacterial species, including E. amylovora, suggesting that pollinators play a role in
30 mmercially available bacterial antagonist of E. amylovora (BlightBan, Pseudomonas fluorescens A506) c
31 r exopolysaccharide, is a virulent factor of E. amylovora.
32 Erwinia species, we cloned the rsmB genes of E. amylovora (rsmB(Ea)) and E. herbicola pv. gypsophilae
33    The genome of apple, an important host of E. amylovora, has been sequenced, creating new opportuni
34                                      HrpW of E. amylovora consists of two domains connected by a Pro
35                        We identified hrpW of E. amylovora, which encodes a protein similar to known h
36 ive detection and absolute quantification of E. amylovora live cells in apple and pear cankers collec
37 er than that in the wild type (WT) strain of E. amylovora.
38 quence of Ea273, a highly virulent strain of E. amylovora.
39 ns restored pathogenicity to dspE strains of E. amylovora, although restored strains were low in viru
40 egree of genetic uniformity among strains of E. amylovora, suggesting that the pathogen has undergone
41 al proteins confirms that the hrp systems of E. amylovora and P. syringae are closely related to each
42 und to be required for the full virulence of E. amylovora.
43 future, this methodology could shed light on E. amylovora population dynamics in cankers and provide
44 ation of the impact of floral microbiomes on E. amylovora colonization, and summarize the current kno
45                     This study suggests that E. amylovora utilizes a variety of strategies during pla
46 ations of extensive similarities between the E. amylovora and P. syringae hrp systems in pathogenesis
47 look on the genetic elements contributing to E. amylovora pathogenesis, including an exploration of t
48 f flagellar motility appears to be unique to E. amylovora and may have recently evolved.
49  ug/mL against the growth of A. tumefaciens, E. amylovora, and P. atrosepticum respectively, which we
50              In response to inoculation with E. amylovora, the BIS3 gene was expressed in stems of cv