ライフサイエンスコーパス: E

1 E-Health interventions reported significant improvements

2 E-selectin and ICAM-1 partially mediated the association

3 E-selectin and intercellular adhesion molecule (ICAM)-1

4 E. chaffeensis cannot synthesize phosphatidylcholine or

5 E. coli DeltaL YA constitutively co-expressing alpha-L-f

6 generate (E)-1-cyano-1,3-butadiene (1) (10:1 E/Z) via tandem S(N)2 and E2' reactions.

7 t ca. 242 cm(-1) (A(1g)) and ca. 280 cm(-1) (E(2g)(1)) with the thickness of the deposits acquired fr

8 We first analyze the E(T(1)), E(S(1)), and E(T(2)) of benzene and cyclobutadiene (CBD)

9 ion of key inflammatory factors (eg, ICAM-1, E-selectin, MCP-1) in endothelial cells or vascular smoo

10 tors, fos-1 (Fos), egl-43 (EVI1/MEL), hlh-2 (E/Daughterless) and nhr-67 (NR2E1/TLX), that mediate anc

11 clovamide, (2S)-3-(3,4-dihydroxyphenyl)-2-[[(E)-3-(3,4-dihydroxyphenyl)prop-2-enoyl]amino]propanoic a

12 longitudes (81 degrees 01' to 129 degrees 28'E).

13 se pair resolution dissection of more than a E. coli promoters in 12 growth conditions.

14 Laurokanols A-E (1-5) comprise an unreported tricyclic core with a [6,

15 d the highest antimicrobial activity against E. coli, S. aureus, and S. typhi in in vitro antimicrobi

16 ulsions, and chlorhexidine solutions against E. coli and S. aureus.

17 Instead, serine alters E. coli's 1C-metabolism, reduces the provision of nucleo

18 Although E(4)(H(2),2H) has not been trapped, cryogenic photolysis

19 In this system, an E. coli recording strain is exposed to a microbial sampl

20 Neurog1 directly regulates cxcr4b through an E-box cluster located just upstream of the cxcr4b transc

21 rombin and prethrombin-2, consistent with an E*-E equilibrium and providing no evidence that free thr

22 tput at minimal promoters with or without an E-box.

23 We first analyze the E(T(1)), E(S(1)), and E(T(2)) of benzene and cyclobutadiene (CBD) as excited-s

24 quently contaminated with E. coli (69%), and E. coli levels were the highest during the wet season.

25 dia (blank broth, Staphylococcus aureus, and E. coli).

26 ooxime-modified lysine, C' = citrulline, and E = glutamic acid).

27 ith serovars D (UW-3/Cx), D (UCI-96/Cx), and E (IOL-43) but not F (N.I.1).

28 In contrast, comparisons between Def and E- or E+ accessions showed a number of significant diffe

29 ant differences were recorded between E- and E+accessions.

30 PCR (capable of detecting E. histolytica and E. dispar), our tetraplex real-time PCR assay demonstrat

31 apply pico-Newton scale forces to lipids and E-cadherin molecules at the living cell surface.

32 dinated by hydrophobic residues of the M and E transmembrane helices that form a binding pocket not p

33 ic defences effective against both Ma549 and E. muscae.

34 assay was performed on Ab-modified MBs, and E. coli could be quantified in tap water and milk.

35 RNA-binding protein CUGBP1 with occludin and E-cadherin genes in DRA-KO mouse colon, suggesting that

36 Resolvins (D-series and E-series) are pro-resolving lipid mediators involved in

37 rsible cathodic process (E(pc) = -1.58 V and E(pa) = -1.47 V vs SHE) that is followed by an irreversi

38 (E)-2-octenal, nonanal, (E)-2-decenal, and (E,E)-2,4-octadienal), which did not occur with free PSO.

39 ncreased the presentation of HLA-B, -C, and -E on lung epithelial cells.

40 Most importantly, in an apolipoprotein E deficient (ApoE(-/-)) atherosclerosis progression muri

41 We identified apolipoprotein E (ApoE) as one of the proteins that are found in comple

42 otide polymorphisms (SNPs) in Apolipoprotein E (APOE) are known to increase risk for developing Alzhe

43 ated with genetic variants in apolipoprotein E (APOE), forkhead box class O3a, clusterin, and phospha

44 The E4 variant of apolipoprotein E (APOE4), the main susceptibility gene for Alzheimer's

45 The apolipoprotein E (APOE) epsilon4 allele is the most common genetic risk

46 bumin (alpha-La), was achieved after 4 h, at E/S ratios of 1/150 U/mg, regardless the initial protein

47 cess to the congeneric (+)-actinoallolides B-E.

48 ntaining E-cadherin extend into gaps between E-cadherin clusters on neighboring cells, while reformat

49 o suggesting a recalcitrant mismatch between E. coli physiology and growth on citrate.

50 ignificant differences were recorded between E- and E+accessions.

51 and KLF15, but not KLF4, occupied the bICP0 E promoter at late times during productive infection of

52 PLZF, and GR had little effect on the bICP0 E promoter.

53 trating PER removes CRY from the CLOCK-BMAL1-E-box complex.

54 s pure compounds: (E)-1-cyano-1,3-butadiene (E-1), (Z)-1-cyano-1,3-butadiene (Z-1), 4-cyano-1,2-butad

55 at the discrimination between RNA ligands by E. coli ProQ and Hfq depends both on positive determinan

56 The short U-C distances and obtuse U-C-E angles suggest significant U=C double bond character.

57 Mongolia from the late second millennium B.C.E. through the Mongol Empire are traditionally assumed,

58 compared with the acid-mediated [(64)Cu]Cu-c[E(4)W(5)C] tracer.

59 ing the convenient cell isolation from Ccl17(E/+) reporter mice; it also exploited both CCL17-depende

60 el constrained by a large set of single-cell E. coli flagellar synthesis data from different strains

61 The class-E group demonstrated improved survival and a high respon

62 Results were interpreted according to CLSI E. coli breakpoints, with 49.0 to 85.8% considered susce

63 charge leached at the slowest rate from CNT/E films.

64 bition zone method against Escherichia coli (E. coli) and Staphylococcus aureus (S. aureus), which de

65 n (DIP) Human, Drosophila, Escherichia coli (E. coli), and Caenorhabditis elegans (C. elegan) dataset

66 Antibiotics eliminated collagenolytic E faecalis and P mirabilis but did not reduce tumor form

67 good yields and isolated as pure compounds: (E)-1-cyano-1,3-butadiene (E-1), (Z)-1-cyano-1,3-butadien

68 through direct Twist1 binding to a conserved E-box element at the IL-6 promoter.

69 ve imaging shows that microspikes containing E-cadherin extend into gaps between E-cadherin clusters

70 e, only a handful of metabolites containing (E)-Dhb have been found thus far.

71 e apparent for each manufacturer for control E. coli strains.

72 duce the Tactile Resistive Annularly Cracked E-Skin (TRACE) sensor to address the inherent trade-off

73 bolA potentiated carbapenem efficacy in CRE E. coli, whereas inhibition of the genes flhC and ygaC c

74 fficiently and selectively captured known CS-E-binding proteins in vitro and in cells.

75 sa with serovars D (UW-3/Cx), D (UCI-96/Cx), E (IOL-43), or F (N.I.1).

76 essed in the placenta between embryonic day (E) 9.5 and E12.5.

77 ngle-cell transcriptomes from embryonic day (E)7.5, when endoderm progenitors are specified, to E10.5

78 factors that regulate stimulation-dependent E-P interactions.

79 treatment that thins F-actin bands, depletes E-cadherin, and stimulates proliferation in long-quiesce

80 e also determined that this assay can detect E. histolytica DNA in the presence of 10-fold more DNA f

81 d duplex real-time PCR (capable of detecting E. histolytica and E. dispar), our tetraplex real-time P

82 king activity is more likely to occur during E/I imbalances.

83 bin and prethrombin-2, consistent with an E*-E equilibrium and providing no evidence that free thromb

84 cooperatively transactivate the bICP0 early (E) promoter.

85 in a multivariate regression model, KEi(EDV) E/A ratio and 4D flow derived tricuspid valve stroke vol

86 he accompanying Explanation and Elaboration (E&E) document, which serves (1) to explain the rationale

87 anic carbon source, pointing to S. elongatus-E. coli K-12 as the most active community.

88 tuted BC dihydrodipyrrin afforded the enone (E, 70%; Z, 3%); subsequent double-ring cyclization of th

89 ng layers-the thalamo-recipient entopallium (E), an overlaying nidopallial stripe called intermediate

90 Amino acid 159 of the envelope (E) protein is reportedly implicated in the different lev

91 asibility of using this approach to estimate E/E (m).Supplemental material is available for this arti

92 types of estrogens-planar [17beta-estradiol (E(2))] and angular triphenylethylene (TPE) derivatives-w

93 ation-evoked, temporally dynamic excitatory (E) and inhibitory (I) activity.

94 utyric acid (GABA) are the major excitatory (E) and inhibitory (I) neurotransmitters in the brain, re

95 In ovalbumin (OVA)-expressing E.G-7 tumor-bearing immune-deficient mice, intravenously

96 of adipose FABP (A-FABP) and epidermal FABP (E-FABP) in the fields of obesity, chronic inflammation,

97 ve LPA(1) receptor agonist described so far (E(max) = 118%, EC(50) = 0.24 muM, K(D) = 19.6 nM; inacti

98 Epichloe coenophialum)-infected tall fescue (E+) and are hallmark signs of fescue toxicosis (FT), a d

99 lity K at moderate normalized electric field E/N and field asymmetric waveform IMS (FAIMS) relying on

100 nes encoding aggregative adherence fimbriae, E. coli common pilus, flagellin and EAEC heat-stable ent

101 suppressed growth and colonisation by focal E. coli but also prevented it from evolving antibiotic r

102 anchors and Z-ring regulators described for E. coli.

103 Curli expression is required for E. coli to exacerbate alphaSyn-induced behavioral defici

104 Nonetheless, the accumulation from E-cigs (24%) was less than that from C-cigs (32%) in bot

105 died how frameshift-inducing stem-loops from E. coli dnaX mRNA and the gag-pol transcript of Human Im

106 o paddy soils covering redox potentials from E(H) -260 to +200 mV.

107 ous bacteria could obtain these vectors from E. coli through several mechanisms of horizontal gene tr

108 ction of papers offers perspectives on how G-E interplay operates contingently within and against a b

109 lective synthesis was developed to generate (E)-1-cyano-1,3-butadiene (1) (10:1 E/Z) via tandem S(N)2

110 structures failed to recognize glycoprotein E, the well-known binding partner of gI.

111 across 400 melanoma hematoxylin and eosin (H&E) specimens from The Cancer Genome Atlas.

112 roteinaceous fluorophores, immunostaining, H&E staining, and light-sheet fluorescence microscopy.

113 CRImage) was used to classify cells in the H&E specimen into stromal, immune, or cancer cells.

114 al cancer specimens with dMMR or MSI using H&E-stained slides; it detected tissues with dMMR with an

115 oped enzymatic-cleavage site between helices E and F and pulled from the top of the E helix using a s

116 colin(R), the licensed vaccine for hepatitis E virus (HEV).

117 inst volatile compound formation (heptanal, (E,E)-2,4-heptadienal, (Z)-2-heptenal, octanal, pentanal,

118 presentative green odorants (i.e., hexanal, (E)-2-nonenal, (E,E)-2,4-decadienal) of soy drink, among

119 heptenal, octanal, pentanal, (E)-2-hexenal, (E)-2-octenal, nonanal, (E)-2-decenal, and (E,E)-2,4-octa

120 FAIMS) relying on the evolution of K at high E/N causing strong ion heating.

121 Higher E-selectin (n = 1,810) and ICAM-1 (n = 1,548) at Y7 were

122 les expressed in humans, HLA-E*01:01 and HLA-E*01:03, can lead to the activation of unconventional T

123 een the two alleles expressed in humans, HLA-E*01:01 and HLA-E*01:03, can lead to the activation of u

124 xpressed high levels of the NKG2C ligand HLA-E.

125 a the nonclassical MHC class Ib molecule HLA-E, with nearly complete identity between the two alleles

126 monstrated that EBV peptides can bind to HLA-E and block inhibition of NK cell effector function.

127 ly differential genomic regions, identifying E-box motifs common to epithelial-mesenchymal transition

128 interleukin-5 (IL-5), IL-13, immunoglobulin E and eosinophils.

129 nical sequence motifs and copy numbers as in E. maclovinus, showing limited impact of relaxed selecti

130 evice using an engineered genetic circuit in E.coli, which responded to microgravity by changing the

131 sm by which spacer orientation is defined in E. coli.

132 This suggests fly populations differ in E. muscae-specific resistance mechanisms as well as gene

133 Severe developmental errors in E- embryos were characterized by improper nervous system

134 first 5' to 3' RNA exonuclease identified in E. coli.

135 the knob-into-hole and WT IgG4 molecules in E. coli.

136 ntributes to mutagenesis of S (p)-Me-PTEs in E. coli.

137 data show that endogenous DNA gap repair in E coli supports efficient multiplex site-directed mutage

138 udy defines an antifolate stress response in E. coli and links its associated metabolites to a major

139 hosphate buffered saline (PBS) and spiked in E. coli cell lysate.

140 f ribosome recycling on protein synthesis in E. coli.

141 the effectiveness of antibiotic treatment in E. coli-induced myositis and a clinically relevant S. au

142 energy-adjusted Dietary Inflammatory Index (E-DII) and Healthy Eating Index (HEI)-2015 scores were c

143 energy-adjusted Dietary Inflammatory Index (E-DII) score during pregnancy with emotional and behavio

144 Balance between excitation and inhibition (E-I balance) in neural circuits is believed to be tightl

145 between synaptic excitation and inhibition (E/I balance) maintained within a narrow window is widely

146 eurotransmission, and excitation-inhibition (E-I) balance.

147 strated alteration of excitation/inhibition (E/I) balance and aberrant synaptic plasticity at the cor

148 dy of postsynaptic excitation to inhibition (E/I ratio) imbalances in human brain diseases, is a high

149 Cryo-EM reveals that DNAs transported into E-S/E-K compartments are 'clamped' in a sub-compartment

150 nd genotypes observed among the investigated E. coli isolates.

151 less, maturation and migration of LC lacking E-cadherin was not altered, neither under steady-state n

152 ta E . B term in electromagnetic Lagrangian (E and B are the electric and magnetic field, respectivel

153 B-lines, E/e', and BNP significantly increased during exercise (P

154 ively characterize gene repression in living E. coli by a collection of individual TALED loops with s

155 s with a stretched exponential decay model (<E(FRET)(exp)> = 0.25 +/- 0.05) and those calculated from

156 SFs for nontransitioned aerosols in the RH(<E), RH(E-D), and RH(>D) zones were 0.18 +/- 0.06, 0.05 +

157 ed from the molecular dynamics simulations (<E(FRET)(MD)> = 0.18 +/- 0.14).

158 y (94.5%) and was a better predictor of FA-M/E positivity than abnormal protein.

159 protein expression of the epithelial marker E-cadherin either remained unaltered or increased.

160 ILT), and lymphocyte antigen 6 family member E (LY6E), the three cellular proteins identified to func

161 eria with costained nucleoids and membranes (E. coli) on surfaces with nanopillars.

162 ted by MHC class II and the nonclassical MHC-E molecule in RM.

163 PAgs can be microbial [(E)-4-hydroxy-3-methyl-but-2-enyl pyrophosphate, HMBPP] o

164 The cell adhesion molecule E-cadherin is a major component of adherens junctions an

165 he actin in actin waves. In contrast, Myo1D, E, and F are enclosed by the actin wave.

166 be strengthened by a rare mutation (D839Y/N/E) from a European strain of SARS-CoV-2.

167 ks using novel engineered carbon nanosheets (E-CNS) derived from sub-bituminous coal.

168 unds, made by nonanoic acid, 2,4-nonadienal (E,E), 1-hexanol, 1-heptanol, and 3-octen-2-one, expresse

169 nal, (E)-2-hexenal, (E)-2-octenal, nonanal, (E)-2-decenal, and (E,E)-2,4-octadienal), which did not o

170 een odorants (i.e., hexanal, (E)-2-nonenal, (E,E)-2,4-decadienal) of soy drink, among of which hexana

171 6F10 murine melanoma cells expressing or not E-cadherin.

172 redicted all-cause hospitalization by 20% (O:E, 1.20) and cardiovascular hospitalization by 70% (O:E,

173 O:E, 1.70) and overpredicted death by 21% (O:E, 0.79); adding SDOH brought O:E near 1 for all outcome

174 and cardiovascular hospitalization by 70% (O:E, 1.70) and overpredicted death by 21% (O:E, 0.79); add

175 n groups (O:E = 0.65 vs 1.00, P = 0.11 and O:E = 0.79 vs 1.00, P = 0.15, respectively).

176 th by 21% (O:E, 0.79); adding SDOH brought O:E near 1 for all outcomes.

177 nth and 1 year was similar between groups (O:E = 0.65 vs 1.00, P = 0.11 and O:E = 0.79 vs 1.00, P = 0

178 g whites, models 1 to 4 all showed similar O:E ratios, suggesting high accuracy in risk prediction us

179 In the absence of E-cadherin-mediated cell adhesion, LC numbers remained s

180 d that changes in the functional activity of E-cadherin expressed on tumor cells in response to envir

181 t to assess the longitudinal associations of E-selectin and ICAM-1 with subclinical alterations in ca

182 ung adults, we evaluated the associations of E-selectin and ICAM-1, obtained at year (Y) 7 (Y7) and Y

183 The binding of E. coli to the M13 phage on the cytosensor surface incre

184 Sele(-/-) hosts) or therapeutic blockade of E-selectin using small molecule mimetic GMI-1271/Uproles

185 ZOflL We determined the lipid composition of E. gracilis strain Z mitochondria and plastids, and of p

186 Concentrations of E(39)PAHs were 1004 ngg(-1) freeze-dried weight (fdw) an

187 major driver of the physiological ecology of E. huxleyi in this system and further suggest that the a

188 pment, associated with reduced expression of E-cadherin.

189 Extract of E. camaldulensis was partitioned into aqueous and ethano

190 corresponding to a logarithmic negativity of E(N) = 0.35.

191 e re-examined the experimental parameters of E. coli in-cell NMR and found that the detectability and

192 as not been trapped, cryogenic photolysis of E(4)(4H) provides a means to address this question.

193 study, we provide a comprehensive picture of E. faecium transmission in an entire sampled patient pop

194 AV1 co-inmunoprecipitated in the presence of E-cadherin in B16F10 melanoma and other cancer cells.

195 en-starved bacteria as a biological probe of E. coli cell function during nitrogen starvation, we dem

196 Results indicated the recovery of E. coli 13457 from four MacConkey agar manufacturers was

197 IHC confirmed the down-regulation of E-cadherin (CDH1) and up-regulation of CXCL12 in endomet

198 eas the expression of negative regulators of E-cadherin was decreased.

199 ere, we describe the extent of resistance of E. dermatitidis to acute gamma-radiation exposure and th

200 study in which the daptomycin resistance of E. faecium isolated from rectal swabs from daptomycin-ex

201 foci have a role in the adaptive response of E. coli to long-term nitrogen starvation.

202 the fore-, mid- and hindbrain and somites of E- embryos at 24 hpf.

203 The 70S ribosome structure of E. faecalis now extends our knowledge of bacterial ribos

204 which will inform the future translation of E. faecium sequencing into routine outbreak detection an

205 r an electric field suggest that the optimum E(0) is determined by characteristics of the surface-act

206 contrast, comparisons between Def and E- or E+ accessions showed a number of significant differences

207 in prostates challenged with either UPEC or E. faecalis was equal or greater than primary infection

208 Two other E. coli proteins that contain SecA-like MBDs, YecA and Y

209 ins) and outside (extraintestinal pathogenic E. coli, or ExPEC).

210 s followed by an irreversible cathodic peak (E(pc) = -2.17 V vs SHE).

211 adienal, (Z)-2-heptenal, octanal, pentanal, (E)-2-hexenal, (E)-2-octenal, nonanal, (E)-2-decenal, and

212 composition of whole cells of the pigmented E. gracilis strain Z and two bleached mutants that lack

213 ay facilitate transmission of mcr-1 positive E. coli to humans and livestock through fecal contaminat

214 ls are readily colonized with mcr-1 positive E. coli, their shedding patterns, transmission among con

215 We found that vaccination with Ad4-prM-E leads to the development of a strong anti-ZIKV T-cell

216 V antibodies, while vaccination with Ad5-prM-E leads to the development of both anti-ZIKV antibody an

217 vaccine research has focused on the E or prM-E proteins and the induction of high levels of neutraliz

218 of 1 exhibits a reversible cathodic process (E(pc) = -1.58 V and E(pa) = -1.47 V vs SHE) that is foll

219 imipenem activity against an NDM-1-producing E. coli clinical strain.

220 ptor activated with U46619 and prostaglandin E(2) receptor subtype 3 activated with iloprost showed a

221 nflammatory mediators, such as prostaglandin E(2) (PGE(2)), bradykinin (BK), and nerve growth factor

222 d confirmed by prolongation of prostaglandin E(2) (PGE(2)) hyperalgesia.

223 f the SARS-CoV-2 virus, the envelope protein E, forms a homopentameric cation channel that is importa

224 e and arrangement of their surface proteins (E and prM in immature virus or M in mature virus).

225 imaging of the local density of states N(r, E) (where E is the energy) reveals electronic modulation

226 lizing the topological defects in these N(r, E) density waves at 2Q, we find them to be concentrated

227 he first characterisation of the loss of Rab-E function.

228 peak early-diastolic annular velocity ratio, E/E': sham, 13.6+/-2.1, banding, 18.5+/-4.1, P=0.014) ac

229 th undifferentiated levels of CTC-recruiting E-selectin under DF vs UF conditions.

230 mperate dynamics at environmentally relevant E. huxleyi host densities rather than virulent dynamics,

231 udies, the M. tuberculosis Tam also replaced E. coli BioC both in vivo and in vitro and complemented

232 fecal prevalence of ciprofloxacin-resistant E. coli.

233 r nontransitioned aerosols in the RH(<E), RH(E-D), and RH(>D) zones were 0.18 +/- 0.06, 0.05 +/- 0.02

234 ction of the Not5 subunit with the ribosomal E-site in Saccharomyces cerevisiae This interaction occu

235 hat such sRNAs are natural products of RNase E-mediated mRNA decay and associate with major RNA-bindi

236 ctive electrophilic aromatic substitution (S(E)Ar) reactions that are enabled via the application of

237 yo-EM reveals that DNAs transported into E-S/E-K compartments are 'clamped' in a sub-compartment crea

238 onize new niches, interrogation of sequenced E. coli O157:H7 genomes showed a high level of CycA cons

239 ne control strains and (ii) 288 human-source E. coli strains classified by PCR as ExPEC and non-ExPEC

240 is and function, we generated CD11c-specific E-cadherin knockout mice (CD11c-Ecad(del)).

241 We show that genes that form stable E-P hubs have less cell-to-cell variability in gene expr

242 nique two open states, we report a two-stage E-M coupling mechanism in voltage-dependent gating of K(

243 received anti-VEGF therapy according to a T&E (n = 163) or PRN (n = 101) regimen.

244 vely tuning the gap between a hot-emitter (T(E) ~ 880 K) and the cold photodetector (T(D) ~ 300 K) fr

245 thin LGE VZ progenitors, whereas Ascl1-Tcf3 (E-protein) interactions predominate in the subventricula

246 Here, we test E(p) using a variety of general biomarkers along a trans

247 ttractors in the system are equilibria, that E and M states dominate across parameter space, but that

248 We find that E-cadherin expression persists in metastatic lung nodule

249 Here, we find that E. tracheiphila has horizontally acquired an operon with

250 psoriatic skin inflammation, indicating that E-cadherin on LC does not influence their ability to orc

251 Altogether, the results show that (E)FtsN interacts with PBP1b and that this interaction pl

252 virus via an antibody that binds across the E and prM proteins, resulting in a subnanometer resoluti

253 We first analyze the E(T(1)), E(S(1)), and E(T(2)) of benzene and cyclobutadi

254 ete GEM that has been developed to date: the E. coli GEM.

255 scovered that during ligule development, the E class MADS-box TF GRCD5 activates GhCYC3 expression.

256 Our findings implicate the E. coli host 3'-5' exonucleases DnaQ and ExoT in spacer

257 Fe-2S] cluster in Fur protein is ~31% in the E. coli iscA/sufA mutant cells and is decreased to ~4% i

258 ct with LL materials induces IL-1beta in the E. granulosus infection setting.

259 duction of CRISPR cleavage activity into the E-DNA sensor, a more apparent signal change between with

260 Moreover, the investigation of the E + LW samples exposed for longer times revealed the pre

261 lices E and F and pulled from the top of the E helix using a site-specific, covalent attachment.

262 amine the doubly reduced FeMo-co core of the E(4)(2H)* limiting-state by (1)H, (57)Fe, and (95)Mo END

263 es presented here, are potent binders of the E-box DNA engaged by Myc for transcriptional activation

264 The principle of the E-DNA sensor is the target induced conformational change

265 ); subsequent double-ring cyclization of the E-enone (via Nazarov, electrophilic aromatic substitutio

266 onships and hence predicted functions of the E. gracilis plastid proteome.

267 stions on the function and regulation of the E/I balance.

268 Dysregulation of the E/I ratio is associated with numerous neurological disor

269 s (ZIKV) vaccine research has focused on the E or prM-E proteins and the induction of high levels of

270 Integrative analyses revealed the E+ microbiota correlated and co-varied with the metabolo

271 ctor pSF-OXB15-p450camfusion showed that the E. coli cells died after five days but a variety of bact

272 ted RNA and is structurally unrelated to the E. coli McrB DNA-binding domain.

273 wavelength range of 360-410 nm, whereas the E-isomer cyclization (Phi(c) = 0.006-0.008) and cyclorev

274 The isomerization of the (E)-azobenzene to its Z isomer enhances diol binding, and

275 en observed and presented by the axial theta E . B term in electromagnetic Lagrangian (E and B are th

276 e marrow-derived dendritic cells compared to E. coli LPS.

277 human brains as well as the transcriptional E/I (tE/I) ratio.

278 ntermolecular FRET measurements of wild-type E-cadherin and cis-interaction mutants combined with sim

279 edure led to cholesterol enrichment, the UAE-E:W conditions favoured the lowest concentration.

280 Ubiquitinated E is present on infectious virions of ZIKV when they are

281 stingly, we find that in B. subtilis, unlike E. coli where multiple enzymes have a biochemical activi

282 partial electron-density redistribution upon E(4)(H(2),2H) formation during catalysis, complementing

283 Vitamin E (VitE) deficiency results in embryonic lethality.

284 Vitamin E acetate was associated with EVALI in a convenience sam

285 tigate the effects of DHA (22:6n-3), vitamin E, and their probable interactions in men with asthenozo

286 c cancers were not different between vitamin E-exposed patients and controls.

287 A or delta-T3-13'COOH), is a natural vitamin E metabolite that has preliminarily been identified as a

288 Unlike for genistein and the other vitamin E compounds, the GA-induced restoration of ApoE efflux w

289 measure several priority toxicants: vitamin E acetate, plant oils, medium-chain triglyceride oil, co

290 riction and antioxidant therapy with vitamin E prevented liver disease.

291 th and without nicotine aerosolized at 40 W (E-smoke) with respect to particle number concentrations,

292 f the local density of states N(r, E) (where E is the energy) reveals electronic modulations with wav

293 sia were decreased in all 3 studies, whereas E coli was increased in 4 of 9 studies.

294 To determine whether E-cadherin plays a role in regulating LC homeostasis and

295 Acidosis is associated with E. coli induced pyelonephritis but whether bacterial cel

296 er samples were frequently contaminated with E. coli (69%), and E. coli levels were the highest durin

297 noculating petroleum-polluted sediments with E. coli carrying the vector pSF-OXB15-p450camfusion show

298 A mutant cells and is decreased to ~4% in WT E. coli cells.

299 hich is evidenced also to take part in the Z/E isomerization of the product.

300 ield, >98:2 alpha:gamma selectivity, >98:2 Z:E selectivity, and >99:1 enantiomeric ratio.