ライフサイエンスコーパス: EAN

1 ced murine experimental autoimmune neuritis (EAN).

2 protic ionic liquid (ethylammonium nitrate, EAN).

3 egeneration in the periphery in animals with EAN.

4 ement of MMP activity in the pathogenesis of EAN.

5 nce of the macrophage polarization status in EAN.

6 sensory neuropathy type la, can also induce EAN.

7 previously in the pathology associated with EAN.

8 ay a multifactorial role in the aetiology of EAN.

9 ur MMPs were differentially regulated during EAN.

10 tion of peripheral nerves that characterizes EAN.

11 te the P0106-125-specific immune response in EAN.

12 isease accelerated the rate of recovery from EAN.

13 amage and may even protect neural tissues in EAN.

14 nd)CC(CH(3))=C(CH(3))C(triple bond)CTol] (10-Eand 10-Z), showing no intrinsic bias toward trans-enedi

15 can induce experimental autoimmune neuritis (EAN), a model of human inflammatory neuropathy.

16 pean Association of Neurosurgical Societies (EANS) an online survey was performed from February 2023

17 B-1101, in experimental autoimmune neuritis (EAN), an animal model of Guillain-Barre syndrome.

18 cantly increased early in the development of EAN and continued to rise, peaking at day 15 coincident

19 lective upregulation of specific MMPs during EAN and their varied cellular localization suggests that

20 work (DMN), the executive attention network (EAN), and the medial visual network (MVN), with the MVN

21 NF-alpha are involved in the pathogenesis of EAN, and that drugs of this type may have potential as n

22 of immunization prevented the development of EAN, and when given from the onset of symptoms, it signi

23 tological examination of the tibial nerve of EAN animals revealed that flecainide provided significan

24 Enteric-associated neurons (EANs) are closely associated with immune cells and conti

25 mal model, experimental autoimmune neuritis (EAN), are typically acute monophasic diseases of the PNS

26 ray of embedded annular nanoband electrodes (EANEs), at which sufficient overpotential drives highly

27 reater connectivity within the DMN and right EAN (corrected P [P(corr)] < 0.05 versus controls), and

28 immune cell infiltration over the course of EAN enables accumulation of circulating nanoparticles.

29 infection-induced inflammation on intrinsic EANs (iEANs) and the role of intestinal muscularis macro

30 The clinical course of P0106-125-induced EAN in mice deficient for IL-10(0/0), IL-4(0/0), or STAT

31 me, namely experimental autoimmune neuritis (EAN), induced in Lewis rats by immunization with bovine

32 Experimental autoimmune neuritis (EAN) is an animal model of Guillain-Barre syndrome.

33 Experimental allergic neuritis (EAN) is considered the in vivo model of Guillain-Barre s

34 reclinical experimental autoimmune neuritis (EAN) model with systemically administered therapeutic co

35 2020, and the European Academy of Neurology (EAN) Neuro-COVID Registry (ENERGY) from March to October

36 etween the insula and both the DMN and right EAN (P(corr) < 0.05).

37 ormed a retrospective study of fulfilment of EAN/PNS 2021 criteria on 120 consecutive patients with a

38 The sensitivity of EAN/PNS criteria for 'CIDP' was 83.3%.

39 The sensitivity and specificity of new EAN/PNS criteria for CIDP is equivalent to that of previ

40 ademy of Neurology/Peripheral Nerve Society (EAN/PNS) for chronic inflammatory demyelinating polyneur

41 athology (onset, effector phase, and peak of EAN severity), intravenously administered small molecule

42 relating to the effector phase of Lewis rat EAN that may be relevant to C. jejuni-induced GBS are di

43 macrophages as pathogenic effector cells in EAN, these data suggest that M2-differentiated macrophag

44 localization in sciatic nerve tissue during EAN, using a semiquantitative competitive reverse transc

45 EAN was induced in Lewis rats (n = 116, in three indepen

46 ase (MMP) inhibitor, prevents development of EAN when given from the day of immunization and, more im