ライフサイエンスコーパス: ECD

1 ECD biopsies showed significantly higher expression of I

2 ECD colocalizes and coimmunoprecipitates with PERK and G

3 ECD fragments indicated unfolding started at the N-termi

4 ECD movements were modulated by binding of the glycosphi

5 ECD transplantation in children is associated with a hig

6 ECD values were significantly higher in donors <80 years

7 ECD was measured before and directly after preparation a

8 etween age and CCT (r = - 0.215, P < 0.001), ECD (r = - 0.496, P < 0.001),and hexagonality (r = - 0.2

9 % at the 4(th) month after surgery and 69.6% ECD remained at the 15(th) month postoperatively while s

10 ecipients data from 1987-2017 to identify 96 ECD and 92 > 85 KDPI kidney recipients (<18 years).

11 After adjustment, ECD recipients were at significantly increased risk of g

12 Up to 2 years of age, ECD was more inversely correlated with CD than with age

13 e found that the BMP9/BMP10 ligand trap ALK1(ECD) administered in monocrotaline or Sugen/hypoxia (SuH

14 For all ECD outcomes, there was not a statistical interaction be

15 show stable visual acuity and CCT, although ECD decreases.

16 /=25% in ECD, and percentage of eyes with an ECD <1500 cells/mm(2).

17 In multivariable analyses, ECD at 6 months and operative complications were both as

18 There was no correlation between CCT and ECD (P = 0.106).

19 h enabled us to separate and analyze CID and ECD fragmentation simultaneously.

20 tutional cohort of 189 patients with ECD and ECD overlapping with Langerhans cell histiocytosis (so-c

21 lpha-tubulin as a result of MSU exposure and ECD-shedding reduced MSU phagocytosis in murine and huma

22 sification of histiocytoses in which LCH and ECD belong to the "L" group.

23 cells was indistinguishable between LCH and ECD, although the histiocytic disorders were distinct to

24 immediate cellular precursor in both LCH and ECD.

25 to the end of follow-up and showed an annual ECD decline of 48 cells/mm(2) (standard error, 3.14) and

26 lation between both plasma anti-RBD and anti-ECD IgG titers and in vitro VN titers.

27 lationship among anti-spike ectodomain (anti-ECD), anti-receptor-binding domain (anti-RBD) IgG titers

28 We conclude that anti-RBD or anti-ECD IgG titers can serve as a surrogate for VN titers to

29 Plasma anti-RBD or anti-ECD titers of >=1:1350 may provide critical information

30 9 treatment, was >=80% when anti-RBD or anti-ECD titers were >=1:1350.

31 omatic adults found 73 individuals with anti-ECD IgG titers of >=1:50 and strong positive correlation

32 The average ECD of the 30 corneoscleral discs, which later underwent

33 nce of heterophilic cell-cell adhesion-based ECD regulation during animal morphogenesis.

34 Cell-cell adhesion-based ECD, however, has not yet been clearly demonstrated.

35 activation of the hemoglobin tetramer before ECD was reflected in the observed fragmentation pattern,

36 Removal of the beta3-ECD abrogated both the depolarizing shift of steady-stat

37 for the beta3-Glu-176 residue and the beta3-ECD in regulating the conformational changes of the volt

38 observed between eyes with FECD and BK, but ECD was significantly lower in eyes with previous failed

39 uggested 3 as the most likely structure, but ECD calculations did not match the experimental values.

40 bsolute configurations have been assigned by ECD spectroscopy.

41 whose thin films have been characterized by ECD and/or CPL, as well other chiroptical spectroscopies

42 to estimate treatment effect modification by ECD.

43 )-mediated G(s) activation is Undisturbed by ECD-scFvhFc binding.

44 The mean CCT, ECD, CV, CA, and hexagonality was 533.80 +/- 33.00 mum,

45 hology in healthy Thai eyes showed that CCT, ECD, and hexagonality were significantly decreased, whil

46 CD44 ECD-shedding reduced the conversion of procaspase-1 to a

47 Central ECD was determined by a central image analysis reading c

48 Finally, we report CIU-ECD experiments for the alcohol dehydrogenase tetramer,

49 ting improved sequence coverage by combining ECD fragmentation integrated over multiple CIU intermedi

50 but one-third of ASM corneas were compliant (ECD > 2000/mm).

51 A total of 63 adult patients with confirmed ECD were identified.

52 In contrast, ECD-scFvhFc potently inhibits beta-arrestin-2 recruitmen

53 insertion, we evaluated the central corneal ECD (CCECD) and peripheral corneal ECD (PCECD) in the ar

54 l corneal ECD (CCECD) and peripheral corneal ECD (PCECD) in the area of the tube; anterior chamber (A

55 dy, we report two cases of GPA whose corneal ECD decreased significantly after phacoemulsification ca

56 salmon calcitonin-bound, GlcNAc-bearing CTR ECD at 1.78 and 2.85 angstrom resolutions and analyze th

57 shed by a combination of spectroscopic data, ECD analysis, and total synthesis.

58 copy, we demonstrate that unlike DDR1b, DDR2 ECD and DDR2-GFP do not undergo collagen-induced recepto

59 We show that the oligomeric form of DDR2 ECD displayed enhanced binding to collagen and inhibitio

60 ) the impact of the oligomeric state of DDR2 ECD on collagen binding and fibrillogenesis, (ii) the ef

61 re conducted using purified recombinant DDR2 ECD proteins in monomeric, dimeric or oligomeric state,

62 We evaluated electrochemical degradation (ECD) and photocatalytic degradation (PCD) technologies f

63 uctural equivalence of missense and deletion ECD changes in GBMs.

64 y and at 3 months: endothelial cell density (ECD in cells/mm), corneal transparency and thickness.

65 l thickness (CCT), endothelial cell density (ECD) and complication rates.

66 raocular pressure, endothelial cell density (ECD) and patient impairment.

67 Donor endothelial cell density (ECD) decline was evaluated for 351 eyes of 275 patients

68 nation revealed an endothelial cell density (ECD) of 1532/mm(2) in patient 1 and 1620/mm(2) in patien

69 cuity (BSCVA), and endothelial cell density (ECD) prior to DMEK and at 1, 3, 6, 12, and 24 months pos

70 Endothelial cell density (ECD) was measured without any additional manipulation by

71 ual acuity (BCVA), endothelial cell density (ECD), and complications.

72 al acuity (BSCVA), endothelial cell density (ECD), and graft survival.

73 l thickness (CCT), endothelial cell density (ECD), and need for regraft.

74 al acuity (BSCVA), endothelial cell density (ECD), central corneal thickness (CCT) at 3 and 6 months

75 l thickness (CCT), endothelial cell density (ECD), coefficient of variation in cell size (CV), cell a

76 s from baseline in endothelial cell density (ECD), coefficient of variation, and percentage of hexago

77 ual acuity (BCVA), endothelial cell density (ECD), postoperative complications, and graft survival.

78 , micrometers) and endothelial cell density (ECD).

79 When the corneal endothelial cells density (ECD) drops, the HCEC may decompensate to keep cornea deh

80 an enhanced equivalent circulation density (ECD), the dynamic density condition, for efficient oil a

81 ty [BCVA], central endothelial cell density [ECD], and central corneal thickness [CCT]) were evaluate

82 Genetic analyses of a newly developing ECD lesion revealed a somatic KRAS(Q61H) mutation withou

83 d feeding (IYCF) on early child development (ECD) among children enrolled in the Sanitation Hygiene I

84 Early childhood development (ECD) programmes can help address early disadvantages for

85 spite progress, early childhood development (ECD) remains a neglected issue, particularly in resource

86 biased GPCR activation in drug development, ECD-scFvhFc could be a valuable tool to study PTH1R sign

87 re, using a WO3 -film electrochromic device (ECD) array (10 x 10 pixels) and a ZnO-nanowire-matrix pr

88 is as well as electronic circular dichroism (ECD) calculation.

89 lation of the electronic circular dichroism (ECD) spectra.

90 th calculated electronic circular dichroism (ECD) spectral data.

91 hroism (VCD), electronic circular dichroism (ECD), and optical rotatory dispersion (ORD).

92 solution NMR, electronic circular dichroism (ECD), and single-crystal X-ray diffraction analyses.

93 namic and the electronic circular dichroism (ECD)-silent 2,5-di(1-naphthyl)-terephthalaldehyde-based

94 Electronic circular dichroism, ECD, and its emission counterpart, circularly polarized

95 ivo data demonstrate that entirely different ECD variants all converge to remove N-TR1 steric hindran

96 he presence of extracranial carotid disease (ECD) is associated with less favorable clinical outcomes

97 he non-LCH neoplasm Erdheim-Chester disease (ECD) are heterogeneous neoplastic disorders marked by in

98 iocytosis (LCH) and Erdheim-Chester disease (ECD) are rare histiocytic disorders induced by somatic m

99 Erdheim-Chester disease (ECD) is a rare non-Langerhans cell histiocytosis that mo

100 Erdheim-Chester disease (ECD) is characterized by the infiltration of tissues by

101 t, and treatment of Erdheim-Chester disease (ECD).

102 neoplasms, such as Erdheim-Chester disease (ECD).

103 that had their circadian rhythms disrupted (ECD) had higher Chlamydia loads, more tissue alterations

104 used an Environmental circadian disruption (ECD) model mimicking circadian disruption occurring duri

105 of mAbs using electron capture dissociation (ECD) and 157 nm ultraviolet photodissociation (UVPD).

106 d analyzed by electron capture dissociation (ECD) and Fourier transform ion cyclotron resonance (FT-I

107 y (IRMPD) and electron capture dissociation (ECD) as fragmentation techniques, and D-alpha-tocopheryl

108 que nature of electron capture dissociation (ECD) for cleaving protein backbone bonds while preservin

109 s produced by electron capture dissociation (ECD) or 193 nm ultraviolet photodissociation (UVPD) were

110 ll to perform electron capture dissociation (ECD) within a quadrupole/ion mobility/time-of-flight mas

111 and targeted electron capture dissociation (ECD), yielding high sequence coverage.

112 atic cell for electron capture dissociation (ECD).

113 ide antibody to the gI extracellular domain (ECD) (amino acids [aa] 110 to 202) both failed to stain

114 pocket located on the extracellular domain (ECD) and allosteric regulation of the transmembrane doma

115 s binding to the large extracellular domain (ECD) and the orthosteric pocket, inducing conformational

116 e is known about their extracellular domain (ECD) conformations in the absence of orthosteric ligands

117 asparagine 130 in its extracellular domain (ECD) enhances calcitonin hormone affinity with the proxi

118 mational change of the extracellular domain (ECD) from a closed, self-inhibited tethered monomer, to

119 ntibody that sheds the extracellular domain (ECD) of CD44 to study the role of the receptor and its a

120 The extracellular domain (ECD) of class B1 G-protein-coupled receptors (GPCRs) pla

121 TcsL recognizes the extracellular domain (ECD) of SEMA6A/6B via a region homologous to the recepto

122 ue organization of its extracellular domain (ECD) relative to its 7-transmembrane (TM) core, forming

123 (TM1 and TM2), and an extracellular domain (ECD).

124 rgizing with the beta3 extracellular domain (ECD).

125 partly mediated by its extracellular domain (ECD).

126 tide encompassing the extracellular domains (ECD) of PLP.

127 the extracellular and transmembrane domains (ECD and TMD, respectively).

128 nits, adjacent to the extracellular domains (ECDs).

129 Donor ECD decreased by 37% (n = 14), 60% (n = 11), and 71% (n

130 Pre- and postoperative central donor ECD were determined by a central image analysis reading

131 Preoperative donor ECD decreased by 59% at 5 years and 68% at 10 years post

132 cell loss as compared to preoperative donor ECD for the entire study group was 33 (+/-16)%, 36 (+/-1

133 Use of expanded criteria donor (ECD) kidneys, which are associated with a reduced graft

134 es associated with expanded-criteria donors (ECD) and high Kidney Donor Profile Index (KDPI) kidneys

135 Kidneys from extended-criteria donors (ECDs) and kidneys with >=85% kidney donor profile indexe

136 ar outcomes in recipients of single and dual ECD kidneys.

137 cell sizes-or epithelial cell size dynamics (ECD)-during morphogenesis entail interplay between two o

138 NaNO(3) was used as supporting electrolyte), ECD resulted in an apparent zero-order rate of 30 mumol

139 Also, newly added modules deal with ETD/ECD analysis, multimodal mass spectra analysis, and pres

140 ch consists of three domains: extracellular (ECD), transmembrane (TMD), and intracellular domain (ICD

141 substudy from 206 clusters and evaluated for ECD at 2 years of age using the Malawi Developmental Ass

142 ary outcomes included prognostic factors for ECD loss: tube insertion entry site position relative to

143 Significant changes were observed for ECD values (1741+/-274.5 cells/mm(2) at last visit befor

144 a structure of hormone-bound, N-glycan-free ECD, which suggested that the GlcNAc might affect CTR dy

145 Preimplantation kidney biopsies from ECD (n = 41) and standard-criteria donor (n = 39) were e

146 y coupled with electron capture detector (GC-ECD), and validated for screening and quantification of

147 ctivity, although traditional techniques (GC-ECD and HPLC-DAD) are still commonly used due to their a

148 the dynamics of the glucagon receptor (GCGR) ECD in the presence of native-like membrane bilayers.

149 tants between gI and gD revealed that the gI ECD is the critical determinant, whereas the transmembra

150 ess which covariates were related to greater ECD decline.

151 ) showed the strongest relation with greater ECD decline.

152 is HPLC with electrochemical detection (HPLC-ECD).

153 sSEC/FT-ICR ECD facilitated the identification and sequence characte

154 We identified ECD-scFvhFc, which exhibited high affinity binding to bo

155 A levels were increased, suggesting impaired ECD translation as the mechanism for reduced protein lev

156 Ten years after implantation, ECD had decreased by >/=25% in 7.9% and 6.3%, whereas EC

157 centage of eyes with a decrease of >/=25% in ECD, and percentage of eyes with an ECD <1500 cells/mm(2

158 ences in changes from baseline to month 3 in ECD, CV, or %HEX between either of the netarsudil groups

159 t 6 and 12 months after DSAEK, the change in ECD from preoperative to 6 and 12 months, surgeon-report

160 At month 60, the mean percent of changes in ECD was -20.4% (95% CI, -23.5% to -17.5%) in the Micro-S

161 No statistically significant changes in ECD were seen in any group.

162 Changes in ECD, CV, and %HEX from baseline to 3 months were compare

163 No significant differences in ECD were observed between eyes with FECD and BK, but ECD

164 verall, robust, and reproducible efficacy in ECD, with no acquired resistance to date, but their use

165 These large-scale fluctuations in ECD conformation may affect the ligand binding and recep

166 ctivating mutation during BRAF inhibition in ECD.

167 imilar gene transcripts as those observed in ECD kidneys, except that tumor necrosis factor alpha and

168 iopsies, and gauge the treatment response in ECD patients.

169 AF/MEK inhibition as a promising strategy in ECD.

170 1, and CASP1 were exclusively upregulated in ECD kidneys.

171 Also, infected ECD mice elicited higher proinflammatory cytokines compa

172 roup-based delivery models for an integrated ECD responsive stimulation and nutrition education inter

173 ndicates that the alpha1 helix of the ECD is ECD-scFvhFc's epitope which may partially overlap with t

174 d high affinity binding to both the isolated ECD and to the full-length receptor in styrene-maleic ac

175 is characterized by the rotation of a large ECD fragment (N-TR1), deleted in the most common GBM mut

176 the neoplastic clone in 20 adults with LCH, ECD, and HCL.

177 the second case of 54-year old female, left ECD dropped 63.9% at the 4(th) month after surgery and 6

178 tive ECD is unrelated to LEGF, whereas lower ECD at 6 months is associated with LEGF.

179 Mean ECD, CV, and %HEX values were comparable between groups

180 The mean ECD loss at 6 months, 1 year, 2 years, 3 years, and 5 ye

181 - 1.4 years (range 0.1-5 years) and the mean ECD was 3746 +/- 370 cells/mm(2) (range 3145-5013 cells/

182 Median ECD decreased from 2646.0 mm(2) to 2497.5 mm(2) (p = 0.0

183 In the chlorine mediated ECD approach, significant quantities of free chlorine (h

184 % (0.0%, 2.4%) for 310 grafts with a 6-month ECD between 1200 and 2000 cells/mm(2), and 0.6% (0.1%, 2

185 % (0.1%, 2.7%) for 589 grafts with a 6-month ECD greater than 2000 cells/mm(2).

186 % (3.0%, 14.0%) for 97 grafts with a 6-month ECD less than 1200 cells/mm(2), 0.3% (0.0%, 2.4%) for 31

187 ouse embryonic fibroblasts (MEFs); moreover, ECD mRNA levels were increased, suggesting impaired ECD

188 directly compared with a conventional GC-MS/ECD detection system.

189 rometry or electron capture detection (GC-MS/ECD) as of yet, which often limits the ability to conduc

190 res, we created comparison groups of 375 non-ECD and 357 <= 85 KDPI recipients for comparisons with E

191 lity (aHR = 1.33; P = .15) compared with non-ECD recipients.

192 l-cell adhesion induce cell sorting, but not ECD.

193 g(abs) and g(lum)) and on the application of ECD and CPL to derive structural information on aggregat

194 uential Cox approach for survival benefit of ECD and > 85 KDPI transplantationvs remaining on the wai

195 We observed no survival benefit of ECD transplants vs remaining on the waitlist (aHR = 1.05

196 t, we shall discuss some general concepts of ECD, CPL, and other chiroptical spectroscopies, with a f

197 sition associated with the complete decay of ECD signals upon acidification.

198 patients with confirmed tissue diagnosis of ECD that showed intraocular manifestations and were imag

199 In all eligible cases, the diagnosis of ECD was made using clinical criteria in conjunction with

200 a large degree of conformational dynamics of ECD that is necessary for binding GLP-1.

201 of GRP78 reversed the attenuating effect of ECD overexpression on PERK signaling.

202 Rare intraocular manifestations of ECD confirmed on histopathology can be imaged with multi

203 Intraocular manifestations of ECD seen on multimodal imaging include histiocytic choro

204 alterations, as well as the co-occurrence of ECD with LCH in 15% of patients with ECD, led to the 201

205 Reciprocally, overexpression of ECD led to marked decreases in p-PERK, p-eIF2alpha, and

206 Ten percent of ECD cases are associated with myeloproliferative neoplas

207 Because of the relative rarity of ECD, key clinical features of the disease may not be wel

208 hilic cell-cell adhesion-based regulation of ECD.

209 Some of the most striking signs of ECD are the long bone involvement (80%-95%), as well as

210 has been assigned using TD-DFT simulation of ECD spectra.

211 ha seems to be the best initial treatment of ECD.

212 showed no clinically significant effects on ECD, CV, or %HEX when dosed QD or BID for 3 months in ey

213 It is not known if adult LCH or ECD arises from hematopoietic stem cells (HSCs), nor whi

214 ntly associated with 3-year graft success or ECD.

215 es and laboratory data of diabetic patients, ECD showed a significant negative correlation with diabe

216 In contrast, PLP(ECD) induced EAE not only in WT mice, but in B cell-suff

217 We demonstrate here that PLP(ECD)-immunized B cell-deficient mice failed to exhibit E

218 Mean postoperative ECD decreased significantly (P < .001) from eye bank val

219 Preoperative ECD was determined for 1209 eyes that did not fail and 1

220 In eyes undergoing DSAEK, preoperative ECD is unrelated to LEGF, whereas lower ECD at 6 months

221 The mean preoperative ECD was 2761 +/- 285 cells/mm(2).

222 time-to-event analyses, whereas preoperative ECD was not (P = 0.55).

223 Preoperative ECDs in the microstent group (2,432.6 cells/mm(2) [95% c

224 ian (interquartile range [IQR]) preoperative ECDs were similar for eyes with LEGF (2523; 2367-3161) c

225 ewed for patients with pathologically proven ECD.

226 ere, we used phage display to generate PTH1R ECD-specific antibodies with the aim of modulating recep

227 ER stress induction led to a reduced ECD protein level, but this effect was not seen in PKR-l

228 nts worldwide with multisystem or refractory ECD have benefitted from highly effective therapy with B

229 of heterophilic cell-cell adhesion regulate ECD: higher cell-cell adhesion results in cell size enla

230 At the final follow-up, the residual ECD was only 55% in the right eye in the 49(th) month, a

231 probable structure (6epi-1) showed the right ECD spectrum and high DP4/DP4+ probabilities obtained af

232 nd 209 eyes in the 8-14d PT group (mean [SD] ECD, 1537 [683] cells/mm2 and mean [SD] ECL, 44% [23%])

233 the 203 eyes in the 0-7d PT group (mean [SD] ECD, 1620 [673] cells/mm2 and mean [SD] ECL, 41% [23%])

234 Finally, administration of SEMA6A-ECD proteins protects human cells from TcsL toxicity and

235 nd short TL were associated with significant ECD loss with time.

236 e 15(th) month postoperatively while similar ECD of right eye before and after left eye surgery.

237 comparison of the experimental and simulated ECD data generated at different theory levels.

238 ansfer studies, we demonstrated that soluble ECD disrupts endogenous DR5-DR5 interactions.

239 the investigated compounds exhibited strong ECD signals, with Deltaepsilon values of up to 24 M(-1)

240 Available extension study ECD results at 4 years mirrored those at 3 years in the

241 roscopic analysis, in combination with TDDFT ECD calculations for determining the absolute configurat

242 es cross-reactive VNAR antibody against TfR1-ECD that does not compete with transferrin or ferritin f

243 t recombinant human TfR1 ectodomain (rh-TfR1-ECD) followed by in vivo selection in mouse for brain pa

244 r removal of non-purgeable hydrocarbons than ECD.

245 Here we present evidence that ECD regulates the endoplasmic reticulum (ER) stress resp

246 The ECD and hexagonal cell ratio were significantly lower, w

247 The ECD at 6 and 12 months after DSAEK, the change in ECD fr

248 The ECD at 6 months was associated with LEGF (P < 0.001) in

249 The ECD, CV, and %HEX were determined using a standardized p

250 se and deletion mutations concentrate at the ECD for unclear reasons.

251 inhibition of TRAIL-induced apoptosis by the ECD predominantly comes from the disruption of DR5 oligo

252 6, and April 30, 2017, were eligible for the ECD substudy.

253 gs is that POPC-only membranes frustrate the ECD-TMD coupling in such a way that the "conformational

254 first case of 69-year-old male with GPA, the ECD dropped 39.6% (OD) four months after phacoemulsifica

255 liganded-receptor gating takes place in the ECD and the interfacial M2-M3 linker but fails to penetr

256 bserved marked conformational changes in the ECD-TMD interface and the TMD.

257 d directly expressed by color changes in the ECD.

258 % of those eligible) were recruited into the ECD substudy from 206 clusters and evaluated for ECD at

259 3-subunit and in a beta3-subunit lacking the ECD.

260 asive method was presented for measuring the ECD of the EDML.

261 X-MS) indicates that the alpha1 helix of the ECD is ECD-scFvhFc's epitope which may partially overlap

262 In contrast, removal of the ECD significantly altered the dynamics of both DIII and

263 sentence 'This conformational change of the ECD triggers a clockwise rotation of the TMD.' In Extend

264 The rearrangement of the ECD-TMD interface is characterized by the movement of th

265 reveals a unique closed conformation of the ECD.

266 and advancement of diabetic retinopathy, the ECD and hexagonal cell ratio decreased, while the averag

267 After the cornea reaches adult size, the ECD decreases at a rate similar to that reported in adul

268 The analysis indicates that the ECD community faces two primary challenges in advancing

269 Simulations showed that the ECD could move about a hinge region formed by residues Q

270 ial binding of the peptide C-terminus to the ECD.

271 resolved by enantioselective HPLC, and their ECD spectra have been recorded online by stopped-flow me

272 parison between experimental and theoretical ECD spectra.

273 satisfactory, and a high proportion of these ECD kidneys are discarded.

274 ad their circadian rhythms disrupted in this ECD model will have a higher Chlamydia load, more pathol

275 Thus, ECD during morphogenesis in the heminotal epithelia of D

276 Demographics, death-to-preservation time, ECD, lens status, medical history, time on mechanical ve

277 disease (no change in symptoms attributed to ECD), and progressive disease (worsening of symptoms att

278 omplete resolution of symptoms attributed to ECD), partial response (partial resolution of symptoms a

279 partial resolution of symptoms attributed to ECD), stable disease (no change in symptoms attributed t

280 disease (worsening of symptoms attributed to ECD).

281 lementary ion mobility measurements prior to ECD provided orthogonal evidence of monomer unfolding wi

282 t quantum mechanical predictions of the VCD, ECD, and ORD for monomeric tert-butylphenylphosphinoamid

283 In addition, at day 88, viable ECD was measured using a live/dead assay, and endothelia

284 ecreased by >/=25% in 7.9% and 6.3%, whereas ECD was <1500 cells/mm(2) in 3.9% and 4.0% in the myopic

285 on of novel somatic mutation associated with ECD enabled treatment with a new-targeted systemic agent

286 ior chamber BGI insertion is associated with ECD loss greatest close to the tube.

287 ions are rarely observed in association with ECD.

288 7 <= 85 KDPI recipients for comparisons with ECD and > 85 KDPI transplants, respectively.

289 Given that corneas with ECD > 1600/mm were also usable for emergency, 58% of ASM

290 We report a patient with ECD who carried the BRAF(V600E) mutation and developed t

291 ti-institutional cohort of 189 patients with ECD and ECD overlapping with Langerhans cell histiocytos

292 nce of myeloid neoplasms among patients with ECD and MH.

293 ay be at least as effective in patients with ECD as in those without ECD, and it should not be withhe

294 Some 10.1% (19/189) of patients with ECD have an overlapping myeloid neoplasm, most commonly

295 n the prespecified subgroup of patients with ECD versus 1.3 (CI, 0.9 to 1.9) in patients presenting w

296 ence of ECD with LCH in 15% of patients with ECD, led to the 2016 revision of the classification of h

297 are found in more than 80% of patients with ECD, mainly the BRAFV600E activating mutation in 57% to

298 , 0.9 to 1.9) in patients presenting without ECD.

299 ive in patients with ECD as in those without ECD, and it should not be withheld in these complex pati

300 1, 11.53]), but not with a lower mean 3-year ECD (mean difference 61 (-479, 601) cells/mm(2), P = .77