ライフサイエンスコーパス: ERN

1 ERN activity was largest under conditions of high respon

2 ERN amplitude correlated with fMRI activation in both th

3 onflict-detection theory claims that ACC and ERN activity represent the detection of response conflic

4 negative (ERN) expression, rates for ERP and ERN diverged; that is, rates for ERP increased with adva

5 ied by ER expression, rates for both ERP and ERN flattened after 50 years of age.

6 f adaptive behavior differs between TRNs and ERNs.

7 inding was due to a significantly attenuated ERN amplitude in lesion patients compared with both sibl

8 s-regulation among two fuel-driven DNA-based ERNs regulated by a concatenated RNA transcription regul

9 moderator such that a more enhanced baseline ERN was associated with greater reduction in anxiety sym

10 Results indicated that baseline ERN was a significant treatment moderator such that a mo

11 ppears that the dynamics of coupling between ERN and post-error slowing between men and women is comp

12 A blunted ERN was associated with more severe negative symptoms an

13 e unaffected by pharmacogenetic factors, but ERN was decoupled from behavioral adaptation by SSRI adm

14 believe that concatenation of multiple CRNs/ERNs provides a basis for the design of more elaborate a

15 n chemical/enzymatic reaction networks (CRNs/ERNs) via cross-regulation to hierarchically control bio

16 nective Tissue and Musculoskeletal Diseases (ERN ReCONNET).

17 Network for Rare Endocrine Conditions (Endo-ERN), and provide recommendations for future research.

18 specially among individuals with an enhanced ERN.

19 determine the neural source of the enhanced ERN.

20 social interaction typically elicit enhanced ERNs.

21 l ITC (AITC), phenethyl ITC (PEITC), erucin (ERN), and sulforaphane (SFN), incorporated in water and

22 Sulforaphane(SFN) and erucin(ERN) are isothiocyanates (ITCs) bearing, respectively, m

23 Here, we show that monkeys exhibit ERN and Pe components when they commit errors during a s

24 Except for ERN, all three ITCs become more labile when the pH incre

25 reased with advancing age, whereas rates for ERN flattened after 50 years.

26 In controls, error trials generated greater ERN activity than correct trials.

27 eyes of 4 patients were identified as having ERN or ERNM.

28 However, ERN and error positivity were unaffected by pharmacogene

29 the broccoli extract, so a rapid decrease in ERN was observed while SFN experienced an increase befor

30 While individual differences in ERN magnitude have been implicated in a wide variety of

31 ents with OCD showed significantly increased ERN amplitudes.

32 Both the intracranial ERN (iERN) and error neuron responses appeared first in

33 to design and control sizable and intricate ERNs.

34 pants who avoided negative events had larger ERNs than those who were biased to learn more from posit

35 ct of conflict, positive learners had larger ERNs when having to choose among two good options (win/w

36 utational model further predicts that larger ERNs should be associated with better learning to avoid

37 r negativity or error-related negativity (Ne/ERN), a correlate of errors in choice tasks, is related

38 on errors, the error-related negativity (Ne/ERN), in a task in which two types of errors could occur

39 etection are responsible for the observed Ne/ERN amplitude reductions.

40 Our data revealed a pattern of Ne/ERN amplitudes that closely mirrored the amount of monet

41 at the error monitoring system scales the Ne/ERN according to the strength of error precursors to sel

42 ential error sources and then scaling the Ne/ERN according to the strength of the error precursor upo

43 ndent change in posterror slowing and the Ne/ERN amplitude, questioning a direct link between the amp

44 The Ne/ERN in turn predicted alpha asymmetry on the next trial

45 tractor on error trials and predicted the Ne/ERN on a single-trial level.

46 slowing and whether the amplitude of the Ne/ERN predicts posterror slowing in the current task setti

47 s, we found an amplitude reduction in the Ne/ERN, contradicting the existence of a direct relationshi

48 ested whether it predicts the size of the Ne/ERN.

49 ant correlations between the single-trial Ne/ERN amplitude and error-related reaction times.

50 The single-trial Ne/ERN distribution showed a reduced variance for middle-ag

51 rogen receptor-positive (ERP) and -negative (ERN) expression, rates for ERP and ERN diverged; that is

52 upling between the error-related negativity (ERN) and consecutive behavioural slowing.

53 ave focused on the error-related negativity (ERN) and error positivity (Pe) on error trials, as well

54 The error-related negativity (ERN) and error-related functional MRI (fMRI) activation

55 ng, indexed by the error-related negativity (ERN) and manifested by performance adaptations.

56 been quantified as error-related negativity (ERN) and may reflect abnormal neurophysiological mechani

57 The error-related negativity (ERN) and positivity (Pe) are components of event-related

58 processing, namely error-related negativity (ERN) and positivity.

59 ration of both the error-related negativity (ERN) and the novelty-related frontocentral N2.

60 as measured by the error-related negativity (ERN) in the event-related potential, is a reliable findi

61 The error-related negativity (ERN) is a well-established macroscopic scalp EEG correla

62 The error-related negativity (ERN) is an electrophysiological marker thought to reflec

63 e amplitude of the error-related negativity (ERN), a negative deflection in the electroencephalogram

64 in schizophrenia; error-related negativity (ERN), an electrophysiological index, for prediction of f

65 We recorded the error-related negativity (ERN), an event-related brain potential proposed to refle

66 ies found that the error-related negativity (ERN), an event-related potential (ERP) originating in th

67 oring, such as the Error-Related Negativity (ERN), are considerably influenced by situational factors

68 ted with a blunted error-related negativity (ERN), indicating a deficit in error monitoring.

69 as measured by the error-related negativity (ERN), is a transdiagnostic neurobiological marker of anx

70 c of an error, the error-related negativity (ERN).

71 The error-related negativity (ERN, or N(E)), an event-related brain potential, reflect

72 2, response-locked error-related negativity (ERN/Ne), and response-locked error positivity (Pe), meas

73 erroneous trials (error-related negativity--ERN) was diminished in patients with unilateral and bila

74 Neither ERN nor SFN inhibit the oxidation of bulk linolenic acid

75 e local resection of early rectal neoplasms (ERNs) remains debated, with limited data comparing trans

76 ng a redox-based enzymatic reaction network (ERN), enabling the dissipative disulfide formation for m

77 lies selected by European Reference Network (ERN) experts.

78 stabilize the epigenetic regulatory network (ERN) may converge into common phenotypes.

79 d a resilient epigenetic regulatory network (ERN) remains poorly understood.

80 lex features of enzymatic reaction networks (ERNs) play a key role in the emergence and sustenance of

81 ign of in vitro enzymatic reaction networks (ERNs) requires a detailed analysis of network kinetics a

82 simvastatin, plus extended-release niacin ([ERN], 1,500 to 2,000 mg/day), with ezetimibe added as ne

83 ription factor, ERF Required for Nodulation (ERN), which contains a highly conserved AP2 DNA binding

84 Patients with nonpedunculated ERNs >20 mm and <=cT1N0 were randomized to TAMIS or ESD.

85 However, an oxidation of ERN to SFN was observed in the broccoli extract, so a ra

86 and acid conditions promote the oxidation of ERN.

87 nsights into the chemical characteristics of ERNs while also delving into their potential application

88 noninferior to TAMIS for local resection of ERNs regarding 12-month recurrence.

89 o tested for effects of response conflict on ERN magnitude.

90 e and beyond other measures (i.e., N2, P300, ERN/Ne, age, sex, IQ, impulsivity, depression, anxiety,

91 : 1.21 [p = 0.017]) and the simvastatin plus ERN group (baseline HR: 1.25 [p = 0.001] and on-study HR

92 ed spike rate in L2/3 but not L5/6 predicted ERN magnitude.

93 adaptive networks with enzymatic regulation (ERNs) have been investigated in detail.

94 ore, we studied antioxidant behaviour of SFN/ERN at elevated temperature in two lipid systems.

95 Moreover, uncertain responses showed similar ERN activity as aware errors, in comparison with decreas

96 P2; error-monitoring, as indexed by smaller ERN/Ne; and adjusting response strategy posterror, as in

97 ough reduced compared with control subjects, ERN amplitude was greater in patients with higher neurot

98 Reference Network on Tumour Risk Syndromes (ERN GENTURIS).

99 However, there was no evidence that ERN reduced CV risk, despite favorable lipoprotein chang

100 ogether, these novel findings highlight that ERN may help guide treatment decisions regarding engagem

101 This theory predicts that ERN and ACC activity should increase directly with the d

102 We propose that ERN is a component of the Nod factor signal transduction

103 We show that ERN is necessary for Nod factor-induced gene expression

104 The ERN and the error positivity (Pe) were recorded from 33

105 The ERN can be customized to operate with aliphatic and arom

106 The ERN modestly increased 1-year apoA-1 (7%), decreased apo

107 The ERN occurs only when subjects are aware of making an err

108 The ERN was profoundly blunted in the patient group, regardl

109 The ERN, hemodynamic responses following errors, and intrain

110 ver the error-detection theory, although the ERN was not associated with posterror slowing, as predic

111 roves sufficiently predictive to control the ERN output after a single optimally designed experiment.

112 and quantitatively monitored by diluting the ERN output with nonlabeled standards of known concentrat

113 A flanker task was used to elicit the ERN at baseline and 12 weeks later, following either CBT

114 bility and score agreement were high for the ERN, CRN, and Pe.

115 findings suggest that the PCC generates the ERN and communicates with the dACC to subserve error pro

116 However, the ERN reflects the increase in phase-locked activities, pa

117 acity to alter individual differences in the ERN, providing evidence that the ERN is not entirely sta

118 confidence responses that might increase the ERN amplitude.

119 nd magnetoencephalography data localized the ERN to the posterior cingulate cortex (PCC).

120 Moreover, the ERN allows for programming hydrogel lifetimes by utilizi

121 Moreover, the ERN has been assessed in relation to a number of persona

122 Increased intraindividual correlation of the ERN and activity of the presupplementary motor area was

123 to be at odds with prominent theories of the ERN and aMCC.

124 rors, and intraindividual correlation of the ERN and blood oxygen level-dependent activity were compa

125 s demonstration of macaque homologues of the ERN and Pe forms a keystone in the bridge linking human

126 Additionally, increased correlation of the ERN and presupplementary motor area may indicate stronge

127 eveal a human single-neuron correlate of the ERN and suggest that dACC synthesizes error information

128 al limiting membrane disruption, area of the ERN in square millimeters, and central macular thickness

129 ar, the reinforcement learning theory of the ERN may need to be modified because it may not suffice a

130 cifically, the error-detection theory of the ERN states that the ERN reflects ACC processing that is

131 ow a linear increase in the amplitude of the ERN with increasingly late responses.

132 to date has assessed the reliability of the ERN, Pe, and CRN.

133 oring and confirm one cortical source of the ERN.

134 cortex (aMCC), the neuronal generator of the ERN.

135 he dACC as the hemodynamic reflection of the ERN.

136 s of response similarity and conflict on the ERN, using a task that involved hand and foot movements.

137 etition and interpersonal cooperation on the ERN.

138 es about the neural system that produces the ERN - one based on principles of reinforcement learning

139 uals with schizophrenia, indicating that the ERN and Pe are differentially related to psychotic illne

140 However, recent studies demonstrate that the ERN decreases after unaware errors.

141 Results also revealed that the ERN increased pre- to post-treatment among patients rand

142 Together these findings suggest that the ERN is activated by aware motor errors as well as sensor

143 We hypothesized that the ERN is dependent upon awareness, and predicted that prev

144 nces in the ERN, providing evidence that the ERN is not entirely static in patients with anxiety diso

145 Furthermore, we found that the ERN predicted consecutive behavioural slowing within sub

146 These results demonstrate that the ERN predicts the degree to which participants are biased

147 -detection theory of the ERN states that the ERN reflects ACC processing that is directly related to

148 We conclude that the ERN reflects medial frontal activity involved in the det

149 Thus, the ERN motif and residue Asp-142 in the loop 2 are of criti

150 nderstanding of the mechanism underlying the ERN.

151 To date, little is known about whether the ERN can inform the choice between first-line anxiety dis

152 anxiety disorder treatments and whether the ERN changes following treatment completion.

153 he study was to therefore assess whether the ERN is a treatment moderator and index of symptom change

154 eal homeostatic mechanisms through which the ERN sustains somatic cell fitness and uncover how the ne

155 layer of functional compensation within the ERN, with intra- or inter-class interactions buffering t

156 13 referral centers belonging to the ToxiTEN ERN-skin subgroup was conducted.

157 al prefrontal damage, however, correct-trial ERN activity was equal to error-trial ERN activity.

158 -trial ERN activity was equal to error-trial ERN activity.

159 ment, leading to a communication between two ERNs.