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1 equence analysis of a 919-bp sequence of the ehrlichial 16S rRNA gene amplified by PCR from the blood
2                                 Granulocytic ehrlichial 16S rRNA gene DNAs from six dogs were amplifi
3  the majority of polyreactive IgM recognized ehrlichial Ag(s), including an immunodominant outer memb
4                                           An ehrlichial agent (Venezuelan dog Ehrlichia [VDE]) was is
5 olation and antigenic characterization of an ehrlichial agent from a dog in South America, as well as
6                                          The ehrlichial agent was isolated from the blood of experime
7 ood rats serve as reservoirs of granulocytic ehrlichial agents in certain areas of northern Californi
8  United States and tested against a panel of ehrlichial and rickettsial pathogens.
9                                              Ehrlichial antibodies were measured by IFA using Ehrlich
10 ell responses, (iii) enhanced secondary anti-ehrlichial antibody response, (iv) accelerated bacterial
11 ehrlichial burden, and cytokine responses to ehrlichial antigen in primary splenic cultures during th
12 is presently infected or has been exposed to ehrlichial antigens in the past.
13 hree dogs infected with VDE by using several ehrlichial antigens revealed that the antigenic profile
14 ibed because of similarity of titers to both ehrlichial antigens.
15 nd protective mechanisms involve blocking of ehrlichial attachment or complement and Fcgamma-receptor
16 ny of these three cytokines, indicating that ehrlichial binding is required for IL-1beta mRNA express
17                  These results indicate that ehrlichial binding to host cells is sufficient to inhibi
18  the infection, yet had significantly higher ehrlichial burden both in the blood and tissues than C57
19 sayed for hepatic histopathological lesions, ehrlichial burden, and cytokine responses to ehrlichial
20 e by regulating liver injury and controlling ehrlichial burden.
21                                              Ehrlichial cell division was initiated soon after, resul
22 protein, OMP-19, protects mice against fatal ehrlichial challenge infection, and we identified a CD4
23 d and SLP mice were protected against lethal ehrlichial challenge infection.
24 oduce only IgM, were protected against fatal ehrlichial challenge infection.
25 lls, contributed to protection against fatal ehrlichial challenge.
26 ition was HGE agent dose dependent, required ehrlichial contact with the host cells, and was reversib
27 ear accumulation of NF-kappaB and TRIM21 and ehrlichial destruction.
28 during a febrile condition characteristic of ehrlichial disease.
29               Attempts to amplify and detect ehrlichial DNA from the limited tissues available (n = 4
30                        PCR amplifications of ehrlichial DNA targeting the 16S rRNA gene, the variable
31                                              Ehrlichial DNA was identified in 17 (68%) of the 25 sero
32 ing a number of host cell processes, but the ehrlichial effector proteins involved are unknown.
33 that these reagents did not directly inhibit ehrlichial energy metabolism.
34 his is in contrast to the MSP2 homologues in ehrlichial genogroup I pathogens, Ehrlichia chaffeensis,
35 mplexity requires efficient use of the small ehrlichial genome.
36                        Consistent with other ehrlichial glycoproteins, the gp19 protein exhibited a l
37 morula membrane is the interface between the ehrlichial growing environment and the host cytoplasm, M
38 anscription-PCR analysis, the mRNA levels of ehrlichial HSP70 and HSP60 were examined after temperatu
39                                          The ehrlichial HSP70 gene encoded a 637-amino-acid protein,
40 of E. coli dnaK was observed upstream of the ehrlichial HSP70 gene.
41 s promote immunopathology and defective anti-ehrlichial immunity, possibly via decreasing the protect
42                    Similar to other reported ehrlichial immunoreactive glycoproteins, carbohydrate wa
43                   These results suggest that ehrlichial inclusions are early endosomes which selectiv
44         After 6 h postinfection, 100% of the ehrlichial inclusions became TfR positive and the intens
45 nt Dishevelled induced lysosomal fusion with ehrlichial inclusions corresponding to p62 degradation a
46                                              Ehrlichial inclusions did not fuse with lysosomes, since
47 t colocalization of predominately SUMO1 with ehrlichial inclusions was observed.
48                                         Some ehrlichial inclusions were labeled positive with antibod
49 ed that up to 3 h postinfection, most of the ehrlichial inclusions were negative for TfRs.
50                                          The ehrlichial inclusions were slightly acidic, since they w
51 roteins and PLC-gamma2 were colocalized with ehrlichial inclusions, as determined by double-immunoflu
52 ocalization of tyrosine phosphoproteins with ehrlichial inclusions.
53 n TRP120-interacting host protein, PCGF5, to ehrlichial inclusions.
54 etes established CD11c(+)T-bet(+) B cells in ehrlichial-infected mice, in a B cell-intrinsic manner.
55                                              Ehrlichial infection also inhibited spleen IgG responses
56 nstrating that DCs play an important role in ehrlichial infection and immunity.
57 is inoculum size can influence the course of ehrlichial infection and should be regarded as important
58 fection (peritoneum) following lethal murine ehrlichial infection compared to innocuous ehrlichial in
59          The study indicated that persistent ehrlichial infection contributes to heterologous protect
60      In conclusion, protection against fatal ehrlichial infection correlates with strong expansion of
61                              To test whether ehrlichial infection disrupts Janus kinase (Jak) and sig
62                                              Ehrlichial infection in dogs was defined as concurrence
63                                       Lethal ehrlichial infection in wild-type mice induced a decline
64  primary function of T-bet in B cells during ehrlichial infection is to promote appropriate class swi
65 In this study, signaling events required for ehrlichial infection of human monocytic cell line THP-1
66 rabbits there, but the identity of the other ehrlichial infection remains undescribed.
67   CD11c(+) T-bet(+) B cells generated during ehrlichial infection require CD4(+) T cell help and IL-2
68 p a tick vector transmission animal model of ehrlichial infection using a human pathogen, Ehrlichia m
69 ted in the reduction of TRP120-Ub, decreased ehrlichial infection, and reduced recruitment of a known
70 small molecules also significantly decreased ehrlichial infection, indicating that the Ub pathway is
71                    In dogs that developed an ehrlichial infection, thrombocytopenia occurred by 28 da
72 ted with the intermediate doses developed an ehrlichial infection.
73 he two highest dose of E. canis developed an ehrlichial infection.
74 locks Wnt secretion, significantly decreased ehrlichial infection.
75 e ehrlichial infection compared to innocuous ehrlichial infection.
76  antibodies can protect SCID mice from fatal ehrlichial infection.
77  homologous and heterologous immunity during ehrlichial infection.
78  an animal model for the study of persistent ehrlichial infection.
79 leotide in THP-1 cells significantly blocked ehrlichial infection.
80 m immunopathological mechanisms initiated by ehrlichial infection.
81 standing of the mechanisms of persistence of ehrlichial infections in humans, domestic animals, and r
82 se of infection than previously reported for ehrlichial infections induced by intravenous inoculation
83                             We conclude that ehrlichial infections may be as common as spotted fever
84 cent) were obtained from patients with fatal ehrlichial infections.
85 les from patients with other rickettsial and ehrlichial infections.
86 10(5) bacterial genomes/mouse, respectively) ehrlichial inocula.
87 hosphorylation is specific and essential for ehrlichial internalization, replication, and spreading i
88 dicating that the Ub pathway is critical for ehrlichial intracellular replication and survival.
89 rlichia spp. suggests that genes involved in ehrlichial iron acquisition have been subject to reducti
90 ong otherwise indistinguishable granulocytic ehrlichial isolates.
91 ng, and the distribution and quantity of the ehrlichial load by immunohistochemistry, polymerase chai
92  time, hematological changes, PCR detection, ehrlichial load, seroconversion time, and titer range we
93 s, resulted in significant reductions in the ehrlichial load.
94 ion of rickettsemia, and initial and maximal ehrlichial loads between the two routes of infection.
95                                  The highest ehrlichial loads were present between days 4 and 14 afte
96 logical grading in these organs and relative ehrlichial loads.
97                   A gene encoding a 23.5-kDa ehrlichial morula membrane protein designated MmpA was c
98                         LC3 colocalized with ehrlichial morulae on days 1, 2, and 3 postinfection, an
99                                              Ehrlichial myocarditis is an unusual finding in nonimmun
100 arge repertoire of the p28 genes in a single ehrlichial organism and antigenic diversity of the P28 a
101                             Strain HF565, an ehrlichial organism closely related to E. chaffeensis is
102      Identical 16S rRNA gene sequences among ehrlichial organisms from dogs, ticks, and a human in th
103 ernalization and continuous proliferation of ehrlichial organisms or the production of ehrlichial pro
104 rne pathogen, one of several closely related ehrlichial organisms that cause disease in animals and h
105      We have shown that immunization with an ehrlichial outer membrane protein, OMP-19, protects mice
106 sis of these interactions and their roles in ehrlichial pathobiology are not well defined.
107 ctor alpha, Stat1, and CD48) associated with ehrlichial pathobiology were strongly upregulated during
108 n 2 (MSP2) and MSP3 of the persistent bovine ehrlichial pathogen Anaplasma marginale are immunodomina
109              Protective immunity against the ehrlichial pathogen Anaplasma marginale has been hypothe
110 Native major surface protein 1 (MSP1) of the ehrlichial pathogen Anaplasma marginale induces protecti
111                    Anaplasma marginale is an ehrlichial pathogen of cattle that establishes lifelong
112    Anaplasma marginale, an intraerythrocytic ehrlichial pathogen of cattle, establishes persistent in
113                    Anaplasma marginale is an ehrlichial pathogen of cattle, in the order Rickettsiale
114                             Isolation of the ehrlichial pathogen was achieved by inoculating patient
115 cture of Anaplasma marginale, a genogroup II ehrlichial pathogen, in both an acute outbreak and in pe
116 o valid small animal model that uses a human ehrlichial pathogen.
117       Although specific associations between ehrlichial pathogenicity and genotype were not identifie
118 port the use of IL-12 coadsorbed with OMP of ehrlichial pathogens in alum to amplify both antibody an
119                  Protective immunity against ehrlichial pathogens is believed to require induction of
120                                   Tick-borne ehrlichial pathogens of animals and humans require a mam
121 ocytes participate in protective immunity to ehrlichial pathogens through production of gamma interfe
122  of TRP activation of Wnt pathways to induce ehrlichial phagocytosis.
123 ocytes, which have a life span too short for ehrlichial proliferation.
124 ntified a 1884-bp open reading frame with an ehrlichial promoter.
125 re required for antiapoptotic signaling, but ehrlichial protein synthesis and/or proliferation is not
126 of ehrlichial organisms or the production of ehrlichial proteins are required for the up-regulation o
127 ar and antigenic characterization of a human ehrlichial species in South America.
128                      Recent data for several ehrlichial species show differential expression of the O
129 nant outer membrane proteins (OMPs) in other ehrlichial species.
130 e effector proteins involved in facilitating ehrlichial survival in mononuclear phagocytes.
131 onents of PI3K-GSK3-mTOR signaling decreased ehrlichial survival.
132 pathway, and activation was induced by three ehrlichial tandem repeat protein (TRP) effectors, with T
133                        Consistent with other ehrlichial TRPs, the TRPs identified in ehrlichial whole
134                                              Ehrlichial vacuoles did not colocalize with the lysosoma
135 ng early infection, autophagosomes fuse with ehrlichial vacuoles to form an amphisome indicated by th
136 ther ehrlichial TRPs, the TRPs identified in ehrlichial whole-cell lysates and the recombinant protei

 
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