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1 er inoculation of their endophytic bacteria (Enterobacter cloacae).
2 sistance (AMR) are Klebsiella pneumoniae and Enterobacter cloacae.
3  deployed by the type VI secretion system of Enterobacter cloacae.
4 hich is identical to plasmid pNDM-HF727 from Enterobacter cloacae.
5 gle D(H) gene segment (D-limited mice), with Enterobacter cloacae.
6 onse to alpha-1,3 dextran (DEX) expressed on Enterobacter cloacae.
7 ence analysis revealed that the organism was Enterobacter cloacae.
8  antibiotic for treatment of infections with Enterobacter cloacae.
9 robacter koseri but 97.0% similar to that of Enterobacter cloacae.
10 d by blood feeding, restored the immunity to Enterobacter cloacae.
11 illus, which most laboratories recognized as Enterobacter cloacae.
12 escens after injection of prepupae with live Enterobacter cloacae.
13  unique collective behaviors of the bacteria Enterobacter cloacae.
14 ia coli, 0.25/1; Klebsiella pneumoniae, 2/4; Enterobacter cloacae 1/4; and Stenotrophomonas maltophil
15 i (14.3%), Klebsiella pneumoniae (10.9%) and Enterobacter cloacae (16.3%) were the main MDRE species
16              Pseudomonas aeruginosa (27.5%), Enterobacter cloacae (16.8%), and Enterobacter asburiae
17 m patients were Pseudomonas aeruginosa (22), Enterobacter cloacae (21), Acinetobacter spp. (13), Ente
18 including Escherichia coli (14 patients) and Enterobacter cloacae (7 patients).
19 coli (18.8%), Klebsiella pneumoniae (14.2%), Enterobacter cloacae (9.1%), Acinetobacter spp. (6.2%),
20 hogens with interpretive criteria, excluding Enterobacter cloacae (98.3% S) and E. faecalis (86.0% S)
21 inst Escherichia coli, Klebsiella pneumonia, Enterobacter cloacae, Acinetobacter baumannii, and methi
22  a transmission cluster of blaKPC-2-positive Enterobacter cloacae among patients treated in a highly
23 linically important enzymes CTX-M-15, KPC-2, Enterobacter cloacae AmpC, Pseudomonas aeruginosa AmpC,
24 usly determined for the class C enzymes from Enterobacter cloacae and Citrobacter freundii.
25 rial cell wall biosynthetic enzyme MurA from Enterobacter cloacae and Escherichia coli in vitro.
26               A case of ventriculitis due to Enterobacter cloacae and Pseudomonas fulva following pla
27 microbial bacteremia, and seven of these had Enterobacter cloacae and S. marcescens in the same cultu
28 stant (IC(50), approximately 10,000 nM), and Enterobacter cloacae and Serratia marcescens were highly
29 bacteria (that is, Klebsiella pneumoniae and Enterobacter cloacae) and their corresponding antimicrob
30 luding Escherichia coli, Citrobacter koseri, Enterobacter cloacae, and clinical isolates of non-typho
31  348 Klebsiella pneumoniae, one (<1%) of 890 Enterobacter cloacae, and one (1%) of 162 Enterobacter a
32 d -negative strains), Klebsiella pneumoniae, Enterobacter cloacae, and Pseudomonas aeruginosa.
33 a: Escherichia coli, Pseudomonas aeruginosa, Enterobacter cloacae, and Yersinia enterocolitica.
34 erved, while ETEST FO should not be used for Enterobacter cloacae, because of low EA and a high VME r
35 L49 antibodies were chemically conjugated to Enterobacter cloacae beta-lactamase (bL), and their abil
36                            Here, we discover Enterobacter cloacae CD-NTase-associated protein 4 (Cap4
37 d of clinically significant AmpC production (Enterobacter cloacae, Citrobacter freundii, and Klebsiel
38 treptococcus pneumoniae (10/11 [90.9%]), and Enterobacter cloacae complex (2/4 [50%]).
39            The 3 most prevalent species were Enterobacter cloacae complex (42%), Klebsiella pneumonia
40 ively enrolled patients with K. aerogenes or Enterobacter cloacae complex (Ecc) BSI from 2002 to 2015
41                                          The Enterobacter cloacae complex (ECC) consists of closely r
42 sis (n = 117), Escherichia coli (n = 75) and Enterobacter cloacae complex (n = 57) also detected.
43 nd the increasing clinical importance of the Enterobacter cloacae complex have often been discussed.
44 cherichia coli, 55 Klebsiella pneumoniae, 21 Enterobacter cloacae complex, 18 Serratia marcescens, 12
45 nt Escherichia coli, Enterobacter aerogenes, Enterobacter cloacae complex, Klebsiella pneumoniae, or
46 iae, n = 44; Pseudomonas aeruginosa, n = 17; Enterobacter cloacae complex, n = 9; and Acinetobacter b
47  baumannii, E. coli, Serratia marcescens and Enterobacter cloacae complex.
48 m Staphylococcus aureus, and aac(3)-VIa from Enterobacter cloacae (conferring resistance to kanamycin
49 s following induction of the SOS response in Enterobacter cloacae decreased the amount of DNA measura
50 oci from isolates of Serratia marcescens and Enterobacter cloacae, demonstrating the presence of in-f
51  Escherichia coli, Klebsiella pneumoniae and Enterobacter cloacae/E. cloacae complex, the most common
52  type VI secretion system (T6SS) activity in Enterobacter cloacae (ECL).
53 tation of an alternative nitroreductase from Enterobacter cloacae enables the tailoring of a photoenz
54 cytosis of 11 clinical isolates representing Enterobacter cloacae, Enterobacter bugandensis, Enteroba
55 y Citrobacter freundii, Clostridium species, Enterobacter cloacae, Enterococcus faecalis, Klebsiella
56 fied in the Enterobacteria Escherichia coli, Enterobacter cloacae, Erwinia herbicola, and Salmonella
57 ing probes for pathogenic bacteria including Enterobacter cloacae, Escherichia coli J96, Pseudomonas
58  isolates of important Gram-negative species-Enterobacter cloacae, Escherichia coli, Klebsiella pneum
59               As in other bacterial species, Enterobacter cloacae form macroscopic aggregates.
60                                          The Enterobacter cloacae GC1 enzyme is an example of a class
61        The crystallographic structure of the Enterobacter cloacae GC1 extended-spectrum class C beta-
62  structure of the nitroreductase enzyme from Enterobacter cloacae has been determined for the oxidize
63                                              Enterobacter cloacae has been implicated as one of the c
64         Recently, enteric commensal bacteria Enterobacter cloacae has been recognized as a helper in
65 rganisms, intraocular infection secondary to Enterobacter cloacae infection is a devastating disease
66 ch has best been described in the context of Enterobacter cloacae infections.
67                                              Enterobacter cloacae is a clinically important Gram-nega
68                                              Enterobacter cloacae is a Gram-negative nosocomial human
69                               We describe an Enterobacter cloacae isolate harbouring a minor subpopul
70 present in K. oxytoca, Escherichia coli, and Enterobacter cloacae isolates from unlinked patients wit
71                                              Enterobacter cloacae isolates were resistant to all firs
72 iae isolates, 3 Escherichia coli isolates, 5 Enterobacter cloacae isolates, 2 S. marcescens isolates,
73                      This protein was termed Enterobacter cloacae lectin A (EclA) and found to bind l
74  the x-ray structures of the D305A mutant of Enterobacter cloacae MurA and the D313A mutant of Escher
75 tured and depicted the Cys-115-PEP adduct of Enterobacter cloacae MurA in various reaction states by
76 ablished that Cys115 of Escherichia coli and Enterobacter cloacae MurA is the active site nucleophile
77 s been determined to be 8.3, by titration of Enterobacter cloacae MurA with the alkylating agent iodo
78 d the x-ray structure of the C115S mutant of Enterobacter cloacae MurA, which was crystallized in the
79 (n = 1,127), Escherichia coli (n = 149), and Enterobacter cloacae (n = 110).
80 moniae (n = 236), Escherichia coli (n = 22), Enterobacter cloacae (n = 23), Klebsiella oxytoca (n = 8
81 ther Staphylococcus epidermidis (n = 100) or Enterobacter cloacae (n = 60).
82 1.9%), Escherichia coli (n = 129, 30.0%) and Enterobacter cloacae (n = 62, 14.4%) were the main Enter
83 ies of the flavin mononucleotide cofactor of Enterobacter cloacae nitroreductase (NR), determined und
84   The oxygen-insensitive nitroreductase from Enterobacter cloacae (NR) catalyzes two-electron reducti
85                                 A library of Enterobacter cloacae P99 beta-lactamase mutants was prod
86 ed reaction, cephalosporin hydrolysis by the Enterobacter cloacae P99 cephalosporinase (beta-lactam h
87 dase of Streptomyces sp. R61, a PBP, and the Enterobacter cloacae P99 cephalosporinase, a class C bet
88  inhibition of the class C beta-lactamase of Enterobacter cloacae P99 determined.
89 e) inactivates the class C beta-lactamase of Enterobacter cloacae P99 in a covalent fashion.
90  to react with the class C beta-lactamase of Enterobacter cloacae P99 in two ways, by acylation and b
91 stants for hydrolysis by beta-lactamase from Enterobacter cloacae P99 indicated kcat values of 476 +/
92                The class C beta-lactamase of Enterobacter cloacae P99 is closely similar in structure
93                The class C beta-lactamase of Enterobacter cloacae P99 is competitively inhibited by l
94         The class C serine beta-lactamase of Enterobacter cloacae P99 is irreversibly inhibited by O-
95 cts on V/K for the class C beta-lactamase of Enterobacter cloacae P99 suggest an acyl-transfer transi
96                The class C beta-lactamase of Enterobacter cloacae P99 was employed.
97 hibited typical class A (TEM-2) and class C (Enterobacter cloacae P99) beta-lactamases in a time-depe
98 , catalyzed by the class C beta-lactamase of Enterobacter cloacae P99, have been studied in order to
99 -2 antibody detected Escherichia coli CMY-2, Enterobacter cloacae P99, Klebsiella pneumoniae ACT-1, a
100 nactivators of the class C beta-lactamase of Enterobacter cloacae P99.
101 by class C beta-lactamases such as that from Enterobacter cloacae P99.
102 omyces R61 and the class C beta-lactamase of Enterobacter cloacae P99.
103 rally very similar class C beta-lactamase of Enterobacter cloacae P99.
104 on with K. pneumoniae, Proteus mirabilis, or Enterobacter cloacae promoted greater recruitment of neu
105 several gram-negative bacteria, specifically Enterobacter cloacae, Pseudomonas aeruginosa, and Pantoe
106 ed with the P solubilizing bacterial strains Enterobacter cloacae, Pseudomonas pseudoalcaligenes, and
107 lysaccharides (e.g., Helicobacter pylori and Enterobacter cloacae), revealing it to be a promising pr
108 LY]) and Enterobacter lignolyticus (formerly Enterobacter cloacae) SCF1 (enterolysin [ELY]).
109 , invasive aspergillosis (20%, 3 of 15), and Enterobacter cloacae, Serratia marcescens, Pneumocystis
110 mis, E. coli O157:H7, Klebsiella pneumoniae, Enterobacter cloacae, Shigella dysenteriae, Salmonella e
111 ia (including only Klebsiella pneumoniae and Enterobacter cloacae) showed the following performance:
112 neage - accumulates LDs when challenged with Enterobacter cloacae, Sindbis, and Dengue viruses.
113     The membrane-bound selenate reductase of Enterobacter cloacae SLD1a-1 is purified in low yield an
114 erial species such as Klebsiella pneumoniae, Enterobacter cloacae, Stenotrophomonas maltophilia, and
115                                              Enterobacter cloacae strain G6809 with reduced susceptib
116  C beta-lactamase from a clinical isolate of Enterobacter cloacae strain GC1 with improved hydrolytic
117 fication of a Shiga toxin 1 (Stx1)-producing Enterobacter cloacae strain, M12X01451, from a human cli
118 nterobacter aerogenes and 13,954 isolates of Enterobacter cloacae tested using a Vitek system; for th
119 ing the melibiose-H(+) symporter (MelY) from Enterobacter cloacae that had enhanced fermentation on 1
120 t on four cases of endophthalmitis caused by Enterobacter cloacae: two in patients with acute postope
121 rates higher than the totals were noted with Enterobacter cloacae versus ampicillin-sulbactam, aztreo
122 e given 1 x 10(6) colony-forming units/mL of Enterobacter cloacae with the third feeding.
123 roducing bacteria (Klebsiella pneumoniae and Enterobacter cloacae) with a detection limit of 10 3 bac
124 infections caused by carbapenemase-producing Enterobacter cloacae within 21 days of cefiderocol thera

 
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