ライフサイエンスコーパス: Enterococcus

1 r flagella), and a motile Streptococcus (now Enterococcus).

2 aphylococcus aureus and vancomycin-resistant Enterococcus.

3 s aureus infections and vancomycin-resistant enterococcus.

4 Prevotella, and reduction of Escherichia and Enterococcus.

5 This isothermal assay targets the same Enterococcus 23S rRNA gene region as the existing quanti

6 iotic for patients with vancomycin-resistant Enterococcus (4.2 versus 43.7 h; P=0.006) and viridans g

7 01), enriched in Bacteroides and depleted in Enterococcus, Acinetobacter, Pseudomonas, and Hydrogenop

8 li, Klebsiella spp., Pseudomonas aeruginosa, Enterococcus aerogenes, Proteus vulgaris and Enterobacte

9 We found that Enterococcus also expands in the mouse gastrointestinal

10 rculating seawater carries fecal indicators, Enterococcus and bird-associated Catellicoccus, through

11 es C1q, CL-11, and murine ficolin-A bind the enterococcus and drive the CP and the LP in human and mo

12 11 genera and with increased proportions of Enterococcus and Enterobacteriaceae.

13 uid cultures were preliminarily positive for enterococcus and gram-negative rods.

14 bundances of Streptococcus, Escherichia, and Enterococcus and increased primary bile acids.

15 nt of Bifidobacterium spp., and reduction of Enterococcus and Klebsiella spp. in vaginally delivered

16 discriminated E. faecalis from 20 different Enterococcus and non-Enterococcus spp.

17 as associated with a significant increase of Enterococcus and Peptostreptococcus sp and a reduction o

18 wo gram positive (Staphyllococcus aureus and Enterococcus) and two gram negative pathogens (E. coli a

19 hemolytic streptococci, vancomycin-resistant Enterococcus, and Enterobacteriaceae.

20 fidobacterium and depleted of Lactobacillus, Enterococcus, and Lachnospira.

21 cluding genera Lactobacillus, Streptococcus, Enterococcus, and Listeria.

22 fections (CAUTI) caused by Escherichia coli, Enterococcus, and Staphylococcus aureus we observed that

23 y facultative anaerobic genera (Escherichia, Enterococcus, and Streptococcus), with multiple ciproflo

24 ow more accurate microbial identification of Enterococcus avium from metagenomic samples with FDA-ARG

25 of patients, with Staphylococcus aureus and enterococcus bacteremia associated with worse outcomes.

26 terococcus is an independent risk factor for Enterococcus bacteriuria.

27 Vancomycin-resistant Enterococcus bloodstream infections (VRE-BSIs) are assoc

28 Vancomycin-resistant Enterococcus bloodstream infections (VRE-BSIs) are becom

29 bility to C. difficile, vancomycin-resistant Enterococcus, carbapenem-resistant Klebsiella pneumoniae

30 of environmentally adapted species including Enterococcus casseliflavus.

31 d-type (ATCC 29212) and vancomycin-resistant Enterococcus cells were incubated at five different vanc

32 ary outcome was MRSA or vancomycin-resistant enterococcus clinical cultures attributed to participati

33 id was enhanced by anti-CRISPRs derived from Enterococcus conjugative elements, highlighting a role f

34 lus and Clostridiales) and increased risk of Enterococcus domination (odds ratio [OR], 5.50; 95% conf

35 owed compromised clearance of postantibiotic Enterococcus domination.

36 Escherichia coli (E. coli) and Gram-positive Enterococcus durans (E. durans) and Staphylococcus epide

37 ted with the hospital environment (including Enterococcus, Enterobacter and Klebsiella species), in b

38 epeat therapy was a significant predictor of Enterococcus expansion (P = 0.006), independently of ant

39 In the present study, an Enterococcus faecalis (E. faecalis) DNA biosensor (ef-bi

40 Enterococcus faecalis (E. faecalis) is one of the indica

41 hat Streptococcus gordonii (S. gordonii) and Enterococcus faecalis (E. faecalis) were frequent isolat

42 This approach is demonstrated on Enterococcus faecalis (E. faecalis), which served as tar

43 inosa (Pa), Legionella pneumophila (Lp), and Enterococcus faecalis (Ef) by using anti-infective, anti

44 applied single-molecule FRET methods to the Enterococcus faecalis (Efa) Cas1-Cas2 system to establis

45 ng a collagenolytic rodent-derived strain of Enterococcus faecalis (strain E2), and on the second day

46 cus anginosus group (</=0.06 microg/mL), and Enterococcus faecalis (vancomycin susceptible, </=0.25 m

47 occus aureus (MRSA) and vancomycin-resistant Enterococcus faecalis (VRE), and another undergoes spont

48 Vancomycin-resistant Enterococcus faecalis (VREfs) is an important nosocomial

49 Vancomycin-resistant Enterococcus faecalis (VREfs) is an important nosocomial

50 n this study, we investigated the ability of Enterococcus faecalis 2/28, isolated from artisan cheese

51 lococcus aureus, Pseudomonas aeruginosa, and Enterococcus faecalis although PSO had an antimicrobial

52 three most common species, Escherichia coli, Enterococcus faecalis and Bacteroides vulgatus, did not

53 acterial biofilm and faecal samples included Enterococcus faecalis and Enterobacter hormaechei.

54 f clinical strains of Staphylococcus aureus, Enterococcus faecalis and Enterococcus faecium.

55 greater potency against vancomycin resistant Enterococcus faecalis and methicillin-resistant Staphylo

56 For Enterococcus faecalis and other enterococcal species, re

57 -positive bacteria Staphylococcus aureus and Enterococcus faecalis and two Gram-negative bacteria Esc

58 -negative Escherichia coli and Gram-positive Enterococcus faecalis applied during the first 7 months

59 combinations was assessed by application on Enterococcus faecalis as a model organism for Gram-posit

60 sk only), S. aureus ATCC 29213 (broth only), Enterococcus faecalis ATCC 29212 (broth only), Streptoco

61 ccus aureus ATCC 29213, 0.25 to 2 mug/ml for Enterococcus faecalis ATCC 29212, 1 to 4 mug/ml for Esch

62 4028, Staphylococcus epidermidis ATCC 12228, Enterococcus faecalis ATCC 29212, and Escherichia coli D

63 of synergistic gentamicin for uncomplicated Enterococcus faecalis bacteremia in children.

64 ective endocarditis (IE) among patients with Enterococcus faecalis bacteremia.

65 tural snapshots from the type II-A system of Enterococcus faecalis Cas1 and Cas2 during spacer integr

66 Enterococcus faecalis Cas1-Cas2 selectively binds to a s

67 Upon sensing of the peptide pheromone cCF10, Enterococcus faecalis cells carrying pCF10 produce three

68 sis revealed that hsdS allelic variations in Enterococcus faecalis exert significant impact on gene e

69 es to PG composition in vancomycin-resistant Enterococcus faecalis following the growth in presence o

70 -positive bacteria Staphylococcus aureus and Enterococcus faecalis have lost either all or most polya

71 positive organisms Staphylococcus aureus and Enterococcus faecalis in comparison with known analogues

72 lar function in Streptococcus agalactiae and Enterococcus faecalis In conclusion, the elucidation of

73 Enterococcus faecalis is a commensal and pathogen of hum

74 Enterococcus faecalis is a commensal bacterium of the hu

75 Enterococcus faecalis is a Gram-positive commensal bacte

76 Enterococcus faecalis is a gram-positive organism respon

77 Enterococcus faecalis is a human intestinal pathobiont w

78 Since Enterococcus faecalis is a natural heme auxotroph and ca

79 e (SAS) RelQ from the Gram-positive pathogen Enterococcus faecalis is a sequence-specific RNA-binding

80 The Gram-positive bacterium Enterococcus faecalis is both a colonizer of the gastroi

81 Enterococcus faecalis is both a common commensal of the

82 Enterococcus faecalis is considered to be the most impor

83 sphate-dependent tyrosine decarboxylase from Enterococcus faecalis is followed by transformation of d

84 Enterococcus faecalis is frequently associated with poly

85 The Gram-positive opportunistic pathogen Enterococcus faecalis is frequently responsible for noso

86 Enterococcus faecalis is one of the most frequently isol

87 Expansion of intestinal Enterococcus faecalis is sufficient to exacerbate ethano

88 Enterococcus faecalis is the third most frequent cause o

89 e visualization of conformational changes in Enterococcus faecalis MDD that describe sequential steps

90 lope homeostasis), from daptomycin-resistant Enterococcus faecalis not only reversed resistance to 2

91 l-prolyl cis-trans isomerase, as well as the Enterococcus faecalis polysaccharide diheteroglycan, are

92 Multidrug-resistant Enterococcus faecalis possess numerous mobile elements t

93 d the GIT microbiota of MAT mothers, whereas Enterococcus faecalis predominated within the MAT infant

94 EfbA is a PavA-like fibronectin adhesin of Enterococcus faecalis previously shown to be important i

95 Enterococcus faecalis PrgA, encoded by the conjugative p

96 ost & Microbe, Keogh et al. (2016) show that Enterococcus faecalis promotes Escherichia coli biofilm

97 uconostoc mesenteroides, Bacillus cereus and Enterococcus faecalis proving its antimicrobial action.

98 occi, as homologs from Bacillus subtilis and Enterococcus faecalis retain this ability.

99 R (D191N) first identified from the pathogen Enterococcus faecalis S613.

100 The Enterococcus faecalis strain, which was not inhibited, p

101 Enterococcus faecalis strains are resident intestinal ba

102 of five E. faecium strains but none of five Enterococcus faecalis strains consistently developed res

103 ve Escherichia coli Symbio and Gram-positive Enterococcus faecalis Symbio or placebo from week 5 unti

104 ) from the opportunistic nosocomial pathogen Enterococcus faecalis synthesizes a specific lysoform li

105 rk, we identify LiaR-independent pathways in Enterococcus faecalis that regulate cell membrane adapta

106 y for growth of the human bacterial pathogen Enterococcus faecalis The final enzyme in this pathway,

107 ic activity of SrtA is key to the ability of Enterococcus faecalis to bind mucin (a major component o

108 usly, our research demonstrated that dietary Enterococcus Faecalis UC-100 substituting antibiotics en

109 ons were tested against Escherichia coli and Enterococcus faecalis urinary tract infection isolates.

110 l and pathogenic host-microbe interaction of Enterococcus faecalis was explored using a Caenorhabditi

111 Under sterile conditions, 1 muL Enterococcus faecalis was inoculated inside the implants

112 omonas gingivalis and the endodontic species Enterococcus faecalis were grown to early log phase and

113 However, the AcpAs of Lactococcus lactis and Enterococcus faecalis were inactive.

114 n-a two-subunit exotoxin that is secreted by Enterococcus faecalis(5,6)-as a cause of hepatocyte deat

115 g a microbe with host-protective properties (Enterococcus faecalis) and a pathogen (Staphylococcus au

116 both Gram positive ( Staphylococcus aureus, Enterococcus faecalis) and Gram negative bacteria (e.g.,

117 A protocol was developed for Gram-positive (Enterococcus faecalis) and Gram-negative (Escherichia co

118 Expression of ace (adhesin to collagen of Enterococcus faecalis), encoding a virulence factor in e

119 (predominant microorganism in institution A: Enterococcus faecalis, 18 cultures [51.4%]; institution

120 Enterococcus faecalis, a Gram-positive bacterium, and Ca

121 ve characterization of collateral effects in Enterococcus faecalis, a gram-positive opportunistic pat

122 Enterococcus faecalis, a leading cause of hospital-acqui

123 Multidrug-resistant Enterococcus faecalis, an opportunistic human pathogen,

124 eant dye, YOYO-1, were first developed using Enterococcus faecalis, an organism that has previously b

125 us aureus (MRSA), Listeria monocytogenes and Enterococcus faecalis, and against the Gram-negative bac

126 57, Listeria innocua, Staphylococcus aureus, Enterococcus faecalis, and Bacillus anthracis, on sample

127 ve bacteria, including Bacillus subtilis and Enterococcus faecalis, and drug-sensitive and drug-resis

128 occus epidermidis, Streptococcus pneumoniae, Enterococcus faecalis, and Enterococcus faecium) and thr

129 tion by a pure facultative anaerobic strain, Enterococcus faecalis, and fresh mixed anaerobic sludge,

130 9.65, and 100.00% for Staphylococcus aureus, Enterococcus faecalis, and streptococci, respectively.

131 photoinactivation of a laboratory strain of Enterococcus faecalis, but depressed photoinactivation o

132 In Enterococcus faecalis, conjugation of a Cas9-targeted pl

133 In other gram-positive bacteria, such as Enterococcus faecalis, disulfide bonds are formed in sec

134 y named celBA) of the opportunistic pathogen Enterococcus faecalis, encodes a 6-phospho-beta-glucosid

135 s (Bacillus cereus group, Enterococcus spp., Enterococcus faecalis, Enterococcus faecium, Staphylococ

136 am-positive pathogens Staphylococcus aureus, Enterococcus faecalis, Enterococcus faecium, Streptococc

137 ides thetaiotaomicron, Campylobacter jejuni, Enterococcus faecalis, Escherichia coli K12, E. coli O15

138 robial CAUTI and frequently cocolonizes with Enterococcus faecalis, Escherichia coli, Providencia stu

139 TDB, Escherichia coli, Bacillus subtilis and Enterococcus faecalis, from the guts of the desert woodr

140 idium perfringens, Escherichia coli), except Enterococcus faecalis, human milk was more antimicrobial

141 e wetland, while for the bacterial indicator Enterococcus faecalis, inactivation results were compara

142 , Clostridium species, Enterobacter cloacae, Enterococcus faecalis, Klebsiella oxytoca, Klebsiella pn

143 ecent clinical isolates of Escherichia coli, Enterococcus faecalis, Klebsiella pneumoniae, and Pseudo

144 : Pseudomonas aeruginosa, Proteus mirabilis, Enterococcus faecalis, Klebsiella pneumoniae, Escherichi

145 ntestinal microbiota (Lactobacillus reuteri, Enterococcus faecalis, Lactobacillus crispatus and Clost

146 Enterococcus faecalis, long implicated in serious system

147 species such as Staphylococcus epidermidis, Enterococcus faecalis, Pseudomonas aeruginosa, and Klebs

148 the AMP-modifications with Escherichia coli, Enterococcus faecalis, Pseudomonas aeruginosa, Staphyloc

149 induced stresses (Brochothrix thermosphacta, Enterococcus faecalis, Pseudomonas fluorescens, Salmonel

150 t side of the heart caused by streptococcus, Enterococcus faecalis, Staphylococcus aureus, or coagula

151 fect of the sealer to multispecies bacteria (Enterococcus faecalis, Streptococcus gordonii, Actinomyc

152 In Enterococcus faecalis, the regulatory nucleotides pppGpp

153 Here, using a laboratory strain of Enterococcus faecalis, we developed a novel Caenorhabdit

154 nic Escherichia coli (UPEC) or Gram-positive Enterococcus faecalis, we used a mouse transurethral ins

155 adenovirus 41, Phi X 174) and the bacterium Enterococcus faecalis, which are relevant for water hygi

156 ms are absent from multidrug-resistant (MDR) Enterococcus faecalis, which only possess an orphan CRIS

157 Escherichia coli, Pseudomonas aeruginosa and Enterococcus faecalis.

158 r times at room temperature in comparison to Enterococcus faecalis.

159 leukemia cells, yeast, Escherichia coli and Enterococcus faecalis.

160 ion of IL-10 deficient (Il10(-/-)) mice with Enterococcus faecalis.

161 itogenic, protease-secreting enteric microbe Enterococcus faecalis.

162 tococcus mutans, Actinomyces naeslundii, and Enterococcus faecalis.

163 istic pathogens Clostridioides difficile and Enterococcus faecalis.

164 lococcus aureus (MRSA), vancomycin-resistant Enterococcus faecalis/faecium (VREfc/VREfm), and ciprofl

165 cocci (16.9%), Escherichia coli (11.8%), and Enterococcus faecium (11.4%).

166 We discovered that Enterococcus faecium (E. faecium), a ubiquitous commensa

167 g a predicted fibronectin-binding protein of Enterococcus faecium (fnm), a homologue of Streptococcus

168 reaks of linezolid- and vancomycin-resistant Enterococcus faecium (LR-VRE) in solid organ transplant

169 ee important pathogens: vancomycin-resistant Enterococcus faecium (n=19), methicillin-resistant Staph

170 Vancomycin-resistant Enterococcus faecium (VRE) is a leading cause of hospita

171 Vancomycin-resistant Enterococcus faecium (VRE) is a major cause of nosocomia

172 tion rule to guide anti-vancomycin-resistant Enterococcus faecium (VRE) therapy.

173 onization of the gut by vancomycin-resistant Enterococcus faecium (VRE), a leading cause of hospital-

174 lococcus aureus (MRSA), vancomycin-resistant Enterococcus faecium (VRE), and beta-lactam-resistant Kl

175 to marked expansion of vancomycin-resistant Enterococcus faecium (VRE), Klebsiella pneumoniae, and E

176 occus aureus (MRSA) and vancomycin-resistant Enterococcus faecium (VREF) with MIC values of 1.4 and 2

177 population structure of vancomycin-resistant Enterococcus faecium (VREfm) in Latin America (LATAM).

178 Vancomycin-resistant Enterococcus faecium (VREfm) is a leading cause of healt

179 Vancomycin-resistant Enterococcus faecium (VREfm) is a leading cause of nosoc

180 Vancomycin-resistant Enterococcus faecium (VREfm) is an important cause of he

181 ion and transmission of vancomycin-resistant Enterococcus faecium (VREfm) is the driver for E. faeciu

182 nt organisms, including vancomycin-resistant Enterococcus faecium (VREfm).

183 rcent of bacteria were MDR, including 95% of Enterococcus faecium and 55% of Enterobacteriaceae; 82%

184 stant microbes, such as vancomycin-resistant Enterococcus faecium and carbapenem-resistant Klebsiella

185 ia (high priority) were vancomycin-resistant Enterococcus faecium and meticillin-resistant Staphyloco

186 drug-sensitive and drug-resistant strains of Enterococcus faecium and Staphylococcus aureus.

187 ront-line antibiotic for multidrug-resistant Enterococcus faecium bloodstream infections (BSIs).

188 We identify Acinetobacter baumannii and Enterococcus faecium co-association on multiple surfaces

189 We tested this idea in a mouse model of Enterococcus faecium gastrointestinal tract colonization

190 The incidence of multidrug-resistant Enterococcus faecium hospital infections has been steadi

191 e focus on the important nosocomial pathogen Enterococcus faecium in a hospital system where resistan

192 In particular, vancomycin-resistant Enterococcus faecium infections have been increasing in

193 Enterococcus faecium is a common cause of nosocomial inf

194 Enterococcus faecium is an important cause of hospital-a

195 nical settings, non-susceptibility to DAP by Enterococcus faecium is correlated frequently with a mut

196 We show that secreted antigen A (SagA) from Enterococcus faecium is sufficient to protect Caenorhabd

197 dynamic modeling that bisected the wild-type Enterococcus faecium MIC distribution.

198 Previously, we have shown that 2 Enterococcus faecium proteins, the secreted antigen A an

199 ted to be efficacious against a DAP-tolerant Enterococcus faecium strain (HOU503).

200 re induced in one Campylobacter coli and one Enterococcus faecium strain, while these strains plus th

201 ; however, smaller segments were detected in Enterococcus faecium strains.

202 precedes infection with antibiotic-resistant Enterococcus faecium We used a mouse GIT colonization mo

203 occus pneumoniae, Enterococcus faecalis, and Enterococcus faecium) and three associated genetic resis

204 nized cluster of 10 genetically related VRE (Enterococcus faecium) infections was discovered.

205 ined individually and in combination against Enterococcus faecium, Acinetobacter baumannii and Klebsi

206 that the silver/platinum combination against Enterococcus faecium, and silver/copper combination agai

207 ation demonstrated platinum and gold against Enterococcus faecium, platinum against Klebsiella pneumo

208 rug-resistant organisms (MDROs), and ESKAPE (Enterococcus faecium, Staphylococcus aureus, Klebsiella

209 identifies the most common ESKAPE bacteria (Enterococcus faecium, Staphylococcus aureus, Klebsiella

210 he rapid identification of ESKAPE pathogens (Enterococcus faecium, Staphylococcus aureus, Klebsiella

211 Mortality was also analyzed for Enterococcus faecium, Staphylococcus aureus, Klebsiella

212 p, Enterococcus spp., Enterococcus faecalis, Enterococcus faecium, Staphylococcus spp., Staphylococcu

213 taphylococcus aureus, Enterococcus faecalis, Enterococcus faecium, Streptococcus pyogenes, Streptococ

214 in a clinical strain of daptomycin-resistant Enterococcus faecium, using a murine model of peritoniti

215 These effects do not occur in Enterococcus faecium, which lacks the respiratory chain

216 hylococcus aureus, Enterococcus faecalis and Enterococcus faecium.

217 patients were Clostridium sp. 7 2 43FAA and Enterococcus faecium.

218 of the bacteria such as Escherichia coli and Enterococcus faecium.

219 idermidis in synthetic urine also containing Enterococcus faecium.

220 ectrum activity against vancomycin-resistant Enterococcus faecium.

221 and vancomycin, such as vancomycin-resistant Enterococcus faecium.

222 beum (Staphylococcus aureus: MIC: 16 mug/mL; Enterococcus faecium: MIC: 32 mug/mL) and Harungana mada

223 , Streptococcus, Clostridium, Desulfovibrio, Enterococcus, Fusobacterium, and several new genera desc

224 describe the immunostimulatory properties of Enterococcus gallinarum MRx0518, a candidate live biothe

225 solated K. pneumoniae, Proteus mirabilis and Enterococcus gallinarum, which were prevalently detected

226 s belonging to the phylum Proteobacteria and Enterococcus genus have also been linked to increased tr

227 Enterococcus growth is dependent on the disaccharide lac

228 Staphylococcus aureus and enterococcus had the highest 1-year mortality (adjusted

229 performance, we conclude that the developed Enterococcus HDA assay has great potential as a qualitat

230 The developed Enterococcus HDA assay successfully discriminated 15 ent

231 Enterococcus hirae accounted for 92% of the fecal isolat

232 Here, we identified two bacterial species, Enterococcus hirae and Barnesiella intestinihominis that

233 age found in the genome of the bacteriophage Enterococcus hirae Mice bearing E. hirae harboring this

234 irst high-resolution view of ATP activity in Enterococcus hirae V1-ATPase.

235 structure of the related A-ATP synthase from Enterococcus hirae, the arrangements of the ScDF molecul

236 a and UTI and a 1% relative gut abundance of Enterococcus is an independent risk factor for Enterococ

237 d dense colonization by vancomycin-resistant Enterococcus, K. pneumoniae, and E. coli.

238 sed responses compared with those possessing Enterococcus, Klebsiella, and Enterobacter isolates from

239 f eight strains (four Campylobacter and four Enterococcus) obtained macrolide-resistant mutants, incl

240 se, and dietary lactose depletion attenuates Enterococcus outgrowth and reduces the severity of GVHD

241 on resistance and increases vulnerability to Enterococcus outgrowth.

242 olically active cells (E. coli, B. subtilis, Enterococcus, P. aeruginosa and Salmonella typhi) to ant

243 In a mouse model of enterococcus peritonitis, BALB-C mice were challenged wi

244 Escherichia-, Enterobacteria-, and Enterococcus phages were over-represented in fecal sampl

245 ylococcus species (61% vs 42%, P = .001) and Enterococcus species (20% vs 9%, P = .002) were more fre

246 , including due to Escherichia coli (16.3%), Enterococcus species (3.9%), and group B Streptococcus (

247 al inoculation consisting of twelve cultured Enterococcus species combined with conventional intestin

248 Among patients with early endocarditis, Enterococcus species were the most frequently isolated m

249 ring the early period, is commonly caused by Enterococcus species, and results in considerable risks

250 Escherichia coli and Enterococcus species, both indicators of fecal contamina

251 cal indicator bacteria measured in seawater (Enterococcus species, fecal coliforms, total coliforms)

252 on by clinically vexing vancomycin-resistant Enterococcus species.

253 Enterococcus spp (20%) occurred as urinary tract pathoge

254 The combinations of Bacillus spp and Enterococcus spp, and 1 or more Bifidobacterium spp and

255 almonella spp. < E. coli < total coliforms < Enterococcus spp.

256 beta-glucosidase (beta-gluco) is produced by Enterococcus spp.

257 in mice by increasing numbers of intestinal Enterococcus spp.

258 calis from 20 different Enterococcus and non-Enterococcus spp.

259 Streptococcus pneumoniae (1.07 to 0.26) and Enterococcus spp. (0.60 to 0.17) declined.

260 Enterobacteriaceae (42%), Enterococcus spp. (24%), and Candida spp. (15%) were pre

261 erobacteriaceae (43.0%, 52/121), followed by Enterococcus spp. (32.2%, 39/121), and Candida spp. (9.1

262 faecal indicator bacteria (FIB; E. coli and Enterococcus spp. (ENT)), Pseudomonas spp., and ARGs (bl

263 lococcus aureus (MRSA), vancomycin-resistant Enterococcus spp. (VRE), extended-spectrum beta-lactamas

264 s observed by flow cytometry, and inoculated Enterococcus spp. and Salmonella typhimurium during the

265 Staphylococcus aureus, vancomycin-resistant Enterococcus spp. and several multidrug-resistant Gram-n

266 ) was much lower for Gram positive bacteria (Enterococcus spp. and Staphylococcus spp., including two

267 d 3.93 (95% CI: 1.57 to 9.84; p = 0.003) for Enterococcus spp. and Staphylococcus spp., respectively.

268 phylococcus aureus, and vancomycin-resistant Enterococcus spp. in the environment.

269 quality, and with multiresistant E. coli and Enterococcus spp. rising in concern, the quantification

270 a greater abundance of Escherichia spp. and Enterococcus spp. than healthy controls (n = 26).

271 itute revised the daptomycin breakpoints for Enterococcus spp. twice in rapid succession.

272 oli vs P. aeruginosa tau = 0.090, p = 0.027; Enterococcus spp. vs P. aeruginosa tau = 0.126, p = 0.00

273 ith valve surgery, Staphylococcus aureus and Enterococcus spp. were associated with valve culture gro

274 acteria (FIB) Escherichia coli (E. coli) and Enterococcus spp. were enumerated using culture-based me

275 Enterococcus spp. were the most frequent bacterial isola

276 orms, Escherichia coli, Salmonella spp., and Enterococcus spp.) naturally occurring in UWW and 74 org

277 monas aeruginosa, daptomycin breakpoints for Enterococcus spp., and ceftaroline breakpoints for Staph

278 ram-positive targets (Bacillus cereus group, Enterococcus spp., Enterococcus faecalis, Enterococcus f

279 Enterococcus spp., Escherichia coli and Klebsiella pneum

280 Enterococcus spp., Escherichia coli, Salmonella enterica

281 robial susceptibility test (AST) results for Enterococcus spp., Escherichia coli, Staphylococcus aure

282 comprises the group of Enterobacteriacae and Enterococcus spp., with a high proportion of multiresist

283 This review highlights the ability of Enterococcus, Staphylococcus, Klebsiella, Acinetobacter,

284 of a gene specific to a vancomycin-resistant Enterococcus strain was performed on the developed micro

285 he four identified genes, found in Listeria, Enterococcus, Streptococcus and Staphylococcus genomes,

286 er, swarm cells rarely tumbled, and cells of Enterococcus tended to swim in loops when moving slowly.

287 on MRSA, C. difficile, vancomycin-resistant Enterococcus (VRE) and ICU-acquired bloodstream infectio

288 Vancomycin-resistant Enterococcus (VRE) are highly antibiotic-resistant and r

289 examined the impact of vancomycin-resistant Enterococcus (VRE) bloodstream infection (BSI) on outcom

290 infection (BSI) to due vancomycin-resistant Enterococcus (VRE) is an important complication of hemat

291 ections attributable to vancomycin-resistant Enterococcus (VRE) strains have become increasingly prev

292 on MRSA, C. difficile, vancomycin-resistant Enterococcus (VRE), and ICU-acquired bloodstream infecti

293 lococcus aureus (MRSA), vancomycin-resistant Enterococcus (VRE), and MDR Enterobacteriaceae Fecal met

294 lococcus aureus (MRSA), vancomycin-resistant enterococcus (VRE), extended-spectrum cephalosporin resi

295 estinal domination with vancomycin-resistant Enterococcus (VRE), leading to bloodstream infection in

296 lococcus aureus (MRSA), vancomycin-resistant Enterococcus (VRE), Pseudomonas aeruginosa (PA), and Can

297 contact precautions for vancomycin-resistant Enterococcus (VRE).

298 Enterococcus was measured both by culture (cENT) and DNA

299 appears to promote overgrowth of intestinal Enterococcus, which promotes liver disease, based on dat

300 p postoperative increase in the abundance of Enterococcus, which was also cultured from wound drainag