ライフサイエンスコーパス: Ets transcription factor

1 33, Ccl20, and SAM pointed domain-containing Ets transcription factor.

2 ein with the DNA binding domain of FLI-1, an ETS transcription factor.

3 encompasses multiple binding motifs for the ETS transcription factor.

4 thway regulates G-CSF expression through the Ets transcription factor.

5 nd synergistically activated by Forkhead and Ets transcription factors.

6 ulation by the absence of a binding site for Ets transcription factors.

7 sors, including a core component of PRC1 and ETS transcription factors.

8 E-3), a novel member of the ESE subfamily of Ets transcription factors.

9 s may function as dominant negative forms of ets transcription factors.

10 s of Ras1 and two Ras1/MAP kinase-responsive ETS transcription factors.

11 inding sites to target genes associated with ETS transcription factors.

12 rol the activity of this subset of oncogenic Ets transcription factors.

13 canonical ETS motifs by displacing wild-type ETS transcription factors.

14 ge effects are dependent on de-repression of Ets transcription factors.

15 result in overexpression of any one of four ETS transcription factors.

16 des an erythroblast transformation-specific (ETS) transcription factor.

17 L sequences, an E26-transformation-specific (ETS) transcription factor.

18 ccurred via the E26 transformation-specific (ETS) transcription factor.

19 ding site of an E26 transformation-specific (ETS) transcription factor.

20 alysis revealed that the epithelium-specific ETS transcription factor-1 (ESE-1 or ELF3), known to reg

21 Previously, E74 like ETS transcription factor 3 (ELF3) was identified to gove

22 ers of EHF and the 5' adjacent gene E47 like ETS transcription factor 5 (ELF5) in reporter gene assay

23 SAP-1 is a member of the Ets transcription factors and cooperates with SRF protei

24 FOXR2 extends the mechanisms known to active ETS transcription factors and highlights how transcripti

25 e precise spatial relationship between these Ets transcription factors and the FGF signal originating

26 nscriptional control of Bcl-xl that involves ETS transcription factors and the HGF/Met axis.

27 the anterior one producing the Pointed (Pnt) ETS transcription factors and the posterior one the Odd-

28 a complex interplay between the Ras pathway, ETS transcription factors, and enhancer binding as a cru

29 Here we report that beta-catenin, Lef-1, Ets transcription factors, and the AP-1 protein c-Jun ea

30 o consensus binding motifs for E-twenty-six (ETS) transcription factors, and in reporter assays, the

31 ETS transcription factors are commonly deregulated in ca

32 cations leading to deregulated expression of ETS transcription factors are frequent in prostate tumor

33 ETS transcription factors are hypothesized to play a sim

34 Ets transcription factors are important downstream targe

35 Ets transcription factors are known to be important medi

36 Here, members of two groups of ETS transcription factors are shown to act directly at t

37 The DA motif is activated by the ETS transcription factor AST-1.

38 eal disparate, but overlapping, functions of Ets transcription factors at distinct stages of megakary

39 ted transcription and activate expression of ETS transcription factors at the early stages of more th

40 assays demonstrated that IRF-8 and the PU.1 Ets transcription factor bind to this element in vivo.

41 This region contains an Ets transcription factor binding motif, and a 2-base pai

42 scription initiation site and includes three Ets transcription factor binding sequences.

43 n by deltaRaf-1:ER required a composite AP-1/Ets transcription factor binding site located between bp

44 -responsive element (HRE) in proximity to an ETS transcription factor binding site.

45 appaB consensus site, it does contain tandem Ets transcription factor binding sites previously shown

46 ell enhancer contains four perfect consensus Ets transcription factor binding sites that are efficien

47 omoters show significant enrichment for NRF2/ETS transcription factor binding sites.

48 Consistent with the predicted loss of ETS transcription factor binding, we observed that recur

49 reate a de novo E26 transformation-specific (ETS) transcription factor binding motif.

50 tivity is dependent on a likely Fgf-mediated Ets transcription factor-binding site.

51 mutations are predicted to disrupt consensus ETS transcription factor-binding sites and are correlate

52 ndrogen-stimulated overexpression of ERG, an ETS transcription factor, but critical downstream effect

53 We further demonstrate that Ets transcription factors, but not Nrf2, are regulated d

54 pecific transcription factors, including the ETS transcription factors, C/EBP, GATA, and CREB.

55 pression of the E26 transformation-specific (ETS) transcription factors characterizes numerous human

56 Ets transcription factors control multiple biological pr

57 Transcriptional analysis revealed that Ets transcription factors controlled VEGFR2 expression i

58 )-8, PSMB9, and PSMB10, which occurred in an ETS transcription factor-dependent manner.

59 with a key role for GABPA/B1 as the critical ETS transcription factors deregulating SDHD expression i

60 d dosage of ERF, which encodes an inhibitory ETS transcription factor directly bound by ERK1/2 (refs.

61 tion codon after Arg254 within the conserved ETS transcription factor domain confirmed the lethality

62 this study, we report that the E-twenty six (ETS) transcription factor E74-like factor 4 (ELF4) suppr

63 ction studies and ChIP-seq, we show that the Ets transcription factor EHF promotes cornea epithelial

64 The ETS transcription factor Elf-4 is an important regulator

65 wth factor family members), induction of the ETS transcription factor Elf3, which transactivates gene

66 During pregnancy, the ETS transcription factor ELF5 establishes the milk-secre

67 Additional experiments indicate that the ETS transcription factor ELK is required for HIF-2alpha

68 racellular signal-regulated kinase (ERK1/2), ETS transcription factor Elk-1, and activity-regulated c

69 e mitogen-activated protein kinase-regulated ETS transcription factor Elk-1, but not by multiple othe

70 erative p38 mitogen-activated protein kinase/ETS transcription factor ELK1 (Elk1)/cFos signaling is m

71 ugh the EGR1 (Early Growth Response 1)-ELK1 (ETS Transcription Factor ELK1)-ERK (Extracellular Signal

72 cancer-associated E26 transformed specific (ETS) transcription factor ELK4, and comprises numerous g

73 f, stimulate hTERT promoter activity via the ETS transcription factor ER81 and ERK mitogen-activated

74 In this study, we show that the ETS transcription factor ER81 directly binds to and acti

75 The regulated expression of the ETS transcription factor ER81 is a prerequisite for norm

76 The ETS transcription factor ER81 is activated in response t

77 The ETS transcription factor ER81 is expressed in sensory ne

78 ied a novel interaction partner of ACTR, the ETS transcription factor ER81 that is also heavily impli

79 ein HER2/Neu is able to collaborate with the ETS transcription factor ER81 to activate Smad7 transcri

80 in, however, contains cells that express the ETS transcription factor Er81, which is also expressed i

81 cluding the subpopulation that expresses the ETS transcription factor ER81.

82 is similar to the defect in mice lacking the ETS transcription factor ER81.

83 Two ETS transcription factors, ER81 and PEA3, are expressed

84 In previous studies we found that the ets transcription factor ERG and its alternatively-splic

85 cy data, we identified and characterized the ETS transcription factor ERG as a direct transcriptional

86 To examine the hypothesis that the Hsa21 ETS transcription factor ERG cooperates with GATA1s in t

87 ow that mice with homozygous deletion of the Ets transcription factor Erg die between embryonic day 1

88 The ETS transcription factor ERG drives expression of VE-cad

89 251-262) show an essential role for the ETS transcription factor ERG in the self-renewal of embr

90 The ETS transcription factor ERG is aberrantly expressed in

91 The ETS transcription factor Erg is required for endothelial

92 Overexpression of KLF2 or the ETS transcription factor ERG is sufficient to induce ect

93 The ETS transcription factor ERG plays a central role in def

94 The endothelial ETS transcription factor Erg plays an important role in

95 nduces phosphorylation and activation of the ETS transcription factor ERG, a prerequisite for DLL4 in

96 s in transcriptional activation of truncated ETS transcription factors ERG and ETV1, the first candid

97 normally silent E26 transformation-specific (ETS) transcription factor ERG in prostate cells.

98 sions involving E26 transformation-specific (ETS) transcription factors ERG, ETV1, ETV4, or ETV5 have

99 nd Ikaros were most common in T-ALL, whereas ETS transcription factors (Erg and Ets1) were targeted i

100 ement resulting in the overexpression of the ETS transcription factor, ERG; however, the functional s

101 Two ETS transcription factors, ERG and ETV1, were identified

102 Mechanistically, FUS collaborates with the ETS transcription factor ERM to stimulate transcription

103 The newly identified epithelium-specific ets transcription factor ERT/ESX/ELF-3/ESE-1/jen binds t

104 ently isolated the first epithelium-specific Ets transcription factor (ESE-1).

105 ort the involvement of a novel member of the ETS transcription factor, ESE-1, in mediating vascular i

106 ere, we show that the endogenously expressed ETS transcription factor ESE3/EHF controls prostate epit

107 cer, we investigated connections between the ETS transcription factor ESE3/EHF, the Lin28/let-7 micro

108 sent study, we investigated whether ER71, an Ets transcription factor essential for hematopoietic and

109 utant lung, two E26 transformation-specific (ETS) transcription factors essential for normal lung bra

110 The Ets transcription factor, ESX, exhibits a unique pattern

111 In this report, we demonstrate that the ETS transcription factor ETS variant 5 (ETV5) promotes I

112 The genes encoding the E-twenty-six (ETS) transcription factors Ets related gene (Erg) and Et

113 urally related E-26 transformation-specific (ETS) transcription factors, ETS-related gene (ERG) and F

114 ting IL-2 production as proposed for another ETS transcription factor, ETS1.

115 DA motifs that can be activated by the mouse ETS transcription factor Etv1 (also known as ER81), and

116 al muscles differ in their dependence on the ETS transcription factor Etv1 for their survival and dif

117 The ETS transcription factor ETV1 is frequently overexpresse

118 r, combined overexpression of JMJD2A and the ETS transcription factor ETV1, a JMJD2A-binding protein,

119 cular basis of DNA-binding autoinhibition of ETS transcription factors ETV1, ETV4 and ETV5, which are

120 The ETS-transcription factor ETV1 is involved in epithelial-

121 regulatory element and mutually repress the ETS transcription factor, ETV1.

122 By transient expression of the ETS transcription factor ETV2 for 2 weeks and constituti

123 An ETS transcription factor Etv2 functions as an evolutiona

124 The ETS transcription factor Etv2 is necessary and sufficien

125 e for Forkhead transcription factors and the Ets transcription factor Etv2, for activity in vivo.

126 ETS transcription factors ETV2, FLI1, and ERG1 specify p

127 Overexpression of key endothelial ETS transcription factors (ETV2, ERG, and FLI1) reprogra

128 Although the ETS transcription factors Etv4 and Etv5 are known to be

129 Here we show that the ETS transcription factors Etv4 and Etv5 are positively r

130 we show that FGF-dependent activation of the ETS transcription factors Etv4 and Etv5 contributes to p

131 Prior binding of the repressive Ets transcription factor Etv6 predicts cohesin binding a

132 Here, we show that the ETS transcription factor, Etv6, positively regulates veg

133 Prostate-derived Ets factor (PDEF) is an ETS transcription factor expressed in normal tissues wit

134 ty are unclear, the STAT, Rel/NF-kappaB, and Ets transcription factor families have recently been rep

135 Fli-1 belongs to the Ets transcription factor family and is expressed primari

136 The ER71 protein belongs to the ETS transcription factor family and is testis-specifical

137 of BLCA-4 reveals that it is a member of the ETS transcription factor family and that it seems to ass

138 Members of the Ets transcription factor family are widely expressed in

139 First, by screening the entire ETS transcription factor family for rearrangements, we f

140 defining a role for selected members of the ETS transcription factor family in the regulation of vas

141 The ETS transcription factor family is characterized by a co

142 The Ets transcription factor family is involved in a variety

143 novel target of MK2 has been identified, the ETS transcription factor family member ER81, whose dysre

144 cbp-1 may function in concert with LIN-1, an Ets transcription factor family member that is one of th

145 iend leukemia virus integration 1 (FLI1), an Ets transcription factor family member, is linked to acu

146 viously isolated different isoforms of a new Ets transcription factor family member, NERF/ELF-2, NERF

147 the androgen-regulated gene TMPRSS2 and the ETS transcription factor family members ERG, ETV1, and E

148 of androgen-regulated TMPRSS2 promoter with ETS transcription factor family members is found frequen

149 nslocations fuse the EWS gene to a subset of ets transcription factor family members, most commonly t

150 ibody disruption assays demonstrated that an Ets transcription factor family protein, GA-binding prot

151 ERG is a member of the ETS transcription factor family that is highly enriched

152 a prototypic member of a novel subset of the ETS transcription factor family that is predominantly ex

153 hermore, we found that PU.1 (a member of the Ets transcription factor family that plays a crucial rol

154 fies ETS-related gene (ERG), a member of the ETS transcription factor family, as the most frequently

155 PU.1, a member of the ets transcription factor family, has been previously sho

156 integration factor 1 (Fli1), a member of the Ets transcription factor family, has been shown to play

157 ERG, a member of the ETS transcription factor family, is frequently overexpre

158 PU.1 (spi-1), a member of the Ets transcription factor family, is predominantly expres

159 egulated TMPRSS2 promoter to a member of the ETS transcription factor family, most commonly ERG.

160 is an epithelially restricted member of the ETS transcription factor family, which is involved in a

161 he isolation of Tel-2, a novel member of the Ets transcription factor family, with high homology to T

162 e fusions between TMPRSS2 and members of the ETS transcription factor family.

163 uggest two distinct roles for members of the ETS transcription factor family.

164 Ets1 is a member of the Ets transcription factor family.

165 strongly stimulated by Elf3, a member of the Ets transcription factor family.

166 e consensus-binding site for a member of the ets transcription factor family.

167 comprise the most divergent subfamily of the Ets transcription factor family.

168 POU domain protein Pit-1 and members of the ETS transcription factor family.

169 ETS-related gene (ERG) is a member of the ETS transcription factor family.

170 ctor Lame duck and is likely a member of the ETS transcription factor family.

171 erythroblast transformation-specific domain (Ets) transcription factor family member Spi-C, and oncog

172 ne megakaryoblastic cells and identified the ets transcription factor FLI-1 as a novel in vivo-associ

173 DNA-binding and COOH-terminal regions of the Ets transcription factor FLI-1.

174 rminal EWS with the DNA binding moiety of an ETS transcription factor (FLI-1 in 90% of cases).

175 chanism whereby the fusion of EWSR1 with the ETS transcription factor FLI1 contributes to malignant t

176 FOXF1 synergized with ETS transcription factor FLI1 to activate ACVRL1 promote

177 EWS(LCD)) with the DNA binding domain of the ETS transcription factor FLI1.

178 for 2 weeks and constitutive expression the ETS transcription factors FLI1 and ERG1, concomitant wit

179 actor 1, Fli-1 proto-oncogene, E-twenty-six (ETS) transcription factor (friend leukemia integration 1

180 Interfering with Ets transcription factor function reverses multiple aspe

181 Here we show that ETS transcription factor fusions resulting from disease

182 The E-twenty six (ets) transcription factor GA binding protein (GABP) is a

183 Binding sites for the ets transcription factor, GA-binding protein (GABP), and

184 include interactions of the widely expressed ETS transcription factor, GA-binding protein (GABP), wit

185 us and that this activity is mediated by the ets transcription factor, GABPalpha.

186 ed loss-of-function variants in the X-linked ETS transcription factor gene ELF4 in multiple unrelated

187 eobox transcription factor gene DUX4 and the ETS transcription factor gene ERG is a hallmark of a sub

188 ssion of the human FEV (fifth Ewing variant) ETS transcription factor gene impacts the level of CNS s

189 1), an erythroblast transformation-specific (ETS) transcription factor, governs pericyte viability in

190 Etv2, an Ets-transcription factor, governs the specification of t

191 The current paradigm of ETS transcription factors holds that their DNA-binding (

192 Here we report that transcripts of PEA3, an ETS transcription factor implicated in oncogenesis, were

193 are activated by the combination of ZicL and ETS transcription factors in Ciona embryos.

194 tween the EWS gene and one of five different ETS transcription factors in Ewing's family tumors (EFTs

195 n pattern, we analyzed the roles of Runx and ETS transcription factors in the laminar targeting of se

196 get of AR action in TMPRSS2, a gene fused to ETS transcription factors in the majority of prostate ca

197 ion of PARP expression levels, and implicate ETS transcription factors in the radiation response of E

198 he Raf kinase, suggesting the involvement of Ets transcription factors in the regulation of GlcNAc-T

199 In this study, we investigated the role of Ets transcription factors in the regulation of NOS2 by L

200 thus implicating a novel function for these ETS transcription factors in the regulation of the Egr1

201 further implicate a novel function for these ETS transcription factors in the regulation of the Egr1

202 dentified highly conserved binding sites for Ets transcription factors in the Tie2 promoter.

203 tal lineage dependency mediated by oncogenic ETS transcription factors in this malignancy.

204 K homolog) and upstream of lin-1 (encodes an Ets transcription factor) in the anchor cell signaling p

205 Lef-1, however, was considerably enhanced by Ets transcription factors including Ets-1, Ets-2, ERM, a

206 Recurrent gene fusions involving oncogenic ETS transcription factors (including ERG, ETV1, and ETV4

207 Several ETS transcription factors, including ELF4/MEF, can funct

208 Motifs for several ETS transcription factors, including GABPA and ELF1, are

209 protein interaction domain, found in several ETS transcription factors, including TEL (translocation

210 The LIN-1 ETS transcription factor inhibits vulval cell fates duri

211 The aberrant expression of an oncogenic ETS transcription factor is implicated in the progressio

212 Fli-1 (friend leukemia integration 1), an Ets transcription factor, is required for the normal mat

213 We show here that Pointed, an ETS transcription factor, is required in dorsal follicle

214 -9 expression is regulated by AP-1, Sp1, and Ets transcription factors, KiSS-1 did not alter the bind

215 Recent evidence suggests that Ets transcription factors may contribute to NOS2 inducti

216 Thus, downstream targets of Neu, including Ets transcription factors, may be useful points for ther

217 ETS transcription factors mediate a wide array of cellul

218 To identify which ETS transcription factors might be involved in Gata4 reg

219 the RNA binding protein EWS and one of five ETS transcription factors, most commonly FLI1.

220 e that 1 of 3 distinct isoforms of the novel Ets transcription factor NERF, NERF2, is expressed in en

221 ne expression dynamics controlled by an AP-1/ETS transcription factor network, where JUN is necessary

222 d lipid signaling pathways and activation of ETS transcription factors occur in oligodendrocytes only

223 itical AR-tethering proteins led to ELK1, an ETS transcription factor of the ternary complex factor s

224 ovel, highly tissue-restricted member of the ets transcription factor/oncogene family, ESE-1 (for epi

225 We recently isolated a novel member of the Ets transcription factor/oncogene family, ESE-1/ESX/ELF3

226 with an expression vector encoding the PEA3 Ets transcription factor, or its close relatives ER81 an

227 Pdef is an Ets transcription factor originally identified in prosta

228 a correlated with the expression of multiple ETS transcription factors, particularly in SDHD promoter

229 ion of a novel, prostate epithelium-specific Ets transcription factor, PDEF (prostate-derived Ets fac

230 The epithelium-specific Ets transcription factor, PDEF, plays a role in prostate

231 skeletal muscle, triggers expression of the ETS transcription factor Pea3 in a subset of motor neuro

232 essential for interaction of SRC-3 with the ETS transcription factor PEA3, which promotes upregulati

233 Of particular interest were the Ets transcription factors Pea3 and Erm, which function a

234 motor neuron survival factor, GDNF, and the ETS transcription factor, PEA3, as key components of a s

235 The ETS transcription factors perform distinct biological fu

236 Ets transcription factors play important roles during th

237 ETS transcription factors play important roles in hemato

238 y quantifying the expression dynamics of the ETS transcription factors Pnt and Yan.

239 Here we show that one isoform of the Ets transcription factor Pointed (Pnt), PntP1, is specif

240 ow that Btd functions cooperatively with the Ets transcription factor Pointed P1 to promote the gener

241 The ETS-transcription factor pointed (pnt) is expressed in a

242 ing a genetic screen, we have identified the ETS-transcription factor pointed as a key regulator of c

243 target of Spi/DER signaling mediated by the ETS transcription factor PointedP1 (PntP1).

244 at, contrary to ETS1, EWS/FLI-1, an aberrant ETS transcription factor present in most EWS cells, is a

245 The ETS transcription factor PU.1 has a potent ability to co

246 The Ets transcription factor PU.1 is a master regulator for

247 The ets transcription factor PU.1 is an important regulator

248 of neutrophil terminal differentiation, the ets transcription factor PU.1, was significantly decreas

249 pression through heterodimerization with the ETS transcription factor PU.1.

250 interference experiments, we found that the Ets transcription factors PU.1 and GA-binding protein (G

251 Although the ets transcription factor, PU.1, bound to this ets site,

252 Because ETS transcription factors regulate expression of TGFBR2

253 emonstrate that at subthreshold levels, this Ets transcription factor regulates a mixed pattern (macr

254 he ternary complex factor (TCF) subfamily of ETS-transcription factors represent key nuclear targets

255 (HMG-I(Y)) is a proposed co-activator of the ETS transcription factors required for mu enhancer activ

256 expression and suggest the participation of Ets transcription factor(s) in this control.

257 and Ets-2 proteins, thus confirming that an ETS transcription factor(s) recognizes the -12 motif.

258 te or modify binding sites for E-twenty-six (ETS) transcription factors, similar to mutations in the

259 ression of the SAM pointed domain containing ETS transcription factor (SPDEF or prostate-derived ETS

260 expression of SAM-pointed domain containing ETS transcription factor (SPDEF) in stratified conjuncti

261 monstrate that SAM pointed domain-containing ETS transcription factor (SPDEF) inhibited prostate canc

262 nt work, mouse SAM pointed domain-containing ETS transcription factor (SPDEF) mRNA and protein were d

263 gests that the SAM pointed domain containing ETS transcription factor (SPDEF) plays a significant rol

264 The SAM pointed domain containing ETS transcription factor (SPDEF) suppresses formation of

265 Furthermore, we isolated spib, an ETS transcription factor, specifically expressed in prim

266 Thus, Ets transcription factors specify non-vascular, amniotic

267 The DNA-binding ETS transcription factor Spi-1/PU.1 is of central import

268 inal deoxynucleotidyl transferase (TdT), the ETS transcription factor Spi-B, the nuclear factor-kappa

269 The Ets transcription factor Spi-C, expressed in B cells and

270 y, we report our analysis of the role of the Ets transcription factor, Spi-B, in this process.

271 ogram, including the sustained expression of Ets transcription factors such as ETV1 Together, our dat

272 Chromosomal rearrangements involving ETS transcription factors, such as ERG and ETV1, occur f

273 vo accessibility of distal enhancers binding ETS transcription factors, targeting important leukemoge

274 TEL2/ETV7 is highly homologous to the ETS transcription factor TEL/ETV6, a frequent target of

275 We show that the leukemia-associated Ets transcription factor, Tel1/ETV6, specifies the first

276 ETS1 is the archetype of the ETS transcription factor (TF) family.

277 ESE-1 is an epithelium-specific ETS transcription factor that contains two distinguishin

278 ERG (Ets-related gene) is an ETS transcription factor that has recently been shown to

279 etv2 is an endothelial-specific ETS transcription factor that is essential for vascular

280 ER71, also known as ETV2, is an ETS transcription factor that is expressed during embryo

281 SPIB is an Ets transcription factor that is expressed exclusively i

282 Moreover, we found that PU.1, an Ets transcription factor that is oncogenic in erythroid

283 ified prostate derived Ets factor (PDEF), an Ets transcription factor that is overexpressed in both p

284 GABP is an ets transcription factor that regulates genes that are r

285 SPI-B is a B lymphocyte-specific Ets transcription factor that shares a high degree of si

286 ESE-1 is an epithelium-specific ETS transcription factor that transforms human breast ep

287 polyoma enhancing activity-3/E26 virus (PEA3/ETS) transcription factors through consensus binding sit

288 s both the LIN-31 winged-helix and the LIN-1 Ets transcription factors to specify the vulval cell fat

289 g subtractive cloning, we identified ERM, an Ets transcription factor, to be a Th1-specific, IL-12-in

290 Ha-ras promoter resembles a binding site for Ets transcription factors, we did not detect the binding

291 Indeed, among the ETS transcription factors, we identified ELK1 as being m

292 ontaining the binding sites for the AP-1 and Ets transcription factors, we observed that IGF-I stimul

293 ctions of PU.1 and Spi-B, two highly related Ets transcription factors, we previously generated PU.

294 The GATA and Ets transcription factors were shown to function as acti

295 wing Family Tumors (EFT) harbor chimeric EWS/ETS transcription factors which are thought to aberrantl

296 from 22q-12 to FLI1 and genes encoding other ETS transcription factors (which bind DNA through the co

297 ression of the SAM pointed domain-containing ETS transcription factor, which contributes to goblet ce

298 Ets transcription factors, which share the conserved Ets

299 Among these is a novel ets transcription factor with a dual DNA-binding specifi

300 We find that the ETS transcription factor Yan is required for border cell