ライフサイエンスコーパス: FFA

1 FFA content of the crude and stabilized bran fractions t

2 FFA produces a significantly greater enhancement of curr

3 FFAs activated both the ERalpha and mTOR pathways and re

4 FFAs increased MIR122 expression in livers of mice by ac

5 FFAs induced collagen deposition and MMP2-9 activity red

6 s of magnitude if an internal standard (15:0 FFA) was added.

7 Among 20 FFAs identified in Fucus algae, 14 could be confirmed by

8 Thereby, 31 and 22 FFA signals were annotated by exact mass and identified

9 the crystal structure of ADIPOR2 bound to a FFA molecule and show that ADIPOR2 possesses intrinsic b

10 ile reducing utilization of free fatty acid (FFA) and branched-chain amino acid (BCAA).

11 HOMA-IR and positively with free fatty acid (FFA) and HDL after control for age and sex.

12 weight and elevated plasma free fatty acid (FFA) concentrations.

13 ride and glycogen contents, free fatty acid (FFA) content and release, and cholesterol and cholestero

14 riacylglycerol composition, free fatty acid (FFA) content, peroxide index, thermal properties, meltin

15 zed for 3-MCPD esters, GEs, free fatty acid (FFA) contents, specific extinction at 232 and 268 nm (K2

16 icantly reduced insulin and free fatty acid (FFA) levels (P < 0.001) and ameliorated the oxidative da

17 lipolysis, elevated plasma free fatty acid (FFA) levels, and impaired insulin signaling.

18 id translocase and elevated free fatty acid (FFA) levels.

19 sed 'lyso'-lipid as well as free fatty acid (FFA) levels.

20 stance and dysregulation of free fatty acid (FFA) metabolism are core defects in type 2 diabetic (T2D

21 ctly, determine the rate of free fatty acid (FFA) oxidation.

22 d dysfunction in a model of free fatty acid (FFA) palmitate-induced oxidative stress.

23 nses through members of the free fatty acid (FFA) receptor family, which includes FFA4.

24 Leptin receptor and free fatty acid (FFA) receptor, GPR120, are upregulated only in macrophag

25 triacylglyceride (TAG) and free fatty acid (FFA) species to be significantly increased.

26 a novel near-infrared (NIR) free fatty acid (FFA) tracer suitable for in vivo imaging of deep tissues

27 6 facilitates cell membrane free fatty acid (FFA) transport, but its role in human metabolism is not

28 Alterations in hepatic free fatty acid (FFA) uptake and metabolism contribute to the development

29 p4/Fabp5) impairs exogenous free fatty acid (FFA) uptake by CD8(+) TRM cells and greatly reduces thei

30 When applied to free fatty acid (FFA) uptake in 3T3-L1 adipocytes, this muChopper permitt

31 membrane, and thus greater free fatty acid (FFA) uptake, in adipocyte cell models.

32 a (PPARalpha) activation by free fatty acid (FFA), and cAMP response element-binding protein (CREB) a

33 uld be exacerbated by acute free fatty acid (FFA)-induced insulin resistance.

34 calpain-1 degrades Erk5 in free fatty acid (FFA)-stressed cardiomyocytes, whereas the prevention of

35 o produce sphingosine and a free fatty acid (FFA).

36 In vitro, fenofibric acid (FFA) rescued high glucose-induced (25 mmol/L) impairment

37 into the crystallization of flufenamic acid (FFA) in a confined environment of mesoporous silica mate

38 gatively charged activator, flufenamic acid (FFA) is critical in the search for more potent and selec

39 f a small organic molecule, flufenamic acid (FFA), a common pharmaceutical.

40 asting and mean OGTT plasma free fatty acid [FFA] x insulin concentrations), peripheral IR (1/[Matsud

41 in the form of circulating free fatty acids (FFA) also have toxic effects, and that the combination o

42 thymus weights, increased free fatty acids (FFA) and produced hyperglycemia and glucose intolerance.

43 cylglycerols, DAG, MAG and free fatty acids (FFA) and the concentration of saturated, mono- and polyu

44 n vitro assays, identified free fatty acids (FFA) as circulating plasma factors that correlated with

45 hibits ATGL expression and free fatty acids (FFA) beta-oxidation.

46 l extraction, (ii) Omega-3 free fatty acids (FFA) concentration (low temperature winterization), (iii

47 Free fatty acids (FFA) content of beer affects the ability to form a stabl

48 nherent increase in plasma free fatty acids (FFA) in the HFD together with an HFD-induced alteration

49 suppression of lipolysis, free fatty acids (FFA), and endogenous glucose production (EGP) in humans

50 e levels of triglycerides, free fatty acids (FFA), and leptin?

51 lipids in the fillets were free fatty acids (FFA), lipid hydroperoxides (PV) and thiobarbituric acid

52 a reduction in circulating free fatty acids (FFA).

53 SCs) cells were exposed to free fatty acids (FFAs) alone or in combination with OCA or INT-767.

54 nificant increase in serum free fatty acids (FFAs) and decrease in subcutaneous/peritoneal fat depots

55 in increased intracellular free fatty acids (FFAs) and elevated expression of uncoupling protein 2 (U

56 dance of saturated C16-C20 free fatty acids (FFAs) and long polyunsaturated complex lipids.

57 Free fatty acids (FFAs) are known to induce lipoapoptosis in liver cells i

58 podocytes, the presence of free fatty acids (FFAs) associated with serum albumin stimulated macropino

59 Nutrients such as free fatty acids (FFAs) contribute to precise regulation of beta cell mass

60 ree amino acids (FAAs) and free fatty acids (FFAs) during ripening of raw sheep's milk Tulum cheeses

61 duced by extraction of the free fatty acids (FFAs) from flaxseed oil, concentration of PUFAs, and enz

62 l as increased circulating free fatty acids (FFAs) in NAFLD, we hypothesized the involvement of chola

63 h its power in analysis of free fatty acids (FFAs) is limited.

64 ons of the polyunsaturated free fatty acids (FFAs) linoleic and alpha-linolenic acid, which we detect

65 Faba bean LOX preferred free fatty acids (FFAs) over triacylglycerols as substrates, and together

66 Serum free fatty acids (FFAs) profile is highlighted in its association with obe

67 ted the mechanism by which free fatty acids (FFAs) regulate MIR122 expression and the effect of MIR12

68 n elevated plasma ratio of free fatty acids (FFAs) to albumin when proteinuria reached nephrotic rang

69 did not affect myocardial free fatty acids (FFAs) uptake but reduced myocardial glucose uptake by 57

70 Circulating free fatty acids (FFAs) were less suppressed in IR than IS subjects.

71 hat acyl-CoAs, rather than free fatty acids (FFAs), are the preferred substrate for CvFAP.

72 ons of intestinal origin), free fatty acids (FFAs), insulin, glucose, glucagon, glucagon-like peptide

73 WFOs) with high content of free fatty acids (FFAs), otherwise unsuitable for biodiesel production.

74 cleavage of TGs generates free fatty acids (FFAs), which can serve as energy substrates, precursors

75 or the slow suppression of free fatty acids (FFAs), which in turn is responsible for delayed suppress

76 his led to elevated plasma free fatty acids (FFAs), which were transported to the adipose tissue for

77 ated by nonesterified or "free" fatty acids (FFAs).

78 n plasma phospholipids and free fatty acids (FFAs).

79 ia with elevated levels of free fatty acids (FFAs).

80 ents (hydrocarbons - HCs, free fatty acids - FFAs, free fatty alcohols - FALs and wax esters - WEs) o

81 metabolic mediators (e.g., free fatty acids, FFA) associated with excess adiposity and implicated in

82 lipid digestion products (free fatty acids, FFAs, and monoacylglycerides, MAGs) during in vitro dige

83 We find that fast flicker adaptation (FFAd) shifts the tuning of face perception to higher spa

84 Additional FFA analysis confirmed that the purified effector lipase

85 but are also effective in protecting against FFA-induced oxidative stress; thus, EMP function is refl

86 Frontal fibrosing alopecia (FFA) is a recently described inflammatory and scarring t

87 nd performed bioluminescence imaging with an FFA probe.

88 cerides [SMD=0.97 (95% CI: 0.53, 1.40)], and FFA [SMD=0.86 (95% CI: 0.50, 1.22)], and a nonsignifican

89 highlighting the specific roles of CD36 and FFA uptake.

90 f adipose tissue IR to increase glycerol and FFA availability to the liver in both receptor and postr

91 in beta-cells possibly comprise glycerol and FFA formation and release extracellularly and the divers

92 od levels, the interplay between insulin and FFA was studied with regard to hepatocyte proliferation

93 type 2 diabetes mellitus (T2DM), HOMA-IR and FFA.

94 is substantial disagreement between OCT and FFA findings in detecting active disease in patients wit

95 nts (99.9%) had agreement between SD OCT and FFA in detecting CNV activity.

96 of lipids as confirmed by low PV, TBARS and FFA.

97 e found stronger connectivity between V1 and FFA before face reports for low-frequency oscillations.

98 Interestingly, fenofibrate in vivo and FFA in vitro reversed high glucose-induced expression of

99 ing whole-body turnover rates of glucose and FFAs in L-AktFoxo1TKO mice also confirmed that hepatic E

100 Coadministration of insulin and FFAs, however, abolished hepatocyte proliferation and tr

101 NASH, based on a set of short-chain TAGs and FFAs.

102 aditionally, fundus fluorescein angiography (FFA) has been considered the reference standard to detec

103 ing, SS-OCT, fundus fluorescein angiography (FFA), and indocyanine green angiography (ICG) by 2 indep

104 graphy (FP), fundus fluorescein angiography (FFA), and optical coherence tomography (OCT).

105 ompared with fundus fluorescein angiography (FFA), and swept-source optical coherence tomography (SS-

106 d temporal lobe, and the fusiform face area (FFA) and anterior temporal lobe play key roles in the re

107 sensory responses in the fusiform face area (FFA) and parahippocampal place area (PPA), respectively.

108 ts connectivity with the fusiform face area (FFA) during eye contact with a speaker predicted the lev

109 ural styles included the fusiform face area (FFA) in addition to several scene-selective regions.

110 tches in monkeys and the fusiform face area (FFA) in humans.

111 the right face-selective fusiform face area (FFA) was closely associated with individual differences

112 ive areas, including the fusiform face area (FFA), occipital face area (OFA), amygdala, fusiform body

113 ssing network comprising fusiform face area (FFA), superior temporal sulcus, amygdala, and intraparie

114 sulcus (pSTS) and to the fusiform face area (FFA), using a searchlight approach to reveal interaction

115 pecific regions like the fusiform face area (FFA).

116 f the category-sensitive fusiform face area (FFA).

117 d our first paper on the fusiform face area (FFA): how we chose the question, developed the methods,

118 adjacent regions (e.g., fusiform face area, FFA) within the temporal visual cortex.

119 oscillations with specialized areas such as FFA and PPA.

120 c strengths using a fluorescent focus assay (FFA).

121 The relation between FFA-pSTS connectivity and the attention to detail abilit

122 -entrant processing loop involving bilateral FFA and LOC.

123 isual cortical network composed of bilateral FFA and bilateral object-selective lateral occipital cor

124 s the channel in a conformation which blunts FFA activation.

125 ll randomized study eyes in HARBOR with both FFA and SD OCT data were analyzed for (1) evidence of CN

126 1-dependent GPR40 signaling relative to both FFAs.

127 erence standard to detect nAMD activity, but FFA is costly and invasive.

128 oss-tabulation of CNV activity identified by FFA and SD OCT by office visit.

129 , and 24, 92% to 100% of cases identified by FFA only were occult CNV lesions.

130 ases (n = 50) had CNV activity identified by FFA only.

131 naling, was able to block changes induced by FFA and was more effective in the presence of FFA.

132 ld be also be reversed pharmacologically, by FFA.

133 ed in pancreatic beta cells and activated by FFAs.

134 ong complex lipid subclasses and the C16-C20 FFAs but directly associated with short complex lipids w

135 hat increased abundance of saturated C16-C20 FFAs coupled with impaired beta-oxidation of FFAs and in

136 uptake, and 3) is needed for normal cardiac FFA uptake over a range of FFA concentrations from low t

137 , and micellar incorporation of carotenoids, FFAs and MAGs.

138 ocytes, this muChopper permitted single-cell FFA uptake rates to be quantified at 3.5 +/- 0.2 x 10(-1

139 le for the analysis of medium and long chain FFAs in beer.

140 Saturated long chain FFAs induced apoptosis and JNK activation in primary rat

141 t into disease pathogenesis and characterise FFA as a genetically predisposed immuno-inflammatory dis

142 y 45%, associated with decreased circulating FFAs and adipokines/cytokines including IGF-1, VEGF, and

143 siderable evidence suggests that circulating FFAs promote beta cell expansion by direct and indirect

144 As enzymatic treatment, Omega-3 concentrate FFA (Omega-3>600mg Omega-3 per g oil) were esterified wi

145 quid-like layer besides crystalline confined FFA form I.

146 analysis of Certified Reference Material CTA-FFA-1 (Fine Fly Ash) and six natural water samples with

147 During one study day, FFA was elevated by infusion of Intralipid plus heparin.

148 acological blockade of Gq activity decreased FFA-induced insulin secretion.

149 diet, weight loss with an ADF diet decreases FFA concentrations through potentially different mechani

150 A novel diphenylalaninamid (FFA) based peptide nanoparticles (PNPs) modified pencil

151 e tested this hypothesis by comparing direct FFA storage in subcutaneous adipose tissue during insuli

152 We measured direct FFA storage in abdominal and femoral subcutaneous fat in

153 and subcortical face-selective areas (i.e., FFA and amygdala) remained intact.

154 e inferior frontal junction, IFJ, and either FFA or PPA, depending on which object was attended.

155 Although obesity is manifested as elevated FFA levels, the degree of EMT was not associated with th

156 irect evidence associating CD36 and elevated FFAs with HCC progression.

157 Defective suppression of endogenous FFA is one common link between impaired potentiation and

158 riven structural modifications to endogenous FFAs, focused on breaking planarity and reducing lipophi

159 tive metabolism in the presence of exogenous FFAs; this increase was not seen in Fabp4/Fabp5 double-k

160 suggest that CD8(+) TRM cells use exogenous FFAs and their oxidative metabolism to persist in tissue

161 FABP5 expression and enhanced extracellular FFA uptake were also demonstrated in human CD8(+) TRM ce

162 ed UCP2 expression, suggesting that the FABP-FFA equilibrium controls UCP2 expression.

163 We conclude that CD36 1) facilitates FFA transport into muscle and adipose tissue in humans w

164 connectivity (i.e., Granger causality) from FFA to V1 predicted not only the category of the upcomin

165 Whether insulin stimulates greater FFA clearance into adipose tissue in vivo is unknown.

166 In the ADF-HF group, FFA concentrations were positively correlated with waist

167 associated with changes in FAT%, TC, HbA1c, FFA and HDL-c.

168 mponents dictating the gel strength are HCs, FFAs and WEs in a descending order of importance.

169 th FALs and a positive correlation with HCs, FFAs and WEs.

170 levated, 2) is not rate limiting for hepatic FFA uptake, and 3) is needed for normal cardiac FFA upta

171 for studying the dynamic changes in hepatic FFA flux in models of liver disease.

172 Throughout the two study days, higher FFA levels were significantly associated with lower (inc

173 However, FFA may still be of value in those with occult lesions t

174 convincing evidence that the alterations in FFA levels occurring during progression to diabetes are

175 ys of shelf life without a notable change in FFA content of rice bran fraction which was obtained fro

176 pproach enabled us to observe the changes in FFA hepatic uptake under different physiological conditi

177 However, detecting dynamic changes in FFA uptake by the liver in live model organisms has prov

178 using this method, we detected a decrease in FFA accumulation in the liver after mice were given inje

179 Despite the postprandial decrease in FFA-driven esterification and oxidation, VLDL-TAG secret

180 associated with a progressive impairment in FFA suppression during OGTT, whereas the rise in mean pl

181 is associated with a progressive increase in FFA and fasting Adipo-IR.

182 er between face vs. vase reports in V1 or in FFA, indicating similar levels of neural excitability.

183 In conclusion, insulin does not increase FFA storage in adipose tissue compared with niacin, whic

184 re subjected to conditions known to increase FFA uptake in the heart (fasting) and BAT [cold exposure

185 sociated with hyperinsulinemia and increased FFA-blood levels, the interplay between insulin and FFA

186 rophage-specific migration because increased FFA induce leptin receptors, whereas higher leptin cause

187 decreased circulating lymphocytes, increased FFA, and induced hypeerglycemia and glucose intolerance.

188 ates a metabolic reprogramming by increasing FFA and glucagon levels.

189 Total and individual FFA and plasma lipid concentrations were measured before

190 consistent with two interrelated influences: FFAd reduces the responsiveness of magnocellular neurons

191 We present a novel near-infrared FFA tracer, AlexaFFA, that is suitable for in vivo quant

192 t FABP4/aP2 directly regulates intracellular FFA levels and indirectly controls macrophage inflammati

193 opinocytosis through a pathway that involves FFA receptors, the Gbeta/Ggamma complex, and RAC1.

194 In receptor-level IR, FFA oxidation drives gluconeogenesis rather than being r

195 In contrast, in postreceptor IR, FFA contributes to both gluconeogenesis and hepatic stea

196 ode were determined for mixtures of isolated FFAs to values in the low 10 pmol range.

197 t instead switched toward utilization of KB, FFA, and BCAA (increased myocardial uptake of these 3 me

198 uel utilization away from glucose toward KB, FFA, and BCAA, thereby improving myocardial energetics,

199 e enzymes implicated in the metabolism of KB/FFA/BCAA).

200 d insulin-mediated suppression of lipolysis, FFA, and EGP.

201 ations in the zonation of proteins mediating FFA uptake or triglyceride release as very low density l

202 ly diminished by inhibition of mitochondrial FFA beta-oxidation in vivo.

203 group, decreases were found in several more FFAs than in the ADF-HF group.

204 , SGLT2 inhibition did not affect myocardial FFA uptake, but channeled myocardial substrate utilizati

205 In this structure, no FFA is observed and the ceramide binding pocket and puta

206 mp recordings in the presence and absence of FFA.

207 crease in incidence the aetiopathogenesis of FFA remains unknown.

208 -1_A286C channels by repeated application of FFA.

209 ces of FFA: the first principal component of FFA shows differential connectivity with occipital and p

210 h work has shown that high concentrations of FFA can be very damaging to beta-cells when used for in

211 ion in vitro, indicating that the effects of FFA are mediated by TXNIP.

212 f TXNIP abrogated the restorative effects of FFA on high glucose-impaired endothelial cell function i

213 However, the fate of FFA diverges in these populations.

214 To enable noninvasive real-time imaging of FFA flux in the liver, we generated transgenic mice with

215 High temporospatial imaging of FFA in its native environment, an organic solvent, sugge

216 n, WD-feeding results in increased levels of FFA and microbiota that, even in absence of hyperglycaem

217 This panel of FFA ratios could be used for identification and early in

218 m male mice when cultured in the presence of FFA to mimic hyperlipidemia of obesity.

219 FA and was more effective in the presence of FFA.

220 at is suitable for in vivo quantification of FFA metabolism and can be applied in the context of a lo

221 or normal cardiac FFA uptake over a range of FFA concentrations from low to slightly elevated.

222 addition, we observed diurnal regulation of FFA hepatic uptake in living mice.

223 Replacement of FFA by OCT can be justified if there is a substantial ag

224 with different representational subspaces of FFA: the first principal component of FFA shows differen

225 In myotubes, UT attenuated the ability of FFAs to induce insulin resistance and PP2A hyperactivity

226 gels increased with the increasing amount of FFAs and HCs and the decreasing amount of WEs and FALs.

227 tion, which led to a higher concentration of FFAs in the mixed micelles.

228 was regulated by insulin-mediated control of FFAs.

229 n analytical method for the determination of FFAs in beer.

230 The extraction of FFAs in beer was achieved via Liquid-Liquid Cartridge Ex

231 ydrolysis of triacylglycerides, formation of FFAs and MAGs, and micellar incorporation of carotenoids

232 fundamental and physiological importance of FFAs, an oversupply can trigger lipotoxicity with impair

233 was a positive correlation between level of FFAs and level of MIR122 in plasma samples from 6 health

234 C to remove glycerol completely and most of FFAs; and the second distillation at optimized TE 155 de

235 FFAs coupled with impaired beta-oxidation of FFAs and inverse partitioning into complex lipids may be

236 proliferative insulin effect in presence of FFAs and prevented EGFR/CD95 association, CD95 tyrosine

237 Likewise, in presence of FFAs insulin increased apoptosis in hepatocytes from wil

238 dentification and relative quantification of FFAs in biological samples of different origins.

239 ided a 2-fold advantage: i) the reduction of FFAs content resulting into an upgrading of the "exhaust

240 The release of FFAs and MAGs from TAGs proceeded faster than their inco

241 Conversely, impaired release of FFAs and other lipid mediators can also disrupt key cell

242 of rapeseed oil, especially of rapeseed oil FFAs, remarkably increased the amounts of volatile produ

243 oid spaces); (2) evidence of CNV activity on FFA identified by the presence of leakage, and (3) cross

244 Macular ischemia on FFA correlated with ischemia in the SCP layer only.

245 , as this type of lesion may show leakage on FFA.

246 cts murine and human adipocytes from HFD- or FFA-elicited cell death through NF-kappaB-dependent upre

247 d cardiomyocytes challenged with TNFalpha or FFA, we demonstrate that 2-AG improves insulin sensitivi

248 nd release and lipid synthesis (particularly FFA, triglycerides, and cholesterol), whereas glycogen p

249 -months of DAPA therapy, HbA1c, FBG, 2h-PBG, FFA, TG, blood pressure, BMI, WHR, body weight, FAT%, FI

250 Plasma FFA and LPS levels were assessed, in addition to colonic

251 high (HF) or low (LF) in fat affects plasma FFA profiles in the context of weight loss, and changes

252 pimox 1) markedly reduced the fasting plasma FFA concentration and enhanced suppression of plasma FFA

253 al metabolites and concomitant higher plasma FFA.

254 ongly correlated with the decrease in plasma FFA and increase in insulin-mediated glucose disposal (b

255 Reduction in plasma FFA in obese NGT and T2DM individuals improves mitochond

256 entration and enhanced suppression of plasma FFA during oral glucose tolerance tests and insulin clam

257 Preventing the fall in plasma FFAs during insulin infusion either by administering int

258 oreover, mice with elevated levels of plasma FFAs as the result of a high-fat diet were more suscepti

259 Inhibiting the fall of plasma FFAs in these mice prevented the suppression of EGP duri

260 ctively analyzed 24 [product FFA]/[precursor FFA] ratios in fasting sera and clinical data from 481 i

261 als, we retrospectively analyzed 24 [product FFA]/[precursor FFA] ratios in fasting sera and clinical

262 the SCFA receptor free fatty acid receptor (FFA)3, one of the free fatty acid receptor family member

263 an SCFA receptor, free fatty acid receptor (FFA)3, to the enteric nervous system is unknown.

264 rved by neural computations within the right FFA as well as a re-entrant processing loop involving bi

265 ) coefficients for the analysis of saturated FFAs were found to be generally close to 0.98 over about

266 insulinemia or after oral niacin to suppress FFA compared with 11 saline control experiments.

267 e spherical nanostructure of the synthesized FFA based PNPs while attenuated total reflectance-fourie

268 It is concluded that FFAs can shift insulin-induced hepatocyte proliferation

269 and adipose tissues from mice, we found that FFAs increase hepatic expression and secretion of MIR122

270 The FFA and ICG in 7 and 11 patients, respectively, showed s

271 The FFA and ICG were screened for any vascular abnormalities

272 The FFA family of receptors is a recently deorphanized set o

273 e methods, and followed the data to find the FFA and subsequently many other functionally specialized

274 nd adipose tissues of C57BL/6 mice given the FFA-inducer CL316243.

275 on: with increasing neural activation in the FFA, direct-gaze faces entered awareness more readily th

276 Based on the FFA, ICG, and SS-OCT imaging, there were 3 patients with

277 We conclude that the FFA is involved in fine-grained neural encoding of scene

278 of error patterns further revealed that the FFA participated to a much larger extent in the neural e

279 pective is to raise doubts about whether the FFA levels found in real-life situations are ever high e

280 quid-Liquid Cartridge Extraction (LLCE), the FFAs extract was purified by Solid Phase Extraction (SPE

281 edominantly molecular [M + K](+) ions of the FFAs, whereas other alkali metal adducts can be generate

282 The results showed that the % FFA was reduced by 44.3, PV by 50.2, and FOS reading by

283 um ischemic insult following RVO compared to FFA, hence represents a more efficient grader for ischem

284 ctions carrying face information from EVC to FFA and EVC to pSTS.

285 from EVC to the occipital face area, EVC to FFA, and EVC to posterior superior temporal sulcus (pSTS

286 ion presented a higher conversion of MAGs to FFAs during digestion, which led to a higher concentrati

287 The response to FFAs may function in the development of nephrotic syndro

288 Total FFA concentrations also decreased (P < 0.001).

289 The levels of total FFA and total OA were higher in GST-cheese in comparison

290 Traditionally, FFA was frequently performed in clinical practice, but i

291 difference in postprandial triacylglycerol, FFA, insulin, glucose, glucagon, or GIP related to prote

292 analyses of mixtures containing unsaturated FFAs are also possible but require more effort on the ca

293 -beta signaling pathways were activated upon FFA treatment, potentially acting as upstream activators

294 Using FFA as the reference standard, the sensitivity and speci

295 en magnocellular responses are mitigated via FFAd, human form perception is transiently sharpened bec

296 the disruption of the filtration barrier via FFAs bound to albumin and respond by enhancing fluid-pha

297 tection rate was 50% clinically: 52.94% with FFA, 82.35% with ICG, and 86.36% with SS-OCT.

298 rve genome-wide significant association with FFA at four genomic loci: 2p22.2, 6p21.1, 8q24.22 and 15

299 tained migratory capacity when cultured with FFA, whereas female macrophages failed to migrate.

300 atment of human liver cancer cell lines with FFAs exacerbated the EMT phenotype, whereas chemical inh