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1 e growth factor, fibroblast growth factor-5 (FGF-5).
2 s overexpressing fibroblast growth factor-5 (FGF-5).
3 nized nonmutated fibroblast growth factor-5 (FGF-5).
4 FGF receptor, the preferred FGF receptor for FGF-5.
5                                   Exogeneous FGF-5 (0.37 nM) increased the growth of COLO-357 cells b
6     Neither alpha(2)M nor alpha(2)M* bind to FGF-5, -7, -9, or -10.
7 eficient adenoviral construct overexpressing FGF-5 (AdvFGF-5) to improve flow and function in swine w
8 ults demonstrate that N-CAM, tenascin-C, and FGF-5 are dispensable for major aspects of synaptic deve
9 thelial cells exhibit expression in vitro of FGF-5 at the messenger RNA and protein levels.
10                                Expression of FGF-5 by BCME cells was studied by a combination of Nort
11         Northern blot analysis with a 306 bp FGF-5 cDNA revealed the presence of 4.0 kb and 1.6 kb FG
12 ransgenic mice was substantially reversed in FGF-5-deficient mice.
13          Because nonglycosylated recombinant FGF-5 does not appear to be a mitogen in BCME cells in v
14                                              FGF-5 expression by quantitative real-time reverse trans
15 ific analyses revealed lower group levels of FGF-5, FGF-19 and SPOCK1 in multiple system atrophy comp
16             Differentiated myofibers express FGF-5, FGF-7, and reduced levels of FGF-6 mRNA.
17 human pancreatic cancer was investigated, as FGF-5 has a classical signal sequence for secretion not
18                    In this study the role of FGF-5 in human pancreatic cancer was investigated, as FG
19  and in situ hybridization demonstrated that FGF-5 localized in the cancer cells, stromal fibroblast
20              These observations suggest that FGF-5 may participate in autocrine and paracrine pathway
21 A revealed the presence of 4.0 kb and 1.6 kb FGF-5 mRNA transcripts in both normal and cancerous panc
22 ic analysis indicated that 4.0 kb and 1.6 kb FGF-5 mRNA transcripts levels were increased 2.4- and 2.
23                                              FGF-5 mRNA was also detected in COLO-357 human pancreati
24  the mitogenic activity of human recombinant FGF-5 on a variety of cell types was evaluated, using a
25 ygous mutant mice lacking N-CAM, tenascin-C, FGF-5, or both N-CAM and tenascin-C.
26  class I molecules presenting a nine-residue FGF-5 peptide generated by protein splicing.
27                        Furthermore, secreted FGF-5 protein was present in conditioned medium of COLO-
28 cinomas and little normal tissue expression, FGF-5 represents an immunotherapy target with potential
29 expressing human fibroblast growth factor-5 (FGF-5) resulted in messenger RNA and protein expression
30 rivascular and endothelial cell staining for FGF-5 seen previously in choroidal neovascular membranes
31 ected against the aminoterminus of the human FGF-5 sequence was used in Western blot analyses to iden
32 ded evidence for the expression of two major FGF-5 transcripts at 4 kb and 3 kb and two minor transcr
33 y Northern blot analysis for the presence of FGF-5 transcripts, using a 1-kb human complemetary DNA.
34 ular proliferation assays, human recombinant FGF-5 was not mitogenic in BCME cells but exhibited an a
35 time reverse transcription PCR revealed that FGF-5 was overexpressed in the majority of renal cell ca
36 xpression of the differentiation marker gene FGF-5 whereas the expression of the stem cell marker gen
37 RS cells produce fibroblast growth factor-5 (FGF-5), which acts in a paracrine fashion to terminate p
38  Studies with fully glycosylated recombinant FGF-5 will be required, however, to assess the biologic